Bibliographies thématiques / Rheumatoid arthritis

Littérature scientifique sur le sujet « Rheumatoid arthritis »

Auteur : Grafiati
Publié le 4 juin 2021
Mis à jour le 7 décembre 2023

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Articles de revues sur le sujet "Rheumatoid arthritis"

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Genth, E. « Rheumatoide Arthritis. Rheumatoid Arthritis ». Laboratoriums Medizin 26, no 3-4 (avril 2002) : 130–36. http://dx.doi.org/10.1046/j.1439-0477.2002.02025.x.

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Genth, E. « Rheumatoide Arthritis/Rheumatoid Arthritis ». LaboratoriumsMedizin 26, no 3/4 (1 janvier 2002) : 130–36. http://dx.doi.org/10.1515/labmed.2002.018.

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Kazmi, Zehra. « The Influence of Early Menopause in Women with Rheumatoid Arthritis ; A Systematic Review ». Healer Journal of Physiotherapy and Rehabilitation Sciences 1, no 1 (30 juin 2021) : 27–37. http://dx.doi.org/10.55735/thjprs.v1i1.22.

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Abstract Background: Rheumatoid arthritis is a chronic disease affecting the immune system and it particularly targets synovial joints, bringing about inflammation and causing functional limitations. Objective: To explore the evidence showing the influence of early menopause in women with rheumatoid arthritis. Methods: In this systematic review, all the literature from 2010 to 2019 was searched with specific keywords “menopause, premature menopause, early menopause, rheumatoid arthritis, rheumatic diseases” with the use of Boolean operators. Databases that were thoroughly searched included Google Scholar, PubMed and HEC digital library. After careful screening, 10 studies that met the inclusion criteria were included in the review. The quality of these articles was assessed using the AXIS tool for that cross-sectional studies. Results: These studies indicated that post-menopausal women are at higher risk of developing rheumatoid arthritis and they are more likely to have early or premature menopause (in ≤45 years). At baseline, the post-menopausal women had high health assessment questionnaire scores than pre-menopausal women. Conclusion: This review states that menopausal women are highly at risk of developing rheumatoid arthritis, especially those experiencing this at an early age and this condition can also worsen arthritic symptoms. Hormonal changes may influence pathways that are distinct from those leading to severe and progressive rheumatoid arthritis. *Corresponding author: Zehra Kazmi, University Institute of Physical Therapy, University of Lahore, Lahore, Pakistan; Email: zaza.kazmi110@gmail.com Citation: Kazmi Z. 2021. The influence of early menopause in women with rheumatoid arthritis; a systematic review. The Healer Journal of Physiotherapy and Rehabilitation Sciences, 1(1):27-37.
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Wekoadi, Galih Mahendra, et Budi Widiyanto. « Management of Non Pharmacological Pain in Clients With Rheumathoid Arthritis : A Literature Review ». Journal of Applied Health Management and Technology 2, no 3 (31 juillet 2020) : 104–15. http://dx.doi.org/10.31983/jahmt.v2i3.5725.

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Rheumathoid Arthritis is a disease that occurs due to abnormalities in the joints, causing pain and stiffness in the musculoskeletal system such as skeletal muscles, joints, ligaments and bones and generally attacks clients over the age of 40 years (Syapitri, 2018). Rheumatoid Arthritis has typical symptoms in sufferers, one of which is pain. If not handled properly, pain in rheumathoid arthritis can disrupt the activities of sufferers, paralysis and impaired balance. Therefore, pain in reumathoid arthritis must be treated. The aim of writing this literature review is to provide information about the non-pharmacological management of pain in clients with rheumatoid arthritis. The method used in this study is to use the Literature review method. Literature review is carried out by analyzing articles relevant to the title taken, namely the management of non-pharmacological pain in clients with Rheumathoid arthritis. The stages in conducting a Literature review are based on 5 stages of literature review that cover the eligibility criteria, information sources, selection of literature, data collection and selection of data items. With pain rheumatic arthritis include: back massage, cupping, benson relaxation and warm compresses, cinnamon compresses, giving of the gnome, ginger compresses, lemongrass compresses, Swedish massases, giving keroncong music, grated ginger grater, tai chi exercise, and yoga interventions.
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Tri Novana, Vanesa, Firman Faradisi et Nuniek Nizmah Fajriyah. « Pengaruh Senam Rematik Terhadap Penurunan Nyeri Pada Pasien Rheumatoid Arthritis ». Prosiding Seminar Nasional Kesehatan 1 (23 décembre 2021) : 2084–89. http://dx.doi.org/10.48144/prosiding.v1i.977.

