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Letteratura scientifica selezionata sul tema "Periodontitis"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 29 luglio 2024

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Articoli di riviste sul tema "Periodontitis"

Lee, Young-Soo, e Hye-Sun Shin. "Association between Diet Quality and Periodontitis in Korean Population". Korean Society of Oral Health Science 10, n. 3 (30 settembre 2022): 32–42. http://dx.doi.org/10.33615/jkohs.2022.10.3.32.

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Objectives: The aim of this study was to examine the association between the diet quality and periodontiti for Korean population. Methods: The total number of participants in the KNHANES was 7,943. Periodontitis was defined as Community Periodontal Index. Diet quality was evaluated using the Korean Healthy Eating Index (KHEI) following adequacy, moderation, and balance components. Results: The lowest KHEI total score was associated with periodontitis and severe periodontitis. Especially, lower fruit intake was associated with periodontitis, while lower have breakfast, lower total fruits, meat fish, eggs, and beans intake, and lower carbohydrate and fat energy balance were associated with severe periodontitis. Conclusions: Diet quality had a more significant effect on severe periodontitis than on periodontitis. It is necessary to pay attention to diet quality to better oral health.

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Rams, Thomas E., Charles E. Hawley, Eugene J. Whitaker, John E. Degener e Arie J. van Winkelhoff. "Centipeda periodontii in human periodontitis". Odontology 103, n. 3 (19 luglio 2014): 286–91. http://dx.doi.org/10.1007/s10266-014-0166-1.

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Pischon, N., N. Heng, J. P. Bernimoulin, B. M. Kleber, S. N. Willich e T. Pischon. "Obesity, Inflammation, and Periodontal Disease". Journal of Dental Research 86, n. 5 (maggio 2007): 400–409. http://dx.doi.org/10.1177/154405910708600503.

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The prevalence of obesity has increased substantially over the past decades in most industrialized countries. Obesity is a systemic disease that predisposes to a variety of co-morbidities and complications that affect overall health. Cross-sectional studies suggest that obesity is also associated with oral diseases, particularly periodontal disease, and prospective studies suggest that periodontitis may be related to cardiovascular disease. The possible causal relationship between obesity and periodontitis and potential underlying biological mechanisms remain to be established; however, the adipose tissue actively secretes a variety of cytokines and hormones that are involved in inflammatory processes, pointing toward similar pathways involved in the pathophysiology of obesity, periodontitis, and related inflammatory diseases. We provide an overview of the definition and assessment of obesity and of related chronic diseases and complications that may be important in the periodontist’s office. Studies that have examined the association between obesity and periodontitis are reviewed, and adipose-tissue-derived hormones and cytokines that are involved in inflammatory processes and their relationship to periodontitis are discussed. Our aim is to raise the periodontist’s awareness when treating obese individuals.

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Paty, Diana Sofia Patrocinio, Nara Santos Araujo, Guilherme Nunes de Carvalho, Pedro Yoshito Noritomi, Jorge Vicente Lopes da Silva, Daniel Takanori Kemmoku, Jean Nunes dos Santos e Patricia Ramos Cury. "Using the Response Surface Methodology for Periodontitis Diagnosis". Journal of Health Sciences 25, n. 3 (25 ottobre 2023): 153–58. http://dx.doi.org/10.17921/2447-8938.2023v25n3p153-158.

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AbstractResponse surface methodology (RSM) consists of mathematical and statistical techniques to develop models which help to understand the influence of various factors on a dependent variable of interest. The feasibility of RSM use to detect cases of periodontitis and its correlated factors has not yet been evaluated. This study developed mathematical models for periodontitis diagnosis independent of periodontal probing using the RSM. Demographic, socioeconomic, behavioral, systemic, local factors, and periodontitis were assessed in 176 volunteers. Periodontitis case was defined according to three different definitions: 1) ≥3 sites with clinical attachment level (CAL) ≥4 mm; 2) at least one site with CAL ≥4 mm and bleeding on probing; 3) ≥2 proximal sites with CAL ≥3 mm and ≥2 proximal sites with probing depth (PD) ≥4 mm (not on the same tooth) OR 1 site with PD ≥5 mm. 4th-degree polynomial equations showed high coefficients of determination (R²= 1) and were used to represent the mathematical models of periodontitis cases. According to definition 1, the diagnosis of periodontitis was accurate by including in the model: age, sex, education level, plaque index (PI), number of missing teeth, previous hygiene instructions, and body mass index (BMI). According to definition 2, the diagnosis of periodontitis was accurate by including in the model: age, sex, education level, income, PI, previous oral hygiene instructions, frequency of brushing and type of toothbrush, and use of mouthwash in the model. For an accurate diagnosis of periodontitis according to definition 3, the model included: age, education level, IP, number of missing teeth, previous oral hygiene instruction, BMI, and diabetes. The multifactorial mathematical models were able to diagnosis periodontitis according to different periodontitis case definitions using only variables of easy evaluation and non-invasive. Keywords: Periodontal Diseases. Diagnosis. Theoretical Model. Logistic Models. ResumoA metodologia de superfície de resposta (MSR) consiste em técnicas matemáticas e estatísticas para desenvolver modelos que ajudam a entender a influência de vários fatores em uma variável dependente de interesse. A viabilidade do uso da MSR para detectar casos de periodontite e seus fatores correlacionados ainda não foi avaliada. Este estudo desenvolveu modelos matemáticos para diagnóstico de periodontite independente da sondagem periodontal usando a MSR. Fatores demográficos, socioeconômicos, comportamentais, sistêmicos, locais e periodontite foram avaliados em 176 voluntários. O caso de periodontite foi definido de acordo com três definições diferentes: 1) ≥3 locais com nível de inserção clínica (NIC) ≥4 mm; 2) Um local com NIC ≥4 mm e sangramento à sondagem; 3) ≥2 locais proximais com NIC ≥3 mm e ≥2 locais proximais com profundidade de sondagem (PS) ≥4 mm (não no mesmo dente) OU 1 local com PS ≥5 mm. Equações polinomiais de 4º grau apresentaram altos coeficientes de determinação (R²= 1) e foram utilizadas para representar os modelos matemáticos dos casos de periodontite. De acordo com a definição 1, o diagnóstico de periodontite foi preciso ao incluir no modelo: idade, sexo, escolaridade, índice de placa (IP), número de dentes perdidos, instruções de higiene anteriores e índice de massa corporal (IMC). De acordo com a definição 2, o diagnóstico de periodontite foi preciso ao incluir no modelo: idade, sexo, escolaridade, renda, IP, instruções prévias de higiene bucal, frequência de escovação e tipo de escova dental e uso de enxaguatório bucal no modelo. Para um diagnóstico preciso de periodontite de acordo com a definição 3, o modelo incluiu: idade, escolaridade, IP, número de dentes perdidos, instrução prévia de higiene oral, IMC e diabetes. Os modelos matemáticos multifatoriais foram capazes de diagnosticar a periodontite de acordo com diferentes definições de casos de periodontite usando apenas variáveis de fácil avaliação e não invasivas. Palavras-chave: Doenças Periodontais. Diagnóstico. Modelo Teórico. Modelos Logísticos.

