Bibliografie tematiche / Blood-vessels

Letteratura scientifica selezionata sul tema "Blood-vessels"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 7 luglio 2024

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Articoli di riviste sul tema "Blood-vessels"

1

Chen, Chiu-Yu, Cara Bertozzi, Zhiying Zou, Lijun Yuan, John S. Lee, MinMin Lu, Stan J. Stachelek et al. "Blood flow reprograms lymphatic vessels to blood vessels". Journal of Clinical Investigation 122, n. 7 (2 luglio 2012): 2702. http://dx.doi.org/10.1172/jci65314.

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Chen, Chiu-Yu, Cara Bertozzi, Zhiying Zou, Lijun Yuan, John S. Lee, MinMin Lu, Stan J. Stachelek et al. "Blood flow reprograms lymphatic vessels to blood vessels". Journal of Clinical Investigation 122, n. 6 (1 giugno 2012): 2006–17. http://dx.doi.org/10.1172/jci57513.

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3

Lawson, Jeffrey. "Engineered Blood Vessels". Blood 130, Suppl_1 (7 dicembre 2017): SCI—12—SCI—12. http://dx.doi.org/10.1182/blood.v130.suppl_1.sci-12.sci-12.

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Abstract (sommario):
Abstract The field of tissue engineering has has made significant progress in the past 20 years. Of the many tissues and organs in development, the area of vascular tissue engineering is now one of the most mature. In that regard, we have developed a novel tissue engineered vascular graft (human acellular vessel [HAV]) that addresses many of the limitations of native vein harvest and the performance of both synthetic ePTFE and autologous vein grafts. The HAV is manufactured in a laboratory by culturing human vascular cells within a biodegradable scaffold that forms a mechanically robust tissue engineered blood vessel. The cells are then completely removed (decellularization), leaving behind a non-immunogenic human collagen-based vascular tissue that can be stored on the shelf for months at a time and ready for immediate implantation into any patient. The HAV is currently being evaluated in Phase II and Phase III clinical trials in the U.S., E.U. and Israel as an arteriovenous vascular access graft for hemodialysis in patients with end-stage renal failure and as an arterial substitute for patients in need of vascular bypass for peripheral arterial disease or vascular trauma. Following clinical implantation, we have observed repopulation and remodeling of the manufactured vessel with the hosts' own cells. We hypothesize that the biological composition of the HAV, compared to synthetic vascular grafts, promotes its physiological integration into host tissue including support of normal host cell infiltration and function. Host cells that identify histologically similar to vascular smooth muscle cells appear to repopulate the middle of the vessel and recipient cells characterized as endothelial cells appear to cover the luminal surface of the implanted vessel. Clinical observations of the Phase II trials have demonstrated excellent vessel durability and a freedom from both early and delayed infection. Based on the success of the Phase II studies, a Global Phase III study is underway for patients in need of dialysis access shunts and vascular programs for vascular (arterial) bypass and trauma are expanding. Disclosures Lawson: InnaVasc: Patents & Royalties; Humacyte, Inc.: Employment.
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Wong, W. "Broken Blood Vessels". Science Signaling 2, n. 57 (10 febbraio 2009): ec49-ec49. http://dx.doi.org/10.1126/scisignal.257ec49.

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Frederickson, Robert. "Rebuilding blood vessels". Nature Biotechnology 17, n. 11 (novembre 1999): 1051. http://dx.doi.org/10.1038/15025.

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Iruela-Arispe, M. Luisa. "LUMENating Blood Vessels". Developmental Cell 20, n. 4 (aprile 2011): 412–14. http://dx.doi.org/10.1016/j.devcel.2011.03.020.

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Hindson, Jordan. "Eternal blood vessels". Nature Reviews Materials 3, n. 5 (26 aprile 2018): 4. http://dx.doi.org/10.1038/s41578-018-0015-x.

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Lou, Kai-Jye. "Building blood vessels". Science-Business eXchange 7, n. 44 (novembre 2014): 1283. http://dx.doi.org/10.1038/scibx.2014.1283.

