Letteratura scientifica selezionata sul tema "Antigènes T"
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Articoli di riviste sul tema "Antigènes T":
Delafosse, Arnaud, Zakaria Bengaly e Gérard Duvallet. "Absence d'interaction des infections à Trypanosoma theileri avec le diagnostic des trypanosomoses animales par détection des antigènes circulants". Revue d’élevage et de médecine vétérinaire des pays tropicaux 48, n. 1 (1 gennaio 1995): 18–20. http://dx.doi.org/10.19182/remvt.9480.
Desquesnes, Marc, e Stéphane De La Rocque. "Comparaison de la sensibilité du test de Woo et d'un test de détection des antigènes de Trypanosoma vivax chez deux moutons expérimentalement infectés avec une souche guyanaise du parasite". Revue d’élevage et de médecine vétérinaire des pays tropicaux 48, n. 3 (1 marzo 1995): 247–53. http://dx.doi.org/10.19182/remvt.9454.
Diall, O., V. M. Nantulya, Antony George Luckins, B. Diarra e Boubacar Kouyaté. "Evaluation des tests immune-enzymatiques de détection des antigènes au moyen des anticorps mono- et polyclonaux pour le diagnostic de l’infection à Trypanosoma evansi chez le dromadaire (Camelus dromedarius)". Revue d’élevage et de médecine vétérinaire des pays tropicaux 45, n. 2 (1 febbraio 1992): 149–53. http://dx.doi.org/10.19182/remvt.8941.
Brown, W. C., S. Zhao, V. M. Woods, D. A. E. Dobbelaere e A. C. Rice Ficht. "Des clones de cellules T CD4+ spécifiques pour Babesia bovis, de bovins immunisés, expriment le profil de cytokines des cellules Th0 ou des Th1". Revue d’élevage et de médecine vétérinaire des pays tropicaux 46, n. 1-2 (1 gennaio 1993): 65–69. http://dx.doi.org/10.19182/remvt.9400.
Doko, A., A. Verhulst, V. S. Pandey, Philippe Büscher e Veerle Lejon. "Détection d'antigènes circulants au cours d'une infection expérimentale à T. brucei brucei chez des bovins Borgou, Lagunaire et zébus Bororo blancs". Revue d’élevage et de médecine vétérinaire des pays tropicaux 49, n. 3 (1 marzo 1996): 207–11. http://dx.doi.org/10.19182/remvt.9514.
Barry, A. M., François Roger, M. B. Diallo e S. Geerts. "Evaluation de la séroprévalence de la trypanosomose bovine en Guinée". Revue d’élevage et de médecine vétérinaire des pays tropicaux 61, n. 3-4 (1 marzo 2008): 177. http://dx.doi.org/10.19182/remvt.9985.
Laetitia, Delort, Hermine Billard, Marie-Paule Vasson e Florence Caldefie-Chézet. "42: Glycosylations aberrantes et cancer du sein: évaluation in vitro de l’expression des antigènes Tn et T". Bulletin du Cancer 97, n. 1 (marzo 2010): S37. http://dx.doi.org/10.1016/s0007-4551(15)31135-8.
Sanfo, N., A. Ciree, C. Giraut, P. Diot, H. Watier, S. Marchand-Adam e G. Desoubeaux. "Antigènes recombinants d’Aspergillus fumigatus : existe-t-il des corrélations cliniques, biologiques et radiologiques chez les patients présentant une hypersensibilité aspergillaire ?" Revue des Maladies Respiratoires 36 (gennaio 2019): A42. http://dx.doi.org/10.1016/j.rmr.2018.10.075.
Tartour, Eric. "Vaccins anti-cancer : quel avenir dans les stratégies d’immunothérapie anti-cancéreuse ?" Biologie Aujourd'hui 212, n. 3-4 (2018): 69–76. http://dx.doi.org/10.1051/jbio/2019002.
Desquesnes, Marc, Jean-François Michel, Stéphane De La Rocque, Philippe Solano, Leopold Millogo, Zakaria Bengaly e I. Sididé. "Enquête parasitologique et sérologique (Elisa-indirect) sur les trypanosomoses des bovins dans la zone de Sidéradougou, Burkina Faso". Revue d’élevage et de médecine vétérinaire des pays tropicaux 52, n. 3-4 (1 marzo 1999): 223–32. http://dx.doi.org/10.19182/remvt.9667.
Tesi sul tema "Antigènes T":
Guillaume, Yves. "Caractérisation fonctionnelle de la molécule CD277 dans les lymhocytres T Vγ9Vδ2". Aix-Marseille 2, 2009. http://www.theses.fr/2009AIX20654.
Favre, Cédric. "Rôle de la molécule d'adhésion CD28 dans l'activation lymphocytaire T". Aix-Marseille 2, 2003. http://www.theses.fr/2003AIX22031.
