Tesi sul tema "Pathogen"
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Panagoda, Gehan Joseph. "Pathogenic features of Candida parapsilosis : an emerging fungal pathogen /". Thesis, Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20377770.
Testo completoHovhannisyan, Hrant 1992. "Comparative transcriptomics of host-pathogen interactions and hybridization in Candida pathogens". Doctoral thesis, Universitat Pompeu Fabra, 2020. http://hdl.handle.net/10803/670316.
Testo completoLas levaduras patógenas Candida representan un problema de salud global. Este grupo de levaduras, comprenden especies filogenéticamente diversas, e incluye patógenos emergidos recientemente. La forma en que las interacciones entre humanos y Candida varían de una especie a otra y qué procesos subyacen a la aparición de nuevos patógenos son poco conocidos. La tesis actual aborda estos problemas utilizando una aproximación de transcriptómica comparativa y bioinformática. Establecimos los patrones globales de las interacciones huésped-patógeno entre el huésped humano y las principales especies de Candida, proporcionando nuevas ideas mecanicistas sobre su interacción. También exploramos los lncRNA de estos patógenos, evaluando sus implicaciones en la infección. Además, diseñamos y validamos un enfoque de enriquecimiento de ARN pan-Candida, abriendo nuevas posibilidades para estudiar las interacciones huésped-patógeno in vivo. Luego, evaluamos el impacto de la hibridación en los transcriptomas de levaduras híbridas, explorando los vínculos entre la hibridación y la aparición de virulencia. En su conjunto, los resultados de esta tesis amplían nuestro conocimiento sobre aspectos relevantes de las interacciones humano-Candida y la evolución de las levaduras.
Gibson, Josie. "In vivo imaging and analysis of host-pathogen interactions of intracellular pathogens". Thesis, University of Sheffield, 2017. http://etheses.whiterose.ac.uk/19047/.
Testo completoYan, Ling. "Phagocyte-pathogen interactions". [Lincoln, Neb. : University of Nebraska-Lincoln], 2004. http://www.unl.edu/libr/Dissertations/2004/YanDis.pdf.
Testo completoFeldmann, Friederike. "Implication of extraintestinal pathogenic Escherichia coli siderophore receptors in host pathogen interaction". kostenfrei, 2008. http://mediatum2.ub.tum.de/doc/649951/649951.pdf.
Testo completoKärkkäinen, Riikka M. "Production of DNA aptamers with specificity for bacterial food pathogens". Thesis, University of Chester, 2012. http://hdl.handle.net/10034/620695.
Testo completoAhmad, Sarah. "Identification of pathogen-specific protein-encoding genes from microbial pathogens based on bioinformatic analysis". Thesis, University of Exeter, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425246.
Testo completoAlmiman, Bandar F. "Molecular genetic and genomic characterization of an emerging mycotoxigenic pathogen Fusarium proliferatum". Thesis, University of Bedfordshire, 2018. http://hdl.handle.net/10547/622835.
Testo completoBerriri, Souha. "Identification of constitutively active forms of Arabidopsis MAP Kinases : brings more evidence on MPK4 function in plant immunity". Thesis, Evry-Val d'Essonne, 2012. http://www.theses.fr/2011EVRY0024.
Testo completoProtein phosphorylations and dephosphorylations are common events occurring duringintracellular signaling processes. Among plant kinases, Mitogen-Activated Protein Kinases (MAPKs) are involved in signaling of many important biological processes, including biotic and abiotic stresses, development and cytoskeleton organization. Despite an abundant literature on MAPKs, the exact roles and direct targets of many Arabidopsis thaliana MAPKs are not clear yet. The activation of kinases using phospho-mimicking mutations of the phosphorylated residues was a successful approach in the case of MAP2Ks, helping to elucidate their functions. This strategy failed in the case of MAPKs since the necessary residues to mutate remain unclear. To bypass this problem, we adapted a screen based on the functional complementation of a MAPK yeast mutant with randomly mutated Arabidopsis MPK6 in order to identify the ones mutants showing constitutive activity. We identified several clones and showed that these constitutively active (CA) of MPK6 candidates are indeed active without phosphorylation by MAP2Ks. Interestingly, mutations of the equivalent residues in other MAPKs triggered constitutive activity as well, indicating that this strategy may be used as a general approach to activate MAPKs and identify their functions. Interaction and phosphorylation assays indicatedthat CA MAPKs retain their substrate and interactor specificity. As proof-of-concept, we generated active versions of MPK4. CA MPK4 expressed under itsown promoter successfully complements mpk4 mutant plants. Characterization of CA MPK4 lines further confirmed the negative role of MPK4 in plant pathogen defense responses and its implication in both PTI (PAMP Triggered Immunity) and ETI (Effector Triggered Immunity). Overall, the work will help to provide direct information on all MAPK targets and should be an important contribution to the overall understanding of signal transduction in plants
Su, Fan. "Modifications physiologiques induites par Burkholderia phytofirmans chez Arabidopsis thaliana. Applications à la protection contre les stress biotique et abiotique". Thesis, Reims, 2015. http://www.theses.fr/2015REIMS032.
