Bibliografie tematiche / CD2BP2

Letteratura scientifica selezionata sul tema "CD2BP2"

Autore: Grafiati

Pubblicato: 15 giugno 2024

Cita una fonte nei formati APA, MLA, Chicago, Harvard e in molti altri stili

Scegli il tipo di fonte:

Consulta la lista di attuali articoli, libri, tesi, atti di convegni e altre fonti scientifiche attinenti al tema "CD2BP2".

Accanto a ogni fonte nell'elenco di riferimenti c'è un pulsante "Aggiungi alla bibliografia". Premilo e genereremo automaticamente la citazione bibliografica dell'opera scelta nello stile citazionale di cui hai bisogno: APA, MLA, Harvard, Chicago, Vancouver ecc.

Puoi anche scaricare il testo completo della pubblicazione scientifica nel formato .pdf e leggere online l'abstract (il sommario) dell'opera se è presente nei metadati.

Articoli di riviste sul tema "CD2BP2":

Guo, Xiaobo, Gang Li, Yufeng Zhao e Bo Zhao. "TGFB Induced Factor Homeobox 2 Induces Deterioration of Bladder Carcinoma via Activating CD2 Cytoplasmic Tail Binding Protein 2". Journal of Biomedical Nanotechnology 19, n. 9 (1 settembre 2023): 1670–76. http://dx.doi.org/10.1166/jbn.2023.3657.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

Bladder carcinoma is a complex and aggressive malignancy with limited treatment options. In this study, we aimed to investigate the expression pattern of TGIF2 in bladder carcinoma and its clinical significance, as well as its functional role and interaction with CD2BP2 in disease progression. Through quantitative reverse transcription-polymerase chain reaction (qRT-PCR) analysis, we found that TGIF2 was highly expressed in bladder carcinoma tissues compared to normal bladder mucosa. Furthermore, elevated TGIF2 levels were associated with advanced tumor stage and larger tumor size, indicating its potential as a prognostic marker in bladder carcinoma. Using knockdown models in bladder carcinoma cell lines (253j and J82), we observed that the inhibition of TGIF2 resulted in decreased proliferation and migration rates, suggesting a critical role of TGIF2 in promoting these malignant phenotypes. Additionally, our dual-luciferase reporter assay revealed a direct interaction between TGIF2 and CD2BP2, with CD2BP2 being upregulated in bladder carcinoma tissues and positively correlated with TGIF2 expression. Notably, the overexpression of CD2BP2 reversed the suppressed malignant phenotypes caused by TGIF2 knockdown. Collectively, our findings highlight the abundant expression of TGIF2 in bladder carcinoma tissues and its association with malignant characteristics. We demonstrate that TGIF2 promotes proliferative and metastatic capacities in bladder carcinoma by positively regulating CD2BP2. These insights provide a basis for further investigations into the potential of TGIF2 and CD2BP2 as therapeutic targets and prognostic markers in bladder carcinoma management.

