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Articles de revues sur le sujet « Rapid decompression »

Auteur : Grafiati

Publié le 10 mars 2023

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1

Hanson, B. "GEOLOGY: Rapid Decompression." Science 303, no. 5657 (2004): 435b—435. http://dx.doi.org/10.1126/science.303.5657.435b.

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Christopher, Gabriel, and Suchard Jeffrey. "Hematuria Following Rapid Bladder Decompression." Clinical Practice and Cases in Emergency Medicine 1, no. 4 (2017): 443–45. http://dx.doi.org/10.5811/cpcem.2017.9.35803.

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Alidibirov, Mikhail, and Donald B. Dingwell. "Magma fragmentation by rapid decompression." Nature 380, no. 6570 (1996): 146–48. http://dx.doi.org/10.1038/380146a0.

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4

Pflugrath, Brett D., Craig A. Boys, and Bruce Cathers. "Predicting hydraulic structure-induced barotrauma in Australian fish species." Marine and Freshwater Research 69, no. 12 (2018): 1954. http://dx.doi.org/10.1071/mf18137.

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When fish pass downstream through river infrastructure, such as dams and weirs, barotrauma may occur as a result of rapid decompression. In severe cases, barotrauma may lead to mortality. Different species are likely to respond differently to these decompressions. Therefore, to predict barotrauma for a specific species, surrogate species may not be a valid approach, and it may be necessary to examine each species individually. For the present study, Australian bass and carp gudgeon were exposed to a range of rapid decompressions using hyper- and hypobaric hydro-chambers and examined for injuries and mortality. Rapid decompression data from these two fish species, in addition to previously examined Murray cod and silver perch, were evaluated to determine which injuries were highly associated with and likely to predict mortality. Dose–response logistic regression models were developed for each species to predict injury and mortality over a range of rapid decompressions. These models are valuable for estimating injury and mortality rates for fish passing though river infrastructure and can be applied to specific sites where pressure profiles have been developed. Applying these models to current and future infrastructure can provide important insight into what measures or design alterations may be necessary to reduce negative effects of infrastructure on fish populations.

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Pratt, J. D. "Rapid decompression of pressurized aircraft fuselages." Journal of Failure Analysis and Prevention 6, no. 6 (2006): 70–74. http://dx.doi.org/10.1361/154770206x156268.

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Hudson, Steven J., and Jeffrey S. Todd. "Rapid Decompression in the EA-6B." Military Medicine 163, no. 8 (1998): 572–74. http://dx.doi.org/10.1093/milmed/163.8.572.

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Lu, Jianfeng, Lin Li, and Lunjun Chen. "STUDY ON THE DECOMPRESSION TIME OF THE HYPOBARIC RAPID DECOMPRESSION CHAMBER." International Journal of Heat and Technology 33, no. 2 (2015): 75–78. http://dx.doi.org/10.18280/ijht.330212.

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HE DUAN-WEI, ZHANG FU-XIANG, LIU RI-PING, ZHANG MING, XU YING-FAN, and WANG WEN-KUI. "A NEW METHOD FOR RAPID SOLIDIFICATION QUENCHING BY RAPID DECOMPRESSION." Acta Physica Sinica 47, no. 2 (1998): 183. http://dx.doi.org/10.7498/aps.47.183.

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He, D. W., F. X. Zhang, M. Zhang, et al. "Quenching with rapid decompression—a new method for rapid solidification." Applied Physics Letters 71, no. 26 (1997): 3811–13. http://dx.doi.org/10.1063/1.120542.

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Tian, Zifeng, Jianfeng Lu, Zhao Gu, Lihua Yu, Hao Zhou, and Lijun Yang. "Theoretical and Experimental Study on Rapid Decompression Oscillation in Altitude Chamber." International Journal of Aerospace Engineering 2022 (November 18, 2022): 1–16. http://dx.doi.org/10.1155/2022/9258503.

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Given the frequent airflow excitation phenomenon caused by the rapid decompression of the altitude chamber, the mathematical model of the nonlinear system of the rapid decompression of the altitude chamber is established. The polynomial parameter method is used to evaluate the characteristics of airflow oscillation, and a rapid decompression test system is built. The test results verify the pressure oscillation phenomenon of the numerical simulation. This paper proves the phenomenon of fluid-induced vibration in the process of rapid decompression and determines that the main factors affecting the induced oscillation are the diameter of the pipe (throat), pressure difference between the two chambers, and initial pressure conditions. Specifically, this study establishes safety and reliability for preventing engineering accidents caused by resonance in the altitude chamber.