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Abstract Rheumatoid arthritis is an autoimmune disease when a person’s immune system attacks the body’s cells. Signs and symptoms of rheumatoid arthritis include joint inflammation and joint deformity. In most cases, patients with rheumatoid arthritis experience joint pain. Rheumatic Gymnastics is an alternative therapy that has been proven to reduce joint pain in rheumatic patients. The purpose of this case study is to describe the use of rheumatic exercise therapy in arthritic patients. The purpose of this case study is to examine therapeutic gymnastic in reducing pain among patients with rheumatoid arthritis. Two patients were taught to exercise therapeutic gymnastic. The research instrument is a pain scale observation sheet (Numerical Rating Scale). Two patients reported that there was a decreasing intensity of joint pain after doing exercise. This study concludes this particular exercise may reduce joint pain. Nurses are suggested to implement therapeutic gymnastics exercise in reducing pain among patients with Rheumatoid arthritis.Keywords : rheumatoid arthritis; pain; therapeutic gymnastics exercise Abstrak Rematik merupakan penyakit auto imun ketika sistem imun pada tubuh seseorang menyerang sel-sel tubuhnya sendiri. Gejala rematik yaitu inflamasi, deformitas, dan nyeri sendi yang paling dirasakan oleh penderita rematik. Senam Rematik merupakan terapi alternative yang sudah terbukti dapat menurunkan nyeri sendi pada pasien rematik. Tujuan studi kasus ini adalah untuk menggambarkan penggunaan terapi senam rematik pada pasien rematik. Metode yang digunakan adalah asuhan keperawatan dengan menerapkan terapi senam rematik. Instrumen penelitian berupa lembar observasi skala nyeri (Numerical Rating Scale). Hasil yang didapatkan pada klien 1 maupun 2 yaitu mengalami penurunan nyeri. Kesimpulan pada studi kasus ini bahwa senam rematik dapat menurunkan skala nyeri. Saran bagi perawat diharapkan dapat menerapkan tindakan senam rematik untuk menurunkan skala nyeri pada pasien rematik.Kata kunci : Rematik, nyeri, senam rematik
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Wulandari, Sri, Warda Anil Masyayih, Rista Dian Anggraini et Hany Puspita Aryani. « Hubungan Rheumatoid Arthritis dengan Kejadian Insomnia pada Usia Lanjut ». Journal of Holistics and Health Science 5, no 1 (9 mars 2023) : 45–54. http://dx.doi.org/10.35473/jhhs.v5i1.256.

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One of the health problems often experienced by the elderly is rheumatoid arthritis. Joint disorders experienced by people with rheumatoid arthritis will have an impact on physical problems such as insomnia and will later affect sleep quality. Design correlational analytical research with a cross-sectional approach. The sample was all elderly people with rheumatoid arthritis as many as 30 respondents using the smapling purposive sampling technique. Data collection using KSPBJ insomnia rating scale questionnaires. Analyse data with Spearman’s Rho test with 0.05 significance. The results of the study were known that in respondents who experienced rheumatoid arthritis with severe pain all experienced severe insomnia, which was 13.3%. Meanwhile, in respondents who experienced rheumatoid arthritis with mild pain, all experienced mild insomnia, which was 36.7%. The results of the data analysis with the Spearman Rho test found that there was a relationship between rheumatoid arthritis and insomnia in old age in Kreteranggon Village, Sambeng District, Lamongan Regency (ρ =0,000, α=0.05, r=0.699). Based on the description above, it can be concluded that there is a relationship in the strong category between rheumathoid arthritis and the incidence of insomnia. ABSTRAK Salah satu masalah kesehatan yang sering dialami oleh lansia adalah rheumatoid arthritis. Gangguan sendi yang dialami penderita rheumatoid arthritis akan berdampak pada masalah fisik seperti insomnia dan nantinya akan berpengaruh pada kualitas tidur. Rancangan penelitian analitik korelasional dengan pendekatan cross sectional. Sampelnya semua lansia penderita penyakit rheumatoid arthritis sebanyak 30 responden dengan menggunakan teknik sampling purposive sampling. Pengumpulan data menggunakan kuesioner KSPBJ insomnia rating scale. Analisa data dengan uji Spearman’s Rho dengan signifikasi 0,05. Hasil Penelitian diketahui bahwa pada responden yang mengalami rheumatoid arthritis dengan nyeri berat seluruhnya mengalami insomnia berat yaitu sebanyak 13,3%. Sedangkan pada responden yang mengalami rheumatoid arthritis dengan nyeri ringan seluruhnya mengalami insomnia ringan yaitu sebanyak 36,7%. Hasil analisa data dengan uji Spearman Rho didapatkan ada hubungan rheumatoid arthritis dengan kejadian insomnia pada usia lanjut di Desa Kreteranggon Kecamatan Sambeng Kabupaten Lamongan ( ρ =0,000, α=0,05, r=0,699). Berdasarkan uraian diatas dapat disimpulkan bahwa Ada hubungan dalam kategori kuat antara rheumathoid Arthritis dengan kejadian insomnia.
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Chaudhary, Archana, et Pandit Vinay. « Rheumatoid Arthritis : Etiology, Treatment and Animal Models ». Journal of Drug Delivery and Therapeutics 10, no 5-s (15 octobre 2020) : 290–98. http://dx.doi.org/10.22270/jddt.v10i5-s.4357.

