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Articoli di riviste sul tema "B cells"

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Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 15 giugno 2024

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1

Tangye, Stuart G., e Kim L. Good. "Human IgM+CD27+B Cells: Memory B Cells or “Memory” B Cells?" Journal of Immunology 179, n. 1 (19 giugno 2007): 13–19. http://dx.doi.org/10.4049/jimmunol.179.1.13.

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2

MacConmara, Malcolm, e James A. Lederer. "B cells". Critical Care Medicine 33, Suppl (dicembre 2005): S514—S516. http://dx.doi.org/10.1097/01.ccm.0000190616.15952.4b.

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3

Dörner, Thomas, e Peter E. Lipsky. "B cells". Current Opinion in Rheumatology 26, n. 2 (marzo 2014): 228–36. http://dx.doi.org/10.1097/bor.0000000000000000.

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4

Ollila, Juha, e Mauno Vihinen. "B cells". International Journal of Biochemistry & Cell Biology 37, n. 3 (marzo 2005): 518–23. http://dx.doi.org/10.1016/j.biocel.2004.09.007.

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5

Haas, Karen M. "Noncanonical B Cells: Characteristics of Uncharacteristic B Cells". Journal of Immunology 211, n. 9 (1 novembre 2023): 1257–65. http://dx.doi.org/10.4049/jimmunol.2200944.

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Abstract (sommario):

Abstract B lymphocytes were originally described as a cell type uniquely capable of secreting Abs. The importance of T cell help in Ab production was revealed soon afterward. Following these seminal findings, investigators made great strides in delineating steps in the conventional pathway that B cells follow to produce high-affinity Abs. These studies revealed generalized, or canonical, features of B cells that include their developmental origin and paths to maturation, activation, and differentiation into Ab-producing and memory cells. However, along the way, examples of nonconventional B cell populations with unique origins, age-dependent development, tissue localization, and effector functions have been revealed. In this brief review, features of B-1a, B-1b, marginal zone, regulatory, killer, NK-like, age-associated, and atypical B cells are discussed. Emerging work on these noncanonical B cells and functions, along with the study of their significance for human health and disease, represents an exciting frontier in B cell biology.

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6

Hananeh, W., R. Al Rukibat e M. Daradka. "Primary splenic diffuse large B-cell lymphoma with multinucleated giant cells in a horse". Veterinární Medicína 66, No. 2 (2 febbraio 2021): 76–79. http://dx.doi.org/10.17221/61/2020-vetmed.

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A diagnosis of a diffuse splenic large B-cell lymphoma with multinucleated giant cells in a 5-year-old mare was made based upon the clinical, pathological, and immunohistochemical findings. The enormous primary splenic mass weighed 51.75 kg. To the best of our knowledge, this is the biggest reported splenic mass and the first case of an equine diffuse large B-cell lymphoma with multinucleated giant cells.

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7

ANDREW, ANN. "DEVELOPMENTAL RELATIONSHIPS OF NEUROENDOCRINE CELLS ". Biomedical Research 6, n. 4 (1985): 191–96. http://dx.doi.org/10.2220/biomedres.6.191.

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8

Yeo, Seung Geun, Joong Saeng Cho, Dong Choon Park e Thomas L. Rothstein. "B-1 Cells Differ from Conventional B (B-2) Cells: Difference in Proliferation". Immune Network 4, n. 3 (2004): 155. http://dx.doi.org/10.4110/in.2004.4.3.155.

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9

YANABA, Koichi. "Regulatory B cells". Japanese Journal of Clinical Immunology 32, n. 3 (2009): 135–41. http://dx.doi.org/10.2177/jsci.32.135.

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10

Cory, Suzanne. "Masterminding B Cells". Journal of Immunology 195, n. 3 (17 luglio 2015): 763–65. http://dx.doi.org/10.4049/jimmunol.1501277.

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11

Desiderio, Stephen. "Becoming B cells". Nature 361, n. 6409 (gennaio 1993): 202–3. http://dx.doi.org/10.1038/361202a0.

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12

Mollejo, Manuela, Javier Menárguez, Eva Cristóbal, Patrocinio Algara, Esther Sánchez-Díaz, Máximo Fraga e Miguel A. Piris. "Monocytoid B Cells". American Journal of Surgical Pathology 18, n. 11 (novembre 1994): 1131–39. http://dx.doi.org/10.1097/00000478-199411000-00007.

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13

Dart, Anna. "Bad B cells". Nature Reviews Cancer 18, n. 2 (febbraio 2018): 66. http://dx.doi.org/10.1038/nrc.2018.7.

