Livres : « Susceptibilité au stress »

1

Gillis, A. R. Cultural susceptibility to crowding: An empirical analysis. Centre for Urban and Community Studies, University of Toronto, 1986.

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Lyle, Fred F. Stress-corrosion cracking susceptibility of weldments in duplex stainless steels. Materials Technology Institute of the Chemical Process Industries, 1989.

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Madra, Moneek. Interactions between the BDNF (Val66Met) variant and stress as a model of susceptibility to Anorexia Nervosa. [publisher not identified], 2015.

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4

International Berzelius Symposium. (6th 1985 Malmö, Sweden). Social support--health and disease. Edited by Isacsson Sven-Olof, Janzon Lars, and Sweden. Delegationen för social forskning. Almqvist & Wiksell International, 1986.

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5

J, Sholar Clyde. Evaluation of the drought susceptibility of water supplies used in the Kentucky River Basin in 1988. U.S. Dept. of the Interior, U.S. Geological Survey, 1991.

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Sholar, Clyde J. Evaluation of the drought susceptibility of water supplies used in the Kentucky River Basin in 1988. U.S. Dept. of the Interior, U.S. Geological Survey, 1991.

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B, Ludvigsen J., European Association for Animal Production. Commission on Animal Management and Health., European Association for Animal Production. Commission on Pig Production., and European Association for Animal Production. Meeting, eds. Stress susceptibility and meat quality in pigs: Proceeding of Commis[s]ion on Animal Management and Health and Commission on Pig Production joint session, Halkidiki, Greece, 30. Sept.-5. Oct. 1985. European Association for Animal Production, 1985.

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8

L, Power Michael, and Schulkin Jay, eds. Birth, distress, and disease: Placental-brain interactions. Cambridge University Press, 2005.

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9

Tsai, Jack, Natalie Jones, Robert H. Pietrzak, Ilan Harpaz-Rotem, and Steven M. Southwick. Susceptibility, Resilience, and Trajectories. Edited by Frederick J. Stoddard, David M. Benedek, Mohammed R. Milad, and Robert J. Ursano. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457136.003.0019.

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Nearly everyone experiences a highly stressful or traumatic event during their lifetime. However, individual responses to such events vary widely from person to person. Some people respond with symptoms of anxiety, depression, acute stress, or posttraumatic stress disorder, yet others experience minimal or no psychiatric symptoms after trauma. What makes one person more susceptible and another more resilient to the negative effects of trauma? What are the different adaptive trajectories of trauma survivors and what determines their trajectory? These are some of the questions that are examined in this chapter, which focuses on what is currently known about resilience to stress. The chapter is divided into five sections: definition, prevalence, and measurement of resilience; longitudinal studies on trajectories after trauma exposures; research on factors that are predictive of resilience and different trajectories; interventions that have been developed to increase resilience; and discussion about future directions for research on resilience.

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Who Gets Ptsd?: Issues of Posttraumatic Stress Vulnerability. Charles C. Thomas Publisher, 2006.

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M, Violanti John, and Paton Douglas, eds. Who gets PTSD?: Issues of posttraumatic stress vulnerability. Charles C Thomas, 2005.

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(Editor), John M. Violanti, and Douglas Paton (Editor), eds. Who Gets PTSD?: Issues of Posttraumatic Stress Vulnerability. Charles C. Thomas Publisher, 2006.

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13

Kraft, Patricia Maria. The effect of suggestion and stress inoculation on the reduction of pain and the modification of hypnotic suggestibility with the Carleton Skills Training Program. 1991.

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Swift, Adrienne. Anxiety and college women: Exploring a diathesis-stress model. 1996.

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Swift, Adrienne. Anxiety and college women: Exploring a diathesis-stress model. 1996.

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Inst, Materials Technology. Stress Corrosion Cracking Susceptibility of Weldments in Duplex Stainless Steels. Natl Assn of Corrosion, 1989.

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Alcantara, Lyonna F., Eric M. Parise, and Carlos A. Bolaños-Guzmán. Animal Models of Mood Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0026.

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Animal modeling has advanced our understanding of the underlying pathophysiology of human neuropsychiatric disorders and facilitated development of safer, more efficient medications. Similar to humans with depression, rodents exposed to various stress paradigms exhibit aberrant responses to rewarding stimuli, along with hormonal and immunological dysregulation. Development of more complex models, such as social defeat, has led to a firmer grasp of the mechanisms mediating resilience and susceptibility to stress; and adapted versions of social defeat have yielded insights into how emotional stress influences development of mood disorders. This chapter focuses on stress-induced models of mood disorders and outlines how a depression-like phenotype is induced and tested in rodents.

