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Letteratura scientifica selezionata sul tema "IgE"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 11 marzo 2023

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Articoli di riviste sul tema "IgE"

Mastrangelo, G., C. Veller Fornasa, S. Pavanello, G. Marcer, M. Lazzaro, G. Milan, E. Fadda, U. Fedeli e E. Clonfero. "Polyaromatic Hydrocarbons Administered in Humans by Dermal Route Increase Total IgE". International Journal of Immunopathology and Pharmacology 16, n. 2 (maggio 2003): 145–50. http://dx.doi.org/10.1177/039463200301600208.

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Inhalation of polyaromatic hydrocarbons (PAHs) extracted from diesel exhaust particles (DEP) enhances local (nasal) production of IgE in humans. The aim of the present research is to investigate whether in humans dermal exposure to PAHs which are not extracted from DEPs increases serum IgE and whether host factors modify the immunologic effect. In thirty-two patients with acute psoriatic lesions, a cream containing 3% of coal tar (which holds a variety of PAHs) was applied to the skin for 24 hours. Serum IgE were measured before (IgE0) and four (IgE4) and eight (IgE8) days after application. Replicated means were compared by analysis of variance for repeated measures and by the Newman-Keuls' test. IgE0, IgE4 and IgE8 were 151.19, 159.69 (a 6% excess) and 170.90 kU/L (a 13% excess) respectively; pairwise comparison showed IgE8 was significantly higher than IgE0 (p<0.05). At multiple linear regression analysis, the percentage increase in serum IgE across observation days was the dependent variable against age, sex, cigarettes/day, urinary 1-pyrenol, atopy, skin area treated, and grams of cream. Of the independent variables, only age had a significant (p<0.028) influence: the younger the age, the higher the IgE response to PAHs. We conclude that whatever the source and the route of entry (skin or respiratory tract), PAHs increase total serum IgE, mainly in younger age groups.

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Ledford, Dennis K. "Increased IgE in IgA Deficiency". Journal of Allergy and Clinical Immunology: In Practice 5, n. 6 (novembre 2017): 1795. http://dx.doi.org/10.1016/j.jaip.2017.07.013.

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Kolopp-Sarda, M. N., e G. C. Faure. "Pourquoi les immunoglobulines s’appellent-elles IgG, IgA, IgM, IgD et IgE ?" Revue Francophone des Laboratoires 2016, n. 484 (luglio 2016): 57–58. http://dx.doi.org/10.1016/s1773-035x(16)30250-7.

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Abbal, Michel. "IgE totales et IgE spécifiques". Bio Tribune Magazine 6, n. 1 (giugno 2003): 20–22. http://dx.doi.org/10.1007/bf03010285.

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Corrado, Alessia, Richard P. Ramonell, Matthew C. Woodruff, Christopher Tipton, Sarah Wise, Joshua Levy, John DelGaudio et al. "Extrafollicular IgD+ B cells generate IgE antibody secreting cells in the nasal mucosa". Mucosal Immunology 14, n. 5 (28 maggio 2021): 1144–59. http://dx.doi.org/10.1038/s41385-021-00410-w.

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AbstractIncreased IgE is a typical feature of allergic rhinitis. Local class-switch recombination has been intimated but B cell precursors and mechanisms remain elusive. Here we describe the dynamics underlying the generation of IgE-antibody secreting cells (ASC) in human nasal polyps (NP), mucosal tissues rich in ASC without germinal centers (GC). Using VH next generation sequencing, we identified an extrafollicular (EF) mucosal IgD+ naïve-like intermediate B cell population with high connectivity to the mucosal IgE ASC. Mucosal IgD+ B cells, express germline epsilon transcripts and predominantly co-express IgM. However, a small but significant fraction co-express IgG or IgA instead which also show connectivity to ASC IgE. Phenotypically, NP IgD+ B cells display an activated profile and molecular evidence of BCR engagement. Transcriptionally, mucosal IgD+ B cells reveal an intermediate profile between naïve B cells and ASC. Single cell IgE ASC analysis demonstrates lower mutational frequencies relative to IgG, IgA, and IgD ASC consistent with IgE ASC derivation from mucosal IgD+ B cell with low mutational load. In conclusion, we describe a novel mechanism of GC-independent, extrafollicular IgE ASC formation at the nasal mucosa whereby activated IgD+ naïve B cells locally undergo direct and indirect (through IgG and IgA), IgE class switch.

