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Articoli di riviste sul tema "Insulin resistance"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 19 ottobre 2024

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1

Kumar, Sinha Ritesh, e Chandra Satish. "Insulin resistance". Asian Pacific Journal of Health Sciences, Supplimentary 2014 (2014): 71–78. http://dx.doi.org/10.21276/apjhs.2014.1.1s.15.

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2

Sowers, James R., e Edward D. Frohlich. "Insulin and insulin resistance:". Medical Clinics of North America 88, n. 1 (gennaio 2004): 63–82. http://dx.doi.org/10.1016/s0025-7125(03)00128-7.

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3

Jeffery, Alison. "Insulin resistance". Nursing Standard 17, n. 32 (23 aprile 2003): 47–53. http://dx.doi.org/10.7748/ns2003.04.17.32.47.c3381.

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4

Tomono, Syouichi. "Insulin Resistance :". Kitakanto Medical Journal 62, n. 1 (2012): 73–74. http://dx.doi.org/10.2974/kmj.62.73.

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5

Jeffery, Alison. "Insulin resistance". Nursing Standard 17, n. 32 (23 aprile 2003): 47–55. http://dx.doi.org/10.7748/ns.17.32.47.s62.

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6

Park, Kyong Soo. "Insulin Resistance". Journal of the Korean Medical Association 44, n. 3 (2001): 302. http://dx.doi.org/10.5124/jkma.2001.44.3.302.

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7

Bell, David S. H. "Insulin resistance". Postgraduate Medicine 93, n. 7 (15 maggio 1993): 99–107. http://dx.doi.org/10.1080/00325481.1993.11701704.

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8

Carroll, P. B., e R. C. Eastman. "Insulin Resistance". Endocrinologist 1, n. 2 (aprile 1991): 89–97. http://dx.doi.org/10.1097/00019616-199104000-00005.

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9

DeFronzo, Ralph A. "Insulin resistance". Current Opinion in Cardiology 5, n. 5 (ottobre 1990): 592–98. http://dx.doi.org/10.1097/00001573-199010000-00003.

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10

Whitelaw, D. C., e S. G. Gilbey. "Insulin Resistance". Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 35, n. 5 (settembre 1998): 567–83. http://dx.doi.org/10.1177/000456329803500501.

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11

Sinaiko, Alan R., e Sonia Caprio. "Insulin Resistance". Journal of Pediatrics 161, n. 1 (luglio 2012): 11–15. http://dx.doi.org/10.1016/j.jpeds.2012.01.012.

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12

VOLL, ARTUR, e JOHAN KLOSTER. "Insulin Resistance". Acta Medica Scandinavica 157, n. 3 (24 aprile 2009): 223–32. http://dx.doi.org/10.1111/j.0954-6820.1957.tb14430.x.

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13

El Ray, Ahmed, Tarik Asselah, Rami Moucari, Maged El Ghannam, Alaa A. Taha, Mohamed A. Saber, Maha Akl et al. "Insulin resistance". European Journal of Gastroenterology & Hepatology 25, n. 4 (aprile 2013): 421–27. http://dx.doi.org/10.1097/meg.0b013e32835c9f69.

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14

O'Doherty, R., D. Stein e J. Foley. "Insulin resistance". Diabetologia 40 (19 settembre 1997): S10—S15. http://dx.doi.org/10.1007/s001250051389.

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15

El-Zayadi, Abdel-Rahman. "Insulin resistance". Arab Journal of Gastroenterology 11, n. 2 (giugno 2010): 66–69. http://dx.doi.org/10.1016/j.ajg.2010.04.010.

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16

Altunoglu, Esma Guldal. "Insulin resistance". Istanbul Medical Journal 13, n. 3 (2012): 0. http://dx.doi.org/10.5505/1304.8503.2012.78941.

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17

Krenkel, Jessica A. "Insulin Resistance". Topics in Clinical Nutrition 17, n. 5 (dicembre 2002): 85. http://dx.doi.org/10.1097/00008486-200212000-00011.

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18

Nesbitt, Alexander. "Insulin Resistance". Journal of Advanced Nursing 44, n. 3 (20 ottobre 2003): 327–28. http://dx.doi.org/10.1046/j.1365-2648.2003.02826_3.x.

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19

Wolfrum, C., E. Asilmaz, E. Luca, J. M. Friedman e M. Stoffel. "Insulin Resistance". Journal of the American Society of Nephrology 16, n. 3 (marzo 2005): 569–71. http://dx.doi.org/10.1681/01.asn.0000926692.88991.55.