Kofler, Michael, Kathrin Motzny, Michael Beyermann e Christian Freund. "Novel Interaction Partners of the CD2BP2-GYF Domain". Journal of Biological Chemistry 280, n. 39 (6 luglio 2005): 33397–402. http://dx.doi.org/10.1074/jbc.m503989200.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Heinze, M., M. Kofler e C. Freund. "Investigating the functional role of CD2BP2 in T cells". International Immunology 19, n. 11 (6 settembre 2007): 1313–18. http://dx.doi.org/10.1093/intimm/dxm100.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Albert, Gesa I., Christoph Schell, Karin M. Kirschner, Sebastian Schäfer, Ronald Naumann, Alexandra Müller, Oliver Kretz et al. "The GYF domain protein CD2BP2 is critical for embryogenesis and podocyte function". Journal of Molecular Cell Biology 7, n. 5 (16 giugno 2015): 402–14. http://dx.doi.org/10.1093/jmcb/mjv039.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Nielsen, Tine K., Sunbin Liu, Reinhard Lührmann e Ralf Ficner. "Structural Basis for the Bifunctionality of the U5 snRNP 52K Protein (CD2BP2)". Journal of Molecular Biology 369, n. 4 (giugno 2007): 902–8. http://dx.doi.org/10.1016/j.jmb.2007.03.077.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Kofler, Michael, Katja Heuer, Tobias Zech e Christian Freund. "Recognition Sequences for the GYF Domain Reveal a Possible Spliceosomal Function of CD2BP2". Journal of Biological Chemistry 279, n. 27 (22 aprile 2004): 28292–97. http://dx.doi.org/10.1074/jbc.m402008200.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Andujar-Sanchez, Montserrat, Eva S. Cobos, Irene Luque e Jose C. Martinez. "Thermodynamic Impact of Embedded Water Molecules in the Unfolding of Human CD2BP2-GYF Domain". Journal of Physical Chemistry B 116, n. 24 (4 giugno 2012): 7168–75. http://dx.doi.org/10.1021/jp303495b.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Piotukh, K., e C. Freund. "A novel hSH3 domain scaffold engineered to bind folded domains in CD2BP2 and HIV capsid protein". Protein Engineering Design and Selection 25, n. 10 (17 settembre 2012): 649–56. http://dx.doi.org/10.1093/protein/gzs062.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Gan, Zhen, Bei Wang, Yishan Lu, Shuanghu Cai, Jia Cai, JiChang Jian e Zaohe Wu. "Molecular characterization and expression of CD2BP2 in Nile tilapia (Oreochromis niloticus) in response to Streptococcus agalactiae stimulus". Gene 548, n. 1 (settembre 2014): 126–33. http://dx.doi.org/10.1016/j.gene.2014.07.032.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Kang, Yuanyuan, Bhavita Patel, Kairong Cui, Keji Zhao, Yi Qiu e Suming Huang. "A T-Cell Specific Element Activates the TAL1 Oncogene Via an Interchromosomal Interaction During Leukemogenesis". Blood 120, n. 21 (16 novembre 2012): 3507. http://dx.doi.org/10.1182/blood.v120.21.3507.3507.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

Abstract Abstract 3507 T-cell acute lymphoblastic leukemia (T-ALL) is an aggressive malignant disease of thymocytes that mainly affects children and has very poor prognosis with high rates of relapse. A prominent feature observed in 60% of T-ALL childhood patients is the ectopic expression of a key hematopoietic transcription factor TAL1/SCL. Although several enhancers has been identified to play an important role in normal hematopoietic differentiation, the histone modification patterns and chromatin organization over the whole TAL1 locus reveled that none of them is active in T-ALL cell lines such as Jurkat and Rex cells. It remains currently unknown how TAL1 is activated in the majority of T-ALL patients lacking the TAL1 locus rearrangements. To understand the molecular mechanism underlying regulation of the TAL1 oncogene in leukemic T-cells, we employed circularized chromosome conformation capture (4C) methodology to identify new regulatory elements that activate TAL1 specifically in T-ALL leukemia. Using the TAL1 promoter 1a as the bait, we discovered that the TAL1 promoter 1a interacts with the TIL16 element (TAL1 interacting locus in chromosome 16) that is located at ∼15 Kb downstream of T-cell specific CD2BP2 gene in T-ALL cell line Jurkat, but not in erythroid progenitor K562 cells. The CD2BP2 protein is a cellular adapter protein that was originally identified as a binding partner of the T cell adhesion protein CD2 in the context of T cell signaling. The TIL16 element contains the bind sites for several transcription factors that are important for hematopoiesis such as C-Maf, Pax5, HoxA7 and USF2. The inter-chromosomal interaction between the TIL16 and the TAL1 promoter 1a was further confirmed by chromosome conformation capture (3C) assay in three TAL1 over-expressing T-ALL cell lines, Jurkat, REX and Molt4, but not in K562 cells. Recent genome wide study has correlates H3K4 mono- or dimethyl marks with distal enhancers while trimethyl H3K4 is enriched in promoters of active genes. To further test if the TIL16 acts as T-cell specific enhancer for TAL1 activation in T-ALL cells, we carried out ChIP-seq and ChIP analysis in CD4 T cells, Jurkat, and K562 cells. We found that the TIL16 element is specifically marked by H3K4me1 in Jurkat and CD4+ T-cells but not in K562 cells. The enrichment of H3K4me1 is correlated with the binding of c-Maf, a T-cell specific transcription factor. To further test whether the TIL16 element contributes to transcription activity, a DNA fragments containing the TIL16 element were cloneed into SV40 minimal promoter driven luciferase reporter and introduced into K562 and several T-ALL cell lines. Compared to the pGL3-SV40 vector that showed only minimal luciferase activity, the 1 Kb TIL element specifically activated transcription of the luciferase reporter in T-ALL cells, but not in erythroid progenitor K562 cells suggesting that the TIL16 element functions as a T-cell specific TAL1 enhancer. Thus, our data revealed a novel epigenetic mechanism by which the TAL1 oncogene is ectopically activated in T-cell leukemia. Disclosures: No relevant conflicts of interest to declare.

