Bibliografie tematiche / Olfactions

Letteratura scientifica selezionata sul tema "Olfactions"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 21 febbraio 2023

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Articoli di riviste sul tema "Olfactions"

1

Wen, Tengteng, Dehan Luo, Jiafeng He e Kai Mei. "The Odor Characterizations and Reproductions in Machine Olfactions: A Review". Sensors 18, n. 7 (18 luglio 2018): 2329. http://dx.doi.org/10.3390/s18072329.

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Machine olfaction is a novel technology and has been developed for many years. The electronic nose with an array of gas sensors, a crucial application form of the machine olfaction, is capable of sensing not only odorous compounds, but also odorless chemicals. Because of its fast response, mobility and easy of use, the electronic nose has been applied to scientific and commercial uses such as environment monitoring and food processing inspection. Additionally, odor characterization and reproduction are the two novel parts of machine olfaction, which extend the field of machine olfaction. Odor characterization is the technique that characterizes odorants as some form of general odor information. At present, there have already been odor characterizations by means of the electronic nose. Odor reproduction is the technique that re-produces an odor by some form of general odor information and displays the odor by the olfactory display. It enhances the human ability of controlling odors just as the control of light and voice. In analogy to visual and auditory display technologies, is it possible that the olfactory display will be used in our daily life? There have already been some efforts toward odor reproduction and olfactory displays.
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2

Gros, A. "Olfaction, émotions et comportements". European Psychiatry 30, S2 (novembre 2015): S32. http://dx.doi.org/10.1016/j.eurpsy.2015.09.094.

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Le nez a ses raisons que la raison ignore. Les émotions olfactives sont souvent inconscientes et vont pourtant diriger nombreux de nos comportements. Ce lien étroit entre émotion et olfaction est dû au fait que certaines régions cérébrales sont à la fois impliquées dans les processus olfactifs et émotionnels (cortex frontal et amygdale). Dans les pathologies neurodégénératives, dont la maladie d’Alzheimer (MA), ces structures vont être atteintes à des stades plus ou moins sévères de la maladie. Au-delà d’une aide au diagnostic précoce, nous avons montré que l’utilisation d’odorants pourrait aider au diagnostic différentiel entre certains troubles de l’humeur et la maladie d’Alzheimer . Sentir et ressentir, humer et humeur : la langue française nous apporte déjà des preuves d’un lien étroit entre nos émotions et notre olfaction. De manière objective nous avons mis en évidence que les odorants étaient des générateurs d’émotion puissants et stables . Ainsi les odeurs pourraient constituer une alternative non pharmacologique de prise en charge des troubles émotionnels de manière pratique, cette présentation abordera, en premier lieu, le lien précoce entre atteinte olfactive et psycho-comportementale dans la maladie d’Alzheimer puis l’intérêt de la stimulation olfactive dans la prise en charge non médicamenteuse des perturbations émotionnelles et comportementales dans cette pathologie. Pour finir, nous nous interrogerons sur l’apport de l’étude de l’olfaction dans la prise en charge des principaux syndromes émotionnels et thymiques du champ de la pathologie psychiatrique.
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3

Jacquot, L. "Olfaction et troubles cognitifs. Application aux pathologies neurodégénératives". European Psychiatry 30, S2 (novembre 2015): S31. http://dx.doi.org/10.1016/j.eurpsy.2015.09.093.