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Yıldırım, Hatice Selin, Nimet Gül Görgülü, Kübra Kundak e Leyla Kuru. "Kronik Periodontitis ile Agresif Periodontitisin Farklılıkları". European Journal of Research in Dentistry 2, n. 3 (30 dicembre 2019): 119–23. http://dx.doi.org/10.35333/erd.2019.100.

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Koffi-Coulibaly, Nadin Therese, Zocko Ange Desire Pockpa e Gnaba Samson Mobio. "Prevalence and severity of periodontitis among adults in Côte d’Ivoire according to the new EFP/AAP periodontal disease classification". Journal of Advanced Periodontology & Implant Dentistry 13, n. 2 (28 novembre 2021): 76–83. http://dx.doi.org/10.34172/japid.2021.018.

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Background. To describe the prevalence and severity of periodontitis in patients attending the Periodontics Service of the Cocody University Hospital at Abidjan.Methods. This retrospective observational study reviewed records of patients aged 20-80 years who attended the Periodontics Service between January 2014 and December 2018. Periodontitis was diagnosed, according to the 2018 EFP/AAP new classification of Periodontal and Peri-Implant Diseases and Conditions. Chi-square test, 1-factor Anova test, and logistic regression were performed for analysis.Results. A total number of 596 patients were included. The mean age was 44.94 ± 14.34 years and 59.20% of were males. 2 (0.40%) patients were classified as Stage I, 221 (37.08%) as Stage II, and 373 (62.58%) as Stage III/V; the extent of periodontitis was generalized in 39.77% of patients. PD ≥ 6 mm, missing teeth ≥ 5 and mobile teeth were present in 47.15%, 26.35% and 25.50% of the sample, respectively.Severity of periodontitis were associated with age (p < 0.001), socio-economic status (p=0.001), diabetes (p < 0.001), missing teeth (p < 0.001) and smoking (p=0.009). Age (OR= 1.59, 95% CI: 1.11-2.26) and missing teeth (OR= 2.31, 95% CI: 1.08-4.89) were identified as independent risks indicators.Conclusion. The prevalence and severity of periodontitis were high. Risks indicators identified may allow early detection and management of groups at high risk in Côte d’Ivoire.

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Laine, M. L., S. A. Morré, L. S. Murillo, A. J. van Winkelhoff e A. S. Peña. "CD14 and TLR4 Gene Polymorphisms in Adult Periodontitis". Journal of Dental Research 84, n. 11 (novembre 2005): 1042–46. http://dx.doi.org/10.1177/154405910508401114.

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Bacterial deposits, smoking, and host genetic factors play a major role in an individual’s predisposition to periodontitis. Bacterial components are recognized by CD14 and toll-like receptor 4 (TLR4), resulting in a NF-κB-based inflammatory response. We hypothesized that functional CD14 and TLR4 polymorphisms contribute to periodontitis susceptibility. We aimed to investigate the occurrence of CD14-260C>T, TLR4 299Asp>Gly, and 399Thr>Ile gene polymorphisms in adult periodontititis. DNA was collected from 100 patients with severe periodontitis and from 99 periodontally healthy controls. The gene polymorphisms were determined by the PCR technique. The presence of the periodontal pathogens Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, and whether the subjects smoked, was included in the analyses. The CD14-260T/T genotype was found in 34.0% of periodontitis patients and in 20.2% of controls. Logistic regression analysis adjusted for gender, age, smoking, and prevalence of P. gingivalis and A. actinomycetemcomitans showed an association between the CD14-260T/T genotype and periodontitis (P = 0.004, OR 3.0, 95% CI 1.4–6.9). We conclude that the CD14-260T/T genotype contributes to the susceptibility to severe periodontitis in Dutch Caucasians.

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Herawati, Dahlia. "Terapi Kombinasi Root Debridement dan Antibiotik terhadap Periodontitis Agresif". Majalah Kedokteran Gigi Indonesia 19, n. 1 (20 ottobre 2016): 200. http://dx.doi.org/10.22146/majkedgiind.15426.

