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Libri sul tema "Metabolic Skin Diseases"

Autore: Grafiati
Pubblicato: 29 luglio 2024
Ultima modifica: 31 luglio 2024

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1

J, Vermeer B., e European Society for Dermatological Research., a cura di. Metabolic disorders and nutrition correlated with skin. Basel: Karger, 1991.

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2

Smoller, Bruce R., e Franco Rongioletti, a cura di. Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-181-3.

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3

1949-, Schröder Jens-Michael, a cura di. Fatty acids and inflammatory skin diseases. Basel: Birkhäuser Verlag, 1999.

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4

I, Maibach Howard, a cura di. Toxicology of skin. New York: Taylor & Francis, 2000.

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5

Fuchs, Jürgen. Oxidative injury in dermatopathology. Berlin: Springer-Verlag, 1992.

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6

Fuchs, Jürgen, 1957 June 28- e Packer Lester, a cura di. Oxidative stress in dermatology. New York: Dekker, 1993.

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7

Jenifer, Swanson, a cura di. Physical and mental issues in aging sourcebook: Basic consumer health information on physical and mental disorders associated with the aging process, including concerns about cardiovascular disease, pulmonary disease, oral health, digestive disorders, musculoskeletal and skin disorders, metabolic changes, sexual and reproductive issues, and changes in vision, hearing, and other senses; along with data about longevity and causes of death, information on acute and chronic pain, descriptions of mental concerns, a glossary of terms, and resource listings for additional help. Detroit, MI: Omnigraphics, 1999.

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8

Keith, Lierre. The vegetarian myth: Food, justice and sustainability. Crescent City, Ca: Flashpoint Press, 2009.

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9

Smoller, Bruce R., e Franco Rongioletti. Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease. Springer New York, 2016.

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10

Clinical And Pathological Aspects Of Skin Diseases In Endocrine Metabolic Nutritional And Deposition Disease. Springer, 2010.

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11

Smoller, Bruce R., e Franco Rongioletti. Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease. Springer, 2011.

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12

Smoller, Bruce R., e Franco Rongioletti. Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease. Springer, 2010.

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13

Beattie, R. Mark, Anil Dhawan e John W.L. Puntis. Metabolic liver disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0054.

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Abstract (sommario):
Background 390Clinical features 390Investigations 392Clinical presentations of metabolic disorders 394General management 402The diagnostic approach to a child with metabolic liver disease requires a high degree of suspicion, detailed history and physical examination, and extensive blood and urine tests; liver, skin, and muscle biopsy are usually necessary to establish the diagnosis....
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14

Skin Diseases Nutrition and Metabolism. Elsevier, 2013. http://dx.doi.org/10.1016/c2013-0-06725-4.

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15

Urbach, Erich. Skin Diseases Nutrition and Metabolism. Elsevier Science & Technology Books, 2015.

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16

Golper, Thomas A., Andrew A. Udy e Jeffrey Lipman. Drug dosing in acute kidney injury. A cura di William G. Bennett. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0364.

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Drug dosing in acute kidney injury (AKI) is one of the broadest topics in human medicine. It requires an understanding of markedly altered and constantly changing physiology under many disease situations, the use of the drugs to treat those variety of diseases, and the concept of drug removal during blood cleansing therapies. Early in AKI kidney function may be supraphysiologic, while later in the course there may be no kidney function. As function deteriorates other metabolic pathways are altered in unpredictable ways. Furthermore, the underlying disorders that lead to AKI alter metabolic pathways. Heart failure is accompanied by vasoconstriction in the muscle, skin and splanchnic beds, while brain and cardiac blood flow proportionally increase. Third spacing occurs and lungs can become congested. As either kidney or liver function deteriorates, there may be increased or decreased drug sensitivity at the receptor level. Acidosis accompanies several failing organs. Protein synthesis is qualitatively and quantitatively altered. Sepsis affects tissue permeability. All these abnormalities influence drug pharmacokinetics and dynamics. AKI is accompanied by therapeutic interventions that alter intrinsic metabolism which is in turn complicated by kidney replacement therapy (KRT). So metabolism and removal are both altered and constantly changing. Drug management in AKI is exceedingly complex and is only beginning to be understood. Thus, we approach this discussion in a physiological manner. Critically ill patients pass through phases of illness, sometimes rapidly, other times slowly. The recognition of the phases and the need to adjust medication administration strategies is crucial to improving outcomes. An early phase involving supraphysiologic kidney function may be contributory to therapeutic failures that result in the complication of later AKI and kidney function failure.
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17

Takeshita, Junko, e Joel M. Gelfand. Epidemiology of psoriasis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0002.

