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1

Baron-Faust, Rita. Sleep disorders : Common problems & treatments. Norwalk, CT : Belvoir Media Group, 2009.

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Baron-Faust, Rita. Sleep disorders : Common problems & treatments. Norwalk, CT : Belvoir Media Group, 2009.

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3

Behavioral treatments for sleep disorders : A comprehensive primer of behavioral sleep medicine interventions. Amsterdam : Academic, 2011.

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4

Ian, St James-Roberts, Harris Gillian et Messer David J. 1952-, dir. Infant crying, feeding, and sleeping : Devleopment, problems, and treatments. New York : Harvester Wheatsheaf, 1993.

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5

Gillian, Harris, Messer David J. 1952- et St James-Roberts Ian, dir. Infant crying, feeding and sleeping : Development, problems and treatments. New York : Harvester Wheatsheaf, 1993.

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6

Steven, Poceta J., et Mitler Merrill Morris 1945-, dir. Sleep disorders : Diagnosis and treatment. Totowa, N.J : Humana Press, 1998.

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7

Can't sleep, won't sleep. Milverton [England] : Capall Bann, 2005.

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8

Sacchetti, Lisandro M., et Priscilla Mangiardi. Obstructive sleep apnea : Causes, treatment and health implications. New York : Nova Science Publishers, 2012.

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9

Sleep solutions. London : Ward Lock, 1999.

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10

Guglietta, Antonio, dir. Drug Treatment of Sleep Disorders. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-11514-6.

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1922-, Williams Robert L., Karacan Ismet 1927- et Moore Constance A, dir. Sleep disorders : Diagnosis and treatment. 2e éd. New York : Wiley, 1988.

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12

Eugene, Lefebvre, et Moreau Renaud, dir. Snoring : Causes, diagnosis, and treatment. Hauppauge, N.Y : Nova Science Publishers, 2009.

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13

Pedr, Fulke, et Vaughan Sior, dir. Sleep deprivation : Causes, effects, and treatment. Hauppauge, NY : Nova Science Publishers, 2009.

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14

Pressman, Mark R., et William C. Orr, dir. Understanding sleep : The evaluation and treatment of sleep disorders. Washington : American Psychological Association, 1997. http://dx.doi.org/10.1037/10233-000.

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R, Pressman Mark, et Orr William C, dir. Understanding sleep : The evaluation and treatment of sleep disorders. Washington, DC : American Psychological Association, 1997.

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16

Wauquier, A. Dr. Drowsy's sleep prescription. Dallas : Somnus Press, 2003.

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17

Estivill, Eduard. Dormir sin descanso : Trastornos del sueño. Barcelona : Oceano Grupo Editorial, 1999.

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18

Estivill, Eduard. Dormir sin descanso : Trastornos del sueño. Barcelona : Oceano Grupo Editorial, 1999.

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19

Chao kuai shui mian shi dian. Taibei Shi : Hua wen wang gu fen you xian gong si, 2002.

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20

Nardo, Don. Sleep problems. Detroit : Lucent Books, a part of Gale, Cengage Learning, 2014.

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21

Therapy in sleep medicine. Philadelphia, PA : Elsevier/Saunders, 2012.

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22

Smolik, Petr. Kleine-Levin syndrome : Ethiopathogenesis and treatment. Praha : Univerzita Karlova, 1989.

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23

Slumbertime : A parent's guide for children's sleep and sleep disorders. Corpus Christi, Tex : Pearson Publishing Co., 2008.

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24

Giles, Stephen. How to go to sleep ... : And stay there. Great Ambrook : White Ladder, 2007.

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25

Tuckey, C. Lloyd. Psycho-therapeutics, or, Treatment by sleep and suggestion. Bristol, U.K : Thoemmes Press, 1998.

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26

Pigeon, Wilfred R. Sleepmanual : Training your mind and body to achieve the perfect night's sleep. Sous la direction de Sateia Michael J. 1948-. Hauppague, NY : Barron's, 2010.

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27

Ankang, Lü, et Lu Xiaolan, dir. Dui fu shi mian 50 fa. Shanghai : Shanghai ke xue ji shu wen xian chu ban she, 2000.

