Accedi

Bibliografie tematiche / Inbred mice / Articoli di riviste

Articoli di riviste sul tema "Inbred mice"

Segui questo link per vedere altri tipi di pubblicazioni sul tema: Inbred mice.

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 6 febbraio 2022

Cita una fonte nei formati APA, MLA, Chicago, Harvard e in molti altri stili

Scegli il tipo di fonte:

Vedi i top-50 articoli di riviste per l'attività di ricerca sul tema "Inbred mice".

Accanto a ogni fonte nell'elenco di riferimenti c'è un pulsante "Aggiungi alla bibliografia". Premilo e genereremo automaticamente la citazione bibliografica dell'opera scelta nello stile citazionale di cui hai bisogno: APA, MLA, Harvard, Chicago, Vancouver ecc.

Puoi anche scaricare il testo completo della pubblicazione scientifica nel formato .pdf e leggere online l'abstract (il sommario) dell'opera se è presente nei metadati.

Vedi gli articoli di riviste di molte aree scientifiche e compila una bibliografia corretta.

1

Nishioka, Yutaka. "The origin of common laboratory mice". Genome 38, n. 1 (1 febbraio 1995): 1–7. http://dx.doi.org/10.1139/g95-001.

Testo completo

Abstract (sommario):

The house mouse is one of the model organisms in genetics and more than 400 inbred strains have been established. However, many of the strains are related and their ancestry can be traced back to European fancy mice inbred in the 1920s. Recent molecular studies corroborate the early historical records that assert that Japanese fancy mice were introduced into European stocks and thus contributed to the development of "old" inbred strains. Consequently, many inbred strains have genomic DNA derived from more than one subspecies of Mus musculus. The subspecific hybrid origin of common inbred strains has important bearings on the interpretation of genetic data, and the limitations that history imposes upon the currently available strains make it necessary to establish new inbred strains representing specific wild populations.

Gli stili APA, Harvard, Vancouver, ISO e altri

2

West, John D., e Graham Fisher. "Inherited cataracts in inbred mice". Genetical Research 46, n. 1 (agosto 1985): 45–56. http://dx.doi.org/10.1017/s0016672300022448.

Testo completo

Abstract (sommario):

SUMMARYExamination of the eyes with a slit lamp revealed that 101/H mice had a coloured cataract. Crosses to C3H/HeH indicated that this was inherited as a single recessive gene which we have designated lop-2 (lens opacity-2). The related strain 129/Sv-SlJ-CP had a phenotypically identical cataract and presumably also carries the lop-2 gene. CBA/H, CBA/CaH- + /p, CBA/H-kd and CBA/H-T6 mice had a bright white or white/green cataract that typically extended from the nucleus to the anterior cortex of the lens. Crosses to C3H/HeH indicated that this was inherited as a semi-dominant gene. However, other crosses raise the possibility that the CBA cataract is also caused by lop-2. If so, the expressivity (and penetrance of the heterozygote) is affected by genetic background. Neither lop-2 nor the gene responsible for the CBA cataract was linked to contrasted (Slcon) on chromosome 10, so these are distinct from the Lop (lens opacity) gene. Further studies of genetic linkage are needed to clarify whether lop-2 is responsible for both the 101/H and CBA/H cataracts.

Gli stili APA, Harvard, Vancouver, ISO e altri

3

Sundberg, J. P., C. A. Hanson, D. R. Roop, K. S. Brown e H. G. Bedigian. "Myoepitheliomas in Inbred Laboratory Mice". Veterinary Pathology 28, n. 4 (luglio 1991): 313–22. http://dx.doi.org/10.1177/030098589102800408.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

4

Passino, Enrica, e Martine Ammassari–Teule. "Visual Discrimination in Inbred Mice". Physiology & Behavior 67, n. 3 (settembre 1999): 393–99. http://dx.doi.org/10.1016/s0031-9384(99)00076-1.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

5

Iwasaki, Akiko. "Antiviral responses of inbred mice". Nature Reviews Immunology 16, n. 6 (25 aprile 2016): 339. http://dx.doi.org/10.1038/nri.2016.44.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