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Abstract (sommario):
Psoriasis is a common chronic inflammatory disorder of the skin that is associated with multisystem effects. Approximately 125 million people worldwide are affected by psoriasis, nearly one quarter of whom have moderate to severe disease. The majority of patients with psoriasis have a waxing and waning course with variable periods of spontaneous disease improvement or clearance. A rapidly expanding body of epidemiologic literature suggests psoriasis to be associated with a greater comorbid disease burden than patients without psoriasis. In addition to psoriatic arthritis, cardiometabolic diseases, including metabolic syndrome and its component disorders, as well as major adverse cardiovascular events are the most common comorbidities of psoriasis; together they are the primary cause of premature mortality among moderate to severe psoriasis patients. Continued efforts to better understand currently known and identify other emerging comorbidities of psoriasis are critical.
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18

Born, Gustav V. R., e Martin G. M\xf6hrle. Pharmacology of the Skin II: Methods, Absorption, Metabolism and Toxicity, Drugs and Diseases. Springer, 2012.

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19

Maibach, Howard I., e Jacob P. Thyssen. Filaggrin: Basic Science, Epidemiology, Clinical Aspects and Management. Springer London, Limited, 2014.

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20

Maibach, Howard I., e Jacob P. Thyssen. Filaggrin: Basic Science, Epidemiology, Clinical Aspects and Management. Springer, 2016.

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21

Maibach, Howard I., e Jacob P. Thyssen. Filaggrin: Basic Science, Epidemiology, Clinical Aspects and Management. Springer, 2014.

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22

Toxicology of skin. Philadelphia, PA: Taylor & Francis, 2001.

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23

Waldek, Stephen. Fabry disease. A cura di Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0335_update_001.

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Abstract (sommario):
Fabry disease is a rare X-linked disorder of glycosphingolipid metabolism caused by a deficiency of the lysosomal acid hydrolase enzyme, alpha-galactosidase A. The resulting accumulation of substrate, mostly globotriaosylceramide, leads to a progressive, multiorgan disease affecting predominantly the kidneys, skin, heart, and nervous system. It is one of over 50 lysosomal storage diseases. It is typically diagnosed in young men after many years of ‘acral pain’ syndrome, when the diagnosis is made through identification of characteristic abnormalities of skin, kidney or heart, or of other organs. Renal failure has been a common outcome. Females may also develop manifestations, usually later in life. Renal biopsy shows vacuoles/deposits in podocytes and other renal cell types with progressive scarring. The diagnosis can be made by measuring enzyme levels in men, or by genetic testing. This latter is the more reliable test in women. Fabry disease can now be treated where affordable by regular (every 2 weeks) intravenous infusions of recombinant preparations of the deficient enzyme. These are burdensome and expensive, but are transforming the outlook for the condition.
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24

Pharmacology of the Skin II: Methods, Absorption, Metabolism and Toxicity, Drugs and Diseases. Springer, 2012.

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25

Atherton, D. J. Pharmacology of the Skin II: Methods, Absorption, Metabolism and Toxicity, Drugs and Diseases. Springer, 2012.

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26

Maibach, Howard I. Toxicology of the Skin (Target Organ Toxicology Series). CRC, 2001.

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27

Melanins and melanogenesis. San Diego: Academic Press, 1992.

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28

Khavandi, Kaivan, Halima Amer, Sarah Withers e Behdad Afzali. Pleiotropic effects of vitamin D. A cura di David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0127.

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Abstract (sommario):
Vitamin D is a fat-soluble steroid pro-hormone integral to physiological health, fulfilling a central role in skeletal mineralization, bone metabolism, and immune biology. Although vitamin D is synthesized photochemically in the skin and some is absorbed from dietary sources, vitamin D insufficiency and deficiency are very common. Epidemiological studies have demonstrated a strong association between vitamin D and kidney and heart disease, and some supplementation studies have suggested that repletion may prevent and/or ameliorate cardiorenal injury. This chapter focuses on vitamin D biology and discusses the many associations of vitamin D perturbation with diseases of humans.
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29

The Biological Role of Proteinases and their Inhibitors in Skin. Elsevier Science Publishing Company, 1986.

