Littérature scientifique sur le sujet « Underlying cognitive deficits »
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Articles de revues sur le sujet "Underlying cognitive deficits"
Stanford, George, et Thomas Oakland. « Cognitive Deficits Underlying Learning Disabilities ». School Psychology International 21, no 3 (août 2000) : 306–21. http://dx.doi.org/10.1177/0143034300213007.
Texte intégralDodd, Barbara, Judi Leahy et Gail Hambly. « Phonological disorders in children : Underlying cognitive deficits ». British Journal of Developmental Psychology 7, no 1 (mars 1989) : 55–71. http://dx.doi.org/10.1111/j.2044-835x.1989.tb00788.x.
Texte intégralLoe, Irene M., Heidi M. Feldman, Enami Yasui et Beatriz Luna. « Oculomotor Performance Identifies Underlying Cognitive Deficits in Attention-Deficit/Hyperactivity Disorder ». Journal of the American Academy of Child & ; Adolescent Psychiatry 48, no 4 (avril 2009) : 431–40. http://dx.doi.org/10.1097/chi.0b013e31819996da.
Texte intégralWilmer, Jeremy B., Alexandra J. Richardson, Yue Chen et John F. Stein. « Two Visual Motion Processing Deficits in Developmental Dyslexia Associated with Different Reading Skills Deficits ». Journal of Cognitive Neuroscience 16, no 4 (mai 2004) : 528–40. http://dx.doi.org/10.1162/089892904323057272.
Texte intégralSagar, H. J. « Specificity of Cognitive Impairment in Neurological Disease : A Methodological Critique of Parkinson’s Disease ». Behavioural Neurology 4, no 2 (1991) : 89–102. http://dx.doi.org/10.1155/1991/625647.
Texte intégralCory-Slechta, Deborah. « Delineating behavioral domains underlying lead-induced cognitive deficits ». Neurotoxicology and Teratology 32, no 4 (juillet 2010) : 497. http://dx.doi.org/10.1016/j.ntt.2010.04.005.
Texte intégralMcPhilemy, Genevieve, Leila Nabulsi, Liam Kilmartin, Denis O’Hora, Stefani O’Donoghue, Giulia Tronchin, Laura Costello et al. « Neuroanatomical Dysconnectivity Underlying Cognitive Deficits in Bipolar Disorder ». Biological Psychiatry : Cognitive Neuroscience and Neuroimaging 5, no 2 (février 2020) : 152–62. http://dx.doi.org/10.1016/j.bpsc.2019.09.004.
Texte intégralKalezic, Nevena, Ivan Dimitrijevic, Ljubica Leposavic, Mladen Kocica, Vesna Bumbasirevic, Cedomir Vucetic, Ivan Paunovic, Nemanja Slavkovic et Jelena Filimonovic. « Postoperative cognitive deficits ». Srpski arhiv za celokupno lekarstvo 134, no 7-8 (2006) : 331–38. http://dx.doi.org/10.2298/sarh0608331k.
Texte intégralNieto, Rodrigo R., Hernán Silva, Alejandra Armijo, Rubén Nachar, Alfonso González, Carmen Paz Castañeda, Cristián Montes et Manuel Kukuljan. « BDNF and Cognitive Function in Chilean Schizophrenic Patients ». International Journal of Molecular Sciences 24, no 13 (24 juin 2023) : 10569. http://dx.doi.org/10.3390/ijms241310569.
Texte intégralZulkifli, Nathratul Ayeshah, JesJeet Singh Gill Jeswant Singh et Ng Chong Guan. « Sudden Cognitive Decline in Bipolar Mood Disorder Patient with Underlying Severe Tardive Dyskinesia After the Failure of His Deep Brain Stimulation Device Battery ». Malaysian Journal of Science Health & ; Technology 7, no 3 (1 septembre 2021) : 79–81. http://dx.doi.org/10.33102/mjosht.v7i3.163.
Texte intégralThèses sur le sujet "Underlying cognitive deficits"
Bull, Rebecca. « Cognitive deficits underlying children's mathematical difficulties ». Thesis, University of St Andrews, 1998. http://hdl.handle.net/10023/15456.