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Il est maintenant clairement établi que de nombreuses maladies neurodégénératives, en particulier la maladie de Parkinson et la maladie d’Alzheimer, sont associées à des troubles olfactifs qui peuvent même précéder l’apparition des symptômes moteurs ou cognitifs. Décrits pour la première fois il y a près de quatre décennies [1,2], les déficits de l’olfaction dans ces pathologies ont depuis fait l’objet de nombreuses études qui mettent notamment en avant leur importante prévalence (autour de 95 % pour la maladie de Parkinson ) et leur apparition dans les stades précoces de la maladie . Des travaux récents soulignent ainsi l’intérêt de l’évaluation clinique des déficiences olfactives dans l’établissement du diagnostic précoce ou différentiel. L’objectif de cette présentation est de faire une synthèse de l’état des connaissances sur les déficits olfactifs dans les pathologies neurodégénératives et, en particulier, dans la maladie de Parkinson. La première partie de l’exposé abordera de façon générale les troubles de l’olfaction, leurs étiologies les plus fréquentes et présentera les différents tests permettant l’examen des fonctions olfactives. La deuxième partie portera plus spécifiquement sur la nature et la physiopathologie des altérations olfactives dans la maladie de Parkinson et sur les analogies et les différences avec d’autres pathologies neurodégénératives, notamment la maladie d’Alzheimer. Enfin, une dernière partie présentera les résultats de quelques études récentes montrant les effets bénéfiques potentiels de l’entraînement olfactif sur la récupération de certaines fonctions olfactives.
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4

Pajot-Augy, Édith. "L’haleine et les capteurs d’odeurs". médecine/sciences 35, n. 2 (febbraio 2019): 123–31. http://dx.doi.org/10.1051/medsci/2019001.

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Grâce à leurs capacités olfactives développées, des animaux sont capables de détecter des odeurs associées à certaines pathologies. Récemment, des nez électroniques, consistant en des réseaux de senseurs artificiels générant une signature caractéristique aux odorants présents, par exemple dans l’haleine, permettent à l’heure actuelle de diagnostiquer des pathologies variées, à des stades précoces, et de façon fiable et non invasive. Pour bénéficier de la capacité intrinsèque et naturellement optimisée du système olfactif animal à détecter et discriminer des traces d’odorants, des dispositifs hybrides bioélectroniques sont également en développement pour le diagnostic médical. Ces divers senseurs olfactifs sont en plein essor avec des résultats prometteurs.
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Leopold, Donald A. "Olfaction". Current Opinion in Otolaryngology & Head and Neck Surgery 2 (febbraio 1994): 22–26. http://dx.doi.org/10.1097/00020840-199402000-00005.

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Doty, Richard L. "Olfaction". Annual Review of Psychology 52, n. 1 (febbraio 2001): 423–52. http://dx.doi.org/10.1146/annurev.psych.52.1.423.

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Pinto, J. M. "Olfaction". Proceedings of the American Thoracic Society 8, n. 1 (1 marzo 2011): 46–52. http://dx.doi.org/10.1513/pats.201005-035rn.

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8

McCormick, James. "OLFACTION". Lancet 341, n. 8853 (maggio 1993): 1126–27. http://dx.doi.org/10.1016/0140-6736(93)93134-m.

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9

de Weert, Ch M. M. "Olfaction". Acta Psychologica 75, n. 2 (novembre 1990): 177. http://dx.doi.org/10.1016/0001-6918(90)90092-t.

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Yates, Darran. "Connecting olfaction". Nature Reviews Neuroscience 15, n. 1 (4 dicembre 2013): 5. http://dx.doi.org/10.1038/nrn3652.

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Più fonti

Tesi sul tema "Olfactions"

1

Lecoq, Jérôme. "Métabolisme cérébral et olfaction : Étude des réponses olfactives et leur consommation d'énergie dans le bulbe olfactif du rat anesthésié". Phd thesis, Université Pierre et Marie Curie - Paris VI, 2008. http://tel.archives-ouvertes.fr/tel-00812501.