6

Galkin, O. Yu, A. G. Komar e O. B. Besarab. "Different mice inbred strains humoral immune response against human prostate-specific antigen". Ukrainian Biochemical Journal 91, n. 1 (28 gennaio 2019): 30–37. http://dx.doi.org/10.15407/ubj91.01.030.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

7

Darlington, T. M., M. A. Ehringer, C. Larson, T. L. Phang e R. A. Radcliffe. "Transcriptome analysis of Inbred Long Sleep and Inbred Short Sleep mice". Genes, Brain and Behavior 12, n. 2 (22 febbraio 2013): 263–74. http://dx.doi.org/10.1111/gbb.12018.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

8

Dagnaes-Hansen, Frederik, Jacob M. Moser, Thomas Smith-John e Mie Aarup. "Sudden death in lactating inbred mice". Lab Animal 39, n. 7 (luglio 2010): 205. http://dx.doi.org/10.1038/laban0710-205.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

9

Liebelt, Robert A. "Inbred mice fed only bee pollen". Journal of ApiProduct and ApiMedical Science 2, n. 4 (1 ottobre 2010): 156–60. http://dx.doi.org/10.3896/ibra.4.02.4.04.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

10

Haimrich, B., H. Schwegler, W. E. Crusio e W. Buselmaier. "Substrain divergence in C3H inbred mice". Behavior Genetics 18, n. 6 (novembre 1988): 671–74. http://dx.doi.org/10.1007/bf01066849.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

11

Beau, Jacques. "Activity rhythms in inbred mice. I. Genetic analysis with recombinant inbred strains". Behavior Genetics 21, n. 2 (marzo 1991): 117–29. http://dx.doi.org/10.1007/bf01066331.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

12

Grasperge, Britton J., Kathryn E. Reif, Timothy D. Morgan, Piyanate Sunyakumthorn, Joseph Bynog, Christopher D. Paddock e Kevin R. Macaluso. "Susceptibility of Inbred Mice to Rickettsia parkeri". Infection and Immunity 80, n. 5 (5 marzo 2012): 1846–52. http://dx.doi.org/10.1128/iai.00109-12.

Testo completo

Abstract (sommario):

ABSTRACTRickettsia parkeri, a member of the spotted fever groupRickettsia, is the causative agent of American boutonneuse fever in humans. Despite the increased recognition of human cases, limited information is available regarding the infection of invertebrate and vertebrate hosts for this emerging tick-borne disease. Toward the development of a viable transmission model and to further characterize the pathology associated withR. parkeriinfection, inbred mouse strains (A/J, BALB/c, C3H/HeJ, and C3H/HeN) were intravenously and intradermally inoculated with 105low-passage-numberR. parkeri(Portsmouth strain), and infection, gross pathology, and histopathology were scored. Additionally, a quantitative real-time PCR (qPCR) was performed to estimate rickettsial load in heart, lung, spleen, and liver tissues of infected mice at 19 days postinoculation. Of the A/J, BALB/c, and C3H/HeN mice, none displayed universal pathology consistent with sustained infection. Compared to age-matched control mice, the intravenously inoculated C3H/HeJ mice exhibited marked facial edema and marked splenomegaly upon gross examination, while the intradermally inoculated mice developed characteristic eschar-like lesions. The C3H/HeJ mice also exhibited the greatest concentrations of rickettsial DNA from heart, lung, liver, and spleen samples when examined by qPCR. The similarity of the pathology of human disease and sustained infection suggests that the C3H/HeJ strain of mice is a promising candidate for subsequent experiments to examine the tick transmission, dissemination, and pathology ofR. parkeririckettsiosis.