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28

Woodson, B. Tucker. Obstructive sleep apnea syndrome : Diagnosis and treatment. Alexandria, VA : American Academy of Otolaryngology--Head and Neck Surgery Foundation, 1996.

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29

Giora, Pillar, et Malhotra Atul, dir. Sleep disorders : Diagnosis, management and treatment : a handbook for clinicians. London : Martin Dunitz, 2002.

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30

Behavioral Treatments for Sleep Disorders. Elsevier, 2011. http://dx.doi.org/10.1016/c2009-0-62216-9.

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31

Durand, V. Mark. Pediatric Sleep Disorders : Therapist Guide (Treatments That Work). Oxford University Press, USA, 2008.

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Durand, V. Mark. Pediatric Sleep Disorders : Parent Workbook (Treatments That Work). Oxford University Press, USA, 2008.

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33

Innominato, Pasquale F., et David Spiegel. Circadian rhythms, sleep, and anti-cancer treatments. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0016.

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The circadian timing system temporally regulates biological functions relevant for psycho-physical wellbeing, spanning all the systems related to health. Hence, disruption of circadian rhythms, along with sleep cycles, is associated with the development of several diseases, including cancer. Moreover, altered circadian and sleep functions negatively impact on cancer patients’ quality of life and survival, above and beyond known determinants of outcome. This alteration can occur as a consequence of cancer, but also of anti-cancer treatments. Indeed, circadian rhythms govern also the ability of detoxifying chemotherapy agents across the 24 hours. Hence, adapting chemotherapy delivery to the molecular oscillations in relevant drug pathways can decrease toxicity to healthy cells, while increasing the number of cancer cells killing. This chronomodulated chemotherapy approach, together with the maintenance of proper circadian function throughtout the whole disease challenge, would finally result in safer and more active anticancer treatments, and in patients experiencing better quality and quantity of life.
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Graci, Gina, et Kathleen J. Sexton-Radek. Sleep Disorders : Elements, History, Treatments, and Research. ABC-CLIO, LLC, 2021.

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35

Lipskis, Dr Lynn, et Dr Edmund Lipskis. Breathe, Sleep, Live, Smile : Integrative Treatments For TMJ, Sleep Apnea, And Orthodontics. Advantage Media Group, 2019.

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36

Behavioral Treatments for Sleep Disorders : A Comprehensive Primer of Behavioral Sleep Medicine Interventions. Academic Press, 2010.

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37

Perlis, Michael L., Brett Kuhn et Mark Aloia. Behavioral Treatments for Sleep Disorders : A Comprehensive Primer of Behavioral Sleep Medicine Interventions. Elsevier Science & Technology Books, 2010.

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38

Cooper, Remi. Depression and Nervous/Sleep Disorders : Natural Treatments (Woodland Health). Woodland Publishing, 1997.

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39

Pittman, Marcus, et Adrian Williams. Central sleep apnoea. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0005.

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Central sleep apnoea and Cheyne-Stokes respiration are common forms of sleep-disordered breathing, particularly in patients with co-morbidities such as cardiac and renal disease which, however, often do not require specific treatment. Physicians may encounter such patients in their outpatient clinics or as ward referrals in hospital. A typical case is presented to aid the approach to such patients, including how to make an accurate diagnosis, which of the various treatment modalities to use, and what to do if a treatment fails. The evidence for the different interventions is explored, including oxygen, modes of non-invasive positive airway pressure, and drug treatments, with particular attention to groundbreaking studies.
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40

Burman, Deepa, Daniel J. Buysse et Charles F. Reynolds. Treatment of Sleep Disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0021.

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This chapter provides an evidence-based review of the seven categories of DSM-5 sleep-wake disorders. Specific sleep-wake disorders vary widely in their clinical manifestations, etiologies, treatment, and the level of evidence supporting those treatments. Sleep-wake disorders are prevalent, increase the risk of many common mental and substance use disorders, and exacerbate these disorders, with which they typically co-occur. Unless properly treated in their own right, sleep-wake disorders can impair treatment response and worsen the long-term course of mental disorders. Thus, it is important for psychiatrists and other mental health practitioners to understand the diagnosis and treatment of sleep-wake disorders and when to refer patients to a sleep medicine specialist.
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41

Toh, Song Tar. Prevention, Screening and Treatments for Obstructive Sleep Apnea : Beyond PAP, an Issue of Sleep Medicine Clinics. Elsevier - Health Sciences Division, 2019.