Più fonti

Tesi sul tema "CD2BP2":

Mansour, Hala. "Characterization of GEXP15 as a potential regulator of protein phosphatase 1 in Plasmodium falciparum". Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS068.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

Le paludisme est l'une des maladies infectieuses les plus répandues, menaçant 40% de la population mondiale, provoquant environ 300 millions de cas et 450 000 décès chaque année, touchant principalement les enfants de moins de 5 ans. En l'absence d'un vaccin efficace et face à l'émergence de la résistance aux médicaments, il y a un besoin urgent pour mieux comprendre la biologie du parasite afin de proposer des traitements innovants. Le parasite du paludisme, Plasmodium, responsable de la maladie, présente un cycle de vie complexe et un processus de division cellulaire unique. Par rapport aux systèmes bien étudiés, la connaissance limitée de la biologie du Plasmodium empêche le développement thérapeutique. La phosphorylation des protéines, un mécanisme de régulation important, est moins comprise dans Plasmodium que dans les cellules mammifères ou de levure. Les kinases et les phosphatases impliquées dans la phosphorylation et la déphosphorylation respectivement sont des cibles potentielles de médicaments. La sous-unité catalytique de la protéine phosphatase de type 1 (PP1c) (PF3D7_1414400) opère en combinaison avec diverses protéines régulatrices pour diriger et contrôler spécifiquement son activité phosphatase. Cependant, peu d'informations sont disponibles sur cette phosphatase et ses régulateurs dans le parasite du paludisme humain, Plasmodium falciparum. Pour combler cette lacune de connaissances, nous avons mené une étude approfondie sur les caractéristiques structurelles et fonctionnelles d'un régulateur spécifique du Plasmodium appelé, Gametocyte EXported Protein 15, GEXP15 (PF3D7_1031600). Par analyses in silico, nous avons identifié trois régions d'intérêt significatives dans GEXP15 : une région N-terminale couvrant un motif RVxF interagissant avec PP1, un domaine conservé dont la fonction est inconnue, et un domaine de type GYF qui facilite potentiellement des interactions spécifiques protéine-protéine. Pour élucider davantage le rôle de GEXP15, nous avons réalisé des études d'interaction in vitro qui ont démontré une interaction directe entre GEXP15 et PP1 via le motif de liaison RVxF. Cette interaction avec PfGEXP15 a été montrée capable d'augmenter l'activité phosphatase de PP1 in vitro. De plus, en utilisant une lignée transgénique de P. falciparum exprimant la GEXP15-GFP, nous avons observé une forte expression de GEXP15 dans les stades asexués tardifs du parasite, avec une localisation principalement dans le noyau. Des expériences d'immunoprécipitation suivies d'analyses en spectrométrie de masse ont révélé l'interaction de GEXP15 avec des protéines de liaison aux ribosomes et à l'ARN. De plus, grâce à des analyses de capture de domaines fonctionnels recombinants de GEXP15 marqués avec un tag His, nous avons confirmé sa liaison avec PfPP1et au complexe ribosomal via le domaine GYF. Dans l'ensemble, notre étude éclaire l'interaction PfGEXP15-PP1-ribosome, qui joue un rôle crucial dans la traduction des protéines. Ces découvertes suggèrent que PfGEXP15 pourrait être une cible potentielle pour le développement de médicaments contre le paludisme