Texte intégralWaters, Flavie. « Cognitive dysfunction underlying auditory hallucinations in schizophrenia : a combined-deficits model ». University of Western Australia. School of Psychiatry and Clinical Neurosciences, 2005. http://theses.library.uwa.edu.au/adt-WU2005.0047.
Texte intégralWong, Hon-kwan, et 黃漢鈞. « Cognitive factors underlying pragmatic deficits in children with autism spectrum disorder ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/209676.
Texte intégralpublished_or_final_version
Educational Psychology
Master
Master of Social Sciences
Knapman, Alana. « Cognitive Deficits and their Underlying Structural and Functional Alterations in Mice Selectively Bred for High Stress Reactivity ». Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-125411.
Texte intégralMorgan, Amanda. « Developmental Dyslexia and/or co-occurring Attention Deficit : Investigation of prevalence, underlying cognitive deficits, and family risk in a self-selected sample of parents and children ». Thesis, Griffith University, 2017. http://hdl.handle.net/10072/376518.
Texte intégralThesis (Professional Doctorate)
Doctor of Philosophy in Clinical Psychology (PhD ClinPsych)
School of Applied Psychology
Griffith Health
Full Text
Harrar-Eskinazi, Karine. « Dyslexie développementale et méthodes de remédiation : Conception et évaluation d'un programme d'intervention multimodale et multi-componentielle fondé sur les approches phonologique, visuo-attentionnelle et intermodalitaire ». Electronic Thesis or Diss., Université Côte d'Azur, 2023. http://www.theses.fr/2023COAZ2025.
Texte intégralDyslexia is a neurodevelopmental disorder that affects children's ability to read and spell words accurately and effectively. Most remediation studies are based on the hypothesis that the core deficit in dyslexic children is based on a single underlying cognitive deficit. However, a large number of studies suggest that most dyslexic children exhibit multiple underlying cognitive deficits that are simultaneously present to varying degrees, leading to a wide range of clinical disorders in reading and spelling. Thus, etiological multifactoriality in developmental dyslexia leads to semiological heterogeneity which explains clinical variability and leads to complex diagnosis, which we called nosographic variability. In line with multi-deficit models of developmental dyslexia, we designed and evaluated a remediation study with a multimodal and multi-componential protocol, which aimed at enhancing both underlying cognitive processes (audio-phonological, visual-attentional, and crossmodal) and reading and spelling procedures, using several training programs and taking into account the child's semiological profile.We assessed benefits of the protocol through a multicenter, longitudinal, randomized, crossover and clinical trial including 3 stages that lasted for a total duration of 16 months. An overall of 94 speech and language therapists and 144 dyslexic readers (aged around 8-13 years) participated in the study.In the first phase, participants were randomly assigned to 2 groups and received weekly speech and language therapy for 2 months without intensive training. In the second phase, in addition to weekly follow-up sessions with the speech therapist, participants received 3 types of intensive computer-based interventions for 2 months each. The first 2 interventions focused on audio-phonological and visual-attentional processes (the order of which was counterbalanced between the 2 groups) and were followed by a third intervention that focused on cross-modal integration processes. The construction of the 3 training programs was based on the scientific literature, the expertise of the speech and language therapists, the patient's complaint (shared care decision) and the environmental context. In the third phase, intensive interventions were discontinued and weekly speech therapy consultations were continued for two months. At the end of the remediation protocol, the multimodal and multi-componential intensive intervention lead to significant improvement in reading efficiency (Cohen's d=2.3), reading comprehension (Cohen's d=0.9), and spelling (Cohen's d=0.78), compared to the weekly speech and language therapy (first phase), and regardless of the order of the interventions. Multiple-case analysis revealed that 52 % of participants were reading disorder free.In conclusion, our results show that an intensive intervention based on a multi-componential and multimodal training program produces major benefits in the treatment of developmental dyslexia. These findings are consistent with a curative (rather than a compensatory) approach for remediation and open up a new avenue for developmental dyslexia treatment
Thompson, Hannah Elizabeth. « Deficits of semantic cognition in stroke aphasia : underlying causes and ameliorating factors ». Thesis, University of York, 2012. http://etheses.whiterose.ac.uk/3721/.