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42

Selim, Bernardo, et Kannan Ramar. Beyond positive airway pressure therapy : experimental and non-conventional treatments in sleep apnoea. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0259.

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With decreased adherence to positive airway pressure therapy to treat sleep apnoeas, non-conventional treatments based on new therapeutic targets are emerging. In central sleep apnoea syndrome associated with heart failure, phrenic nerve stimulation and non-conventional pharmacological treatments such as carbonic anhydrase inhibitors, gas therapies, and cardiac devices are novel alternative therapies. In obstructive sleep apnoea, a better understanding of predominant pathophysiological pathways is characterizing diverse clinical phenotypes. For patients with low arousal threshold, sedatives or hypnotics might be effective, whereas for those with unstable ventilatory control, carbonic anhydrase inhibitors or oxygen might improve obstructive sleep apnoea. For patients with upper airway muscle dysfunction, an increase in pharyngeal tone might be beneficial. This chapter describes ‘experimental’ therapies and novel technologies to treat these disorders.
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Miller, Michelle A. The genetics of sleep. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0006.

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The genetic regulation of normal sleep and sleep disorders is complex and often shows strong environmental interactions. This is a relatively new, and rapidly expanding, area of research, and the number of sleep conditions with established, underlying genetic components is growing. The genetic basis regulating the sleep–wake cycle has identified the Period genes. Their polymorphisms appear to determine the morning/night preferences of individuals. At present, the public health benefits are limited, but will increase as the identification and understanding of genetic causes for sleep conditions improve. This may lead to new diagnostic and treatment options including genetic counselling, improved therapeutic regimes, and new drug treatments.
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44

Hertenstein, Elisabeth, Christoph Nissen et Dieter Riemann. Pharmacological and non-pharmacological treatments of insomnia. Sous la direction de Sudhansu Chokroverty, Luigi Ferini-Strambi et Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0020.

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This chapter evaluates evidence-based treatment options for chronic insomnia. Insomnia is a common sleep disorder characterized by sleep onset and maintenance difficulties and daytime impairment such as reduced concentration and motivation. Cognitive behavioral therapy for insomnia (CBTI) is the first-line treatment for chronic primary and comorbid insomnia. CBTI comprises behavioral treatment (sleep restriction, stimulus control), relaxation, cognitive therapy, and sleep education. Its effects are of medium to large size and are stable up to two years after treatment. Benzodiazepines and benzodiazepine receptor agonists are equally effective for short-term treatment. However, because of their adverse effects, especially in the elderly, and their potential for tolerance and dependence, they are only recommended for a treatment period up to four weeks. Low doses of sedating antidepressants are commonly prescribed for treating chronic insomnia and have shown promising results in clinical trials. However, more research on their long-term efficacy and safety is needed.
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45

Carney, Colleen E., et Taryn G. Moss. Sleep Disorders and Depression. Sous la direction de C. Steven Richards et Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.012.

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Major depressive disorder (MDD) commonly occurs with several sleep disorders, including hypersomnia, breathing or limb-related sleep disturbances, and most notably chronic insomnia. A bidirectional relationship exists between sleep and mood problems, and both issues often warrant timely clinical management. However, there are several assessment- and treatment-related complexities that complicate the clinical management of such patients. For example, there are several overlapping symptoms for MDD and both insomnia and hypersomnia, and the two sleep conditions are both listed as possible symptoms in the diagnostic criteria for MDD. This has led to a well-documented problem of underrecognizing and undertreating these significant disorders in the context of MDD. Moreover, certain effective depression treatments can actually worsen the coexisting sleep disorder. Understanding and treating both disorders (i.e., MDD and the co-occurring sleep disorder) is imperative for effective clinical care. Almost all (i.e., up to 90%) of those with depression report sleep problems. This chapter provides an overview of the etiologic, assessment, and treatment issues inherent in this very large, highly prevalent group.
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46

Braley, Tiffany J., et Alon Y. Avidan. Sleep Disorders in Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0021.