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30

Greaves, Malcolm W. Pharmacology of the Skin II: Methods, Absorption, Metabolism and Toxicity, Drugs and Diseases (Handbook of Experimental Pharmacology). Springer, 1989.

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31

Pediatric ICD-10-CM. American Academy of Pediatrics, 2015. http://dx.doi.org/10.1542/9781581109016.

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Abstract (sommario):
In this first edition, Pediatric ICD-10-CM: A Manual for Provider-Based Coding strives to bring to the pediatric provider, coder, and biller the most accurate and easy-to use manual on ICD-10-CM yet. Composed entirely with a pediatrics focus, this manual exclusively features codes and guidelines for physician- and provider-based coding, all in a simplified yet familiar format. The full draft of the ICD-10-CM code set comes in at well over 1,000 pages. This book condenses that large and potentially cumbersome book into 400 pages of the most relevant,pediatrics-related codes and guidelines. It also fully integrates into the tabular list specific chapter and code guidelines. Guideline are now included directly at the chapter and code level, ensuring that coders will always use the right codes in right circumstances Features include Integrated codes and guidelines Simplified yet familiar layout Tabular and indexed navigation Pediatric-focused and provider-based guidance And more... Contents Include: ICD-10-CM Official Guidelines for Coding and Reporting: FY 2015 Certain Infectious and Parasitic Diseases (A00-B99) Neoplasms (C00-D49) Diseases of the Blood and Blood-Forming Organs and Certain Disorders Involving the Immune Mechanism (D50-D89) Endocrine, Nutritional and Metabolic Diseases (E00-E89) Mental, Behavioral and Neurodevelopmental Disorders (F01-F99) Diseases of the Nervous System (G00-G99) Diseases of the Eye and Adnexa (H00-H59) Diseases of the Ear and Mastoid Process (H60-H95) Diseases of the Circulatory System (I00-I99) Diseases of the Respiratory System (J00-J99) Diseases of the Digestive System (K00-K95) Diseases of the Skin and Subcutaneous Tissue (L00-L99) Diseases of the Musculoskeletal System and Connective Tissue (M00-M99) Diseases of the Genitourinary System (N00-N99) Pregnancy, Childbirth, Certain Conditions Originating in the Perinatal Period (P00-P99) Congenital Malformations, Deformations and Chromosomal Abnormalities (Q00-Q99) Symptoms, Signs, and Abnormal Clinical and Laboratory Findings (R00-R99) Injury, Poisoning and Consequences of Certain Other External Causes (S00-T88) External Causes of Morbidity (V00-Y99) Factors Influencing Health Status and Contact With Health Services (Z00-Z99)
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32

Kragballe, Knud. Vitamin D in Dermatology. Informa Healthcare, 2000.

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33

Bioengineering of the skin: Water and stratum corneum. 2a ed. Boca Raton: CRC Press, 2005.

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34

(Editor), Joachim W. Fluhr, Peter Elsner (Editor), Enzo Berardesca (Editor) e Howard I. Maibach (Editor), a cura di. Bioengineering of the Skin: Water and the Stratum Corneum, 2nd Edition (Dermatology: Clinical & Basic Science). 2a ed. Informa Healthcare, 2004.

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35

Groothoff, Jaap W. Primary Hyperoxaluria. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0065.

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Abstract (sommario):
Primary hyperoxalurias (PH) are rare autosomal recessive metabolic disorders characterized by an increased endogenous oxalate production which leads to the development of urolithiasis, nephrocalcinosis, and ultimately to renal failure.PH patients with severe renal failure develop life-threatening systemic oxalosis, which affects many organs such as bone, skin, retina, myocardium, vessel walls, and the central nervous system. So far, combined or sequential liver-kidney transplantation is the only therapeutic option for patients with advanced disease. Contrary to the former impression of a relatively mild course of disease in patients diagnosed as adults, recent data have shown that patients diagnosed in adulthood mostly present with established ESRD and systemic oxalosis. The fact that some of these patients respond to pyridoxine therapy underlines the importance of early diagnosis and measures to prevent renal failure and systemic oxalosis. All children with stone disease or nephrocalcinosis and all adults with recurrent stone disease should therefore be screened for PH.
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36

Mutyambizi, Kudakwashe. Dermatologic Complications. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0034.