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Yehuda, Rachel, and Nikolaos P. Daskalakis, eds. Programming HPA-axis by early life experience: Mechanisms of stress susceptibility and adaptation. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-308-0.

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Stoddard Jr., Frederick J., Robert J. Ursano, and Stephen J. Cozza. Population Trauma. Edited by Frederick J. Stoddard, David M. Benedek, Mohammed R. Milad, and Robert J. Ursano. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457136.003.0010.

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This chapter reviews trauma- and stressor-related disorders (TSRDs) as they relate to disaster, defined by the World Health Organization as “a severe disruption, ecological and psychosocial, which greatly exceeds the coping capacity of the affected community.” Some are human-made such as a terrorist event or shooting, while others are due to natural events such as earthquake or hurricane. Humanitarian emergencies are also a class of disasters. Since most but not all people and communities are resilient, the prevalence of TSRDs after disaster and what interventions are optimal is highly relevant to disaster recovery. The chapter discusses the impact of disaster preparedness, factors that influence how communities cope with disaster, and the effect of trauma and stress on populations. It goes on to review factors that influence susceptibility and resilience to disaster trauma, the range of psychological consequences of disaster, and early interventions for TSRDs in response to disaster.

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Test methods for assessing the susceptibility of prestressing steels to hydrogen induced SCC. Published for the European Federation of Corrosion by Maney on behalf of the Institute of Materials, Minerals and Mining, 2004.

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21

Jenney, Colin, and Angela Liegey Dougall. Immune System Functioning and Mental Health. Edited by Sara Maltzman. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199739134.013.45.

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During the past few decades, research from multiple disciplines has elucidated the profound connections between the immune system and mental health. This chapter provides a review of this literature, placing emphasis on the connections between inflammation and cytokines, and stress, depression, posttraumatic stress disorder, schizophrenia, bipolar disorder, and social support. Additionally, brief overviews of the role of the natural immune system and adaptive immunity, as well as past research investigating stress are included. Further attention is focused on the physical health consequences of immune system dysregulation, ranging from increased susceptibility to infectious diseases, delayed wound healing, autoimmune responses, age-related elevations in proinflammatory cytokines, and decreased effectiveness of protective vaccinations. The chapter concludes with a discussion of the health effects and clinical implications of the relationships discussed throughout, as well as future directions to advance this field of study.

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Isacsson, S. O., and L. Janzon. Social Support: Health & Disease. Coronet Books, 1986.

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23

Christensen, Daniel K. Effects of dry flotationn [i.e. flotation] rest, natural environment video presentation, and hypnotizability on recovery from induced stress. 1994.

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McCarty, Richard. Stress and Mental Disorders: Insights from Animal Models. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190697266.001.0001.

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Stress has now been recognized as an important factor in the development or recurrence of various mental disorders, from major depressive disorder to bipolar disorder to anxiety disorders. Stressful stimuli appear to exert their effects by acting upon individuals with susceptible genotypes. Over the past 50 years, animal models have been developed to study these dynamic interactions between stressful stimuli and genetically susceptible individuals during prenatal and postnatal development and into adulthood. This book begins with a discussion of the history of psychiatric diagnosis and the recent goal of moving toward precision psychiatry, followed by a review of clinical research on connections between stressful stimuli and the development of psychiatric disorders. Chapters are also included on neuroendocrine, immune, and brain systems involved in responses to stress. Additional chapters focus on the development of animal models in psychiatry and the susceptibility of the developing organism to stressful stimuli. Subsequent chapters are devoted to animal models of specific stress-sensitive psychiatric disorders, including schizophrenia, autism spectrum disorders, bipolar disorder, anxiety disorders, depression, and post-traumatic stress disorder. These chapters also focus on the identification of promising molecular targets for development of new drug therapies; a chapter examines animal models of resilience to stress-induced behavioral alterations as a newer approach to understand why some animals (e.g., inbred mice) are susceptible to stress and others are resilient, even if they are essentially genetically identical. The final chapter discusses how these basic laboratory animal models are providing promising leads for future breakthroughs in the diagnosis, treatment, and prevention of mental disorders.

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Leinonen, Heikki. Evaluation of stress corrosion cracking susceptibility of austenitic stainless steels in CaCl2 solution by the constant load method. 1995.

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26

Franke, Barbara, and Jan K. Buitelaar. Gene–environment interactions. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0005.