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Rheumatoid arthritis is an autoimmune disease that can cause joint pain and damage throughout your body. About 75% of Rheumatoid arthritis patients are women. In fact, 1 – 3% of women may get rheumatoid arthritis in their lifetime. The disease most often begins between the ages of 30 and 50. Rheumatoid arthritis occurs when your immune system attacks the synovium, the lining of the membranes that surround your joints. The resulting inflammation thickens the synovium, which can eventually destroy the cartilage and bone within the joint. The tendons and ligaments that hold the joint together weaken and stretch. Gradually, the joint loses its shape and alignment. It also affects other organs of body like: skin, eyes, heart, kidneys, and lungs. The main risk factors that cause Rheumatoid arthritis are Age, Gender, Genetics, weight, smoking, diet, etc. Three main ways to treat rheumatoid arthritis are Drugs, physical therapies and surgery. There are four main groups of drugs that are used to treat rheumatoid arthritis are non-steroidal anti-inflammatory drugs , disease-modifying anti-rheumatic drugs and steroids (also known as corticosteroids). Collagen induced arthritis and Adjuvant arthritis are the most commonly used standard animal models in Rheumatoid arthritis. This literature review assessed the sign & symptoms, risk factors, etiology, treatment and standard animal models for Rheumatoid arthritis. Keywords: Rheumatoid arthritis, Inflammation, Antirheumatic drugs, Adjuvants, Rat, Mice.
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Konyshevskaya, A. A., N. V. Vaizer et L. P. Shelestova. « Clinical characteristic of juvenile rheumatoid arthritis debut and course ». Likarska sprava, no 5-6 (30 septembre 2017) : 143–49. http://dx.doi.org/10.31640/ls-2017(5-6)26.

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The article is devoted to juvenile rheumatoid arthritis – one of the most common rheumatic diseases with children. High activity and aggressive course of juvenile rheumatoid arthritis are common with children with systemic and polyarticular disease variants debut. The article presents case studies of children with systemic and polyarticular variants debut of juvenile rheumatoid arthritis.
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Dariushnejad, Hassan, Leila Chodari, Mehrnoosh Sedighi, Soheila Akbari et Vajihe Ghorbanzadeh. « Rheumatoid arthritis : current therapeutics compendium ». Endocrine Regulations 56, no 2 (1 avril 2022) : 148–62. http://dx.doi.org/10.2478/enr-2022-0016.

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Abstract Rheumatoid arthritis is a common chronic inflammatory disease with substantial economic, social, and personal costs. Its pathogenesis is multifactorial and complex. The ultimate goal of rheumatoid arthritis treatment is stopping or slowing down the disease progression. In the past two decades, invention of new medicines, especially biologic agents, revolutionized the management of this disease. These agents have been associated with an improved prognosis and clinical remission, especially in patients who did not respond to traditional disease-modifying anti-rheumatic drugs (DMARDs). Improvement in the understanding of the rheumatoid arthritis pathogenesis leads to the development of novel biologic therapeutic approaches. In the present paper, we summarized the current therapeutics, especially biologic agents, available for the treatment of rheumatoid arthritis.
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Tkachenko, L. A., U. A. Kostrikova, T. I. Yarmola, G. L. Pustovoit et V. V. Talash. « KIDNEY DAMAGE IN PATIENTS WITH RHEUMATOID ARTHRITIS ». Актуальні проблеми сучасної медицини : Вісник Української медичної стоматологічної академії 19, no 2 (19 juillet 2019) : 246–50. http://dx.doi.org/10.31718/2077-1096.19.2.246.

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The purpose of this work is to perform a general analysis of relevant literature on the issue of kidney damage in patients with rheumatoid arthritis. Kidney damage in patients with rheumatic diseases is potentially dangerous, as it can lead to the development of terminal renal failure that may require replacement renal therapy. Amyloidosis often leads to kidney failure in patients with rheumatoid arthritis. Renal amyloidosis more often develops in patients with acute course of rheumatoid arthritis and under maximal immunological disorders. In patients with renal amyloidosis against the background of rheumatoid arthritis, manifestations of joint affection decrease, while the renal-uremic syndrome takes a predominant role. Signs of nephrotic syndrome and chronic renal failure develop gradually. Kidney damage can be caused by medications for rheumatoid arthritis. The choice of the optimal scheme of individual-centred therapy is vitally important for patients, since every aggravation of both rheumatic disease and secondary renal damage leads to the progression of chronic renal failure.
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Thèses sur le sujet "Rheumatoid arthritis"

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Ulfgren, Ann-Kristin. « Cytokines in rheumatoid arthritis / ». Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-3823-7/.