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14

Fend, Falko, David Nachbaur e Heinz Huber. "Thymic B Cells". American Journal of Clinical Pathology 96, n. 1 (1 luglio 1991): 148–49. http://dx.doi.org/10.1093/ajcp/96.1.148.

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15

CHEIKH, MARCIA CURY, MIREILLE-HONTEBEYRIE JOSKOWICZ, ANTONIO COUTINHO e PAOLA MINOPRIO. "CD5 B Cells". Annals of the New York Academy of Sciences 651, n. 1 (maggio 1992): 557–63. http://dx.doi.org/10.1111/j.1749-6632.1992.tb24662.x.

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16

Tierens, Anne, Jan Delabie e Chris De Wolf-Peeters. "Monocytoid B cells". Blood 96, n. 4 (15 agosto 2000): 1612–14. http://dx.doi.org/10.1182/blood.v96.4.1612.

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17

Tierens, Anne, Jan Delabie e Chris De Wolf-Peeters. "Monocytoid B cells". Blood 96, n. 4 (15 agosto 2000): 1612–14. http://dx.doi.org/10.1182/blood.v96.4.1612.h8001608c_1612_1614.

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18

Dempsey, Laurie A. "Clipping B cells". Nature Immunology 14, n. 3 (15 febbraio 2013): 204. http://dx.doi.org/10.1038/ni.2562.

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19

Fehervari, Zoltan. "Thymic B cells". Nature Immunology 14, n. 12 (15 novembre 2013): 1211. http://dx.doi.org/10.1038/ni.2777.

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20

Papatriantafyllou, Maria. "ChATty B cells". Nature Reviews Immunology 13, n. 2 (25 gennaio 2013): 70. http://dx.doi.org/10.1038/nri3396.

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21

Kurosaki, Tomohiro, Kohei Kometani e Wataru Ise. "Memory B cells". Nature Reviews Immunology 15, n. 3 (13 febbraio 2015): 149–59. http://dx.doi.org/10.1038/nri3802.

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22

Sinkorova, Z., J. Sinkora, L. Zarybnicka, Z. Vilasova e J. Pejchal. "Radiosensitivity of peripheral blood B cells in pigs". Veterinární Medicína 54, No. 5 (1 giugno 2009): 223–35. http://dx.doi.org/10.17221/59/2009-vetmed.

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Abstract (sommario):

: Swine are here introduced to biodosimetry in an attempt to develop a large animal model allowing for comparison of <I>in vitro</I> experiments with the <I>in vivo</I> processes occurring after exposure to gamma radiation. This work investigates the radiosensitivity of the B cell compartment in peripheral blood. Four-week-old piglets were irradiated using the whole body protocol or full blood samples were irradiated <I>in vitro</I> in the dose range of 0–10 Gy. Relative radioresistance of B cell subpopulations and subsets was determined by measuring their relative numbers in leukocyte preparations at selected time intervals after irradiation using two color immunophenotyping and flow cytometry. Porcine B cells represent the most radiosensitive lymphocyte population in peripheral blood. Among B cell subpopulations and subsets investigated, the CD21+SWC7+ and CD21+CD1+ cells are highly radiosensitive and possess biodosimetric potential, at least in the range of low doses. Differences between cultures irradiated <I>in vitro</I> and lymphocyte dynamics in peripheral blood of irradiated animals clearly document the limits of <I>in vitro</I> data extrapolation in biodosimetry. We have shown that pigs can successfully be used in radiobiology and experimental biodosimetry due mainly to their availability, size and a relatively broad spectrum of available immunoreagents for lymphocyte classification.

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23

Quách, Tâm D., Thomas J. Hopkins, Nichol E. Holodick, Raja Vuyyuru, Tim Manser, Ruthee-Lu Bayer e Thomas L. Rothstein. "Human B-1 and B-2 B Cells Develop from Lin−CD34+CD38loStem Cells". Journal of Immunology 197, n. 10 (7 ottobre 2016): 3950–58. http://dx.doi.org/10.4049/jimmunol.1600630.

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24

DeFranco, Anthony L. "Between B cells and T cells". Nature 351, n. 6328 (giugno 1991): 603–4. http://dx.doi.org/10.1038/351603a0.

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25

Rodríguez-Pinto, Daniel. "B cells as antigen presenting cells". Cellular Immunology 238, n. 2 (dicembre 2005): 67–75. http://dx.doi.org/10.1016/j.cellimm.2006.02.005.

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26

Bell, Elaine. "Turning B cells into T cells". Nature Reviews Immunology 7, n. 11 (novembre 2007): 838–39. http://dx.doi.org/10.1038/nri2196.

