Letteratura scientifica selezionata sul tema "Intraplaque neovascularization"

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Articoli di riviste sul tema "Intraplaque neovascularization"

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Koole, Dave, Jan Heyligers, Frans L. Moll, and Gerard Pasterkamp. "Intraplaque neovascularization and hemorrhage." Journal of Cardiovascular Medicine 13, no. 10 (2012): 635–39. http://dx.doi.org/10.2459/jcm.0b013e3283590cd2.

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Akkus, Zeynettin, Gerard van Burken, Stijn C. H. van den Oord, et al. "Carotid Intraplaque Neovascularization Quantification Software (CINQS)." IEEE Journal of Biomedical and Health Informatics 19, no. 1 (2015): 332–38. http://dx.doi.org/10.1109/jbhi.2014.2306454.

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Evdokimenko, A. N., A. O. Chechetkin, L. D. Druina, and M. M. Tanashyan. "Contrast-enhanced ultrasonography for assessing neovascularization of carotid atherosclerotic plaque." Exosomes, no. 4 (July 31, 2019): 24–31. http://dx.doi.org/10.24075/brsmu.2019.057.

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Abstract (sommario):
Neovascularization of a carotid atherosclerotic plaque (AP) is associated with an increased risk of stroke. Contrast-enhanced ultrasonography (CEUS) is a widely used method for imaging intraplaque neovascularization in vivo. Unfortunately, there are no standardized guidelines for CEUS interpretation. The aim of this study was to identify the most reliable method for CEUS-based assessment of AP neovascularization. Seventy-eight AP were removed during carotid endarterectomy in 73 patients, of whom 5 had AP on both sides, and examined morphologically. All patients underwent preoperative duplex sc
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Ugusman, Azizah, Nur Syahidah Nor Hisam, Nur Syakirah Othman, et al. "Pharmacological interventions for intraplaque neovascularization in atherosclerosis." Pharmacology & Therapeutics 261 (September 2024): 108685. http://dx.doi.org/10.1016/j.pharmthera.2024.108685.

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Perrotta, Paola, Bieke Van der Veken, Pieter Van Der Veken, et al. "Partial Inhibition of Glycolysis Reduces Atherogenesis Independent of Intraplaque Neovascularization in Mice." Arteriosclerosis, Thrombosis, and Vascular Biology 40, no. 5 (2020): 1168–81. http://dx.doi.org/10.1161/atvbaha.119.313692.

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Abstract (sommario):
Objective: Intraplaque neovascularization is an important feature of unstable human atherosclerotic plaques. However, its impact on plaque formation and stability is poorly studied. Because proliferating endothelial cells generate up to 85% of their ATP from glycolysis, we investigated whether pharmacological inhibition of glycolytic flux by the small-molecule 3PO (3-[3-pyridinyl]-1-[4-pyridinyl]-2-propen-1-one) could have beneficial effects on plaque formation and composition. Approach and Results: ApoE −/ − (apolipoprotein E deficient) mice treated with 3PO (50 µg/g, ip; 4×/wk, 4 weeks) show
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Filis, Konstantinos, Levon Toufektzian, George Galyfos, et al. "Assessment of the vulnerable carotid atherosclerotic plaque using contrast-enhanced ultrasonography." Vascular 25, no. 3 (2016): 316–25. http://dx.doi.org/10.1177/1708538116665734.

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Abstract (sommario):
Carotid atherosclerosis represents a primary cause for cerebrovascular ischemic events and its contemporary management includes surgical revascularization for moderate to severe symptomatic stenoses. However, the role of invasive therapy seems to be questioned lately for asymptomatic cases. Numerous reports have suggested that the presence of neovessels within the atherosclerotic plaque remains a significant vulnerability factor and over the last decade imaging modalities have been used to identify intraplaque neovascularization in an attempt to risk-stratify patients and offer management guid
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Schinkel, Arend F. L., Johan G. Bosch, Daniel Staub, Dan Adam, and Steven B. Feinstein. "Contrast-Enhanced Ultrasound to Assess Carotid Intraplaque Neovascularization." Ultrasound in Medicine & Biology 46, no. 3 (2020): 466–78. http://dx.doi.org/10.1016/j.ultrasmedbio.2019.10.020.

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Ichibori, Yasuhiro, Daisaku Nakatani, Yasushi Sakata, et al. "Cardiac Allograft Vasculopathy Progression Associated With Intraplaque Neovascularization." Journal of the American College of Cardiology 61, no. 9 (2013): e149. http://dx.doi.org/10.1016/j.jacc.2012.08.1036.

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Kashiwazaki, Daina, Masaki Koh, Haruto Uchino, et al. "Hypoxia accelerates intraplaque neovascularization derived from endothelial progenitor cells in carotid stenosis." Journal of Neurosurgery 131, no. 3 (2019): 884–91. http://dx.doi.org/10.3171/2018.4.jns172876.