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Les techniques modernes d'imagerie fonctionnelle du cerveau utilisent le métabolisme cérébral comme marqueur d'activité neuronale. En effet le cerveau dépend intimement des apports sanguins en métabolites pour son fonctionnement. Cependant les mécanismes de régulation du métabolisme sont encore mal connus. Dans cette étude nous avons utilisé le modèle du bulbe olfactif chez le rat anesthésié pour caractériser la consommation d'oxygène en réponse à une stimulation physiologique. La quantification précise de la vascularisation du bulbe olfactif a pu mettre en évidence que la couche glomérulaire, très dense en synapses, est l'une des zones les plus vascularisées du cerveau. Cette couche est aussi le lieu d'une intense consommation d'oxygène lors du traitement de l'information olfactive. Par contraste, la couche du nerf, complètement dénuée d'interactions synaptiques et très peu vascularisée, consomme peu d'oxygène. L'étude pharmacologique de ces réponses métaboliques nous a permis de montrer que le compartiment post-synaptique du glomérule est le siège de cette intense activité métabolique. Cette dernière est aussi dépendante du traitement de l'information olfactive qui est effectué à la fois dans le bulbe olfactif et à la périphérie, dans la cavité nasale. Ceci nous a permis de caractériser l'effet de l'adaptation périphérique sur la consommation d'oxygène et le traitement local de l'information olfactive. Enfin, nous avons décrit en détail l'importance des phénomènes de diffusion au niveau du réseau microvasculaire dans le rééquilibrage transitoire du taux d'oxygène local.
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2

Matton, Cécile. "Olfaction et parfums". Paris 5, 2001. http://www.theses.fr/2001PA05P006.

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3

Honoré, Axel. "Effet des Cellules Gliales Olfactives issues des Bulbes Olfactifs sur les cellules souches épendymaires et leur progénie après une lésion médullaire". Thesis, Normandie, 2017. http://www.theses.fr/2017NORMR060/document.

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Les lésions médullaires traumatiques (LMT) conduisent à une atteinte des voies nerveuses sensitives et motrices. Leur taux de mortalité reste très élevé, d'où la nécessité de trouver de nouveaux traitements. Les Cellules Gliales Olfactives (CGOs) représentent un candidat intéressant de par leur fonction au sein du système olfactif primaire. La découverte d'une population de cellule souche neurale bordant le canal central de la moelle spinale (MS) adulte, appelées cellules épendymaires, suscite un nouvel espoir dans le domaine des biothérapies. Ce travail de thèse a permis d'étudier l'effet d'une transplantation de CGOs sue le comportement des cellules résidentes de la moelle spinale et notamment les cellules souches épendymaires qui, en association avec les astrocytes et les péricytes, participent aux mécanismes de guérison des LMT. L'utilisation du modèle murin hFoxJ1-CreERT2::YFP (permettant le suivi spécifique des cellules épendymaires et de leur progénie), a montré que les CGOs augmentaient in vitro le potentiel d'auto-renouvellement des cellules souches de la MS et modifiaient leur voie de différenciation vers un type neural. In vivo, la transplantation de CGOs augmente la prolifération des cellules épendymaires ainsi que leur différenciation en astrocytes hypo-réactifs conduisant à la formation d'un environnement post-lésionnel bénéfique à la survie neuronal et l'établissement d'une neurogenèse. Nos travaux ont montré pour la première fois que la transplantation de CGOs après LMT permettait la génération de nouveaux neurones. Ceci constitue un nouvel espoir dans l'établissement de stratégies thérapeutiques pour le traitement des LMT chez l'Homme
The spinal cord injuries (SCI) lead to the damages of the spinal cord or nerves and often cause permanent changes in body functions leading to the death. Cell therapies have raised great hope for regenerative medicine. Clinical data showed that the olfactory ensheathing cells (OECs) enhanced functional recovery after SCI and could be a very attractive therapeutic approach. Moreover, the discovery of a new endogenous resident stem cell population, lining the central canal of the spinal cord, named ependymal stem cells, represents a new hope for the therapy. This thesis analyzed the role of OECs transplantation, on the behaviour of ependymal stem cells since these cells, together with astrocytes and pericytes significantly contribute to the recovery of SCI. The use of the mouse model hFoxJ1-CreERT2::YFP (allowing to specifically follow the ependymal stem cells ant their progeny) showed that OECs increased in vitro the self-renewal potential of spinal cord stem cells and modified their differentiation pathway towards a neural type. In vivo, OECs transplantation significantly increases the proliferation of ependymal cells and their differenciation into hypo-reactive astrocytes leading to the formation of a beneficial environment to neuronal survival and the neurogenesis establishment. Our results also showed for the first time that OECs transplantation after SCI allows the generation of new neurons by non-ependymal cell-derived progenitors. These results represent a new hope in the establishment of therapeutic strategies for the treatment of SCI in humans
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4

Steiger, Silke. "Evolution of avian olfaction". Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-91757.

