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Libri sul tema "Fetal growth retardation"

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Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 3 marzo 2023

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1

Jacques, Senterre, a cura di. Intrauterine growth retardation. [Vevey, Switzerland]: Nestlé Nutrition, 1989.

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2

Asim, Kurjak, e Beazley John M, a cura di. Fetal growth retardation: Diagnosis and treatment. Boca Raton, Fla: CRC Press, 1989.

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3

Y, Divon Michael, a cura di. Abnormal fetal growth. New York: Elsevier, 1991.

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4

A, Hanson Mark, Spencer John A. D e Rodeck C. H, a cura di. Growth. Cambridge: Cambridge University Press, 1995.

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5

1937-, Carrera José María, a cura di. Ultrasound and fetal growth. New York: Parthenon Pub. Group, 2001.

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6

Manning, F. A. Fetal medicine: Principles and practice. Norwalk, Conn: Appleton & Lange, 1995.

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7

Hamilton, Miriam. Sweet Caroline. Cork: Mercier Press, 2012.

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8

W, Kiess, Chernausek Steven D e Hokken-Koelega Anita C. S, a cura di. Small for gestational age: Causes and consequences. Basel: Karger, 2008.

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9

Mahendran, Dhushyanthan. Transcellular transport in human placenta: Changes in fetal growth retardation and during gestation. Manchester: University of Manchester, 1993.

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10

Detmer, Ann. Intrauterine growth retardation: An experimental study of fetal growth, regional blood flow and hepatic lipid metabolism in the anaesthetized guinea pig. Uppsala: Sveriges Lantbruksuniversitet, 1992.

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11

1932-, Battaglia Frederick C., Nestlé Nutrition Services e Nestlé Nutrition Workshop (39th : 1996 : East Sussex, England), a cura di. Placental function & fetal nutrition. [Vevey, Switzerland]: Nestlé Nutrition Services, 1997.

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12

Misra, Dawn Phillips. The effect of the hypertensive disorders of pregnancy upon fetal growth. [New York]: [Columbia University], 1993.

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13

Young, Maureen. What is baby expecting?: How we are fed to grow before we are born. Toft: M. Young, 2001.

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14

Anderson, Kim. Aboriginal approaches to fetal alcohol syndrome-effects. A cura di Ontario Federation of Indian Friendship Centres. Toronto, Ont: Ontario Federation of Indian Friendship Centres, 2002.

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15

Nametz, Patricia. Low birthweight in Wisconsin. [Madison, Wis.]: Wisconsin Dept. of Health and Social Services, Division of Health, Center for Health Statistics, 1989.

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16

Barker, D. J. P. Mothers, babies, and health in later life. 2^a ed. Edinburgh: Churchill Livingstone, 1998.

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17

Twilley, Leslie. Predictors of preterm and small-for-gestational-age births in Alberta: Report. Edmonton, Alta: Public Health Surveillance & Environmental Health, 2007.

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18

Das, Undurti N. A perinatal strategy for preventing adult disease: The role of long-chain polyunsaturated fatty acids. Boston, Mass: Kluwer Academic Publishers, 2002.

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19

Cohen, Alice Eve. What I thought I knew. New York: Viking, 2009.

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20

Institute of Medicine (U.S.). Committee to Study the Prevention of Low Birthweight. Preventing low birthweight: Summary. Washington, D.C: National Academy Press, 1985.

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21

Institute of Medicine (U.S.). Committee to Study the Prevention of Low Birthweight. Preventing low birthweight. Washington, D.C: National Academy Press, 1985.

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22

Cohen, Alice Eve. What I Thought I Knew. New York: Penguin USA, Inc., 2009.

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23

Kurjak, Asim, e J. M. Beazley. Fetal Growth Retardation. Taylor & Francis Group, 2019.

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24

Spencer, John A. D., Mark A. Hanson e Charles H. Rodeck. Growth. University of Cambridge ESOL Examinations, 2000.

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25

Kurjak, Asim, e J. M. Beazley. Fetal Growth Retardation: Diagnosis and Treatment. A cura di Asim Kurjak e John M. Beazley. CRC Press, 2020. http://dx.doi.org/10.1201/9781003068198.

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26

Kurjak, Asim, e J. M. Beazley. Fetal Growth Retardation: Diagnosis and Treatment. Taylor & Francis Group, 2020.

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27

Beazley, J. M., e Asim Kurjak. Fetal Growth Retardation: Diagnosis and Treatment. CRC, 1989.

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28

Kurjak, Asim, e J. M. Beazley. Fetal Growth Retardation: Diagnosis and Treatment. Taylor & Francis Group, 2020.

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29

Kurjak, Asim, e J. M. Beazley. Fetal Growth Retardation: Diagnosis and Treatment. Taylor & Francis Group, 2020.

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30

Kurjak, Asim, e J. M. Beazley. Fetal Growth Retardation: Diagnosis and Treatment. Taylor & Francis Group, 2020.

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31

Intrauterine growth retardation: A practical approach. Chicago: Year Book Medical Publishers, 1989.

