Littérature scientifique sur le sujet « Rfc1 »
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Articles de revues sur le sujet "Rfc1"
Amin, Neelam S., K. Michelle Tuffo et Connie Holm. « Dominant Mutations in Three Different Subunits of Replication Factor C Suppress Replication Defects in Yeast PCNA Mutants ». Genetics 153, no 4 (1 décembre 1999) : 1617–28. http://dx.doi.org/10.1093/genetics/153.4.1617.
Texte intégralCullmann, G., K. Fien, R. Kobayashi et B. Stillman. « Characterization of the five replication factor C genes of Saccharomyces cerevisiae. » Molecular and Cellular Biology 15, no 9 (septembre 1995) : 4661–71. http://dx.doi.org/10.1128/mcb.15.9.4661.
Texte intégralCui, Kan, Lei Qin, Xianyu Tang, Jieying Nong, Jin Chen, Nan Wu, Xin Gong, Lixiong Yi, Chenghuizi Yang et Shitou Xia. « A Single Amino Acid Substitution in RFC4 Leads to Endoduplication and Compromised Resistance to DNA Damage in Arabidopsis thaliana ». Genes 13, no 6 (9 juin 2022) : 1037. http://dx.doi.org/10.3390/genes13061037.
Texte intégralGong, Maokai, James Yess, Tatiana Connolly, S. Percy Ivy, Takao Ohnuma, Kenneth H. Cowan et Jeffrey A. Moscow. « Molecular Mechanism of Antifolate Transport-Deficiency in a Methotrexate-Resistant MOLT-3 Human Leukemia Cell Line ». Blood 89, no 7 (1 avril 1997) : 2494–99. http://dx.doi.org/10.1182/blood.v89.7.2494.
Texte intégralNaiki, Takahiro, Tae Kondo, Daisuke Nakada, Kunihiro Matsumoto et Katsunori Sugimoto. « Chl12 (Ctf18) Forms a Novel Replication Factor C-Related Complex and Functions Redundantly with Rad24 in the DNA Replication Checkpoint Pathway ». Molecular and Cellular Biology 21, no 17 (1 septembre 2001) : 5838–45. http://dx.doi.org/10.1128/mcb.21.17.5838-5845.2001.
Texte intégralPanda, Debasis, Daniel J. Fernandez, Madhu Lal, Eugen Buehler et Bernard Moss. « Triad of human cellular proteins, IRF2, FAM111A, and RFC3, restrict replication of orthopoxvirus SPI-1 host-range mutants ». Proceedings of the National Academy of Sciences 114, no 14 (20 mars 2017) : 3720–25. http://dx.doi.org/10.1073/pnas.1700678114.
Texte intégralKai, Mihoko, Hiroyuki Tanaka et Teresa S. F. Wang. « Fission Yeast Rad17 Associates with Chromatin in Response to Aberrant Genomic Structures ». Molecular and Cellular Biology 21, no 10 (15 mai 2001) : 3289–301. http://dx.doi.org/10.1128/mcb.21.10.3289-3301.2001.
Texte intégralMa, David W. L., Richard H. Finnell, Laurie A. Davidson, Evelyn S. Callaway, Ofer Spiegelstein, Jorge A. Piedrahita, J. Michael Salbaum et al. « Folate Transport Gene Inactivation in Mice Increases Sensitivity to Colon Carcinogenesis ». Cancer Research 65, no 3 (1 février 2005) : 887–97. http://dx.doi.org/10.1158/0008-5472.887.65.3.
Texte intégralXie, Yali, Chris Counter et Eric Alani. « Characterization of the Repeat-Tract Instability and Mutator Phenotypes Conferred by a Tn3 Insertion in RFC1, the Large Subunit of the Yeast Clamp Loader ». Genetics 151, no 2 (1 février 1999) : 499–509. http://dx.doi.org/10.1093/genetics/151.2.499.
