Letteratura scientifica selezionata sul tema "Pagents"
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Articoli di riviste sul tema "Pagents"
Twardy, Chuck. "Pagers, Pageants and Powwows". Afterimage 26, n. 6 (maggio 1999): 17. http://dx.doi.org/10.1525/aft.1999.26.6.17.
Testo completoMantovani, Giulia, Anna Fagotti, Massimo Franchi, Giovanni Scambia e Giorgia Garganese. "Reviewing vulvar Paget’s disease molecular bases. Looking forward to personalized target therapies: a matter of CHANGE". International Journal of Gynecologic Cancer 29, n. 2 (23 gennaio 2019): 422–29. http://dx.doi.org/10.1136/ijgc-2018-000080.
Testo completoZiyanak, Sebahattin, e Steve Aicinena. "Native American Princess Pageants and Their Role in Maintaining Tribal Cultures". Great Plains Research 34, n. 1 (marzo 2024): 25–43. http://dx.doi.org/10.1353/gpr.2024.a933408.
Testo completoKhan, Shana Makhmood, e Suprayogi Suprayogi. "SCHEMATIC STRUCTURE AND METADISCOURSE ANALYSIS OF BEAUTY PAGEANT-WINNING ANSWERS". JOURNAL OF LANGUAGE 6, n. 1 (22 maggio 2024): 112–24. http://dx.doi.org/10.30743/jol.v6i1.9104.
Testo completoAnja, Kerschen, Dano Hélène, Van Eeckhout Pascal, Marot Liliane e Van Bockstal Mieke. "Not All Cases of Mammary Paget’s Disease are Cytokeratin-7 Positive: A Challenging Diagnosis!" International Journal of Surgical Pathology 29, n. 6 (22 marzo 2021): 631–34. http://dx.doi.org/10.1177/10668969211002920.
Testo completoReid, Ellie. "Dressing the Pageanteers: The Local People and Theatre Professionals who Costumed Edwardian Historical Pageants". Costume 58, n. 1 (marzo 2024): 21–47. http://dx.doi.org/10.3366/cost.2024.0285.
Testo completoShrestha, Tara Lal. "Body Politics in Beauty Pageants: A Study of Miss Nepal Discourse". SCHOLARS: Journal of Arts & Humanities 5, n. 1 (15 febbraio 2023): 1–15. http://dx.doi.org/10.3126/sjah.v5i1.52470.
Testo completoTourangeau, Rémi, e Marcel Fortin. "Le Phénomène des pageants au Québec". Theatre Research in Canada 7, n. 2 (gennaio 1986): 215–38. http://dx.doi.org/10.3138/tric.7.2.215.
Testo completoBae, Soo Youn, Ji Eun Lee, Woo-Chan Park, Chang Ik Yoon, Dooreh Kim, Kabsoo Shin e Youngjoo Lee. "Clinical subtypes and survival outcomes in invasive breast cancer with Paget’s disease: A SEER population-based study." Journal of Clinical Oncology 41, n. 16_suppl (1 giugno 2023): e12564-e12564. http://dx.doi.org/10.1200/jco.2023.41.16_suppl.e12564.
Testo completoHavird, David. "Pageants". Sewanee Review 115, n. 3 (2007): 331–34. http://dx.doi.org/10.1353/sew.2007.0073.
Testo completoTesi sul tema "Pagents"
Williams, Caroline. "It's Not a Beauty Pageant!: An Examination of Leadership Development through Alaska Native Pageants". Diss., The University of Arizona, 2013. http://hdl.handle.net/10150/293488.
Testo completoAndrade, José Costa de [UNESP]. "Perfil imunoistoquímico do carcinoma de Paget da mama". Universidade Estadual Paulista (UNESP), 2004. http://hdl.handle.net/11449/103714.
