Littérature scientifique sur le sujet « Skeletal muscle fibrosis »
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Articles de revues sur le sujet "Skeletal muscle fibrosis"
Mahdy, Mohamed A. A. « Skeletal muscle fibrosis : an overview ». Cell and Tissue Research 375, no 3 (12 novembre 2018) : 575–88. http://dx.doi.org/10.1007/s00441-018-2955-2.
Texte intégralAmani, Majid, Masoud Rahmati, Mohammad Fathi et Hasan Ahmadvand. « Reduce Muscle Fibrosis through Exercise via NRG1/ErbB2 Modification in Diabetic Rats ». Journal of Diabetes Research 2020 (14 mai 2020) : 1–8. http://dx.doi.org/10.1155/2020/6053161.
Texte intégralMeyer, Gretchen A., et Richard L. Lieber. « Skeletal muscle fibrosis develops in response to desmin deletion ». American Journal of Physiology-Cell Physiology 302, no 11 (1 juin 2012) : C1609—C1620. http://dx.doi.org/10.1152/ajpcell.00441.2011.
Texte intégralZhao, Na, Bo Liu, Si-Wen Liu, Wei Zhang, Hua-Nan Li, Geng Pang, Xiong-Fei Luo et Jin-Gui Wang. « The Combination of Electroacupuncture and Massage Therapy Alleviates Myofibroblast Transdifferentiation and Extracellular Matrix Production in Blunt Trauma-Induced Skeletal Muscle Fibrosis ». Evidence-Based Complementary and Alternative Medicine 2021 (7 juillet 2021) : 1–10. http://dx.doi.org/10.1155/2021/5543468.
Texte intégralLieber, Richard L., et Samuel R. Ward. « Cellular Mechanisms of Tissue Fibrosis. 4. Structural and functional consequences of skeletal muscle fibrosis ». American Journal of Physiology-Cell Physiology 305, no 3 (1 août 2013) : C241—C252. http://dx.doi.org/10.1152/ajpcell.00173.2013.
Texte intégralMoyer, Adam L., et Kathryn R. Wagner. « Regeneration versus fibrosis in skeletal muscle ». Current Opinion in Rheumatology 23, no 6 (novembre 2011) : 568–73. http://dx.doi.org/10.1097/bor.0b013e32834bac92.
Texte intégralLi, Zhao Bo, Helen D. Kollias et Kathryn R. Wagner. « Myostatin Directly Regulates Skeletal Muscle Fibrosis ». Journal of Biological Chemistry 283, no 28 (3 mai 2008) : 19371–78. http://dx.doi.org/10.1074/jbc.m802585200.
Texte intégralPidlisetskyy, Andriy, Serhii Savosko, Igor Gayovich, Oleksii Dolhopolov et Volodymyr Biliavskyi. « THE ULTRASONOGRAPHY EXAMINATION OF SKELETAL MUSCLES IN TRAUMATIC ISCHEMIA (EXPERIMENTAL STUDY) ». Wiadomości Lekarskie 76, no 1 (janvier 2023) : 175–81. http://dx.doi.org/10.36740/wlek202301124.
Texte intégralChen, Wan-Jing, I.-Hsuan Lin, Chien-Wei Lee et Yi-Fan Chen. « Aged Skeletal Muscle Retains the Ability to Remodel Extracellular Matrix for Degradation of Collagen Deposition after Muscle Injury ». International Journal of Molecular Sciences 22, no 4 (20 février 2021) : 2123. http://dx.doi.org/10.3390/ijms22042123.
Texte intégralTonogai, Ichiro, et Ichiro Tonogai. « Influence of Platelet Rich Plasma on the Skeletal Muscle Fibrosis after Limb Lengthening in Mice ». Foot & ; Ankle Orthopaedics 5, no 4 (1 octobre 2020) : 2473011420S0046. http://dx.doi.org/10.1177/2473011420s00468.
Texte intégralThèses sur le sujet "Skeletal muscle fibrosis"
Smith, Cheryl A. « Skeletal muscle injury, fibrosis and transforming growth factor-[beta] ». Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1744.
Texte intégralTitle from document title page. Document formatted into pages; contains xii, 146 p. : ill. (some col.). Includes abstract. Includes bibliographical references.
van, Erp Christel. « Modifying function and fibrosis of cardiac and skeletal muscle from mdx mice ». University of Southern Queensland, Faculty of Sciences, 2005. http://eprints.usq.edu.au/archive/00001521/.
Texte intégralPuliti, Elisa. « Role of sphingosine 1-phosphate metabolism and signalling in skeletal muscle atrophy and fibrosis ». Doctoral thesis, Università di Siena, 2022. http://hdl.handle.net/11365/1195603.
Texte intégralCai, Weisong, et 蔡蔚松. « Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B49618088.