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Niu, Guoguang, Etai Sapoznik e Shay Soker. "Bioengineered blood vessels". Expert Opinion on Biological Therapy 14, n. 4 (25 gennaio 2014): 403–10. http://dx.doi.org/10.1517/14712598.2014.880419.

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Hendry, Charles, Alistair Farley e Ella McLafferty. "Blood vessels, circulation and blood pressure". Nursing Standard 27, n. 11 (14 novembre 2012): 35–40. http://dx.doi.org/10.7748/ns.27.11.35.s48.

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Tesi sul tema "Blood-vessels"

1

Carter, A. J. "Thromboxane synthesis in human blood platelets and blood vessels". Thesis, University of Nottingham, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.355288.

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Au, Pakwai. "Engineering functional blood vessels in vivo". Thesis, Massachusetts Institute of Technology, 2008. http://hdl.handle.net/1721.1/43867.

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Thesis (Ph. D.)--Harvard-MIT Division of Health Sciences and Technology, 2008.
Includes bibliographical references.
At the present time, there are many hurdles to overcome in order to create a long-lasting and engineered tissue for tissue transplant in patients. The challenges include the isolation and expansion of appropriate cells, the arrangement of assorted cells into correct spatial organization, and the development of proper growth conditions. Furthermore, the creation of a three dimensional engineered tissue is limited by the fact that tissue assemblies greater than 100-200 micrometers, the limit of oxygen diffusion, require a perfused vascular bed to supply nutrients and to remove waste products and metabolic intermediates. To overcome this limitation, this thesis aims to pre-seed a tissue engineered construct with vascular cells (both endothelial and perivascular cells), so the vascular cells could readily form functional vessels in situ. Previous work in the laboratory had successfully demonstrated the formation of functional microvascular network by co-implantation of human umbilical cord vein endothelial cells (HUVECs) and 10 T 1/2 cells, a line of mouse embryonic fibroblasts. To translate this concept to the clinic, we need to utilize cells that can be secured and used in clinic. To this end, we systematically replace each individual vascular cell type with a readily available source of cells. First, we investigated human embryonic stem cells (hESCs) derived endothelial cells. We demonstrated that when hESCs derived endothelial cells were implanted into SCID mice, they formed blood vessels that integrated into the host circulatory system and served as blood conduits. Second, we compared the formation and function of engineered blood vessels generated from circulating endothelial progenitor cells (EPCs) derived from either adult peripheral blood or umbilical cord blood.
(cont.) We found that adult peripheral blood EPCs formed blood vessels that were unstable and regressed within three weeks. In contrast, umbilical cord blood EPCs formed normal-functioning blood vessels that lasted for more than four months. These vessels exhibited normal blood flow, perm-selectivity to macromolecules and induction of leukocyte-endothelial interactions in response to cytokine activation similar to normal vessels. Third, we evaluated human bone marrow-derived mesenchymal stem cells (hMSCs) as a source of vascular progenitor cells. hMSCs expressed a panel of smooth muscle markers in vitro and cell-cell contact between endothelial cells and hMSCs up-regulated the transcription of smooth muscle markers. hMSCs efficiently stabilized nascent blood vessels in vivo by functioning as perivascular precursor cells. The engineered blood vessels derived from HUVECs and hMSCs remained stable and functional for more than 130 days in vivo. On the other hand, we could not detect differentiation of hMSCs to endothelial cell in vitro and hMSCs by themselves could not form conduit for blood flow in vivo. Similar to normal perivascular cells, hMSCs-derived perivascular cells contracted in response to endothelin-1 in vivo. Thus, our work demonstrates the potential to generate a patent and functional microvascular network by pre-seeding vascular cells in a tissue-engineered construct. It serves as a platform for the addition of parenchymal cells to create a functional and vascularized engineered tissue.
by Pakwai (Patrick) Au.
Ph.D.
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HYNES, MICHAEL RAY. "ENDOTHELIUM-DEPENDENT RELAXATION OF BLOOD VESSELS". Diss., The University of Arizona, 1987. http://hdl.handle.net/10150/184134.