Gli stili APA, Harvard, Vancouver, ISO e altri

13

Salcedo, Tovah, Armando Geraldes e Michael W. Nachman. "Nucleotide Variation in Wild and Inbred Mice". Genetics 177, n. 4 (dicembre 2007): 2277–91. http://dx.doi.org/10.1534/genetics.107.079988.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

14

Stevens, James C., Gareth T. Banks, Michael F. W. Festing e Elizabeth M. C. Fisher. "Quiet mutations in inbred strains of mice". Trends in Molecular Medicine 13, n. 12 (dicembre 2007): 512–19. http://dx.doi.org/10.1016/j.molmed.2007.10.001.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

15

Liu, Wing-Keung, Nai-Ki Mak e Chuen-Chuen Wong. "Immunomodulatory Effect of Methimazole on Inbred Mice". Immunobiology 180, n. 1 (novembre 1989): 23–32. http://dx.doi.org/10.1016/s0171-2985(89)80027-0.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

16

Lewin, R. "Why do inbred mice evolve so quickly?" Science 228, n. 4704 (7 giugno 1985): 1187. http://dx.doi.org/10.1126/science.4039848.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

17

Sundberg, J. P., D. L. Adkison e H. G. Bedigian. "Skeletal Muscle Rhabdomyosarcomas in Inbred Laboratory Mice". Veterinary Pathology 28, n. 3 (maggio 1991): 200–206. http://dx.doi.org/10.1177/030098589102800303.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

18

Eisen, H. N., e E. B. Reilly. "Lambda Chains and Genes in Inbred Mice". Annual Review of Immunology 3, n. 1 (aprile 1985): 337–65. http://dx.doi.org/10.1146/annurev.iy.03.040185.002005.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

19

Pratte, Michel, e Marc Jamon. "Detection of social approach in inbred mice". Behavioural Brain Research 203, n. 1 (ottobre 2009): 54–64. http://dx.doi.org/10.1016/j.bbr.2009.04.011.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

20

Alexander, R. C., J. Duda, D. Garth, W. Vogel e W. H. Berrettini. "Morphine and cocaine preference in inbred mice". Psychiatric Genetics 3, n. 1 (1993): 33–38. http://dx.doi.org/10.1097/00041444-199321000-00006.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

21

Maeda, Nobuko, Satoshi Arai, Akira Ozaki, Tsutomu Oowada, Akiko Takahashi, Hiroshi Fujita e Takeo Mizutani. "Experimental Dental Caries on Gnotobiotic Inbred Mice". Microbiology and Immunology 39, n. 1 (gennaio 1995): 71–73. http://dx.doi.org/10.1111/j.1348-0421.1995.tb02171.x.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

22

Otobe, Takayuki, e Junshiro Makino. "Impulsive choice in inbred strains of mice". Behavioural Processes 67, n. 1 (luglio 2004): 19–26. http://dx.doi.org/10.1016/j.beproc.2004.02.001.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

23

Elmer, Gregory I., Richard A. Meisch e Frank R. George. "Oral ethanol reinforced behavior in inbred mice". Pharmacology Biochemistry and Behavior 24, n. 5 (maggio 1986): 1417–21. http://dx.doi.org/10.1016/0091-3057(86)90204-2.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

24

Crow, James F. "C. C. Little, Cancer and Inbred Mice". Genetics 161, n. 4 (1 agosto 2002): 1357–61. http://dx.doi.org/10.1093/genetics/161.4.1357.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

25

Kirillicheva, G. B., M. S. Solov'eva, I. G. Baturina, T. N. Shchukovskaya e G. A. Maslennikov. "Immunomodulating action of histamine in inbred mice". Bulletin of Experimental Biology and Medicine 113, n. 3 (marzo 1992): 386–88. http://dx.doi.org/10.1007/bf00783122.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

26

Hamada, Akiko, Kiyotoshi Inenaga, Shuichi Nakamura, Masamichi Terashita e Hiroshi Yamashita. "Disorder of salivary secretion in inbred polydipsic mouse". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 278, n. 4 (1 aprile 2000): R817—R823. http://dx.doi.org/10.1152/ajpregu.2000.278.4.r817.