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A., Roman, and Tverezovska N. "Features of rapid diagnostics in the process of decompression measures." HUMANITARIAN STUDIOS: PEDAGOGICS, PSYCHOLOGY, PHILOSOPHY 12, no. 4 (2021): 89–94. http://dx.doi.org/10.31548/hspedagog2021.04.089.

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The article reveals the primary social and psychological rehabilitation (decompression) at the stage of the march of the unit from the area of environmental protection, both permanent locations and permanent locations. The commander's account of certain information when deciding on decompression in a certain unit is shown. The drawing up of an individual plan of a serviceman in the process of decompression is shown, as well as the stages preceding this rehabilitation.

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Elias, E., and P. L. Chambre´. "Flashing Inception in Water During Rapid Decompression." Journal of Heat Transfer 115, no. 1 (1993): 231–38. http://dx.doi.org/10.1115/1.2910654.

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A phenomenological model is presented for predicting the thermodynamic conditions at the onset of flashing in liquid undergoing a static or flow depressurization transient. It is shown that at extremely high rates of decompression (pulse expansion), a liquid may reach the homogeneous nucleation limit of superheat before appreciable phase transition occurs. A criterion for pulse expansion is derived by an asymptotic solution of the mass and energy equations in conjunction with the equation for spontaneous bubble nucleation and growth near the flashing inception point. The effect of impurities and dissolved gases is considered by an empirically defined heterogeneous nucleation coefficient. The model predicts the minimum pressure at the flashing point with a probable error of less than 11 percent, using 83 experimental data points.

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Wu, Meng-Yu, Jer-Ruey Chang, Yi-Kung Lee, Po-Chen Lin, and Tou-Yuan Tsai. "The Effect and Safety of Rapid and Gradual Urinary Decompression in Urine Retention: A Systematic Review and Meta-Analysis." Medicina 58, no. 10 (2022): 1441. http://dx.doi.org/10.3390/medicina58101441.

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Background and objectives: Trials to evaluate the effect and safety of rapid and gradual urinary decompression have been published for decades. Due to inconclusive results, this study aimed to assess whether rapid bladder decompression increased complications in adults with acute urinary retention. Materials and Methods: We searched the Cochrane Library, EMBASE, Google Scholar, and PubMed databases for articles published from the database inception to 31 August 2021. Studies that compared the effects and complication rates of rapid and gradual urinary decompression in adults with acute urinary retention were included. The primary outcome was post-decompression hematuria, while the secondary outcome was circulatory collapse. Meta-analyses were conducted using random effects models. Sensitivity analyses, tests for publication bias, and trial sequential analyses were conducted. The PROSPERO registration number is CRD42021233457. Results: Overall, four articles were included in the comprehensive analysis, and 435 participants met all the eligibility criteria. In the primary meta-analysis of all four study groups, rapid urinary decompression did not increase the risk of post-decompression hematuria (RR = 0.91; 95% CI: 0.62 to 1.35; p = 0.642). The I2 statistic was 0.0% (p = 0.732), indicating no substantial heterogeneity. In the meta-analysis of randomized controlled studies, the result did not change (RR = 0.89; 95% CI: 0.31 to 2.52; p = 0.824). The Egger’s test and Begg test (p = 0.339 and 0.497, respectively) indicated the absence of statistical evidence of publication bias. Leave-one-out sensitivity analysis was conducted and showed the pooled results were robust. In secondary outcome, there were no reported events of circulatory collapse in the current studies. Conclusions: The currently available data suggest that rapid urinary decompression is an effective and safe method with a complication rate similar to that of gradual decompression in an acute urinary retention population. Further large-scale randomized studies are required.

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Shevchuk, M. P., M. A. Dudchenko, D. M. Ivashchenko, M. I. Kravtsiv, and R. A. Prikhidko. "SYNDROME OF "RAPID" BILIARY DECOMPRESSION IN COMMON BILE DUCT OBSTRUCTION OF VARIOUS ETIOLOGY." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 21, no. 4 (2021): 94–97. http://dx.doi.org/10.31718/2077-1096.21.4.94.

