Littérature scientifique sur le sujet « Trauma hemorrhagic shock »
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Articles de revues sur le sujet "Trauma hemorrhagic shock"
Horton, J. W. « Cardiac contractile effects of ethanolism and hemorrhagic shock ». American Journal of Physiology-Heart and Circulatory Physiology 262, no 4 (1 avril 1992) : H1096—H1103. http://dx.doi.org/10.1152/ajpheart.1992.262.4.h1096.
Texte intégralD'Alessandro, Angelo, Hunter B. Moore, Ernest E. Moore, Matthew Wither, Travis Nemkov, Eduardo Gonzalez, Anne Slaughter et al. « Early hemorrhage triggers metabolic responses that build up during prolonged shock ». American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 308, no 12 (15 juin 2015) : R1034—R1044. http://dx.doi.org/10.1152/ajpregu.00030.2015.
Texte intégralGODINHO, MAURICIO, PEDRO PADIM, PAULO ROBERTO B. EVORA et SANDRO SCARPELINI. « Curbing Inflammation in hemorrhagic trauma : a review ». Revista do Colégio Brasileiro de Cirurgiões 42, no 4 (août 2015) : 273–78. http://dx.doi.org/10.1590/0100-69912015004013.
Texte intégralNapolitano, Lena M. « Resuscitation following trauma and hemorrhagic shock ». Critical Care Medicine 23, no 5 (mai 1995) : 795–97. http://dx.doi.org/10.1097/00003246-199505000-00001.
Texte intégralLaserna, Anna Karen Carrasco, Yiyang Lai, Guihua Fang, Rajaseger Ganapathy, Mohamed Shirhan Bin Mohamed Atan, Jia Lu, Jian Wu, Mahesh Uttamchandani, Shabbir M. Moochhala et Sam Fong Yau Li. « Metabolic Profiling of a Porcine Combat Trauma-Injury Model Using NMR and Multi-Mode LC-MS Metabolomics—A Preliminary Study ». Metabolites 10, no 9 (16 septembre 2020) : 373. http://dx.doi.org/10.3390/metabo10090373.
Texte intégralLiu, Fu-Chao, Chih-Wen Zheng et Huang-Ping Yu. « Maraviroc-Mediated Lung Protection following Trauma-Hemorrhagic Shock ». BioMed Research International 2016 (2016) : 1–8. http://dx.doi.org/10.1155/2016/5302069.
Texte intégralChu, Xiaogang, Kumar Subramani, Marie Warren et Raghavan Pillai Raju. « Innate immune response in acute lung injury following hemorrhagic shock ». Journal of Immunology 198, no 1_Supplement (1 mai 2017) : 70.9. http://dx.doi.org/10.4049/jimmunol.198.supp.70.9.
Texte intégralSchneider, Christian P., Martin G. Schwacha, T. S. Anantha Samy, Kirby I. Bland et Irshad H. Chaudry. « Androgen-mediated modulation of macrophage function after trauma-hemorrhage : central role of 5α-dihydrotestosterone ». Journal of Applied Physiology 95, no 1 (juillet 2003) : 104–12. http://dx.doi.org/10.1152/japplphysiol.00182.2003.
Texte intégralGauss, Tobias, Justin E. Richards, Costanza Tortù, François-Xavier Ageron, Sophie Hamada, Julie Josse, François Husson et al. « Association of Early Norepinephrine Administration With 24-Hour Mortality Among Patients With Blunt Trauma and Hemorrhagic Shock ». JAMA Network Open 5, no 10 (7 octobre 2022) : e2234258. http://dx.doi.org/10.1001/jamanetworkopen.2022.34258.
Texte intégralRushing, G. D., R. C. Britt, J. N. Collins, F. J. Cole, L. J. Weireter et L. D. Britt. « Adrenal Insufficiency in Hemorrhagic Shock ». American Surgeon 72, no 6 (juin 2006) : 552–54. http://dx.doi.org/10.1177/000313480607200619.