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ADHD is highly heritable, but environmental factors also play significant roles in disease aetiology and outcome. Genetic and environmental influences are likely to show different types of interplay, with gene–environment interactions (G×E) playing a part. Different models of G×E exist, with the most frequently investigated in ADHD up to the present being the diathesis–stress and differential susceptibility models. The most frequently studied have been monoaminergic genes, often based on a single genetic variant. Only a single genome-wide study has been reported thus far. Environmental factors investigated include prenatal and postnatal risk factors for ADHD, in particular prenatal exposure to smoking or alcohol and aspects of parenting.

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Siebert, Stefan, Sengupta Raj, and Alexander Tsoukas. The genetics of axial spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198755296.003.0004.

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Family and twin studies have long suggested a large genetic component in ankylosing spondylitis (AS). The genetic association with HLA-B27 remains one of the strongest single gene variant associations reported in any complex polygenic disease. The exact mechanism by which HLA-B27 contributes to AS remains unknown, with three main theories proposed: the arthritogenic peptide, endoplasmic reticulum stress with unfolded protein response, and homodimerization theories. Genome-wide association studies have identified a number of other important susceptibility genes for AS, several of which overlap with other spondyloarthritis conditions. Of these, ERAP1 and IL-23R, are covered in more detail, highlighting their functional importance.

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Jones, Eric Merwin. Investigation of the stress corrosion cracking susceptibility of annealed and heat treated alloy 625 castings and forgings in sea water. 1987.

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Test Methods for Assessing the Susceptibility of Prestressing Steel to Hydrogen Induced Scc (European Federation of Corrosion). Maney Publishing, 2004.

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Furst, Eric M., and Todd M. Squires. Magnetic bead microrheology. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199655205.003.0008.

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Magnetism is a convenient force for actively pulling colloidal particles in a material. Many materials of interest in a microrheology experiment have a negligible magnetic susceptibility, and so embedded magnetic particles can be subject to relatively strong forces by fields imposed from outside of the sample. These are usually generated by electromagnets, but can also include the use of permanent magnets, or a combination of both. Such “magnetic tweezers” are used as sensitive force probes, capable of generating forces ranging from femtonewtons to nanonewtons. Magnetic forces and magnetic materials are reviewed and magnetic tweezer designs discussed. Linear and non-linear measurements using magnetic tweezers are presented, including studies yield stress and shear thinning. The operating regime of magnetic tweezer microrheology is presented, which enables microrheology experiments to access stiffer materials.

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Razzoli, Maria, Alessandro Bartolomucci, and Valeria Carola. Gene-by-Environment Mouse Models for Mood Disorders. Edited by Turhan Canli. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.013.

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Much of the impact of genes on mood disorders likely depends on interactions between genes and the environment. Recent studies demonstrating an interaction between specific genes and life stressful events (early and/or adult) in the modulation of several mood disorders (e.g., serotonin transporter and brain-derived neurotrophic factor genes) have compelled researchers to incorporate information about adverse environmental experiences into the study of genetic risk factors; these same gene-by-environment (G×E) interactions have been identified in mouse models. Notably, G×E not yet described in humans (e.g., serotonin 1A receptor gene) have been uncovered, providing helpful indications to discover similar interactions in humans. Accurate knowledge of the modality of expression of gene-by-stress interaction may help design prevention protocols aimed at identifying susceptibility to mood disorders on the basis of genetic predisposition and exposure to environmental stressful conditions, thus providing patients with appropriate pharmacological and psychological support.

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Molina, Brooke S. G., Margaret H. Sibley, Sarah L. Pedersen, and William E. Pelham. The Pittsburgh ADHD Longitudinal Study (PALS). Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190213589.003.0005.

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The Pittsburgh ADHD Longitudinal Study (PALS) is a prospective study of children diagnosed with ADHD and a comparison group of demographically similar youth without ADHD. The study tested hypotheses regarding the development of alcohol and other substance use in children with ADHD. Assessments offered comprehensive coverage of theoretically important variables, including frequency and quantity of alcohol and drug consumption, age at first substance use, ADHD symptom persistence, conduct problems and delinquency, social functioning, stress and coping, risky behaviors, depression and anxiety, academic and vocational outcomes, and cost. Findings revealed differences in developmental pathways and outcomes between the ADHD and comparison groups. Childhood ADHD increased the risk of developing alcohol and substance use disorders by adulthood, particularly when ADHD symptoms persisted past childhood and conduct/antisocial tendencies developed. Children with ADHD also demonstrated susceptibility to stressors, weaker coping skills, unfavorable cognitive patterns, risky behaviors, and impaired functioning across a range of domains.

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Robeck, Ilene R., Stephen C. Hunt, Lucile Burgo-Black, Jeremiah McKelvey, Afreen Siddiqui, and Anthony J. Mariano. Pain and Addiction in Military Personnel and Veterans (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0033.