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DeLaura, Angela. « Rheumatoid arthritis : an overview / ». Online version of thesis, 1989. http://hdl.handle.net/1850/11502.

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Thomson, W. « Immunogenetics of rheumatoid arthritis ». Thesis, University of Manchester, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383908.

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Kalla, Asgar Ali. « Osteoporosis in rheumatoid arthritis ». Master's thesis, University of Cape Town, 1989. http://hdl.handle.net/11427/26297.

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The literature is replete with reports of osteoporosis in rheumatoid arthritis, but the mechanism of bone loss remains obscure. This is probably due to the overlap with bone loss of aging and the menopause, whose exact mechanisms are also poorly understood. Against this background, a study was designed to evaluate generalised bone loss in young, premenopausal (if female), patients with rheumatoid arthritis. The protocol was designed to record demographic data, as well as information pertaining to the disease. Cortical bone mass was measured at the metacarpals and left femur, using an automated, computer-controlled technique. Trabecular bone was evaluated at the left femur (Singh index) as well as at the 3rd lumbar vertebra (Saville index). Bone kinetics were studied by the measurement of urinary excretion of calcium, phosphate and hydroxy-praline (resorption) and serum alkaline phosphatase (formation). Disease activity was measured clinically and with laboratory indices. Physical activity was indirectly measured by quantitating the disability, using the Keitel function test as well as a modified health assessment questionnaire (HAQ). The radiograph of the right wrist was scored by the Larsen index. The carpometacarpal ratio was also calculated from the radiograph. Numerous statistical techniques were applied in the analysis of the data. Healthy volunteers were used as controls. Patients with SLE were also studied, in order to compare the 2 inflammatory diseases. Patients with RA had generalised cortical bone loss (metacarpal and femur) (p < 0.001). Trabecular bone measurements were not significantly different from normals, using the crude radiographic techniques. Duration of disease was the most important clinical determinant of this bone loss. The relative contributions of disease activity and lack of physical activity to the loss of bone could not be adequately separated using conventional statistical techniques. Corticosteroid therapy did not promote metacarpal bone loss in these subjects, but may have contributed to thinning of the femoral cortex. Nonsteroidal anti-inflammatory drugs and disease modifying agents did not seem to influence the extent of the bone loss. Nutritional status and skinfold thickness did not correlate with bone mass. Dietary factors played no role in the genesis of bone loss, but may have had some effect on disease activity. Metacarpal measurements showed a sensitivity of 80% and specificity of 85% in discriminating between osteopaenic and normopaenic groups with RA. Osteopaenia could not be adequately predicted in the absence of metacarpal measurements. Metacarpal bone loss in RA was due to endosteal resorption, while in SLE it was due to periosteal resorption. The semi-automatic technique for measurement of metacarpal bone mass showed good reproducibility among 5 observers and at 2 different centres. The pathogenesis of bone loss in RA was multifactorial, the largest contribution probably coming from a humoral factor in the circulation, closely related to disease activity. Ionised calcium was elevated in 55% of RA patients, but only 5% of SLE patients. Serum PTH levels were normal in 99% of the RA subjects. Elevations in alkaline phosphatase. (25%) probably reflected disease activity rather than increased bone formation. Factor analysis of 27 variables showed that disease activity was central to the development of OP in RA. CS therapy tended to be used in the presence of active disease. Disability was not an important determinant of bone loss in RA, but may be a useful measure of activity of the disease. This study did not evaluate the relationships with sex hormonal status or vitamin D metabolism. Future research should aim at cohort analysis at 2 different periods, in order to improve our understanding of the pathogenesis of bone loss in RA.
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Toms, Tracey. « Dyslipidaemia in rheumatoid arthritis ». Thesis, University of Manchester, 2012. https://www.research.manchester.ac.uk/portal/en/theses/dyslipidaemia-in-rheumatoid-arthritis(e7808bd7-52e6-40a0-84cb-e4aadbf7505c).html.