Texte intégralpublished_or_final_version
Physiology
Master
Master of Philosophy
Lu, Lin, et 鹿琳. « The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208017.
Texte intégralpublished_or_final_version
Physiology
Doctoral
Doctor of Philosophy
Pinto, Priscilla Avelino Ferreira. « Treino de baixa intensidade retarda a deposi??o de fibras col?genas no m?sculo gastrocn?mio distr?fico de modelo mdx ». UFVJM, 2017. http://acervo.ufvjm.edu.br/jspui/handle/1/1608.
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Funda??o de Amparo ? Pesquisa do Estado de Minas Gerais (FAPEMIG)
Introdu??o: A Distrofia Muscular de Duchenne (DMD) ? marcada pela falta da distrofina, e sua aus?ncia leva a altera??es mec?nicas da fibra muscular, resultando num processo de necrose muscular e altera??es histol?gicas. O treinamento f?sico de baixa intensidade vem sendo empregado como forma de retardar a progress?o da DMD, entretanto, ainda n?o se sabe os par?metros ben?ficos ao m?sculo distr?fico. Assim, objetivou-se elucidar os efeitos do exerc?cio terap?utico de baixa intensidade em esteira no m?sculo esquel?tico distr?fico do modelo mdx nos par?metros morfol?gicos, na morfometria dos marcadores de les?o muscular, da fibrose musclar e na constitui??o da matriz extracelular pelos col?genos tipo I e III. Metodologia: Foram estudados tr?s grupos: camundongos mdx exerc?cio (mdxE), mdx sedent?rio (mdxC) e controle saud?veis (Cc) (n=8/grupo). O grupo mdxE foi estimulado a correr em esteira horizontal motorizada para ratos, em baixa intensidade, 9m/min por 30 minutos/dia, 3 vezes/semana, por 8 semanas. Ap?s o protocolo de exerc?cio foi realizada a eutan?sia dos animais e coletado o m?sculo gastrocn?mio. Foi realizada a colora??o Hematoxilina-Eosina para an?lise dos marcadores de les?o muscular: N?cleo Central e Di?metro M?nimo de Feret; a rea??o de Picrossirius red para an?lise das fibras col?genas na fibrose muscular e a imuno-localiza??o das fibras col?genas tipo I e III. Resultados: Foram observadas altera??es histol?gicas distr?ficas caracter?sticas nos grupos mdxE e mdxC e feixes de fibras col?genas espessas no grupo mdxC. A imuno-histoqu?mica revelou presen?a de col?geno do tipo I principalmente no grupo mdxC. N?o houve diferen?a significativa entre os grupos mdxE e mdxC para fibras com n?cleo central e coeficiente de varia??o do Di?metro m?nimo de Feret. O grupo mdxE n?o apresentou diferen?a significativa em rela??o ao Cc para a porcentagem da ?rea de fibras col?genas na fibrose muscular. Conclus?es: O treino de baixa intensidade reduz a deposi??o de fibras col?genas na fibrose muscular, com fibras delgadas do col?geno tipo I e n?o altera os marcadores de les?o muscular.
Disserta??o (Mestrado Profissional) ? Programa de P?s-Gradua??o em Reabilita??o e Desempenho Funcional, Universidade Federal dos Vales do Jequitinhonha e Mucuri, 2017.
Introduction: Duchenne muscular dystrophy (DMD) is marked by lack of dystrophin, and its absence leads to muscle fiber rupture, resulting in muscular necrosis and histological changes. Physical training of low intensity has been used to delay the progression of DMD, however, parameters of the dystrophic muscle are not yet known. The objective of this study was to elucidate the effects of a treadmill low intensity exercise on morphology, morphometric parameters of the muscle injury markers and the composition of the extracellular matrix by type I and III collagens of the dystrophic skeletal muscle of the mdx model. Methods: Three groups were studied: exercised mdx (mdxE), sedentary mdx (mdxC) and healthy controls (Cc) (n =8/group). The mdxE group was stimulated to run on a motorized horizontal treadmill for rats, with a low intensity (9 m/min for 30 minutes/day, 3 times/week) for 8 weeks. After the exercise protocol, the animals were euthanized and the gastrocnemius muscle was collected. Hematoxylin-Eosin staining was performed for analysis of muscle injury markers, Central Nucleus and Minimum Diameter of Feret, Picrossirius red reaction for analysis of collagen fibers in muscle fibrosis and immuno-localization of type I and III collagen fibers. Results: Dystrophic histopathological changes were observed in the mdxE and mdxC groups and thicker collagen fiber bundles in the mdxC group. Immunohistochemistry revealed the presence of type I collagen mainly in the mdxC group. There was no significant difference between the mdxE and mdxC groups for fibers with centrally located nuclei and coefficient of variation of the Minimum Feret Diameter. The mdxE group showed no significant difference in relation to Cc for the percentage of the area of collagen fibers in muscle fibrosis. Conclusions: The low intensity training reduced the deposition of collagen fibers in muscle fibrosis with thin fibers of type I collagen and did not alter the markers of muscle injury.