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Dickinson, Keith. "Receptor mechanisms in olfaction". Thesis, University of Warwick, 1987. http://wrap.warwick.ac.uk/98464/.

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This thesis is a study of receptor-mediated events occurring in olfaction. Potential enzymes involved in olfactory transduction were investigated using standard biochemical techniques, and an investigation of the fatty acid receptor/s was attempted using psychophysical methods. 1. Other investigators in olfaction have recently demonstrated an odorant modulated adenylate cyclase in the frog. We have shown that tissue preparations from the rat have high levels of the enzyme adenylate cyclase. This activity was stimulated in the presence of odorants by up to two times the basal value. The concentrations of odorants used were in the range expected to be physiologically relevant. Both basal and stimulated adenylate cyclase were inhibited by μM calcium ion. Tissue preparations from brain showed no odorant activation. Guanylate cyclase was present at a tenth the concentration of adenylate cyclase and showed no odorant simulation. 2. There are no studies in the literature characterising the cyclic nucleotide phosphodiesterases in olfactory tissue. The Sutherland criteria require the presence of a cyclic nucleotide phosphodiesterase (PDE) in olfactory tissue, if cyclic AMP is to be a second messenger in olfaction. It is possible that olfactory PDE is directly regulated by a receptor, as is found in vision. It is important, therefore, that the PDE’s in olfactory tissue are characterised. Tissue preparations of the rat and sheep were shown to have high levels of cyclic nucleotide phosphodiesterase. The activities were not stimulated in the presence of physiologically relevant concentrations of odorants; inhibition of these activities by odorants was not significantly different from that of brain homogenates. Characterisation of the phosphodiesterase isoenzymes separated by DEAE chromatography established that they resembled the isoenzymes characterised from other tissues. The evidence presented indicates that olfactory tissue does not resemble visual tissue, which has a receptor-linked phosphodiesterase. All evidence suggests that PDE’s in olfactory tissue serve to return cyclic nucleotide concentrations to resting levels after stimulation. The observation that the Type 1 (calcium/calmodulin stimulated) phosphodiesterase is present in high concentrations in olfactory tissue suggests that calcium ion concentration in vivo may be an important regulator of phosphodiesterase activity. 3. The threshold values of various short chain fatty acids, of high purity, were determined using human subjects. The results were one order of magnitude greater than found by Amoore (1970) but showed a similar group trend. Descriptive analysis was also performed on these acids by generating a series of descriptors sufficient to discriminate between the acids. Analysis of the results by principal component analysis yielded a three dimensional map that showed the acids clustering into four groups. These four groups could indicate the presence of four receptors. Attempts to demonstrate structure-activity relationships between the thresholds, and descriptive analysis results with various physical and chemical parameters, failed. This was probably due to the interaction of the acids with more than one receptor. So far, the lack of success in identifying olfactory receptors by researchers has meant that investigations of olfactory receptors have had to be done using psychophysical techniques such as those used in this report. It will probably not be known if these methods are useful for identifying receptors, until the receptors can be purified and characterised biochemically.
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Silveira, Moriyama L. "Olfaction in Parkinson's Disease". Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/18728/.