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The hallmark of HIV infection is immune dysregulation and immunosuppression. As the immune system deteriorates, inflammatory dermatoses, metabolic dysregulation, adverse drug reactions, opportunistic infections, and cutaneous malignancies become more common, atypical in presentation, and recalcitrant to therapy. Both acute and chronic skin complaints contribute significantly to reduced quality of life for HIV patients. The Centers for Disease Control and Prevention recommends that individuals between ages 13 and 64 years be tested for HIV at least once in their lifetime, with increased screening of high-risk individuals and testing based on symptoms. The presence of dermatoses uncommon in the general population but concentrated in the HIV population, or dermatoses strikingly recalcitrant to therapy, should warrant suspicion and testing for HIV.
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37

Kragballe, Knud. Vitamin D in Dermatology. Taylor & Francis Group, 2000.

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38

Deegan, Patrick. Porphyria. A cura di Patrick Davey e David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0179.

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This chapter discusses six diseases caused by inborn errors of metabolism affecting the biosynthesis of haem. Haem is a tetracyclic metal-binding compound involved in oxygen transport (in haemoglobin and myoglobin) and redox reactions (e.g. in the cytochrome P450 system). Each of these conditions is caused by a single gene defect in one of the enzymes involved in the biosynthesis of haem. Inheritance is usually autosomal dominant with incomplete penetrance. The enzyme defect results in disease, not as a result of deficiency of the reaction product, but as a result of accumulation of precursors. Early, soluble precursors, 5-aminolaevulinic acid, and porphobilinogen (not porphyrins as such) are neurotoxic and, when present in great excess, as occurs when flux through the haem synthetic pathway is increased in response to particular medications or hormones, lead to acute neurovisceral crises. Later cyclical precursors (porphyrins) in the pathway are also water soluble and excreted in urine, but are susceptible to activation by electromagnetic radiation in the visible spectrum and are converted to free-radical metabolites that cause pain, inflammation, and tissue damage in the skin. The final haem precursors (also porphyrins) are hydrophobic and excreted in the bile and faeces and are also activated by light to toxic metabolites.
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39

Denton, Nathan. Waisted: The Biology of Body Fat. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198865278.001.0001.

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Abstract (sommario):
Waisted outlines the fascinating and misunderstood biology of fat (i.e., adipose tissue). This controversial, much-maligned organ plays a crucial yet curiously overlooked role in the global obesity crisis currently wreaking havoc on the world’s healthcare systems and economies. Attaining a better appreciation of the biology of fat, its social meanings, and how these intersect is essential for improving the world’s physical and mental health. Far from being a passive layer of blubber under the skin, fat plays a highly dynamic role in energy metabolism, reproductive health, and immunity, with these links having ancient origins in the evolution of modern humanity. Waisted provides a comprehensive, evidence-based perspective on the biology of fat and its crucial role in human evolution, health, disease, and society. Waisted draws upon biomedical, epidemiological, and evolutionary research to understand adipose tissue biology and the striking relationship between body fat distribution and health outcomes. Waisted demonstrates the practical implications of key conceptual points through relatable real-world cases and highlights how seemingly disparate common and rare diseases may be underpinned by adipose tissue dysfunction. Overall, Waisted covers a wide breadth of material that challenges and reframes the generally negative perspective of fat to highlight the underappreciated importance of adipose tissue biology in humans.
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40

Coates, Laura C., e Philip S. Helliwell. Psoriatic arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0114.

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Abstract (sommario):
Psoriasis is a chronic skin condition affecting about 3% of Europeans and North Americans. About 15% of people afflicted with psoriasis will develop psoriatic arthritis—cutaneous risk factors for this are psoriasis of the nails, scalp, and flexures. Since most cases of arthritis develop in people with psoriasis, new screening tools, both clinical and imaging, are available. Some genetic factors may also explain susceptibility and severity. Historically, five clinical subgroups have been described but these may be simplified to axial and peripheral involvement, the latter dividing into oligo- and polyarticular patterns. The importance of these clinical subdivisions is still under debate and research but it is clear that there is marked heterogeneity in all manifestations of this disease. In recent times the importance of extra-articular features has gained prominence such that the metabolic syndrome and cardiovascular morbidity are now seen as important features of 'psoriatic disease'. The diverse changes seen in bone on imaging reflect both the underlying pathogenic mechanisms and the ways in which the disease progresses. Recent work with animal models and immunohistochemistry has further advanced our understanding of these features. In the biologic era renewed interest in psoriatic arthritis has stimulated research into outcome assessment and permitted clearer understanding of how these new drugs work on the different aspects of the disease. In addition, improved recognition of the impact of the disease on the person has stimulated the development of new patient-reported outcome tools.
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41

Coates, Laura C., e Philip S. Helliwell. Psoriatic arthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0114_update_003.

