Bibliographies thématiques / Underlying cognitive deficits

Sommaire

Articles de revues

Littérature scientifique sur le sujet « Underlying cognitive deficits »

Auteur : Grafiati

Publié le 13 avril 2024

Créez une référence correcte selon les styles APA, MLA, Chicago, Harvard et plusieurs autres

Choisissez une source :

Consultez les listes thématiques d’articles de revues, de livres, de thèses, de rapports de conférences et d’autres sources académiques sur le sujet « Underlying cognitive deficits ».

À côté de chaque source dans la liste de références il y a un bouton « Ajouter à la bibliographie ». Cliquez sur ce bouton, et nous générerons automatiquement la référence bibliographique pour la source choisie selon votre style de citation préféré : APA, MLA, Harvard, Vancouver, Chicago, etc.

Vous pouvez aussi télécharger le texte intégral de la publication scolaire au format pdf et consulter son résumé en ligne lorsque ces informations sont inclues dans les métadonnées.

Articles de revues sur le sujet "Underlying cognitive deficits"

Stanford, George, and Thomas Oakland. "Cognitive Deficits Underlying Learning Disabilities." School Psychology International 21, no. 3 (2000): 306–21. http://dx.doi.org/10.1177/0143034300213007.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

Dodd, Barbara, Judi Leahy, and Gail Hambly. "Phonological disorders in children: Underlying cognitive deficits." British Journal of Developmental Psychology 7, no. 1 (1989): 55–71. http://dx.doi.org/10.1111/j.2044-835x.1989.tb00788.x.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

Loe, Irene M., Heidi M. Feldman, Enami Yasui, and Beatriz Luna. "Oculomotor Performance Identifies Underlying Cognitive Deficits in Attention-Deficit/Hyperactivity Disorder." Journal of the American Academy of Child & Adolescent Psychiatry 48, no. 4 (2009): 431–40. http://dx.doi.org/10.1097/chi.0b013e31819996da.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

Wilmer, Jeremy B., Alexandra J. Richardson, Yue Chen, and John F. Stein. "Two Visual Motion Processing Deficits in Developmental Dyslexia Associated with Different Reading Skills Deficits." Journal of Cognitive Neuroscience 16, no. 4 (2004): 528–40. http://dx.doi.org/10.1162/089892904323057272.

Texte intégral

Résumé :

Developmental dyslexia is associated with deficits in the processing of visual motion stimuli, and some evidence suggests that these motion processing deficits are related to various reading subskills deficits. However, little is known about the mechanisms underlying such associations. This study lays a richer groundwork for exploration of such mechanisms by more comprehensively and rigorously characterizing the relationship between motion processing deficits and reading subskills deficits. Thirty-six adult participants, 19 of whom had a history of developmental dyslexia, completed a battery of visual, cognitive, and reading tests. This battery combined motion processing and reading subskills measures used across previous studies and added carefully matched visual processing control tasks. Results suggest that there are in fact two distinct motion processing deficits in developmental dyslexia, rather than one as assumed by previous research, and that each of these deficits is associated with a different type of reading subskills deficit. A deficit in detecting coherent motion is selectively associated with low accuracy on reading subskills tests, and a deficit in discriminating velocities is selectively associated with slow performance on these same tests. In addition, evidence from visual processing control tasks as well as self-reports of ADHD symptoms suggests that these motion processing deficits are specific to the domain of visual motion, and result neither from a broader visual deficit, nor from the sort of generalized attention deficit commonly comorbid with developmental dyslexia. Finally, dissociation between these two motion processing deficits suggests that they may have distinct neural and functional underpinnings. The two distinct patterns of motion processing and reading deficits demonstrated by this study may reflect separable underlying neurocognitive mechanisms of developmental dyslexia.

Styles APA, Harvard, Vancouver, ISO, etc.

Sagar, H. J. "Specificity of Cognitive Impairment in Neurological Disease: A Methodological Critique of Parkinson’s Disease." Behavioural Neurology 4, no. 2 (1991): 89–102. http://dx.doi.org/10.1155/1991/625647.

Texte intégral

Résumé :

Multiple cognitive deficits have been recognized in many neurological disorders but the specificity of the findings and the relationship to the underlying neuropathology remain obscure. Definitions of dementia have been proposed based on symptom profiles of the cognitive disorder and qualitative differences have been claimed between dementias of different aetiology. Some conditions have been claimed to show patterns of cognitive deficit that are distinguished from dementia and related to specific neuropathology or psychological processes, e.g. frontal lobe deficits in Parkinson's disease. Sometimes, a relationship has been established between certain cognitive deficits and particular neurochemical deficits which has led to the notion of specific drug treatment, e.g. cholinergic deficits and memory failure in Alzheimer's disease. However, these conclusions are often potentially flawed by methodological inadequacies. This critique presents some methodological issues relevant to the study of brain-behaviour and drug-behaviour relationships in syndromes of multiple cognitive deficit, using Parkinson's disease as the model. The following recommendations are made: rigid diagnostic criteria; representative patient groups; avoidance of arbitrary quantitative criteria to limit definitions of dementia; matching of groups for overall level of cognitive impairment in the search for qualitative cognitive differences related to neuropathology or effects of particular drugs; the use of suitable controls in patient groups, neuropsychological tests and treatment regimes; the use of specific quantitative tests of cognition, affect and motor disability; and longitudinal, compared with cross-sectional, study design.

Styles APA, Harvard, Vancouver, ISO, etc.