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This thesis examines the clinical and pathological involvement of the olfactory system in Parkinson’s Disease (PD). The main aim is to investigate the practical use of smell identification tests (SITs) in parkinsonism and tremor. A secondary objective is to investigate the pathological involvement of the rhinencephalon. Commercially available SITs were used to differentiate PD patients from control subjects in the UK, Brazil and Sri Lanka, showing SITs have combinations of sensitivity and specificity greater than 80%. Based on the data obtained a traffic light ruler was devised to determine the likelihood of a patient having PD at the time of the initial consultation. This was then used to interpret SITs in 34 patients with possible parkinsonism, showing 86.4% sensitivity and 80.0% specificity of SITs when compared to dopamine transporter imaging using single photon emission computed tomography (SPECT) as the gold-standard for detecting nigrostriatal dopamine denervation. Olfaction was shown to be severely impaired in parkinsonism related to LRRK2 mutations, moderately impaired in subjects with pure autonomic failure, multiple system atrophy and progressive supranuclear palsy (PSP) and normal in patients with essential tremor, dystonia and in subjects who had been diagnosed as having PD, but were found to have normal scans. This indicates that SITs will be more useful in differentiating PD from non-degenerative tremors than from atypical parkinsonism. Neuropathological changes were investigated in the rhinencephalon and it was demonstrated that α-synuclein accumulation in the primary olfactory cortex is heterogeneous, being more severe in the temporal subdivision of the piriform cortex. The piriform cortex had Lewy body pathology in all 10 PD cases studied, as well as in 7 control cases who presented incidental Lewy body pathology and four cases of LRRK2 related parkinsonism. The piriform cortex had abnormal tau accumulation in 6 PSP patients, suggesting tauopathy in the rhinencephalon is a possible substrate for hyposmia in PSP.
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Österbauer, Robert Alexander. "Multisensory integration of olfaction". Thesis, University of Oxford, 2007. http://ora.ox.ac.uk/objects/uuid:34764b4d-7d12-461b-8a88-c3aa3418d228.

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The primary aim of this thesis was to investigate, using functional magnetic resonance imaging (fMRI), the neurophysiological basis of multisensory integration involving smell and vision. To achieve this goal, several technical challenges had to be addressed: the attainment of sufficiently high quality fMRI images in olfactory brain regions within the orbitofrontal cortex (OFC), the construction of a stimulus delivery system adequate for rapid and controlled odour delivery in the MRI environment, and optimal strategies for delivering and perceiving liquid flavour stimuli in the scanner. In two initial fMRI experiments, strategies including sensitivity encoding and passive shimming to improve OFC image quality were explored. The results demonstrated that both methods can improve signal detection in OFC, a brain area particularly sensitive to susceptibility artefacts. In a further fMRI study, the effectiveness of two methods of delivering odorants dissolved in liquids was compared. In this study, the same set of participants was required to either swallow the liquid immediately after delivery or hold it in their mouths for a brief period of time. The results indicated that while both methods allowed detection of activity in primary olfactory and gustatory cortices, activation of the OFC was not observed when participants swallowed the liquids immediately. This was presumed to be due to the increased head motion associated with swallowing. Finally, the mechanisms underlying visual-olfactory integration were investigated using a combination of behavioural and imaging methods. An initial behavioural study revealed strong colour-odour associations for certain smells associated with fruits (e.g. lemon - yellow). In a subsequent fMRI study, volunteers were presented with a selection of the most colour-associated odours from the prior behavioural study either in isolation or in the presence of congruent and incongruent colours. Analysis of the fMRI data revealed that a highly left lateralised network of brain areas comprising of the OFC and insular showed increasingly stronger responses to odour-colour combinations of higher congruency. In a follow-up fMRI study, this same network was also found to be responsible for integrating odours, not only with colours, but also with their corresponding visual images (objects). In sum, the series of fMRI studies undertaken in this thesis argue for a fundamental role of the OFC in the integration of olfactory-visual inputs in the human brain.
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8

Steiger, Silke S. Fidler Andrew Eric Kempenaers B. Mueller Jakob C. "Evolution of avian olfaction". Connect to this title online (Universität München site) Connect to this title online (Deutsche Nationalbibliothek site), 2008. http://d-nb.info/991247264/34.

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Thesis (doctoral)--Ludwig-Maximilians-Universität München, 2008.
Title from PDF t.p. (viewed on Jan. 8, 2009). Some chapters co-authored with others. Includes bibliographical references (p. 117-127).
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Steiger, Silke S. "Evolution of avian olfaction". kostenfrei, 2008. http://edoc.ub.uni-muenchen.de/9175/.