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Veterans of military service pose a special case both in their susceptibility to addiction and their risk for a co-occurring disorder, posttraumatic stress disorder. The elements of a military history are provided; this is not intuitive, and is salient both for determining entitlement status and to assess the cumulative injury. Numerous pain syndromes can occur as the result of both long-term wear and tear and abrupt injury, requiring consideration for disability compensation. PTSD and traumatic brain injury are more common here than perhaps among any other occupational group. Unique risks and concerns accompany the experience of warfare in different theaters; special sections are devoted to Iraq and Afghanistan; the first and second Gulf wars; and to Vietnam. Women constitute a newly enfranchised military group as subject to injury as their male cohorts. Several pain care innovations have been championed by the Veterans Administration, and these are described.

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Barrett, Catherine E., and Larry J. Young. Molecular Neurobiology of Social Bonding. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.001.

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Many psychiatric illnesses, including autism spectrum disorders (ASD), schizophrenia, and depression, are characterized by impaired social cognition and a compromised ability to form social relationships. Although drugs are currently available to treat other symptoms of these disorders, none specifically target the social deficits. In order to develop pharmacotherapies to enhance social functioning, particularly for ASD where social impairment is a core symptom, we must first understand the basic neurobiology underlying complex social behaviors. The socially monogamous prairie vole (Microtus ochrogaster) has been a remarkably useful animal model for exploring the neural systems regulating complex social behaviors, including social bonding. Prairie voles form enduring social bonds between mated partners, or pair bonds, and display a biparental familial structure that is arguably very similar to that of humans. Here we discuss the neural systems underlying social bonding in prairie voles, including the neuropeptides oxytocin and vasopressin, opioids, dopaminergic reward and reinforcement, and stress-related circuitry, as well as the susceptibility of social functioning to early life experiences. We highlight some of the remarkable parallels that have been discovered in humans, and discuss how research in prairie voles has already led to novel therapies to enhance social functioning in ASD.

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Rodrigo, Olivares-Caminal, Douglas John, Guynn Randall, Kornberg Alan, Paterson Sarah, and Singh Dalvinder. Part II Bank Resolution, 5 Legal Aspects of Banking Regulation in the UK and US. Oxford University Press, 2016. http://dx.doi.org/10.1093/law/9780198725244.003.0005.

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The chapter starts by describing what the banking industry does and how it is regulated and managed. The susceptibility of banks to collapse or failure is the result of the ‘maturity mismatch’ between their borrowing and lending. The fallout from failure can have very wide systematic consequences, and efficient regulation is vital. This chapter sets out the principal features of the UK and US bank regulation. First, the UK proposals for reform and key features of the Financial Conduct Authority (FCA) and the Prudential Regulation Authority (PRA) supervisory regime are outlined. Secondly, the US federal bank regulatory system and the impact of the Dodd-Frank Wall Street Reform and Consumer Act 2010, as well as the impact of the Act on areas of the regulatory and supervisory regime are described. The chapter examines how significant reform has improved the safety and soundness of banking and brought supervisory measures to the fore.

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Otgaar, Henry, and Mark L. Howe, eds. Finding the Truth in the Courtroom. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190612016.001.0001.

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The focus of this book is on how legal professionals, legal/forensic psychologists, and memory researchers can decide when statements or identifications are based on truthful or fabricated experiences and whether if fabricated, can we distinguish between lies, deception, and false memories. The ultimate focus is to assemble recent experimental work and case studies in which deception or false memory plays a dominant role. That is, in many criminal trials, forensic technical evidence is lacking and triers of fact must rely on the reliability of eyewitness statements, identifications, and testimony. However, such reports can be riddled with deceptive statements or erroneous recollections. Based on such considerations, the question arises as to how one should weigh such eyewitness accounts given the theoretical and empirical knowledge in this field. Topics discussed are, for example, related to the susceptibility to suggestive pressure (e.g., “Under which circumstances are children or adults the most vulnerable to suggestion?”), the fabrication of symptoms (e.g., “How to detect whether PTSD symptoms are malingered?”), or the detection of deceit (e.g., “Which paradigms are promising in deception detection?”). By using this approach, this book unites diverse streams of research (i.e., deception, malingering, false memory) that are involved in the reliability of eyewitness statements.

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Schulkin, Jay, and Michael L. Power. Birth, Distress and Disease: Placental-Brain Interactions. Cambridge University Press, 2005.

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Schulkin, Jay, and Michael L. Power. Birth, Distress and Disease: Placental-Brain Interactions. Cambridge University Press, 2009.

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Schulkin, Jay, and Michael L. Power. Birth, Distress and Disease: Placental-Brain Interactions. Cambridge University Press, 2010.

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