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Sabra, Aderbal, Joseph A. Bellanti, Jonathan M. Rais, Henry J. Castro, Julia Mendez de Inocencio e Selma Sabra. "IgE and non-IgE food allergy". Annals of Allergy, Asthma & Immunology 90, n. 6 (giugno 2003): 71–76. http://dx.doi.org/10.1016/s1081-1206(10)61664-x.

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Boltansky, Howard, Jean Dyer, Steve Esworthy e Michael Kaliner. "IgE-immunotoxins. I. IgE-intact ricin". Immunopharmacology 14, n. 1 (settembre 1987): 35–45. http://dx.doi.org/10.1016/0162-3109(87)90007-5.

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Xie, Liping, John T. Schroeder, Jacqueline M. Langdon, Rebecca S. Sora-Scott, Toshiaki Kawakami e Susan M. MacDonald. "Human IgE+ and IgE− are not equivalent to mouse highly cytokinergic IgE". Journal of Allergy and Clinical Immunology 121, n. 4 (aprile 2008): 1027–33. http://dx.doi.org/10.1016/j.jaci.2007.12.1157.

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DECLERCK, L., M. WESTEDT, A. CATS, B. VERMEER, E. WELTEVREDEN, C. BRIDTS e W. STEVENS. "391 IgE, IgE-containing circulating immune complexes (IgE-CIC) IgE-rheumatoid factor (RF) and skin IgE-deposition in rheumatoid arthritis (RA) patients (pts)". Journal of Allergy and Clinical Immunology 75, n. 1 (gennaio 1985): 202. http://dx.doi.org/10.1016/0091-6749(85)90527-5.

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Pankovics, Gergő. "Az ige jelentésének összefüggése az igei vonzatstruktúrával". Magyar Nyelvőr 145, n. 2 (2021): 211–19. http://dx.doi.org/10.38143/nyr.2021.2.211.

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Più fonti

Tesi sul tema "IgE"

Suzuki, Lisandra Akemi. "Resposta imune humoral na neurocisticercose". [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308741.