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Dilation of blood vessels in response to a large number of agents has been shown to be dependent on an intact vascular endothelium. The present studies examine some aspects of endothelium-dependent vasodilation in blood vessels of the rabbit and rat. Using the rabbit ear artery and the subtype-selective muscarinic antagonist pirenzepine, muscarinic receptors of the endothelium and smooth muscle cells were shown to be of the low affinity M₂ subtype. Inhibition of [³H](-)quinuclidinyl benzilate was used to determine affinity for the smooth muscle receptors while antagonism of methacholine induced vasodilation yielded the endothelial cell receptor affinity. The effect of increasing age (1-27 months) on endothelium-dependent relaxation was studied in aortic rings, perfused tail artery and perfused mesenteric bed of the Fisher 344 rat. Both aortic ring segments and perfused caudal arteries showed an age-related increase in sensitivity of endothelium-mediated relaxation to the cholinergic agonist methacholine. This increased sensitivity occurs between the ages of 6 and 12 months, with no further significant increase up to 27 months of age, suggesting this is a consequence of growth and development rather than old age. No difference with age in cholinergic relaxation was observed in the perfused mesenteric bed indicating either no change of sensitivity in smaller resistance vessels or an effect which is hidden in this more complex perfused system. In contrast to findings with cholinergic stimution, responses of the perfused caudal artery to the calcium ionophore A23187 were not altered with age. This suggests that the alteration with age in response to methacholine involves the muscarinic receptor or receptor coupling mechanism rather than the generation of, or response to, endothelium-derived relaxing factor (EDRF). The influence of endothelium on contractile responses was examined using the perfused caudal artery. Endothelium removal significantly increased contraction to the α-adrenergic agonists methoxamine and BH-T 920 as well as to transmural nerve stimulation. Inhibition of contraction to agents which must first cross the smooth muscle layer before reaching the endothelium suggests that a continuous or basal level of EDRF release is responsible for decreased contraction rather than an receptor stimulated release of EDRF.
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Mancini, Maria L. "Novel Roles for Endoglin in Vascular Development and Maintenance of Vascular Integrity". Fogler Library, University of Maine, 2007. http://www.library.umaine.edu/theses/pdf/ManciniML2007.pdf.

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Arief, Melissa Suen. "Human Tissue Engineered Small Diameter Blood Vessels". Yale University, 2010. http://ymtdl.med.yale.edu/theses/available/etd-03152010-144428/.

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The engineering of human vascular grafts is an intense area of study since there is crucial need for alternatives to native vein or artery for vascular surgery. This current study sought to prove that a tissue engineered blood vessel (TEBV) 1mm in diameter could be developed from human smooth muscle cells and that endothelial progenitor cells (EPCs) could be cultured and used to endothelialize these grafts. This project had four specific aims: the isolation and characterization of EPCs, the seeding of a novel scaffold with EPCs and exposure to physiologic shear stress in vitro, the development of TEBV from human smooth muscle cells that are strong enough to implant in vivo, and the in vivo implantation of TEBV into the rat aortic model with a comparison of EPC seeded TEBVs pretreated with shear stress and unseeded TEBVs. The results yielded isolation of four EPC lines and a flow system design capable of seeding EPCs onto a novel scaffold with preliminary studies indicating that it is capable of exposing the EPCs to physiologic shear stress, although further studies require more optimization. The development of mechanically strong TEBV was highly successful, yielding TEBVs comparable to native vessels in collagen density and burst pressure, but with much lower compliance. Current implantation studies indicated that unseeded TEBV grafts implanted into the rat aorta without anticoagulation is highly thrombogenic. However, anticoagulation using Plavix may be capable of maintaining graft patency. These TEBVs did not rupture or form aneurysm in vivo and the future completion of the in vivo studies are likely to demonstrate the high potential of these grafts.
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Dritsoula, A. "Regulation of NKX2-5 in blood vessels". Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/1551692/.