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32

Gross, Thomas L., e Robert J. Sokol. Intrauterine Growth Retardation: A Practical Approach. Year Book Medical Publishers, Incorporated, 1989.

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33

(Editor), Mark A. Hanson, John A. D. Spencer (Editor) e Charles H. Rodeck (Editor), a cura di. Fetus and Neonate: Physiology and Clinical Applications. Cambridge University Press, 1995.

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34

(Editor), John Kingdom, Philip Baker (Editor) e M. Whittle (Preface), a cura di. Intrauterine Growth Restriction: Aetiology and Management. Springer, 2000.

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35

Sloan, Nancy L. Effects of maternal protein consumption on fetal growth and gestation. 1985.

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36

Ultrasound and Fetal Growth (Progress in Obstetric and Gynecological Sonography). Informa Healthcare, 2001.

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37

Prenatal and perinatal factors associated with brain disorders. [Bethesda, Md.]: National Institute of Child Health and Human Development, 1985.

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38

McLeod, Rima, Kelsey Wheeler, Pauline Levigne e Kenneth Boyer. Toxoplasma gondii. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0017.

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Abstract (sommario):

Mother-to-child transmission (MTCT) of the parasite Toxoplasma gondii can result in congenital toxoplasmosis. Untreated congenital toxoplasmosis presents considerable potential risks to patients and costs for society, with manifestations recurring throughout life. Infection with T. gondii, acquired at any time during pregnancy can damage the fetus, but especially during early gestation. Fetal infection with T. gondii can cause fetal loss, intrauterine growth retardation, and damage to organs (especially the brain and eyes). Treatment with pyrimethamine and sulfadiazine improves manifestations of active infection in the fetus, congenital infection in infants, and recurrent disease when manifested later in life in those congenitally infected. Key components of the prevention and treatment of congenital toxoplasmosis include prompt, correct diagnosis and treatment with effective anti–T. gondii medications. Several countries have gestational screening programs to detect newly acquired T. gondii infections. In the future, development of new medications, including those for chronic infection, and vaccines for prevention will be important.

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39

Das, Undurti N. A Perinatal Strategy for Preventing Adult Disease: The Role of Long-Chain Polyunsaturated Fatty Acids. Springer, 2002.

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40

Das, Undurti N. Perinatal Strategy for Preventing Adult Disease: The Role of Long-Chain Polyunsaturated Fatty Acids. Springer, 2012.

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41

Das, Undurti N. Perinatal Strategy for Preventing Adult Disease: The Role of Long-Chain Polyunsaturated Fatty Acids. Springer London, Limited, 2011.

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42

Waldmann, Carl, Neil Soni e Andrew Rhodes. Obstetric emergencies. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199229581.003.0031.

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Abstract (sommario):

Pre-eclampsia 518Eclampsia 520HELLP syndrome 522Postpartum haemorrhage 524Amniotic fluid embolism 526Pre-eclampsia is a common complication of pregnancy, UK incidence is 3–5%, with a complex hereditary, immunological and environmental aetiology.Abnormal placentation is characterized by impaired myometrial spiral artery relaxation, failure of trophoblastic invasion of these arterial walls and blockage of some vessels with fibrin, platelets and lipid-laden macrophages. There is a 30–40%, reduction in placental perfusion by the uterine arcuate arteries as seen by Doppler studies at 18–24 weeks gestation. Ultimately the shrunken, calcified, and microembolized placenta typical of the disease is seen. The placental lesion is responsible for fetal growth retardation and increased risks of premature labour, abruption and fetal demise. Maternal systemic features of this condition are characterized by widespread endothelial damage, affecting the peripheral, renal, hepatic, cerebral, and pulmonary vasculatures. These manifest clinically as hypertension, proteinuria and peripheral oedema, and in severe cases as eclamptic convulsions, cerebral haemorrhage (the most common cause of death due to pre-eclampsia in the UK), pulmonary oedema, hepatic infarcts and haemorrhage, coagulopathy and renal dysfunction....

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43

Horowitz, Sandra L. “I Am Pregnant; Why Can’t I Sleep?”. A cura di Angela O’Neal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190609917.003.0029.

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Abstract (sommario):

This chapter reviews the common sleep disorders of pregnancy. During pregnancy and postpartum, 84% of women report poor sleep at least a few nights a week. These problems are common, disruptive to daytime and nighttime activity, and may have multiple causes. This chapter covers aspects of insomnia and restless leg syndrome. It also discusses sleep apnea in pregnancy with related hormonal changes that may increase the incidence. There is an association of sleep apnea and pregnancy-induced hypertension, with increased adverse outcomes of pregnancy, including fetal growth retardation and premature birth. It has been suggested that treating nocturnal airflow limitation may improve gestational hypertension. The recommended therapies in this chapter may also be applied to non-pregnant patients with similar complaints.

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44

Committee to Study the Prevention of Low Birthweight e Division of Health Promotion and Disease Prevention. Preventing Low Birthweight: Summary. National Academies Press, 1985.

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45

Cohen, Alice Eve. What I Thought I Knew: A Memoir. Penguin (Non-Classics), 2010.

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