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Surgical interventions performed on the background of obstructive jaundice are characterized by a significant risk of complications and characterized by high post-surgical mortality. The modern approach to the treatment of patients of this category regulates a certain sequence of treatment measures, provides for the implementation of minimally invasive interventions at the first stage of treatment. In some patients, despite adequate and minimally invasive bile excretion, the general condition can worsen. This condition, known as “rapid” biliary decompression syndrome, is a severe complication is difficult to treat and often fatal and requires further study. The aim of this study is to improve the results of treatment of patients with obstructive jaundice through timely prediction, early diagnosis and the use of effective therapy for the syndrome of "rapid" biliary decompression. The results of treatment of 76 patients with obstructive jaundice of various origins, who underwent 98 minimally invasive surgical interventions to eliminate biliary hypertension, were investigated. Complications of following the interventions were analyzed, including the “rapid” biliary decompression syndrome. Clinical and laboratory criteria and signs of “rapid” biliary decompression syndrome were formulated using the methods for calculating the rate of biliary decompression and the intensity of the decrease in bilirubin. When performing bile draining operations in patients with obstructive jaundice, one should expect the development of a syndrome of "rapid" biliary decompression. It can manifest both through individual signs and their combination that requires the immediate implementation of all the necessary therapeutic measures.

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L'Abbate, Antonio, Claudia Kusmic, Marco Matteucci, et al. "Gas embolization of the liver in a rat model of rapid decompression." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, no. 2 (2010): R673—R682. http://dx.doi.org/10.1152/ajpregu.00699.2009.

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Occurrence of liver gas embolism after rapid decompression was assessed in 31 female rats that were decompressed in 12 min after 42 min of compression at 7 ATA ( protocol A). Sixteen rats died after decompression ( group I). Of the surviving rats, seven were killed at 3 h ( group II), and eight at 24 h ( group III). In group I, bubbles were visible in the right heart, aortic arch, liver, and mesenteric veins and on the intestinal surface. Histology showed perilobular microcavities in sinusoids, interstitial spaces, and hepatocytes. In g roup II, liver gas was visible in two rats. Perilobular vacuolization and significant plasma aminotransferase increase were present. In g roup III, liver edema was evident at gross examination in all cases. Histology showed perilobular cell swelling, vacuolization, or hydropic degeneration. Compared with basal, enzymatic markers of liver damage increased significantly. An additional 14 rats were decompressed twice ( protocol B). Overall mortality was 93%. In addition to diffuse hydropic degeneration, centrilobular necrosis was frequently observed after the second decompression. Additionally, 10 rats were exposed to three decompression sessions ( protocol C) with doubled decompression time. Their mortality rate decreased to 20%, but enzymatic markers still increased in surviving rats compared with predecompression, and perilobular cell swelling and vacuolization were present in five rats. Study challenges were 1) liver is not part of the pathophysiology of decompression in the existing paradigm, and 2) although significant cellular necrosis was observed in few animals, zonal or diffuse hepatocellular damage associated with liver dysfunction was frequently demonstrated. Liver participation in human decompression sickness should be looked for and clinically evaluated.

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He, Wenbin, Xi Yang, Ding Luo, et al. "Decompression Load Analysis in Large Passenger Planes with Windshield Cracked." Aerospace 9, no. 9 (2022): 517. http://dx.doi.org/10.3390/aerospace9090517.

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To prevent the possible accident of a large passenger plane due to rapid decompression, transient load analysis is of vital importance in the assessment of structure strength and also an important clause of airworthiness standard. A 0-D isentropic model and a 1-D model based on the characteristic line are developed to simulate the rapid decompression process of the cockpit-cabin model due to a cracked windshield. The accuracy of these models is presented by comparing them with experiments and 3-D CFD simulations. Then, the 1-D model is applied to study the influence of cabin and cockpit volume, windshield and decompression panel area, compartments, and environment pressure on the decompression load. The non-dimensional decompression time and the non-dimensional decompression load are developed to evaluate the decompression characteristics, and the correlation equations are established. The relative deviation between the results of the correlation equation fit and the results of the one-dimensional simulation is less than 3%. This work provides a new engineering method for structure strength design and decompression load analysis with high accuracy and low resource consumption.

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Wang, Junwei, Lei Zhang, Guohua Li, et al. "Research and Verification of Key Techniques in the Simulation of Space Extremely Rapid Decompression in Millisecond." International Journal of Aerospace Engineering 2021 (April 5, 2021): 1–11. http://dx.doi.org/10.1155/2021/6634468.

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The research of rapid decompression with its effect assessment and protection technology is the problem that must be faced by the future exploration projects such as near space exploration, deep space exploration, and long-term lunar or Mars base. A new reusable quick opening mechanism which can be opened in millisecond is designed to meet the testing requirement of ground simulator for extremely rapid decompression, and the testing results show that the quick opening mechanism can be opened within 0.1 s. The mathematical formulation is also developed, and the comparisons with the results from the literature demonstrate its validity. The CFD simulation and the verification system are established for the airflow in the rapid decompression process under different opening degrees. The simulation results show that the effect of the opening on the decompression process is very obvious and the decompression time corresponding to 50%, 75%, and 100% opening is 479.1 ms, 320.7 ms, and 290.1 ms, respectively. The testing results also show a consistent trend which is 583 ms, 450 ms, and 384 ms, respectively, to reach the equilibrium state.

