Bibliografie tematiche / Jprom

Letteratura scientifica selezionata sul tema "Jprom"

Autore: Grafiati

Pubblicato: 25 luglio 2024

Ultima modifica: 30 luglio 2024

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Articoli di riviste sul tema "Jprom"

Calvete, Juan J. "JPROT=∑3Y>1017m5.074IF2Tu". Journal of Proteomics 74, n. 10 (settembre 2011): 1827–28. http://dx.doi.org/10.1016/j.jprot.2011.06.030.

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Thomson, Scott C., Ali Kashkouli e Prabhleen Singh. "Glucagon-like peptide-1 receptor stimulation increases GFR and suppresses proximal reabsorption in the rat". American Journal of Physiology-Renal Physiology 304, n. 2 (15 gennaio 2013): F137—F144. http://dx.doi.org/10.1152/ajprenal.00064.2012.

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Abstract (sommario):

The incretin hormone glucagon-like peptide-1 (GLP-1) is released from the gut in response to fat or carbohydrate and contributes to negative feedback control of blood glucose by stimulating insulin secretion, inhibiting glucagon, and slowing gastric emptying. GLP-1 receptors (GLP-1R) are also expressed in the proximal tubule, and possibly elsewhere in the kidney. Presently, we examined the effect of a GLP-1R agonist on single-nephron glomerular filtration rate (GFR; SNGFR), proximal reabsorption ( Jprox), tubuloglomerular feedback (TGF) responses, and urine flow rate in hydropenic male Wistar and Wistar-Froemter rats. Micropuncture and whole-kidney data were obtained before and during infusion of the GLP-1 agonist exenatide (1 nmol/h iv). SNGFR and Jprox were measured by late proximal collection at both extremes of TGF activation, which was achieved by perfusing Henle's loop at 0 or 50 nl/min. Primary changes in Jprox were revealed by analysis of covariance for Jprox with SNGFR as a covariate. Effects on TGF activation were determined in a separate set of experiments by comparing early distal and late proximal collections. Exenatide increased SNGFR by 33–50%, suppressed proximal tubular reabsorption by 20–40%, doubled early distal flow rate, and increased urine flow rate sixfold without altering the efficiency of glomerulotubular balance, TGF responsiveness, or the tonic influence of TGF. This implies that exenatide is both a proximal diuretic and a renal vasodilator. Since the natural agonist for the GLP-1R is regulated by intake of fat and carbohydrate, but not by salt or fluid, the control of salt excretion by the GLP-1R system departs from the usual negative-feedback paradigm for regulating salt balance.

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Thomson, Scott C., Timo Rieg, Cynthia Miracle, Hadi Mansoury, Jean Whaley, Volker Vallon e Prabhleen Singh. "Acute and chronic effects of SGLT2 blockade on glomerular and tubular function in the early diabetic rat". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 302, n. 1 (gennaio 2012): R75—R83. http://dx.doi.org/10.1152/ajpregu.00357.2011.

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Tubuloglomerular feedback (TGF) stabilizes nephron function from minute to minute and adapts to different steady-state inputs to maintain this capability. Such adaptation inherently renders TGF less efficient at buffering long-term disturbances, but the magnitude of loss is unknown. We undertook the present study to measure the compromise between TGF and TGF adaptation in transition from acute to chronic decline in proximal reabsorption (Jprox). As a tool, we blocked proximal tubule sodium-glucose cotransport with the SGLT2 blocker dapagliflozin in hyperglycemic rats with early streptozotocin diabetes, a condition in which a large fraction of proximal fluid reabsorption owes to SGLT2. Dapagliflozin acutely reduced proximal reabsorption leading to a 70% increase in early distal chloride, a saturated TGF response, and a major reduction in single nephron glomerular filtration rate (SNGFR). Acute and chronic effects on Jprox were indistinguishable. Adaptations to 10–12 days of dapagiflozin included increased reabsorption by Henle's loop, which caused a partial relaxation in the increased tone exerted by TGF that could be explained without desensitization of TGF. In summary, TGF contributes to long-term fluid and salt balance by mediating a persistent decline in SNGFR as the kidney adapts to a sustained decrease in Jprox.

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Miracle, Cynthia M., Timo Rieg, Hadi Mansoury, Volker Vallon e Scott C. Thomson. "Ornithine decarboxylase inhibitor eliminates hyperresponsiveness of the early diabetic proximal tubule to dietary salt". American Journal of Physiology-Renal Physiology 295, n. 4 (ottobre 2008): F995—F1002. http://dx.doi.org/10.1152/ajprenal.00491.2007.