Testo completoUniversidade Estadual Paulista (UNESP)
O objetivo deste trabalho foi estudar através do exame Imunoistoquimico, a correlação entre alguns fatores Anatomopatológicos (tipo histológico e grau nuclear) e alguns fatores biológicos (receptores hormonais de estrógeno e de progesterona, proteína c-erbB-2 e proteína p53, no carcinoma de Paget da mama, doença rara e especial. No período entre Janeiro de 1980 a Dezembro de 1998 foram tratados no Instituto Brasileiro de Controle do Câncer, 6.303 casos de câncer de mama, deste total 98 casos foram diagnosticados como carcinoma de Paget, cuja incidência foi de 1,55%. Estudamos retrospectivamente 60 casos, sendo que 38 deles foram excluídos da análise, devido a limitação e escassez da amostra no material disponível, a idade das pacientes variou entre 26 e 84 anos, com média de 55,4 anos, as informações clínicas e terapêuticas foram obtidas dos prontuários das pacientes considerando, idade na época do diagnóstico, tamanho do tumor quando palpável, estadiamento clínico, e o tipo de cirurgia realizada. As amostras de tecido mamário foram recuperadas dos arquivos do Departamento de Anatomia Patológica do I.B.C.C. / MATTOSINHO. Após a revisão histológica, realizada por dois patologistas os casos foram classificados em quatro grupos: Grupo A- Doença de Paget (forma pura) n = 09 Grupo B- Doença de Paget + neoplasia ductal “in situ”, n = 12 Grupo C- Doença de Paget, neoplasia ductal invasora, n = 30 Grupo D- Doença de Paget + neoplasia ductal “in situ”+ neoplasia ductal invasora, n = 09. Entre as variáveis anatomopatológicas, o grupo C prevaleceu com 30 casos (50%). O grau nuclear (GN-II) predominou com 45 casos (75%). Em relação as variáveis biológicas o receptor de estrógeno negativo predominou com 41 casos (68,3%), seguido pelo receptor de progesterona negativo com 40 casos (66,7%), podemos dizer que...
The objective of this paper was to study the immunohistochemical efects of Paget’s disease, a rare and special carcinoma of the breast, by correlating anatomopathological (histological and nuclear grade) and biological (estrogen and progesterone hormonal receptors, c-erbB-2 protein and p53 protein) factors. Between January 1980 and December 1998, 6303 cases of breast cancer were treated at the Brazilian Institute of Cancer Control; 98 of these were diagnosed with Paget’s carcinoma, an incidence of 1.55%. We retrospectively studied 60 of these cases; 38 were excluded due to lack of available sample material. Patient age varied between 26 and 84 years (mean 55.4), clinical and therapeutic information were obtained from patient’s medical records considering age at time of diagnosis, tumor size when palpable, clinical stage, and type of surgery performed. Samples of breast tissue were retrieved the Anatomical Pathology Department, I.B.C.C. / MATTOSINHO. After histological review, by two different pathologists, they were classified into four groups: Group A- Paget’s disease (pure form) n = 09 Group B- Paget’s disease + “in situ” duct neoplasia n = 12 Group C- Paget’s disease, invasive duct neoplasia n = 30 Group D- Paget’s disease + “in situ” duct neoplasia + invasive duct neoplasia n = 09.Invasive duct neoplasia was the most prevalent anatomopathological variable (Group C, n = 30, 50%). Nuclear grade (GNII) was found in 45 cases (75%). In relation to the biological variables, the negative estrogen receptor was predominant with 41 cases (68.3%), followed by the negative progesterone receptor with 40 cases (66.7%); this correlation had good concordance by the Kappa test. The c-erbB-2 protein was positive in 53 cases (88.3%) and p53 was negative in 47 cases (78.3%). From these results, we concluded that there was no statistical... (Complete abstract click electronic address below)
Lewis, Heidi D. ?UNAUTHORIZED. ""Speaking out of the dust" : religious reenactments with the specific iconic identity of place /". Diss., CLICK HERE for online access, 2006. http://contentdm.lib.byu.edu/ETD/image/etd1478.pdf.