Texte intégralZhao, Rongbao, Feng Gao et I. David Goldman. « Reduced folate carrier transports thiamine monophosphate : an alternative route for thiamine delivery into mammalian cells ». American Journal of Physiology-Cell Physiology 282, no 6 (1 juin 2002) : C1512—C1517. http://dx.doi.org/10.1152/ajpcell.00547.2001.
Texte intégralThèses sur le sujet "Rfc1"
Breitenbücher, Frank. « Allgemeine und funktionelle Untersuchungen zur grossen Untereinheit des humanen Replikationsfaktors C (RFC1), sowie initiale Untersuchungen zur Regulation der Expression von RFC1 durch das BCR-ABLp210-Onkogen ». [S.l.] : [s.n.], 2005. http://archiv.ub.uni-marburg.de/diss/z2005/0114/.
Texte intégralHinken, Matthias. « Gewebeverteilung und Lokalisation des Transportproteins für reduzierte Folate (RFC1) der Ratte ». Doctoral thesis, Universitätsbibliothek Leipzig, 2007. http://nbn-resolving.de/urn:nbn:de:bsz:15-20071107-143055-8.
Texte intégralChazelas, Pauline. « Syndrome de CANVAS – Analyse moléculaire des répétitions pentanucléotidiques du gène RFC1 et étude de leurs conséquences physiopathologiques ». Electronic Thesis or Diss., Limoges, 2024. http://www.theses.fr/2024LIMO0006.
Texte intégralThe CANVAS syndrome defines a group of clinical entities - cerebellar ataxia, sensory neuronopathy and vestibular areflexia - that together or separately are responsible for a variety of symptoms, the most common of which is a disturbance of balance. Interestingly, it has now been shown that the majority of patients present with a chronic refractory cough decades before the onset of neurological disorders. In 2019, the genetic cause of this syndrome was described. It involves the RFC1 gene, in which a biallelic expansion of AAGGG pentanucleotide repeats in intron 2 of the gene has been implicated in clinically affected patients. This large AAGGG motif replaces the AAAAG conformation, which is classically repeated 11 times in the general population. The pathophysiological cause of the disease is still unknown. Several studies have shown variability in the repeat zone, both in terms of motif conformation and number of repeats. The implications for pathology are not always clear.In this study, we focused on three aspects of this syndrome: (a) description of the variability of the repeated zone in a cohort of controls and in a cohort of patients referred for neuropathy, (b) histological study of peripheral nerve damage, particularly that of small nerve fibers, which has not been previously described, (c) study of the prevalence of biallelic expansion of the pentanucleotide AAGGG in a cohort of refractory chronic cough patients.Our results revealed novel complex repeats in both controls and patients with neuropathy. The involvement of small nerve fibers in CANVAS was objectified for the first time. Our cohort study of 68 patients with refractory chronic cough identified 16% of patients with a pathogenic AAGGG biallelic expansion, leading us to suggest that the cough of CANVAS patients is neurogenic.Taken together, the results of this thesis represent a breakthrough in the molecular and pathophysiological knowledge of CANVAS syndrome. Further work is required to understand the underlying pathophysiological mechanisms with a view to developing personalized therapies
Halwachs, Sandra. « Regulation des Reduced Folate Carrier (RFC1) in HPCT-1E3-Ratten-Hepatocytoma-Zellen durch Cytochrom P450-Induktoren vom Phenobarbital-Typ ». Doctoral thesis, Universitätsbibliothek Leipzig, 2006. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-34397.
Texte intégralLopes, Tairine Zara. « Avaliação de polimorfismos nos genes MTHFR, MTR, RFC1 e CßS envolvidos no metabolismo do folato em pacientes com câncer de tireoide ». Faculdade de Medicina de São José do Rio Preto, 2015. http://hdl.handle.net/tede/272.