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Abstract (sommario):
OBJECTIVEThe relationship between intraplaque hypoxia and intraplaque hemorrhage (IPH) has been reported, but the details remain obscure. In this study, the authors aimed to clarify the relationship among intraplaque hypoxia, endothelial progenitor cells (EPCs), and neovascularization, which causes IPH. The histological findings of specimens obtained from carotid endarterectomy were assessed.METHODSThis study included 49 patients who underwent carotid endarterectomy. Magnetic resonance plaque imaging was performed to analyze the components of the carotid plaques, and surgical specimens were su
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Koganti, S., A. Karanasos, S. Tu, R. D. Rakhit, and E. Regar. "Visualization of extensive intraplaque neovascularization by optical coherence tomography." Hellenic Journal of Cardiology 58, no. 1 (2017): 87–88. http://dx.doi.org/10.1016/j.hjc.2017.01.011.

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Più fonti

Tesi sul tema "Intraplaque neovascularization"

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Piechel, Lise. "Rôle du récepteur nucléaire Rev-erbα dans le contrôle de la fonction des cellules musculaires lisses au cours de l'athérosclérose". Electronic Thesis or Diss., Université de Lille (2022-....), 2024. http://www.theses.fr/2024ULILS078.

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Abstract (sommario):
Les maladies cardiovasculaires, principalement causées par l'athérosclérose, demeurent la première cause de mortalité dans le monde. L'athérosclérose est une maladie inflammatoire chronique de la paroi vasculaire provoquée par l'internalisation de lipides dans l'espace sous-endothéliale. Cette accumulation anormale déclenche l'infiltration de leucocytes et l'activation des cellules musculaires lisses (CML). Plus que l'obstruction des vaisseaux, l'instabilité et la rupture des plaques sont considérées comme les événements les plus délétères à l'origine d'infarctus du myocarde par exemple. Les d
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Bellengier, Cécilia. "Identification du rôle du récepteur nucléaire Rev-erb-α dans la néovascularisation de la plaque d'athérosclérose". Electronic Thesis or Diss., Université de Lille (2022-....), 2024. http://www.theses.fr/2024ULILS081.

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Abstract (sommario):
Malgré des décennies d'administration de traitements hypocholestérolémiants, de stratégies de prévention et d'efforts de recherche, les maladies cardiovasculaires, principalement causées par l'athérosclérose, restent la principale cause de décès dans le monde. De nouvelles thérapies sont donc indispensables pour réduire le risque cardiovasculaire résiduel et prévenir les événements athéro-thrombotiques. Plus que la sténose de la lumière artérielle, il est désormais admis que l’instabilité et la rupture de la plaque d'athérome sont les événements les plus délétères au cours de l’athérogenèse. P
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Capitoli di libri sul tema "Intraplaque neovascularization"

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Joner, Michael, Maria Isabel Castellanos, Anna Bulin, and Kristin Steigerwald. "Pathology of stable coronary artery disease." In ESC CardioMed, edited by William Wijns. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0325.

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Abstract (sommario):
Coronary artery disease remains the major cause of morbidity and mortality on a global scale. Intimal thickening and fatty streaks represent early adaptive vascular changes, which are often regressive. Pathological intimal thickening represents the earliest progressive atherosclerotic lesion characterized by a focal accumulation of smooth muscle cells and acellular areas, often associated with lipid pools. Fibroatheroma is characterized by a necrotic core and can be split into early and late fibroatheroma, where macrophage apoptosis and defective efferocytosis play important roles in lesion pr
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Joner, Michael, Maria Isabel Castellanos, Anna Bulin, and Kristin Steigerwald. "Pathology of stable coronary artery disease." In ESC CardioMed, edited by William Wijns. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0325_update_001.

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Abstract (sommario):
Coronary artery disease remains the major cause of morbidity and mortality on a global scale. Intimal thickening and fatty streaks represent early adaptive vascular changes, which are often regressive. Pathological intimal thickening represents the earliest progressive atherosclerotic lesion characterized by a focal accumulation of smooth muscle cells and acellular areas, often associated with lipid pools. Fibroatheroma is characterized by a necrotic core and can be split into early and late fibroatheroma, where macrophage apoptosis and defective efferocytosis play important roles in lesion pr
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Atti di convegni sul tema "Intraplaque neovascularization"

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Akkus, Zeynettin, Guillaume Renaud, Nico de Jong, et al. "New quantification methods for carotid intraplaque neovascularization in contrast enhanced ultrasound." In 2013 IEEE International Ultrasonics Symposium (IUS). IEEE, 2013. http://dx.doi.org/10.1109/ultsym.2013.0316.

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Rapporti di organizzazioni sul tema "Intraplaque neovascularization"

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Ugusman, Azizah, Nur Syahidah Nor Hisam, Nur Syakirah Othman, et al. PHARMACOLOGICAL INTERVENTIONS FOR INTRAPLAQUE NEOVASCULARIZATION IN ATHEROSCLEROSIS. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.3.0005.

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