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Orientador: Claudio Lucio Rossi
Tese (doutorado) - Universidade Estadual de Campinas. Faculdade de Ciencias Medicas
Made available in DSpace on 2018-08-15T09:51:57Z (GMT). No. of bitstreams: 1 Suzuki_LisandraAkemi_D.pdf: 1217880 bytes, checksum: 4ceb1f773f112c88145bc69e02976ef2 (MD5) Previous issue date: 2010
Resumo: A neurocisticercose (NC) e uma importante causa de doença neurológica em muitos paises em desenvolvimento, incluindo o Brasil. O diagnostico clinico da NC e dificultado pelo polimorfismo e pela não especificidade dos sintomas. As tecnicas de neuroimagem e pesquisa de anticorpos específicos tem contribuído para o diagnostico da NC e uma melhor compreensão dos processos fisiopatológicos dessa infecção. O presente trabalho teve como objetivo avaliar, por meio de técnicas imunoenzimaticas (ELISA), a resposta imune humoral na NC, utilizando como preparações antigênicas o liquido vesicular (LV) e uma fração glicoproteica obtida do extrato bruto de cisticercos de Taenia solium (T. solium) com afinidade por lentil-lectina (fração Gp). Cinquenta e seis amostras de liquido cefalorraquidiano (LCR), 22 de pacientes com NC e 34 de pacientes com outros problemas neurológicos, foram utilizadas para a pesquisa de IgG e suas subclasses, com os seguintes resultados: IgG-LV: 100% de sensibilidade e especificidade; IgG1 -LV: 72,73% de sensibilidade e 100% de especificidade; IgG2-LV: 81,81% de sensibilidade e 100% de especificidade; IgG3-LV: 59,09% de sensibilidade e 97,06% de especificidade; IgG4-LV: 90,91% de sensibilidade e 97,06% de especificidade; IgG-fração Gp: 90,91% de sensibilidade e 97,06% de especificidade; IgG1-fração Gp: 59,09% de sensibilidade e 91,18% de especificidade; IgG2-fração Gp: 68,18% de sensibilidade e 94,12% de especificidade; IgG3-fração Gp: 36,36% de sensibilidade e 100% de especificidade; IgG4-fração Gp: 86,36% de sensibilidade e 100% de especificidade. Quarenta e sete amostras de LCR, 16 de pacientes com NC e 31 de pacientes com outros problemas neurológicos foram utilizadas para a pesquisa de IgE, com os seguintes resultados: IgE-LV e IgE-fração Gp: 93,75% de sensibilidade e 100% de especificidade. Cinquenta e sete amostras de soros, 22 de pacientes com NC, 18 de pacientes com outras infecções e 17 de pessoas presumivelmente sadias, foram utilizadas para a pesquisa da IgG e suas subclasses, IgE, IgA e IgM, com os seguintes resultados: IgG-LV: 100% de sensibilidade e especificidade; IgG1-LV: 86,36% de sensibilidade e 94,28% de especificidade; IgG2-LV: 90,91% de sensibilidade e 97,14% de especificidade; IgG3-LV: 86,36% de sensibilidade e 97,14% de especificidade; IgG4-LV: 100% de sensibilidade e de especificidade; IgG-fração Gp: 95,45% de sensibilidade e 100% de especificidade; IgG1-fração Gp: 63,64% de sensibilidade e 94,28% de especificidade; IgG2-fração Gp: 68,18% de sensibilidade e 97,14% de especificidade; IgG3-fração Gp: 54,54% de sensibilidade e 88,57% de especificidade; IgG4-fração Gp: 90,91% de sensibilidade e 100% de especificidade; IgELV: 90,91% de sensibilidade e 97,14% de especificidade; IgE-fração Gp: 86,36% de sensibilidade e 100% de especificidade; IgA-LV: 54,54% de sensibilidade e 94,28% de especificidade; IgA-fração Gp: 13,63% de sensibilidade e 100% de especificidade. Anticorpos IgM não foram detectados com as preparações de LV e fração Gp. Nossos resultados mostraram que, com ambas as preparações antigênicas, tanto em amostras de LCR quanto em amostras de soros, a maior positividade foi obtida na detecção de anticorpos das classes IgG e IgE, seguida da positividade da IgA. Anticorpos IgM não foram detectados em amostras de soros com reações de ELISA realizadas com LV e fração Gp. Com relação as subclasses da IgG, a IgG4 apresentou, tanto em amostras de LCR como em amostras de soros, valores de positividade e concentração iguais ou superiores as outras subclasses. As reações ELISA realizadas com LV mostraram sensibilidades iguais ou superiores aquelas obtidas com a fração Gp. Considerando a complexidade e o custo final da obtenção da fração Gp, o LV pode ser considerado mais adequado para a pesquisa de anticorpos em amostras de LCR e soros de pacientes com NC.
Abstract: Neurocysticercosis (NC) is an important cause of neurological disease in many developing countries, including Brazil. The clinical diagnosis of NC is hindered by the polymorphism and non-specificity of the symptoms. Neuroimaging techniques and detection of specific antibodies have contributed to the diagnosis of NC and a better understanding of the physiopathological processes of this infection. The purpose of this study was to evaluate the humoral immune response in NC by using immunoenzymatic techniques (ELISA) in which vesicular fluid (VF) and a glycoprotein fraction purified from a crude extract of Taenia solium cysticerci with affinity for lentil-lectin (fraction Gp) were used as antigenic preparations. Fifty-six cerebrospinal fluid (CSF) samples, 22 from patients with NC and 34 from patients with other neurological disorders, were assayed for IgG and IgG subclasses, with the following results: IgG-VF: 100% sensitivity and specificity, IgG1 - VF: 72.73% sensitivity and 100% specificity, IgG2 -VF: 81.81% sensitivity and 100% specificity, IgG3 -VF: 59.09% sensitivity and 97.06% specificity, IgG4 -VF: 90.91% sensitivity and 97.06% specificity, IgG-fraction Gp: 90.91% sensitivity and 97.06% specificity, IgG1- fraction Gp: 59.09% sensitivity and 91.18% specificity, IgG2-fraction Gp: 68.18% sensitivity and 94.12% specificity, IgG3 -fraction Gp: 36.36% sensitivity and 100% specificity, IgG4 - fraction Gp: 86.36% sensitivity and 100% specificity. Forty-seven CSF samples, 16 from patients with NC and 31 from patients with other neurological disorders, were assayed for IgE, with the following results: IgE-VF and IgE-fraction Gp: 93.75% sensitivity and 100% specificity. Fifty-seven serum samples, 22 from patients with NC, 18 from patients with other infections and 17 from presumably healthy individuals, were assayed for IgG, IgG subclasses, IgE, IgA and IgM, with the following results: IgG-VF: 100% sensitivity and specificity, IgG1-VF: 86.36% sensitivity and 94.28% specificity, IgG2 -VF: 90.91% sensitivity and 97.14% specificity, IgG3 -VF: 86.36% sensitivity and 97.14% specificity, IgG4 -VF:100% sensitivity and specificity, IgG-fraction Gp: 95.45% sensitivity and 100% specificity, IgG1- fraction Gp: 63.64% sensitivity and 94.28% specificity, IgG2 -fraction Gp: 68.18% sensitivity and 97.14% specificity, IgG3 -fraction Gp: 54.54% sensitivity and 88.57% specificity, IgG4 - fraction Gp: 90.91% sensitivity and 100% specificity, IgE-VF: 90.91% sensitivity and 97.14% specificity, IgE-fraction Gp: 86.36% sensitivity and 100% specificity, IgA-VF: 54.54% sensitivity and 94.28% specificity, IgA-fraction Gp: 13.63% sensitivity and 100% specificity. No specific IgM antibodies were detected with VF and fraction Gp antigenic preparations. These results show that with the two antigenic preparations the highest positivity in CSF and serum samples was obtained for IgG and IgE antibodies, followed by positivity for IgA. No IgM antibodies were detected in serum samples assayed with VF and fraction Gp. With regard to IgG subclasses, IgG4 positivity and concentration in CSF and serum samples were higher than or equal to the other subclasses. ELISA reactions done with VF showed equal or higher sensitivities than those obtained with fraction Gp. Considering the complexity and high cost of obtaining fraction Gp, VF could be more suitable for detecting specific antibodies in CSF and serum samples from patients with NC.
Doutorado
Ciencias Biomedicas
Doutor em Ciências Médicas