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NKX2-5 is a transcription factor required for the formation of the heart and vessels during development. Postnatal expression is significantly downregulated, and then re-activated in diseased conditions characterised by vascular remodelling. However, the mechanisms regulating NKX2-5 activation in diseased vessels remain unknown. The aim of this thesis is to identify these mechanisms and provide information on how the gene contributes to cardiovascular pathologies, such as sclerodermaassociated pulmonary hypertension. A case-control genetic association study was performed in two independent cohorts of scleroderma patients. Associated SNPs located in the NKX2-5 genomic region were cloned into reporter vectors, and transcriptional activity was assessed by reporter-gene assays. Associated SNPs were further evaluated through proteinDNA binding assays, chromatin immunoprecipitation and RNA silencing. Signalling mechanisms activating NKX2-5 expression were investigated in vascular endothelial and smooth muscle cells using a panel of selective inhibitors. Meta-analysis across the two independent cohorts revealed that rs3131917 was associated with scleroderma. Rs3132139, downstream of NKX2-5, was significantly associated with pulmonary hypertension in both cohorts. The region containing rs3132139 and rs3131917 was shown to be a novel functional enhancer, which increased NKX2-5 transcriptional activity through the binding of GATA6, c-JUN, and MEF-2c. An activator TEAD/YAP1 complex was shown to bind at rs3095870, another functional SNP upstream of NKX2-5 transcription start site, which showed marginal association with scleroderma. Signalling mechanisms, involving TGF-β, ERK5, AKT and hypoxia, stimulated NKX2-5 expression during phenotypic modulation of vascular endothelial and smooth muscle cells. Overall, the data showed that NKX2-5 is genetically associated with scleroderma and pulmonary hypertension. Functional evidence revealed a regulatory mechanism, activated by TGF-β, which results in NKX2-5 transcription in human vascular smooth muscle cells through the interaction of an upstream promoter and a novel downstream enhancer. These regulatory mechanisms can act as a model for NKX2-5 activation in cardiovascular disease characterised by vascular remodelling.
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Kaur, Amaratpal. "Optical remote sensing of hypertensive blood vessels". Thesis, Loughborough University, 1997. https://dspace.lboro.ac.uk/2134/32228.

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The aim of this research is to investigate the effects of hypertension on small artery structure by extending the instrumentation which is available for optical analysis of vessels in-vitro. Conventionally, arterial vessels are pressurised in a perfusion myograph and are analysed by video microscopy. A key objective of this research is to enable the examination of vessels by absorption spectroscopy.
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Omatuku, Emmanuel Ngongo. "Modelling of Residual Stresses of Blood Vessels". Thesis, University of Cape Town, 2015. http://hdl.handle.net/11427/21604.

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The diagnosis of vascular diseases can be achieved with a suitably determined circum­ferential stress at arterial walls. The stress distribution over arterial walls in blood vessels is affected by residual stresses and stresses due to blood pressure. However, residual stresses are still not reliably determined. For this reason, a suitable incorporation of these stresses is required in order to establish the wall stress as a reliable diagnostic indicator. Thus this study aims to model residual stresses by incorporating them into the wall stress distribution, and to investigate the effect that parameters defining the study constitutive model have on the stress distribution. The constitutive model makes use of the Cosserat fibre continuum in order to account for mechanics of arterial walls. It was developed for cardiac tissues by Skatulla et al. (2014), but it can also be used for a preliminary investigation on arterial tissues as these two types of tissues exhibit comparable mechanics. Residual stresses are incorporated by using three problem definitions, which are derived from the opening angle method, into a three dimensional two-layer artery con­sisting of the media and adventitia. The first problem incorporates residual stresses that are locked within individual load-free layers. The second problem continues the first problem by incorporating residual stresses acting at the interface surface between arterial layers, and then determine the artery wall stress distribution under blood pressure. The third problem determines the wall stress in the stress-free artery under blood pressure. On the other hand, the effect of parameters defining the constitutive model is investi­gated by varying the size of parameters in these problems. However, the second problem is not analysed in this study because it requires an analysis implementation that could not be achieved within the study timeline. Similarly, model parameters of problems are not calibrated to available experimental data. There­fore, this study only provides qualitative results. The investigation results on the incorporation of residual stresses into the stress distribution are found to be inconclusive as they provide contradictory results. The char­acteristic scaling parameters are found to influence the magnitude and gradient of the stress distribution. However, these results are not conclusive to clearly define the influ­ence. Thus it is recommended that further research be conducted in order to gain con­clusive results.
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Williams, Chrysanthi. "Perfusion bioreactor for tissue-engineered blood vessels". Diss., Available online, Georgia Institute of Technology, 2004:, 2003. http://etd.gatech.edu/theses/available/etd-06072004-131410/unrestricted/williams%5Fchrysantyhi%5F200405%5Fphd.pdf.