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Dromsky, David M., Charles B. Toner, Shalini Survanshi, Andreas Fahlman, Erich Parker, and Paul Weathersby. "Natural history of severe decompression sickness after rapid ascent from air saturation in a porcine model." Journal of Applied Physiology 89, no. 2 (2000): 791–98. http://dx.doi.org/10.1152/jappl.2000.89.2.791.

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We developed a swine model to describe the untreated natural history of severe decompression sickness (DCS) after direct ascent from saturation conditions. In a recompression chamber, neutered male Yorkshire swine were pressurized to a predetermined depth from 50–150 feet of seawater [fsw; 2.52–5.55 atmospheres absolute (ATA)]. After 22 h, they returned to the surface (1 ATA) at 30 fsw/min (0.91 ATA/min) without decompression stops and were observed. Depth was the primary predictor of DCS incidence ( R = 0.52, P < 0.0001) and death ( R = 0.54, P < 0.0001). Severe DCS, defined as neurological or cardiopulmonary impairment, occurred in 78 of 128 animals, and 42 of 51 animals with cardiopulmonary DCS died within 1 h after surfacing. Within 24 h, 29 of 30 survivors with neurological DCS completely resolved their deficits without intervention. Pretrial Monte Carlo analysis decreased subject requirement without sacrificing power. This model provides a useful platform for investigating the pathophysiology of severe DCS and testing therapeutic interventions. The results raise important questions about present models of human responses to similar decompressive insults.

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Alkhuzai, Ahmed. "Prospective Comparative Analysis Study of Minimally Invasive Surgical Decompressions versus Traditional Surgical Decompression of Carpal Tunnel Syndrome." Open Access Macedonian Journal of Medical Sciences 10, B (2022): 1882–88. http://dx.doi.org/10.3889/oamjms.2022.9900.

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Objective: Carpal tunnel syndrome (CTS) occurs when there is compression on the median nerve within the carpal tunnel in the wrist joint. It commonly affects those who make frequent, repetitive hand movements, with women being more affected than men. The present study was aimed at comparing minimally invasive and classical surgical decompression techniques for carpal tunnel syndrome. Materials and Methods: One hundred and four patients with CTS who were scheduled for carpal tunnel decompression were chosen at random. The patients were divided into two groups. Group A had standard classical long incision decompression, whereas Group B had minimally invasive decompression. The patients were evaluated with the Boston Functional Score Scale. Between the two groups, a comparison of visit reports was made at 3, 6, and 12 months after surgery. Results: There was no significant difference in patient age (Group A was 38 years old and Group B was 36 years old) and availability of bilateral CTS status (A was 34 and B was 31). The female-male ratios in groups A and B were 3.6 to 0.2 and 3.1 to 0.3, respectively, with a p-value of p<0.05 indicating significant difference. Comparing both groups, the minimally invasive decompression technique showed a high rate of functional hand grip and a low rate of complications including early wound healing, rapid resumption of palmar strength grip, and rapid return to daily activities. Conclusion: The findings indicate that patients who underwent the minimally invasive surgical decompression one-stitch technique showed significant improvement over the traditional method.

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Bhat, AbdulRashid, UdaySingh Raswan, and AltafRehman Kirmani. "Intracranial extradural hematoma: Spontaneous rapid decompression - not resolution." Journal of Pediatric Neurosciences 10, no. 3 (2015): 266. http://dx.doi.org/10.4103/1817-1745.165698.

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Qiu, Ning, and Robert E. Apfel. "Rapid decompression of seeded melts for materials processing." Review of Scientific Instruments 66, no. 5 (1995): 3337–43. http://dx.doi.org/10.1063/1.1145504.

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Zhang, Xian, Wei Leng, Ying Zhou, Ran Liu, and Junwei Wang. "Rapid Decompression Technology for the Oxygen Supply System." IOP Conference Series: Materials Science and Engineering 449 (November 29, 2018): 012018. http://dx.doi.org/10.1088/1757-899x/449/1/012018.

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Choy, Daniel S. J., Peter Wolf Ascher, William Liebler, et al. "Rapid Communication: Percutaneous Laser Decompression of Intervertebral Disc." Laser Medicine and Surgery News and Advances 7, no. 5 (1989): 25. http://dx.doi.org/10.1089/lms.1989.7.5.25.