Texte intégralCléry-Melin, Galichon Marie-Laure. « Étude des fonctions neurocognitives dans la dépression : caractérisation de déficits motivationnels et cognitifs, évaluation de leur valeur pronostique Why don't you try harder ? An investigation of effort production in major depression Neural mechanisms underlying motivation of mental versus physical effort Psychomotor retardation is a scar of past depressive episodes, revealed by simple cognitive tests Are cognitive deficits in major depressive disorder progressive ? A simple attention test in the acute phase of a major depressive episode is predictive of later functional remission Progress in elucidating biomarkers of antidepressant pharmacological treatment response : a systematic review and meta-analysis of the last 15 years Stability of the diagnosis of seasonal affective disorder in a long-term prospective study ». Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCB218.
Texte intégralHese deficits coexist in the acute phase of a depressive episode and interfere with decision-making and goal-directed behaviors, and the associated feeling of effort. They appear to persist in periods of clinical remission, decreasing the quality of the therapeutic and functional response and lately worsening the prognosis of the disorder. The aim of this work is to identify objectively measurable neurocognitive markers in clinical practice, and to study their association with the prognosis of a depressive episode, in order to better predict remission and potentially to optimize therapeutic prescribing strategies for patients accordingly. The impairment of neurocognitive processes related to reward constitutes a first vulnerability marker for major depressive disorder (MDD): in a study assessing the production of motor effort in order to obtain a reward, depressed patients had a deficit in production of effort, unlike healthy subjects. Such deficit in incentive motivation - a process underpinned by the activation of ventral cortico-striatal circuits in healthy subjects - may constitute a specific dimension of MDD. It participates in the decision-making and action processes impairments and is associated with – and possibly a consequence of- more specifically cognitive deficits. In a study assessing several cognitive functions in a large cohort of depressed patients, the persistence of psychomotor retardation after 6 to 8 weeks of treatment - in patients considered as being in clinical remission - was positively and independently correlated with the number of past depressive episodes, thus constituting a marker of "cumulative" marker of past depressive episodes. Finally, in a literature review on the progressive evolution of cognitive deficits in MDD, we discussed the existence of a “neurotoxic” effect of the lifetime accumulation of depressive episodes on neurocognitive deficits and its consequences on disease prognosis (increased risk of incomplete functional/clinical remission, relapses, evolution towards dementia). One of the main interest in identifying clinical and cognitive markers of vulnerability is to highlight their capacity to predict the course of a depressive episode-or disorder. In a study based on a cohort of more than 500 depressed patients, a measurement of attention (d2 attention test) was able to significantly and independently predict the subsequent course towards complete remission (clinical and functional) and to constitute a trait -marker of depression, easy to use in clinical practice. Other cognitive markers (such as executive functions) have shown high predictive values for therapeutic response, comparable to those provided by imaging or electrophysiology markers, according to the results of a recent meta-analysis, that emphasizes the interest of using them in patient’s follow-up. Finally, in order to better assess the prognosis of depressive disorder, we have shown that Seasonal Affective Disorder (SAD) diagnosis criteria - which nevertheless represents a specific depressive disorder with well-known physiopathology substrates (construction validity) - had a low predictive validity, prompting to consider this disorder as a temporary expression of a mood disorder, rather than a specific disorder. The identification of clinical tools measuring motivational and cognitive deficits in clinical routine and predicting the course of a depressive episode or disorder represents a major challenge in the improvement of personalized therapeutic management and the long-term prognosis in depressed patients
Araújo, Susana. « Cognitive processes underlying reading and naming deficits in dyslexic readers ». Doctoral thesis, 2011. http://hdl.handle.net/10400.1/5580.
Texte intégralOs estudos apresentados nesta tese sugerem que um défice no sistema de processamento fonológico por si só não é suficiente para explicar o padrão de resultados que caracterizam os leitores disléxicos, pelo menos no contexto de uma ortografia intermédia como o português. É sugerido que, nestes leitores, a existência de problemas ao nível do reconhecimento visual possa contribuir adicionalmente para os seus défices de leitura e de nomeação visual. Estes problemas parecem situar-se não apenas ao nível do processamento lexical das representações ortográficas (por exemplo, uma pobre representação ortográfica dos estímulos linguísticos) mas também nas etapas mais precoces de processamento visual, pré-lexical.