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Kuzborska, Zyta. "Research of blood flow and stresses in the pathological blood vessels". Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2012. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2012~D_20120131_135725-24904.

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Abstract (sommario):
Physical load, age and gender influence to blood pressure and maximal stresses in the pathological blood vessel palaces was studies in this work. The main research subject – pathological blood vessel and its blood flow processes that depend on physical load, pathology degree and type, age, gender and blood vessels stress characteristics. The main aim of this work – to examine blood flow characteristics, local blood pressure, stress distribution in the pathological blood vessels dependent physical load assessing blood vessels mechanical properties variations due to age, gender, blood vessel pathology type; to make simplified human efficiency evaluation methodology. The paper analyse a few main tasks: to explore physical load, blood vessels pathology degree, age and gender influence to blood pressure and tensions increase in the pathological blood vessels locations; experimentally determine blood flow rates changes in pathological blood vessels assessing; additionally investigate blood pressure and heart rate characteristics variations during set physical load and human working age range. This paper consists of introduction, four chapters, summary, literature and authors publications theses lists and two annexes. Introductory chapter discusses the test problem, work topicality, research subject, also formulates work subject and tasks, and describes research methodology, work scientific novelty, practical value of the work results, defended propositions. In the introduction end... [to full text]
Disertacijoje nagrinėjama fizinio krūvio, amžiaus bei lyties įtaka kraujo spaudimui ir didžiausiems įtempiams pažeistoje kraujagyslės vietoje. Pagrindinis tyrimo objektas – ligos pažeista kraujagyslė ir joje vykstantys kraujo tėkmės procesai, priklausantys nuo fizinio krūvio dydžio, pažeidimo laipsnio ir rūšies, amžiaus, lyties, bei šių veiksnių įtaka didžiausiems įtempiams ir spaudimui pažeistose vietose. Pagrindinis disertacijos tikslas – ištirti kraujo tėkmės charakteristikas, lokalinį kraujo spaudimą, įtempių pasiskirstymą pažeistose kraujagyslėse priklausomai nuo fizinio krūvio, įvertinant kraujagyslių mechaninių savybių pokytį dėl amžiaus, lyties, kraujagyslės pažeidimo rūšies, ir sudaryti supaprastintą darbingumo verti-nimo metodiką. Darbe sprendžiami keli uždaviniai: ištirti fizinės apkrovos dydžio, kraujagyslių pažeidimų laipsnio, amžiaus ir lyties įtaką kraujo spaudimui ir įtempių padidėjimui pažeistose kraujagyslių vietose; ekspe-rimentiniu būdu nustatyti kraujo tėkmės rodiklių pokyčius pažeistose kraujagyslėse; ištirti kraujo spaudimo ir širdies susitraukimų dažnio charakteristikų pokyčius nustatytame fizinio krūvio ir žmogaus darbingo amžiaus intervale. Disertaciją sudaro įvadas, keturi skyriai, rezultatų apibendrinimas, naudotos literatūros ir autorės publikacijų disertacijos tema sąrašai ir du priedai. Įvadiniame skyriuje aptariama tiriamoji problema, darbo aktualumas, aprašomas tyrimų objektas, formuluojamas darbo tikslas bei uždaviniai, aprašoma tyrimų... [toliau žr. visą tekstą]
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Libri sul tema "Blood-vessels"

1

Robinson, Lorraine. The blood vessels. [Kelowna, BC]: Creativity Advocate Publications, 2004.

