Littérature scientifique sur le sujet « TLR Genes »
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Articles de revues sur le sujet "TLR Genes"
Bergman, Ingrid-Maria, Amelie Johansson, Caroline Fossum, Leif Andersson et Inger Edfors-Lilja. « Genetic analysis of porcine TLR genes ». Veterinary Immunology and Immunopathology 128, no 1-3 (mars 2009) : 218–19. http://dx.doi.org/10.1016/j.vetimm.2008.10.022.
Texte intégralLiu, Long, Yu-Shan Wei et Dun Wang. « Identification of Core Genes of Toll-like Receptor Pathway from Lymantria dispar and Induced Expression upon Immune Stimulant ». Insects 12, no 9 (14 septembre 2021) : 827. http://dx.doi.org/10.3390/insects12090827.
Texte intégralPryimenko, Nataliia O., Tetiana M. Kotelevska, Tetiana I. Koval, Vadym A. Bodnar, Liudmyla M. Syzova et Stanislav S. Rudenko. « EFFICACY OF SPECIFIC PREVENTION OF INFLUENZA IN INDIVIDUALS WITH POLYMORPHISMS ARG753GLN OF TLR-2, LEU412PHE OF TLR-3, ASP299GLY OF TLR-4 GENES ». Wiadomości Lekarskie 73, no 9 (2020) : 1944–49. http://dx.doi.org/10.36740/wlek202009209.
Texte intégralCui, Jian, Greta J. Frankham, Rebecca N. Johnson, Adam Polkinghorne, Peter Timms, Denis O’Meally, Yuanyuan Cheng et Katherine Belov. « SNP Marker Discovery in Koala TLR Genes ». PLOS ONE 10, no 3 (23 mars 2015) : e0121068. http://dx.doi.org/10.1371/journal.pone.0121068.
Texte intégralRamos Aguila, Luis Carlos, Hafiza Javaira Ashraf, Jessica Paola Sánchez Moreano, Komivi Senyo Akutse, Bamisope Steve Bamisile, Liuyang Lu, Xiaofang Li, Jingyi Lin, Qing Wu et Liande Wang. « Genome-Wide Identification and Characterization of Toll-like Receptors (TLRs) in Diaphorina citri and Their Expression Patterns Induced by the Endophyte Beauveria bassiana ». Journal of Fungi 8, no 8 (22 août 2022) : 888. http://dx.doi.org/10.3390/jof8080888.
Texte intégralFajar, Jonny Karunia. « H1 antihistamines in allergic rhinitis : The molecular pathways of interleukin and toll - like receptor systems ». Journal of Health Sciences 6, no 1 (25 mars 2016) : 1. http://dx.doi.org/10.17532/jhsci.2016.272.
Texte intégralTantia, M. S., Bina Mishra, P. Banerjee, J. Joshi, S. Upasna et R. K. Vijh. « Phylogenetic and sequence analysis of toll like receptor genes (TLR-2 and TLR-4) in buffaloes ». Indian Journal of Animal Sciences 82, no 8 (14 août 2012) : 875–78. http://dx.doi.org/10.56093/ijans.v82i8.23016.
Texte intégralSharbafi, Mohammad Hossein, Sara Assadiasl, Fatemeh Pour‐reza‐gholi, Saeed Barzegari, Peyman Mohammadi Torbati, Shiva Samavat, Mohammad Hossein Nicknam et Aliakbar Amirzargar. « TLR‐2, TLR‐4 and MyD88 genes expression in renal transplant acute and chronic rejections ». International Journal of Immunogenetics 46, no 6 (9 juillet 2019) : 427–36. http://dx.doi.org/10.1111/iji.12446.
Texte intégralShaik-Dasthagirisaheb, Yazdani, Steve Shen, Caroline Genco et Frank Gibson III. « Ageing and expression of TLR pathway associated genes in macrophages to Porphyromonas gingivalis challenge. (55.26) ». Journal of Immunology 188, no 1_Supplement (1 mai 2012) : 55.26. http://dx.doi.org/10.4049/jimmunol.188.supp.55.26.