Texte intégralMade available in DSpace on 2016-06-21T19:17:07Z (GMT). No. of bitstreams: 1 tairinezaralopes_dissert.pdf: 1511634 bytes, checksum: 2203122e0b39cf1687115059c3e19447 (MD5) Previous issue date: 2015-10-29
Introduction: Thyroid cancer is the most common malignancy of the endocrine system and has been presenting continuous increase in the last years. Studies suggest that folate deficiency in the body decrease DNA repair, resulting in malignant cells changes that alter expression of genes, and may induce several kinds of cancer development. Polymorphisms in genes involved in folate pathway have been investigated as risk factors for susceptibility to cancer, among them MTHFR, MTR, RFC1 and CßS. Objectives: To investigate association of polymorphisms in the MTHFR (C677T), MTR (A2756G), RFC1 (A80G) and CßS (844ins68) genes in risk thyroid cancer in a case-control study; to evaluate the association of polymorphisms with gender, age, alcohol and tobacco consumption, body-mass index in thyroid cancer development; and to evaluated the association between polymorphisms and clinical-histopathological parameters. Methods: This study included 462 individuals (151 patients with thyroid cancer and 311 controls). The peripheral blood was collected and genomic DNA was extracted. The MTHFR (C677T), MTR (A2756G) and RFC1 (A80G) were evaluated by PCR-RFLP and CßS (844ins68) by conventional PCR without enzymatic digestion. For statistical analysis chi-square and multiple logistic regression were used. Results: The results showed that MTHFR C677T (OR=2.87, 95% CI=1.50-5.48, p< 0.01, codominant model), (OR=1.76, 95% CI=1.18-2.64, p< 0.01, dominant model), (OR=2.37, 95% CI=1.28-4.39, p< 0.01, recessive model) and RFC1 A80G (OR: 1.55; 95% CI: 1.02-2.38; p=0.04, recessive model) were associated with thyroid cancer. The alcohol (OR=1.56, 95% CI=1.36-1.89, p< 0.01) and tobacco consumption (OR=1.97, 95% CI=1.28-3.04, p< 0.01) were statistically significant, being associated with increased risk. The MTR A2756G is associated with tumor extension (OR=2.69, 95% CI=1.27-5.71, p< 0.01) and aggressiveness (OR= 4.51, 95% CI=1.67-12.1, p< 0.01). Conclusions: The MTHFR (C677T) and RFC1 (A80G) polymorphisms were involved in risk for thyroid cancer. Additionally, alcohol and tobacco consumption increase risk for disease development.
Introdução: O câncer de tireoide é a neoplasia maligna mais comum do sistema endócrino e vem apresentando contínuo aumento nos últimos anos. Estudos sugerem que a deficiência de folato no organismo diminui a reparação do DNA, resultando em alterações celulares malignas que modulam a expressão gênica, podendo levar ao desenvolvimento de vários tipos de câncer. Polimorfismos em genes envolvidos na via do folato têm sido investigados como fatores de risco para suscetibilidade ao câncer, entre eles, polimorfismos nos genes MTHFR, MTR, RFC1 e CßS. Objetivos: Investigar a associação dos polimorfismos nos genes MTHFR (C677T), MTR (A2756G), RFC1 (A80G) e CßS (844ins68) no risco de câncer de tireoide em um estudo caso-controle; Avaliar a associação dos polimorfismos com o gênero, idade, consumo de álcool e tabaco, índice de massa corpórea (IMC) no desenvolvimento do câncer de tireoide; Avaliar a associação entre os polimorfismos e os parâmetros clínico-histopatológicos do câncer de tireoide. Casuística e Método: Este estudo incluiu 462 indivíduos (151 pacientes com câncer de tireoide e 311 indivíduos controles). Foi coletado sangue periférico e extraído o DNA genômico. Os