Texte intégralThèses sur le sujet "Trauma hemorrhagic shock"
Talving, Peep. « Aspects of hepatoduodenal trauma and fluid therapy in hemorrhagic shock / ». Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-740-5/.
Texte intégralAlexander, Geoffrey C. « The Effect of Adding Drag-Reducing Polymers to Resuscitation Fluid During Hemorrhagic Shock on Skeletal Muscle Microcirculation ». VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1522.
Texte intégralLabruto, Fausto. « Modifications of cardiovascular response to ischemia and trauma / ». Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-379-5/.
Texte intégralJi, Soo-Yeon. « COMPUTER-AIDED TRAUMA DECISION MAKING USING MACHINE LEARNING AND SIGNAL PROCESSING ». VCU Scholars Compass, 2008. http://scholarscompass.vcu.edu/etd/1628.
Texte intégralLundin, Sandra, et Jonas Molin. « Räddar liv eller slösar tid ? : prehospital vätskebehandlings effekter på patienter i hemorragisk chock ». Thesis, Sophiahemmet Högskola, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:shh:diva-3742.
Texte intégralTrauma is the leading cause of death in Sweden for people between the age of 15 and 44 years and a large proportion of people die because of bleeding that occurs at the time of the injury. Bleeding also continues to be the leading cause of trauma-related death that could have been preventable both in a civilian and military setting. Trauma care is complex, often time-critical, and the ambulance nurses are often the first to care for these patients on the scene and therefore the first assessment and care of these patients is of great importance. Fluid resuscitation for critically injured trauma patients in hemorrhagic shock or threatening hemorrhagic shock is a debated topic and Sweden lacks national guidelines for trauma care and treatment prehospital. The regional guidelines sometimes for some manner differ between the counties in Sweden. The aim of this study was to determine the impact of fluid resuscitation for patients with hemorrhagic shock after trauma. As a method, a literature review was carried out, which included a total of 15 studies published between 2009 and 2019. The result revealed two main themes - the impact of fluid resuscitation on mortality and the impact of fluid resuscitation on coagulation. All four studies that examined how coagulation ability is affected by crystalloid hypertonic and/or isotonic fluid resuscitation in patients at risk of hemorrhagic shock after trauma, the severity seems to be dependent on the amount of fluid infused, the more fluid the more severe coagulation abnormalities. The result showed unclear evidence of the effect of fluid resuscitation in mortality for trauma patients in hemorrhagic shock. However, none of the studies showed it decreased in mortality. In conclusion, the majority of articles show that large amount of fluid given in prehospital care have no impact or did have a negative impact on survival of critically injured trauma patients in hemorrhagic shock. Many factors come into play and it is difficult to draw any conclusions based on the results and more research are needed.
Dufour-Gaume, Frédérique. « Enjeux, préparation et évaluation de produits sanguins labiles innovants adaptés aux blessés de guerre ». Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASQ076.
Texte intégralWar casualties associate multiple injuries with shock hemorrhage. Despite the therapeutic progress of recent years, hemorrhage is the leading cause of preventable deaths and secondary multiple organ failure can lead to vital and functional prognosis. Management of war-injured patients based on damage control resuscitation and massive transfusion of whole blood reduced considerably the number of deaths. Nevertheless, during foreign operations whole blood is sometimes lacking because of logistic limitations and massive casualties. Modified blood products that are free from constraints could help war-injured patients to survive. To achieve this objective, we developed two French hyper-concentrated lyophilized plasmas with (FLYP-H/LP) or not (FLYP-H) lyophilized platelets. The production of these products is of a high quality. FLYP-H and FLYP-H/LP are high protein products, especially albumin, which confer them a hyperosmolarity twice that of plasma. In FLYP-H/LP, platelets were lysed during the manufacturing process and liberated high quantities of coagulation factors, such as fibrinogen. The therapeutic effects of FLYP-H and FLYP-H/LP were evaluated thanks to our war-injured porcin model. Statistical analysis highlighted the beneficial effects of FLYP-H and FLYP-H/LP on cardiovascular function and hemostasis. These results open the door to more analysis but on human FLYP-H and FLYP-H/LP
Prunet, Bertrand. « Contusion pulmonaire : aspects physiopathologiques et conséquences thérapeutiques ». Thesis, Aix-Marseille, 2015. http://www.theses.fr/2015AIXM5001.