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Noma, S., M. Shimoda, and I. Hayakawa. "Inactivation of Vegetative Bacteria by Rapid Decompression Treatment." Journal of Food Science 67, no. 9 (2002): 3408–11. http://dx.doi.org/10.1111/j.1365-2621.2002.tb09598.x.

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Linnarsson, D., H. Ornhagen, M. Gennser, and H. Berg. "Breathing volumes and gas exchange during simulated rapid free ascent from 100 msw." Journal of Applied Physiology 74, no. 3 (1993): 1293–98. http://dx.doi.org/10.1152/jappl.1993.74.3.1293.

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The crew of a disabled submarine can be rescued by means of free ascent through the water to the surface. Pulmonary gas exchange was studied during simulated rapid free ascent in subjects standing immersed to the neck in a pressure chamber. The pressure was rapidly increased to 1.1 MPa [100 meters seawater (msw)] followed by decompression at 0.03 MPa/s (3 msw/s). Effective inspired tidal volume, as estimated by an Ar dilution method, fell gradually to zero during decompression from 20 to 0 msw. Directly determined expired tidal volumes were increased up to two to three times at the time of return to surface pressure compared with pre- and postdecompression volumes. End-tidal PCO2 was increased on compression and fell to a nadir of 3.4 kPa (25 Torr) at the time of return to surface pressure. Thus, intrapulmonary gas expansion caused simultaneous inspiratory hypoventilation and expiratory hyperventilation. If O2-enriched gas is to be used to reduce the risk of decompression sickness, it should be administered early during decompression to alter the intrapulmonary gas composition. The time course of arterial PCO2 changes as reflected by end-tidal values during short-lasting compression/decompression would act to promote inert gas supersaturation in the brain.

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Park, Chan Beum, and Douglas S. Clark. "Rupture of the Cell Envelope by Decompression of the Deep-Sea Methanogen Methanococcus jannaschii." Applied and Environmental Microbiology 68, no. 3 (2002): 1458–63. http://dx.doi.org/10.1128/aem.68.3.1458-1463.2002.

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ABSTRACT The effect of decompression on the structure of Methanococcus jannaschii, an extremely thermophilic deep-sea methanogen, was studied in a novel high-pressure, high-temperature bioreactor. The cell envelope of M. jannaschii appeared to rupture upon rapid decompression (ca. 1 s) from 260 atm of hyperbaric pressure. When decompression from 260 atm was performed over 5 min, the proportion of ruptured cells decreased significantly. In contrast to the effect produced by decompression from hyperbaric pressure, decompression from a hydrostatic pressure of 260 atm did not induce cell lysis.

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Beirão, Bernardo V., Luiz G. M. Silva, Richard S. Brown, and Ricardo W. Walker. "Determining barotrauma in the Pictus catfish, Pimelodus pictus, experimentally exposed to simulated hydropower turbine passage." Marine and Freshwater Research 69, no. 12 (2018): 1913. http://dx.doi.org/10.1071/mf18142.

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Hydropower development poses severe threats to the aquatic diversity and ecosystem services. One such threat is the exposure of fish to extreme conditions within hydropower facilities. Fish may suffer rapid decompression when passing through turbines or when entering the draft tubes, which can lead to barotrauma and mortality. We aimed to evaluate the effects of rapid decompression on the Amazonian benthic species Pimelodus pictus (Pictus catfish), by simulating in hypo–hyperbaric chambers. The most frequent injuries in Pictus catfish exposed to simulated rapid decompression were swim-bladder rupture, intestine rupture, internal haemorrhage and embolism. The occurrence and magnitude of internal haemorrhaging and emboli were related to the ratio of pressure change and the decompression timespan, whereas swim-bladder rupture occurred even at relatively low ratios. Emboli was present almost entirely among fish with a ruptured swim bladder. Importantly, all fish were negatively buoyant before exposure to decompression, posing challenges to data analysis. Therefore, barotrauma studies with benthic fish species are deemed to be challenging and are likely to require the use of complementary approaches. Research is needed to understand the state of buoyancy of benthic fish in the wild and to develop methods to accurately replicate these in a controlled testing environment.

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Zhang, Lei, Xiao Han, Xinbin Zhang, and Jihong Yan. "Door-Triggering Mechanism for Large-Scale Rapid-Decompression Experiments." International Journal of Aerospace Engineering 2020 (August 1, 2020): 1–9. http://dx.doi.org/10.1155/2020/6841651.