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Latar Belakang. Kerusakan periodontal yang signifikan secara klinis selama dewasa atau awal masa dewasa dikenal sebagai periodontitis agresif. Perawatan standar scaling dan root planing sering kurang memuaskan hasilnya sehingga perlu mempelajari periodontitis agresif secara tuntas dan terapi yang harus diberikan sehingga perawatan bisa memberikan hasil yang optimal. Tujuan. Untuk mengupas tentang periodontitis agresif agar bisa menegakkan diagnosis, serta mendapatkan hasil yang optimal dalam perawatannya. Ringkasan Pembahasan. Gigi goyah disebabkan oleh sedikit atau rapuhnya tulang alveoler pendukung gigi sehingga gigi tidak bisa menjalankan fungsinya. Periodontitis agresif menyerang seseorang, diketahui oleh dokter gigi sering tidak dari awal, akan tetapi setelah penyakit tersebut berlanjut. Skrening melalui foto Rontgen pada penderita periodontitis usia awal dewasa berguna untuk mengetahui secara dini periodontitis agresif. Pada perawatan regeneratif dengan mengganti tulang alveoler yang hilang, terlebih dahulu menghentikan aktivitas periodontitis agresif, yaitu dengan memberikan antibiotik dikombinasi dengan root debridement baik secara bedah maupun non bedah. Kesimpulan. 1. Mengenali dan merawat periodontitis agresif secara dini dapat mencegah kerusakan jaringan periodontal yang berat. 2. Perawatan periodontitis agresif terutama mengeliminir bakteri dengan kombinasi tindakan mekanis root debridement dan pemberian antibiotik yang tepat dalam jagka waktu yang cukup secara konsisten. 3. Pemberian antibiotik sebaiknya berdasarkan tes laboratorium bakteri resiten. Background. Periodontal destruction is clinically significant during adulthood or early adulthood is known as aggressive periodontitis. Nursing standard scaling and root planing is often less satisfactory result, so need to study of periodontits aggressive thoroughly and therapy should be given so that treatments can provide result that optimal. The Purpose. To investigated the aggresive periodontitis in order to establish the diagnosis, and obtain optimal results in treatment. Summary of Discussion. Wobbly tooth caused by a slightly or bone fragility alveoler supporting the teeth so the teeth can not perform its function. Aggressive periodontitis someone attack, is is known by dentist often not from the beginning, but after the disease continues. Screening through X-ray in periodontitis someone attack, it is known by dentists often not from the beginning, but after the disease continues. Screening through X-ray in periodontitis patients with adult early age is useful to know early aggresive periodontitis. In the regenerative treatment by replacing the lost bone alveoler, first stop the activities of aggresive periodontitis, namely by giving antibiotics combination with root debridement either surgery or non surgical. Conclusion. 1. Recognizing and treating aggressive periodontitis early can prevent severe damage to periodontal tissues. 2. Treatment of aggressive periodontitis, especially action to eliminate the bacteria with a combination of mechanical root debridement and giving appropriate antibiotics within a sufficient period of time is concictently. 3. The given antibiotics should be based on laboratory tests of resistant bacteria.

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Sudhakar, U., JA Sivaranjani e MC Dayan. "Interleukin-1 as a Marker of Periodontitis with Oral Carcinoma – A Biochemical Study". Journal of Oral Health and Community Dentistry 7, n. 3 (2013): 137–39. http://dx.doi.org/10.5005/johcd-7-3-137.

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ABSTRACT AIM This biochemical study aimed at evaluating the level of interleukin-1beta in patients with oral squamous cell carcinoma as a reflection of amount of bone destruction leading to periodontitis in comparison with the normal healthy individuals. MATERIAL & METHOD The subjects were grouped under four major categories like chronic periodontits, chronic periodontitis with oral squamous cell carcinoma and squamous cell carcinoma alone and the control. Each group consisted of 10 subjects each. Unstimulated whole saliva was collected from each subject. IL-1 in saliva was measured by enzyme linked immunosorbent assay. RESULTS In the present study, IL-1 was significantly more in patients with cancer and periodontitis than patients with only cancer. When compared with the normal subjects IL-1 value was significantly more in subjects with carcinoma (0.016). CONCLUSION This study presents the first evidence of association between level of salivary biomarker interleukin 1 in subjects with periodontitis and in oral squamous cell carcinoma.

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Еловикова, Татьяна, Tat'yana Elovicova, Елена Гайсина, Elena Gaysina, Артем Приходкин e Artem Prihodkin. "USE OF ANTIBACTERIAL DRUGS FOR THE TREATMENT OF AGGRESSIVE PERIODONTITIS". Actual problems in dentistry 15, n. 1 (6 maggio 2019): 10–15. http://dx.doi.org/10.18481/2077-7566-2019-15-1-10-15.

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Background. Aggressive forms of periodontitis are a group of periodontal diseases characterized by rapid development, significant bone loss, few clinical manifestations, weak inflammatory and immune response, a small number of microorganisms, not proportional to the severity of destruction. According to modern ideas about the etiology of aggressive forms of periodontitis, the main reason for their development – the penetration of periodontal tissue Aggregatibacter actinomycetemcomitans. Therefore, aggressive forms of periodontitis may require adjunct antibacterial therapy. Objective ― to determine and analyze the most effective methods of antibiotic therapy in patients with aggressive forms of periodontitis. Methods. A literary review of the studies of Russian and foreign scientists on the development of approaches to improving the effectiveness of antibiotic therapy of aggressive forms of periodontitis in patients, using scientific search library databases PubMed, Medline, Cochrane, Elibrary. The main selection of materials was carried out by keywords. 37 publications were found. Results. The review describes the main methods of modern antibiotic therapy of aggressive forms of periodontitis, the main advantages of antibacterial drugs. Conclusion. In modern periodontics, there is a significant interest in the problem of antibiotic therapy of aggressive forms of periodontitis; various modes of antibiotics are studied. Most studies of this pathology are devoted to the use of moxifloxacin, azithromycin and a combination of amoxicillin and metronidazole. Additional use of antibiotics leads to better results in the treatment of aggressive forms of periodontitis.

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Più fonti

Tesi sul tema "Periodontitis"

Velasco, Fernanda Guerra. "Analise da doença periodontal como indicador de risco para o parto prematuro e recem-nascidos de baixo peso". [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290389.