Hauck, James Spencer. « Mineralocorticoid Receptor Signaling in Acute and Chronic Muscle Injury ». The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1565089935933727.
Texte intégralNeves, Juliana de Carvalho. « Envolvimento da neuraminidase-1 na regeneração muscular ». Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-06052014-091743/.
Texte intégralNeuraminidase-1 (Neu1) participates in sialoglycoconjugates catabolism in lysosomes. Congenital Neu1 deficiency is the basis of sialidosis, a severe neurosomatic disorder associated with osteoskeletal deformities, hypotonia and muscle weakness. Mice with Neu1 deficiency (Neu1-/-) develop an atypical form of muscle degeneration characterized by abnormal fibroblast proliferation and expanded extracellular matrix (ECM), invasion of muscle fibers by fibroblast, cytosolic fragmentation, vacuolar formation and muscle atrophy. Despite muscle degeneration is well characterized in these animals, myogenesis has not been studied so far. The aim of this study was to evaluate the involvement of Neu1 in muscle regeneration process after cardiotoxin (CTX) injection in Neu1-/- mice and normal controls. CTX was applied in the right tibialis anterior muscle, and the animals were euthanized by cervical dislocation 1, 3, 5, 7, 10, 14, 21 and 28 days after injury. The muscles were analyzed through histology; cross-sectional area of regenerative muscle fibers; quantification of BrdU labeling; immunohistochemistry labelling for inflammation, regenerative fibers, and fibrosis; and gene and protein expression of muscle transcription factors. The data were compared and variances considered statistically significant in case p <= 0.05. In animals with Neu1 deficiency, both inflammatory process (mainly macrophagic response) and proliferative potential were increased in the initial stages, accompanied by overexpression of Pax7. We observed delay in muscle maturation characterized by higher expression of embryonic myosin later in muscle regeneration. MyoD and MyoG genes were overexpressed from 5 to 10 days after injury, though the expression of these proteins was reduced. At the end of muscle regeneration, reticulin deposition in ECM was increased, indicating fibrotic process. Neu1 seems to participate in all stages of muscle regeneration, since acute injury phase through the control of cell proliferation, towards muscle maturation, and at the final stages when it would regulate the deposition of ECM components
Ranasinghesagara, Janaka C. Yao Gang. « Optical reflectance in fibrous tissues and skeletal muscles ». Diss., Columbia, Mo. : University of Missouri--Columbia, 2008. http://hdl.handle.net/10355/6629.
Texte intégralHuber, Adrian Thomas. « Multi-organ non-invasive tissue characterization of fibrosis, adipose tissue, edema and inflammation with magnetic resonance (MR) imaging : applications to myocardium, skeletal muscle and liver interactions Cardiac MR strain : a noninvasive biomarker of fibro-fatty remodeling of the left atrial myocardium Comparison of MR T1 and T2 mapping parameters to characterize myocardial and skeletal muscle involvement in systemic Idiopathic Inflammatory Myopathy (IIM) Non-invasive differentiation of acute viral myocarditis and idiopathic inflammatory myopathy with cardiac involvement using magnetic resonance imaging T1 and T2 mapping CT predicts liver fibrosis : Prospective evaluation of morphology- and attenuationbased quantitative scores in routine portal venous abdominal scans ». Thesis, Sorbonne université, 2019. http://www.theses.fr/2019SORUS135.
Texte intégralThis thesis provides a proof of concept for MR atrial strain, as well as MR relaxometry in the myocardium, in skeletal muscles and in the liver. Thanks to a close interaction between radiologist and software engineers, two different softwares were developed, applied and validated: one for multiorgan T1 mapping in the myocardium, skeletal muscle and liver, another one for cardiac four-chamber strain analysis and volumetry. The first publication showed a strong correlation of LA strain with the degree of fibro-fatty replacement in histology. Such functional imaging biomarker in combination with LA volumetry could help to guide clinical decisions, since myocardial structural remodeling is a known morphologic substrate of LA dysfunction, atrial fibrillation and adverse outcome. In the second publication, MR relaxometry parameters applied to the myocardium and skeletal muscles in IIM patients and healthy volunteers were used as a model to demonstrate influences of different tissue composition and vascularization on T1 mapping parameters. ΔT1 and EHF were introduced as simple alternatives to ECV in highly vascularized tissues such as the myocardium. In the third publication, MR relaxometry parameters applied to the skeletal muscls allowed for an accurate discrimination of AVM and IIM with cardiac involvement. However, when applied to the myocardium, parametric mapping did not separate between the two groups. The fourth publication introduced native T1 of the liver an easily accessible and accurate non-invasive imaging associate of congestive HF in IDCM patients with better performance than established functional parameters such as LV volumes, ejection fraction or strain
Chapitres de livres sur le sujet "Skeletal muscle fibrosis"
Pessina, Patrizia, et Pura Muñoz-Cánoves. « Fibrosis-Inducing Strategies in Regenerating Dystrophic and Normal Skeletal Muscle ». Dans Methods in Molecular Biology, 73–82. New York, NY : Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3810-0_7.