Malaria is one of the most prevalent vector-borne infectious diseases threatening 40% of the global population, causing around 300 million cases and 450,000 deaths annually, mostly affecting children under 5. With no effective vaccine and drug resistance emerging, there is an urgent need for innovative treatments. The malaria-causing Plasmodium parasite has a complex life cycle and unique cell division process. Compared to well-studied systems, limited knowledge of Plasmodium biology hampers therapeutic development. Protein phosphorylation, a key regulatory mechanism, is less understood in Plasmodium than in mammalian or yeast cells. Kinases and phosphatases involved in phosphorylation and dephosphorylation processes respectively are potential drug targets. The Protein Phosphatase type 1 catalytic subunit (PP1c) (PF3D7_1414400) operates in combination with various regulatory proteins to specifically direct and control its phosphatase activity. However, there is little information about this phosphatase and its regulators in the human malaria parasite, Plasmodium falciparum. To address this knowledge gap, we conducted a comprehensive investigation into the structural and functional characteristics of a conserved Plasmodium-specific regulator called Gametocyte EXported Protein 15, GEXP15 (PF3D7_1031600). Through in silico analysis, we identified three significant regions of interest in GEXP15: an N-terminal region hous-ing a PP1-interacting RVxF motif, a conserved domain whose function is unknown, and a GYF-like domain that potentially facilitates specific protein-protein interactions. To further elucidate the role of GEXP15, we conducted in vitro interaction studies that demonstrated a direct interaction between GEXP15 and PP1 via the RVxF-binding motif. This interaction was found to enhance the phosphatase activity of PP1. Additionally, utilizing a transgenic GEXP15-tagged line and live microscopy, we observed high expression of GEXP15 in late asexual stages of the parasite, with localization predominantly in the nucleus. Immunoprecipitation assays followed by mass spectrometry analyses revealed the interaction of GEXP15 with ribosomal- and RNA-binding proteins. Furthermore, through pull-down analyses of recombinant functional domains of His-tagged GEXP15, we confirmed its binding to PfPP1 and to the ribosomal complex via the GYF domain. Collectively, our study sheds light on the PfGEXP15-PP1-ribosome interaction, which plays a crucial role in protein translation. These findings suggest that PfGEXP15 could serve as a potential target for the development of malaria drugs

Oliveira, João Bosco Lucena de. "Determinação dos tensores polares de CH2C12/CD2C12 e os clorofluorcarbonos". [s.n.], 1991. http://repositorio.unicamp.br/jspui/handle/REPOSIP/249373.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

Orientador : Roy Edward Bruns
Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Quimica
Made available in DSpace on 2018-07-14T01:41:45Z (GMT). No. of bitstreams: 1 Oliveira_JoaoBoscoLucenade_D.pdf: 2836729 bytes, checksum: f689a1a625f22aa90eefb1f02c9d107d (MD5) Previous issue date: 1991
Doutorado

Monzo, Pascale. "Fonctions cellulaires de CD2AP et son implication dans la cytokinese". Nice, 2004. http://www.theses.fr/2004NICE4067.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Walker, Jennifer Anne. "CD22, autoimmunity and the B cell". Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612192.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Barrett, Anna. "Molecular and cellular investigation of Alzheimer's disease associated risk loci : BIN1 and CD2AP". Thesis, Cardiff University, 2017. http://orca.cf.ac.uk/111373/.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