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Abstract (sommario):
Psoriasis is a chronic skin condition affecting about 3% of Europeans and North Americans. About 15% of people afflicted with psoriasis will develop psoriatic arthritis—cutaneous risk factors for this are psoriasis of the nails, scalp, and flexures. Since most cases of arthritis develop in people with psoriasis, new screening tools, both clinical and imaging, are available. Some genetic factors may also explain susceptibility and severity. Historically, five clinical subgroups have been described but these may be simplified to axial and peripheral involvement, the latter dividing into oligo- and polyarticular patterns. The importance of these clinical subdivisions is still under debate and research but it is clear that there is marked heterogeneity in all manifestations of this disease. In recent times the importance of extra-articular features has gained prominence such that the metabolic syndrome and cardiovascular morbidity are now seen as important features of ’psoriatic disease’. The diverse changes seen in bone on imaging reflect both the underlying pathogenic mechanisms and the ways in which the disease progresses. Recent work with animal models and immunohistochemistry has further advanced our understanding of these features. In the biologic era renewed interest in psoriatic arthritis has stimulated research into outcome assessment and permitted clearer understanding of how these new drugs work on the different aspects of the disease. In addition, improved recognition of the impact of the disease on the person has stimulated the development of new patient-reported outcome tools.
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42

Barsoum, Rashad S. Schistosomiasis. A cura di Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0182_update_001.

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AbstractSchistosomiasis is a parasitic disease that affects millions of people in 78 countries, where it is held responsible for considerable morbidity and mortality. It is caused by a blood fluke, which provokes an immunological response to hundreds of its antigens. This induces multi-organ pathology through the formation of tissue granulomata or circulating immune complexes. In addition, it is amyloidogenic and carcinogenic, through the interaction of immunological perturbation with confounding metabolic and genetic factors. The primary targets of schistosomiasis are urinary and hepatointestinal.The lower urinary tract is mainly affected in S. haematobium infection, and may lead to chronic pyelonephritis and/or obstructive nephropathy. The colon and liver are the targets of S. mansoni and S. japonicum infection, leading to hepatic fibrosis, portal hypertension, and liver failure. S. mansoni may also lead to immune complex glomerulonephritis, which is discussed elsewhere. Both S. haematobium and S. mansoni ova may be carried with the venous circulation to the lungs, where they provoke granulomatous and immune-mediated endothelial injury leading to cor-pulmonale. Ova may be subsequently carried with the arterial circulation to form ‘metastatic’ granulomas in other tissues, notably the brain (S. japonicum), spinal cord (S. haematobium), skin, conjunctiva, and genital organs.Schistosomiasis is preventable. World Health Organization programmes have successfully eradicated or reduced the incidence of infection in many countries, particularly Egypt and China. Prevention strategies include health education, raising hygiene standards, and interruption of the parasite’s life cycle by snail control and mass treatment. The search for a vaccine continues. Effective antiparasitic treatment is now possible with high elimination rates. Available agents include praziquantel and artemether for all species, metrifonate for S. haematobium, and oxamniquine for S. mansoni. Successful outcome correlates with early intervention, before fibrosis has occurred.
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43

Gonder, Ulrike, a cura di. Ethisch Essen mit Fleisch: Eine Streitschrift über nachhaltige und ethische Ernährung mit Fleisch und die Missverständnisse und Risiken einer streng vegetarischen und veganen Lebensweise. Riva, 2021.

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44

El mito vegetariano: Comida, Justicia, Sostenibilidad. Capitán Swing Libros, 2018.

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45

Le Mythe végétarien: Nourriture, justice et pérennité. Les Editions Pilule Rouge, 2013.

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46

Keith, Lierre. Vegetarian Myth: Food, Justice, and Sustainability. PM Press, 2010.

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47

El Mito Vegetariano: Alimento, justicia y sustentabilidad. FisicalBook, 2012.

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48

Keith, Lierre. Vegetarian Myth: Food, Justice, and Sustainability. PM Press, 2009.

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49

Keith, Lierre. The Vegetarian Myth: Food, Justice, and Sustainability. ReadHowYouWant, 2013.

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