Texte intégralPulmonary contusion is often associated with hemorrhagic shock, constituting a challenge in trauma care. For patients who have sustained lung contusions, fluid resuscitation should be carefully performed, because injured lungs are particularly vulnerable to massive fluid infusions with an increased risk of pulmonary edema and compliance impairment. Fluid administration should be included in an optimized and goal directed resuscitation, based on blood pressure objectives and hemodynamical monitoring. The use of fluids with high volume-expanding capacities (hypertonic saline, colloids) is probably interesting, as well as early introduction of vasopressors. Hemodynamic monitoring will allow to conduct resuscitation on blood pressure objectives, on preload parameters and on extravascular lung water measurement.Our work, based on experimental and clinical studies, objective to characterize the current modalities of ventilatory and hemodynamical aspect of pulmonary contusion care
Morrison, C. Anne Horwitz Irwin Hwang Lu-Yu. « Hypotensive resuscitation versus standard fluid resuscitation for the management of trauma patients in hemorrhagic shock : the safety phase of a randomized controlled trial ». 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1465583.
Texte intégralFraga, Caroline Anne Pires. « The use of Tranexamic acid in trauma patients with hemorrhagic shock - a retrospective study in a tertiary care hospital ». Master's thesis, 2020. https://hdl.handle.net/10216/128787.
Texte intégralIntroduction - Hemorrhagic shock is the major complication and cause of morbi-mortality in trauma patients. We aimed to study the impact of tranexamic acid in trauma patients with hemorrhagic shock in a tertiary hospital. Material and methods - Our study, performed at Centro Hospitalar e Universitário São João, included adult trauma patients with hemorrhagic shock admitted to the Intensive Care Department. We defined hemorrhagic shock as Systolic Blood Pressure ≤90 mmHg and/or Heat Rate ≥110 bpm. In the absence of these, the clinical judgment of the attending physician prevailed. We did patients cohort with and without tranexamic acid use: we assessed global mortality, invasive mechanical ventilation duration, Intensive Care Unit and hospital length of stay, red blood cells units transfused and vascular occlusive events. Results - 105 patients were included from the 1045 admitted for trauma between January 2017 and December 2018: of those, 38 received tranexamic acid (36,2%) versus 67 without it (63,8%). Tranexamic acid group registered a higher severity (APACHE II p=0,038) and mortality in the Intensive Care Unit either in the global population (p=0,003) or in the stratified Traumatic Brain Injury subgroup (p=0,037). We found no statistically significant differences in the secondary outcomes. Discussion - Trauma patients with hemorrhagic shock admitted in the Intensive Care Department and treated with tranexamic acid had higher mortality, probably explained by the higher severity observed. We didnt find increased vascular occlusive events in tranexamic acid group. Conclusions - It is necessary to complement these findings with more studies and larger cohorts to assess the real benefits of tranexamic acid use in these patients.
Fraga, Caroline Anne Pires. « The use of Tranexamic acid in trauma patients with hemorrhagic shock - a retrospective study in a tertiary care hospital ». Dissertação, 2020. https://hdl.handle.net/10216/128787.