Testo completoAmilibia, Cabeza Emilio. "Caracterización de la hipoacusia en la enfermedad de Paget". Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/664352.
Testo completoIntroduction and objectives: Paget’s disease of bone (PDB) may lead to hearing loss. The present study is conducted with the aim of measuring, characterizing, determining the risk factors for hearing loss and analyse the possible relation of hearing loss with tomography findings in a group of subjects with PDB. Methods: An observational, transversal, case-control study was conducted, a cohort of 76 subjects diagnosed of Paget’s disease of bone (PDB) in the case group and a control group of 134 subjects were included. Clinical, demographic, audiometric and tomographic data (by means of a CT scan study) were analysed. Results: The comparative analysis between the subjects in the PDB group and the control group found that the case group showed higher hearing thresholds (39,51dB) comparing with the control group (37,28 dB) (p=0,069) and presented a greater rate of conductive hearing loss (22,76%) than the control group (12,05%) (p=0,0062). The study of risk factors for hearing loss found that skull involvement in bone scintigraphy, age and high blood pressure were risk factors for higher hearing impairment in PDB. The CT scan data showed that subjects in the PDB group had lower temporal bone density (Hounsfield Units, HU) comparing with the control group. It was also found that the otic capsule bone density in the PDB group correlated with both the air and bone conduction hearing thresholds (p<0,05) and that the finding of an air-bone gap in the audiogram correlated with Paget’s disease temporal bone involvement. Conclusions: − The 63,45% of subjects with Paget’s disease of bone (PDB) suffer hearing loss with a hearing threshold of 39,51dB on average. − The subjects with PDB showed a deeper hearing loss and a higher proportion of conductive type than the control group. − The patients with PDB and skull involvement presented a more severe hearing loss compared with the subjects without skull involvement. Skull involvement and age were found to be risk factors for hearing loss. − In the PDB group both the air and bone conduction hearing thresholds correlated with the otic capsule bone density.
Conesa, Mateos Arántzazu. "Caracterización clínica, epidemiológica y molecular de la enfermedad Ósea de Paget". Doctoral thesis, Universitat Autònoma de Barcelona, 2021. http://hdl.handle.net/10803/673198.
Testo completoLa enfermedad ósea de Paget (EOP) se caracteriza por ser un trastorno crónico y focal del remodelado óseo que conlleva a la aparición de complicaciones como deformidades óseas, artropatía por vecindad y fracturas. La EOP presenta una gran variabilidad en su distribución en función de la edad, género, etnia y área geográfica. Recientes estudios han descrito cambios seculares en cuanto a la prevalencia, incidencia y gravedad al diagnóstico de la EOP, mostrando una disminución en las últimas décadas. La EOP se considera actualmente una enfermedad multifactorial con participación de factores ambientales y genéticos. El gen que ha mostrado mayor susceptibilidad es el Sequestosoma1(SQSMT1), las mutaciones descritas en él no explicarían de forma completa la patogenia de la EOP. En la actualidad, son escasos los estudios acerca de variaciones genéticas en los genes TNFRSF11B y TNFRSF11A asociados a EOP clásica así como la existencia de interacciones genéticas entre ambos genes y el gen SQSMT1, que conduzca a un incremento de riesgo de EOP e incluso que influya en el fenotipo. Con la finalidad de investigar el patrón de comportamiento clínico-epidemiológico como genético de la EOP de nuestra población de baja prevalencia, nos planteamos los siguientes objetivos. Los objetivos se centraron en evaluar cambios en la evolución del porcentaje de nuevos diagnósticos de EOP durante el periodo 1970-2009 y describir en dichos pacientes diferencias en cuanto a la severidad al diagnóstico. Así como así como analizar la importancia de alteraciones moleculares en el gen SQSTM1 y la presencia de variaciones alélicas en los genes TNFRSF11B y TNFRSF11A, tanto en la susceptibilidad para el desarrollo de la EOP como la influencia en su fenotipo. Pacientes y Métodos: Estudio descriptivo ambispectivo observacional de pacientes procedentes del Hospital del Mar-Parc de Salut Mar (Barcelona), un área de baja prevalencia. Se incluyeron 393 pacientes, diagnosticados entre 1970 y 2009. En base al estudio molecular se llevó a cabo el genotipado de las 21 variantes polimórfica para el estudio los genes TNFRSF11B y TNFRSF11A, así como la secuenciación del gen SQSTM1 en una población de 200 pacientes afectos de EOP y 200 controles hipernormales. Resultados: En el estudio se observa un descenso progresivo del porcentaje de nuevos diagnósticos de EOP en relación a la población de referencia, entre 