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Orientador: Antonio Wilson Sallum
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: Recentemente, vários estudos sugerem que a reação inflamatória causada pela doença periodontal está associada ao aumento do risco de nascimentos prematuros e recém-nascidos de baixo peso. Partindo dessa premissa, o objetivo do presente trabalho foi avaliar a prevalência da doença periodontal em uma amostra de puérperas atendidas no Hospital Universitário da Faculdade de Medicina de Jundiaí, bem como correlacionar sua ocorrência com resultados perinatais adversos descritos anteriormente. Para tanto, a amostra foi composta de 400 gestantes que realizaram o parto no hospital acima referida. Os dados coletados, até 48 horas pós-parto foram: anamnese, informações relativas ao parto e ao recém-nascido e exame clínico periodontal. Os parâmetros clínicos periodontais analisados foram: índice de placa, índice de sangramento à sondagem, profundidade de sondagem e nível de inserção clínica. Após a coleta, a amostra foi dividida em 2 grupos: sem doença periodontal (saudável, com gengivite ou com periodontite leve - P1) ou com doença periodontal (periodontite moderada - P2 ou periodontite severa - P3). Os dados mostraram que a prevalência da doença periodontal de moderada a severa (P2-P3) na população estudada foi de 38,7%, e a prevalência da doença periodontal de leve a severa (P1-P3) foi de 77,7%. Das 400 mulheres estudadas, 69 (17,2%) apresentaram parto prematuro (< 37 semanas) e 45 (11,2%) apresentaram recém-nascidos de baixo peso. Dentre os prematuros, 58% apresentaram mães com doença periodontal e, dentre os recém-nascidos de baixo peso, 60% apresentaram mães com a mesma condição. Estatisticamente houve uma associação entre doença periodontal e prematuridade (p = 0,0003) gerando uma estimativa de risco de 2,59 (IC 1,53 a 4,4), e entre doença periodontal e recém-nascidos de baixo peso (p = 0,0019) com estimativa de risco de 2,66 (IC 1,41 a 5,02). Dentre os limites do presente estudo pode-se concluir que a doença periodontal foi um indicador de risco tanto para a prematuridade quanto para o baixo peso ao nascer
Abstract: Recently, several studies suggest that inflammation caused by periodontal disease is associated with increased risk of premature births and infants of low birth weight. Thus the aim of this study was to evaluate the prevalence of periodontal disease in a sample of postpartum women at the Hospital Universitário da Faculdade de Medicina de Jundiaí and correlate their occurrence with adverse perinatal outcomes described above. Therefore, the sample consisted of 400 women who delivered at the hospital above. The data collected up to 48 hours postpartum were: history, information on delivery and newborn and periodontal clinical examination. The clinical periodontal parameters evaluated were: plaque index, bleeding on probing index, probing depth and clinical attachment level. After data collection, the sample was divided into 2 groups: no periodontal disease (healthy or with gingivitis or mild periodontitis - P1) or periodontal disease (moderate periodontitis - P2 or severe periodontitis - P3). The data showed that the prevalence of periodontal disease, moderate to severe (P2-P3) in the study population was 38.7%, and prevalence of periodontal disease from mild to severe (P1-P3) was 77,7%. Out of the 400 women studied, 69 (17.2%) had preterm delivery (<37 weeks) and 45 (11.2%) had infants of low birth weight. Among the premature, 58% had mothers with periodontal disease and among newborns of low birth weight, 60% had mothers with the same condition. There was a statistically significant association between periodontal disease and prematurity (p = 0.0003) generating a risk estimate of 2.59 (IC 1.53 to 4.4), and between periodontal disease and infants of low birth weight (p = 0.0019) with an estimated risk of 2.66 (IC 1.41 to 5.02). Within the limits of this study, it can be concluded that periodontal disease was a predictor for both preterm delivery and low birth weight
Mestrado
Periodontia
Mestre em Clínica Odontológica

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Nadeem, Muhammad. "The association between periodontitis and end-stage renal disease". Thesis, University of the Western Cape, 2006. http://etd.uwc.ac.za/index.php?module=etd&action=viewtitle&id=gen8Srv25Nme4_6748_1194349692.

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Patients who are in end-stage renal disease (ESRD) experience a significantly increased rate of atherosclerotic complications. Inflammation plays a central role in the pathogenesis of these complications. The major acute phase protein, C-reactive protein (CRP) has been found to predict all-cause and cardiovascular mortality in ESRD patients. Many patients in ESRD experience elevated CRP levels without an overt infection. Periodontal diseases in the general population have been associated with both an increased prevalence of atherosclerotic complications and an elevation in serum CRP values. The aim of this present study was to investigate whether periodontal disease is associated with increased systemic inflammation reflected by CRP values, in patients with ESRD on maintenance haemodialysis (HD) or perioneal dialysis (PD).

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Semenoff, Segundo Alex [UNESP]. "Efeito do estresse crônico na modulação da periodontite induzida por ligadura em ratos". Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/104711.

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O objetivo do estudo foi comparar o efeito modular do estresse crônico variável e do estresse crônico físico sobre a periodontite induzida por ligadura. Utilizaram-se 48 ratos Wister adultas, divididas aleatoriamente nos grupos: estresse físico GEF (n=2), estresse variável GEV (n=2), controle positivo GCP (n=2) e controle negativo GCN (n=2). Iniciou-se ensaio de estresse físico associado a frio (contenção) e estresse variável (exposição à luz piscante, isolamento, exame da cavidade bucal, ambiente congestionado, odor de sangue e barulho). Decorridos dez dias de estresse, submeteram-se os grupos GEF, GEV e GCP à anestesia, acondicionando fio de seda em volta do segundo molar superior direito, servindo o lado esquerdo com controle. Após processamento laboratorial, realizaram-se a digitalização e a mensuração histométrica das imagens, usando-se dois parâmetros: perda de inserção histológica e perda óssea. No lado com indução de doença, observaram-se maior perda de inserção e perda óssea para GEF, comparado aos demais grupos. O GEV não apresentou diferenças estatísticas, comparado ao grupo GCP. O lado, sem indução de doença, demonstrou ausência de diferença estatística. Em relação ao peso do timo, o GEV teve um aumento e diferenciou-se do GCP e GCN. O timo do GEF também aumentou, diferenciando-se do GCP. O peso das adrenais diminuiu no GEF, diferenciando-se do GCP e GCN. Com relação ao peso do fígado as diferenças estatísticas foram notadas no GEF, se comparado ao GCP e ao GCN. Também se observou diferença estatística entre GEV e GCN. Concluindo, o estresse físico modulou o padrão de resposta da periodontite induzida.
Stress is an intrinsic factor in modern society, though its relation with periodontal disease is contraversial. In the light of this problem, the modulatory effect of variable chronic stress and chronic physical stress on ligature-induced periodontitis was compared. Forty eight adult Wister rats were used, randomly divided into four groups: physical stress (PSG n=2), variable stress (VSG n=2), control positive (PC n=2), and control negative (NC n=2). Experiments for physical stress, immobilization associated with cold, and variable stress, including flickering light exposure, isolation, buccal cavity exam, overcrowded environment, blood odor and noise, were realized. After ten days of stress, the PSG, VSG and PC groups were anesthetized and a suture thread was placed around the upper right second molar, with the left side serving as control. The experimental results revealed a greater loss of insertion and bone destruction for the PSG compared to the remaining groups. The VSG in relation to the PC, though with no statistical difference. On the left side with no ligature-induced disease, no statistical differences were observed between the groups. The thymus was found to be enlarged in the VSG, differentiating this group from the PC and NC. The thymus in the PSG was different from the PC. The adrenals were diminished in the PSG, differentiating this group from the PC and NC. Statistical differences were found in the liver between the PSG and the PC and NC, with differences also observed between the VSG and NC. In conclusion, stress models physical modulated the standard response to ligature-induced periodontitis in rats.