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Heightened sensitivity of the diabetic proximal tubule to dietary salt leads to a paradoxical effect of salt on glomerular filtration rate (GFR) via tubuloglomerular feedback. Diabetic hyperfiltration is a feedback response to growth and hyperreabsorption by the proximal tubule. The present studies were performed to determine whether growth and hyperfunction of the proximal tubule are essential for its hyperresponsiveness to dietary salt and, hence, to the paradoxical effect of dietary salt on GFR. Micropuncture was performed in four groups of inactin-anesthetized Wistar rats after 10 days of streptozotocin diabetes drinking tap water or 1% NaCl. Kidney growth was suppressed with ornithine decarboxylase (ODC) inhibitor, DFMO (200 mg·kg−1·day−1), or placebo. Single nephron GFR (SNGFR) was manipulated by perfusing Henle's loop so that proximal reabsorption ( Jprox) could be expressed as a function of SNGFR in each nephron, dissociating primary effects on the tubule from the effects of glomerulotubular balance. Alone, DFMO or high salt reduced SNGFR and suppressed Jprox independent of SNGFR. Suppression of Jprox was eliminated and SNGFR increased when high salt was given to rats receiving DFMO. ODC is necessary for hyperresponsiveness of the proximal tubule to dietary salt and for the paradoxical effect of dietary salt on GFR in early diabetes. This coupling of effects adds to the body of evidence that feedback from the proximal tubule is the principal governor of glomerular filtration in early diabetes.

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Singh, Prabhleen, Aihua Deng, Roland C. Blantz e Scott C. Thomson. "Unexpected effect of angiotensin AT1 receptor blockade on tubuloglomerular feedback in early subtotal nephrectomy". American Journal of Physiology-Renal Physiology 296, n. 5 (maggio 2009): F1158—F1165. http://dx.doi.org/10.1152/ajprenal.90722.2008.

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After subtotal nephrectomy (STN), the remaining nephrons engage in hyperfiltration, which may be facilitated by a reduced sensitivity of the tubuloglomerular feedback (TGF) response to increased distal delivery. However, a muted TGF response would contradict the notion of remnant kidney as a prototype of angiotensin II (ANG II) excess, since ANG II normally sensitizes the TGF response and stimulates proximal reabsorption. We examined the role of ANG II as a modulator of TGF and proximal reabsorption in 7 days after STN in male rats. Single-nephron glomerular filtration rate (SNGFR) and proximal reabsorption ( Jprox) were measured in late proximal collections while perfusing Henle's loop for minimal and maximal TGF stimulation in rats treated with the angiotensin type 1 (AT1) receptor blocker losartan or placebo in drinking water for 7 days. Perfusion of Henle's loop yielded a robust TGF response in sham-operated rats. In STN, the feedback responses were highly variable and nil, on average. Paradoxical TGF responses to augmented late proximal flow were confirmed in SNGFR measurements from the early distal nephron. Chronic losartan treatment normalized the average TGF response without reducing the variability. Jprox was subtly affected by chronic losartan in sham surgery or STN, after controlling for differences in SNGFR. However, when administered acutely into the early S1 segment, losartan potently suppressed Jprox in STN and sham-operated rats alike. Chronic losartan stabilizes the TGF system in remnant kidneys. This cannot be explained by currently known actions of AT1 receptors but is commensurate with a salutary effect of an intact TGF system on dynamic autoregulation of intraglomerular flow and pressure.

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Castillo, Jonathan, Judy K. Thibadeau, Tim Brei e Heidi Castillo. "From prenatal care to spina bifida related mortality: The lifespan is marked by transitions experienced by increasing immigrant and international populations". Journal of Pediatric Rehabilitation Medicine 16, n. 4 (26 dicembre 2023): 581–82. http://dx.doi.org/10.3233/prm-239020.

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Whether it is for collaboration on folic acid fortification or the standardization of care efforts concerning neurogenic bowel dysfunction, a global forum on neural tube defects related issues is needed. Propitiously, the 2023 Spina Bifida World Congress sponsored by the Spina Bifida Association (SBA) was a catalyst for transnational dialog in the field of spina bifida (SB) research. Concurrently, the Journal of Pediatric Rehabilitation Medicine (JPRM) provides a platform for both international research as well as numerous clinical and educational projects, such as The Lifespan Bowel Management Protocol, and social interventions taught through the American Academy of Pediatrics’ Spina Bifida Transition ECHO. Through this open access issue, work by colleagues in Ethiopia, the Nordic countries, and Switzerland, as well as among other transnational populations is highlighted. The development of the Spina Bifida Global Learning Collaborative is also showcased, representing a training initiative across four continents. Correspondingly in this issue, JPRM published an update to the Transition Guidelines for the Care of People with Spina Bifida. The clinical guidelines are a product of the SBA Collaborative Care Network cooperative agreement with the National Center on Birth Defects and Developmental Disabilities in the Centers for Disease Control and Prevention. While colleagues across the globe remain committed to native, immigrant, and displaced populations of individuals affected by SB, JPRM will continue to distribute premier research in multidisciplinary care, education, and advocacy.

