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Bibliografie tematiche / Instability mechanisms

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Articoli di riviste

Letteratura scientifica selezionata sul tema "Instability mechanisms"

Autore: Grafiati

Pubblicato: 7 settembre 2024

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Articoli di riviste sul tema "Instability mechanisms"

1

Baird, D. M. "Mechanisms of telomeric instability." Cytogenetic and Genome Research 122, no. 3-4 (2008): 308–14. http://dx.doi.org/10.1159/000167817.

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2

Thompson, Sarah L., Samuel F. Bakhoum, and Duane A. Compton. "Mechanisms of Chromosomal Instability." Current Biology 20, no. 6 (2010): R285—R295. http://dx.doi.org/10.1016/j.cub.2010.01.034.

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3

He Bai and M. Arcak. "Instability Mechanisms in Cooperative Control." IEEE Transactions on Automatic Control 55, no. 1 (2010): 258–63. http://dx.doi.org/10.1109/tac.2009.2036301.

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4

Sharma, G., R. V. Ramanujan, and G. P. Tiwari. "Instability mechanisms in lamellar microstructures." Acta Materialia 48, no. 4 (2000): 875–89. http://dx.doi.org/10.1016/s1359-6454(99)00378-x.

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5

Venkatesan, Shriram, Adayapalam T. Natarajan, and M. Prakash Hande. "Chromosomal instability—mechanisms and consequences." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 793 (November 2015): 176–84. http://dx.doi.org/10.1016/j.mrgentox.2015.08.008.

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6

Gollin, Susanne M. "Mechanisms leading to chromosomal instability." Seminars in Cancer Biology 15, no. 1 (2005): 33–42. http://dx.doi.org/10.1016/j.semcancer.2004.09.004.

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7

Shah, Prediman K. "Molecular mechanisms of plaque instability." Current Opinion in Lipidology 18, no. 5 (2007): 492–99. http://dx.doi.org/10.1097/mol.0b013e3282efa326.

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8

Sirignano, William A. "Driving Mechanisms for Combustion Instability." Combustion Science and Technology 187, no. 1-2 (2014): 162–205. http://dx.doi.org/10.1080/00102202.2014.973801.

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9

Gallaire, F., and J. M. Chomaz. "Instability mechanisms in swirling flows." Physics of Fluids 15, no. 9 (2003): 2622–39. http://dx.doi.org/10.1063/1.1589011.

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10

Huang, Jinhua, Jinping Liang, Lijia Huang, and Tingting Li. "Mechanisms of Atherosclerotic Plaque Instability." International Journal of Biology and Life Sciences 5, no. 1 (2024): 9–12. http://dx.doi.org/10.54097/83r6jq74.

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Abstract (sommario):

Cardiovascular disease (CVD) is the leading cause of mortality in humans worldwide. The main cause of CVD is the formation of thrombi due to by unstable atherosclerotic plaque rupture on the arterial wall. Long-term accumulation of thrombi results in vascular remodeling, and subsequent-stenosis of the lumen obstructs the blood flow, thereby leading to myocardial tissue ischemia and hypoxia. Sustained ischemia and hypoxia lead to myocyte necrosis, resulting in irreversible myocardial injury. Many molecular and cellular mechanisms are associated with atherosclerotic plaque instability (API). For example, macrophages can produce various inflammatory factors, adhesion factors, chemokines and matrix metalloproteinases (MMPs), which play important roles in the pathophysiological mechanisms of API and in maintaining plaque stability. These molecules may help predict unstable atherosclerotic plaques. If the plaque is stable, it will not be prone to rupture or thrombosis. Accordingly, in this review, we will discuss the different pathophysiological mechanisms of API and the related roles of macrophages in the mechanisms of API mainly in animal models and humans. We believe this review will provide a theoretical basis for the development of treatments and diagnostic approaches for the management of API.

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