1994 y 2009, más marcado en el grupo de ≥ de 65 años. Asimismo, se ha constatado que los pacientes afectos de EOP presentan una disminución en la actividad biológica de la enfermedad, una menor extensión de la enfermedad y una mayor edad de presentación al diagnóstico, en función del año de nacimiento y año del diagnóstico. En contraposición, se ha observado mayor severidad en el momento del diagnóstico en los pacientes con edades al diagnóstico de <45 años y ≥75 años. Se han identificado 5 factores independientes de mal pronóstico al diagnóstico, que van a favorecer peores desenlaces. Por lo que podemos concluir, que en el momento de diagnóstico de EOP, la expresión de la enfermedad es menos severa y acontece de forma más tardía. Los resultados del estudio molecular destacan la asociación significativa entre los SNPs rs3018362 y rs1805034 del gen TNFRSF11A y rs11573871 del gen TNFRSF11B con el incremento de riesgo a desarrollar EOP así como la influencia en su fenotipo. Asimismo se observó que la presencia del alelo T en el polimorfismo rs6567274 del gen TNFRSF11A se asoció con una disminución de riesgo a desarrollar la EOP, así como a un fenotipo más leve. En el estudio molecular del gen SQSTM1 se identificaron seis variantes genéticas "missense" de nueva descripción, asociadas todas ellas a una mayor susceptibilidad en el desarrollo de la EOP.
Paget’s Disease of Bone (PDB) is characterized by a chronic and focal disorder of bone remodeling that leads to the appearance of complications as bone deformities, osteoarthritis and fractures. The PDB presents a great variability in its distribution depending on the age, gender, ethnics and geographic area. Recent studies have described secular changes in the prevalence, incidence and severity at diagnosis of PDB, showing a decline in the last decades. PDB is currently considered a multifactorial disease involving environmental and genetic factors. The gene which has shown higher susceptibility is the Sequestosome-1 (SQSTM1), the mutations described in it would not be able to explain completely the PDB pathogenesis. Nowadays, there are few studies on genetic variations in the TNFRSF11B and TNFRSF11A genes associated with classic PDB, as well as the existence of genetic interactions between both genes and the SQSMT1 gene, leading to an increased risk of PDB or even influencing the phenotype. To investigate the pattern of clinical-epidemiological and genetic behavior of the PDB in our low prevalence population, we propose the following objectives. The objectives were focused on evaluating changes in the evolution of the percentage of new PDB diagnoses during the period 1970-2009 and to describing in those patients differences in terms of severity at diagnosis. As well as to analyze the importance of molecular alterations in the SQSTM1 gene and the presence of allelic variations in the TNFRSF11B and TNFRSF11A genes, both in the susceptibility for the development of PDB and its influence on the phenotype. Patients and Methods: Descriptive, ambispective observational study of patients from Hospital del Mar-Parc de Salut Mar (Barcelona), a low prevalence area. 393 patients, diagnosed between 1970 and 2009, were included. Based on the molecular study, the genotyping of the 21 polymorphic variants was carried out for the study of the TNFRSF11B and TNFRSF11A genes, as well as the sequencing of the SQSTM1 gene in a population of 200 PDB affected patients and 200 hypernormal controls. Results: The study observed a progressive decrease in the percentage of new PDB diagnoses in relation to the reference population, between 1994 and 2009, more marked in the group ≥65 years old group. Thereby, it has been confirmed that the PDB-affected patients show a decrease in the biological activity of the disease, a lower extension of the disease, and a higher age of presentation at diagnosis, depending on the year of birth and the year of diagnosis. In contrast, it has been observed higher severity at diagnosis in patients with ages at diagnosis <45 years and ≥75 years. Five independent factors of poor prognosis at diagnosis have been identified, which will favor worse outcomes. We can conclude, that at the time of diagnosis of PDB, the expression of the disease is less severe and occurs later. The results of the molecular study highlight the significant association between SNPs rs3018362 and rs1805034 of the TNFRSF11A gene and rs11573871 of the TNFRSF11B gene with the increased risk of developing PDB as well as the influence on its phenotype. Thereby, it was observed that the presence of the T allele in the polymorphism rs6567274 of the TNFRSF11A gene was associated with a decreased risk of developing PDB, as well as a milder phenotype. In the molecular study of the SQSTM1 gene, six newly described “missense” genetic variants were identified, all associated with greater susceptibility in the development of PDB.