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Hjelm, Fredrik. "Early Immunostimulatory Effects of IgE- and IgG Antibodies". Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7209.

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Renault, Neil. "Construction, method development and comparative testing of an 'All-Diet' protein microarray to measure IgA, IgM, IgG and IgE in human sera and milk". Thesis, University of Nottingham, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503929.

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Existing immunoglobulin (Ig) tests only give a limited picture of the immunological response to food antigens. Furthermore, existing tests require large volumes of sample, over a limited number of foods, are not amenable to a high sample through-put system and the results are limited to normally just one immunoglobulin class. In order to investigate the global immune response towards food products we have developed the "all diet" microarray concept. The "all-diet food protein microarray contains extracts of over 400 food ingredients that cover most of the food products found in the UK. Using this system we have retrospectively determined food specific IgE, IgA, IgG and IgM from 17 well characterized sera. The results were analyzed by multivariate techniques and parametric methods. The proof-of-concept of the ''all diet microarray to investigate the relationships between food antigen specificity and multiple Ig type was demonstrated here. The novelity of this protein microarray is the use of arrayed food samples sequentially extracted with detergent and chaotropic agents. The array system possesses many advantages over traditional systems such as requirement of low sample volume, high sensitivity and a global view of the immune response. Notwithstanding these potential advantages to clinical practices, these benefits remain yet to be demonstrated. The development of the technique will allow further expansion into areas of research such as conjugation of the microarray with sensitized human basophils and also immunoglobulin binding to extracts of parasites.

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Gilbert, Sophie. "Studies on feline IgE". Thesis, University of Edinburgh, 1998. http://hdl.handle.net/1842/29776.

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Ten normal cats were immunised with Dermatophagoides farinae (DF) antigen and intradermal skin tests (IDSTs) were performed weekly. Sera from the latter were also assessed for DF-specific IgE by ELISA and PK tests. Detectable DF-specific IgE was induced in all of the 10 cats, however levels were found not to be correlated with the development of positive IDSTs nor with the levels of IgE as assessed by PK tests. Sera from 10 cats with symptoms consistent with atopy, from 15 normal household cats and from 11 laboratory maintained cats were assessed for allergen-specific IgE and IgG to DF by ELISA. Although DF-specific IgE was detectable in all the atopic cats, there was no significant difference between the levels in this group and in the clinically normal household cats. However levels in both these groups were significantly higher than those in the laboratory maintained cats. The influence of vaccination and endoparasitism on the IgE response to a food antigen was assessed in 34 cats. Seventeen kittens experimentally infected with T. cati and 17 parasite-free kittens were dosed with human serum albumin (HSA) daily for 3 weeks. Seven cats from both groups were given two injections of a live attenuated viral vaccine. The group of parasitised cats has significantly higher levels of HSA-specific IgE, IgG and IgA than did the group of parasite-free cats. Vaccinated cats had also higher levels than non vaccinated cats but only in the group of parasite-free cats. None of the cats developed clinical signs of food allergy.