polimorfismos MTHFR (C677T), MTR (A2756G) e RFC1 (A80G) foram avaliados por meio da PCR-RFLP e o polimorfismo CßS (844ins68) foi analisado por PCR convencional sem corte enzimático. Para análise estatística utilizou-se o teste do qui-quadrado e regressão logística múltipla. Resultados: Os resultados mostraram que os polimorfismos MTHFR C677T (OR=2.87, 95% IC=1.50-5.48, p< 0.01, modelo codominante), (OR=1.76, 95% IC=1.18-2.64, p< 0.01, modelo dominante), (OR=2.37, 95% IC=1.28-4.39, p< 0.01, modelo recessivo) e RFC1 A80G (OR: 1.55; 95% IC: 1.02-2.38; p=0.04, modelo recessivo) estão associados ao câncer de tireoide. O consumo de álcool (OR=1.56, 95% IC=1.36-1.89, p< 0.01) e tabaco (OR=1.97, 95% IC=1.28-3.04, p< 0.01) foram estatisticamente significantes, sendo associados ao aumento do risco. O polimorfismo MTR A2756G está associado à extensão do tumor (OR=2.69, 95% IC=1.27-5.71, p< 0.01) e à agressividade (OR= 4.51, 95% IC=1.67-12.1, p< 0.01). Conclusões: Os polimorfismos MTHFR (C677T) e RFC1 (A80G) estão envolvidos no risco de câncer de tireoide. Adicionalmente, o consumo de álcool e tabaco aumenta o risco de desenvolvimento da doença.
Tomida, Junya. « DNA damage induced ubiquitylation of RFC2 subunit of RFC complex ». Kyoto University, 2008. http://hdl.handle.net/2433/135870.
Texte intégralNg, Chin Chin. « Performance analysis of the mobile IP protocol (RFC 3344 and related RFCS) ». Thesis, Monterey, Calif. : Naval Postgraduate School, 2006. http://bosun.nps.edu/uhtbin/hyperion.exe/06Dec%5FNg%5FChin.pdf.
Texte intégralThesis Advisor(s): George W. Dinolt, J. D. Fulp. "December 2006." Includes bibliographical references (p. 121-125). Also available in print.
Hakim, Chantal DDS MSD, Marie M. MD PhD DrSc Tolarová et Miroslav MD PhD Tolar. « Association of Type and Severity of Nonsyndromic Orofacial Clefts with Combined Genotypes of RFC1 80 GA, MTHFR 677 CT, IRF6 (rs642961) and IRF6 (rs2235371) Gene Polymorphisms in an Indian Population ». Scholarly Commons, 2020. https://scholarlycommons.pacific.edu/dugoni_etd/2.
Texte intégralGiusti, Kelma Cordeiro da Silva. « Associação entre polimorfismos em genes relacionados ao metabolismo de folato (RFC1, GCP2, MTHFR e MTHFD1) e alterações nas concentrações de folato, cobalamina e homocisteína em mulheres com história de abortos espontâneos recorrentes ». Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-08032013-115754/.
Texte intégralThe recurrent spontaneous abortion (RSA) is characterized by the occurrence of three or more consecutive miscarriages and affects 2-4% of women of childbearing age. The etiology is associated with several risk factors such as uterine abnormalities, chromosomal aberrations, autoimmunity, thrombophilia, increased concentration of homocysteine (tHcy). About 40% of cases remains unknown cause. The units of carbon metabolism plays an essential role in the availability of the cell folate, is essential for the placental and fetal development. A deficiency of the vitamins that regulate this metabolism, like folic acid, and polymorphisms in genes encoding enzymes related to folate metabolism (MTHFR, RFC1, and GCP2 MTHFD1) may lead to decreased concentrations of this vitamin and increased concentrations of tHcy. Objective was to evaluate the association between polymorphisms in genes related to folate metabolism (RFC1, GCP2, MTHFD1 and MTHFR) and the risk of having AER, and to evaluate the association between these polymorphisms and changes in concetranções folate, cobalamin, and homocysteine. Three