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Introduction: Rheumatoid arthritis (RA) is known to be associated with an increased risk of cardiovascular disease (CVD), resulting in excess mortality. Dyslipidaemia has been identified as a major CVD risk factor in the general population. Current evidence would suggest that lipid metabolism is altered in RA due to inflammation, and that use of anti-inflammatory therapy may reverse some of these changes. However, the impact of such lipid changes on CVD in RA remains unknown. Data regarding the effects of RA/drug therapy on lipid structure and function are sparse. Genetic factors are important in the pathogenesis of RA and play a central role in the regulation of lipid metabolism. To date, no studies have assessed the impact of genetic polymorphisms on lipids in RA.The aim of this thesis is to: 1) assess the prevalence of dyslipidaemia in RA and the CVD risk this confers 2) establish the effects of inflammation on lipid levels and lipid ratios 3) assess the impact of anti-inflammatory drug therapy (anti-TNF, rituximab and intravenous glucocorticoids) on lipid levels, structure and function 4) assess the prevalence and associations of particular genetic polymorphisms (RA susceptibility and lipid metabolism regulating genes) with lipids in RA.Methods: Data from 400 RA patients were used to address aims 1, 2 and 4 in cross-sectional studies. All patients had a clinical assessment and fasting blood taken. Blood was processed to provide data on the lipid profile, ESR, CRP and DNA was extracted for genotyping. Aim 2 and 4 also utilised a retrospective longitudinal cohort of 550 RA patients and the DNA from 400 healthy controls, respectively. Aim 3 was addressed using a longitudinal cohort including: patients due to commence anti-TNF (n=35), rituximab (n=10), intravenous glucocorticoids (n=12); 15 RA controls on stable therapy; and 40 healthy controls. Assessments and blood samples were taken at baseline, 2 weeks and 3 months. Results: Dyslipidaemia was highly prevalent (56.8%), but undertreated in many RA patients at risk of developing CVD. Systemic inflammation associated with many of the changes in lipid levels and structure. Lipid ratios were found to be less susceptible to fluctuations due to inflammation. The use of anti-inflammatory drug therapy produced changes in lipid structure and function through both generic suppression of inflammation and drug specific mechanisms (particularly in the case of glucocorticoids). The prevalence of cholesterol ester transfer protein (CETP) and Apolipoprotein C3 genetic polymorphisms differed between RA patients and local population controls. RA susceptibility genes (HLA-DRB1-SE and TRAF1C5) and several ’lipid metabolism genes’ (Apolipoprotein E, ATP-binding cassette transporter 1 (ABCA1) and CETP) were found to associate with lipid levels within the RA population. Conclusion: Dyslipidaemia is highly prevalent in RA and currently undertreated. Dyslipidaemia in RA is regulated by numerous factors including inflammation, drug therapy and genetic factors. Further longitudinal studies are required to assess whether these findings have an impact on hard CVD endpoints.
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Vingsbo, Lundberg Carina. « Chronic autoimmune arthritis in rats pathogenesis and genetic factors / ». Lund : Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/68945081.html.

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Riding, S. Barbara. « The arthritic pain experience of children with juvenile rheumatoid arthritis ». Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/27731.

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This study was designed to investigate the experience of having arthritic pain from the children's perspective. Previous research on how Canadian children perceive and manage arthritic pain and how it affects their daily lives is nonexistent. Therefore the purpose of this qualitative descriptive study was to explore and describe the arthritic pain experience of school age children with juvenile rheumatoid arthritis (JRA) and to understand the impact/influence of various factors on the construction of that experience. Ten children, aged 10 to 13 years, with either early (at 2 to 4 years) or late (at 7 to 11 years) onset arthritis participated in this study. Descriptive data were obtained during two open-ended in depth interviews with the children in their homes. Using content analysis, data were analyzed for themes and their elements. An analytical framework of themes and their elements was developed that reflected the children's descriptions of and explanations for arthritic pain in the context of their day to day in the context of their day to day living with arthritis, both in the past and currently. The children perceived pain to be synonymous with arthritis and the mediating factor in how they functioned. They described arthritic pain in relation to distinguishing factors: intensity, duration, and frequency. Intermittent arthritic pain was attributed to cessation of medications, arthritis "flare-ups," inactivity, and activity. A current concern for most children was pain attributed to activity because it meant limitations in activities with peers. The children identified strategies they used to manage pain and cope with pain's unpredictability. The findings of this study were discussed in relation to selected research studies that either supported or refuted the findings of this study. Implications for nursing practice and research were addressed.
Applied Science, Faculty of
Nursing, School of
Graduate
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Gutowska-Owsiak, Danuta. « NKT Cells in Rheumatoid Arthritis ». Thesis, University of Liverpool, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526938.

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Eurenius, Eva. « Physical activity in rheumatoid arthritis / ». Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-697-2/.

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Reynolds, Sophie L. « Vascular dysfunction in rheumatoid arthritis ». Thesis, Cardiff University, 2010. http://orca.cf.ac.uk/54162/.

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These findings suggest that systemic and vascular wall levels of matrix metalloproteinase-9, related to inflammation at the joint site, may play a prominent role in the development of vascular dysfunction in this experimental model. This thesis goes someway to elucidating the potential mechanisms of vascular dysfunction in rheumatoid arthritis.
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Livres sur le sujet "Rheumatoid arthritis"

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D, Harris Edward. Rheumatoid arthritis. Philadelphia : Saunders, 1997.

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Rothermich, Norman O. Rheumatoid arthritis. Orlando : Grune & Stratton, 1985.

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Liu, Shuang, dir. Rheumatoid Arthritis. New York, NY : Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8802-0.

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Smolen, Josef S., Joachim R. Kalden et Ravinder N. Maini, dir. Rheumatoid Arthritis. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8.

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Luqmani, Raashid. Rheumatoid arthritis. Oxford : Oxford University Press, 2010.

