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Libri sul tema "Neurophysiology"

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Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 1 giugno 2024

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1

Carpenter, R. H. S. Neurophysiology. 4a ed. London: Arnold, 2003.

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2

Blankenship, James E. Neurophysiology. St. Louis: Mosby, 2003.

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3

Carpenter, R. H. S. Neurophysiology. 3a ed. London: Arnold, 1996.

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4

Carpenter, R. H. S. Neurophysiology. 2a ed. London: Edward Arnold, 1991.

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5

Carpenter, R. H. S. Neurophysiology. 2a ed. London: Edward Arnold, 1990.

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6

Simon, Mirela V., a cura di. Intraoperative Neurophysiology. New York, NY: Springer Publishing Company, 2018. http://dx.doi.org/10.1891/9781617052941.

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7

Tranquillo, Joseph V. Quantitative Neurophysiology. Cham: Springer International Publishing, 2009. http://dx.doi.org/10.1007/978-3-031-01628-8.

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8

D, Binnie C., a cura di. Clinical neurophysiology. Amsterdam: Elsevier, 2003.

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9

R, Daube Jasper, a cura di. Clinical neurophysiology. Philadelphia: F.A. Davis, 1996.

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10

Daube, Jasper R., e Devon I. Rubin. Clinical neurophysiology. 3a ed. New York: Oxford University Press, 2009.

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11

Tranquillo, Joseph Vincent. Quantitative neurophysiology. San Rafael, Calif. (1537 Fourth St, San Rafael, CA 94901 USA): Morgan & Claypool Publishers, 2008.

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12

name, No. Clinical neurophysiology. 2a ed. New York: Oxford University Press, 2003.

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13

David, Grundy, e Read N. W, a cura di. Gastrointestinal neurophysiology. London: Baillière Tindall, 1988.

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14

R, Daube Jasper, e Rubin Devon I, a cura di. Clinical neurophysiology. 3a ed. New York: Oxford University Press, 2009.

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15

Galbraith, G. C., M. L. Kietzman e E. Donchin. Neurophysiology and Psychophysiology. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003164647.

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16

Libet, Benjamin. Neurophysiology of Consciousness. Boston, MA: Birkhäuser Boston, 1993. http://dx.doi.org/10.1007/978-1-4612-0355-1.

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17

Schmidt, Robert F., a cura di. Fundamentals of Neurophysiology. New York, NY: Springer New York, 1985. http://dx.doi.org/10.1007/978-1-4613-9553-9.

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18

van Putten, Michel J. A. M. Essentials of Neurophysiology. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-69890-6.

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19

V, Simon Mirela, a cura di. Intraoperative clinical neurophysiology. New York: Demos Medical Pub., 2010.

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20

1938-, Booth D. A., a cura di. Neurophysiology of ingestion. Oxford: Pergamon Press, 1993.

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21

V, Simon Mirela, a cura di. Intraoperative clinical neurophysiology. New York: Demos Medical Pub., 2010.

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22

1932-, Schmidt Robert F., e Dudel Josef, a cura di. Fundamentals of neurophysiology. 3a ed. New York: Springer-Verlag, 1985.

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23

Atwood, Harold L. Essentials of neurophysiology. Toronto: Decker, 1989.

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24

Galloway, Gloria M., a cura di. Clinical Neurophysiology in Pediatrics. New York, NY: Springer Publishing Company, 2015. http://dx.doi.org/10.1891/9781617052118.

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25

Arduini, Arnaldo. Principles of Theoretical Neurophysiology. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71468-9.

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26

Blum, Andrew S., e Seward B. Rutkove, a cura di. The Clinical Neurophysiology Primer. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-271-7.

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27

Armstrong, William E., e Jeffrey G. Tasker, a cura di. Neurophysiology of Neuroendocrine Neurons. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118606803.

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28

Rana, Abdul Qayyum, Ali T. Ghouse e Raghav Govindarajan. Neurophysiology in Clinical Practice. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-39342-1.

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29

Misulis, Karl E. Essentials of clinical neurophysiology. 2a ed. Boston: Butterworth-Heinemann, 1997.

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30

Carpenter, Roger. Neurophysiology. Taylor & Francis Group, 2002.