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For large-scale rapid-decompression experiments, a new door-triggering mechanism is proposed for a 750 mm diameter pressure relief channel. Quick opening of the door is realized by utilizing a spring-based release mechanism to instantly convert large amounts of elastic potential energy into kinetic energy. To counteract the significant inertial effect of the high-speed door on the chamber, a flywheel-based cushioning mechanism is designed to absorb the kinetic energy of the door after opening. This carefully designed mechanism consists of the closing mechanism, energy storage unit, locking/releasing mechanism, and cushioning mechanism. Kinetic models are established to analyze the dynamic properties. Simulation results reveal that it takes approximately 280 ms for the door to open from 0° to 90°. This work can provide insights for the development of large-scale rapid-decompression equipment in the future.

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Chen, Ye, Elizabeth Montcalm-Smith, Christine Schlaerth, Charles Auker, and Richard M. McCarron. "Acclimation to decompression: stress and cytokine gene expression in rat lungs." Journal of Applied Physiology 111, no. 4 (2011): 1007–13. http://dx.doi.org/10.1152/japplphysiol.01402.2010.

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Previous studies demonstrated that animals exposed to repeated compression-decompression stress acclimated (i.e., developed reduced susceptibility) to rapid decompression. This study endeavored to characterize inflammatory and stress-related gene expression and signal transduction associated with acclimation to rapid decompression. Rats were divided into four groups: 1) control-sham: pressure naïve rats; 2) acclimation-sham: nine acclimation dives [70 feet seawater (fsw), 30 min]; 3) control-dive: test dive only (175 fsw, 60 min); and 4) acclimation-dive: nine acclimation dives and a test dive. After the test dive, rats were observed for decompression sickness (DCS). Expression of 13 inflammatory and stress-related genes and Akt (or PKB, a serine/threonine protein kinase) and MAPK phosphorylation of lung tissue were examined. The expression of immediate early gene/transcription factor early growth response gene 1 (Egr-1) was observed in both control and acclimation animals with DCS but not in animals without DCS. Increased Egr-1 in control-dive animals with DCS was significantly greater than in acclimation-dive animals with DCS. TNF-α, IL-1β, IL-6, and IL-10 were significantly elevated in control-DCS animals. Acclimation-DCS animals had increased TNF-α, but there was no change in IL-1β, IL-6, and IL-10. High levels of Akt phosphorylation were observed in lungs of acclimation-sham, acclimation-dive, and control-dive animals; phosphorylated ERK1/2 was only observed in animals with DCS. This study suggests that activation of ERK1/2 and upregulation of Egr-1 and its target cytokine genes by rapid decompression may play a role in the initiation and progression of DCS. It may be that the downregulated expression of these genes in animals with DCS is associated with previous exposure to repeated compression-decompression cycles. This study represents an initial step toward understanding the molecular mechanisms associated with acclimation to decompression.

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Etafy, MohamedH, FatmaH Saleh, Cervando Ortiz-Vanderdys, et al. "Rapid versus gradual bladder decompression in acute urinary retention." Urology Annals 9, no. 4 (2017): 339. http://dx.doi.org/10.4103/0974-7796.216320.

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TODA, Saburo, Kazuhisa OKADA, Yutaka HORI, and Masaaki KUROKAWA. "Rapid Vapor Bubble Growth during Decompression of Superheated Water." JSME international journal. Ser. 2, Fluids engineering, heat transfer, power, combustion, thermophysical properties 31, no. 3 (1988): 520–28. http://dx.doi.org/10.1299/jsmeb1988.31.3_520.

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Wang, Shugang, Derek Elsworth, and Jishan Liu. "Rapid decompression and desorption induced energetic failure in coal." Journal of Rock Mechanics and Geotechnical Engineering 7, no. 3 (2015): 345–50. http://dx.doi.org/10.1016/j.jrmge.2015.01.004.

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Wheeler, R. M., C. A. Wehkamp, M. A. Stasiak, M. A. Dixon, and V. Y. Rygalov. "Plants survive rapid decompression: Implications for bioregenerative life support." Advances in Space Research 47, no. 9 (2011): 1600–1607. http://dx.doi.org/10.1016/j.asr.2010.12.017.

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Bass, Thomas, Larry E. White, Richard D. Wood, Alice L. Werner, and Frank P. Schinco. "Rapid Decompression of Congenital Hydrocephalus Associated with Parenchymal Hemorrhage." Journal of Neuroimaging 5, no. 4 (1995): 249–51. http://dx.doi.org/10.1111/jon199554249.

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Pflugrath, Brett, Ryan Harnish, Briana Rhode, et al. "The Susceptibility of Juvenile American Shad to Rapid Decompression and Fluid Shear Exposure Associated with Simulated Hydroturbine Passage." Water 12, no. 2 (2020): 586. http://dx.doi.org/10.3390/w12020586.