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20

Chaoji, Sunanda Ashutosh. "Insulin Resistance". Vidarbha Journal of Internal Medicine 33 (6 aprile 2023): 27–31. http://dx.doi.org/10.25259/vjim_41_2022.

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Abstract (sommario):

The twin epidemic of ‘Diabesity’-diabetes and obesity, all over the world, both in developed and developing countries, has brought the issue of insulin resistance (IR) into new focus of research. Apart from Type 2 diabetes mellitus (DM), IR is implicated in many other clinical syndromes because of its varied metabolic and mitogenic actions. IR has been found to play important pivotal role in pathophysiology of diabesity. IR is defined as when a normal or higher insulin level fails to produce expected biological response; one predominantly affecting insulin mediated glucose disposal 20–30% reduction in the number of insulin receptors on the target cells is observed in majority of Type 2 DM patients but 1/3 of the patients may not manifest loss of number of receptors; therefore, defective post-receptor signalling is considered as the main cause of IR. Main sites of IR are liver, adipose tissue and skeletal muscles. Apart from Type 2 DM, many other clinical and genetic syndromes are associated with IR.

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21

O’Doherty, R., D. Stein e J. Foley. "Insulin resistance". Diabetologia 40, S3 (marzo 1997): B10—B15. http://dx.doi.org/10.1007/bf03168180.

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22

Perkins, Amanda. "Insulin resistance". Nursing Made Incredibly Easy! 22, n. 3 (10 aprile 2024): 5–13. http://dx.doi.org/10.1097/nme.0000000000000046.

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Abstract (sommario):

Insulin resistance is believed to be a precursor to type 2 diabetes mellitus, metabolic syndrome, and nonalcoholic fatty liver disease. Through education, nurses can understand their role in helping patients manage this condition.

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23

Beale, Elmus G. "Insulin Signaling and Insulin Resistance". Journal of Investigative Medicine 61, n. 1 (1 gennaio 2013): 11–14. http://dx.doi.org/10.2310/jim.0b013e3182746f95.

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24

OʼHare, James A. "Insulin, insulin resistance, and hypertension". Current Opinion in Endocrinology and Diabetes 1, n. 1 (gennaio 1994): 147–52. http://dx.doi.org/10.1097/00060793-199400010-00027.

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25

Stumvoll, Michael, e Hans Häring. "Insulin Resistance and Insulin Sensitizers". Hormone Research in Paediatrics 55, n. 2 (2001): 3–13. http://dx.doi.org/10.1159/000063466.

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26

HOWARD, BARBARA V. "Insulin, Insulin Resistance, and Dyslipidemia". Annals of the New York Academy of Sciences 683, n. 1 Dietary Lipid (giugno 1993): 1–8. http://dx.doi.org/10.1111/j.1749-6632.1993.tb35687.x.

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27

Petersen, Max C., e Gerald I. Shulman. "Mechanisms of Insulin Action and Insulin Resistance". Physiological Reviews 98, n. 4 (1 ottobre 2018): 2133–223. http://dx.doi.org/10.1152/physrev.00063.2017.

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Abstract (sommario):

The 1921 discovery of insulin was a Big Bang from which a vast and expanding universe of research into insulin action and resistance has issued. In the intervening century, some discoveries have matured, coalescing into solid and fertile ground for clinical application; others remain incompletely investigated and scientifically controversial. Here, we attempt to synthesize this work to guide further mechanistic investigation and to inform the development of novel therapies for type 2 diabetes (T2D). The rational development of such therapies necessitates detailed knowledge of one of the key pathophysiological processes involved in T2D: insulin resistance. Understanding insulin resistance, in turn, requires knowledge of normal insulin action. In this review, both the physiology of insulin action and the pathophysiology of insulin resistance are described, focusing on three key insulin target tissues: skeletal muscle, liver, and white adipose tissue. We aim to develop an integrated physiological perspective, placing the intricate signaling effectors that carry out the cell-autonomous response to insulin in the context of the tissue-specific functions that generate the coordinated organismal response. First, in section II, the effectors and effects of direct, cell-autonomous insulin action in muscle, liver, and white adipose tissue are reviewed, beginning at the insulin receptor and working downstream. Section III considers the critical and underappreciated role of tissue crosstalk in whole body insulin action, especially the essential interaction between adipose lipolysis and hepatic gluconeogenesis. The pathophysiology of insulin resistance is then described in section IV. Special attention is given to which signaling pathways and functions become insulin resistant in the setting of chronic overnutrition, and an alternative explanation for the phenomenon of ‟selective hepatic insulin resistanceˮ is presented. Sections V, VI, and VII critically examine the evidence for and against several putative mediators of insulin resistance. Section V reviews work linking the bioactive lipids diacylglycerol, ceramide, and acylcarnitine to insulin resistance; section VI considers the impact of nutrient stresses in the endoplasmic reticulum and mitochondria on insulin resistance; and section VII discusses non-cell autonomous factors proposed to induce insulin resistance, including inflammatory mediators, branched-chain amino acids, adipokines, and hepatokines. Finally, in section VIII, we propose an integrated model of insulin resistance that links these mediators to final common pathways of metabolite-driven gluconeogenesis and ectopic lipid accumulation.