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Ding, Zhoujie. "Feedback Enhancement of Immune Responses by IgE, IgM, and IgG3 Antibodies". Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-237337.

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Antibodies can enhance or suppress the immune responses against their specific antigens. This phenomenon is known as antibody-mediated feedback regulation. We have studied the mechanisms underlying IgE-, IgM-, and IgG3-mediated enhancement of immune responses in mouse models using intravenous immunization. We attempted to answer the following questions: 1) Which cell type presents IgE-complexed antigens to CD4+ T cells? 2) Is complement activation required for specific IgM to enhance antibody responses? 3) Does IgM enhance CD4+ T-cell responses? 4) How are IgG3-antigen complexes transported into B-cell follicles? We found that CD23+ B cells transporting IgE-antigen complexes into B-cell follicles were not required to prime the antigen-specific CD4+ T cells in vivo, whereas CD11c+ cells were indispensable. After examining the three most common subpopulations of CD11c+ cells in the spleen, we determined that it was CD8α- conventional dendritic cells migrating into the T-cell zone following immunization that presented IgE-complexed antigens to CD4+ T cells. Next, we showed that specific IgM from Cµ13 mice, which is unable to activate complement, failed to enhance either antibody or germinal center responses whereas wild-type IgM enhanced both responses. Therefore, specific IgM must activate complement to enhance humoral responses. In addition, wild-type IgM did not up-regulate CD4+ T-cell responses. Finally, we showed that IgG3-antigen complexes were transported by marginal zone B cells into B-cell follicles via binding to complement receptors 1 and 2 (CR1/2) on those cells. The immune complexes were captured by follicular dendritic cells as early as 2 h after immunization. Germinal center responses were also enhanced by IgG3. Using bone marrow chimeric mice, we found that CR1/2 expression was required on both marginal zone B cells and follicular dendritic cells to provide an optimal enhancement of antibody responses.

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Ashburn, David. "The relevance of IgA and IgE assays, IgG avidity and western blotting in the diagnosis of Toxoplasma infection". Thesis, University of Aberdeen, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361776.

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To improve diagnosis of Toxoplasma gondii in difficult patient groups, IgA- and IgE-immunosorbent agglutination assays (ISAGA), IgG avidity and Western blotting were developed and assessed. In the ISAGA, rabbit anti-human IgA or anti-human IgE (for the IgA-ISAGA and IgE-ISAGA respectively) was adsorbed onto microtitre plates; formaldehyde fixed tachyzoites were used to identify specific antibody. Both ISAGAs were specific; only 1/482 (0.2%) and 1/513 (0.19%) false positive results were recorded for the IgA and IgE-ISAGA respectively. Both were produced early in infection and were detected for up to 11 (IgA) and 10 (IgA) months. In the avidity ELISA, the performance of excretory/secretory, surface, cytoplasmic and mixed antigen was similar when tested with sera from patients with known duration of infection. Thirteen pregnant women were tested. Specific IgA was detected in all and so was not useful to time infection but specific IgE was absent in 5 women and may therefore have been useful to exclude recent infection. Specific IgE however, was not detected in one woman who seroconverted; detection of IgE may indicate severity of infection. High IgG avidity was measured in 4 patients and could have been useful in conjunction with IgE to exclude recent infection. In immunocompromised patients, IgA and IgE were detected with similar frequency to IgM, but their presence in some IgM negative patients makes them a useful addition to the repertoire of testing. High avidity in immunocompromised patients was not of use to improve diagnosis. Using Western blotting it was possible to differentiate between acute (< 4 months) and recent (4-10 months) infection. This test was less useful for timing infection of infection in pregnancy.

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Zimmer, Anja. "Futtermittel-spezifisches IgG und IgE vor und nach Eliminationsdiäten bei allergischen Hunden". Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-148673.

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Guerra, Fernanda Garcia. "Anticorpos igg e ige para auto-antígenos nucleares no lúpus eritematoso sistêmico". Instituto de Ciências da Saúde, 2005. http://repositorio.ufba.br/ri/handle/ri/21734.