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Throughout many areas of their native range, American shad (Alosa sapidissima) and other Alosine populations are in decline. Though several conditions have influenced these declines, hydropower facilities have had significant negative effects on American shad populations. Hydropower facilities expose ocean-migrating American shad to physical stressors during passage through hydropower facilities, including strike, rapid decompression, and fluid shear. In this laboratory-based study, juvenile American shad were exposed separately to rapid decompression and fluid shear to determine their susceptibility to these stressors and develop dose–response models. These dose–response relationships can help guide the development and/or operation of hydropower turbines and facilities to reduce the negative effects to American shad. Relative to other species, juvenile American shad have a high susceptibility to both rapid decompression and fluid shear. Reducing or preventing exposure to these stressors at hydropower facilities may be a potential method to assist in the effort to restore American shad populations.

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Tanoue, K., Y. Mano, K. Kuroiwa, H. Suzuki, M. Shibayama, and H. Yamazaki. "Consumption of platelets in decompression sickness of rabbits." Journal of Applied Physiology 62, no. 5 (1987): 1772–79. http://dx.doi.org/10.1152/jappl.1987.62.5.1772.

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Platelet behavior was studied in rabbit decompression sickness which was brought about by the exposure to 6 ATA for 40 min (bottom time) followed by rapid decompression. Platelet counts significantly decreased after the decompression. Kinetic studies with 111In-oxine-labeled platelets revealed shortened survivals of circulating platelets, and audioradiograms indicated the accumulation of radioactivity in the lungs after the decompression. Although there was no change in the mode volume of platelets after the decompression, the transient appearance of circulating smaller or fragmented platelets suggested a random overdestruction of platelets. Whole and releasable adenine nucleotide contents of platelets were decreased significantly after the decompression. There were no significant changes in cytoplasmic adenine nucleotide contents. Therefore, in decompression sickness, the circulating platelets behaved similarly to those in acquired storage pool disease. Platelet thrombi were found in the pulmonary arteries, compatible with the accumulation of 111In-oxine-labeled platelets. These findings suggest that circulating air bubbles interact with platelets, causing the platelet release reaction, and these activated platelets participate in the formation of thrombi in experimental decompression sickness.

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Montcalm-Smith, E. A., R. M. McCarron, W. R. Porter, R. S. Lillo, J. T. Thomas, and C. R. Auker. "Acclimation to decompression sickness in rats." Journal of Applied Physiology 108, no. 3 (2010): 596–603. http://dx.doi.org/10.1152/japplphysiol.00596.2009.

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Protection against decompression sickness (DCS) by acclimation to hyperbaric decompression has been hypothesized but never proven. We exposed rats to acclimation dives followed by a stressful “test” dive to determine whether acclimation occurred. Experiments were divided into two phases. Phase 1 rats were exposed to daily acclimation dives of hyperbaric air for 30 min followed by rapid decompression on one of the following regimens: 70 ft of seawater (fsw) for 9 days (L70), 70 fsw for 4 days (S70), 40 fsw for 9 days (L40), 40 fsw for 4 days (S40), or unpressurized sham exposure for 9 days (Control). On the day following the last exposure, all were subjected to a “test” dive (175 fsw, 60 min, rapid decompression). Both L70 and S70 rats had significantly lower incidences of DCS than Control rats (36% and 41% vs. 62%, respectively). DCS incidences for the other regimens were lower than in Control rats but without statistical significance. Phase 2 used the most protective regimen from phase 1 (L70); rats were exposed to L70 or a similar regimen with a less stressful staged decompression. Another group was exposed to a single acclimation dive (70 fsw/30 min) on the day before the test dive. We observed a nonsignificant trend for the rapidly decompressed L70 dives to be more protective than staged decompression dives (44% vs. 51% DCS incidence). The single acclimation dive regimen did not provide protection. We conclude that protection against DCS can be attained with acclimating exposures that do not themselves cause DCS. The deeper acclimation dive regimens (70 fsw) provided the most protection.

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FUJII, Hitoshi, Akiko FUJIWARA, Nobuyuki WAKABAYASHI, and Yutaka ABE. "Bubble Forming Behavior of High Viscous Fluid under Rapid Decompression." Transaction of the Visualization Society of Japan 28, no. 5 (2008): 27–32. http://dx.doi.org/10.3154/tvsj.28.27.

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Thompson, Matthew B., Jan Bon, Mike Vatan, and Mohammad Zaman. "Effects of rapid gas decompression on coal seam gas swellables." APPEA Journal 62, no. 2 (2022): S187—S191. http://dx.doi.org/10.1071/aj21166.