Genome wide association studies have identified genes associated with Late Onset Alzheimer’s disease (AD). Pathway analyses have used this data to implicate a number of biological mechanisms in AD pathogenesis. Endocytosis has been implicated in AD and is a critical biological mechanism involved in the production of Aβ. BIN1 and CD2AP are associated with AD and function in endocytosis. This thesis describes how depletion of BIN1 and CD2AP has contradicting effects on the processing of APP in a human brain cell line and affects endocytosis in different ways, suggesting multiple cellular trafficking mechanisms may be involved in Aβ production. As most genetic variants associated with complex diseases are located in the non-coding region of the genome, they may contribute to disease susceptibility by disrupting gene regulation. Variants at the BIN1 locus associated with AD are located approximately 30 Kb from the coding region, suggesting BIN1 regulation may be a risk mechanism in AD. BIN1 expression is influenced by cis-regulatory mechanisms in prefrontal cortex tissue. Differential allele expression implied that this cis-regulation was influenced by DNA variants. The variant responsible was not identified but there was suggestive evidence for an intronic variant associated with AD. ChIP-Seq and DNase-Seq data identified chromatin modifications and transcription factor binding in immune cell types at the BIN1 risk locus. Gene reporter assays showed that the BIN1 locus was capable of functioning as a genetic enhancer. Furthermore, assays used to investigate DNA protein interactions showed that SPI1, an AD associated transcription factor with a critical immune function, bound to the BIN1 locus. The BIN1 index SNP had no effect on gene expression or protein binding, but may have a greater impact on additional disease relevant immune cell types. Finally, gene-editing techniques were explored as a mechanism for generating isogenic cell models to investigate the effect of AD associated variants.

Wöhner, Miriam [Verfasser], e Lars [Akademischer Betreuer] Nitschke. "Entwicklung einer knockin Mauslinie mit Expression von humanem CD22 zum Test therapeutischer Anwendungen künstlicher CD22-Liganden / Miriam Wöhner. Betreuer: Lars Nitschke". Erlangen : Universitätsbibliothek der Universität Erlangen-Nürnberg, 2012. http://d-nb.info/1024608700/34.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Braae, Anne. "Exploring potential functional variants in the Alzheimer's disease associated genes, CD2AP, EPHA1 and CD33". Thesis, University of Nottingham, 2016. http://eprints.nottingham.ac.uk/33083/.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

Little is known about the molecular biology of late onset Alzheimer’s disease (LOAD), the most common dementia in the elderly. Genetic loci associated with LOAD have been identified through genome-wide association studies (GWAS). However, the functional variants responsible for the observed GWAS association at each of the loci remain unknown. The aim of this project was to identify and assess potential functional rare variants at three associated loci, CD2AP, EPHA1 and CD33. Target enriched, pooled sequencing of 96 post-mortem confirmed LOAD patient samples was used to identify 1273 variants within the three GWAS loci. Variants were prioritised using a combination of in silico functional annotation and putative disease association. Disease association was assessed through comparison to an independent, imputed LOAD GWAS dataset (2067 cases, 7376 controls). 18 coding and untranslated region variants and 9 noncoding variants were prioritised for further investigation. Potential splicing variants in CD2AP (6:47544253A > G) and EPHA1 (rs6967117) were assessed using minigene assays, although neither were found to influence splicing products in vivo. Five untranslated variants from the three genes and a frameshift variant in CD33 (rs201074739) were assessed for potential cis-regulatory consequences using allelic expression imbalance in brain tissues and B-lymphoblast cell lines. Only the frameshift variant displayed significant allelic expression imbalance and was found to be targeted for nonsense-mediated decay. None of the prioritised variants investigated were both functional and significantly associated with LOAD. However, pooled next generation sequencing using target enrichment successfully identified potential functional rare variants in CD2AP, EPHA1 and CD33. Rare variants do have a role to play in late onset Alzheimer’s disease. With the development of additional functional databases and improvements imputing rare variants from GWAS datasets, the combined prioritisation strategy used in this thesis will be useful for similar studies investigating causal GWAS variants.