Texte intégralIntroduction - Hemorrhagic shock is the major complication and cause of morbi-mortality in trauma patients. We aimed to study the impact of tranexamic acid in trauma patients with hemorrhagic shock in a tertiary hospital. Material and methods - Our study, performed at Centro Hospitalar e Universitário São João, included adult trauma patients with hemorrhagic shock admitted to the Intensive Care Department. We defined hemorrhagic shock as Systolic Blood Pressure ≤90 mmHg and/or Heat Rate ≥110 bpm. In the absence of these, the clinical judgment of the attending physician prevailed. We did patients cohort with and without tranexamic acid use: we assessed global mortality, invasive mechanical ventilation duration, Intensive Care Unit and hospital length of stay, red blood cells units transfused and vascular occlusive events. Results - 105 patients were included from the 1045 admitted for trauma between January 2017 and December 2018: of those, 38 received tranexamic acid (36,2%) versus 67 without it (63,8%). Tranexamic acid group registered a higher severity (APACHE II p=0,038) and mortality in the Intensive Care Unit either in the global population (p=0,003) or in the stratified Traumatic Brain Injury subgroup (p=0,037). We found no statistically significant differences in the secondary outcomes. Discussion - Trauma patients with hemorrhagic shock admitted in the Intensive Care Department and treated with tranexamic acid had higher mortality, probably explained by the higher severity observed. We didnt find increased vascular occlusive events in tranexamic acid group. Conclusions - It is necessary to complement these findings with more studies and larger cohorts to assess the real benefits of tranexamic acid use in these patients.
Livres sur le sujet "Trauma hemorrhagic shock"
J, Sugerman Harvey, et DeMaria Eric J. 1959-, dir. Cytokines in trauma and hemorrhage. New York : Chapman & Hall, 1997.
Trouver le texte intégral1950-, Pope Andrew MacPherson, French Geoffrey, Longnecker David E. 1939- et Institute of Medicine (U.S.). Committee on Fluid Resuscitation for Combat Casualties, dir. Fluid resuscitation : State of the science for treating combat casualties and civilian injuries. Washington, D.C : National Academy Press, 1999.
Trouver le texte intégralSUGERMAN, HARVEY J. Cytokines In Trauma & Hemorrhage. Thomson Publishing Group, 1997.
Trouver le texte intégralSabato, Stefan. Massive Transfusion in a Child. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0024.
Texte intégralMedicine, Institute of, et Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation : State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Trouver le texte intégralMedicine, Institute of, et Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation : State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Trouver le texte intégralMedicine, Institute of, et Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation : State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Trouver le texte intégralMedicine, Institute of, et Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation : State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Trouver le texte intégralWeyker, Paul David, Christopher Allen-John Webb et Tricia E. Brentjens. Hypovolemic Shock. Sous la direction de Matthew D. McEvoy et Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0097.
Texte intégralFinn, Patrick C., et Michael C. Reade. Bleeding Emergencies (DRAFT). Sous la direction de Raghavan Murugan et Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0010.
Texte intégralChapitres de livres sur le sujet "Trauma hemorrhagic shock"
Zhao, Zi-Gang, Yu-Ping Zhang, Li-Min Zhang et Ya-Xiong Guo. « Lymph Formation and Transport : Role in Trauma-Hemorrhagic Shock ». Dans Severe Trauma and Sepsis, 67–95. Singapore : Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-3353-8_5.
Texte intégralLivingston, D. H., J. Hseih, T. Murphy et B. F. Rush. « Hemorrhagic Shock Inhibits Myelopoiesis Independent of Bacterial Translocation ». Dans Host Defense Dysfunction in Trauma, Shock and Sepsis, 339–48. Berlin, Heidelberg : Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_36.
Texte intégralOchoa, J. B., T. R. Billiar et A. B. Peitzman. « The Role of Nitric Oxide in Hemorrhagic Shock and Trauma ». Dans Shock, Sepsis, and Organ Failure — Nitric Oxide, 84–101. Berlin, Heidelberg : Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79343-1_4.
Texte intégralLivingston, D. H., et M. A. Malangoni. « Effect of Therapeutic Gamma-Interferon Administration on Wound Infection After Resuscitated Hemorrhagic Shock ». Dans Immune Consequences of Trauma, Shock, and Sepsis, 551–55. Berlin, Heidelberg : Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7_66.