Testo completo

Abstract (sommario):

To find mechanisms of an extreme polydipsia in an inbred strain of mice, STR/N, this study was undertaken using Institute of Cancer Research (ICR) mice as a control. During food deprivation, daily water intake of both strains decreased. The decrement in the STR/N mice was larger than that in the ICR mice. During dehydration, daily food intake of the STR/N mice was smaller than that of the ICR mice. These data indicate that prandial drinking was more severely affected for the STR/N mice. Under anesthesia, the stimulated salivary secretion by pilocarpine of the STR/N mice was significantly smaller than that of the ICR mice. The submandibular gland of the STR/N mice was lighter and harder than that of the ICR mice. After desalivation from the major three salivary glands, the ICR mice drank as much as the STR/N mice. Young STR/N mice with undeveloped polydipsia did not show different salivary secretion stimulated by pilocarpine from the young ICR mice. These findings indicate a dysfunction with age in the salivary glands of the STR/N mice, and they suggest that the decreased saliva induces thirst and triggers extraordinary drinking in the polydipsic mice.

Gli stili APA, Harvard, Vancouver, ISO e altri

27

Chebib, Jobran, Benjamin C. Jackson, Eugenio López-Cortegano, Diethard Tautz e Peter D. Keightley. "Inbred lab mice are not isogenic: genetic variation within inbred strains used to infer the mutation rate per nucleotide site". Heredity 126, n. 1 (31 agosto 2020): 107–16. http://dx.doi.org/10.1038/s41437-020-00361-1.

Testo completo

Abstract (sommario):

AbstractFor over a century, inbred mice have been used in many areas of genetics research to gain insight into the genetic variation underlying traits of interest. The generalizability of any genetic research study in inbred mice is dependent upon all individual mice being genetically identical, which in turn is dependent on the breeding designs of companies that supply inbred mice to researchers. Here, we compare whole-genome sequences from individuals of four commonly used inbred strains that were procured from either the colony nucleus or from a production colony (which can be as many as ten generations removed from the nucleus) of a large commercial breeder, in order to investigate the extent and nature of genetic variation within and between individuals. We found that individuals within strains are not isogenic, and there are differences in the levels of genetic variation that are explained by differences in the genetic distance from the colony nucleus. In addition, we employ a novel approach to mutation rate estimation based on the observed genetic variation and the expected site frequency spectrum at equilibrium, given a fully inbred breeding design. We find that it provides a reasonable per nucleotide mutation rate estimate when mice come from the colony nucleus (~7.9 × 10−9 in C3H/HeN), but substantially inflated estimates when mice come from production colonies.

Gli stili APA, Harvard, Vancouver, ISO e altri

28

Eveleigh, J. R. "Murine cage density: cage ammonia levels during the reproductive performance of an inbred strain and two outbred stocks of monogamous breeding pairs of mice". Laboratory Animals 27, n. 2 (1 aprile 1993): 156–60. http://dx.doi.org/10.1258/002367793780810432.

Testo completo

Abstract (sommario):

The Laboratory Animal Breeders Association guidelines recommend a minimum floor area of 300 cm2 for a monogamous pair of inbred/outbred mice or a trio of inbreds. The mean level of ammonia produced during lactation from BALB/c, TO and CD-1 breeding pairs housed in M2 cages with a floor area of 300 cm2 on Day 4 after cleaning was 30 ppm, 87 ppm and 92 ppm, respectively. All 3 strains of mice, particularly the outbred strains, were subjected to high levels of ammonia as compared with human long-term health and safety occupational exposure limits (25 ppm). However, there is a gradient of ammonia within an M2 breeding cage from the nest (19 ppm), to the food hopper, 77 ppm. By housing CD-1 pairs of mice in RM2 cages which have more than double the floor area of M2 cages (676 cm2), the mean level of ammonia during lactation on Day 4 after cleaning was reduced to 26 ppm. The reproductive performance on inbred/outbred strains of mice has to be equated with cage size (floor area) to maintain acceptable levels of ammonia. It is suggested that the recommended minimum floor areas for breeding mice be reviewed.