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Thousands of non-stimulated coal seam gas (CSG) wells in Queensland’s Surat Basin rely on swellable packers as the first line of defence against interburden solids production and poor well run life. This paper is aimed at understanding some of the impacts of long-term well operations on swellable performance from rapid changes in downhole pressure. For the first time, rapid gas decompression (RGD) effects on CSG swellables were experimented on in a quantitative manner as an analogue to underbalanced workover and pump trip conditions. RGD has the potential to break down swellables due to rapid release of high-pressure methane diffused in the rubber matrix resulting in a flow path for interburden solids. Five commonly available swellables from the CSG market were lab-tested for rapid decompression with methane at operational conditions. Coupon samples were swollen to representative conditions and placed in an autoclave under high-pressure methane, then rapidly decompressed in cycles. Results of this study showed relatively low levels of physical degradation under test conditions but shrinkage effects varied between products largely grouped into material properties, confirmed with separate ambient shrinkage test. As such, the focus on swellable placement geometry remains paramount.

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RUZIBOYZODA, K. R. "DIAGNOSIS AND TREATMENT OF «RAPID DECOMPRESSION SYNDROME» IN OBSTRUCTIVE JAUNDICE." Bulletin of Pirogov National Medical & Surgical Center 17, no. 2 (2022): 36–39. http://dx.doi.org/10.25881/20728255_2022_17_2_36.

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Ogasawara, Kuniaki, Keiji Koshu, Takashi Yoshimoto, and Akira Ogawa. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 45, no. 3 (1999): 484–89. http://dx.doi.org/10.1097/00006123-199909000-00014.

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ISIKAWA, Chihiro, Yutaka ABE, and Akiko KANEKO. "20701 Research on High Viscous Fluids Behavior under Rapid Decompression." Proceedings of Conference of Kanto Branch 2013.19 (2013): 361–62. http://dx.doi.org/10.1299/jsmekanto.2013.19.361.

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Lin, Chuanlong, Jesse S. Smith, Stanislav V. Sinogeikin, and Guoyin Shen. "Experimental evidence of low-density liquid water upon rapid decompression." Proceedings of the National Academy of Sciences 115, no. 9 (2018): 2010–15. http://dx.doi.org/10.1073/pnas.1716310115.

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Water is an extraordinary liquid, having a number of anomalous properties which become strongly enhanced in the supercooled region. Due to rapid crystallization of supercooled water, there exists a region that has been experimentally inaccessible for studying deeply supercooled bulk water. Using a rapid decompression technique integrated with in situ X-ray diffraction, we show that a high-pressure ice phase transforms to a low-density noncrystalline (LDN) form upon rapid release of pressure at temperatures of 140–165 K. The LDN subsequently crystallizes into ice-Ic through a diffusion-controlled process. Together with the change in crystallization rate with temperature, the experimental evidence indicates that the LDN is a low-density liquid (LDL). The measured X-ray diffraction data show that the LDL is tetrahedrally coordinated with the tetrahedral network fully developed and clearly linked to low-density amorphous ices. On the other hand, there is a distinct difference in structure between the LDL and supercooled water or liquid water in terms of the tetrahedral order parameter.

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Alidibirov, M., and D. B. Dingwell. "Three fragmentation mechanisms for highly viscous magma under rapid decompression." Journal of Volcanology and Geothermal Research 100, no. 1-4 (2000): 413–21. http://dx.doi.org/10.1016/s0377-0273(00)00149-9.

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Kim, Choong-Hyun. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 47, no. 3 (2000): 791. http://dx.doi.org/10.1227/00006123-200009000-00057.

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Ogasawara, Kuniaki, Akira Ogawa, and Takashi Yoshimoto. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 47, no. 3 (2000): 792. http://dx.doi.org/10.1227/00006123-200009000-00058.

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Falero, Raul Andrés Pérez, Angeles de Lourdes Cardentey Pereda, and Ivan Francisco Arenas Rodríguez. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 47, no. 6 (2000): 1468. http://dx.doi.org/10.1093/neurosurgery/47.6.1468.

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Ogasawara, Kuniaki, Akira Ogawa, and Takashi Yoshimoto. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 47, no. 6 (2000): 1468. http://dx.doi.org/10.1093/neurosurgery/47.6.1468a.

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Kim, Choong-Hyun. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 47, no. 3 (2000): 791. http://dx.doi.org/10.1097/00006123-200009000-00057.

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Ogasawara, Kuniaki, Akira Ogawa, and Takashi Yoshimoto. "Transient Hyperemia Immediately after Rapid Decompression of Chronic Subdural Hematoma." Neurosurgery 47, no. 3 (2000): 792. http://dx.doi.org/10.1097/00006123-200009000-00058.

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