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C, Hochberg Marc, dir. Rheumatoid arthritis. Philadelphia : Mosby/Elsevier, 2009.

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B, Natvig Jacob, et Winchester R, dir. Rheumatoid arthritis. Heidelberg, Federal Republic of Germany : Springer International, 1988.

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National Institute of Arthritis and Musculoskeletal and Skin Diseases (U.S.). Rheumatoid arthritis. Bethesda, Md.] : Dept. of Health and Human Services, National Institututes of Health, National Institute of Arthritis and Musculoskeletal and Skin Diseases, 2009.

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Moreland, Larry W. Rheumatoid arthritis. Philadelphia, Pa : Lippincott, Williams & Wilkins, 2006.

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Moreland, Larry W. Rheumatoid arthritis. 2e éd. Chicago : Remedica, 2008.

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Chapitres de livres sur le sujet "Rheumatoid arthritis"

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Silman, A. J. « Is Rheumatoid Arthritis a Disappearing Disease ? » Dans Rheumatoid Arthritis, 3–13. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_1.

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Natvig, J. B., I. Randen, K. Thompson et Ø. Førre. « The Genetics of Rheumatoid Factors (RF) : The V-gene Repertoire of RFs in Rheumatoid Arthritis as Analyzed by Hybridoma Clones ». Dans Rheumatoid Arthritis, 137–49. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_10.

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Youinou, P., A. Lamour, A. Dumay et P. Le Goff. « Neutrophil Polymorphonuclear Cell Function in Rheumatoid Arthritis ». Dans Rheumatoid Arthritis, 150–63. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_11.

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van Eden, W., C. J. P. Boog, E. J. M. Hogervorst, M. H. M. Wauben, R. van der Zee et J. D. A. van Embden. « Heat-Shock Proteins and Mycobacterial Antigens ». Dans Rheumatoid Arthritis, 167–79. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_12.

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Holmdahl, R. « Type-II Collagen in the Pathogenesis of Rheumatoid Arthritis ». Dans Rheumatoid Arthritis, 180–201. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_13.

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Crofford, L. J., et R. L. Wilder. « Streptococcal Cell Wall Antigens and Rheumatoid Arthritis ». Dans Rheumatoid Arthritis, 202–15. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_14.

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Peterhans, E., B. Pohl, R. Zanoni et S. Lazary. « Caprine Arthritis-Encephalitis ». Dans Rheumatoid Arthritis, 216–30. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_15.

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Stransky, G., R. E. Gay, A. Trabandt, W. K. Aicher, S. R. Barnum et S. Gay. « Oncogenes and Retroviruses in Rheumatoid Arthritis ». Dans Rheumatoid Arthritis, 231–43. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_16.

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Krapf, E. F. « Are Retroviruses Involved in the Pathogenesis of Autoimmune Diseases ? » Dans Rheumatoid Arthritis, 244–56. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_17.

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Lotz, M., et J. Roudier. « Epstein-Barr Virus and Rheumatoid Arthritis ». Dans Rheumatoid Arthritis, 257–80. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76189-8_18.

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Actes de conférences sur le sujet "Rheumatoid arthritis"

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YAMAUCHI, Morio, Kazuhisa NAKANO, Yoshiya TANAKA et Keiichi HORIO. « Predicting Disease Activity for Biologic Selection in Rheumatoid Arthritis ». Dans 9th International Conference on Signal, Image Processing and Pattern Recognition (SPPR 2020). AIRCC Publishing Corporation, 2020. http://dx.doi.org/10.5121/csit.2020.101913.

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In this article, we implemented a regression model and conducted experiments for predicting disease activity using data from 1929 rheumatoid arthritis patients to assist in the selection of biologics for rheumatoid arthritis. On modelling, the missing variables in the data were completed by three different methods, mean value, self-organizing map and random value. Experimental results showed that the prediction error of the regression model was large regardless of the missing completion method, making it difficult to predict the prognosis of rheumatoid arthritis patients.
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SUN, YANG-BO, et ZHI-LI ZENG. « BIBLIOMETRIC ANALYSIS OF METHOTREXATE IN THE TREATMENT OF RHEUMATOID ARTHRITIS ». Dans 2021 International Conference on Education, Humanity and Language, Art. Destech Publications, Inc., 2021. http://dx.doi.org/10.12783/dtssehs/ehla2021/35713.