Testo completoEndophytic PGPR Burkholderia phytofirmans PsJN (Bp) promotes growth of various plants and triggers protection against several environmental stresses. To get more insights into the interaction between plant and Bp, we focused on leaf physiological and metabolic aspects of Arabidopsis thaliana. We also determined the mechanisms involved in the defense of leaves after inoculation of the bacteria followed by an abiotic (cold) or a biotic (Pseudomonas syringae pv. tomato DC3000, Pst) stress. Our results show that the induction of growth promotion of A. thaliana by Bp could be related to the accumulation of primary metabolite levels (amino acids, soluble carbohydrates and vitamins) and to the variation of hormone levels in the leaves. Leaf physiology and metabolism are changed locally and distally by Bp epi- and endophytic colonization. In addition, changes in metabolite levels are more pronounced after a relatively long interaction between plant and bacteria.Moreover, Bp inoculation can also reduce cold injury on the photosynthetic activity by a non-stomatal limitation of photosynthesis and accumulation of photosynthetic pigments. Finally, the local presence of Bp causes a delay in the development of Pst, but only in the early stages of the infection. However, the inoculation with Bp does not protect the photosynthetic apparatus during Pst attack.Thus, our results emphasize that the time of presence of a PGPR and his location in the plant could influence the plant physiology and stress tolerance
Berg, Femke van den. "The evolution of plant defence against pests : pathogen development and pathogen-induced leaf necrosis". Thesis, University of Reading, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.421209.
Testo completoNurtay, Anel. "Mathematical modelling of pathogen specialisation". Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/667178.
Testo completoLa aparición de nuevos virus causantes de enfermedades está estrechamente ligada a la especialización de las subpoblaciones virales hacia nuevos tipos de anfitriones. La modelizaci ón matemática proporciona un marco cuantitativo que puede ayudar a la predicción de procesos a largo plazo como la especialización. Debido a la naturaleza compleja que presentan las interacciones intra e interespecíficas en los procesos evolutivos, aplicar herramientas matemáticas complejas, tales como el análisis de bifurcación, al estudiar dinámicas de población. Esta tesis desarrolla una jerarquía de modelos de población para poder comprender la aparición y las dinámicas de especialización, y su dependencia de los parámetros del sistema. Utilizando un modelo para un virus de tipo salvaje y un virus mutado que compiten por el mismo anfitrión, se determinan las condiciones para la supervivencia únicamente de la subpoblación mutante, junto con su coexistencia con la cepa de tipo salvaje. Los diagramas de estabilidad que representan regiones de dinámicas diferenciadas se construyen en términos de tasa de infección, virulencia y tasa de mutación; los diagramas se explican en base a las características biológicas de las subpoblaciones. Para parámetros variables, se observa y se describe el fenómeno de intersección e intercambio de estabilidad entre diferentes soluciones sistemáticas y periódicas en el ámbito de las cepas de tipo salvaje y las cepas mutantes en competencia directa. En el caso de que varios tipos de anfitriones estén disponibles para ser disputados por cepas especializadas y generalistas existen regiones de biestabilidad, y las probabilidades de observar cada estado se calculan como funciones de las tasas de infección. Se ha encontrado un raro atractor caótico y se ha analizado con el uso de exponentes de Lyapunov. Esto, combinado con los diagramas de estabilidad, muestra que la supervivencia de la cepa generalista en un entorno estable es un hecho improbable. Además, se estudia el caso de los varias cepas N>> 1 que compiten por diferentes tipos de células anfitrionas. En este caso se ha descubierto una dependencia no monotónica, contraria a lo que se preveía, del tiempo de especialización sobre el tamaño inicial y la tasa de mutación, como consecuencia de la realización de un análisis de regresión sobre datos obtenidos numéricamente. En general, este trabajo hace contribuciones amplias a la modelización matemática y el análisis de la dinámica de los patógenos y los procesos evolutivos.