groups were divided: AER primary: 117 women with RSA and no viable fetus, AER secondary: 139 women with RSA and at least one viable fetus and Control: 264 women with no history of miscarriage. None of the women was pregnant at time of blood collection. Blood samples were taken for biochemical (folate, Cbl, tHcy, etc.), immunological and genomic DNA extraction. The genotyping were carried out by PCR-RFLP or real time PCR. Serum concentrations of folate and Cbl were higher in groups 1 and 2 (p <0.05). The distribution of genotypes of MTHFR c.677C> T, MTHFR c.1298A> C, MTHFD1 c.1958G> A, RFC1 c.80G>GCP2 A and c.1561C> T was similar among the three groups. The increased concentrations of serum folate (OR: 1.05, 95% CI: 1.03 - 1.07, p <0.001), Cbl (OR: 1.00, 95% CI: 1.00 to 1.00, p = 0.016), tHcy (OR: 1.03, 95% CI: 0.97 to 1.11, p = 0.033) and T4 (OR: 1.02, 95% CI: 1.00 to 1.03, p = 0.006) and the presence of ANA (1:160) (OR: 2.90, 95% CI: 1.25 - 6.75, p = 0.013) were considered risk factors primary for abortion. For secondary abortion, were considered risk factors increased the concentrations of serum folate (OR: 1.04, 95% CI: 1.02 - 1.05, p <0.001), cobalamin (OR: 1.00, 95 % CI: 1.00 to 1.00, p = 0.019) and tHcy (OR: 1.05, 95% CI: 1.00 to 1.09, p = 0.039), higher age (OR: 1.02, 95% CI: 0.98 to 1.06, p = 0.031), cigarette smoking (OR: 2.54, 95% CI: 1.41 to 4.60, p = 0.002) and had a higher BMI (OR : 1,42,95% CI: 1.07 to 1.88, p = 0.015). The studied polymorphisms were not associated with increased risk of having RSA when analyzed separately, and were not associated with changes in serum folate, Cbl and tHcy, with the exception of the MTHFR 677TT genotype, whose patients had a higher concentration of total tHcy compared with those with 677CC and 677CT genotypes in the three groups. The variable concentrations of folate, Cbl, tHcy, and T4, presence of ANA and have been associated with increased risk for miscarriage primary. The variables age, BMI, smoking, concentrations of folate, Cbl and tHcy were associated with increased risk of secondary miscarriage.
Alata, Jimenez Nagif. « La deficiencia en los transportadores de folato (FOLR1 y RFC1) causa cambios epigenéticos que afectan el desarrollo neural y de las células de la cresta neural en embriones de pollo Gallus gallus ». Bachelor's thesis, Universidad Nacional Mayor de San Marcos, 2019. https://hdl.handle.net/20.500.12672/10342.
Texte intégralTesis
Livres sur le sujet "Rfc1"
Peter, Loshin, dir. Big book of FYI RFCs. San Francisco, Calif : Morgan Kaufmann, 2000.
Trouver le texte intégralLoshin, Peter. Big book of IPsec RFCs. San Diego, CA : Morgan Kaufmann, 2000.
Trouver le texte intégralSalus, Peter H. Big book of IPv6 addressing RFCs. San Diego : Morgan Kaufmann, 2000.
Trouver le texte intégralPeter, Loshin, dir. Big book of terminal emulation RFCs. San Francisco : Morgan Kaufmann, 2000.
Trouver le texte intégralLin, Tan. Heath course pak rfc. 2e éd. Denver : Counterpath, 2012.
Trouver le texte intégralLoshin, Peter. Big book of Internet file transfer RFCs. San Diego : Morgan Kaufmann, 2000.
Trouver le texte intégralPeter, Loshin, dir. Big book of World Wide Web RFCs. San Francisco, Calif : Morgan Kaufmann, 2000.
Trouver le texte intégralRevell, Alex. No 56 Sqn RAF/RFC. Oxford : Osprey, 2009.
Trouver le texte intégralNo 60 Sqn RFC/RAF. Oxford : Osprey Pub., 2011.
Trouver le texte intégralLoshin, Peter. Essential Ethernet standards : RFCs and protocols made practical. New York : John Wiley & Sons, 2000.