Guirlinger, Marie-Joëlle. "Caractérisation immunochimique des antigènes excrétés-sécrétés par Toxoplasma gondii". Lyon 1, 1993. http://www.theses.fr/1993LYO10073.
Louveau, Antoine. "Rôle de la molécule adaptatrice CD3zeta et de la voie de co-stimulation CTLA4/CD80-CD86 dans le développement et la plasticité neuronale". Nantes, 2013. http://archive.bu.univ-nantes.fr/pollux/show.action?id=428743af-fa66-4821-8b8d-cd4e57adf2dd.
Recent studies have shown that molecules thought to be restricted to the immune system (IS) are expressed in the central nervous system (CNS) where they exert non-immune function on neuronal development and plasticity. We studied the cerebral function of the immunoreceptor CD3zeta and of the CTLA4/CD80-CD86 co-stimulatory pathway. Regarding CD3zeta, we found that CD3zeta inhibits neurite formation at early neuronal developmental stages, through a mechanism involving the ephrinA1/EphA4 pathway. In mature neurons, CD3zeta supports the synaptic localization of the NMDAR subunit GluN2A, and is required for the CaMKII-dependent signaling pathway underlying synaptic plasticity. These mechanisms are associated to a pro-cognitive action of CD3zeta on learning and memory behavior, independently of its immune role on T lymphocytes. Concerning the co-stimulatory pathway, we showed that the molecule CTLA4-Ig promotes axonal growth in vivo in a model of intracerebral transplantation. This effect is correlated with a morphological plasticity of microglial cells mediated through the microglial receptor CD86, and with an increased expression of BDNF and arginase 1. These results suggest that the beneficial effect of CTLA4-Ig on neuronal survival/growth is mediated through the release of microglial BDNF and arginase 1. Our studies reveal unexpected role of CD3zeta and the co-stimulatory molecules in the CNS. Our results highligt new mechanisms important to better understand neuroimmune interactions in normal and pathological conditions
Fornasa, Giulia. "Clivage du CD31 des lymphocytes T dans l'athérosclérose". Paris 7, 2011. http://www.theses.fr/2011PA077024.
Immune responses are controlled by inhibitory immune receptors such as CD31 that belongs to the family of Ig-like ITIM-containing receptors, ITIM for Immune receptor Tyrosine-based Inhibitory Motifs. The hemophilic engagement of the CD31 triggers the phosphorylation of the ITIM motifs leading to the inhibition of cellular activation pathways. During my thesis I have demonstrated that CD31 on T lymphocytes undergoes proteolytic cleavage upon cell activation. The concentration of the CD31 truncated soluble form released after cleavage in the circulation likely reflects the activation state of T lymphocytes. Indeed, it is higher in the plasma of patients with acute coronary syndromes and thus its measure could represent a potential novel diagnostic and prognostic tool for the evaluation of the cardiovascular disease risk. The homophilic engagement of CD31, and thus the inhibitory signaling, is disabled by the cleavage. However I demonstrated that an effective signal transmission can still occur targeting with a CD31-derived peptide the juxtamembrane portion, that remains expressed after the cleavage. The peptide, binding to its homotypic sequence on the truncated membrane-anchored fragment on T cells, restores the ITIM-dependent CD31 signalling and its inhibitory properties in vitro and in vivo. The administration of the CD31 peptide prevents experimental atherosclerosis development, inhibiting the pathologic immune responses favored by the CD31 cleavage on T cells. The CD31 peptide can be envisaged as a therapeutic tool to treat chronic inflammatory diseases characterized by massive lymphocytes activation
Davodeau, François. "Étude de la physiologie et du répertoire de reconnaissance des lymphocytes T [gamma delta] humains : contribution à l'étude des mécanismes générateurs de la diversité des récepteurs à l'antigène des lymphocytes T". Nantes, 1994. http://www.theses.fr/1994NANT12VS.
Harly, Christelle. "Modalités d’activation et fonctions des lymphocytes T gamma-delta humains". Nantes, 2011. http://archive.bu.univ-nantes.fr/pollux/show.action?id=1423324b-316e-46a1-b9c4-b4b7b772a246.
γdelta T lymphocytes (γdelta TL), stand in between innate and adaptative immunity. These lymphocytes are able to respond to various antigenic stimulations which reflects their potential implication in many infectious and tumoral physiopathological contexts. Fine activation modalities of these cells remain unclear, although their phenotypical and functional properties turn them potentially into pivotal players of the immune response. Understanding the modalities of activation of γdeltaTL currently represents an important issue for understanding the biology of these cells and evaluation of their therapeutical potential. The work achieved in this thesis is focused on the activation modalities of several humanγ deltaTL subsets, and leads to the identification of three major molecular players in these processes. (i) ILT2 expressed at the cell surface of B cell lines is essential for the activation of Vdelta2neg γdelta TL, (ii) EphA2 expressed by epithelial tumor cells is a key partner of Vdelta1pos γdeltaTL activation, (iii) expression of CD277 by target cells is mandatory for their specific recognition by Vdelta2pos γdeltaTL. However, the mecanisms implying these molecules in γdeltaTL activation processes remain still to be clearly defined and will be analyzed in further investigations. The expected results should lead to the evaluation of the role of these molecular and cellular players in vivo, along with their immunotherapeutical potential in various human pathologies
Huet, Stéphane. "Etude de l'activation lymphocytaire T par les molécules CD2 et CD3". Compiègne, 1988. http://www.theses.fr/1988COMPD125.