Texte intégralMelnichuk, Nataliia, Vladimir Kashuba, Svitlana Rybalko et Zenoviy Tkachuk. « Complexes of Oligoribonucleotides with d-Mannitol Modulate the Innate Immune Response to Influenza A Virus H1N1 (A/FM/1/47) In Vivo ». Pharmaceuticals 11, no 3 (22 juillet 2018) : 73. http://dx.doi.org/10.3390/ph11030073.
Texte intégralThèses sur le sujet "TLR Genes"
Wlasiuk, Battagliotti Gabriela. « THE MOLECULAR EVOLUTION OF INNATE IMMUNITY GENES ». Diss., The University of Arizona, 2009. http://hdl.handle.net/10150/195184.
Texte intégralMaughan, Willard. « The Effect of Single Nucleotide Polymorphisms (SNPs) in Toll-Like Receptors -2, -4, -9, and CD14 Genes in an African-American Population with Chronic Periodontitis ». VCU Scholars Compass, 2009. http://scholarscompass.vcu.edu/etd/1844.
Texte intégralFranchim, Camila Sommerauer [UNIFESP]. « Mediadores de inflamação e pré-eclâmpsia : análise de polimorfismos de genes codificadores de IL1-R1, IL-12, IL-18, TLR-2 e TLR-4 ». Universidade Federal de São Paulo (UNIFESP), 2009. http://repositorio.unifesp.br/handle/11600/9805.
Texte intégralObjetivo: avaliar a possível relação entre polimorfismos dos genes codificadores de receptor 1 de interleucina (IL) 1 (IL-1R1) (PstI, rs2234650), IL-12 (+1188, rs3212227), IL-18 (-137, rs187238), IL-18 (-607, rs1946519), receptor tipo Toll (TLR) 2 (TLR-2) (+2258, rs5743708) e TLR-4 (+896, rs4986790) e a pré-eclâmpsia (PE). Pacientes e métodos: Este estudo de caráter caso-controle incluiu 109 pacientes com PE e 174 gestantes sem patologia sistêmica ou obstétrica, e com história de duas ou mais gestações sem intercorrências, como controles. O DNA genômico foi extraído de sangue periférico por método de DTAB/CTAB, e os polimorfismos foram genotipados por técnicas de PCR-RFLP ou PCR-ARMS. Para a análise dos resultados, foram utilizados os testes t de Student e exato de Fischer, tendo sido adotado o nível de significância de p<0,05. Resultados: As freqüências genotípicas do polimorfismo do gene IL-1R1 foram 20,9% CC, 59% CT e 20,1% TT em casos de PE; e 24,7% CC, 56,2% CT e 19,1% TT em controles (p=0,82). As freqüências do polimorfismo do gene IL-12 foram 54,7% AA, 32,9% AC e 12,4% CC em casos de PE; e 55,4% AA, 33,8% AC e 10,8% CC em controles (p=0,93). As freqüências genotípicas de IL-18 (-137) foram 5,2% CC, 42,7% CG e 52,1% GG em casos de PE; e 7,6% CC, 43% CG e 49,4% GG em controles (p=0,74). As freqüências genotípicas de IL-18 (-607) foram 41% CC, 52,7% CA e 6,3% AA em pacientes com PE; e 32,2% CC, 56,2% CA e 11,6% AA no grupo controle (p=0,22). As freqüências do polimorfismo do gene TLR-2 foram 84,6% GG e 15,4% GA em casos de PE; e 84,8% GG e 15,2% GA em controles (p=0,97). As freqüências do polimorfismo do gene TLR-4 foram 93,6% AA e 6,4% AG em casos de PE; e 87,6% AA, 11,7% AG e 0,7% GG em controles (p=0,23). Não houve diferenças significantes entre os grupos, quanto às freqüências genotípicas e alélicas. Conclusões: Não foi observada associação entre polimorfismos de genes codificadores de IL- 1R1, IL-12, IL-18, TLR-2 e TLR-4 e a ocorrência de pré-eclâmpsia.