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2

1909-, Horwitz Orville, McCombs Peter e Roberts Brooke, a cura di. Diseases of blood vessels. Philadelphia: Lea & Febiger, 1985.

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Hoffman, Gary S., Cornelia M. Weyand, Carol A. Langford e Jörg J. Goronzy, a cura di. Inflammatory Diseases of Blood Vessels. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118355244.

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Terence, Bennett, e Gardiner Sheila M, a cura di. Nervous control of blood vessels. [Chur, Switzerland]: Harwood Academic Publishers, 1996.

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Geoffrey, Burnstock, e Griffith Susan G. 1957-, a cura di. Nonadrenergic innervation of blood vessels. Boca Raton, Fla: CRC Press, 1988.

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Geoffrey, Burnstock, e Griffith Susan G, a cura di. Nonadrenergic innervation of blood vessels. Boca Raton, Fla: CRC Press, 1988.

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J, Pedley T., Caro Colin Gerald, Nerem Robert M e World Congress of Biomechanics, (1st : 1990 : La Jolla, Calif), a cura di. Blood flow in large vessels. New York: American Society of Mechanical Engineers, 1992.

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Hoffman, Gary S. Inflammatory diseases of blood vessels. 2a ed. Chichester, West Sussex: Blackwell Pub., 2012.

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Jiri, Widimský, e Herget J, a cura di. Pulmonary blood vessels in lung disease. Basel: Karger, 1990.

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B, Towne Jonathan, e Hollier Larry H, a cura di. Complications in vascular surgery. 2a ed. New York: Marcel Dekker, 2004.

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Capitoli di libri sul tema "Blood-vessels"

1

Sampaio, Walyria O., e Rhian M. Touyz. "Blood Vessels". In Angiotensin-(1-7), 105–16. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22696-1_7.

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Nomura, Yasuya, Yasuya Nomura e Yasuya Nomura. "Blood Vessels". In Morphological Aspects of Inner Ear Disease, 51–83. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54204-9_3.

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Giele, Henk, e Richard Barton. "Blood Vessels". In Disorders of the Hand, 1–21. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6554-5_1.

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Montagna, William, Albert M. Kligman e Kay S. Carlisle. "Blood Vessels". In Atlas of Normal Human Skin, 155–89. New York, NY: Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4613-9202-6_5.

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Rakowska, Adriana, Lidia Rudnicka, Malgorzata Olszewska e Marta Kurzeja. "Blood Vessels". In Atlas of Trichoscopy, 95–108. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-4486-1_5.

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Kohli, Vinay Kumar, Chitra Kohli e Akanksha Singh. "Blood Vessels". In Comprehensive Multiple-Choice Questions in Pathology, 39–44. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-08767-7_6.

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Ernst, Linda M., e Michael K. Fritsch. "Blood Vessels and Lymphatic Vessels". In Color Atlas of Human Fetal and Neonatal Histology, 21–32. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11425-1_2.

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Stepien, Kimberly E. "Retinal Blood Vessels". In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-35951-4_125-3.

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Gwyther, Tracy A., e Marsha W. Rolle. "Regenerating Blood Vessels". In Regenerating the Heart, 393–402. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-021-8_21.

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Hibino, Narutoshi, Christopher Breuer e Toshiharu Shinoka. "Restoring Blood Vessels". In Tissue Engineering in Regenerative Medicine, 211–20. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-322-6_11.

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Atti di convegni sul tema "Blood-vessels"

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Zoller, Christian J., e Alwin Kienle. "Visual appearance of blood vessels". In Diffuse Optical Spectroscopy and Imaging, a cura di Hamid Dehghani e Heidrun Wabnitz. SPIE, 2019. http://dx.doi.org/10.1117/12.2526802.

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Yang, Lung-Jieh, e Bo-Hong Chen. "Blood vessels by fractal gelatin". In 2012 7th IEEE International Conference on Nano/Micro Engineered and Molecular Systems (NEMS). IEEE, 2012. http://dx.doi.org/10.1109/nems.2012.6196750.

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Yuto, Hasebe. "The trees, our blood vessels". In SIGGRAPH Asia 2012 Art Gallery. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2413076.2413077.