Trouver le texte intégralChapitres de livres sur le sujet "Rfc1"
Angeli, Axeö, Ulrich Streit et Robi Gonfalonieri. « RFC Remote Function Call ». Dans The SAP R/3® Guide to EDI and Interfaces, 79–85. Wiesbaden : Vieweg+Teubner Verlag, 2000. http://dx.doi.org/10.1007/978-3-663-01091-3_12.
Texte intégralStubbs, David A., et Wally C. Hoppe. « RFC Automated Inspection Overview ». Dans Review of Progress in Quantitative Nondestructive Evaluation, 901–10. Boston, MA : Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-7763-8_97.
Texte intégralKarimi, Kamran, et Howard J. Hamilton. « RFCT : An Association-Based Causality Miner ». Dans Advances in Artificial Intelligence, 334–38. Berlin, Heidelberg : Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/3-540-47922-8_29.
Texte intégralJin, Feng, et Duruo Huang. « Design of RFC Arch Dam ». Dans Hydroscience and Engineering, 179–203. Singapore : Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-8298-8_6.
Texte intégralJin, Feng, et Duruo Huang. « Design of RFC Gravity Dam ». Dans Hydroscience and Engineering, 135–77. Singapore : Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-8298-8_5.
Texte intégralHoppe, Wally C., et David A. Stubbs. « RFC Eddy Current Probe Tests ». Dans Review of Progress in Quantitative Nondestructive Evaluation, 893–900. Boston, MA : Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-7763-8_96.
Texte intégralOke, Muse, Manal S. Zaher et Samir M. Hamdan. « Mechanism of PCNA Loading by RFC ». Dans Molecular Life Sciences, 1–6. New York, NY : Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_137-1.
Texte intégralOke, Muse, Manal S. Zaher et Samir M. Hamdan. « PCNA Loading by RFC, Mechanism of ». Dans Molecular Life Sciences, 861–66. New York, NY : Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_137.
Texte intégralDo, Cao-Phan, Thien-Phuc Nguyen et Anh-Thang Le. « Study on Adhesive Characteristics of RFCC Asphalt Mastic ». Dans Lecture Notes in Civil Engineering, 926–36. Singapore : Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-7434-4_97.
Texte intégralWörfel, René, et Hans Dodel†. « Antragsbeispiele API bzw. RfC (CR/C)/ITU ». Dans Satellitenfrequenzkoordinierung, 391–99. Berlin, Heidelberg : Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-29203-3_13.
Texte intégralActes de conférences sur le sujet "Rfc1"
Hirons, B., K. Rhatigan, R. Curro, B. Rugginini, J. Shaw, H. Abubakar-Waziri, H. Kesavan et al. « P215 RFC1 disorder ; a genetic, neuropathic cause of chronic cough ». Dans British Thoracic Society Winter Meeting 2023, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 22 to 24 November 2023, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2023. http://dx.doi.org/10.1136/thorax-2023-btsabstracts.365.
Texte intégralLaury, John, Lars Abrahamsson et Math Bollen. « Transient Stability of Rotary Frequency Converter Fed Low Frequency Railway Grids : The Impact of Different Grid Impedances and Different Converter Station Configurations ». Dans 2018 Joint Rail Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/jrc2018-6247.
Texte intégralXu, Chang, Jie Zhang et Zhu Sun. « Online Reputation Fraud Campaign Detection in User Ratings ». Dans Twenty-Sixth International Joint Conference on Artificial Intelligence. California : International Joint Conferences on Artificial Intelligence Organization, 2017. http://dx.doi.org/10.24963/ijcai.2017/541.
Texte intégralKolling, Maikel L., Felipe A. Kuentzer, Cristiano Battisti, Cristiano B. Both, Rafael R. dos Santos et Tatiana G. S. dos Santos. « Verificação em Hardware de Componentes de Comunicação ». Dans Simpósio em Sistemas Computacionais de Alto Desempenho. Sociedade Brasileira de Computação, 2008. http://dx.doi.org/10.5753/wscad.2008.17678.