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Altabtbaei, Khaled. "Comparative Analysis of Aggressive Periodontitis". The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1568628251123148.

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Conill, Jorgelina. "Periodontitis agresiva localizada". Trabajo final de especialización, Universidad Nacional de Cuyo. Facultad de Odontología, 2008. http://bdigital.uncu.edu.ar/9012.

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Las periodontitis agresivas comprenden un grupo de formas diferentes, y han sido definidas basándose en características primarias y secundarias. Entre las primeras, se cita una historia clínica sin particularidades, pérdida de inserción y destrucción ósea rápidas y una agregación familiar de los casos. Con respecto a las características secundarias están presentes, aunque no en todos los casos, como una cantidad de depósitos microbianos incompatible con la severidad de la destrucción de los tejidos periodontales.
Fil: Conill, Jorgelina. Universidad Nacional de Cuyo. Facultad de Odontología.

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Codes, Alejandro. "Periodontitis apical persistente". Trabajo final de especialización, Universidad Nacional de Cuyo. Facultad de Odontología, 2020. http://bdigital.uncu.edu.ar/15166.

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Reintervenir un diente ya tratado endodónticamente es complejo y requiere un arduo entrenamiento. Debido a las distintas dificultades que puede surgir durante el tratamiento, la tasa de éxito baja considerablemente sobre un diente tratado en primera instancia. Sumado a esto requiere un esfuerzo adicional aquellos elementos con pernos cementados sobre alguno de los conductos. Debido a esta problemática uno de los objetivos de esta tesina es evaluar la reintervención endodóntica en comparación con la cirugía periapical cuando se presenta una periodontitis periapical persistente. Informar sobre la microbiota que produce esta patología. Además, describir y comparar los distintos métodos de extracción de pernos alojados en el conducto. Muchos dentistas son reacios para intentar la eliminación de estos por miedo a debilitar, perforar o fracturar la estructura radicular restante. Mediante un caso clínico y con ayuda de la evidencia buscaremos herramientas, avances en la tecnología y la comparación entre estas, para elevar el índice de éxito y facilitar el tratamiento. El tiempo necesario para la extracción de un poste depende de la longitud del poste, su adaptación en el conducto radicular y el tipo de cemento de fijación. Es poco probable que ocurran fracturas de raíz con una buena selección de casos.
Fil: Codes, Alejandro. Universidad Nacional de Cuyo. Facultad de Odontología.

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Jofré, Dante. "Periodontitis apical crónica". Trabajo final de especialización, Universidad Nacional de Cuyo. Facultad de Odontología, 2020. http://bdigital.uncu.edu.ar/15351.

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La periodontitis apical es la inflamación y destrucción de los tejidos perirradiculares causada por agentes etiológicos de origen endodóntico. Generalmente es una secuela de la infección endodóntica. Inicialmente, la pulpa dental se infecta por una microflora oral autógena, y el entorno endodóntico proporciona un hábitat selectivo para el establecimiento de una flora mixta, predominantemente anaeróbica. Los invasores microbianos o sus productos pueden migrar hacia el periápice. En respuesta, el huésped monta una serie de defensas que consisten en varias clases de células, mensajeros intercelulares, anticuerpos y moléculas efectoras. Los factores microbianos y las fuerzas de defensa del huésped se encuentran, chocan y destruyen gran parte del tejido periapical, lo que resulta en la formación de una patosis periapical. El tratamiento de la periodontitis apical consiste en eliminar la infección del conducto radicular, esto se logra mediante los procedimientos de limpieza y conformación, y además prevenir la reinfección a través de un correcto sellado del conducto radicular.
Fil: Jofré, Dante. Universidad Nacional de Cuyo. Facultad de Odontología.

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Ribeiro, Ingrid Webb Josephson. "Perfil de expressão gênica de sítios ativos na progressão das doenças periodontais agressiva e crônica". Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/58/58132/tde-06122010-163420/.