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[Objective] To analyze the growth rule of literature, core authors, core institutions and Co-word Analysis in the field of methotrexate in the treatment of rheumatoid arthritis, to draw the knowledge map of key words and the cooperation network of authors, and to get the current frontiers and research hotspots through the research and analysis of these indicators, so as to provide reference for future researchers to choose research directions. [Methods] The three databases including CNKI, Wanfangdata and Cqvip were used as retrieval databases, and the key words of "rheumatoid arthritis" and "methotrexate" and their synonyms were used as retrieval keywords. The literature published in the three databases from 2009 to 2018 were retrieved and analyzed by bibliometrics analysis method. [Result] 2404 valid literature were screened out. The growth curve of literature showed a relatively stable upward trend, and formed a stable core group of high-yield authors. The main direction of research in this field in the past ten years was the effectiveness analysis of methotrexate combined with drug treatment for rheumatoid arthritis. [Conclusion] Domestic research on methotrexate in the treatment of rheumatoid arthritis will continue to be the focus of medical research in the next few years. Rheumatoid arthritis (RA) is a chronic systemic immune disease. Its early manifestations are mainly joint pain and dysfunction. It can lead to joint function loss and accompanied by atrophy of bone and skeletal muscle. It has a high disability rate and seriously threatens human physical and mental health [1].The incidence of rheumatoid arthritis was higher in women than in men [2]. According to statistics, the prevalence of rheumatoid arthritis in China is between 0.32% and 0.36% [3]. Therefore, searching for drugs with less side effects and obvious effects is a research hotspot in the medical field [4]. Methotrexate, as a gold standard antirheumatic drug, has been widely used in combination therapy and randomized controlled clinical trials [5]. In order to clearly understand the research and development trend of methotrexate in the field of rheumatoid arthritis, this paper makes bibliometric analysis of the relevant literature published in this field in the past 10 years, and provides reference for the future research and development of this field with objective and real data.
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Kummerfeld, Sarah K., Jason A. Hackney, Michael J. Townsend et Hilary F. Clark. « Rheumatoid Arthritis molecular heterogeneity ». Dans the First ACM International Conference. New York, New York, USA : ACM Press, 2010. http://dx.doi.org/10.1145/1854776.1854907.

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Diep, Phuong Phuong, et Constance de Koning. « Rheumatoid arthritis-associated ILD ». Dans Nordic Lung Congress 2022, sous la direction de Phuong Phuong Diep et Vibeke Backer. Baarn, the Netherlands : Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/20fe5496.

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Reis Gama, Cipriano, Heitor Furlan Giordano, Victor Matheus Ostrovski Souza Santos, Ana Cristina de Medeiros Ribeiro et Karina Rossi Bonfiglioli. « Arthritis by Paracoccidioides Brasiliensis in rheumatoid arthritis ». Dans SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1930.

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Kaurova, A. V., I. V. Puhteeva, L. A. Malkevich et N. V. Gerasimovich. « ANALYSIS OF CALCIUM IONS IN THE CYTOPLASM OF PERIPHERAL BLOOD LYMPHOCYTES OF PATIENTS WITH RHEUMATOID ARTHRITIS ». Dans SAKHAROV READINGS 2021 : ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute, 2021. http://dx.doi.org/10.46646/sakh-2021-1-262-265.

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The paper analyzes the content of calcium ions in the cytoplasm of peripheral blood lymphocytes in patients with rheumatoid arthritis. It has been shown that there is an increase in the concentration of intracellular ionized calcium in patients with rheumatoid arthritis relative to the control group, which history does not contain information about this disease.
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Figueirôa, Maria de Lourdes Castro de Oliveira, Maria Clara Moura Costa Campos, Viviane Leal Novais, Renata Borges de Lima, Izabela Prado Viana, Ana Paula Rodrigues Oliveira, Gabriel Costa Alves Sousa, Artur Araujo de Moraes, Gustavo Luiz Behrens Pinto et Mittermayer Barreto Santiago. « Atypically Localized Rheumatoid Nodulosis in Seronegative Rheumatoid Arthritis ». Dans XXXIX Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2022. http://dx.doi.org/10.47660/cbr.2022.2227.

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Pauk, Jolanta, Agnieszka Wasilewska, Justyna Chwiećko, Izabela Domysławska, Stanisław Sierakowski, Adam Idźkowski, Kristina Daunoravičienė et Julius Griškevičius. « Relation between treatment duration and temperature factors in rheumatoid arthritis ». Dans Biomdlore. VGTU Technika, 2016. http://dx.doi.org/10.3846/biomdlore.2016.14.

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Introduction: Rheumatoid arthritis (RA) is a chronic autoimmune disease which results in the loss of joint function and several deformities. The aim of the study was to evaluate the influence of duration of the disease and prescribed treatment on the temperature of RA joints. Methods: The evaluation was carried out on 30 patients with rheumatoid arthritis and 30 typical subjects as a control group. Thermograms were taken using thermograph camera. Results: Statistically significant difference was found for ankle joint temperature between the group of patients treated for more than 10 years and healthy subjects. Conclusions: The observations indicate that mean temperature of the skin above the ankle joint tends to decrease with rising duration of disease course. Progression of a RA leads to an erosive destruction in ankle joint in late stages of a disease.
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Y. Al-Attar, Huda, et Eman Sameer Al-Soffi. « Effect of Rheumatoid Arthritis on Alkaline Phosphatase Activity and Some Biochemical Variables ». Dans IX. International Scientific Congress of Pure, Applied and Technological Sciences. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress9-3.