Cory-Slechta, Deborah. "Delineating behavioral domains underlying lead-induced cognitive deficits." Neurotoxicology and Teratology 32, no. 4 (2010): 497. http://dx.doi.org/10.1016/j.ntt.2010.04.005.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

McPhilemy, Genevieve, Leila Nabulsi, Liam Kilmartin, et al. "Neuroanatomical Dysconnectivity Underlying Cognitive Deficits in Bipolar Disorder." Biological Psychiatry: Cognitive Neuroscience and Neuroimaging 5, no. 2 (2020): 152–62. http://dx.doi.org/10.1016/j.bpsc.2019.09.004.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

Kalezic, Nevena, Ivan Dimitrijevic, Ljubica Leposavic, et al. "Postoperative cognitive deficits." Srpski arhiv za celokupno lekarstvo 134, no. 7-8 (2006): 331–38. http://dx.doi.org/10.2298/sarh0608331k.

Texte intégral

Résumé :

Cognitive dysfunctions are relatively common in postoperative and critically ill patients. This complication not only compromises recovery after surgery, but, if persistent, it minimizes and compromises surgery itself. Risk factors of postoperative cognitive disorders can be divided into age and comorbidity dependent, and those related to anesthesia and surgery. Cardiovascular, orthopedic and urologic surgery carries high risk of postoperative cognitive dysfunction. It can also occur in other types of surgical treatment, especially in elderly. Among risk factors of cognitive disorders, associated with comorbidity, underlying psychiatric and neurological disorders, substance abuse and conditions with elevation of intracranial pressure are in the first place in postoperative patients. Preoperative and perioperative predisposing conditions for cognitive dysfunction and their incidence were described in our paper. These are: geriatric patients, patients with substance abuse, preexisting psychiatric or cognitive disorders, neurologic disease with high intracranial pressure, cerebrovascular insufficiency, epilepsia, preeclampsia, acute intermittent porphyria, operation type, brain hypoxia, changes in blood glucose level, electrolyte imbalance, anesthetic agents, adjuvant medication and intraoperative awareness. For each of these factors, evaluation, prevention and treatment strategies were suggested, with special regard on anesthetic technique.

Styles APA, Harvard, Vancouver, ISO, etc.

Nieto, Rodrigo R., Hernán Silva, Alejandra Armijo, et al. "BDNF and Cognitive Function in Chilean Schizophrenic Patients." International Journal of Molecular Sciences 24, no. 13 (2023): 10569. http://dx.doi.org/10.3390/ijms241310569.

Texte intégral

Résumé :

Despite cognitive symptoms being very important in schizophrenia, not every schizophrenic patient has a significant cognitive deficit. The molecular mechanisms underlying the different degrees of cognitive functioning in schizophrenic patients are not sufficiently understood. We studied the relation between brain-derived neurotrophic factor (BDNF) and cognitive functioning in two groups of schizophrenic patients with different cognitive statuses. According to the Montreal Cognitive Assessment (MoCA) results, the schizophrenic patients were classified into two subgroups: normal cognition (26 or more) and cognitive deficit (25 or less). We measured their plasma BDNF levels using ELISAs. The statistical analyses were performed using Spearman’s Rho and Kruskal–Wallis tests. We found a statistically significant positive correlation between the plasma BDNF levels and MoCA score (p = 0.04) in the subgroup of schizophrenic patients with a cognitive deficit (n = 29). However, this correlation was not observed in the patients with normal cognition (n = 11) and was not observed in the total patient group (n = 40). These results support a significant role for BDNF in the cognitive functioning of schizophrenics with some degree of cognitive deficit, but suggest that BDNF may not be crucial in patients with a normal cognitive status. These findings provide information about the molecular basis underlying cognitive deficits in this illness.

Styles APA, Harvard, Vancouver, ISO, etc.

Zulkifli, Nathratul Ayeshah, JesJeet Singh Gill Jeswant Singh, and Ng Chong Guan. "Sudden Cognitive Decline in Bipolar Mood Disorder Patient with Underlying Severe Tardive Dyskinesia After the Failure of His Deep Brain Stimulation Device Battery." Malaysian Journal of Science Health & Technology 7, no. 3 (2021): 79–81. http://dx.doi.org/10.33102/mjosht.v7i3.163.

Texte intégral

Résumé :

Bipolar disorder (BD) is a chronic mood disorder associated with multiple comorbidities. Structural and functional abnormalities in these subjects’ brains have been related to cognitive deficits correlated with the severity of the mood symptoms. These deficits are also present in euthymic states, indicating a trait characteristic. Bipolar subjects would risk developing tardive dyskinesia due to being exposed to long periods of antipsychotic treatment. Unfortunately, the presence of tardive dyskinesia is also associated with worsening cognition. One treatment option for severe tardive dyskinesia is deep brain stimulation that has also been implicated with cognitive decline. We present a case of Bipolar disorder with underlying cognitive impairment, who had a deep brain stimulation device inserted for severe tardive dyskinesia. He was admitted to a depressive phase and was noted to have a sudden worsening of cognitive impairment following his deep brain stimulation device battery running low. Possible explanations discussed for this unexpected observation are attributed to a further deterioration of his tardive dyskinesia following the device failure and depressive episode, which causes added pseudo-cognitive deficit signs.

Styles APA, Harvard, Vancouver, ISO, etc.

Plus de sources

Nous offrons des réductions sur tous les plans premium pour les auteurs dont les œuvres sont incluses dans des sélections littéraires thématiques. Contactez-nous pour obtenir un code promo unique!