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CNPq; PPGIM – UFBA
O Lúpus Eritematoso Sistêmico (LES) é uma doença reumática autoimune, classificada como uma reação de hipersensibilidade tipo III, que cursa com exuberante produção de auto-anticorpos de diferentes especificidades, e reação inflamatória crônica. O LES acomete predominantemente pessoas do sexo feminino, com prevalência de 5-50 casos/100.000. Contudo, estudos epidemiológicos têm mostrado diferenças raciais na prevalência e aspectos clínicos e laboratoriais do LES. Diferenças têm sido demonstradas principalmente na freqüência dos auto-anticorpos contra antígenos nucleares extraíveis (ENA, extractable nuclear antigens) e de anticorpos IgG anti-DNA fita dupla. Existem poucos dados sobre a prevalência destes auto-anticorpos em pacientes brasileiros, principalmente usando imunoensaios sensíveis. Assim, neste estudo foram investigadas as freqüências de anticorpos antinúcleo dos isotipos IgG e IgE com especificidade antigênica para as proteínas nucleares SSA, SSB, Sm e U1-RNP, além de anticorpos IgG anti-DNA fita dupla. Soros de 21 pacientes do sexo feminino e de 26 doadoras sadias, idade entre 15-65 anos foram inicialmente triados para a presença de anticorpos antinucleares IgG e IgE através de reação de imunofluorescência indireta (IFI, FAN-IgG e FAN-IgE) com células HEp-2. Anticorpos IgG anti-DNA fita dupla, e IgG e IgE anti-ENA foram investigados por técnica de ELISA (Enzyme-Linked Immunosorbent Assay) indireto, usando fase sólida coberta com os autoantígenos purificados. A concentração sérica de IgE foi determinada por ELISA de captura. Todos os soros dos pacientes com LES (100%) foram reativos no teste de FAN-IgG (mediana do título = 640), enquanto 15/21 (71%) amostras reagiram no teste de FAN-IgE. Anticorpos IgG anti-DNA fita dupla foram detectados em 12/21 (52%) soros (mediana do título = 777 UI/ml, sensibilidade de 35,5%). Quatorze soros reagiram em ELISA-IgG para anticorpos anti-ENA, apresentando as seguintes sensibilidades: anti-RNP = 40%; anti-SSA e anti-Sm = 20%, e anti SSB = 10,5%. Anticorpos IgE anti-ENA foram detectados em sete soros, apresentando o teste de ELISA-IgE uma sensibilidade de 2,5% para 13 anticorpos anti-SSA e SSB, e de 5,3% e 17,6% para anti-Sm e anti-RNP, respectivamente. Os testes de ELISA-IgE para anticorpos anti-Sm e anti-SSA mostraram 100% de especificidade, enquanto uma especificidade de 95% foi encontrada para os testes com anticorpos anti-SSB e anti-RNP. Um aumento na concentração de IgE sérica for observado em 6 (29%) das amostras (mediana = 426 UI/ml), existindo uma correlação positiva entre os títulos de FAN-IgG e IgG anti-RNP (r = 0.796, P = 0.002), entre os títulos de IgG e IgE anti-RNP (r = 0.594, P = 0.0045) e também entre IgE anti-RNP e IgE total (r = 0.680, P = 0.0007). Concluindo, a freqüência de FAN-IgG e anticorpos IgG anti- SSA, SSB e anti-Sm nos pacientes brasileiros com LES concordou com os resultados de outros estudos internacionais, existindo contudo uma forte predominância de anticorpos anti-RNP nestes indivíduos.

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Bracher, Marguerite. "IgE in immunotherapy of cancer". Thesis, King's College London (University of London), 2006. https://kclpure.kcl.ac.uk/portal/en/theses/ige-in-immunotherapy-of-cancer(08abceea-54a8-436c-9504-24742d57538d).html.

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Karnowski, Alexander. "Post-transcriptional regulation of IgE". [S.l. : s.n.], 2004. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB10990069.

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Più fonti

Libri sul tema "IgE"

Penichet, Manuel L., e Erika Jensen-Jarolim, a cura di. Cancer and IgE. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-451-7.

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Kim, Tong-gil. MB ... ige mwŏmnikka. [Seoul]: Chʻŏng Midiŏ, 2009.

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L, Spiegelberg Hans, a cura di. IgE immunopathology I. New York: Springer Verlag, 1990.

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Tong-gil, Kim. MB ... ige mwŏmnikka. [Seoul]: Chʻŏng Midiŏ, 2009.

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Bruna, Dick. Ŏmma, ige mwŏya? Sŏul Tʻŭkpyŏlsi: Chigyŏngsa, 1993.

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Nam, Mun-hŭi. Ǒmma~ ige mwŏya? Sŏul: Pukk'iang, 2005.

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Chang, Kang-myŏng. Ch'aek, ige mwŏrago. Kyŏnggi-do P'aju-si: Arŭt'e, 2020.