The occurrence of new disease-causing viruses is tightly linked to the specialisation of viral sub-populations towards new host types. Mathematical modelling provides a quantitative framework that can aid with the prediction of long-term processes such as specialisation. Due to the complex nature of intra- and interspecific interactions present in evolutionary processes, elaborate mathematical tools such as bifurcation analysis must be employed while studying population dynamics. In this thesis, a hierarchy of population models is developed to understand the onset and dynamics of specialisation and their dependence on the parameters of the system. Using a model for a wild-type and mutant virus that compete for the same host, conditions for the survival of only the mutant subpopulation, along with its coexistence with the wild-type strain, are determined. Stability diagrams that depict regions of distinct dynamics are constructed in terms of infection rates, virulence and the mutation rate; the diagrams are explained in terms of the biological characteristics of the sub-populations. For varying parameters, the phenomenon of intersection and exchange of stability between different periodic solutions of the system is observed and described in the scope of the competing wild-type and mutant strains. In the case of several types of hosts being available for competing specialist and generalist strains, regions of bistability exist, and the probabilities of observing each state are calculated as functions of the infection rates. A strange chaotic attractor is discovered and analysed with the use of Lyapunov exponents. This, combined with the stability diagrams, shows that the survival of the generalist in a stable environment is an unlikely event. Furthermore, the case of N=1 different strains competing for different types of host cells is studied. For this case, a counterintuitive and non-monotonic dependence of the specialisation time on the burst size and mutation rate is discovered as a result of carrying out a regression analysis on numerically obtained data. Overall, this work makes broad contributions to mathematical modelling and analysis of pathogen dynamics and evolutionary processes.
Akhras, Michael S. "Nucleic Acid Based Pathogen Diagnostics". Doctoral thesis, KTH, Skolan för bioteknologi (BIO), 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-4684.
Testo completoPatogena organismer smittas till värd organismen genom alla möjliga kontaktnätverk och skapar en mångfald olika sjukdomstillstånd. Dock är det fortfarande vanligt förekommande behandlingsbara infektiösa sjukdomar som orsakar den största hälsoförlusten, sett från ett globalt perspektiv. Bill och Melinda Gates Stiftelsen samarbetade med RAND kooperation för att forma “The Global Health Diagnostics Forum”. Deras mål var att etablera och analysera matematiska modeller för vilka effekter en ny diagnostisk metod utrustat för fältarbete skulle ha i utvecklingsländer. Resultaten var häpnadsveckande, med potentiellt miljoner av liv som skulle kunna räddas på en årlig basis. Den etablerade standarden för diagnostik av patogena bakterier har länge varit kultiveringsmedia baserad. Miljö specialiserade biologer har estimerat att mindre än 1 % av alla bakterie arter går att kultivera. Dock erbjuder genetiska analyser potentialen att kunna identifiera alla mikrober från alla de biologiska rikena. Nukleinsyrebaserade diagnostiska metoder har märkbart förbättrats över de senaste årtionden. Nya tekniker erbjuder utökad sensitivitet, selektivitet, sänkta kostnader och parallella analyser av patient prover. Dock är de flesta metoderna begränsade till standardiserade laboratoriemiljöer. För att konstruera en väl fungerande diagnostisk fältutrustning för användning i problem områden, behöver världsledande tekniker identifieras och kombineras. Fokuseringsområdet för denna doktorsavhandling har varit att utveckla och utföra nukleinsyrebaserade metoder för patogen diagnostik. Metoder och experimentella utförande applicerades på två distinkta system i) sökning av antibiotika resistens relaterade mutationer i den patogena bakterien Neisseria gonorrhoeae och ii) genotypning av det cancer orsakande Humana Papillomaviruset (HPV). Den första delen av studien inriktade sig mot utveckling av snabba, direkta och multiplexa Pyrosekvenserings baserade nukleinsyreanalyser. Med förbättrad provprepareringsmetodologi kunde vi detektera multipla HPV infektioner med högre sensitivitet än vad tidigare beskrivits med liknande metodologi. Den andra delen av studien fokuserades på multiplexa nukleinsyre amplifikationer med “Molecular Inversion Probe” tekniken med sista steg Pyrosekvenserings analys. “PathogenMip assay” erbjuder ett komplett detektionsprotokoll för alla kända patogena organismer. Vi introducerar även den nya “Connector Inversion Probe”, en “Padlock Probe” kapabel att genomföra kompletta gap fyllningar för multiplex nukleinsyre amplifiering.