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Merle, Laëtitia. "Impact d’une alimentation maternelle riche en graisse et en sucre pendant les périodes de préconception, gestation et lactation sur la physiologie olfactive de la progéniture : étude expérimentale chez la souris". Thesis, Bourgogne Franche-Comté, 2018. http://www.theses.fr/2018UBFCK056/document.

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L’alimentation maternelle conditionne la santé métabolique et cardiovasculaire de la progéniture, mais l’impact sur les systèmes sensoriels reste peu étudié. L’olfaction joue un rôle comportemental crucial pour l’évitement des dangers et pour la prise alimentaire. Dès le plus jeune âge, les odeurs participent à l’établissement des préférences alimentaires, qui influencent les habitudes alimentaires de l’adulte. Le système olfactif repose sur le fonctionnement de neurones qui se développent pendant la vie embryonnaire, continuent leur maturation après la naissance et sont continuellement régénérés au cours de la vie. Ces neurones sont en permanence modulés par les facteurs métaboliques. Les patients atteints de troubles métaboliques ont plus de risques de présenter des altérations de la perception des odeurs, et l’exposition à une alimentation de type obésogène ou diabétogène induit des perturbations olfactives chez le rongeur.Le but de cette thèse était d’explorer les effets d’une exposition périnatale au gras et au sucre, à travers l’alimentation maternelle pendant les périodes de préconception, gestation et lactation, sur la physiologie olfactive de souris juvéniles.L’alimentation grasse et sucrée (high-fat high-sucrose, HFHS) a modifié la composition lipidique du lait maternel. La caractérisation métabolique des petits a révélé un surpoids, un excès de masse grasse épididymaire et une hyperleptinémie chez les petits provenant d'une mère sous alimentation HFHS. Les capacités olfactives évaluées lors d'un test de nourriture enfouie et par mesure du comportement de flairage induit par les odeurs étaient altérées chez les petits dont la mère a été exposée à l'alimentation HFHS. Cependant, nos résultats n’ont pas montré de modification de la sensibilité de l’épithélium olfactif (EO) ou de l’expression des gènes codant pour les éléments de la cascade de transduction olfactive. L’exploration du traitement central du signal olfactif a révélé un impact de l’alimentation maternelle HFHS sur le degré de ramification dendritique des interneurones du bulbe olfactif. En revanche, l’activation des neurones dans le cortex piriforme après une exposition odorante n’était pas modifiée.Ainsi, l'alimentation maternelle HFHS pendant les périodes de développement des petits induit chez les mâles des altérations de la perception olfactive, sans perturbation de la détection des odeurs par l'EO, mais associées à des modifications neuronales dans les structures olfactives centrales. La leptine, hormone métabolique connue pour son action sur l’olfaction et sur le développement neuronal, pourrait être à l’origine de ces déficits olfactifs
The influence of maternal diet on progeny’s health has been thoroughly investigated regarding metabolic and cardiovascular diseases, but the impact on sensory systems remains unknown. Olfaction is of great behavioral importance for avoiding hazards and for feeding behavior. In childhood especially, olfaction participates in establishing food preferences, which partly determine adult eating habits. The olfactory system is made of sensory neurons that develop during the embryonic life, pursue their maturation after birth and are continuously regenerated over life. Olfactory neurons activity can be modulated by metabolic factors. Patients with metabolic disorders are at risk of impaired olfactory sensitivity. Adult mice exposed to an obesogenic or diabetogenic diet exhibit disrupted olfactory behavior.The aim of this thesis was to investigate the effect of a perinatal exposure to fat and sugar, through maternal diet during preconception, gestation and lactation, on the olfactory system of young mice.Maternal high fat high sugar (HFHS) diet modified milk lipids composition. When investigating pups’ metabolic phenotype, overweight, increased epididymal fat and hyperleptinemia were revealed in pup’s born from dams fed with the HFHS diet. Olfactory abilities were assessed in a buried food test and by measuring odor-induced sniffing behavior and were disrupted in the progeny of HFHS diet fed dams. However, olfactory epithelium sensitivity and gene expression of constituents of the olfactory transduction cascade were not affected by maternal HFHS diet. When investigating olfactory central processing, dendritic complexity of interneurons in the olfactory bulb was found to be affected by maternal HFHS diet. Meanwhile, neuronal activation in piriform cortex was not altered.These results show that maternal HFHS diet during pups’ development alters olfactory perception in male progeny, without impairing odor detection by the OE, and associated with neuronal modifications in olfactory central areas. Leptin is a metabolic hormone known to influence olfaction and neurons development which could have induced the olfactory defects
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Più fonti