Problem: Intense maternal inflammatory response is a central event in the pathogenesis of preeclampsia (PE) Our aim was to assess a possible relation between pro-inflammatory mediators: IL-1R1, IL-12, IL-18, IL-18, TLR-2 and TLR-4 gene polymorphisms and PE. Method of Study: This case-control study included 109 patients with PE and 174 healthy women (controls). Genotyping were performed by PCR-ARMS or PCR-RFLP techniques. Data were analyzed by Student´s t or Fischer´s exact tests, and significance was set at p<0.05. Results: Genotypic and allelic distribution for all six polymorphisms was similar between the study and control groups (p=0.82 for IL-1R1, p=0.93 for IL-12, p=0.74 for IL-18 -137, p=0.22 for IL-18 -607, p=0.97 for TLR-2 and p=0.23 for TLR-4 gene polymorphisms). Conclusions: Our findings suggest that the analyzed pro-inflammatory gene polymorphisms are not associated with the occurrence of PE. Further studies have to be done to confirm these results.
TEDE
BV UNIFESP: Teses e dissertações
Morale, Mirian Galliote. « Efeito da infecção por HPV nas vias de sinalização por toll like receptors ». Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-30112016-100636/.
Texte intégralPrevious studies have shown that E6 and E7 HPV oncoproteins are involved in innate immune system dysregulation, causing alterations on Toll-like receptors (TLR) expression. Considering TLR pathway function, it would be advantageous for a virus to manipulate the response of this pathway so it can persist in cells without being detected by the immune system or to modulate this response to create a better environment for persistence of infection. However, many of the mechanisms leading to HPV infection clearance or persistence are still unknown and matter of active investigation. We analyzed in cervical cancer cell lines expression of genes from TLR pathway; several were differentially expressed between tumor cells lines and normal keratinocytes, including TLR adaptors molecules and genes associated with MAP kinase pathway, NFkappaB activation and antiviral immune response. About 90% of these genes were down regulated. Among them, we selected HMGB1 for further characterization due to its interference with tumor cell viability and proliferation. Altogether, our data indicate that high risk HPV E6 and E7 can inhibit TLR signaling pathway
Das, Avishek. « Frequency and distribution of TLR genes in some human populations of North Bengal and their association with rheumatoid arthritis ». Thesis, University of North Bengal, 2018. http://ir.nbu.ac.in/handle/123456789/2827.
Texte intégralBelo, Vanessa de Almeida. « Expressão de genes da resposta imune em bovinos infestados com carrapatos (Boophilus microplus) ». Universidade Federal de Juiz de Fora (UFJF), 2008. https://repositorio.ufjf.br/jspui/handle/ufjf/2841.
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Nos países tropicais, as perdas causadas pela infestação de carrapatos em bovinos acarretam um grande impacto no sistema de produção animal. Recentes estudos têm mostrado a importância de fatores genéticos ligados a resistência a carrapato em Bos taurus indicus e Bos taurus taurus e que as citocinas têm um papel crítico na prevenção ou progressão de doenças. O objetivo desse trabalho foi avaliar os níveis de expressão dos genes IL-10 e IL-4 relacionados ao perfil imunológico Th2 associado à susceptibilidade ao carrapato e os genes IL-2 e IFN- relacionados ao perfil imunológico Th1 associado à resistência ao parasito. Além destes genes, analisou-se o perfil de expressão do gene TLR-2, importante no processo de reconhecimento de patógenos e os genes IL-8 e TNF-α importantes no processo inflamatório inicial. Seis animais mais resistentes e seis animais mais susceptíveis de uma população F2 de 332 animais, originária do cruzamento de animais F1(½ Holandês: ½ Gir), foram selecionados baseado na contagem de carrapatos e valor genético. Amostras de tecido foram coletadas de pele no 5° e 12° dias após a infestação para extração de RNA total. As PCRs em tempo real foram realizadas usando o gene GAPDH como controle endógeno. Os animais resistentes e susceptíveis apresentaram aumento de expressão do gene IL-10 no 5° (p<0,01) e 12 ° dias após a infestação (p<0,05). O gene IL-2, nos animais resistentes e susceptíveis, no 5° dia após a infestação não apresentou alteração da expressão sendo que 12° dia, em ambos os grupos de animais, este gene passou a ser mais expresso em relação ao animal controle sugerindo um perfil de resposta imunológica do tipo de Th2 nos animais resistentes e susceptíveis nos primeiros dias após a infestação. O gene IL-4 apresentou uma tendência ao aumento de expressão nos animais resistentes e susceptíveis em relação ao controle, sendo o perfil Th2 sugerido atribuído a IL-10 produzida por