Littérature scientifique sur le sujet « Repro-toxicity »
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Articles de revues sur le sujet "Repro-toxicity"
Johnson, E. Marshall. « Perspectives On Reproductive and Developmental Toxicity ». Toxicology and Industrial Health 2, no 4 (octobre 1986) : 453–82. http://dx.doi.org/10.1177/074823378600200408.
Texte intégralCook, Nancy H., et Peter G. Wells. « Toxicity of Halifax Harbour Sediments : an Evaluation of the Microtox® Solid-Phase Test ». Water Quality Research Journal 31, no 4 (1 novembre 1996) : 673–708. http://dx.doi.org/10.2166/wqrj.1996.037.
Texte intégralDalsenter, PR, AS Faqi, J. Webb, H.-J. Merker et I. Chahoud. « Reproductive toxicity and toxicokinetics of lindane in the male offspring of rats exposed during lactation ». Human & ; Experimental Toxicology 16, no 3 (mars 1997) : 146–53. http://dx.doi.org/10.1177/096032719701600303.
Texte intégralPant, N., AK Prasad, SC Srivastava, R. Shankar et SP Srivastava. « Effect of oral administration of carbofuran on male reproductive system of rat ». Human & ; Experimental Toxicology 14, no 11 (novembre 1995) : 889–94. http://dx.doi.org/10.1177/096032719501401106.
Texte intégralKhafaji, Sura. « Antioxidant, anti-inflammatory, and anti-reprotoxic effects of kaempferol and vitamin E on lead acetate-induced testicular toxicity in male rats ». Open Veterinary Journal 13, no 12 (2023) : 1683. http://dx.doi.org/10.5455/ovj.2023.v13.i12.17.
Texte intégralHu, Chelin, Zoey Hsuan Hsiao, Lei Yin et Xiaozhong Yu. « The role of small GTPases in bisphenol AF -induced multinucleation in comparison with dibutyl phthalate in the male germ cells ». Toxicological Sciences, 10 janvier 2023. http://dx.doi.org/10.1093/toxsci/kfad005.
Texte intégralAbo El-Ela, Fatma I., Amr Gamal, Hossny A. El-Banna, Marwa A. Ibrahim, Ahmed H. El-Banna, Abdel-Razik H. Abdel-Razik, Ahmed Abdel-Wahab, Walid Hamdy Hassan et Asmaa K. Abdelghany. « Repro-protective activity of amygdalin and spirulina platensis in niosomes and conventional forms against aluminum chloride–induced testicular challenge in adult rats : role of CYP11A1, StAR, and HSD-3B expressions ». Naunyn-Schmiedeberg's Archives of Pharmacology, 1 novembre 2023. http://dx.doi.org/10.1007/s00210-023-02788-9.
Texte intégralThèses sur le sujet "Repro-toxicity"
Monrose, Mélusine. « Etude des rôles du récepteur nucléaire CAR dans la physiologie et les physiopathologies testiculaires ». Electronic Thesis or Diss., Université Clermont Auvergne (2021-...), 2022. http://www.theses.fr/2022UCFAC114.
Texte intégralFertility disorders are a major public health concern since they concern millions of people of reproductive age all over the world. The male factor is involved in 50% of identified cases of infertility within couples, involving qualitative and quantitative alterations of the sperm production. However, there are still too many unknowns regarding the cause of many male reproductive disorders. For several decades, an increase in these disorders has been detected especially in developed countries, attracting the scientific community attention to the involvement of environmental pollutants in the etiology of infertility. The number and variety of xenobiotics released into the environment are huge and threaten many physiological functions because of the multiple intracellular signalling pathways that they can affect. According to the researchers, perinatal exposure to xenobiotics may induce altered testicular physiology and predispose to the development of fertility disorders. However, It is essential to better understand the signalling pathways involved in the negative environmental impacts of xenobiotics on testicular physiology to identify other causes of infertility. CAR and PXR are xenobiotic receptors expressed in the testis in men as well as rodents. However, their roles within the testis have not been fully established. In this study, we have identified impacts of neonatal modulations of their signalling on the testicular physiology and the reproduction function in mice. Our data showed that the inhibition of CAR signalling leads to fertility disorders in mice, correlated with qualitative sperm production. Indeed, we have detected alterations in the early and late differentiations of germ cells, notably involving FOXO1 signalling and H4 acetylation levels deregulations that can be associated with histones retention within the sperm genome. Moreover, we have highlighted deleterious additive impacts of the neonatal co-activation of CAR and PXR signalling of mice fertility. Our study has identified major roles of CAR and PXR nuclear receptors in testicular physiology and their potential implication in the emergence of fertility disorders associated with environmental exposures