Universitat Autònoma de Barcelona. Programa de Doctorat en Medicina
McManus, Stephen. "Regulation of osteoclast activation and autophagy through altered protein kinase pathways in Paget's disease of bone". Thèse, Université de Sherbrooke, 2016. http://hdl.handle.net/11143/8960.
Testo completoAbstract : Paget’s disease of bone (PDB) is a skeletal disorder characterized by focal and disorganized increases in bone turnover. In PDB, osteoclasts are larger, more active, more numerous, and resistant to apoptotic stimuli. While no single root cause has been identified, mutations to the gene encoding the p62 protein, SQSTM1, have been described in a significant population of patients with PDB. Among these mutations, the P392L substitution is the most prevalent, and overexpression of p62P392L in osteoclasts generates at least a partial pagetic phenotype in vitro. Normally this protein mediates a number of cell functions, from control of NF-κB signaling to autophagy. In human osteoclasts, a multiprotein complex containing p62 and protein kinases PKCζ and PDK1 (the principal kinase of Akt), form in response to stimulation by receptor activator of nuclear factor kappa-B ligand (RANKL), the principal osteoclastogenic-signaling cytokine. We found that PKCζ is involved in RANKL-induced activation of NF-κB, and that it contributed to a basal activation of NF-κB observed in p62P392L mutants. This may be regulated in part by a PKCζ dependent increase in p65 phosphorylation at Ser536 which we characterized, independent of IκB. This could represent one alternative pathway by which mutant p62 leads to increased NF-κB activation. We observed increased basal phosphorylation of survival regulators ERK and Akt in PDB that was reduced upon PDK1 inhibition. The activity of 4EBP1 and Raptor, associated with mTOR activity, were also altered in pagetic osteoclasts and regulated by PDK1 inhibition. We then identified autophagic defects common to pagetic osteoclasts; with higher basal levels of LC3II (associated with autophagic structures), regardless of p62 mutation, and reduced sensitivity to autophagy induction in PDB. These results suggest an accumulation of non-degradative autophagosomes. Inhibition of PDK1 not only induced apoptosis in PDB and controls, but significantly reduced resorption in PDB, and with regards to autophagy, PDK1 inhibition was more potent in PDB than in controls. Therefore PDK1/Akt signaling represents an important checkpoint to PDB osteoclast activation. In sum, these results demonstrate the importance of several p62-associated kinases in the over-activation of pagetic osteoclasts, through increased survival and altered signaling. As p62 mutations alone do not account for most cases of PDB, the characterization of these pathways may identify a common factor linking pagetic osteoclasts. Therefore these studies represent a novel approach to osteoclast apoptosis, activation, and autophagy associated with PDB.
Bounahai, Najib. "Moors in Elizabethan/Jacobean entertainments : race, performance and politics /". Thesis, Connect to Dissertations & Theses @ Tufts University, 2001.