Texte intégralElhussieny, Ahmed, Ken’ichiro Nogami, Fusako Sakai-Takemura, Yusuke Maruyama, AbdElraouf Omar Abdelbakey, Wael Abou El-kheir, Shin’ichi Takeda et Yuko Miyagoe-Suzuki. « Mesenchymal Stem Cells for Regenerative Medicine for Duchenne Muscular Dystrophy ». Dans Muscular Dystrophy - Research Updates and Therapeutic Strategies. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.92824.
Texte intégralLambrechts, Mark. « Musculoskeletal Abnormalities Caused by Cystic Fibrosis ». Dans Advances in Skeletal Muscle Health and Disease [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104591.
Texte intégralLambrechts, Mark. « Musculoskeletal Abnormalities Caused by Cystic Fibrosis ». Dans Advances in Skeletal Muscle Health and Disease [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104591.
Texte intégralSerrano, Antonio L., Christopher J. Mann, Berta Vidal, Esther Ardite, Eusebio Perdiguero et Pura Muñoz-Cánoves. « Cellular and Molecular Mechanisms Regulating Fibrosis in Skeletal Muscle Repair and Disease ». Dans Current Topics in Developmental Biology, 167–201. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-385940-2.00007-3.
Texte intégralBundgaard, Henning, Anna Axelsson, Alex Christensen et Helle Petri. « The heart in neuromuscular disease : myotonic dystrophy ». Dans ESC CardioMed, 1530–34. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0370.
Texte intégralAtkinson, Martin E. « Introduction and surface anatomy ». Dans Anatomy for Dental Students. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199234462.003.0029.
Texte intégralShpadaruk, Volha, et Karen E. Harman. « Cutaneous vasculitis, connective tissue diseases, and urticaria ». Dans Oxford Textbook of Medicine, sous la direction de Roderick J. Hay, 5639–76. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0556.
Texte intégralActes de conférences sur le sujet "Skeletal muscle fibrosis"
Rozenberg, D., M. Sussman, R. G. L. Koh, S. Nourouzpour, L. Wickerson, L. G. Singer, S. Shapera et al. « Skeletal Muscle Size and Fat Infiltration of the Limb Muscles in Idiopathic Pulmonary Fibrosis ». Dans American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3179.
Texte intégralBalañá, Ana, Juana Martínez-Llorens, Diego Agustin Rodríguez, Mireia Admetlló, Anna Salazar, Pilar Ausin, Esther Barreiro et Joaquin Gea. « Skeletal muscle function and structure in patients with non-cystic fibrosis bronchiectasis ». Dans ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.oa265.
Texte intégralVerges, Samuel, Nicolas Decorte, Mathieu Gruet, Boubou Camara, Sébastien Quetant, Laurent Mely, Jean-Marc Vallier et Bernard Wuyam. « Skeletal muscle metabolism in active cystic fibrosis (CF) patients with light/moderate pulmonary dysfunction ». Dans Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2242.
Texte intégralWang, Xuejie, Ana Balañá Corberó, Juana Martínez Llorens, Liyun Qin, Mireia Admetlló, Esmeralda Hernández Leal, Xavier Duran, Antonio Sancho Muñoz et Esther Barreiro Portela. « Skeletal Muscle Dysfunction and Body Composition Alterations in Non-Cystic Fibrosis Bronchiectasis Patients : Gender Differences ». Dans ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1836.
Texte intégralHuang, Alice H., Spencer S. Watson et Ronen Schweitzer. « Lineage Tracing Reveals a New Model for Tendon Growth and Elongation During Development ». Dans ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80915.
Texte intégralUslu, Nazlı Zeynep, Derya Kocakaya, Sehnaz Olgun Yıldızeli, Emel Eryüksel, Özge Keniş Coşkun, Canan Cimşit, Şeyma Görçin Karaketir et Berrin Ceyhan. « Does cystic fibrosis impact skeletal muscles and diaphragm function ? » Dans ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa343.
Texte intégralMarillier, Mathieu, Anne-Catherine Bernard, Onofre Moran-Mendoza, Denis E. O'Donnell, Samuel Verges et J. Alberto Neder. « Beyond the lungs in fibrotic interstitial lung disease : does supplemental O2 improve skeletal muscle oxygenation and fatigue ? » Dans ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.4405.
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