linfócitos T regulatórios (p>0,05). O gene TNF- apresentou aumento de expressão nos animais susceptíveis no 5° dia após a infestação com posterior diminuição no 12° dia após a infestação (p<0,05). Nos animais resistentes não foi observada alteração da expressão deste gene, isto sugere que ele possa estar mais atuante no início do processo inflamatório, logo após a fixação do carrapato. A mesma observação estende-se para o gene IL-8, em que não foi verificada alteração de expressão nos animais resistentes, embora nos animais susceptíveis este gene apresentou diminuição da expressão no 12° dia após a infestação (p<0,05). Quanto ao gene IFN-, não houve diferença de expressão entre os animais resistentes e susceptíveis, sendo que este gene parece não estar relacionado ao mecanismo de resistência. O gene TLR-2 apresentou diminuição da expressão em ambos os grupos de animais. Estes resultados sugerem que a resposta imune adquirida avaliada neste trabalho não apresenta papel preponderante no mecanismo de resistência e que resposta imune inata poderia está envolvida no mecanismo de resistência ao carrapato. Portanto, avaliação da resposta imunológica horas após a fixação do carrapato poderia nos fornecer resultados mais conclusivos.
In tropical countries losses caused by tick infestation in cattle lead to a major impact on animal production systems. Recent studies have shown the importance of genetic factors linked to tick resistance in Bos indicus and Bos taurus as well as the critical role in the prevention or progression of diseases mediated by cytokines. The aim of this work was to evaluate gene expression of IL-10 and IL-4 in relation to tick susceptibility associated with the Th2 profile and gene expression of IL-2 and IFN- in relation to tick resistance associated with the Th1 profile. In addition, the expression of TLR-2, important in the process the recognition of pathogens, and TNF-α and IL-8 genes, important in the initial inflammatory process, were evaluated. Six tick-resistant and six tick-susceptible animals from a F2 population of 332 animals, originated from the cross of F1 animals (½ Holstein: ½ Gir), were selected based on tick count and breeding value for tick resistance. Skin biopsies were collected in the 5th and 12th days after tick infestation. The GAPDH was used as endogenous control to normalize the amount of starting cDNA target in the real-time PCR assay. Both resistant and susceptible animals showed increased gene expression of IL-10 in the 5th and 12th days after infestation in relation to control animal (p<0.05). The IL-2 gene showed no change of expression in the 5th day after infestation for the resistant and susceptible animals. In the 12th post infestation, both resistant and susceptible animals showed increased gene expression in relation to control animal. These results suggest an enhancement of Th2 profile through the increase of IL-10 mRNA levels and a possible inhibition of the Th1 pattern in both groups (resistant and susceptible) starting 5 days after infestation and return to normal by day 12. Despite our results suggest the occurrence of the Th2 profile, the susceptible and resistant animals did not show variation on gene expression for IL-4 in relation to control animal. The susceptible animals showed increased expression of TNF-α in the 5th day after infestation. However, in the 12th day post infestation it was noted a decrease in the gene expression level. The resistant animals showed no change in the expression of this gene in relation to control animals suggesting that TNF-α could be more actively expressed in the early steps of the inflammatory process. Similarly, the resistant animals showed no variation in the expression of IL-8 while the susceptible animals showed increased expression in the 12th day post infestation. There were no differences of expression between resistant and susceptible animals in relation to IFN-γ what suggests that this gene might not be involved in the resistance mechanism. The TLR-2 gene showed decreased expression in both resistant and susceptible animals (p<0.05). Finally, there was no difference in expression between susceptible and resistant animals in relation to all selected genes in the 5th and 12th days after infestation. These results suggest that the acquired immunity evaluated in this work might not have preponderant role in the resistance mechanism. The innate immunity might be playing a major role in the bovine tick resistance/susceptibility mechanism in early hours after infestation.
Brito, Sara Alves Paiva de. « Produção de prostaglandinas pelo endométrio canino ao longo do ciclo éstrico e a sua relação com a transcrição de genes dos toll-like receptors ». Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2011. http://hdl.handle.net/10400.5/3131.
Texte intégralO endométrio canino constitui a primeira linha de defesa contra infecções bacterianas ascendentes, sendo os toll-like receptors (TLRs) elementos chave no reconhecimento de agentes patogénicos por parte do sistema imunitário inato. Um dos objectivos deste trabalho consistiu na avaliação da expressão de mRNA dos TLRs no endométrio da cadela ao longo do ciclo éstrico. Detectou-se a expressão de mRNA dos TLRs 1-7 e 9 por RT-PCR, o que demonstra a sua capacidade de reconhecimento de uma grande variedade de ligandos através da activação via TLRs. Verificou-se ainda que a transcrição dos TLRs 2 e 4 se encontra significativamente diminuída no início de diestro, o que pode justificar a maior susceptibilidade do útero à infecção por E. coli nesta fase. Pelo contrário, no final de diestro, a transcrição de genes destes receptores demonstrou estar bastante aumentada. O outro objectivo consistiu na determinação da produção de PGE2 e de PGF2α por explantes do endométrio de diferentes fases do ciclo éstrico, na ausência e na presença de estímulos como o lipopolissacarídeo (LPS) e o ácido lipoteicóico (LTA). A estimulação com LPS e LTA levou a um aumento da produção de ambas as prostaglandinas em relação ao tecido não estimulado. No entanto, este aumento foi significativamente superior nos explantes de final de diestro, quando comparado com os de fase folicular, diestro e anestro. Estes resultados sugerem, em suma, a capacidade de activação dos TLRs 2 e 4 em modelos de cultura de explantes de endométrio de cadela, resultando na produção de mediadores capazes de regular a resposta inflamatória local.
ABSTRACT - PROSTAGLANDINS PRODUCTION BY CANINE ENDOMETRIUM THROUGH THE OESTRUS CYCLE AND ITS RELATIONSHIP WITH TOLL-LIKE RECEPTORS GENES TRANSCRIPTION - The canine endometrium is the first line of defense against ascending bacterial infections and the toll-like receptors (TLRs) are key elements in pathogen recognition by innate immune system. The first aim of this study was to evaluate the mRNA expression of TLRs in the endometrium of the bitch throughout the oestrus cycle. Gene transcription of TLRs 1-7 and 9 was detected by RT-PCR, which demonstrates its ability to recognize a wide range of ligands through TLR activation. It was also found that gene transcription of TLRs 2 and 4 are downregulated in early diestrus, which might explain the higher susceptibility to uterine infection, induced by Escherichia coli, in this stage. On the other hand, in late diestrus the mRNA expression of these receptors has proven to be greatly increased. The other objective was to assess the production of PGE2 and PGF2α by endometrial explants in different stages of the oestrus cycle, in the absence and presence of stimuli like lipopolysaccharide (LPS) and lipoteichoic acid (LTA). Stimulation with LPS and LTA led to an increased production of both prostaglandins compared with that observed in unstimulated tissues. However, this increase was significantly higher in explants from late diestrus, when compared with those in follicular phase, diestrus and anestrus. These results suggest, briefly, the activation of TLRs 2 and 4 in canine endometrial explants culture models, which result in the mediators production that may further regulate local inflammatory response.
Fornuskova, Alena. « Genes of innate immunity and their significance in evolutionary ecology of free livings rodents ». Phd thesis, Université Montpellier II - Sciences et Techniques du Languedoc, 2013. http://tel.archives-ouvertes.fr/tel-01021258.
Texte intégralБевз, Т., Г. Мартинюк, С. Куляс, О. Попович et Л. Медведєва. « Особливості клінічного перебігу та прогнозу хронічного гепатиту С при поліморфізмі гену TLR4 ». Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/65455.
Texte intégralThe clinical investigation discusses the connection between TLR4-polymorphysm and the liver fibrosis progression degree, HCV viral load among patients with chronic HCV-hepatitis for diagnostics and treatment improvement in such individuals.
Neto, Lídio Gonçalves Lima. « Polimorfismo dos genes CD14, TLR2, TLR4, IL6 e sua associação com o infarto do miocárdio em adultos jovens ». Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-18022014-101606/.
Texte intégralThe objective of this study was to assess the possible association between the polymorphism - 260C/T of the gene CD14, Arg7S3Gln TLR2, Asp299Gli and Thr399lle TLR4 and - 174G/C of the gene IL6 with myocardial infarction in young adults. For that a case control study was conducted and the case group was formed by 102 patients who had myocardial infarction, and the control group by 108 individuals without history of cardiovascular disease. The genotyping was performed by PCR-RFLP. There was no association between the distribution of genotypes of SNPs - 260C/T of the gene CD14, Arg7S3Gln TLR2, Asp299Gli and Thr399lle TLR4 and -174G/C of the gene IL6 between the two groups studied (p<0.05). The serum inflammatory blood profile was related with polymorphism - 174G/C IL-6 in the group IAM. The GG genotype was linked with elevated PAI-1 concentrations (p<0.0001), the genotype GC with larger quantities of red blood cells (p=0.02) and CC genotype with high concentrations of fibrinogênio (p<0.01) and leucocytes increased quantity (p<0.05). The CC genotype of polymorphism of -260C/T CD14 also showed associated with the elevation in PAI-1 concentrations (p<0.0001) and the CT genotype with elevated fibrinogênio concentrations (p<0.01). The GG genotype of the -174G/C IL-6 polymorphism was related with elevated glucose concentrations (p<0.05) and CC genotype with decrease in the apoA (p <0.01) and HDL (p<0001) concentrations and elevation of the LDL (p <0.05), total cholesterol (p <0.05) concentrations. The CT genotype of -260C / T CD14 polymorphism also was Iinked with high total cholesterol concentrations (p <0.0001). In conclusion, the polymorphism is not associated with myocardial infarction in young patients, but the -260CT CD14 and -174 IL-6 SNPs appear to be related to the serum biochemical, hematological and inflammatory blood profile in patients that had myocardial infarction.
Livres sur le sujet "TLR Genes"
Berkel, Klaas van. Spiegelbeeld der wetenschap : Het Genootschap ter Bevordering van Natuur-, Genees- en Heelkunde, 1790-1990. Rotterdam : Erasmus Publishing, 1991.
Trouver le texte intégralBrent, Jonathan Robert. The ALS Genes TDP-43 and FUS/TLS Regulate a Common Pathway in the Nervous System of Drosophila Melanogaster. [New York, N.Y.?] : [publisher not identified], 2012.
Trouver le texte intégralBilański, Piotr. Trypodendron laeve Eggers w Polsce na tle wybranych aspektów morfologicznych i genetycznych drwalników (Trypodendron spp., Coleoptera, Curculionidae, Scolytinae). Publishing House of the University of Agriculture in Krakow, 2019. http://dx.doi.org/10.15576/978-83-66602-38-0.
Texte intégralGeri, Guillaume, et Jean-Paul Mira. Host–pathogen interactions in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0306.
Texte intégralChapitres de livres sur le sujet "TLR Genes"
Pecman, Mojca, et Natalie Kübler. « Chapter 12. Text genres and Terminology ». Dans Theoretical Perspectives on Terminology, 263–90. Amsterdam : John Benjamins Publishing Company, 2022. http://dx.doi.org/10.1075/tlrp.23.12pec.
Texte intégralRhein, Simone, et Neşe Çakmak-Görür. « TCR Gene Therapy for Cancer ». Dans Methods in Molecular Biology, 95–128. New York, NY : Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2441-8_6.
Texte intégralDuarte, Isabel Margarida, et Maria Aldina Marques. « Chapter 11. Vós and other pronominal forms of address ( tu, você, vocês ) ». Dans It's different with you, 272–93. Amsterdam : John Benjamins Publishing Company, 2023. http://dx.doi.org/10.1075/tar.5.11dua.
Texte intégralLefferts, Juliet W., Vera Boersma, Marne C. Hagemeijer, Karima Hajo, Jeffrey M. Beekman et Erik Splinter. « Targeted Locus Amplification and Haplotyping ». Dans Methods in Molecular Biology, 31–48. New York, NY : Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2819-5_2.
Texte intégralKutikhin, Anton G., et Arseniy E. Yuzhalin. « Structural Genomic Variation in TLR4 Gene and Cancer ». Dans Genomics of Pattern Recognition Receptors, 33–55. Basel : Springer Basel, 2013. http://dx.doi.org/10.1007/978-3-0348-0688-6_3.
Texte intégralLarionov, Vladimir. « Direct Isolation of Specific Chromosomal Regions and Entire Genes by TAR Cloning ». Dans Genetic Engineering, 37–55. Boston, MA : Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4707-5_3.
Texte intégralGaynor, Richard. « Role of the TAR Element in Regulating HIV Gene Expression ». Dans Advances in Molecular Biology and Targeted Treatment for AIDS, 79–91. Boston, MA : Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-5928-9_8.
Texte intégralHens, Kristien. « 7. Een procesontologie voor de bio-ethiek ». Dans Toevallige ontmoetingen, 89–100. Cambridge, UK : Open Book Publishers, 2023. http://dx.doi.org/10.11647/obp.0370.07.
Texte intégralSapkota, Sunil, et Michael P. Gantier. « Selecting Therapeutic Antisense Oligonucleotides with Gene Targeting and TLR8 Potentiating Bifunctionality ». Dans Methods in Molecular Biology, 225–34. New York, NY : Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3331-1_17.
Texte intégralZorina, Anna A., Galina V. Novikova et Dmitry A. Los. « Participation of Ser-Thr Protein Kinases in Regulation of Heat Stress Responses inSynechocystis ». Dans Stress and Environmental Regulation of Gene Expression and Adaptation in Bacteria, 766–80. Hoboken, NJ, USA : John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781119004813.ch74.
Texte intégralActes de conférences sur le sujet "TLR Genes"
Liu-Mares, Wen, Jennifer J. Hu, Ioanna Konidari, Glenn O. Allen, Peter E. Clark, M. Craig Hall, Chuanhui Dong, J. Sunil Rao et William K. Scott. « Abstract 2771 : Genetic variation in interleukin, TLR, TNF and TRAF genes of NF-κB activation pathway and risk of prostate cancer ». Dans Proceedings : AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011 ; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2771.
Texte intégralPaniagua, N. Díaz, EG Tranquilino Batres, AP Lόpez Flores, A. Lozano Cardenas, E. Vallarino Reyes, AL Alvarez Grijalva, L. Sanchez Chapul, C. Díaz Hernández, L. Ríos Ventura et A. Lopez Macay. « AB0861 Expression control by methylation of the TLR1, TLR2, TLR4, IL1B, ALPK1 SLC2A9 and SLC22A12 genes in monocytes of patients with gout ». Dans Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.3594.
Texte intégralSouza, Sabrina Pires Maciel de, JULIANA ECHEVARRIA LIMA et RENATA MEIRELLES PEREIRA. « LINFÓCITOS INFECTADOS PELO HTLV-1 INDUZEM A DIFERENCIAÇÃO DE MONÓCITOS EM MACRÓFAGOS \"M1-LIKE\" IN VITRO ». Dans II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/5788.
Texte intégralELIZA DE JESUS BARROS DOS, SANTOS, LIMA ALINE MEDEIROS, SANTOS ADRYANA TRAVASSOS DOS et PAIVA RAFAEL DA SILVA. « A IDENTIFICAÇÃO E CARACTERIZAÇÃO DA FAMILIA DE GENE 1,3-BETAGLUCOSIDASE DE MANIHOT ESCULENTA CRANTZ ». Dans III Congresso Brasileiro de Ciências Biológicas On-line. Revista Multidisciplinar de Educação e Meio Ambiente, 2022. http://dx.doi.org/10.51189/iii-conbracib/6420.
Texte intégralLiu, Tao, Qiu-Ling Xu, Zhao-Xin Yang et Yan Zhao. « Emodin Protects NAFLD Rats via Regulating the Genes Expression of Hepatic TLR4 Genes ». Dans 2015 International Conference on Medicine and Biopharmaceutical. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789814719810_0069.
Texte intégralStuchi, Amanda Maria, Leonardo Paroche de Matos, Allan Luis Barboza Atum et José Antônio Silva Júnior. « EXPOSIÇÃO PRÉ-NATAL AO ÁLCOOL E A EXPRESSÃO DE GENES RELACIONADOS À HIPERTROFIA CARDÍACA EM CAMUNDONGOS ». Dans Congresso Médico Acadêmico da Universidade Nove de Julho. Universidade Nove de Julho, 2022. http://dx.doi.org/10.5585/comamedvg.2022.9.