QC 20100624
Ho, Timothy Boon Leong. "Pathogen polymorphisms of mycobacterium tuberculosis". Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.399538.
Testo completoSealfon, Rachel (Rachel Sima). "Computational investigation of pathogen evolution". Thesis, Massachusetts Institute of Technology, 2015. http://hdl.handle.net/1721.1/99858.
Testo completoCataloged from PDF version of thesis.
Includes bibliographical references (pages 105-118).
Pathogen genomes, especially those of viruses, often change rapidly. Changes in pathogen genomes may have important functional implications, for example by altering adaptation to the host or conferring drug resistance. Accumulated genomic changes, many of which are functionally neutral, also serve as markers that can elucidate transmission dynamics or reveal how long a pathogen has been present in a given environment. Moreover, systematically probing portions of the pathogen genome that are changing more or less rapidly than expected can provide important clues about the function of these regions. In this thesis, I (1) examine changes in the Vibrio cholerae genome shortly after the introduction of the pathogen to Hispaniola to gain insight into genomic change and functional evolution during an epidemic. I then (2) use changes in the Lassa genome to estimate the time that the pathogen has been circulating in Nigeria and in Sierra Leone, and to pinpoint sites that have recurrent, independent mutations that may be markers for lineage-specific selection. I (3) develop a method to identify regions of overlapping function in viral genomes, and apply the approach to a wide range of viral genomes. Finally, I (4) use changes in the genome of Ebola virus to elucidate the virus' origin, evolution, and transmission dynamics at the start of the outbreak in Sierra Leone.
by Rachel Sealfon.
Ph. D.
Sidhu, Jatinder. "Pathogen regrowth in composted biosolids". Thesis, Sidhu, Jatinder (2000) Pathogen regrowth in composted biosolids. PhD thesis, Murdoch University, 2000. https://researchrepository.murdoch.edu.au/id/eprint/52513/.
Testo completole, Roux Marie Cecilia. "Mycoplasma genitalium,passenger or pathogen?" Thesis, University of Limpopo (Medunsa), 2010. http://hdl.handle.net/10386/251.
Testo completoMycoplasma genitalium is the smallest existing self-replicating prokaryote, lacks a cell wall and has a genome consisting of only 580 kilo base pairs. It has characteristic pear/flask-like morphology with a terminal tip organelle used for attachment. Many researchers, mainly in developed countries, have investigated the role the organism plays in the aetiology of male urethritis and the majority of studies show an association between M. genitalium and male urethritis. In this study, the modified Koch’s postulates were applied to answer the question whether M. genitalium is a true pathogen, or merely a passenger, invading already inflamed or damaged cells. A total of 300 urine specimens were collected from adult males with symptoms and/or signs of urethritis and 75 from asymptomatic men. In the first study, three molecular assays; viz, a commercial conventional PCR test, a real-time PCR (q- PCR) test and a transcription mediated amplification (TMA) assay were evaluated for the detection of M. genitalium. The comparison between the assays was based on the extended gold standard concept, where a specimen was deemed positive when any two nucleic acid amplification tests were positive. In the second study, the specimens were tested for four common urethral pathogens (N. gonorrhoeae, C. trachomatis, T. vaginalis and M. genitalium) using TMA assays. Finally, the bacterial loads for M. genitalium were determined using the q-PCR assay. v All three assays tested were highly specific (98-99%) for the detection of M. genitalium. However, where q-PCR and TMA demonstrated high sensitivities (96% and 100%), the sensitivity of the conventional PCR assay was low (78%). One or more pathogens were detected in a total of 129 (43%) men with urethritis. M. genitalium was the most frequently detected pathogen in men with urethritis (129; 43%), and significantly more (p= 0.04) than in asymptomatic men (7; 9.0%). There is a strong association with M. genitalium bacterial load and clinical urethritis. Patients with urethral discharge had significantly higher M. genitalium concentrations than those with only burning on micturition (p<0.001), and the bacterial concentrations in men with symptoms and/or signs of urethritis were significantly higher than that in asymptomatic men (p=0.02). As the number of organisms increased, the severity of the symptoms increased; an indication of the role that the organism plays in disease progression. In conclusion, by applying the modified Koch postulates, it was shown that Mycoplasma genitalium is by no means a passenger, but rather an important cause of adult male urethritis that should be taken into account when making diagnosis and when designing treatment strategies.