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The study included measuring the concentrations of each Alkaline phosphatase enzyme, Calcium, Vitamin D in (45) blood samples (30) of patients with rheumatoid arthritis (15 males, 15 females), ages (55-83) years from arrivals to some laboratories in the city of Mosul/ Iraq. The results indicated a significant increasing the activity of Alkaline phosphatase a significant decrease in the concentration of calcium and vitamin D in rheumatoid arthritis compared to control groups, and the results also showed a significant increase in the activity of Alkaline phosphatase in females compared to males. There were no significant differences in the concentration of calcium and vitamin D between males and females
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Yildirim, Pinar. « Mining Online Drug Reviews Database for the Treatment of Rheumatoid Arthritis by using Deep Learning ». Dans 3rd International Conference on Data Science and Machine Learning (DSML 2022). Academy and Industry Research Collaboration Center (AIRCC), 2022. http://dx.doi.org/10.5121/csit.2022.121509.

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In this paper, a research study for online patient reviews is introduced. Rheumatoid arthritis is a long-term and disabling autoimmune disease. Today, a huge amount of people have rheumatoid arthritis in the world. Considering the importance of the medication of rheumatoid arthritis, we aimed to investigate patient reviews in WebMD database and get some useful information for this disease. Our results revealed that etanercept treatment has the highest number of reviews. Data analysis was applied to discover knowledge on this drug. Deep learning approach was used to predict the effectiveness of etanercept and classification results were compared with other traditional classifiers. According to the comparison of classifiers, deep neural network has better accuracy metrics than others. Therefore, the results highlight that deep learning can be encouraging for medical data analyses. We hope that our study can make contributions to intelligent data analysis in medical domain.
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Rapports d'organisations sur le sujet "Rheumatoid arthritis"

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Kalinina, E. V., A. R. Babaeva, A. V. Levickaya et M. S. Zvonorencko. MULTIMORBIDITY IN RHEUMATOID ARTHRITIS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-184-186.

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Fox, David A. Citrullinated Chemokines in Rheumatoid Arthritis. Fort Belvoir, VA : Defense Technical Information Center, octobre 2014. http://dx.doi.org/10.21236/ada611994.

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Kalinina, E. V., E. V. Krivospitskaya, A. R. Babaeva et M. S. Zvonorenko. COMORBIDITY IN PATIENTS WITH RHEUMATOID ARTHRITIS. "PLANET", 2019. http://dx.doi.org/10.18411/978-5-907192-54-6-2019-xxxvi-112-119.

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Deane, Keivn D. Pathogenesis and Prediction of Future Rheumatoid Arthritis. Fort Belvoir, VA : Defense Technical Information Center, octobre 2014. http://dx.doi.org/10.21236/ada613196.

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Mozgovaya, E. E., A. S. Trofimenko, M. A. Mamus, E. A. Tikhomirova, S. A. Bedina et S. S. Spitsma. FORMATION OF MONOCYTE EXTRACELLULAR TRAPS IN RHEUMATOID ARTHRITIS. Academy of Natural Knowledge, 2019. http://dx.doi.org/10.18411/1996-3955-2019-10-86-89.

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Papichev, E. V., L. E. Seewordova, Y. R. Akhverdyan, Y. V. Polyakova, B. V. Zavodovsky et O. D. Korolik. CLINICAL SIGNIFICANCE OF FETUIN-A IN RHEUMATOID ARTHRITIS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-219-225.

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Spitsina, S. S. Lipid metabolism disorders in patients with rheumatoid arthritis. Actual problems of experimental and clinical medicine : Materials 77th International Scientific and Practical Conference of Young Scientists and students, 2019. http://dx.doi.org/10.18411/makarenko-e-p.

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Trubnikova, N. S., et L. N. Shilova. FACTORS OF CARDIOVASCULAR RISK IN RHEUMATOID ARTHRITIS IN MEN. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-261-263.

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Aleksandrov, A. V., L. N. Shilova, N. V. Aleksandrova, I. V. Cherkachina, E. E. Mozgovaya, N. I. Emelianov, V. A. Aleksandrov et M. V. Nikitin. On the antioxidant status in patients with rheumatoid arthritis. FilodirittoEditore, 2018. http://dx.doi.org/10.18411/978-88-85813-04-5-2018-10-413-419.

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Donahue, Katrina E., Gerald Gartlehner, Elizabeth R. Schulman, Beth Jonas, Emmanuel Coker-Schwimmer, Sheila V. Patel, Rachel Palmieri Weber, Kathleen N. Lohr, Carla Bann et Meera Viswanathan. Drug Therapy for Early Rheumatoid Arthritis : A Systematic Review Update. Agency for Healthcare Research and Quality (AHRQ), juillet 2018. http://dx.doi.org/10.23970/ahrqepccer211.

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