Bhatt, Arshiya [Verfasser]. "Unraveling details of CIN85/CD2AP assistance to SLP65-mediated B cell activation / Arshiya Bhatt". Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2020. http://d-nb.info/1217842810/34.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Klein, Jörg. "Verwendung von Gene-Targeting-Techniken zur Etablierung neuer Mauslinien mit Mutationen in B-Zell-Signalwegen". [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=976107953.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Mari, Muriel. "Caractérisation structurale et fonctionnelle de RABIP4 et CD2AP/CMS, deux effecteurs de la petite GTPase RAB4". Nice, 2002. http://www.theses.fr/2002NICE5727.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Più fonti

Libri sul tema "CD2BP2":

Books, Bchimak. Buchhaltung: Einfaches Einnahmen- und Ausgaben Kassenbuch Für Kleinunternehmen, Einfaches Buchhaltungsbuch Für Selbstständige, Freiberufler und Als Haushaltsbuch Für Die Buchhaltung, über 3300 Einträge Auf 120 Seiten). Cd22. Independently Published, 2021.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Capitoli di libri sul tema "CD2BP2":

Medway, Christopher, e Kevin Morgan. "CD2-Associated Protein (CD2AP)". In Genetic Variants in Alzheimer's Disease, 201–8. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7309-1_11.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Newton, Dianne L., Luke H. Stockwin e Susanna M. Rybak. "Anti-CD22 Onconase: Preparation and Characterization". In Therapeutic Antibodies, 425–43. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-554-1_22.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Kreitman, Robert J., David J. P. FitzGerald e Ira Pastan. "BL22: A Milestone in Targeting CD22". In Next Generation Antibody Drug Conjugates (ADCs) and Immunotoxins, 151–76. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-46877-8_8.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Topp, Max, e Tobias Feuchtinger. "Management of Hypogammaglobulinaemia and B-Cell Aplasia". In The EBMT/EHA CAR-T Cell Handbook, 147–49. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-94353-0_28.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

AbstractThe development and regulatory approval of CAR-T cell therapies targeting B-lineage surface antigens (Maude et al. 2018), such as CD19 or CD22, represents a major milestone in cancer immunotherapy. This treatment results in the depletion of malignant and normal B cells and is associated with hypogammaglobulinaemia. These on-target, off-tumour toxicities may result in an increased risk of infection. Careful long-term follow-up assessment of patients receiving CAR-T cell therapy is important. Management of these on-target, off-tumour effects should be well coordinated between treatment and referring centres if the patient returns to local providers following therapy. Aims of this toxicity management:

Smith, K. G. C., e D. T. Fearon. "Receptor Modulators of B-Cell Receptor Signalling — CD19/CD22". In Current Topics in Microbiology and Immunology, 195–212. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57066-7_6.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Drgona, Lubos, e Lucia Masarova. "CD22, CD30, CD33, CD38, CD40, SLAMF-7 and CCR4". In Infectious Complications in Biologic and Targeted Therapies, 89–112. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-11363-5_6.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Blasioli, J., e C. C. Goodnow. "Lyn/CD22/SHP-1 and Their Importance in Autoimmunity". In Current Directions in Autoimmunity, 151–60. Basel: KARGER, 2001. http://dx.doi.org/10.1159/000060551.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Cornall, R. J., C. C. Goodnow e J. G. Cyster. "Regulation of B Cell Antigen Receptor Signaling by the Lyn/CD22/SHP1 Pathway". In Current Topics in Microbiology and Immunology, 57–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-58537-1_5.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Fearon, Douglas T. "Non-Structural Determinants of Immunogenicity and the B Cell Co-Receptors, CD19, CD21, and CD22". In Advances in Experimental Medicine and Biology, 181–86. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5355-7_20.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Han, Shoufa, Brian E. Collins e James C. Paulson. "Synthesis of 9-Substituted Sialic Acids as Probes for CD22-Ligand Interactions on B Cells". In ACS Symposium Series, 2–14. Washington, DC: American Chemical Society, 2007. http://dx.doi.org/10.1021/bk-2007-0960.ch001.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Atti di convegni sul tema "CD2BP2":