Gli stili APA, Harvard, Vancouver, ISO e altri

29

Ishii, Akira I., e Motohito Sano. "Strain-dependent differences in susceptibility of mice to experimental Angiostrongylus costaricensis infection". Journal of Helminthology 63, n. 4 (dicembre 1989): 302–6. http://dx.doi.org/10.1017/s0022149x00009196.

Testo completo

Abstract (sommario):

ABSTRACTExperimental Angiostrongylus costaricensis infection was carried out in inbred strains of mice (C57BL/6, BALB/c, DBA/2 and C3H/He). All strains became infected with this parasite. Marked differences in mortality and in worm burden were found among inbred strains of mice tested. A significant reduction was shown in worm length from mice compared to that from cotton rats.

Gli stili APA, Harvard, Vancouver, ISO e altri

30

DiGiovanni, John, Akira Imamoto, Masashi Naito, Susan E. Walker, Linda Beltrán, Kristine J. Chenicek e Loren Skow. "Further genetic analyses of skin tumor promoter susceptibility using inbred and recombinant inbred mice". Carcinogenesis 13, n. 4 (1992): 525–31. http://dx.doi.org/10.1093/carcin/13.4.525.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

31

Rogers, Arlin B. "Stress of Strains: Inbred Mice in Liver Research". Gene Expression 19, n. 1 (14 dicembre 2019): 61–67. http://dx.doi.org/10.3727/105221618x15337408678723.

Testo completo

Abstract (sommario):

Inbred mice are the most popular animals used for in vivo liver research. These mice are genetically defined, readily available, less expensive to maintain than larger animals, and enjoy a broad array of commercial reagents for scientific characterization. C57BL/6 mice are the most commonly used strain. However, other strains discussed, including BALB/c, C3H, A/J, and FVB/N, may be better suited to a particular disease model or line of investigation. Understanding the phenotypes of different inbred mouse strains facilitates informed decision making during experimental design. Model systems influenced by strain-dependent phenotype include tissue regeneration, drug-induced liver injury (DILI; e.g., acetaminophen), fibrosis (e.g., carbon tetrachloride, CCl4), Fas-induced apoptosis, cholestasis, alcohol-induced liver disease and cirrhosis, nonalcoholic fatty liver disease and steatohepatitis (NAFLD/NASH), and hepatocellular carcinoma (HCC). Thoughtful consideration of the strengths and weaknesses of each inbred strain in a given model system will lead to more robust data and a clearer understanding of translational relevance to human liver disease.

Gli stili APA, Harvard, Vancouver, ISO e altri

32

Kruckeberg, WC, DI Doorenbos e PO Brown. "Genetic differences in hemoglobin influence on erythrocyte oxidative stress hemolysis". Blood 70, n. 4 (1 ottobre 1987): 909–14. http://dx.doi.org/10.1182/blood.v70.4.909.909.

Testo completo

Abstract (sommario):

Abstract The RBC from mice of certain inbred strains hemolyzed under oxidative stress (2.0 mmol/L hydrogen peroxide), whereas red cells from mice of other strains did not. In the experimental system human erythrocytes did not hemolyze. The rate of formation of malonyldialdehyde (a fatty acid oxidative breakdown product) was fourfold higher in hemolytic v nonhemolytic red cells. There was insufficient variation in the levels of glutathione, peroxidase activity or its substrate, reduced glutathione, to explain these hemolysis differences. On the other hand, the antioxidants butylated hydroxyanisole and hydroxytoluene, and histidine protected the hemolysis-prone red cells from breaking open. The hemolysis trait demonstrated autosomal recessive Mendelian inheritance. When using inbred, recombinant inbred, and congenic inbred mice, this hemolysis/nonhemolysis trait correlated 1:1 with the type of hemoglobin beta chain in the RBC. This experimental system is a potential model for investigating the role of hemoglobin in prehemolytic events.

Gli stili APA, Harvard, Vancouver, ISO e altri

33

Kruckeberg, WC, DI Doorenbos e PO Brown. "Genetic differences in hemoglobin influence on erythrocyte oxidative stress hemolysis". Blood 70, n. 4 (1 ottobre 1987): 909–14. http://dx.doi.org/10.1182/blood.v70.4.909.bloodjournal704909.