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Patrikeev, I., H. P. Brecht, Y. Y. Petrov, I. Y. Petrova, D. S. Prough e R. O. Esenaliev. "Optoacoustic imaging of blood vessels". In Medical Imaging, a cura di Stanislav Y. Emelianov e Stephen A. McAleavey. SPIE, 2007. http://dx.doi.org/10.1117/12.711176.

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Chuang, Winnie, e Astri Handayani. "Retinal Blood Vessels Tortuosity Measurement". In TENCON 2023 - 2023 IEEE Region 10 Conference (TENCON). IEEE, 2023. http://dx.doi.org/10.1109/tencon58879.2023.10322328.

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de Grauw, Cees J., Marc M. van Zandvoort, M. G. oude Egbrink, Dick W. Slaaf e Hans C. Gerritsen. "Two-photon lifetime imaging of blood and blood vessels". In BiOS 2001 The International Symposium on Biomedical Optics, a cura di Ammasi Periasamy e Peter T. C. So. SPIE, 2001. http://dx.doi.org/10.1117/12.424551.

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Astafyeva, Liudmila G., Georgy Zheltov e Wolf-Dieter Schmidt. "Pulse Laser Heating of Blood Vessels". In European Conference on Biomedical Optics. Washington, D.C.: OSA, 2005. http://dx.doi.org/10.1364/ecbo.2005.tuh47.

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Miao, Jingliang, e Haixiang Liu. "Coupling Vibration Analysis of Blood Vessels". In ASME 2001 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/detc2001/vib-21651.

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Abstract This paper proposes and analyzes a simple dynamic model of blood vessel wall. By studying the coupled vibration of blood flow and vessel wall, one can get the natural frequency of a blood vessel. The method used here is generalized calculus of variations. The results show that the flexibility of blood vessels has a greater influence on the fundamental frequency of the coupled vibration and the viscosity of blood vessel has little effect on the frequency of the coupled vibration but has a greater effect on the amplitude of the vibration. Therefore it is important to control both the viscosity and flexibility of blood vessels.
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Connolly, Adam, e Martin Bishop. "The Strength:Interval Curve for Blood Vessels". In 2016 Computing in Cardiology Conference. Computing in Cardiology, 2016. http://dx.doi.org/10.22489/cinc.2016.053-206.

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"WAVELET BASED EXTRACTION OF BLOOD VESSELS". In International Conference on Bio-inspired Systems and Signal Processing. SciTePress - Science and and Technology Publications, 2009. http://dx.doi.org/10.5220/0001558005290534.

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Rapporti di organizzazioni sul tema "Blood-vessels"

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Patrick Buchanan, Patrick Buchanan. Can immune cells prevent 3D printed blood vessels from falling apart? Experiment, settembre 2022. http://dx.doi.org/10.18258/30136.

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Gay, Carol V. Unique Proteins Expressed by Blood Vessels in Skeletal Sites Colonized by Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, agosto 2005. http://dx.doi.org/10.21236/ada446316.

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Gay, Carol V. Unique Proteins Expressed by Blood Vessels in Skeletal Sites Colonized by Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, agosto 2004. http://dx.doi.org/10.21236/ada429149.

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Gay, Carol V. Unique Proteins Expressed by Blood Vessels in Skeletal Sites Colonized by Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, agosto 2006. http://dx.doi.org/10.21236/ada461608.

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Sosa Munguía, Paulina del Carmen, Verónica Ajelet Vargaz Guadarrama, Marcial Sánchez Tecuatl, Mario Garcia Carrasco, Francesco Moccia e Roberto Berra-Romani. Diabetes mellitus alters intracellular calcium homeostasis in vascular endothelial cells: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, maggio 2022. http://dx.doi.org/10.37766/inplasy2022.5.0104.