Human T lymphocyte activation by monoclonal antibodies (mAb) directed against CD2 or CD3 molecules required the presence of accessory cells (AC). It also appeared that the signal delivered to purify T-cells by different CD2 mAb pairs varied according to the CD2 pair used. Paraformaldehyde-fixed AC fully restored purified T-cells mitosis triggered by all CD2 pairs tested or by CD3 mAb. The required interaction between T-cells and AC involved CD18 molecules and HLA class I molecules from AC. In searching for the HLA class I counterpart from AC we found that both CD4+ and CD8+ cells responded to stimulation via CD2 and CD3. However, within the CD4 subsets, the CD4+ CD45R+ subset did not respond while the CD4+ CDw29+ did. Since the first subset is committed for the induction of suppression while the second to the induction of help, one can foresee how the immune response could develop in one direction or the other following the type of interaction that occurs during T-cell activation
Pepin, Elsa. "Étude des conséquences d'un stress hyperthermique sur l'appretement et la présentation des antigènes". Grenoble 1, 1998. http://www.theses.fr/1998GRE10042.
Espinosa, Carrasco Gabriel. "L'activation des cellules T CD8+ et T CD4+ en réponse aux auto-antigènes : du tissu lymphoïde à l'organe cible". Thesis, Montpellier, 2016. http://www.theses.fr/2016MONTT026.
The immune system has evolved multiple mechanisms of peripheral tolerance to control CD8+ T cell responses. Under particular conditions that are not yet well understood, potentially autoreactive T cells may override tolerance and differentiate into effector cells capable of targeting the own components of the organism resulting in self-reactivity. Utilizing transgenic mice expressing a model antigen in the beta cells of the pancreas, I have studied two important processes involved in CD8+ T cells differentiation in response to self-antigens. 1) Role of lipopolysaccharides (LPS) translocation in the breakdown of CD8+ T cell tolerance. It has been previously shown in our laboratory that lymphodepleting protocols, such as total body irradiation, promote breakdown of peripheral CD8+ T cell tolerance. Irradiation induces translocation of commensal bacteria LPS, a potent innate immune system activator, into the bloodstream. My data demonstrated that LPS translocation correlated with systemic activation of CD11c+ dendritic cells (DC), in particular CD8+ DC, responsible for pancreatic self-antigen cross-presentation, in lymphoid tissue. While antibiotic treatment of mice before irradiation prevented LPS translocation, DC activation was only partially affected, and onset of autoimmunity and breakdown of CD8+ T cell tolerance could not be prevented.2) Intra-vital visualization of effector CD8+ and CD4+ T cell cooperation in beta cell destruction in the pancreas. Using two-photon microscopy, I have been able, for the first time, to simultaneously analyze dynamics of fluorescently tagged autoreactive CD8+ and CD4+ T cells as they infiltrated the pancreas and induced autoimmune diabetes. I found that T cell infiltration promoted extracellular matrix remodeling in the pancreas, which in turn served as a scaffold for T cell migration. In addition, I showed that MHC class II dependent arrest of effector CD4+ T cells, due to interactions with antigen presenting cells, occasionally also implicating CD8+ T cells, provided help to effector CD8+ T cells in maintaining their effector functions
Libri sul tema "Antigènes T":
1945-, Mak Tak W., a cura di. The T-cell receptors. New York: Plenum Press, 1988.
Oksenberg, Jorge R. Polymerase chain reaction and the analysis of the t cell receptor repertoire. Austin, Tex: R.G. Landes, 1992.
Cold Spring Harbor Symposia on Quantitative Biology (54th 1989). Immunological recognition. Cold Spring Harbor, N.Y: Cold Spring Laboratory Laboratory Press, 1989.
Marincola, Francesco M., e Dirk Nagorsen. Analyzing T Cell Responses: How to Analyze Cellular Immune Responses Against Tumor Associated Antigens. Springer, 2010.
(Editor), Dirk Nagorsen, e F. M. Marincola (Editor), a cura di. Analyzing T Cell Responses: How to analyze cellular immune responses against tumor associated antigens. Springer, 2005.
Marincola, Francesco M., e Dirk Nagorsen. Analyzing T Cell Responses: How to Analyze Cellular Immune Responses Against Tumor Associated Antigens. Springer London, Limited, 2006.
Reinherz. Leukocyte Typing Ii (Leukocyte Typing II, Vol 3). Springer, 1986.
Immunological Recognition. Cold Spring Harbor Laboratory Press, 1990.