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28

Chandra, Nimai Chand. "Hyperlipidimia, Hyperleptinemia and Insulin Resistance Intercorrelation with Obesity". Diabetes & Obesity International Journal 5, n. 1 (2020): 1–6. http://dx.doi.org/10.23880/doij-16000223.

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Abstract (sommario):

The present study has shown a parallel increase of triacylglycerol, cholesterol, and low density lipoprotein (LDL) along with an increase in body mass and BMI of human obese subjects. Conversely a fall in HDL concentration was observed. Plasma leptin level increased proportionately with increase of body weight as obesity advanced from obese to morbid type. A positive correlation was observed between leptin and insulin concentrations in obese subjects. A parallel increase of plasma glucose concentration was followed with decreased expression of insulin receptor, measured in adipose tissue, with the progress of obesity. The decreased insulin receptor concentration might be the reason for seen insulin resistance and increased insulin level which marked the positive correlation with associated liptin level in obese subjects.

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29

Zhang, Li, Tao Liu, Pei-Yong Zheng e Guang Ji. "Leptin resistance and insulin resistance". World Chinese Journal of Digestology 17, n. 15 (2009): 1534. http://dx.doi.org/10.11569/wcjd.v17.i15.1534.

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30

Sivitz, William I. "Understanding insulin resistance". Postgraduate Medicine 116, n. 1 (luglio 2004): 41–48. http://dx.doi.org/10.3810/pgm.2004.07.1550.

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31

SARUTA, TAKAO. "Insulin resistance syndrome." Nihon Naika Gakkai Zasshi 85, n. 2 (1996): 285–91. http://dx.doi.org/10.2169/naika.85.285.

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32

Bloomgarden, Z. T. "Insulin Resistance Concepts". Diabetes Care 30, n. 5 (27 aprile 2007): 1320–26. http://dx.doi.org/10.2337/dc07-zb05.

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33

Fletcher, Barbara, e Cindy Lamendola. "Insulin Resistance Syndrome". Journal of Cardiovascular Nursing 19, n. 5 (settembre 2004): 339–45. http://dx.doi.org/10.1097/00005082-200409000-00009.

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34

Leslie, M. "Outfoxing Insulin Resistance". Science of Aging Knowledge Environment 2002, n. 36 (11 settembre 2002): 125nw—125. http://dx.doi.org/10.1126/sageke.2002.36.nw125.

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35

Polonsky, William. "Psychological Insulin Resistance". Diabetes Educator 33, n. 7s (luglio 2007): 241S—244S. http://dx.doi.org/10.1177/0145721707305701.

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36

Montminy, Marc, e Seung-Hoi Koo. "Outfoxing insulin resistance?" Nature 432, n. 7020 (dicembre 2004): 958–59. http://dx.doi.org/10.1038/432958a.

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37

Funnell, Martha Mitchell. "Understanding insulin resistance". Nursing 42, n. 3 (marzo 2012): 62. http://dx.doi.org/10.1097/01.nurse.0000411421.44527.bf.

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38

Cheng, Tsung O. "Insulin Resistance Syndrome". Southern Medical Journal 91, n. 7 (luglio 1998): 697. http://dx.doi.org/10.1097/00007611-199807000-00041.

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39

GRANBERRY, MARK C., e VIVIAN A. FONSECA. "Insulin Resistance Syndrome". Southern Medical Journal 92, n. 1 (gennaio 1999): 2–14. http://dx.doi.org/10.1097/00007611-199901000-00002.

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40

Cummings, Doyle M., Sarah Henes, Kathryn M. Kolasa, John Olsson e David Collier. "Insulin Resistance Status". Archives of Pediatrics & Adolescent Medicine 162, n. 8 (1 agosto 2008): 764. http://dx.doi.org/10.1001/archpedi.162.8.764.

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41

DeFronzo, Ralph A. "Introduction: Insulin Resistance". Diabetes / Metabolism Reviews 5, n. 5 (agosto 1989): iii. http://dx.doi.org/10.1002/dmr.5610050505.