Thomas, Graham. "The host-pathogen interface : characterising putative secreted proteins of the honeybee pathogen Nosema ceranae (Microsporidia )". Thesis, University of Exeter, 2015. http://hdl.handle.net/10871/21445.
Testo completoVieBrock, Lauren. "ORIENTIA TSUTSUGAMUSHI ANKYRIN-REPEAT PROTEIN FAMILY TARGETING OF THE HOST ENDOPLASMIC RETICULUM". VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/4023.
Testo completoHennecke, Berthold Rembertus. "Host-pathogen interactions between the fungal pathogen Phloeospora mimosae-pigrae and Mimosa pigra, giant sensitive plant /". [St. Lucia, Qld.], 2002. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17081.pdf.
Testo completoFilippini, Chiara. "Multiple pathogen detection using nanoplasmonic sensing". Master's thesis, Alma Mater Studiorum - Università di Bologna, 2014. http://amslaurea.unibo.it/7344/.
Testo completoPetzold, Katja. "NMR studies of host-pathogen interactions". Doctoral thesis, Umeå universitet, Medicinsk kemi och biofysik, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-25710.
Testo completoRingqvist, Emma. "Host-Pathogen Responses during Giardia infections". Doctoral thesis, Uppsala universitet, Mikrobiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-108980.
Testo completoMcDonald, Allison. "Pathogen-induced inflammation in immunocompromised conditions". Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/47546.
Testo completoScience, Faculty of
Microbiology and Immunology, Department of
Graduate
Rofe, A. P. "Pathogen manipulation of intracellular membrane traffic". Thesis, University of York, 2015. http://etheses.whiterose.ac.uk/12024/.
Testo completoOtten, Lucienne. "Pathogen detection based on carbohydrate adhesion". Thesis, University of Warwick, 2015. http://wrap.warwick.ac.uk/77814/.
Testo completoKathiria, Palak, e University of Lethbridge Faculty of Arts and Science. "Incompatible and compatible plant pathogen interactions". Thesis, Lethbridge, Alta. : University of Lethbridge, Faculty of Arts and Science, 2006, 2006. http://hdl.handle.net/10133/351.
Testo completoxvi, 147 leaves : ill. (some col.) ; 29 cm.
Hann, Dagmar R. "Early events in plant-pathogen interactions". Thesis, University of East Anglia, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.502371.
Testo completoMastorakis, Emmanouil. "Chromatin remodelling during plant-pathogen interactions". Thesis, University of Warwick, 2017. http://wrap.warwick.ac.uk/101423/.
Testo completoGupta, Saurabh Ph D. Massachusetts Institute of Technology Dept of Biological Engineering. "Genetically programmable pathogen sense and destroy/". Thesis, Massachusetts Institute of Technology, 2012. http://hdl.handle.net/1721.1/75839.
Testo completoThis electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.
Cataloged from student submitted PDF version of thesis.
Includes bibliographical references (p. 123-134).
Twenty five percent of all the deaths worldwide are caused by infectious diseases. They are also the biggest cause of mortality among children under five years of age. Among them diarrheal diseases alone cause as many deaths as AIDS or TB and malaria combined. Also up to 80% of traveler's diarrhea is bacterial in nature. Vibrio cholerae (cholera), Salmonella spp (typhoid fever), Shigella spp (shigellosis) and a variety of enteropathogenic Escherichia coli strains are among the principle bacterial agents that cause this type of diarrhea. Improvements in hygiene and access to adequate nutrition are good strategies but immunization against specific diseases still offers the best solution to fight these infections. Unfortunately the wide diversity of bacterial and viral agents that cause diarrhea complicates accurate diagnosis and makes the development of vaccines difficult. Antibiotics used in timely manner and in appropriate doses can be effective but the diagnosis is usually made too late for the therapy to be effective. Moreover frequent use of over-the-counter drugs without any medical supervision has led to multidrug resistance in most of the bacterial strains. To counter this problem I demonstrate a proof of principle of a novel cell therapy against Pseudomonas Aeruginosa (major cause of urinary tract disease and hospital infections). Using principles of Synthetic Biology I genetically modified a probiotic strain of E. coli to specifically detect PAO₁ and respond by secreting a novel, pathogen-specific engineered toxin. Additionally, I translated the bacterial system into mammalian cells and established a foundation for an adaptive system where the sentinel cells secrete an alternate toxin if the pathogen becomes resistant to the first one. Finally, based on this system I proposed designs against highly pathogenic strains of Shigella, Salmonella and Vibrio cholerae. This cell therapy remains inert until a threat is detected, and then serves as an early detection and rapid response agent. Furthermore this platform can be tuned to release minimum but sufficient amounts of very narrow spectrum antimicrobial proteins to control the early stages of infection before the disease becomes systemic. Therefore this system's rapid, automated and highly specific response can be helpful in reducing the occurrence of dose dependent resistance. This approach offers a single integrated solution to eradicating multiple threats with a strategy that is a rapid, selective, and highly sensitive.