Vasconcelos, D. N., A. C. F. Santos, M. A. MacDonald, M. M. Sant’Anna, B. N. C. Tenório, A. B. Rocha, V. Morcelle, N. Appathurai e L. Zuin. "Ultrafast dissociation of CD2Cl2 and CH2Cl2". In 25TH INTERNATIONAL CONFERENCE ON THE APPLICATION OF ACCELERATORS IN RESEARCH AND INDUSTRY. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5127724.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Le, Thuc Duy, Lin Liu, Emre Kiciman, Sofia Triantafyllou e Huan Liu. "The KDD 2022 Workshop on Causal Discovery (CD2022)". In KDD '22: The 28th ACM SIGKDD Conference on Knowledge Discovery and Data Mining. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3534678.3542890.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Eggers, David F., W. Lewis-Bevan e M. C. L. Gerry. "Vibration-rotation infrared spectra of CHDF2". In OSA Annual Meeting. Washington, D.C.: Optica Publishing Group, 1986. http://dx.doi.org/10.1364/oam.1986.wj3.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Abstract (sommario):

CHDF2 gas was synethesized in several steps in high isotopic purity. All nine fundamentals were observed in the infrared spectrum, and the region from 920 to 1200 cm−1 was scanned at 0.005-cm−1 resolution. This region contains four fundamentals with strong Coriolis coupling about several molecular axes: the four band origins are found within a range of 160 cm−1. Two other fundamentals near 1360 cm−1 have origins closer than 10 cm−1. Molecular constants from the rotational analysis are reported and are compared to predictions from the force fields deduced by other workers. A listing of some coincidences with CO2 laser lines is also presented; the isotopomers CH2F2 and CD2F2 both show numerous far-IR laser lines when suitably pumped with CO2 lasers.

Shah, Nirali N., Haneen Shalabi, Bonnie Yates, Constance Yuan, Haiying Qin, Amanda Ombrello, Hao-Wei Wang et al. "Abstract LB-146: Phase I CD22 CAR T-cell trial updates". In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-lb-146.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Zanetti, SR, T. Velazco-Hernandez, F. Gutierrez-Agüera, H. Roca-Ho, D. Sánchez-Martínez, P. Petazzi, R. Torres et al. "CD19 and CD22-directed biespecific CAR for B-cell Acute Lymphoblastic Leukemia". In 32. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1687121.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Vitkina, Tatyana, Karolina Sidletskaya e Yulia Denisenko. "Expression of CD282+/CD284+ on blood granulocytes and its relationship to cytokine status in patients with stable chronic obstructive pulmonary disease". In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa3423.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Yao, Xin, Patricia Burke, Joyce O. Obidi, Xiaoru Chen, Haifeng Bao, Yihong Yao e Jiaqi Huang. "Abstract 4420: Factors potentially contributing to sensitivities of CD22-targeting agents in B-cell malignancies". In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4420.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Gouble, Agnès, Cécile Schiffer-Mannioui, Severine Thomas, Anne-Sophie Gautron, Laurent Poirot e Julianne Smith. "Abstract 3763: UCART22: allogenic adoptive immunotherapy of leukemia by targeting CD22 with CAR T-cells". In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3763.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Mussai, Francis J., Dario Campana, Deepa Bhojwani, Maryalice Stetler-Stevenson, Seth M. Steinberg, Sebastien Morisot, Curt I. Civin, Alan S. Wayne e Ira Pastan. "Abstract 4356: Cytotoxicity of the anti-CD22 immunotoxin HA22 against pediatric acute lymphoblastic leukemia (ALL)". In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4356.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri