Littérature scientifique sur le sujet « Systemic induced resistance »
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Articles de revues sur le sujet "Systemic induced resistance"
Cohen, Y. « Systemic induced resistance ». Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (1 janvier 2002) : S122—S125. http://dx.doi.org/10.17221/10334-pps.
Texte intégralVallad, Gary E., et Robert M. Goodman. « Systemic Acquired Resistance and Induced Systemic Resistance in Conventional Agriculture ». Crop Science 44, no 6 (novembre 2004) : 1920–34. http://dx.doi.org/10.2135/cropsci2004.1920.
Texte intégralPieterse, Corné M. J., Christos Zamioudis, Roeland L. Berendsen, David M. Weller, Saskia C. M. Van Wees et Peter A. H. M. Bakker. « Induced Systemic Resistance by Beneficial Microbes ». Annual Review of Phytopathology 52, no 1 (4 août 2014) : 347–75. http://dx.doi.org/10.1146/annurev-phyto-082712-102340.
Texte intégralBonello, Gordon et Storer. « Systemic induced resistance in Monterey pine ». Forest Pathology 31, no 2 (28 avril 2001) : 99–106. http://dx.doi.org/10.1046/j.1439-0329.2001.00230.x.
Texte intégralvan Loon, L. C., P. A. H. M. Bakker et C. M. J. Pieterse. « SYSTEMIC RESISTANCE INDUCED BY RHIZOSPHERE BACTERIA ». Annual Review of Phytopathology 36, no 1 (septembre 1998) : 453–83. http://dx.doi.org/10.1146/annurev.phyto.36.1.453.
Texte intégralThomashow, Linda S. « Induced systemic resistance : a delicate balance ». Environmental Microbiology Reports 8, no 5 (octobre 2016) : 560–63. http://dx.doi.org/10.1111/1758-2229.12474.
Texte intégralBakker, Peter A. H. M., Rogier F. Doornbos, Christos Zamioudis, Roeland L. Berendsen et Corne M. J. Pieterse. « Induced Systemic Resistance and the Rhizosphere Microbiome ». Plant Pathology Journal 29, no 2 (1 juin 2013) : 136–43. http://dx.doi.org/10.5423/ppj.si.07.2012.0111.
Texte intégralSchweizer, Patrick, Antony Buchala, Robert Dudler et Jean-Pierre Métraux. « Induced systemic resistance in wounded rice plants ». Plant Journal 14, no 4 (mai 1998) : 475–81. http://dx.doi.org/10.1046/j.1365-313x.1998.00141.x.
Texte intégralSimms, Ellen L., et Todd J. Vision. « Pathogen-induced systemic resistance in Ipomoea purpurea ». Oecologia 102, no 4 (1995) : 494–500. http://dx.doi.org/10.1007/bf00341362.
Texte intégralBakker, Peter A. H. M., Corné M. J. Pieterse et L. C. van Loon. « Induced Systemic Resistance by Fluorescent Pseudomonas spp. » Phytopathology® 97, no 2 (février 2007) : 239–43. http://dx.doi.org/10.1094/phyto-97-2-0239.
Texte intégralThèses sur le sujet "Systemic induced resistance"
Borlinghaus, Maria Theresia. « Implications of biochar on UK barley systems : a biological perspective ». Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/17621.
Texte intégralTripathi, Diwaker. « Role of SABP2 in Systemic Acquired Resistance Induced by Acibenzolar-S-Methyl in Plants ». Digital Commons @ East Tennessee State University, 2010. https://dc.etsu.edu/etd/1720.
Texte intégralLe, Thanh Toan, Van Dien Luong, Thi Thuy Nhien Ngo et Van Kim Pham. « Induced systemic resistance against rice grassy stunt virus – a promising field for ecological rice production ». Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-88491.
Texte intégralHầu hết các phương pháp sản xuất lúa hiện nay đều sử dụng các hóa chất độc hại trong việc phòng trừ bệnh và côn trùng gây hại, nên dẫn đến ô nhiễm môi trường. Kích thích tính kháng lưu dẫn giúp kích hoạt cơ chế tự nhiên kháng bệnh của cây có thể là giải pháp bảo vệ thực vật thay thế an toàn với môi trường. Việc ứng dụng tiến bộ này vào trong sản xuất lúa có thể làm giảm lượng hóa chất sử dụng, đóng góp vào việc giảm thiểu ô nhiễm môi trường và sự phát triển của một nền nông nghiệp bền vững. Nghiên cứu đã được thực hiện tại nhà lưới trường Đại học Cần Thơ để tuyển chọn hóa chất và phương pháp sử dụng hóa chất để tăng cường sức khỏe giúp cây lúa vượt qua bệnh vàng lùn. Hóa chất kích kháng được sử dụng ở một nồng độ rất thấp (đơn vị là mM). Kết quả cho thấy chiều cao cây lúa khi xử lý chất kích kháng tốt hơn so đối chứng không xử lý. Bên cạnh đó, số cây lúa nhiễm bệnh giảm, tỉ lệ hạt chắc và năng suất tăng khi cây lúa được xử lý với chất kích kháng. Trong số các chất kích kháng đã sử dụng, acid oxalic cho hiệu quả vượt trội. Với chất acid oxalic, phương pháp ngâm hạt cho hiệu quả kích kháng tốt hơn phương pháp áo hạt
Conn, Vanessa Michelle, et vanessa conn@acpfg com au. « Molecular Interactions of Endophytic Actinobacteria in Wheat and Arabidopsis ». Flinders University. School of Medicine, 2006. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20060320.171412.
Texte intégralChen, Chunquan. « Induced systemic resistance against Pythium aphanidermatum by plant growth-promoting rhizobacteria on cucumber, Cucumis sativus L ». Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ50129.pdf.
Texte intégralChen, Chunquan 1958. « Induced systemic resistance against Pythium aphanidermatum by plant growth-promoting rhizobacteria on cucumber (Cucumis sativus L.) ». Thesis, McGill University, 1998. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=35862.
Texte intégralChanda, Bidisha. « GLYCEROL-3-PHOSPHATE IS A NOVEL REGULATOR OF BASAL AND INDUCED DEFENSE SIGNALING IN PLANTS ». UKnowledge, 2012. http://uknowledge.uky.edu/plantpath_etds/16.
Texte intégralMuzammil, Saima. « Saccharothrix algeriensis NRRL B-24137 : biocontrol properties, colonization and induced systemic resistance towards Botrytis cinerea on grapevine and Arabidopsis thaliana ». Thesis, Toulouse, INPT, 2012. http://www.theses.fr/2012INPT0063/document.
Texte intégralIn this thesis, the desert soil isolate, Saccharothrix algeriensis NRRL B-24137, has been evaluated for its bioactive properties towards the phytopathogenic fungus Botrytis cinerea, for its colonization of Vitis vinifera L., and Arabidopsis thaliana as well as to study the mechanisms of induced systemic resistance (ISR) towards B. cinerea. The results obtained allowed us firstly to show that Sa. algeriensis NRRL B-24137 can exhibit strong antifungal properties towards B. cinerea and that some metabolites can be responsible of this antifungal activity. Although these metabolites are still under consideration and that this study needs further works, we have demonstrated then the colonization properties of the desert soil isolate with grapevine plants. The results showed that the strain can form rhizospheric as well as endophytic subpopulations with grapevine plants (Cabernet Sauvignon cultivar graffed on 44-53 M rootstock) at early step of colonization. Then we have demonstrated that the beneficial strain could induce a systemic resistance towards B. cinerea. Although the mechanisms are not yet well understood, preliminary parts of this work demonstrated that the genes responsible of glucanase production, chitinase as well as inhibitor of polygalacturonase activity do not seems to be primed during the systemic resistance phenomenon. Finally we demonstrated that the interaction between Sa. algeriensis NRRL B-24137 and Arabidopsis thaliana plants results in a close association due also to a rhizo- and endophytic colonization of the model plant. The beneficial strain can also induce a systemic resistance in A. thaliana towards B. cinerea and analyzes of plant mutants have allowed to determine parts of the mechanisms involved in ISR as well as new mechanisms that could be trigerred by beneficial microbes
Zhang, Weizheng. « Disease suppression and systemic-acquired-resistance-induced in plants by compost-amended potting mixes, compost water extracts and no-tillage soil / ». The Ohio State University, 1997. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487943610784806.
Texte intégralNeves, Christian André Fernandes. « Methylglyoxal-induced glycation changes liver lipid content in high-fat diet-fed rats, causing glucose and lipid systemic dysmetabolism ». Master's thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/14597.
Texte intégralFatty liver disease is simultaneously a cause and a consequence of type 2 diabetes. Hepatic lipid metabolism is altered in obese patients, causing insulin resistance. More, inhibition of insulin signaling may also affect hepatic lipid metabolism, causing a feedback that may lead to hepatic steatosis, common in such patients. In this work, we intended to assess the role of glycation (methylglyoxal-induced) in the hepatic lipid metabolism of high-fat diet-fed rats, using lipidomic approaches and magnetic resonance imaging, which identify hepatic lipid species, including phospholipids (PL), triglycerids (TG), diacylglycerols (DAG) and fatty acids (FA). Wistar rats were maintained during 4 months with methylglyoxal (MG) supplementation (100mg/Kg/day) (MG group), a high-fat diet rich in TG (HFD group) or both (HFDMG group) and compared with controls feeding a standard diet (n=6/ group). Lipidomic approaches, namely liquid chromatography - mass spectrometry (LC-MS) and gas chromatography (GC) were used to determine liver composition in PL, TG and FA. Non-invasive 1H nuclear magnetic resonance (NMR) spectroscopy (9 Tesla) of liver tissues in vivo was used to determine lipid species, such as TG and DAG. The total and phosphorylated levels of the mediators of the insulin receptor pathway and lipid oxidation were determined by western blotting. High-fat diet-fed (HFD) rats showed increased body weight in relation to controls, but this effect was partially inhibited by MG supplementation (HFDMG group). Moreover, HFDMG group showed increased plasma free fatty acid levels, hyperinsulinemia, insulin resistance and glucose intolerance. In liver, lipidomic techniques and 1H NMR showed increased fat mass in the liver of HFD and HFDMG rats. HFD rats, but not HFDMG, showed increased total levels of the 18:1 fatty acid (common in high-fat diets). Despite no differences were observed for HFD group, HFDMG rats showed decreased fraction of unsaturated lipids and increased fraction of saturated lipids. This difference was obtained due to a decrease in monounsaturated FA. Regarding lipid esterification, HFDMG group showed lower percentage of esterified glycerol carbons, suggesting an increased concentration of DAG in relation to TG. In accordance, this group showed higher fatty acids/glycerol ratio, suggesting increased liver non-esterified fatty acid levels. Western Blotting analyses showed decreased activation of insulin pathway, especially HFDMG group, as well as decreased activation of the insulin receptor in HFDMG group. Data suggest that glycation changes lipid metabolism in a context of hyperlipidemia, possibly contributing to hepatic lipotoxicity and to accelerate progression of insulin resistance and fatty liver disease.
O fígado gordo é simultaneamente uma causa e consequência da diabetes mellitus tipo 2. O metabolismo lipidico-hepático (MLH) encontra-se alterado em obesos, causando insulino-resistência. A diminuição da sinalização da via da insulina pode igualmente afetar o MLH, estimulando o desenvolvimento de esteatose hepática, comum nos doentes. Neste trabalho, pretende-se analisar o papel da glicação (induzida por metilglioxal) no MLH em ratos com dieta gorda, através de técnicas de lipidómica e ressonância magnética, para identificar as espécies lipídicas hepáticas, tais como fosfolípidos (FL), triglicéridos (TG), diacilgliceróis (DAG) e ácidos gordos (AG). O modelo animal usado foi o rato Wistar, mantido nos últimos 4 meses, antes de completar 1 ano de idade, com metilglioxal (100mg/Kg/dia) (grupo MG), com dieta gorda rica em TG (grupo HFD) ou com ambas (grupo HFDMG) e comparados com os controlos com dieta normal (n=12/grupo). As técnicas de lipidómica usadas foram cromatografia líquida com espetrometria de massa e cromatografia gasosa para determinar a composição hepática de PL, TG e AG. Usou-se também espectroscopia (9 Tesla), não invasiva, de ressonância magnética nuclear 1H (NMR) nos ratos vivos para determinar os TG e DAG hepáticos. Os mediadores proteicos totais e fosforilados da via da insulina e da oxidação lipídica no fígado também foram analisados por western blot. Os ratos, com dieta gorda (HFD), aumentaram o peso corporal, mas o efeito foi parcialmente inibido pelo metilglioxal (HFDMG). Além disso, o grupo HFDMG apresenta um aumento dos ácidos gordos livres no plasma, hiperinsulinemia, insulino-resistência e intolerância à glicose. No fígado, as técnicas de lipidómica e NMR mostraram um aumento da massa gorda no fígado nos grupos HFD e HFDMG, mas apenas no grupo HFD se verifica o aumento do AG 18:1 (comum na dieta). Apesar de não haver diferença significativa no grupo HFD, o grupo HFDMG apresenta uma diminuição dos AG insaturados e aumento dos saturados; isto deve-se à diminuição dos monoinsaturados neste grupo. Quanto à esterificação dos glicerolípidos, o grupo HFDMG apresenta uma menor percentagem da total esterificação dos gliceróis, sugerindo o aumento dos DAG, em relação aos TG. Também, este grupo apresenta um ratio AG/glicerol aumentado, ou seja, com aumento de AG não esterificados. A análise por western blot mostrou uma diminuição da via do receptor da insulina especialmente no grupo HFDMG. Em suma, estes resultados sugerem que a glicação causa alterações do metabolismo lipidico-hepático num contexto de hiperlipidemia, contribuindo possivelmente para a lipotoxicidade hepática, progressão acelerada de insulino-resistência e patologia do fígado gordo.
Livres sur le sujet "Systemic induced resistance"
Choudhary, Devendra K., et Ajit Varma, dir. Microbial-mediated Induced Systemic Resistance in Plants. Singapore : Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0388-2.
Texte intégralTuzun, Sadik, et Elizabeth Bent, dir. Multigenic and Induced Systemic Resistance in Plants. Boston, MA : Springer US, 2006. http://dx.doi.org/10.1007/b101085.
Texte intégralStrömberg, Anita. Induced systemic resistance in potato to late blight. Uppsala, Sweden : Swedish University of Agricultural Sciences, Dept. of Plant and Forest Protection, Section for Plant Pathology, 1994.
Trouver le texte intégralVarma, Ajit, et Devendra K. Choudhary. Microbial-mediated Induced Systemic Resistance in Plants. Springer, 2018.
Trouver le texte intégralSadik, Tuzun, et Bent Elizabeth. Multigenic and Induced Systemic Resistance in Plants. Springer London, Limited, 2006.
Trouver le texte intégralVarma, Ajit, et Devendra K. Choudhary. Microbial-mediated Induced Systemic Resistance in Plants. Springer, 2016.
Trouver le texte intégral(Editor), Tuzun Sadik, et Bent Elizabeth (Editor), dir. Multigenic and Induced Systemic Resistance in Plants. Springer, 2005.
Trouver le texte intégralVarma, Ajit, et Devendra K. Choudhary. Microbial-Mediated Induced Systemic Resistance in Plants. Springer London, Limited, 2016.
Trouver le texte intégralSadik, Tuzun, et Bent Elizabeth. Multigenic and Induced Systemic Resistance in Plants. Springer, 2010.
Trouver le texte intégralLucena, Carlos, Sabine Dagmar Zimmermann, Ricardo Aroca et Jianfei Wang, dir. Beneficial Microbes and the Interconnection Between Crop Mineral Nutrition and Induced Systemic Resistance. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-086-4.
Texte intégralChapitres de livres sur le sujet "Systemic induced resistance"
Van Loon, L. C. « Systemic Induced Resistance ». Dans Mechanisms of Resistance to Plant Diseases, 521–74. Dordrecht : Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-3937-3_13.
Texte intégralNadarajah, Kalaivani K. « Induced Systemic Resistance in Rice ». Dans Microbial-mediated Induced Systemic Resistance in Plants, 103–24. Singapore : Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0388-2_7.
Texte intégralMadamanchi, Nageswara Rao, et Joseph Kuć. « Induced Systemic Resistance in Plants ». Dans The Fungal Spore and Disease Initiation in Plants and Animals, 347–62. Boston, MA : Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-2635-7_16.
Texte intégralKuć, Joseph. « Induced Systemic Resistance — An Overview ». Dans Developments in Plant Pathology, 169–75. Dordrecht : Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-015-8420-3_8.
Texte intégralBeardon, Emily, Julie Scholes et Jurriaan Ton. « How do Beneficial Microbes Induce Systemic Resistance ? » Dans Induced Resistance for Plant Defense, 232–48. Chichester, UK : John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118371848.ch11.
Texte intégralKumar, Manoj, Priyanku Teotia, Ajit Varma, Narendra Tuteja et Vivek Kumar. « Induced Systemic Resistance by Rhizospheric Microbes ». Dans Microbial-mediated Induced Systemic Resistance in Plants, 197–206. Singapore : Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0388-2_13.
Texte intégralBinder, Andres, Gianni Baer, Christina Hofmann et Kati Kováts. « Mechanisms in Systemic Induced Disease Resistance ». Dans NATO ASI Series, 267–72. Berlin, Heidelberg : Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74158-6_32.
Texte intégralStermer, Bruce A. « Molecular regulation of Systemic Induced Resistance ». Dans Developments in Plant Pathology, 111–40. Dordrecht : Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-015-8420-3_5.
Texte intégralBisen, Kartikay, Chetan Keswani, J. S. Patel, B. K. Sarma et H. B. Singh. « Trichoderma spp. : Efficient Inducers of Systemic Resistance in Plants ». Dans Microbial-mediated Induced Systemic Resistance in Plants, 185–95. Singapore : Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0388-2_12.
Texte intégralMagotra, Shanu, Deepika Trakroo, Sneha Ganjoo et Jyoti Vakhlu. « Bacillus-Mediated-Induced Systemic Resistance (ISR) Against Fusarium Corm Rot ». Dans Microbial-mediated Induced Systemic Resistance in Plants, 15–22. Singapore : Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0388-2_2.
Texte intégralActes de conférences sur le sujet "Systemic induced resistance"
« Systemic Acquired Resistance Induced by Some Biotic Agents against Downy Mildew of Cucumber Disease ». Dans International Conference on Plant, Marine and Environmental Sciences. International Institute of Chemical, Biological & Environmental Engineering, 2015. http://dx.doi.org/10.15242/iicbe.c0115022.
Texte intégralKing, A., C. Arnaud, G. Vial, E. Billoir, B. Ozcan, E. Belaidi, C. O'Donnell et S. Ryan. « The effect of the GLP-1 analogue Liraglutide on intermittent hypoxia-induced systemic insulin resistance in vivo ». Dans ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2182.
Texte intégralDelgado, Yamixa, Mallesh Pandrala, Daraishka Perez, Eddian Velazquez, Jaisy Vega, Melissa Milian et Anamaris Torres. « Abstract 6375 : Development of novel Pt-based drugs using Deferasirox as ligand to diminish systemic toxicity and resistance induced by CisPt ». Dans Proceedings : AACR Annual Meeting 2020 ; April 27-28, 2020 and June 22-24, 2020 ; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-6375.
Texte intégral« Bioformulations and Indigenous Plant Protection Measures in Enhancing the Vitalities of Bio-Control Agents for Induced Systemic Resistance Suppressing Asian Soybean Rust in India ». Dans International Conference on Biological, Civil and Environmental Engineering. International Institute of Chemical, Biological & Environmental Engineering, 2014. http://dx.doi.org/10.15242/iicbe.c0314059.
Texte intégralAngelotti, Austin, Rachel Cole, Amy Webb, Maciej Pietrzak et Martha Belury. « Diet-induced Gene Expression Changes of Cachectic Muscle, Adipose, and Liver ». Dans 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/gvbe2596.
Texte intégralSchmidt, B., M. R. Buchanan, F. Ofosu, L. A. Brooker, M. Andrew et McMaster Univ. « ANTITHROMBOTIC PROPERTIES OF HEPARIN IN A NEONATAL MODEL OF THROMBIN INDUCED VENOUS STASIS THROMBOSIS ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643608.
Texte intégralGarattini, E., M. Bolis, A. Vallerga, M. Fratelli, G. Paroni, A. Zanetti, M. Kurosaki, M. Gianni', V. Celestini et M. Terao. « Abstract P5-05-09 : Systemic perturbations induced by all-trans retinoic acid in the gene-expression profiles of sixteen breast cancer cell lines characterized by sensitivity and resistance to the anti-proliferative effects of the retinoid ». Dans Abstracts : 2018 San Antonio Breast Cancer Symposium ; December 4-8, 2018 ; San Antonio, Texas. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-p5-05-09.
Texte intégralSilva, Ines, Annie Tasker, Camelia Quek, Robert Rawson, Su Yin Lim, Kevin Wang, Jordan Conway et al. « Abstract 975 : Liver metastases (mets) induce systemic immunosuppression and immunotherapy resistance in metastatic melanoma ». Dans Proceedings : AACR Annual Meeting 2019 ; March 29-April 3, 2019 ; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-975.
Texte intégralSilva, Ines, Annie Tasker, Camelia Quek, Robert Rawson, Su Yin Lim, Kevin Wang, Jordan Conway et al. « Abstract 975 : Liver metastases (mets) induce systemic immunosuppression and immunotherapy resistance in metastatic melanoma ». Dans Proceedings : AACR Annual Meeting 2019 ; March 29-April 3, 2019 ; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-975.
Texte intégralOnnink, R., J. Gerritsma et J. A. Keuning. « The Delft Systematic Yacht Hull (Series II) Experiments ». Dans SNAME 10th Chesapeake Sailing Yacht Symposium. SNAME, 1991. http://dx.doi.org/10.5957/csys-1991-003.
Texte intégralRapports d'organisations sur le sujet "Systemic induced resistance"
Michel Jr., Frederick C., Harry A. J. Hoitink, Yitzhak Hadar et Dror Minz. Microbial Communities Active in Soil-Induced Systemic Plant Disease Resistance. United States Department of Agriculture, janvier 2005. http://dx.doi.org/10.32747/2005.7586476.bard.
Texte intégralHarman, Gary E., et Ilan Chet. Enhancement of plant disease resistance and productivity through use of root symbiotic fungi. United States Department of Agriculture, juillet 2008. http://dx.doi.org/10.32747/2008.7695588.bard.
Texte intégralSessa, Guido, et Gregory Martin. Role of GRAS Transcription Factors in Tomato Disease Resistance and Basal Defense. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696520.bard.
Texte intégralLoebenstein, Gad, William Dawson et Abed Gera. Association of the IVR Gene with Virus Localization and Resistance. United States Department of Agriculture, août 1995. http://dx.doi.org/10.32747/1995.7604922.bard.
Texte intégralHoitink, Harry A. J., Yitzhak Hadar, Laurence V. Madden et Yona Chen. Sustained Suppression of Pythium Diseases : Interactions between Compost Maturity and Nutritional Requirements of Biocontrol Agents. United States Department of Agriculture, juin 1993. http://dx.doi.org/10.32747/1993.7568755.bard.
Texte intégralHoitink, Harry A. J., Yitzhak Hadar, Laurence V. Madden et Yona Chen. Sustained Suppression of Pythium Diseases : Interactions between Compost Maturity and Nutritional Requirements of Biocontrol Agents. United States Department of Agriculture, juin 1993. http://dx.doi.org/10.32747/1993.7568746.bard.
Texte intégralFluhr, Robert, et Maor Bar-Peled. Novel Lectin Controls Wound-responses in Arabidopsis. United States Department of Agriculture, janvier 2012. http://dx.doi.org/10.32747/2012.7697123.bard.
Texte intégralAvni, Adi, et Kirankumar S. Mysore. Functional Genomics Approach to Identify Signaling Components Involved in Defense Responses Induced by the Ethylene Inducing Xyalanase Elicitor. United States Department of Agriculture, décembre 2009. http://dx.doi.org/10.32747/2009.7697100.bard.
Texte intégralPalukaitis, Peter, Amit Gal-On, Milton Zaitlin et Victor Gaba. Virus Synergy in Transgenic Plants. United States Department of Agriculture, mars 2000. http://dx.doi.org/10.32747/2000.7573074.bard.
Texte intégralNorelli, John L., Moshe Flaishman, Herb Aldwinckle et David Gidoni. Regulated expression of site-specific DNA recombination for precision genetic engineering of apple. United States Department of Agriculture, mars 2005. http://dx.doi.org/10.32747/2005.7587214.bard.
Texte intégral