Testo completo

Abstract (sommario):

The RBC from mice of certain inbred strains hemolyzed under oxidative stress (2.0 mmol/L hydrogen peroxide), whereas red cells from mice of other strains did not. In the experimental system human erythrocytes did not hemolyze. The rate of formation of malonyldialdehyde (a fatty acid oxidative breakdown product) was fourfold higher in hemolytic v nonhemolytic red cells. There was insufficient variation in the levels of glutathione, peroxidase activity or its substrate, reduced glutathione, to explain these hemolysis differences. On the other hand, the antioxidants butylated hydroxyanisole and hydroxytoluene, and histidine protected the hemolysis-prone red cells from breaking open. The hemolysis trait demonstrated autosomal recessive Mendelian inheritance. When using inbred, recombinant inbred, and congenic inbred mice, this hemolysis/nonhemolysis trait correlated 1:1 with the type of hemoglobin beta chain in the RBC. This experimental system is a potential model for investigating the role of hemoglobin in prehemolytic events.

Gli stili APA, Harvard, Vancouver, ISO e altri

34

Fiette, L., C. Aubert, U. Müller, S. Huang, M. Aguet, M. Brahic e J. F. Bureau. "Theiler's virus infection of 129Sv mice that lack the interferon alpha/beta or interferon gamma receptors." Journal of Experimental Medicine 181, n. 6 (1 giugno 1995): 2069–76. http://dx.doi.org/10.1084/jem.181.6.2069.

Testo completo

Abstract (sommario):

The Daniels strain of Theiler's virus causes a persistent infection of the white matter of spinal cord of susceptible mice, with chronic inflammation and primary demyelination. Inbred 129Sv mice are resistant to this infection; they present with mild encephalomyelitis and clear the infection within a matter of days. A very different outcome was observed with inbred 129Sv mice whose receptors for interferon alpha/beta or interferon gamma had been inactivated by homologous recombination. The former presented severe encephalomyelitis with acute infection of neurons, particularly in brain and hippocampus, and extensive infection with necrosis of the choroid plexus. Most animals died of this acute disease. The latter, presented the same early encephalomyelitis as the control 129Sv mice. However, they remained persistently infected and developed a very severe late infection of the white matter with extensive primary demyelination. This late disease looked like an exacerbated form of the chronic demyelinating disease observed in susceptible inbred mice such as the SJL/J or FVB strains. Our results show that the two interferon systems play nonredundant roles in the resistance of the 129Sv mouse to the infection by Theiler's virus. They also lend support to the notion that the Ifg gene is involved in the resistance/susceptibility of inbred strains of mice to persistent infection by this picornavirus.

Gli stili APA, Harvard, Vancouver, ISO e altri

35

Hata, Hidekazu, e Somei Kojima. "Susceptibility of Inbred Mice to Paragonimus miyazakii Infection". Journal of Parasitology 75, n. 3 (giugno 1989): 463. http://dx.doi.org/10.2307/3282607.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

36

Postan, Miriam, James A. Dvorak, James J. Bailey, Erik W. Pottala e James P. McDaniel. "Studies of Trypanosoma cruzi Clones in Inbred Mice". American Journal of Tropical Medicine and Hygiene 37, n. 3 (1 novembre 1987): 541–49. http://dx.doi.org/10.4269/ajtmh.1987.37.541.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

37

Gatti, Daniel M., Lu Lu, Robert W. Williams, Wei Sun, Fred A. Wright, David W. Threadgill e Ivan Rusyn. "MicroRNA expression in the livers of inbred mice". Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 714, n. 1-2 (settembre 2011): 126–33. http://dx.doi.org/10.1016/j.mrfmmm.2011.05.007.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

38

Katafuchi, T., Y. Hattori e K. Koizumi. "Mechanism of water intake in inbred polydipsic mice". Appetite 12, n. 3 (giugno 1989): 217. http://dx.doi.org/10.1016/0195-6663(89)90203-1.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