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This chapter summarizes information about sleep disorders commonly experienced by patients with multiple sclerosis. Detailed reviews of insomnia, motor disorders of sleep (including restless legs syndrome or Willis-Ekbom disease), sleep-disordered breathing, REM sleep behavior disorder, and narcolepsy are presented. The epidemiology, pathophysiology, and clinical presentations of each sleep disorder are discussed, with emphasis placed on clinical features of multiple sclerosis that may exacerbate or mimic these conditions. Each section also includes a comprehensive review of the diagnostic approaches and treatments for each condition, within the context of symptoms related to multiple sclerosis that may influence diagnosis or treatment. An approach is suggested to distinguish symptoms of fatigue from those of hypersomnolence.
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Nobili, Lino, Paola Proserpio, Steve Gibbs et Giuseppe Plazzi. Sleep and epilepsy. Sous la direction de Sudhansu Chokroverty, Luigi Ferini-Strambi et Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0028.

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This chapter on sleep and epilepsy examines the activating and deactivating properties of NREM and REM sleep states on interictal epileptic activity and seizures. It reviews specific epileptic syndromes in which seizures manifest a tendency to present exclusively or predominantly during sleep or upon wakening. Particular attention is paid to the description of the different forms of nocturnal frontal lobe epilepsy: autosomal dominant and lesional. There is also a discussion of the negative bidirectional relationship between epilepsy and sleep disorders (sleep apneas and parasomnias) and the effect of pharmacological and nonpharmacological treatments. Finally, a brief review of the relationship between sleep and sudden unexpected death in epilepsy is given.
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48

Dyken, Mark Eric, Kyoung Bin Im, George B. Richerson et Deborah C. Lin-Dyken. Sleep and stroke. Sous la direction de Sudhansu Chokroverty, Luigi Ferini-Strambi et Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0027.

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The study of stroke and sleep is in its infancy, as exemplified by the fact that polysomnography (PSG) has only recently been used to help confirm that obstructive sleep apnea (OSA) is a stroke risk factor. There is a strong association between stroke and sleep problems, as stroke can cause, and also may result from, some sleep disorders. Symptoms of OSA, the most frequent and dangerous sleep problem associated with stroke, often suggest other primary sleep disorders. OSA should be the first concern, and, if diagnosed, positive airway pressure (PAP) and positional therapies are first-line treatments. If OSA is ruled out, good sleep hygiene through cognitive–behavioral techniques (cognitive, sleep restriction, stimulus control, and progressive relaxation therapies) are often recommended, as stroke patients are prone to the adverse effects of medications routinely used for sleep problems.
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Dedhia, Param. Sleep and Preventive Health—An Integrative Understanding and Approach. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190241254.003.0019.

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This chapter focuses on the role of sleep in optimal prevention and begins by introducing the epidemiology of sleep, the history of sleep, and how sleep has been defined in modern medicine. It emphasizes the importance of sleep stages and cycles and the need for both quality and adequate quantity of sleep, and covers common sleep disorders. It discusses the role sleep plays in waking performance and consequently public safety, and how disrupted or disordered sleep is correlated with disease and illness. Finally, this chapter introduces a variety of interventions and treatments for disrupted and disordered sleep—including the treatment of sleep-related breathing disorder, Willis-Ebkom disease, and insomnia—and integrative approaches and lifestyle habits that can help promote optimal sleep.
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Mellman, Thomas A. Sleep and PTSD. Sous la direction de Charles B. Nemeroff et Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0023.

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The Chapter reviews two long standing concerns of sleep research in PTSD. The first is characterizing dysregulations of arousal that manifest in relation to sleep including disturbances of sleep initiation, maintenance, and depth. The other is characterizing nightmare disturbances in PTSD and establishing the role of the neurophysiological substrate of dreams, rapid eye movement sleep (REMS), in the disorder. While there has been uncertainty and controversy regarding these issues, a comprehensive look at findings and their context (e.g. duration of PTSD, setting of recordings) allows some reconciliation of findings. Recent developments include assessment of autonomic balance during sleep and application of functional brain imaging. Emerging research on the role of sleep, particularly REMS, on emotional learning has been linked to PTSD. Treatments specifically targeting nightmares and other sleep disruptions, such as the alpha adrenergic antagonist prazosin and imagery rehearsal therapy, continue to be developed and evaluated.
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