Cerca il testo completoAdviser: Barbara Freedman. Submitted to the Dept. of Drama. Includes bibliographical references (leaves 191-207). Access restricted to members of the Tufts University community. Also available via the World Wide Web;
Yoshino, Ayako. "The Edwardian historical pageant". Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.614849.
Testo completoNazari, Shekeba. "Maladie de Paget : résistance à l'apoptose et défaut de l'autophagie". Mémoire, Université de Sherbrooke, 2017. http://hdl.handle.net/11143/11869.
Testo completoAbstract : Paget's disease is an osteopathy characterized by a multifocal increase in bone remodeling, which begins with excessive bone resorption followed by increased bone formation. Osteoclasts "OCs" were incriminated in the initiation of the pagetic process. Pagetic OCs are characterized by a resistance to apoptosis, and abnormalities in the process of autophagy “in particular induction defect”. In order to define whether these two characteristics were linked, we hypothesized the role of Bcl2-Beclin1 complexes. Beclin-1 is an autophagy-inducing protein that can bind anti-apoptotic proteins of the Bcl-2 family; Bcl-2 then inhibits Beclin-1 "and thus the induction of autophagy" while retaining its anti-apoptotic functions. To study the impact of Bcl2 expression on autophagy in human OCs, we used an in vitro differentiation model that uses monocytes, which are derived from umbilical cord blood and grown in the presence of RANKL and MCSF for 21 days. These conditions make it possible to obtain multinucleated cells with an osteoclastic phenotype. To increase the expression of Bcl-2 in OCs and analyze its impact on autophagy due to its interaction with Beclin-1, cultures were stimulated with TNFα or RANKL in order to induce NF-κB activation. The expression of Beclin1 and Bcl2 was confirmed by immunoblotting of Ocs cell lysates. Autophagy was induced in cultures carried out under stringent conditions "nutriment-deprived mediun", but we did not observe any variation in the expression of Bcl2 or Beclin 1 according to the culture conditions or TNFα or RANKL stimulation. TNFα significantly stimulated the activation of NF-κB in HEK cells but not in OCs. However, whatever the conditions, results from immunoprecipitaion experiments did not reveal any association between Beclin1 and Bcl2. On the other hand, the classic interaction partner of Beclin1, PI3K type III, was associated with Beclin1. In conclusion, our work did not allow us to demonstrate the formation of Beclin1 / Bcl2 complexes and the relationship between apoptosis and autophagy, partly because of the complexity of the model "multiple effects of NF-κB and TNFα". Our initial hypothesis should thereby be re-evaluated using a more appropriate methodology. On the other hand, the different techniques are now ready for further study.
Beckham, Kathryn Ann. "'The Gate City' artistry and identity in an American historical pageant (Nebraska) /". Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file 14.18 Mb., 74 p, 2006. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pqdiss&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&rft_dat=xri:pqdiss:1435838.
Testo completoLibri sul tema "Pagents"
Thomas, Heywood. Thomas Heywood's pageants: A critical edition. New York: Garland Pub., 1986.
Cerca il testo completoMakartsoŭ, Mikola. Pa svi͡a︡tochnykh shli͡a︡khakh Belarusi: Rėz͡h︡ysi͡o︡rskii͡a︡ natatki. Minsk: Vyd-va "Chatyry chvėrtsi", 1999.
Cerca il testo completoZelov, D. D. Ofit︠s︡ialʹnye svetskie prazdniki kak i︠a︡vlenie russkoĭ kulʹtury kont︠s︡a XVII-pervoĭ poloviny XVIII veka: Istorii︠a︡ triumfov i feĭerverkov ot Petra Velikogo do ego docheri Elizavety. Moskva: Ėditorial URSS, 2002.
Cerca il testo completoLuigi, Allegri, e Di Benedetto Renato, a cura di. La Parma in festa: Spettacolarità e teatro nel Ducato di Parma nel Settecento. Modena, Italia: Mucchi, 1987.