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Introdução: A periodontite é uma doença inflamatória crônica, de alta prevalência na população, que causa perda dentária. A progressão da doença ocorre por meio de surtos, com períodos de remissão e exacerbação. A análise da expressão gênica de lesões ativas na progressão das doenças periodontais pode demonstrar diferenças na resposta do hospedeiro e indicar processos relacionados à atividade destrutiva da doença. Metodologia: Foram selecionados 54 pacientes, sendo 18 com Doença Periodontal Agressiva (DPA), 25 com Doença Periodontal Crônica (DPC) e 11 com ausência de Doença Periodontal (Controle). Medidas clínicas de profundidade de sondagem (PS), nível de inserção relativo (NIR), sangramento à sondagem (SS) e índice de placa (IP) foram obtidas em dois tempos: inicial e após tratamento na reavaliação com dois meses. Nos pacientes com Doença Periodontal (DP), os sítios que apresentaram perda de inserção de um ou mais milímetros foram considerados ativos e os demais, não ativos. O perfil de expressão gênica, de sítios ativos e não ativos, foi obtido pela análise de biópsias gengivais com o Real Time PCR Array. Resultados: Após o tratamento periodontal não cirúrgico, houve melhora significante em todos os parâmetros clínicos (PS, SS, IP; p<0,05) exceto para o NIR (p>0,05). Dos 5112 sítios tratados, aproximadamente 4% apresentam perda de inserção mesmo após o tratamento com ou sem uso de antibiótico. As doenças DPA e DPC demonstraram diferentes perfis de expressão gênica, com sítios ativos apresentando um padrão significantemente up-regulated em relação aos sítios não ativos. Conclusão: A DPA e a DPC são condições clínicas que apresentam diferenças na resposta imune e a atividade destrutiva da lesão periodontal ativa pode ser reconhecida por um padrão inflamatório up-regulated de resposta. Considerando-se as diferenças observadas, abordagens terapêuticas individualizadas e capazes de modular a resposta poderiam potencializar o efeito benéfico da terapia anti-infecciosa e reduzir o número de sítios ativos da DP progressiva.
Aim: Evaluate the gene expression profile under healthy and periodontal disease conditions; identify possible differences in the gene expression profile between chronic and aggressive periodontal diseases; seek evidences of immune regulation differences in active and non active sites during periodontal destruction. Methods: Clinical parameters of probing depth, relative attachment level, bleeding on probing and plaque index were obtained in two stages: initial and two months. The sites that showed attachment loss 1mm were considered active and others non-active. The gene expression profile of active and non-active sites, was obtained by gingival biopsies analysis with Real Time PCR Array. Results: After non-surgical periodontal treatment, significant improvement in all clinical parameters were achieved (P <0.05), except for the relative attachment level (p> 0.05). The disease showed different gene expression profiles with active sites showing a pattern significantly up-regulated compared to non-active sites. Conclusion: Aggressive and Chronic Periodontitis are clinical conditions that showed differences in the immune response profile and that the progressive periodontal lesion can be recognized by an up-regulated inflammatory response. Considering the observed differences, it is possible that individualized therapeutic approaches that modulate the inflammatory response might enhance the efficacy of the anti-infective therapy and reduce active sites number after treatment.

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Dockter, Kathryn M. Williams Karen B. "Relationship between pre-referral periodontal care and periodontal status at referral". Diss., UMK access, 2005.

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Thesis (M.S.)--School of Dentistry. University of Missouri--Kansas City, 2005.
"A thesis in dental hygiene education." Advisor: Karen B. Williams. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed March 12, 2007. Includes bibliographical references (leaves 57-65). Online version of the print edition.

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Macedo, Paula Delello [UNESP]. "Avaliação dos efeitos da terapia fotodinâmica com hipericina-glucamina no tratamento da doen ça periodontal induzida em ratos". Universidade Estadual Paulista (UNESP), 2014. http://hdl.handle.net/11449/123893.

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Made available in DSpace on 2015-06-17T19:34:13Z (GMT). No. of bitstreams: 0 Previous issue date: 2014-03-24. Added 1 bitstream(s) on 2015-06-18T12:49:14Z : No. of bitstreams: 1 000822755_20170324.pdf: 161177 bytes, checksum: d3c62575cb1a09c7a474770bbda3327c (MD5) Bitstreams deleted on 2017-03-24T12:04:42Z: 000822755_20170324.pdf,. Added 1 bitstream(s) on 2017-03-24T12:05:37Z : No. of bitstreams: 1 000822755.pdf: 1027762 bytes, checksum: 3419274eed9057fcc4fccda4041dd643 (MD5)
Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)
The Photodynamic Therapy (TFD) has shown satisfactory results when coupled with mechanical treatment of periodontal diseases. The aim of this work was to evaluate whether PDT with the photosensitizer hypericin-glucamine, enabled by amber LED (34.10 J/cm2) and combined with scaling and root planning (RAR), promotes better repair of the periodontal disease induced experimentally in rats. The periodontal diseases was installed by randomly inserting ligatures in the grooves of the superior second molars. The bandages were removed after 15 days and the animals were randomized into 4 experimental groups (n = 5): IPD group (Induced Periodontal Disease); RAR group; TFD group; and RAR+TDF group. The animals were sacrificed after 7, 15 and 30 days of the performed treatment. Digital microtomography and histometric analyses were carried out on their hemimaxillas. Shapiro-Wilk normality test was applied to data. The groups were assessed among each other and parametric data were evaluated using ANOVA test. All tests were conducted with p<0.05. Statistical differences were not observed among the groups. Therefore, we concluded that TFD combined with RAR or isolated obtained similar results to scaling and root planning.
CNPq: 133436/2012-8

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Libri sul tema "Periodontitis"

Santi-Rocca, Julien, a cura di. Periodontitis. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6.

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Dag, Ørstavik, e Pitt Ford T. R, a cura di. Essential endodontology: Prevention and treatment of apical periodontitis. 2^a ed. Oxford, UK: Blackwell Munksgaard, 2008.

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Kvist, Thomas, a cura di. Apical Periodontitis in Root-Filled Teeth. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57250-5.

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Bosy, Anne. Relationship of oral malodour to periodontitis. Ottawa: National Library of Canada, 1993.

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Frisk, Fredrik. Epidemiological aspects on apical periodontitis: Studies based on the prospective population study of women in Göteborg and the population study on oral health in Jönköping, Sweden. Göteborg [etc.]: Department of Endodontoly/Oral Diagnosis, Institute of Odontology, the Sahlgrenska Academy at Göteborg University, 2007.

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Dag, Ørstavik, e Pitt Ford T. R, a cura di. Essential endodontology: Prevention and treatment of apical periodontitis. Oxford, OX: Blackwell Science, 1998.