39

Kiupel, Matti, Kathy S. Brown e John P. Sundberg. "Bulbourethral (Cowper's) gland abnormalities in inbred laboratory mice". Journal of Experimental Animal Science 40, n. 4 (gennaio 2000): 178–88. http://dx.doi.org/10.1016/s0939-8600(00)80010-3.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

40

Tuttle, Alexander H., Vivek M. Philip, Elissa J. Chesler e Jeffrey S. Mogil. "Comparing phenotypic variation between inbred and outbred mice". Nature Methods 15, n. 12 (30 novembre 2018): 994–96. http://dx.doi.org/10.1038/s41592-018-0224-7.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

41

Frelier, P. F., J. W. Templeton, M. Estes, H. W. Whitford e R. D. Kienle. "Genetic Regulation ofMycobacterium paratuberculosisInfection in Recombinant Inbred Mice". Veterinary Pathology 27, n. 5 (settembre 1990): 362–64. http://dx.doi.org/10.1177/030098589002700509.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

42

Adkison, D. L., e J. P. Sundberg. "“Lipomatous” Hamartomas and Choristomas in Inbred Laboratory Mice". Veterinary Pathology 28, n. 4 (luglio 1991): 305–12. http://dx.doi.org/10.1177/030098589102800407.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

43

Downing, Chris, Christina Balderrama-Durbin, Hali Broncucia, David Gilliam e Thomas E. Johnson. "Ethanol Teratogenesis in Five Inbred Strains of Mice". Alcoholism: Clinical and Experimental Research 33, n. 7 (luglio 2009): 1238–45. http://dx.doi.org/10.1111/j.1530-0277.2009.00949.x.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

44

Harmon, Kelley J., Leslie L. Couper e Volkhard Lindner. "Strain-Dependent Vascular Remodeling Phenotypes in Inbred Mice". American Journal of Pathology 156, n. 5 (maggio 2000): 1741–48. http://dx.doi.org/10.1016/s0002-9440(10)65045-6.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

45

Smith, Donald F. "Lithium Chloride Toxicity and Pharmacodynamics in Inbred Mice". Acta Pharmacologica et Toxicologica 43, n. 1 (13 marzo 2009): 51–54. http://dx.doi.org/10.1111/j.1600-0773.1978.tb02231.x.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

46

Fitch, W., e W. Atchley. "Evolution in inbred strains of mice appears rapid". Science 228, n. 4704 (7 giugno 1985): 1169–75. http://dx.doi.org/10.1126/science.4001935.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

47

Zuo, Baofen, Xiaoyan Du, Jing Zhao, Huixin Yang, Chao Wang, Yanhua Wu, Jing Lu, Ying Wang e Zhenwen Chen. "Analysis of Microsatellite Polymorphism in Inbred Knockout Mice". PLoS ONE 7, n. 4 (11 aprile 2012): e34555. http://dx.doi.org/10.1371/journal.pone.0034555.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

48

Zhong, Su, e Colin Dobson. "Heligmosomoides polygyrus:Resistance in Inbred, Outbred, and Selected Mice". Experimental Parasitology 82, n. 2 (marzo 1996): 122–31. http://dx.doi.org/10.1006/expr.1996.0016.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

49

Seredenin, S. B., T. G. Khlopushina e V. P. Zherdev. "Pharmacokinetics and metabolism of antipyrine in inbred mice". Pharmaceutical Chemistry Journal 24, n. 9 (settembre 1990): 620–23. http://dx.doi.org/10.1007/bf00767024.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

50

Trier, Lotte, e Bent Rubin. "BONE MARROW TRANSPLANTATION IN INBRED STRAINS OF MICE". Acta Pathologica Microbiologica Scandinavica Section B Microbiology and Immunology 82B, n. 5 (15 agosto 2009): 724–32. http://dx.doi.org/10.1111/j.1699-0463.1974.tb00242.x.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Offriamo sconti su tutti i piani premium per gli autori le cui opere sono incluse in raccolte letterarie tematiche. Contattaci per ottenere un codice promozionale unico!