Texte intégralYen, Jane, Ramesh Govindan et Barath Raghavan. « Tools for disambiguating RFCs ». Dans ANRW '21 : Applied Networking Research Workshop. New York, NY, USA : ACM, 2021. http://dx.doi.org/10.1145/3472305.3472314.
Texte intégralMeriem, B. « Performance of self-compacting concrete based on fine recycled concrete aggregate incorporating polyethylene terephtalate fibers ». Dans Advanced Topics in Mechanics of Materials, Structures and Construction. Materials Research Forum LLC, 2023. http://dx.doi.org/10.21741/9781644902592-17.
Texte intégralAhn, HyeongJoon. « Design Procedure of an Eddy Current Damper Type Reaction Force Compensation (RFC) Mechanism for a Linear Motor Motion Stage ». Dans ASME 2015 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/detc2015-48080.
Texte intégralWirasbawa, Nicholas Dwiarto, Yaman Khaeruzzaman et Alexander Waworuntu. « Modern, Secure Application Programming Interface Implementation using RFC 6238 and RFC 7617 ». Dans 2023 International Conference on Smart Computing and Application (ICSCA). IEEE, 2023. http://dx.doi.org/10.1109/icsca57840.2023.10087735.
Texte intégralElkacimi, Younes, et Norbert Mu¨ller. « Regenerative Flow Pumps and Compressors (RFP/RFC) Applications for Water as Refrigerant ». Dans ASME 2006 International Mechanical Engineering Congress and Exposition. ASMEDC, 2006. http://dx.doi.org/10.1115/imece2006-13486.
Texte intégralLaury, John, Lars Abrahamsson et Math Bollen. « Impact of Reduced Share of Rotary Frequency Converters in a Low Frequency Synchronous Railway Grid : A Transient Stability Study ». Dans 2019 Joint Rail Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/jrc2019-1238.
Texte intégralRapports d'organisations sur le sujet "Rfc1"
Brownlee, N. SVG Drawings for RFCs : SVG 1.2 RFC. RFC Editor, décembre 2016. http://dx.doi.org/10.17487/rfc7996.
Texte intégralEggert, L. Moving the Undeployed TCP Extensions RFC 1072, RFC 1106, RFC 1110, RFC 1145, RFC 1146, RFC 1379, RFC 1644, and RFC 1693 to Historic Status. RFC Editor, mai 2011. http://dx.doi.org/10.17487/rfc6247.
Texte intégralDaigle, L., dir. The RFC Series and RFC Editor. RFC Editor, juillet 2007. http://dx.doi.org/10.17487/rfc4844.
Texte intégralHousley, R., et L. Daigle, dir. The RFC Series and RFC Editor. RFC Editor, février 2020. http://dx.doi.org/10.17487/rfc8729.
Texte intégralHoffman, P. HTML Format for RFCs. Sous la direction de J. Hildebrand. RFC Editor, décembre 2016. http://dx.doi.org/10.17487/rfc7992.
Texte intégralMasinter, L., et M. Hardy. PDF Format for RFCs. Sous la direction de T. Hansen. RFC Editor, décembre 2016. http://dx.doi.org/10.17487/rfc7995.
Texte intégralFlanagan, H., dir. Fifty Years of RFCs. RFC Editor, décembre 2019. http://dx.doi.org/10.17487/rfc8700.
Texte intégralHuitema, C., J. Postel et S. Crocker. Not All RFCs are Standards. RFC Editor, avril 1995. http://dx.doi.org/10.17487/rfc1796.
Texte intégralFlanagan, H. Requirements for Plain-Text RFCs. RFC Editor, décembre 2016. http://dx.doi.org/10.17487/rfc7994.
Texte intégralFlanagan, H., et S. Ginoza. RFC Style Guide. RFC Editor, septembre 2014. http://dx.doi.org/10.17487/rfc7322.
Texte intégral