by Saurabh Gupta.
Ph.D.
Arnold, Markus F. F. "Host-pathogen interactions in chronic infections". Thesis, University of Aberdeen, 2012. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=192267.
Testo completoRoss, David. "Data mining for exotic pathogen spread". Thesis, University of Strathclyde, 2016. http://digitool.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=27557.
Testo completoAsif, Muhammad. "Acanthamoeba and the bacterial pathogen interactions". Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/20427.
Testo completoLim, Elisa X. "Host-pathogen interactions during alphavirus infection". Thesis, Griffith University, 2021. http://hdl.handle.net/10072/410163.
Testo completoThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
Institute for Glycomics
Griffith Health
Full Text
Olive, Andrew James. "Immunity to Chlamydia trachomatis and Host-Pathogen Interactions During Infection". Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:11263.
Testo completoDauch, Amélie L. "Velvetleaf-Colletotrichum coccodes pathosystem : molecular monitoring of the pathogen and gene expression analysis during plant pathogen interaction". Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=102492.
Testo completoSastry, J. S. S. G. M. "Molecular studies on genetic variability and plant pathogen interactions in pearl millet downy mildew(sclerospora graminicola) pathogen". Thesis(Ph.D.), CSIR-National Chemical Laboratory, Pune, 1998. http://dspace.ncl.res.in:8080/xmlui/handle/20.500.12252/3384.
Testo completoCrossley, Brian E. "Role of the Exopolysaccharide Alginate in Adherence to and Inflammation of Pulmonary Epithelial Cells". VCU Scholars Compass, 2016. http://scholarscompass.vcu.edu/etd/4473.
Testo completoTerry, Karianne. "Chemotactic Signaling in the human gastric pathogen /". Diss., Digital Dissertations Database. Restricted to UC campuses, 2005. http://uclibs.org/PID/11984.
Testo completoMarotta, Nicole Ella. "Patterned nanoarray sers substrates for pathogen detection". Diss., Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/37274.
Testo completoDourou, Dimitra. "Pathogen responses in foods : underestimated ecophysiological factors". Thesis, Cranfield University, 2009. http://dspace.lib.cranfield.ac.uk/handle/1826/3809.
Testo completoCarlin, Aaron Foster. "Siglec interactions with a sialylated bacterial pathogen". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3263070.
Testo completoTitle from first page of PDF file (viewed April 9, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
Buckee, Caroline O'Flaherty. "The evolution and maintenance of pathogen diversity". Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.433383.
Testo completoLipsitch, Marc. "Pathogen transmission and the evolution of virulence". Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294342.
Testo completoHitchman, Richard B. "Pathogen variability and dynamics in insect populations". Thesis, Oxford Brookes University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.247600.
Testo completoZhang, Hongliang. "The pathogen construct in project risk analysis". Thesis, Lancaster University, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.507279.
Testo completoPatel, Trupti. "Mechanisms of Pathogen Sensing in Cardiac Myocytes". Thesis, Imperial College London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.486584.
Testo completoMarchione, Wesley A. "Pathogen resistance genes and proteins in orchids". Virtual Press, 2003. http://liblink.bsu.edu/uhtbin/catkey/1260625.
Testo completoDepartment of Biology
Weaver, Louise. "Protozoan pathogen removal by wastewater treatment systems". Thesis, University of Portsmouth, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.419025.
Testo completoReade, Brian. "The population dynamics of mixed pathogen infections". Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264017.
Testo completo