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Wouters, Francis Roger. Objective methods of recording gingivitis and marginal periodontitis in man: A laser, radiography- and computer-based study of gingival swelling and interproximal alveolar bone levels. Stockholm: Kongl. Carolinska Medico Chirurgiska Institutet, 1988.

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Asikainen, Sirkka. Subgingival microflora in relation to clinical conditions in juvenile periodontitis. Helsinki: The author, 1986.

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Christersson, Lars A. Actinobacillus actinomycetemcomitans and localized juvenile periodontitis: Clinical, microbiologic and histologic studies. Göteborg: Department of Periodontology, Faculty of Odontology, University of Göteborg, 1993.

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Larivee, Jacinthe. Collagenase activity in sulcal fluid of patients with localized juvenile periodontitis. [Toronto]: Faculty of Dentistry, University of Toronto, 1985.

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Capitoli di libri sul tema "Periodontitis"

Gooch, Jan W. "Periodontitis". In Encyclopedic Dictionary of Polymers, 914. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14453.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber et al. "Periodontitis". In Encyclopedia of Molecular Mechanisms of Disease, 1621. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6548.

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Niemiec, Brook A. "Periodontitis". In Veterinary Periodontology, 51–68. West Sussex, UK: John Wiley & Sons, Inc,., 2013. http://dx.doi.org/10.1002/9781118705018.ch5.

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Tatakis, Dimitris. "Periodontitis". In Periodontal Manifestations of Local and Systemic Diseases, 29–31. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-10828-0_4.

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Harding, Alice, Shalini Kanagasingam, Richard Welbury e Sim K. Singhrao. "Periodontitis as a Risk Factor for Alzheimer’s Disease: The Experimental Journey So Far, with Hope of Therapy". In Periodontitis, 241–60. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_13.

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Giannelli, Marco, e Daniele Bani. "Multi-photonic Adjunctive Therapy for the Management of Periodontitis: Recent Advances and New Treatment Approach". In Periodontitis, 341–52. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_18.

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Hansen, Peter Riis, e Palle Holmstrup. "Cardiovascular Diseases and Periodontitis". In Periodontitis, 261–80. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_14.

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Karkowska-Kuleta, Justyna, Dorota Satala, Magdalena Smolarz, Marcin Zawrotniak e Maria Rapala-Kozik. "Fungi—A Component of the Oral Microbiome Involved in Periodontal Diseases". In Periodontitis, 113–38. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_6.

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Blanco-Pintos, Triana, Alba Regueira-Iglesias, Carlos Balsa-Castro e Inmaculada Tomás. "Update on the Role of Cytokines as Oral Biomarkers in the Diagnosis of Periodontitis". In Periodontitis, 283–302. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_15.

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Salhi, Leila, e Michèle Reners. "Update on the Bidirectional Link Between Diabetes and Periodontitis". In Periodontitis, 231–40. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_12.

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Atti di convegni sul tema "Periodontitis"

Fink, M., J. Waldmann e PM Schumm-Draeger. "Diabetes and Periodontitis – can improvement of glycaemia also ameliorate periodontitis?" In Late Breaking Abstracts: – Diabetes Kongress 2017 – 52. Jahrestagung der DDG. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1603543.

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Efanov, O. I. "Laser therapy for periodontitis". In Low-Level Laser Therapy, a cura di Tatiana I. Solovieva. SPIE, 2001. http://dx.doi.org/10.1117/12.425532.

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Sodikova, Shoira. "PERIODONTITIS IN PREGNANT WOMEN". In SCIENTIFIC PRACTICE: MODERN AND CLASSICAL RESEARCH METHODS. European Scientific Platform, 2021. http://dx.doi.org/10.36074/logos-26.02.2021.v3.15.

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Usher, Adam, Sabrina Parmar, Viren Vithlani, Iain Chapple, Elizabeth Sapey e Robert Stockley. "The relationship between COPD and periodontitis". In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa1116.

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Brahem, M., M. Jguirim, M. Khemis, E. Chebil, D. Chaabouni, M. Younes, T. Ben Alaya et al. "AB0849 Association between periodontitis and osteoporosis". In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.5184.

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Yoo, Wook-Sung, Hwapyeong Song e Hyun-Duck Kim. "Collaborative Research on Rapid Periodontitis Test". In 2022 IEEE 46th Annual Computers, Software, and Applications Conference (COMPSAC). IEEE, 2022. http://dx.doi.org/10.1109/compsac54236.2022.00070.

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Иманов, Э. А. "Использование современных препаратов в комплексном лечении и профилактика воспалительных заболеваний пародонта у детей". In Наука России: Цели и задачи. НИЦ "LJournal", 2021. http://dx.doi.org/10.18411/sr-10-06-2021-04.

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In the study process participated schoolсhildren with diagnosis of chronic catarrhal gingivitis (HKG) and chronic periodontitis light form (HPLC). In total 103 patient, 67 patient with chronic catarrhal gingivitis and 36 patient with chro nic periodontitis light form. The laboratory results showed that in the process of conducting treatment and preventive measures during the use of the drug Loroben the functional activity of the local immune factors were significantly restored. If we pay attention to indicators after professional hygiene and local application of a solution of digluconate of chlorhexidine, after treatment with Loroben values significantly increased . Solution of Loroben improves trophic of the periodontal tissues, promotes the elimination of bleeding gums and inflammation, and these promotes positive treatment outcome.

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Léber, Attila, Erzsébet Csányi e Mária Budai-Szűcs. "Lipid-based delivery systems for periodontitis treatment". In I. Symposium of Young Researchers on Pharmaceutical Technology,Biotechnology and Regulatory Science. Szeged: Institute of Pharmaceutical Technology and Regulatory Affairs, University of Szeged, Faculty of Pharmacy, 2019. http://dx.doi.org/10.14232/syrptbrs.2019.op16.

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Lobo Pereira, J. A., Maria Cristina Ferreira e Teresa Oliveira. "Assessing risk factors for periodontitis using regression". In 11TH INTERNATIONAL CONFERENCE OF NUMERICAL ANALYSIS AND APPLIED MATHEMATICS 2013: ICNAAM 2013. AIP, 2013. http://dx.doi.org/10.1063/1.4825903.