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31

Newman, P. P. Neurophysiology. Springer Netherlands, 2012.

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32

Brandeis, Daniel, Sandra K. Loo, Grainne McLoughlin, Hartmut Heinrich e Tobias Banaschewski. Neurophysiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0009.

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Abstract (sommario):
Neurophysiology allows us to understand and modulate the neural mechanisms in ADHD with high time- and/or frequency-resolution. These non-invasive methods include electroencephalographic recordings at rest and during tasks, with spontaneous and event-related oscillations and potentials tracking covert processing and transcranial neuromodulation through magnetic or electric fields. The findings indicate consistent cognitive and neural deficits in ADHD related to impaired attention and deficient inhibition. Advanced signal processing and source imaging methods often converge with other imaging approaches. Neurophysiological findings also reveal considerable heterogeneity in ADHD regarding cognitive, affective, and genetic subtypes. This illustrates the importance of dimensional approaches and of pathophysiological mechanisms partly shared with other disorders. Although several potential neurophysiological markers of ADHD have been considered, a clinical use for individual diagnostics and classification is not supported to date. More research should clarify the clinical potential of multivariate multimodal classification and prediction of treatment outcome to advance individualized treatment.
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33

Carpenter, Roger, e Benjamin Reddi. Neurophysiology. CRC Press, 2012. http://dx.doi.org/10.1201/b13510.

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34

Newman, P. P. Neurophysiology. Springer, 2012.

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35

Newman, P. P. Neurophysiology. Springer, 2012.

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36

Carpenter, Roger H. S. Neurophysiology. 4a ed. A Hodder Arnold Publication, 2002.

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37

Stevko, Richard. Neurophysiology. Lulu Press, Inc., 2014.

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38

Carpenter, Roger, e Benjamin Reddi. Neurophysiology 5e. Taylor & Francis Group, 2012.

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39

Manji, Hadi, Seán Connolly, Neil Kitchen, Christian Lambery e Amrish Mehta. Clinical neurophysiology. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199601172.003.0008.

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40

Schaible, Hans-Georg, e Rainer H. Straub. Pain neurophysiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0059.

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Abstract (sommario):
Physiological pain is evoked by intense (noxious) stimuli acting on healthy tissue functioning as a warning signal to avoid damage of the tissue. In contrast, pathophysiological pain is present in the course of disease, and it is often elicited by low-intensity stimulation or occurs even as resting pain. Causes of pathophysiological pain are either inflammation or injury causing pathophysiological nociceptive pain or damage to nerve cells evoking neuropathic pain. The major peripheral neuronal mechanism of pathophysiological nociceptive pain is the sensitization of peripheral nociceptors for mechanical, thermal and chemical stimuli; the major peripheral mechanism of neuropathic pain is the generation of ectopic discharges in injured nerve fibres. These phenomena are created by changes of ion channels in the neurons, e.g. by the influence of inflammatory mediators or growth factors. Both peripheral sensitization and ectopic discharges can evoke the development of hyperexcitability of central nociceptive pathways, called central sensitization, which amplifies the nociceptive processing. Central sensitization is caused by changes of the synaptic processing, in which glial cell activation also plays an important role. Endogenous inhibitory neuronal systems may reduce pain but some types of pain are characterized by the loss of inhibitory neural function. In addition to their role in pain generation, nociceptive afferents and the spinal cord can further enhance the inflammatory process by the release of neuropeptides into the innervated tissue and by activation of sympathetic efferent fibres. However, in inflamed tissue the innervation is remodelled by repellent factors, in particular with a loss of sympathetic nerve fibres.
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41

Knoper, Randall. Literary Neurophysiology. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780192845504.001.0001.