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27

Kurtin, Paul J. "Marginal Zone B Cells, Monocytoid B Cells, and the Follicular Microenvironment". American Journal of Clinical Pathology 114, n. 4 (1 ottobre 2000): 505–8. http://dx.doi.org/10.1309/l69g-f64h-4f3j-l2r5.

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28

Küppers, Ralf. "Human memory B cells: Memory B cells of a special kind". Immunology & Cell Biology 86, n. 8 (12 agosto 2008): 635–36. http://dx.doi.org/10.1038/icb.2008.59.

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29

Kantor, Aaron B. "The development and repertoire of B-1 cells (CD5 B cells)". Immunology Today 12, n. 11 (novembre 1991): 389–91. http://dx.doi.org/10.1016/0167-5699(91)90136-h.

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30

Cook, Matthew C. "B cells: B cell back catalogue (remastered)". Immunology & Cell Biology 86, n. 2 (22 gennaio 2008): 109–10. http://dx.doi.org/10.1038/sj.icb.7100162.

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31

Lydyard, Peter M., Andrew P. Jewell, Christoph Jamin e Pierre Y. Youinou. "CD5 B cells and B-cell malignancies". Current Opinion in Hematology 6, n. 1 (gennaio 1999): 30. http://dx.doi.org/10.1097/00062752-199901000-00006.

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32

Scott, David W. "Transduced B cells: B is for ‘beneficial’!" European Journal of Immunology 41, n. 6 (26 maggio 2011): 1528–30. http://dx.doi.org/10.1002/eji.201141649.

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33

KAWANO, Eisuke, Taku TORIUMI, Shinya IGUCHI, Daigo SUZUKI, Shuichi SATO e Masaki HONDA. "Induction of neural crest cells from human dental pulp-derived induced pluripotent stem cells ". Biomedical Research 38, n. 2 (2017): 135–47. http://dx.doi.org/10.2220/biomedres.38.135.

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34

Krieger, J. I., S. F. Grammer, H. M. Grey e R. W. Chesnut. "Antigen presentation by splenic B cells: resting B cells are ineffective, whereas activated B cells are effective accessory cells for T cell responses." Journal of Immunology 135, n. 5 (1 novembre 1985): 2937–45. http://dx.doi.org/10.4049/jimmunol.135.5.2937.

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Abstract (sommario):

Abstract In this study, we have investigated the ability of splenic B cells to act as antigen-presenting cells. Previous data had established that lipopolysaccharide (LPS)-activated B cells were effective antigen-presenting cells; however, the relative capacity of resting B cells to carry out this function remains controversial. Splenic B cells from naive BALB/c mice were depleted of macrophages, dendritic cells, and T cells, and were fractionated on the basis of cell density by using Percoll gradient centrifugation. Fractions were collected from the 50/60, 60/65, and 65/72% interfaces and from greater than 72% (pellet). Cytofluorograph analysis of the fractionated B cells showed that the two lower density fractions (50/60 and 60/65) contained a number of cells which, by cell size determination, appeared to be activated B cells, whereas the two higher density fractions (65/72 and greater than 72) appeared to contain predominantly small resting B cells contaminated by many fewer activated B cells. Functionally, the capacity of fractionated B cells to act as accessory cells for a concanavalin A response or present the antigens chicken ovalbumin (OVA) or OVA-tryptic digest gave similar results, which indicated a striking hierarchy of accessory cell function in the different Percoll fractions. When normalized to the most active low-density fraction (50/60%), the activity of the other fractions were: 60/65 = 78%; 65/72 = 25%; and greater than 72 = 4%. The differences in the functional capacity between the various Percoll fractions did not appear to be due to differences in Ia expression. Although the expression of Ia varied approximately 12-fold within any one fraction, there was little difference in the mean amount of Ia on cells obtained from the various fractions. Kinetic studies showed that activation of B cells with LPS and dextran sulfate resulted in the expression of two stages of functional development. The first stage was an increased efficiency of accessory cell function that was abrogated by irradiation with 4000 rad followed by a second stage, which was characterized by the acquisition of resistance to treatment with 4000 rad. When nonfractionated B cells that had been stimulated with LPS and DexSO4 were sorted on the basis of cell size into a small B cell fraction and a large B cell fraction, only the large B cells were able to present antigen. Taken together, these data suggest that much of the accessory cell function associated with splenic B cells can be accounted for by the relatively small percentage of activated B cells present in the spleen.(ABSTRACT TRUNCATED AT 400 WORDS)

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35

Pattarabanjird, Tanyaporn, Cynthia Li e Coleen McNamara. "B Cells in Atherosclerosis". JACC: Basic to Translational Science 6, n. 6 (giugno 2021): 546–63. http://dx.doi.org/10.1016/j.jacbts.2021.01.006.