Libri sul tema "Olfactions"

1

Takagi, Sadayuki F. Human olfaction. [Tokyo]: University of Tokyo Press, 1989.

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2

Hansson, Bill S., a cura di. Insect Olfaction. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-662-07911-9.

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3

Serby, Michael J., e Karen L. Chobor, a cura di. Science of Olfaction. New York, NY: Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4612-2836-3.

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4

Breipohl, Winrich, e Raimund Apfelbach, a cura di. Ontogeny of Olfaction. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71576-1.

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5

Hawkes, Christopher H. The neurology of olfaction. Cambridge: Cambridge University Press, 2009.

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6

Dickinson, Keith. Receptor mechanisms in olfaction. [s.l.]: typescript, 1987.

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7

L, Payne Thomas, Birch Martin C e Kennedy C. E. J, a cura di. Mechanisms in insect olfaction. Oxford [Oxfordshire]: Clarendon Press, 1986.

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8

1944-, Rouby Catherine, a cura di. Olfaction, taste, and cognition. Cambridge, U.K: Cambridge University Press, 2002.

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9

Farbman, Albert I. Cell biology of olfaction. Cambridge: Cambridge University Press, 1992.

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10

Kurihara, Kenzo, Noriyo Suzuki e Hisashi Ogawa, a cura di. Olfaction and Taste XI. Tokyo: Springer Japan, 1994. http://dx.doi.org/10.1007/978-4-431-68355-1.

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Capitoli di libri sul tema "Olfactions"

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Zasler, Nathan D. "Olfaction". In Encyclopedia of Clinical Neuropsychology, 2506–9. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_55.

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2

Huart, Caroline, Philippe Eloy e Philippe Rombaux. "Olfaction". In Nasal Physiology and Pathophysiology of Nasal Disorders, 113–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-37250-6_10.

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Zasler, Nathan D. "Olfaction". In Encyclopedia of Clinical Neuropsychology, 1–4. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_55-2.

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Galizia, C. Giovanni, e Pierre-Marie Lledo. "Olfaction". In Neurosciences - From Molecule to Behavior: a university textbook, 253–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-10769-6_13.

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Mozell, Maxwell M. "Olfaction". In Sensory Systems: II, 59–62. Boston, MA: Birkhäuser Boston, 1988. http://dx.doi.org/10.1007/978-1-4684-6760-4_26.

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Bakker, Julie. "Olfaction". In Neuroscience in the 21st Century, 815–37. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1997-6_29.

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Zasler, Nathan D. "Olfaction". In Encyclopedia of Clinical Neuropsychology, 1812–15. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_55.

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Linster, Christiane. "Computational Olfaction". In Encyclopedia of Computational Neuroscience, 773–75. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-6675-8_609.

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Linster, Christiane. "Olfaction: Overview". In Encyclopedia of Computational Neuroscience, 76–78. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-6675-8_786.

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Guthrie, Brian. "Machine Olfaction". In Springer Handbook of Odor, 55–56. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-26932-0_21.

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Atti di convegni sul tema "Olfactions"

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Micaroni, Lorenzo, Marina Carulli, Francesco Ferrise, Monica Bordegoni e Alberto Gallace. "Design of a Directional Olfactory Display to Study the Integration of Vision and Olfaction". In ASME 2018 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/detc2018-85972.