Sanchez, Bezanilla Sonia. « Functional deficits after stroke : the key underlying mechanisms and the therapeutic potential of growth hormone ». Thesis, 2020. http://hdl.handle.net/1959.13/1424038.
Texte intégralBackground and aims: Stroke is currently the leading cause of long-term disability worldwide. Despite the efforts to minimise these negative outcomes, few treatments have been approved and have been mainly focused on the acute phase. This has led to a challenging situation where more than 90% of stroke survivors will experience long-lasting impairments in motor function and/or cognition. Therefore, clinical and pre-clinical research should focus on improving the understanding of underlying mechanisms contributing to neurological damage and functional impairment in later stages after stroke. One of the possible mechanisms that could explain these impairments is the development of secondary neurodegeneration (SND) in remote brain areas synaptically connected to the primary infarct site, such as the thalamus and hippocampus. This process is especially interesting because it occurs over a timescale of weeks to years after the primary infarct, providing a very interesting therapeutic target for chronic stroke treatment. This thesis focused on investigating the evolution of the functional outcomes after stroke and the mechanisms associated with these deficits (Chapter 3 and 4). Additionally, I was interested in studying a potential therapeutic intervention such as growth hormone (GH) to promote brain plasticity and alleviate motor and cognitive impairment (Chapter 5 and 6). Methods: The photothrombotic stroke model targeting the motor and sensory cortex was used to induce ischemia in mice. A touchscreen platform was used to analyse cognitive performance, and the grid walk and cylinder test were used to assess motor function. I also analysed the changes occurring after stroke in different brain areas (peri-infarct, thalamus and hippocampus) combining various molecular biology techniques such as Western Blot, immunohistochemistry, immunofluorescence and various histological stainings. In chapters 5 and 6, I used recombinant human GH (rhGH) delivered subcutaneously via a mini-osmotic pump for 28 days. Results: Stroke significantly impairs multiple cognitive domains and these deficits last for months after the primary infarction. Motor impairments were also long-lasting but a modest spontaneous recovery was observed over time. These deficits were associated with SND processes such as neuronal death, neuroinflammation (astrogliosis and microglia activation) and accumulation of neurotoxic proteins (amyloid-β and α-synuclein) in the thalamus and hippocampus (Chapters 3 and 4). Additionally, motor and cognitive impairment could be reversed by using GH as a therapeutic intervention. GH enhanced functional outcomes through a series of neurorestorative mechanisms including neurogenesis, synaptic plasticity and cerebrovascular remodelling (Chapters 5 and 6). Conclusion: Overall, in this thesis, I have deepened our understanding regarding cognitive and motor impairment after stroke and the mechanisms associated with these impairments. These important findings provide potential targets for translational medical research in the future. Finally, I have demonstrated that GH is an effective treatment following experimental stroke to promote brain plasticity and functional performance in the recovery phase after stroke. My results are encouraging and support the idea that GH represents a promising therapeutic intervention, which should be considered for clinical studies.
Livres sur le sujet "Underlying cognitive deficits"
Leeman-Markowski, Beth A., et Kimford J. Meador. Cognitive Enhancement in Epilepsy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0007.
Texte intégralWilson, Maximiliano A., Bernadette Ska et Yves Joanette. Discourse and Social Cognition Disorders Affecting Communication Abilities. Sous la direction de Anastasia M. Raymer et Leslie J. Gonzalez Rothi. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199772391.013.14.
Texte intégralMaren, Stephen. Neural Circuits for Context Processing in Aversive Learning and Memory. Sous la direction de Israel Liberzon et Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0005.
Texte intégralFrost, Randy O., et Jessica L. Rasmussen. Phenomenology and Characteristics of Compulsive Hoarding. Sous la direction de Gail Steketee. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195376210.013.0020.
Texte intégralManohar, Sanjay, Valerie Bonnelle et Masud Husain. Neurological Disorders of Attention. Sous la direction de Anna C. (Kia) Nobre et Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.027.