Cerca il testo completoWood, Stevens Thomas. Magna Carta: A pageant drama. Chicago: American Bar Association, 1994.
Cerca il testo completoPretini, Giancarlo. La piazza delle meraviglie: Il teatro in strada italiano. Reana del Rojale (UD): Trapezio, 1999.
Cerca il testo completoMillones, Luis. El Inca por la coya: Historia de un drama popular en los Andes peruanos. Lima: Fundación Friedrich Ebert, 1988.
Cerca il testo completoCopyright Paperback Collection (Library of Congress), a cura di. The Northeast girls. New York: Berkley Jam Books, 1998.
Cerca il testo completoRandolph, Sallie G. Putting on perfect proms, programs & pageants. New York: F. Watts, 1991.
Cerca il testo completoYves, Pauwels, e Bibliothèque municipale Livrée Ceccano (Avignon, France), a cura di. Les entrées solennelles à Avignon et à Carpentras, XVIe-XVIIIe siècles: 18 septembre-24 octobre 1997, Bibliothèque municipale, Avignon. [Paris]: Ministère [de la] culture [et de la] communication, Direction du livre et de la lecture, 1997.
Cerca il testo completoCapitoli di libri sul tema "Pagents"
Tate, Shirley Anne. "Beauty pageants". In From Post-Intersectionality to Black Decolonial Feminism, 121–53. London: Routledge, 2022. http://dx.doi.org/10.4324/b23223-5.
Testo completoKrasner, David. "African American Performance in the Harlem Renaissance". In A Beautiful Pageant, 1–14. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_1.
Testo completoKrasner, David. "“What Constitutes a Race Drama and How May We Know It When We Find It?”: The Little Theatre Movement and the Black Public Sphere". In A Beautiful Pageant, 207–38. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_10.
Testo completoKrasner, David. "Shuffle Along and the Quest for Nostalgia: Black Musicals of the 1920s". In A Beautiful Pageant, 239–88. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_11.
Testo completoKrasner, David. "Conclusion: The end of “Butter Side Up”". In A Beautiful Pageant, 289–92. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_12.
Testo completoKrasner, David. "Men in Black and White: Race and Masculinity in the Heavyweight Title Fight of 1910". In A Beautiful Pageant, 17–54. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_2.
Testo completoKrasner, David. "Exoticism, Dance, and Racial Myths: Modern Dance and the Class Divide in the Choreography of Aida Overton Walker and Ethel Waters". In A Beautiful Pageant, 55–80. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_3.
Testo completoKrasner, David. "“The Pageant Is the Thing”: Black Nationalism and The Star of Ethiopia". In A Beautiful Pageant, 81–94. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_4.
Testo completoKrasner, David. "Walter Benjamin and the Lynching Play: Mourning and Allegory in Angelina Weld Grimké’s Rachel". In A Beautiful Pageant, 97–111. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_5.
Testo completoKrasner, David. "Migration, Fragmentation, and Identity: Zora Neale Hurston’s Color Struck and the Geography of the Harlem Renaissance". In A Beautiful Pageant, 113–30. New York: Palgrave Macmillan US, 2002. http://dx.doi.org/10.1007/978-1-137-06625-1_6.
Testo completoAtti di convegni sul tema "Pagents"
Shrestha, Eliza, Rupinder Shekon, Shveta Giri e Sudhir Rawal. "Paget’s disease of the vulva in postmenopausal women: A case report". In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685383.
Testo completoTensmeyer, Chris, Brian Davis, Curtis Wigington, Iain Lee e Bill Barrett. "PageNet". In HIP2017: The 4th International Workshop on Historical Document Imaging and Processing. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3151509.3151522.
Testo completoBarros, Emanuelle de Lima, Andre Silva Machado, Josie Haydée Lima Ferreira Paranaguá, Débora Medeiros de Carvalho, Marcos Josué Rocha Cabral de Oliveira, Carlos Eduardo Moura de Lima e Sabas Carlos Vieira. "Mammary Paget’s disease: Clinical–epidemiological profile in a reference oncology clinic in the capital of Piauí". In Brazilian Breast Cancer Symposium 2023. Mastology, 2023. http://dx.doi.org/10.29289/259453942023v33s1090.