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Abstract (sommario):
Writing about neurophysiology more than a century ago, what were US authors doing? Literary Neurophysiology: Memory, Race, Sex, and Representation in U.S. Writing, 1860–1914 examines their use of literature to experiment with the new materialist psychology, which bore upon their efforts to represent reality and was forging new understandings of race and sexuality. Sometimes they emulated scientific epistemology, allowing their art and conceptions of creativity to be reshaped by it. Sometimes they imaginatively investigated neurophysiological theories, challenging and rewriting scientific explanations of human identity and behavior. By enfolding physiological experimentation into literary inquiries that could account for psychological and social complexities beyond the reach of the laboratory, they used literature as a cognitive medium. Mark Twain, W. D. Howells, and Gertrude Stein come together as they probe the effects on mimesis and creativity of reflex-based automatisms and unconscious meaning-making. Oliver Wendell Holmes explores conceptions of racial nerve force elaborated in population statistics and biopolitics, while W. E. B. Du Bois and Pauline Hopkins contest notions of racial energy used to predict the extinction of African Americans. Holmes explores new definitions of “sexual inversion” as, in divergent ways, Whitman and John Addington Symonds evaluate relations among nerve force, human fecundity, and the supposed grave of nonreproductive sex. Carefully tracing entanglements and conflicts between literary culture and mental science of this period, Knoper reveals unexpected connections among these authors and fresh insights into the science they confronted. Considering their writing as cognitive practice, he provides a new understanding of literary realism.
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42

Applied Neurophysiology. Elsevier, 1988. http://dx.doi.org/10.1016/c2013-0-06464-x.

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43

Clinical Neurophysiology. Elsevier, 1995. http://dx.doi.org/10.1016/c2009-0-49554-0.

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44

Rubin, Devon I., e Jasper R. Daube. Clinical Neurophysiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259631.001.0001.

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Abstract (sommario):
Clinical neurophysiologic testing is an important component of evaluating patients with complaints that may be attributed to diseases of the central or peripheral nervous system. This classic volume in the Contemporary Neurology Series covers the basic concepts underlying each of the testing techniques and provides comprehensive descriptions of the methods and wide range of electrophysiologic testing available for patients with epilepsy, neuromuscular diseases, movement disorders, demyelinating diseases, sleep disorders, autonomic disorders and those undergoing orthopedic and neurosurgical procedures. This text details the role of each study, the interpretation of findings, and their application clinical problems. This text describes the multiple diagnostic procedures for diverse diseases of the neuromuscular system, including: electroencephalography (EEG); electromyography and nerve conduction studies; single fiber EMG; polysomnography; surface EMG patterns, blood pressure, pulse, sweat measures; vestibular function testing; deep brain stimulator physiology; and intraoperative monitoring. It is a practical textbook for neurologists, physiatrists and clinical neurophysiologists in clinical or research practice or in training. Key features of the new edition include fully updated chapters to reflect new research and techniques in clinical neurophysiology; updated images illustrating key elements of techniques and basic concepts; case examples for practical application.
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45

Beheiry, Hossam El. Neurophysiology/Neuroprotection. A cura di David E. Traul e Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0027.

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Abstract (sommario):
Intracerebral hemorrhage (ICH) is a catastrophic event that may lead to severe disability or death. It is associated with major disruption of intracranial neurophysiology and subsequent neuronal tissue injury. The etiology of ICH may be primary (e.g., hypertensive) or secondary (e.g., traumatic). Treatment options include conservative management or neurosurgical intervention in selected patients. In order to manage these challenging cases, the anesthesiologist and the intensivist should have thorough knowledge pertaining to neurophysiologic concepts of cerebral blood flow, cerebral autoregulation, and neuroprotection principles. Additionally, the specialized team managing the patient with ICH should be cognizant of the most recent evidence-based guidelines recommended by the pertinent associations.
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46

Daube, Jasper R., e Devon I. Rubin. Clinical Neurophysiology. Oxford University Press, 2009. http://dx.doi.org/10.1093/med/9780195385113.001.0001.

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Abstract (sommario):
This resource provides a didactic, yet accessible, presentation of electrophysiology that describes the analysis of electrophysiological waveforms, the various methods and techniques of electrophysiological testing, and recommendations of symptom complexes and disease entities using electroencephalography, evoked potentials, and nerve conduction studies.
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47

Carpenter. Ise Neurophysiology. Hodder & Stoughton Educational Division, 1995.

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48

Daube, Jasper R. Clinical Neurophysiology. Oxford University Press, 1996.

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49

Osselton. Clinical Neurophysiology. Butterworth-Heinemann Ltd, 1991.

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50

Levin, Kerry H., e Patrick Chauvel. Clinical Neurophysiology. Elsevier, 2019.

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