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Tong-gil, Kim. MB ... ige mwŏmnikka. [Seoul]: Chʻŏng Midiŏ, 2009.

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M, MacDonald Susan, a cura di. IgE immunopathology II. Berlin: Springer International, 1993.

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Endre, Gyökössy. Élet és ige. Budapest: Budapest Szabadságtéri Református Egyház, 1990.

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Più fonti

Capitoli di libri sul tema "IgE"

Mehlhorn, Heinz. "IgE". In Encyclopedia of Parasitology, 1324. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4701.

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Mehlhorn, Heinz. "IgE". In Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4701-1.

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Gooch, Jan W. "IgE". In Encyclopedic Dictionary of Polymers, 900. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13976.

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van Balen, J. A. M., A. A. Demeulemeester, M. Frölich, K. Mohrmann, L. M. Harms, W. C. H. van Helden, L. J. Mostert e J. H. M. Souverijn. "IgE". In Memoboek, 138. Houten: Bohn Stafleu van Loghum, 2012. http://dx.doi.org/10.1007/978-90-313-9129-5_75.

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Gartler, Stanley M., R. Scott Hansen, Vinzenz Oji, Heiko Traupe, Julia Horn, Bodo Grimbacher, Srijita Sen-Chowdhry et al. "IGE". In Encyclopedia of Molecular Mechanisms of Disease, 1031. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8932.

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Poslussny, P., K. Vinzenz e F. Zekert. "Serumimmunglobuline (IgA, IgE, IgG, IgM) bei Patienten mit Kopf-Halskarzinomen". In Chirurgische Therapie von Kopf-Hals-Karzinomen, 335–42. Vienna: Springer Vienna, 1992. http://dx.doi.org/10.1007/978-3-7091-9087-6_39.

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van de Veen, Willem. "IgE Receptor". In Encyclopedia of Medical Immunology, 386–89. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_75.

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van de Veen, Willem. "Specific IgE". In Encyclopedia of Medical Immunology, 622–24. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_77.

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Eiwegger, Thomas, e Zsolt Szépfalusi. "IgE Testing". In Encyclopedia of Medical Immunology, 389–92. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_78.

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Sicherer, Scott H. "IgE- and Non-IgE-Mediated Food Allergy". In Eosinophilic Esophagitis, 219–38. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-515-6_16.

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Atti di convegni sul tema "IgE"

Jones, Thomas, Victoria Jeffery, Scott Elliott, Daniel Neville, Ellie Lanning, Thomas Brown e Anoop Chauhan. "Total IgE thresholds for detection of a positive specific IgE". In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa1087.

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Hoffmann, Hans Jürgen, Pernille M. Frandsen, Peter Oluf Schiøtz e Ronald Dahl. "Modulation By IgE And Anti-IgE Of FceRI Expression On Cultured Mast Cells". In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1431.

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Woolnough, Kerry, Catherine Pashley e Andrew Wardlaw. "LATE-BREAKING ABSTRACT:Aspergillus fumigatusspecific IgE, but not total IgE orA. fumigatusspecific IgG is associated with evidence of lung damage in asthma". In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4357.

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Schneider, F., e T. Breuer. "Kalter Orbitalabszess bei Hyper-IgE-Syndrom". In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686687.

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Mouthuy, Jonathan, Bruno Detry, Françoise Pirson e Charles Pilette. "Local IgE In Asthma: Presence Of D. Pteronyssinus-specific IgE In Induced Sputum From Intrinsic Asthma Patients". In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5623.

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Tora, I. "Idiopathic Pulmonary Fibrosis with Extremely Elevated IgE". In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6349.

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Rohit, Chirag V., Pranav B. Darji e Hitesh R. Jariwala. "Mitigation of IGE using STATCOM with DFIGs". In 2018 IEEE International Conference on Power Electronics, Drives and Energy Systems (PEDES). IEEE, 2018. http://dx.doi.org/10.1109/pedes.2018.8707856.

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Szczeklik, A., K. Sladek e J. Dropinski. "IgE-MEDIATED IMMUNOLOGICAL RESPONSE IN MYOCARDIAL INFARCTION". In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643022.

