Letteratura scientifica selezionata sul tema "Période périopératoire"
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Articoli di riviste sul tema "Période périopératoire":
Dureuil, Bertrand, Bertrand Dautzenberg e Alain-Charles Masquelet. "Tabagisme en période périopératoire". La Presse Médicale 35, n. 6 (giugno 2006): 1009–15. http://dx.doi.org/10.1016/s0755-4982(06)74740-6.
Mérat (photo), Stéphane, Christophe Cazères e Lan N’Guyen. "Gestion des anticoagulants en période périopératoire". Le Praticien en Anesthésie Réanimation 10, n. 6 (dicembre 2006): 472–77. http://dx.doi.org/10.1016/s1279-7960(06)75650-9.
Challet, Corinne, Nicolas Schaad e Alain Delabays. "Médicaments en période périopératoire, que faire?" Revue Médicale Suisse 3, n. 133 (2007): 2599–604. http://dx.doi.org/10.53738/revmed.2007.3.133.2599.
Janssens, M., e M. Lamy. "Place de l'érythropoïétine recombinante en période périopératoire". Annales Françaises d'Anesthésie et de Réanimation 14 (gennaio 1995): 98–106. http://dx.doi.org/10.1016/s0750-7658(05)81808-8.
Albaladejo, P. "Comment gérer les nouveaux antithrombotiques dans la période périopératoire ?" Journal des Maladies Vasculaires 35, n. 2 (marzo 2010): 79–80. http://dx.doi.org/10.1016/j.jmv.2010.01.015.
Pignot, G., T. Lebret, D. Chekulaev, M. Peyromaure, D. Saighi, T. Flam, D. Amsellem-Ouazana, B. Debre e M. Zerbib. "Cicatrisation et thérapies ciblées : quelles précautions en période périopératoire ?" Progrès en Urologie 21, n. 3 (marzo 2011): 166–72. http://dx.doi.org/10.1016/j.purol.2010.09.023.
Robin, E., E. Haddad e B. Vallet. "Le monoxyde d’azote inhalé en période périopératoire et en réanimation". Annales Françaises d'Anesthésie et de Réanimation 21, n. 7 (luglio 2002): 581–90. http://dx.doi.org/10.1016/s0750-7658(02)00677-9.
Debette-Gratien, Marilyne. "Prise en charge nutritionnelle du greffé hépatique en période périopératoire". Nutrition Clinique et Métabolisme 14 (dicembre 2000): 106s—110s. http://dx.doi.org/10.1016/s0985-0562(00)80089-8.
Lemasle, L., e E. Gayat. "Nouveautés dans la prise en charge cardiovasculaire en période périopératoire". Réanimation 23, n. 2 (marzo 2014): 202–10. http://dx.doi.org/10.1007/s13546-014-0862-4.
Bazin, J. E., A. Attias, H. Baghdadi, A. Baumann, P. Bizouarn, F. Claudot, B. Eon et al. "Conflits en période périopératoire : un enjeu collectif, éthique et professionnel". Annales Françaises d'Anesthésie et de Réanimation 33, n. 5 (maggio 2014): 335–43. http://dx.doi.org/10.1016/j.annfar.2014.04.006.
Tesi sul tema "Période périopératoire":
Saxena, Sarah. "Preventive and therapeutic physio-pathological aspects of peri-operative neurocognitive disorders". Electronic Thesis or Diss., Université de Lille (2022-....), 2022. http://www.theses.fr/2022ULILS034.
Neurocognitive disorders present a major postoperative complication. After many years of speculation about the etiology of this complication, recent studies indicate that an inflammatory cascade, leading to the activation of microglia, may be the cause. In this work, we investigated the mechanisms responsible for postoperative cognitive disorders along two axes, one within the framework of clinical studies, the other by a pre-clinical approach on murine models.Thus, in the clinical study, the influence of lifestyle habits on the prevalence of perioperative neurocognitive disorders as well as known peripheral inflammatory markers (IL-6; HMGB1) known to be associated with perioperative neurocognitive disorders was studied. This study suggests that preoperative sdentarybehaviour is not a risk factor but that the absence of depressive tendencies/ presence of multilingualism would be protective factors. In addition, it reveals that the level of serum IL-6, unlike that of HMGB1, varies according to the type of surgery and the age of the patient. From this study, a formula was defined to predict postoperative cognitive decline based on perioperative blood IL-6 and HMGB1 levels.Several K+ channels (Kca3.1; Kv1.3; Kvir) have been identified on the cell surface of microglia, essential for its activation in pro-inflammatory phenotype. Attenuating microglial activation by blocking these channels may be one approach to preventing the development of neuroinflammation and cognitive decline.In our preclinical study, we showed that pharmacological and genetic inhibition of the Kv1.3 channel reduces postoperative neuroinflammation and cognitive decline. Similarly, we investigated the study of the role of the KCa3.1 channel. Our results suggest that TRAM34 administration caused cognitive decline and neuroinflammation compared to baseline.The KCa3.1 -/- model causes cognitive decline and is associated with more peripheral inflammation. Tibia fracture causes cognitive decline, microglial proliferation and peripheral inflammation. Isoflurane-based anesthesia causes cognitive decline and microglial proliferation
Libri sul tema "Période périopératoire":
1957-, Samama Marc, Société française d'anesthésie et de réanimation e Groupe d'étude sur l'hémostase et la thrombose, a cura di. Agents antiplaquettaires et période périopératoire. Amsterdam: Elsevier, 2002.