Texte intégralAlmeida, Jamile Aislin Silva de, et Carla Yasmym De Carvalho Lima. « NUTRIGENÔMICA E AS DOENÇAS CRÔNICAS NÃO TRANSMISSÍVEIS : UMA REVISÃO BIBLIOGRÁFICA ENTRE OS POLIFORMISMOS E COMPOSTOS BIOATIVOS ». Dans II Congresso Brasileiro de Biologia Molecular On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2323.
Texte intégralGinting, Ronistra, Mohammad Basyuni et Yuntha Bimantara. « Bioinformatics Identification of TLR4 Genes in Brahma Chicken (Gallus gallus domesticus) ». Dans 2019 3rd International Conference on Electrical, Telecommunication and Computer Engineering (ELTICOM). IEEE, 2019. http://dx.doi.org/10.1109/elticom47379.2019.8943920.
Texte intégralPannekok, H., A. J. Van Zonneveid, C. J. M. de vries, M. E. MacDonald, H. Veerman et F. Blasi. « FUNCTIONAL PROPERTIES OF DELETION-MUTANTS OF TISSUE-TYPE PLASMINOGEN ACTIVATOR ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643724.
Texte intégralCarvalho, Gabriel Leal, Isadora Ghilardi, Allan Alcará, Felipe Rodrigues, Ângela Zanatta, Giovani Zocche, Giulia Pinzetta et al. « Gene expression of calcium channel CACNA1H in epileptogenesis can be modulated by mesenchymal stem cells ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.593.
Texte intégralRapports d'organisations sur le sujet "TLR Genes"
Fluhr, Robert, et Maor Bar-Peled. Novel Lectin Controls Wound-responses in Arabidopsis. United States Department of Agriculture, janvier 2012. http://dx.doi.org/10.32747/2012.7697123.bard.
Texte intégralLiang, Yuting, Liyou Wu, Ian Clark, Kai Xue, Joy D. Van Nostrand, Ye Deng, Zhili He, Penny Hirsch, Steve Mcgrath et Jizhong Zhou. Taxa-area Relationship (TAR) of Microbial Functional Genes with Long-TGerm Fertilization. Office of Scientific and Technical Information (OSTI), mai 2010. http://dx.doi.org/10.2172/985942.
Texte intégralDickman, Martin B., et Oded Yarden. Role of Phosphorylation in Fungal Spore Germination. United States Department of Agriculture, août 1993. http://dx.doi.org/10.32747/1993.7568761.bard.
Texte intégralDavid, Lior, Yaniv Palti, Moshe Kotler, Gideon Hulata et Eric M. Hallerman. Genetic Basis of Cyprinid Herpes Virus-3 Resistance in Common Carp. United States Department of Agriculture, janvier 2011. http://dx.doi.org/10.32747/2011.7592645.bard.
Texte intégralWeiss, David, et Neil Olszewski. Manipulation of GA Levels and GA Signal Transduction in Anthers to Generate Male Sterility. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7580678.bard.
Texte intégralSplitter, Gary, et Menachem Banai. Microarray Analysis of Brucella melitensis Pathogenesis. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7709884.bard.
Texte intégralDickman, Martin B., et Oded Yarden. Involvement of the PKA and MAPK signal transduction pathways in sclerotial morphogenesis in Sclerotinia sclerotiorum. United States Department of Agriculture, septembre 2007. http://dx.doi.org/10.32747/2007.7695861.bard.
Texte intégralDickman, Martin B., et Oded Yarden. Regulation of Early Events in Hyphal Elongation, Branching and Differentiation of Filamentous Fungi. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7580674.bard.
Texte intégralGalili, Gad, Harry J. Klee et Asaph Aharoni. Elucidating the impact of enhanced conversion of primary to secondary metabolism on phenylpropanoids secondary metabolites associated with flavor, aroma and health in tomato fruits. United States Department of Agriculture, janvier 2012. http://dx.doi.org/10.32747/2012.7597920.bard.
Texte intégralBarash, Itamar, et Robert Rhoads. Translational Mechanisms Governing Milk Protein Levels and Composition. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7696526.bard.
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