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Review question / Objective: What are the effects of diabetes mellitus on the calcium homeostasis in vascular endothelial cells? -To describe the effects of diabetes on the mechanisms that regulate intracellular calcium; -To describe other molecules/mechanisms that alters intracellular Ca2+ homeostasis. Condition being studied: Diabetes mellitus is a pathology with a high incidence in the population, characterized by an increase in blood glucose. People with diabetes are 2-4 times more likely to suffer from a cardiovascular complication, such as total or partial loss of sight, myocardial infarction, kidney failure, among others. Cardiovascular complications have been reported to derive from dysfunction of endothelial cells, which have important functions in blood vessels. In order to understand the etiology of this poor function of endothelial cells, it is necessary to study the molecular mechanisms involved in these functions, to identify the effects of diabetes and thus, develop new research that will mitigate the effects of this pathology.
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Kuzmin, Vyacheslav, Alebai Sabitov, Andrei Reutov, Vladimir Amosov, Lidiia Neupokeva e Igor Chernikov. Electronic training manual "Providing first aid to the population". SIB-Expertise, gennaio 2024. http://dx.doi.org/10.12731/er0774.29012024.

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First aid represents the simplest urgent measures necessary to save the lives of victims of injuries, accidents and sudden illnesses. Providing first aid greatly increases the chances of salvation in case of bleeding, injury, cardiac and respiratory arrest, and prevents complications such as shock, massive blood loss, additional displacement of bone fragments and injury to large nerve trunks and blood vessels. This electronic educational resourse consists of four theoretical educational modules: legal aspects of providing first aid to victims and work safety when providing first aid; providing first aid in critical conditions of the body; providing first aid for injuries of various origins; providing first aid in case of extreme exposures, accidents and poisonings. The electronic educational resource materials include 8 emergency conditions and 11 life-saving measures. The theoretical block of modules is presented by presentations, the text of lectures with illustrations, a video film and video lectures. Control classes in the form of test control accompany each theoretical module. After studying all modules, the student passes the final test control. Mastering the electronic manual will ensure a high level of readiness to provide first aid to persons without medical education.
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Gupta, Shikhar, Mehtab Ahmed, Sayema ., Azam Haseen e Saif Quaiser. Relevance of Preoperative Vessel Mapping and Early Postoperative Ultrasonography in Predicting AV Fistula Failure in Chronic Kidney Disease Patients. Science Repository, febbraio 2024. http://dx.doi.org/10.31487/j.rdi.2023.02.02.

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Introduction: The increasing prevalence of chronic kidney disease (CKD), coupled with advancements in the diagnosis and treatment of renal diseases and improvements in life expectancy, has led to a greater number of patients requiring hemodialysis. The preferred method of vascular access for hemodialysis is AV fistula formation; however, it is associated with a high rate of failure. In our prospective study, we focused on 40 CKD patients planned for initiation of maintenance hemodialysis. Methods: We employed preoperative ultrasound mapping to assess cephalic vein diameter, compressibility, and colour flow, as well as radial and brachial artery diameter, peak systolic velocity, and intimal wall calcification. Postoperatively, ultrasound examinations were conducted on day 7 and at 6 weeks to evaluate fistula blood volume and detect any complications. Results: A significant association between fistula failure and cephalic vein diameter, brachial artery diameter, intimal vessel wall calcification, and comorbid conditions like diabetes mellitus was observed. Furthermore, blood flow at day 7 was notably lower in the failure group compared to those with a functioning fistula and any fistula with blood flow <154 ml/min on day 7 may be predictive of early fistula failure. Conclusion: Preoperative vessel mapping and early postoperative ultrasonography is indispensable for patients who require AV fistula formation for hemodialysis and provide valuable information for selecting suitable vessels for successful fistula creation and enable early intervention to salvage a failing fistula after the surgery. By utilizing these, healthcare professionals can make informed decisions and take necessary steps to optimize the outcomes of AV fistula formation in patients undergoing hemodialysis.
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Surgical removal of fibroids has better long-term outcomes than blocking blood vessels. National Institute for Health Research, settembre 2023. http://dx.doi.org/10.3310/nihrevidence_59961.

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Treating all narrowed blood vessels immediately after a heart attack may be better than just treating the single blocked artery. National Institute for Health Research, marzo 2016. http://dx.doi.org/10.3310/signal-000218.

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