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Andersen, Roger C., Nicolas G. Loebel e Dane M. Andersen. "Photodynamic dosimetry in the treatment of periodontitis". In 12th World Congress of the International Photodynamic Association, a cura di David H. Kessel. SPIE, 2009. http://dx.doi.org/10.1117/12.822941.

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Rapporti di organizzazioni sul tema "Periodontitis"

Zhao, Zepeng, Fengyuan Zhang e Yijin Li. The Relationship Between Il-1 RN intron 2 (VNTR) rs2234663 Gene Polymorphism and The Progression of Periodontitis: A systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzo 2023. http://dx.doi.org/10.37766/inplasy2023.3.0100.

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Review question / Objective: The aim of this systematic review and meta-analysis of case-control studies is to find out the association of IL-1 RN intron 2 (VNTR) rs2234663 Gene Polymorphism and the occurrence and progression of periodontitis(including chronic periodontitis, aggressive periodontitis and early-onset periodontitis). Condition being studied: Periodontitis is one of the most common ailments affecting the teeth, leading to the destruction of the supporting and surrounding tooth structure. Periodontitis is originally a disease originating from the gingival tissue which if left untreated results in penetration of inflammation to the deeper tissues, altering the bone homeostasis causing tooth loss. Periodontal disease has a multifactorial origin. The main culprit identified in periodontitis is the bacterial biofilm growing on the tooth surfaces. The deleterious effects of periodontopathogens are not limited to the periodontium, but they also exude their ill effects on the systemic health of the patients. While the host response determines the progression of the disease, genetics, environmental factors, systemic health of the patient, lifestyle habits and various social determinants also play a role. Interleukin-1 receptor antagonist encoded by this gene IL-1RN is a member of the interleukin 1 cytokine family. This protein inhibits the activities of interleukin 1, alpha (IL1A) and interleukin 1, beta (IL1B), and modulates a variety of interleukin 1 related immune and inflammatory responses, particularly in the acute phase of infection and inflammation. We aim to study their association by conducting a meta-analysis.

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Chen, Yujia, e Wen Li. Periodontitis Increase the Risk of Heart Failure. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, novembre 2022. http://dx.doi.org/10.37766/inplasy2022.11.0084.

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Review question / Objective: Can periodontitis increase the risk of heart failure? Condition being studied: Previous studies reported that periodontal disease is associated with higher risk of HF. However, their conclusions conflicted a little bit with each other. In this case, a systematic review may help drawing a more comprehensive conclusion. Therefore, the purpose of the present analysis is to tell if periodontitis is associated with HF as well as the subtypes of HF.

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Asmat Abanto, Angel Steven, Rosita Elena Espejo Carrera, Tammy Margarita Honores Solano, José Antonio Caballero Alvarado, Oscar Martín Del Castillo Huertas e Carlos Alberto Minchón Medina. Periodontitis associated with ischemic cerebrovascular accident: systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, aprile 2024. http://dx.doi.org/10.37766/inplasy2024.4.0053.

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ZARATE, NASHELY NICKOL CHAVEZ, NICOLL SARAÍ RIVERA LEDEZMA, CESAR ALONSO PONCE LOZANO e Martin Andres Chavez Mendez. Clinical effectiveness of the infrared diode laser (810-980 nm) in conjunction with scaling and root planing in comparison with scaling and root planning alone (placebo) in the non-surgical treatment of periodontitis: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzo 2023. http://dx.doi.org/10.37766/inplasy2023.3.0037.

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Review question / Objective: What is the efficacy of the use of diode laser (810-980 nm) in association with scaling and root planing compared to scaling and root planing alone (placebo) in the non-surgical treatment of periodontitis? Condition being studied: Periodontitis is a chronic multifactorial inflammatory disease associated with a dysbiotic biofilm and characterized by progressive destruction of the tooth-supporting apparatus, which can lead to toothloss.

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Suresh, Sneha, Leena Beevi e Anshuman Gautam. Niveles de Zinc en la Diabetes y la Periodontitis Crónica. Buenos Aires: siicsalud.com, settembre 2018. http://dx.doi.org/10.21840/siic/157295.

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Zhu, Junfei, Suhan Zhang, Jing Shi, Ning Ning, Ying Wei e Ye Zhang. Periodontitis is associated with the increased levels of Visfatin: A Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, agosto 2023. http://dx.doi.org/10.37766/inplasy2023.8.0125.

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Liu, Chun Ching, Patrick Schmidlin, Thomas Attin e Omid Fakheran. Gender differences in non-apnoea sleep disorder among periodontitis patients: A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, aprile 2023. http://dx.doi.org/10.37766/inplasy2023.4.0010.

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Wu, Peiyao, Jieyu Zhou e Lei Zhao. Is periodontitis associated with liver cirrhosis? A protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, luglio 2020. http://dx.doi.org/10.37766/inplasy2020.7.0102.

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Garcia, Martin, e Pedro Tinedo. ADJUVANT EFFECT OF PROPOLIS TO PERIODONTAL THERAPY FOR THE TREATMENT OF PERIODONTAL DISEASE: A SYSTEMATIC REVIEW. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzo 2022. http://dx.doi.org/10.37766/inplasy2022.3.0030.

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Review question / Objective: In patients with periodontal disease, what will be the scientific evidence on the adjuvant effect of Propolis to periodontal therapy for the treatment of periodontal disease? Condition being studied: Periodontal Disease or Periodontitis, an inflammatory disease that affects the supporting tissues that surround the tooth, which are currently being studied with natural products that would work as an adjuvant to periodontal therapy and obtain better results. Information sources: Three digital data sources were used, PUBMED, SCOPUS and EMBASE.

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Silva, Felipe Rodolfo Pereira da. Supplementary Materials - Two-Way Relationship Between Helicobacter Pylori Infection and Periodontitis: Results from A Systematic Review and Meta-Analysis. Science Repository OÜ, gennaio 2020. http://dx.doi.org/10.31487/j.cei.2020.01.03.sup.

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