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Mast cells have been described in adventitia of coronary arteries; their number increases with progression of atherosclerosis. We reasoned that, if these cells were to contribute to cardiac pathology, the signals turning them on should be detectable in the blood. We, therefore, measured systematically serum IgE in 25 patients (18 men and 7 women, mean age 55 years) with unequivocal diagnosis of recent transmural myocardial infarction. All patients survived. When specifically interviewed, only two gave a history compatible with atopy. In 14 patients, whose initial values ranged from 44-750 IU/ml, and on average 172 IU/ml, serum IgE increased by more than 25%. It raised gradually after the infarction,reaching the peak by 7th day (mean increase by 92%), then began to decrease, but was still elevated on 14th and 21st day. Such behaviour was in striking contrast to mean serum IgG which at no time showed changes differing by more than 10% from the initial values. Thus, in at least half of the patients with acute myocardial infarction there occurs an immunological reaction characterized by distinct release of IgE into circulatory blood. It might be that IgE sensitizes mast cells and facilitates the release of chemical mediators. Histamine, prostaglandin D2 and sulfido-peptide leukotrienes are all powerful vasoconstrictors of human arteries. On the other hand, heparin exerts potent anticoagulant effects through activation of antithrombin III, and PGD2 inhibits platelet aggregation. This complex interplay of the mediators at the target site might affect blood clotting, cardiac function and the course of myocardial infarction.

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Clerc, Sebastien, Denis Caillaud, Cara Maesano, Joana Vitte e Isabella Annesi-Maesano. "Specific IgE sensitization and asthma in French farmers". In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa369.

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Chu, Seung Y., Holly M. Horton, Hsing Chen, Sher Karki, Irene Leung, David E. Szymkowski e John R. Desjarlais. "Suppression Of IgE Production By XmAb7195, An Fc-Engineered Antibody That Specifically Coengages Inhibitory Receptor Fc?RIIb With IgE-BCR". In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4068.

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Rapporti di organizzazioni sul tema "IgE"

He, Dan, Hongmei Wu, Yujie Han, Min Liu e Mao Lu. A meta-analysis of topical antifungal drugs to treat atopic dermatitis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, dicembre 2021. http://dx.doi.org/10.37766/inplasy2021.12.0062.

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Review question / Objective: Various bacteria and fungi colonize the skin surface of patients with AD. The colonized fungi mainly include Malassezia, non-Malassezia yeasts, and molds. Among them, Malassezia occupies 63%~86% of the fungal colonization community on the skin surface of AD patients. Although the relationship between the level of Malassezia on the skin surface and disease severity remains controversial, many studies have shown that the level of serum anti-Malassezia-specific immunoglobulin E (IgE) antibodies in AD patients is related to the disease severity, especially in patients with AD in the head and neck. The specific mechanism by which Malassezia causes or aggravates AD is unclear, but damage to the skin barrier in AD patients is a key component of the mechanism. The presence of Malassezia on the skin also seems to change its barrier function, resulting in more Malassezia and its antigens colonizing the skin surface area that is exposed to the immune system. This produces a large number of specific IgE antibodies and cytokines to aggravate the disease.

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Dallimore, S. R. Ice Bonding and Excess Ice. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1991. http://dx.doi.org/10.4095/132231.

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Coon, Max D. Sea Ice Model for Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, settembre 2003. http://dx.doi.org/10.21236/ada615524.

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Coon, Max D. Sea Ice Model for Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, settembre 2001. http://dx.doi.org/10.21236/ada626073.

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da Rosa, Maria Inês. Accuracy of rapid IgM and IgG Antibody Test for SARS-CoV-2 Infection Diagnosis: a systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review Protocols, aprile 2020. http://dx.doi.org/10.37766/inplasy2020.4.0099.

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Karig, Fred. Ice Camp. Fort Belvoir, VA: Defense Technical Information Center, settembre 1999. http://dx.doi.org/10.21236/ada629717.

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Wadhams, Peter. Wave-Ice Interaction and the Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, settembre 2013. http://dx.doi.org/10.21236/ada601220.

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Wadhams, Peter, e Martin Doble. Wave-Ice Interaction and the Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, settembre 2014. http://dx.doi.org/10.21236/ada617951.

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Ackley, S. F., T. Maksym e S. Stammerjohn. Wave-Ice and Air-Ice-Ocean Interaction During the Chukchi Sea Ice Edge Advance. Fort Belvoir, VA: Defense Technical Information Center, settembre 2013. http://dx.doi.org/10.21236/ada601218.

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Perovich, Don, e Bonnie Light. Sunlight, Sea Ice, and the Ice Albedo Feedback in a Changing Arctic Sea Ice Cover. Fort Belvoir, VA: Defense Technical Information Center, settembre 2013. http://dx.doi.org/10.21236/ada601068.

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