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Kim, Heakyung, Christopher Raffi Najarian, Justin W. Ramsey e Sruthi Pandipati Thomas. "JPRM vol. 17 issue 1 Opening Editorial". Journal of Pediatric Rehabilitation Medicine 17, n. 1 (26 marzo 2024): 1. http://dx.doi.org/10.3233/prm-249002.

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Deng, Aihua, e Scott C. Thomson. "Renal NMDA receptors independently stimulate proximal reabsorption and glomerular filtration". American Journal of Physiology-Renal Physiology 296, n. 5 (maggio 2009): F976—F982. http://dx.doi.org/10.1152/ajprenal.90391.2008.

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N-methyl-d-aspartate receptors (NMDA) are expressed in the kidney, where little is known of their functional role. Several series of micropuncture experiments were performed in hydropenic rats using the NMDA channel blocker, MK801, and the NMDA coagonist, l-glycine, to probe NMDA for effects on single-nephron glomerular filtration rate (SNGFR) and proximal reabsorption ( Jprox). During intravenous infusion of MK801 or l-glycine, Henle's loop was perfused to manipulate SNGFR via tubuloglomerular feedback (TGF), thereby facilitating analysis of glomerulotubular balance. To confirm local actions on the kidney, MK801 was delivered to the glomerulus by microperfusion past the macula densa and to the proximal tubule by microperfusion into the early S1 segment. By all measures, MK801 acted on the glomerulus to reduce SNGFR, and acted on the proximal tubule to suppress Jprox, while having no effect on the responsiveness of TGF. l-Glycine raised SNGFR, dampened the TGF response, and could not be proved to independently stimulate proximal reabsorption. NMDA exerts a tonic vasodilatory influence on the glomerulus and a proreabsorptive effect on the proximal tubule. These combined effects allow NMDA to modulate SNGFR with minimal impact on late proximal flow. The full effects of l-glycine infusion on proximal tubule and TGF response do not extrapolate from the response to NMDA blockade.

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Kim, Jung Myoung, e Chan Su Park. "A Comparative Study on the Utilization of Publications metadata in Korean and Japan". Korean Publishing Science Society 109 (31 dicembre 2022): 5–29. http://dx.doi.org/10.21732/skps.2022.109.5.

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The necessity of informatization of Korea's publishing distribution system has been shared by the publishing industry since the 1990s, but it has not changed much. Then, after the bankruptcy of the Songin book in 2017, discussions began from the 5-year plan (2017~2021) for the promotion of the publishing culture industry. On this study is to compare and analyze the cases of Korea's Publication Distribution Integrated Network and Japan Publication Registry Office(JPRO), and to suggest the differences between the two countries and what should be discussed in Korea. JPRO is established and operated in collaboration with publishers and bookstores, and bibliographic information registration is operated for a fee. Japan provides readers with scheduled publication schedules for paper books, e-books, and audiobooks, and Korea provides publishing industry statistics and publishing checkout book statistics. In the future, Korea's publication distribution network necessary to revise the law in accordance with the practical linkage with the ISBN, paid for publication metadata registration fee, and discussion on public-private operation of publication distribution integrated computer network.

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Uchida, Ken-ichi, Hiroto Adachi, Takashi Kikkawa, Akihiro Kirihara, Masahiko Ishida, Shinichi Yorozu, Sadamichi Maekawa e Eiji Saitoh. "Corrections to “Thermoelectric Generation Based on Spin Seebeck Effects” [DOI: 10.1109/JPROC.2016.2535167]". Proceedings of the IEEE 104, n. 7 (luglio 2016): 1499. http://dx.doi.org/10.1109/jproc.2016.2577478.

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Più fonti

Tesi sul tema "Jprom"

Vollmann, Christian [Verfasser], olker [Akademischer Betreuer] Schulz, Leonhard [Akademischer Betreuer] Frerick, Volker [Gutachter] Schulz, Max [Gutachter] Gunzburger e Martin [Gutachter] Siebenborn. "Nonlocal models with truncated interaction kernels - analysis, finite element methods and shape optimization / Christian Vollmann ; Gutachter: Prof. Dr. Volker Schulz, Prof. Dr. Max Gunzburger, JProf. Dr. Martin Siebenborn ; Prof. Dr. Volker Schulz, Prof. Dr. Leonhard Frerick". Trier : Universität Trier, 2019. http://d-nb.info/1203837046/34.

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