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36

Engelhard, Victor, Jose R. Conejo-Garcia, Rafi Ahmed, Brad H. Nelson, Karen Willard-Gallo, Tullia C. Bruno e Wolf H. Fridman. "B cells and cancer". Cancer Cell 39, n. 10 (ottobre 2021): 1293–96. http://dx.doi.org/10.1016/j.ccell.2021.09.007.

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37

Linnebacher, Michael, e Claudia Maletzki. "Tumor-infiltrating B cells". OncoImmunology 1, n. 7 (ottobre 2012): 1186–88. http://dx.doi.org/10.4161/onci.20641.

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38

Gisselbrecht, Christian. "Large B cells lymphoma". Hématologie 20, n. 3 (maggio 2014): 183–88. http://dx.doi.org/10.1684/hma.2014.0947.

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39

Hamel, Keith M., Vladimir M. Liarski e Marcus R. Clark. "Germinal Center B-cells". Autoimmunity 45, n. 5 (2 aprile 2012): 333–47. http://dx.doi.org/10.3109/08916934.2012.665524.

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40

Bashyam, Hema. "Gut-friendly B cells?" Journal of Experimental Medicine 205, n. 6 (2 giugno 2008): 1246. http://dx.doi.org/10.1084/jem.2056iti3.

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41

Pujanandez, Lindsey. "Teaching baby B cells". Science 363, n. 6430 (28 febbraio 2019): 941.6–942. http://dx.doi.org/10.1126/science.363.6430.941-f.

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42

Scanlon, Seth Thomas. "IgE B cells unmasked". Science 362, n. 6420 (13 dicembre 2018): 1259.6–1260. http://dx.doi.org/10.1126/science.362.6420.1259-f.

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43

Dörner, Thomas, Annett M. Jacobi e Peter E. Lipsky. "B cells in autoimmunity". Arthritis Research & Therapy 11, n. 5 (2009): 247. http://dx.doi.org/10.1186/ar2780.

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44

Seton-Rogers, Sarah. "Spotlight on B cells". Nature Reviews Cancer 16, n. 2 (29 gennaio 2016): 67. http://dx.doi.org/10.1038/nrc.2016.7.

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45

Bernard, Nicholas J. "Double-negative B cells". Nature Reviews Rheumatology 14, n. 12 (26 ottobre 2018): 684. http://dx.doi.org/10.1038/s41584-018-0113-6.

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46

Zachary, Andrea A., Dessislava Kopchaliiska, Robert A. Montgomery e Mary S. Leffell. "HLA-Specific B Cells". Transplantation 83, n. 7 (aprile 2007): 982–88. http://dx.doi.org/10.1097/01.tp.0000259017.32857.99.

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47

Zachary, Andrea A., Dessislava Kopchaliiska, Robert A. Montgomery, Joseph K. Melancon e Mary S. Leffell. "HLA-Specific B Cells". Transplantation 83, n. 7 (aprile 2007): 989–94. http://dx.doi.org/10.1097/01.tp.0000259019.68244.d7.

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48

Allie, S. Rameeza, e Troy D. Randall. "Resident Memory B Cells". Viral Immunology 33, n. 4 (1 maggio 2020): 282–93. http://dx.doi.org/10.1089/vim.2019.0141.

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49

Gough, N. R. "Suppressed by B Cells". Science Signaling 7, n. 318 (25 marzo 2014): ec79-ec79. http://dx.doi.org/10.1126/scisignal.2005296.

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50

Cancro, Michael P. "Age-Associated B Cells". Annual Review of Immunology 38, n. 1 (26 aprile 2020): 315–40. http://dx.doi.org/10.1146/annurev-immunol-092419-031130.

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Abstract (sommario):

The age-associated B cell subset has been the focus of increasing interest over the last decade. These cells have a unique cell surface phenotype and transcriptional signature, and they rely on TLR7 or TLR9 signals in the context of Th1 cytokines for their formation and activation. Most are antigen-experienced memory B cells that arise during responses to microbial infections and are key to pathogen clearance and control. Their increasing prevalence with age contributes to several well-established features of immunosenescence, including reduced B cell genesis and damped immune responses. In addition, they are elevated in autoimmune and autoinflammatory diseases, and in these settings they are enriched for characteristic autoantibody specificities. Together, these features identify age-associated B cells as a subset with pivotal roles in immunological health, disease, and aging. Accordingly, a detailed understanding of their origins, functions, and physiology should make them tractable translational targets in each of these settings.

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