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42

Willette, Auriel A., Christine M. Burns e Barbara B. Bendlin. "Insulin Resistance andAPOEε4". JAMA Neurology 72, n. 12 (1 dicembre 2015): 1536. http://dx.doi.org/10.1001/jamaneurol.2015.3285.

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43

Jeffery, Alison. "Prepubertal insulin resistance". Practical Diabetes 29, n. 4 (maggio 2012): 130–31. http://dx.doi.org/10.1002/pdi.1676.

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44

Limoncini, A., C. Smirne, F. Corliano, C. Marconi, E. Scaglia, G. Carnevale, P. Toniutto, C. Fabris e M. Pirisi. "[725] INSULIN RESISTANCE, INSULIN SECRETION AND". Journal of Hepatology 46 (aprile 2007): S273. http://dx.doi.org/10.1016/s0168-8278(07)62323-x.

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45

MAGSINO, CESAR H., e JUDY SPENCER. "Insulin Receptor Antibodies and Insulin Resistance". Southern Medical Journal 92, n. 7 (luglio 1999): 717–19. http://dx.doi.org/10.1097/00007611-199907000-00013.

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46

Gallagher, Emily Jane, e Derek LeRoith. "Insulin, Insulin Resistance, Obesity, and Cancer". Current Diabetes Reports 10, n. 2 (6 marzo 2010): 93–100. http://dx.doi.org/10.1007/s11892-010-0101-y.

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47

Adiga, Usha, Kathyayani P e Nandith P.B. "Association of Insulin Based Insulin Resistance with Liver Biomarkers in Type 2 Diabetes mellitus". Journal of Pure and Applied Microbiology 13, n. 2 (30 giugno 2019): 1199–205. http://dx.doi.org/10.22207/jpam.13.2.60.

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48

Chitturi, Shivakumar, Shehan Abeygunasekera, Geoffrey C. Farrell, Jane Holmes-Walker, Jason M. Hui, Caroline Fung, Rooshdiya Karim et al. "NASH and insulin resistance: Insulin hypersecretion and specific association with the insulin resistance syndrome". Hepatology 35, n. 2 (febbraio 2002): 373–79. http://dx.doi.org/10.1053/jhep.2002.30692.

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49

Pollak, Michael N. "Insulin, insulin-like growth factors, insulin resistance, and neoplasia". American Journal of Clinical Nutrition 86, n. 3 (1 settembre 2007): 820S—822S. http://dx.doi.org/10.1093/ajcn/86.3.820s.

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50

Jahromi, Abdolreza Sotoodeh, e Zhila Rahmanian. "25: ASSOCIATION OF SERUM GAMMA- INTERFERON AND IL-10 CONCENTRATIONS WITH INSULIN RESISTANCE IN MAJOR THALASSEMIA PATIENTS". BMJ Open 7, Suppl 1 (febbraio 2017): bmjopen—2016–015415.25. http://dx.doi.org/10.1136/bmjopen-2016-015415.25.

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Abstract (sommario):

Background and aims:Thalassemia is one of the most prevalent hematologic disorders worldwide. Thalassemia is the most common inherited anemia and genetic disease. Diabetes mellitus and insulin resistance is one of the major endocrine problems in major thalassemia patients. This study was done to evaluate the association of serum γ- interferon and IL-10 concentrations with insulin resistance in splenectomized and non-splenectomized major thalassemia patients.Methods:193 thalassemia patients with rang of years old participated in this study. IFN-γ and IL-10 levels were measured using ELISA method. Insulin resistance was measured using HOMA-IR method. Data was analyzed with SPSS software by Pair T test.Results:the mean age of splenectomized patients in this study was 8.86±7.03 and non- splenectomized patients 7.88±2.39 years. There were significant and inverse associations between serum IL-10 levels and insulin resistance was found in non-splenectomized major thalassemia patients (P=0.002). There were not a significant association between serum IL-10 levels and insulin resistance in splenectomized major thalassemia patients (P=0.079). There were not a significant association between serum γ- interferon levels and insulin resistance in splenectomized major thalassemia patients (P=0.778). There were not significant associations between serum γ- interferon levels and insulin resistance in non- splenectomized major thalassemia patients (P=0.435).Conclusion:In this study, interferon-gamma as an inflammatory cytokine and interlukin-10 as an anti-inflammatory cytokines was studied. Overall these results suggest that interleukin-10 is associated with insulin resistance in non-splenectomized major thalassemia patients and as an anti-inflammatory cytokine may play an important role in sustaining insulin resistance in non-splenectomized major thalassemia patients. With increase in serum IL-10 levels there is a decline in insulin resistance. This data provide new insights into the mechanisms of insulin resistance and support the use of other cytokines with insuln resistance in major thalassemia patients.

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