Texte intégralErtel, W., E. Faist et I. H. Chaudry. « The Biological Characteristics of Cytokines and Their Interactions with Prostaglandins Following Hemorrhagic Shock ». Dans Host Defense Dysfunction in Trauma, Shock and Sepsis, 655–64. Berlin, Heidelberg : Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_83.
Texte intégralTolentino, Rafael, Timothy J. Holleran et Laura S. Johnson. « Futility of Care in Hemorrhagic Shock : When Prolonging the Massive Transfusion Protocol Is of No Benefit ». Dans Difficult Decisions in Trauma Surgery, 35–44. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81667-4_4.
Texte intégralStadlbauer, K. H., H. G. Wagner-Berger, C. Raedler, W. Voelckel, V. Wenzel, A. C. Krismer, K. Rheinberger, K. H. Lindner et A. Koenigsrainer. « Survival with Full Vital Organ Function Recovery after Uncontrolled Hemorrhagic Shock in a Penetrating Liver Trauma Model with Vasopressin in Pigs ». Dans Zurück in die Zukunft, 638. Berlin, Heidelberg : Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55611-1_443.
Texte intégralChaudry, I. H., R. N. Stephan, J. M. Harkema et R. E. Dean. « Immunological Alterations Following Simple Hemorrhage ». Dans Immune Consequences of Trauma, Shock, and Sepsis, 363–73. Berlin, Heidelberg : Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7_47.
Texte intégralChaudry, I. H., A. Ayala, D. Meldrum et W. Ertel. « Hemorrhage-Induced Alterations in Cell-Mediated Immune Function ». Dans Host Defense Dysfunction in Trauma, Shock and Sepsis, 149–59. Berlin, Heidelberg : Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_16.
Texte intégralClemens, M. G. « Hepatic Parenchymal and Nonparenchymal Cells in Hemorrhage and Ischemia ». Dans Host Defense Dysfunction in Trauma, Shock and Sepsis, 127–37. Berlin, Heidelberg : Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_14.
Texte intégralActes de conférences sur le sujet "Trauma hemorrhagic shock"
Rahbar, Elaheh, Bryan A. Cotton, John B. Holcomb et Charles E. Wade. « Reduced Plasma Oncotic Pressure is Indicative of Injury Severity in Trauma Patients ». Dans ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14539.
Texte intégralThau, M. R., T. Liu, N. A. Sathe, G. O'Keefe, C. E. Wade, E. E. Fox, J. B. Holcomb et al. « Latent Class Analysis in a Trauma Cohort with Hemorrhagic Shock Identifies Two Distinct Sub-Phenotypes with a Differential Treatment Response to Blood Transfusion Ratios ». Dans American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5038.
Texte intégralSotudeh-Chafi, M., N. Abolfathi, A. Nick, V. Dirisala, G. Karami et M. Ziejewski. « A Multi-Scale Finite Element Model for Shock Wave-Induced Axonal Brain Injury ». Dans ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192342.
Texte intégralNemeth, Christopher, Adam Amos-Binks, Gregory Rule, Dawn Laufersweiler, Natalie Keeney, Yuliya Pinevich et Vitaly Herasevich. « Real Time Battlefield Casualty Care Decision Support ». Dans 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1002112.
Texte intégralGuzas, Emily L., Stephen E. Turner, Matthew Babina, Brandon Casper, Thomas N. Fetherston et Joseph M. Ambrico. « Validation of a Surrogate Model for Marine Mammal Lung Dynamics Under Underwater Explosive Impulse ». Dans ASME 2019 Verification and Validation Symposium. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/vvs2019-5143.
Texte intégralRapports d'organisations sur le sujet "Trauma hemorrhagic shock"
Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality,Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA : Defense Technical Information Center, décembre 2013. http://dx.doi.org/10.21236/ada612817.
Texte intégralTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA : Defense Technical Information Center, décembre 2012. http://dx.doi.org/10.21236/ada612818.
Texte intégralTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction Using IL-6 as a Resuscitation Adjuvant. Fort Belvoir, VA : Defense Technical Information Center, décembre 2011. http://dx.doi.org/10.21236/ada612819.
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