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Abstract (sommario):
This research aims to design and develop an innovative system, based on an olfactory display, to be used for investigating the directionality of the sense of olfaction. In particular, the design of an experimental setup to understand and determine to what extent the sense of olfaction is directional and whether there is prevalence of the sense of vision over the one of smell when determining the direction of an odor, is described. The experimental setup is based on low cost Virtual Reality (VR) technologies. In particular, the system is based on a custom directional olfactory display, an Oculus Rift Head Mounted Display (HMD) to deliver both visual and olfactory cues and an input device to register subjects’ answers. The VR environment is developed in Unity3D. The paper describes the design of the olfactory interface as well as its integration with the overall system. Finally the results of the initial testing are reported in the paper.
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Latorre-Estivalis, José Manuel. "Neuropeptides and olfaction inRhodnius prolixus". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94896.

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Meloni, M. "Olfaction: an indispensable kitchen "utensil"". In Proceedings of the 5th Italian Conference — Extended to Mediterranean Countries. WORLD SCIENTIFIC, 2000. http://dx.doi.org/10.1142/9789812792013_0038.

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Latham, Peter. "Olfaction As Probabilistic Inference – Abstract". In 27th Conference on Modelling and Simulation. ECMS, 2013. http://dx.doi.org/10.7148/2013-0022.

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Pouria, Pouria, Justin Hui, Mahyar Mohaghegh Montazeri, Kim Tien Nguyen, Abigail Logel, Allen O'Brian e Mina Hoorfar. "Smelling Through Microfluidic Olfaction Technology". In 2018 Canadian Society for Mechanical Engineering (CSME) International Congress. York University Libraries, 2018. http://dx.doi.org/10.25071/10315/35359.

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Orsucci, Franco. "Olfaction: new frontiers for cognitive sciences". In Proceedings of the 5th Italian Conference — Extended to Mediterranean Countries. WORLD SCIENTIFIC, 2000. http://dx.doi.org/10.1142/9789812792013_0039.

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Jacob, Rob. "Session details: Olfaction, breath & biofeedback". In CHI '11: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2011. http://dx.doi.org/10.1145/3248972.

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Lee, Alexander B., Thomas L. Spencer, Jasmine Pillarisetti, Matthew Ersted e David L. Hu. "Mimicking sniffing for improving machine olfaction". In 2017 ISOCS/IEEE International Symposium on Olfaction and Electronic Nose (ISOEN). IEEE, 2017. http://dx.doi.org/10.1109/isoen.2017.7968894.

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Webster, Jason, Pratistha Shakya, Eamonn Kennedy, Michael Caplan, Christopoher Rose e Jacob K. Rosenstein. "TruffleBot: Low-Cost Multi-Parametric Machine Olfaction". In 2018 IEEE Biomedical Circuits and Systems Conference (BioCAS). IEEE, 2018. http://dx.doi.org/10.1109/biocas.2018.8584767.

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Knaden, Markus. "Olfaction-guided navigation of the desert antCataglyphis". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93808.

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Rapporti di organizzazioni sul tema "Olfactions"

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Bower, James M., Linda Buck, William Goddard, Wilson III, Lewis Denise e Nathan S. Understanding Olfaction: From Detection to Classification. Fort Belvoir, VA: Defense Technical Information Center, maggio 2004. http://dx.doi.org/10.21236/ada428676.

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Schmeisser, Elmar, Kimberly A. Pollard e Tomasz Letowski. Olfaction Warfare: Odor as Sword and Shield. Fort Belvoir, VA: Defense Technical Information Center, marzo 2013. http://dx.doi.org/10.21236/ada577342.

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Gelperin, Alan, Gary Beauchamp, Beverly Cowart, Pam Dalton, Graeme Lowe, Johan Lundstrom, George Preti, Johannes Reisert, Charles Wysocki e Kunio Yamazaki. Learning and Olfaction: Understanding and Enhancing a Critical Information Channel. Fort Belvoir, VA: Defense Technical Information Center, maggio 2013. http://dx.doi.org/10.21236/ada603587.

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