Texte intégralYoris, Adrián, Adolfo M. García, Paula Celeste Salamone, Lucas Sedeño, Indira García-Cordero et Agustín Ibáñez. Cardiac interoception in neurological conditions and its relevance for dimensional approaches. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0010.
Texte intégralNaninck, E. F. G., P. J. Lucassen et Aniko Korosi. Consequences of Early-Life Experiences on Cognition and Emotion. Sous la direction de Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.003.
Texte intégralO’Neill, Sarah, Jeffrey M. Halperin et David Coghill. Neuropsychological functioning and ADHD. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0012.
Texte intégralMcCauley, Robert N., et George Graham. Hearing Voices and Other Matters of the Mind. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190091149.001.0001.
Texte intégralCummings, Jeffrey L., et Jagan A. Pillai. Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0001.
Texte intégralChapitres de livres sur le sujet "Underlying cognitive deficits"
Kucinski, Aaron, et Martin Sarter. « Cortico-Striatal, Cognitive-Motor Interactions Underlying Complex Movement Control Deficits ». Dans Innovations in Cognitive Neuroscience, 117–34. Cham : Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42743-0_6.
Texte intégralCalabria, Marco. « Chapter 4. Bilingualism and language control ». Dans Bilingual Processing and Acquisition, 130–58. Amsterdam : John Benjamins Publishing Company, 2023. http://dx.doi.org/10.1075/bpa.17.04cal.
Texte intégralCho, Yoon H., et Yannick Jeantet. « Altered Neural Synchronies Underlying Cognitive Deficits in a Transgenic Mouse Model of Huntington’s Disease ». Dans The Neurobiological Basis of Memory, 321–35. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15759-7_14.
Texte intégralBaune, Bernhard T. « Interventions for social cognitive deficits ». Dans Cognitive Dimensions of Major Depressive Disorder, 83–88. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198835554.003.0010.
Texte intégralHalder, Susmita, et Akash Mahato. « Cognitive Remediation Therapy in Chronic Schizophrenia ». Dans Advances in Psychology, Mental Health, and Behavioral Studies, 292–307. IGI Global, 2017. http://dx.doi.org/10.4018/978-1-5225-0519-8.ch016.
Texte intégralHalder, Susmita, et Akash Mahato. « Cognitive Remediation Therapy in Chronic Schizophrenia ». Dans Research Anthology on Rehabilitation Practices and Therapy, 1337–53. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-3432-8.ch067.
Texte intégralMeghana, S., et Sakshi Shishir. « Social Cognitive Rehabilitation for Neurodegenerative Disorders ». Dans Principles and Clinical Interventions in Social Cognition, 269–87. IGI Global, 2024. http://dx.doi.org/10.4018/979-8-3693-1265-0.ch016.
Texte intégralLee, Chuly, et Richard Brown. « Use of advance information in Parkinson’s disease ». Dans Neuropsychological Disorders Associated with Subcortical Lesions, 190–203. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780198546771.003.0008.
Texte intégralHumphreys, Glyn W., et M. Jane Riddoch. « Fractionating the Intentional Control of Behaviour : A Neuropsychological Analysis ». Dans Agency and Self-Awareness, 201–17. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780199245611.003.0009.
Texte intégralWasserman, Victor, Sheina Emrani, Emily Matusz, Catherine C. Price, Melissa Lamar, Rodney A. Swenson, Kenneth M. Heilman et David J. Libon. « Vascular Cognitive Impairment ». Dans Vascular Disease, Alzheimer's Disease, and Mild Cognitive Impairment, 83–101. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190634230.003.0005.
Texte intégralActes de conférences sur le sujet "Underlying cognitive deficits"
Pinheiro, Mariana Maciel, Victor Albuquerque, Pedro Albuquerque, Eduardo Maranhão, Jonathan Diniz et Breno Barbosa. « CORTICOBASAL SYNDROME DUE TO ALZHEIMER’S DISEASE ». Dans XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda055.
Texte intégralDahmen-Zimmer, Katharina, et Alf C. Zimmer. « Maneuvering in Intersections – What Is the Specific Challenge for Elderly Drivers?Underlying Causes for Violations and a Design for an Assistive System ». Dans Applied Human Factors and Ergonomics Conference (2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1001232.
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