Testo completoWentorf, Deb. "Printed pages vs. web pages". In the 29th annual ACM SIGUCCS conference. New York, New York, USA: ACM Press, 2001. http://dx.doi.org/10.1145/500956.501005.
Testo completo"Pages". In 2010 68th Annual Device Research Conference (DRC). IEEE, 2010. http://dx.doi.org/10.1109/drc.2010.5551914.
Testo completoBelluco, Rosana Zabulon Feijó, Flávio Lúcio Vasconcelos, Paulo Eduardo Silva Belluco, Júllia Eduarda Feijó Belluco e Carmelia Matos Santiago Reis. "NIPPLE MINIMUM PAGET DISEASE: A CASE REPORT". In XXIV Congresso Brasileiro de Mastologia. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s1059.
Testo completoAlzahr, A., M. Mansour e B. Knof. "Paget's disease in temporal bone". In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640234.
Testo completoBen Chehida, R., S. Ben Jemaa, Z. Gassara, A. Feki, M. Ezzeddine, R. Akrout, M. H. Kallel, H. Fourati e S. Baklouti. "AB0281 PAGET’S DISEASE: EPIDEMIOLOGICAL PROFILE". In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4033.
Testo completo"TERN/AusCover - Remote sensing data management for terrestrial ecosystem research". In 19th International Congress on Modelling and Simulation. Modelling and Simulation Society of Australia and New Zealand (MSSANZ), Inc., 2011. http://dx.doi.org/10.36334/modsim.2011.h4.paget.
Testo completo"Title pages". In 2009 International Conference on Biomedical and Pharmaceutical Engineering (ICBPE 2009). IEEE, 2009. http://dx.doi.org/10.1109/icbpe.2009.5384061.
Testo completoRapporti di organizzazioni sul tema "Pagents"
Postel, J., e C. Anderson. White Pages Meeting Report. RFC Editor, febbraio 1994. http://dx.doi.org/10.17487/rfc1588.
Testo completoReddy, Sakamuri. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Fort Belvoir, VA: Defense Technical Information Center, agosto 2010. http://dx.doi.org/10.21236/ada539193.
Testo completoReddy, Sakamuri V. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Fort Belvoir, VA: Defense Technical Information Center, agosto 2012. http://dx.doi.org/10.21236/ada567774.
Testo completoReddy, Sakamuri. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Fort Belvoir, VA: Defense Technical Information Center, agosto 2011. http://dx.doi.org/10.21236/ada553287.
Testo completoHansen, Marc. The Nature of Expansion of Paget's Disease of Bone. Fort Belvoir, VA: Defense Technical Information Center, aprile 2013. http://dx.doi.org/10.21236/ada586286.
Testo completoHansen, Marc F. Understanding the Delay in Onset of Paget's Disease of Bone. Fort Belvoir, VA: Defense Technical Information Center, settembre 2014. http://dx.doi.org/10.21236/ada613442.
Testo completoHansen, Marc. On the Nature of Expansion of Paget's Disease of Bone. Fort Belvoir, VA: Defense Technical Information Center, ottobre 2012. http://dx.doi.org/10.21236/ada573353.
Testo completoResnik, P. Evaluating Multilingual Gisting of Web Pages. Fort Belvoir, VA: Defense Technical Information Center, marzo 1997. http://dx.doi.org/10.21236/ada458592.
Testo completoAlvestrand, H., e P. Jurg. Deployment of the Internet White Pages Service. RFC Editor, settembre 1997. http://dx.doi.org/10.17487/rfc2148.
Testo completoHutchins, Thomas. PR744-22113-Z01 Hydrogen Natural Gas Blends in Existing Natural Gas Systems. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), novembre 2022. http://dx.doi.org/10.55274/r0012245.
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