Littérature scientifique sur le sujet « Radiation Induced Activation »
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Articles de revues sur le sujet "Radiation Induced Activation"
TAKAMASA, Tomoji, Koji OKAMOTO, Kaichiro MISHIMA et Masahiro FURUYA. « Radiation Induced Surface Activation ». Journal of the Atomic Energy Society of Japan / Atomic Energy Society of Japan 45, no 2 (2003) : 112–17. http://dx.doi.org/10.3327/jaesj.45.112.
Texte intégralKIKUCHI, Takayoshi, Yoshinori HIROSE, Tatsuya HAZUKU et Tomoji TAKAMASA. « ICONE15-10316 EFFECT OF RADIATION INDUCED SURFACE ACTIVATION ON SURFACE WETTABILITY IN HIGH-TEMPERATURE CONDITION ». Proceedings of the International Conference on Nuclear Engineering (ICONE) 2007.15 (2007) : _ICONE1510. http://dx.doi.org/10.1299/jsmeicone.2007.15._icone1510_160.
Texte intégralYang, Nong, Xican Gao, Xiaofei Qu, Ruiguang Zhang, Fan Tong, Qian Cai, Jihua Dong, Yu Hu, Gang Wu et Xiaorong Dong. « PIDD Mediates Radiation-Induced Microglia Activation ». Radiation Research 186, no 4 (octobre 2016) : 345–59. http://dx.doi.org/10.1667/rr14374.1.
Texte intégralDong, X., M. Luo, J. Dong et G. Wu. « The Mechanism of Radiation-induced Microglia Activation ». International Journal of Radiation Oncology*Biology*Physics 78, no 3 (novembre 2010) : S632—S633. http://dx.doi.org/10.1016/j.ijrobp.2010.07.1473.
Texte intégralRedd, Priscilla S., Kankana Bardhan, May R. Chen, Amy V. Paschall, Chunwan Lu, Roni J. Bollag, Fengchong Kong et al. « NF-κB acts as a molecular link between tumor cells and Th1/Tc1 T cells in the tumor microenvironment to exert radiation-mediated tumor suppression. » Journal of Immunology 196, no 1_Supplement (1 mai 2016) : 213.3. http://dx.doi.org/10.4049/jimmunol.196.supp.213.3.
Texte intégralJudge, J. L., K. M. Owens, S. J. Pollock, C. F. Woeller, T. H. Thatcher, J. P. Williams, R. P. Phipps, P. J. Sime et R. M. Kottmann. « Ionizing radiation induces myofibroblast differentiation via lactate dehydrogenase ». American Journal of Physiology-Lung Cellular and Molecular Physiology 309, no 8 (15 octobre 2015) : L879—L887. http://dx.doi.org/10.1152/ajplung.00153.2015.
Texte intégralYacoub, Adly, Anna Miller, Ruben W. Caron, Liang Qiao, David A. Curiel, Paul B. Fisher, Michael P. Hagan, Steven Grant et Paul Dent. « Radiotherapy-induced signal transduction ». Endocrine-Related Cancer 13, Supplement_1 (décembre 2006) : S99—S114. http://dx.doi.org/10.1677/erc.1.01271.
Texte intégralFURUYA, Masahiro, Tomoji TAKAMASA, Koji OKAMOTO, David T. YASUNAGA et Susumu UEMATSU. « Corrosion Control Based on Radiation Induced Surface Activation ». Journal of The Japan Institute of Marine Engineering 41, no 2 (2006) : 278–84. http://dx.doi.org/10.5988/jime.41.2_278.
Texte intégralXUE, JUN, JI-HUA DONG, GUO-DONG HUANG, XIAO-FEI QU, GANG WU et XIAO-RONG DONG. « NF-κB signaling modulates radiation-induced microglial activation ». Oncology Reports 31, no 6 (23 avril 2014) : 2555–60. http://dx.doi.org/10.3892/or.2014.3144.
Texte intégralHONJO, Yoshio, Masahiro FURUYA, Tomoji TAKAMASA et Koji OKAMOTO. « Mechanism of Hydrophilicity by Radiation-Induced Surface Activation ». Journal of Power and Energy Systems 3, no 1 (2009) : 216–27. http://dx.doi.org/10.1299/jpes.3.216.
Texte intégralThèses sur le sujet "Radiation Induced Activation"
Sander, David [Verfasser]. « Radiation-Induced Activation of the Epidermal Growth Factor Receptor in Lung Cancer / David Sander ». Hamburg : Staats- und Universitätsbibliothek Hamburg, 2011. http://d-nb.info/1010749641/34.
Texte intégralMeziani, Lydia. « Study of Interaction Between the Inflammatory Response and Radiation-Induced Fibrosis ». Thesis, Paris 11, 2015. http://www.theses.fr/2015PA11T041.
Texte intégralRadiation-induced fibrosis (RIF) is a delayed complication of radiotherapy often associated with chronic inflammatory process and macrophage infiltration. Nowadays, macrophages are suggested to be important cellular contributors to fibrogenic process, but their implication in the context of RIF has never been investigated. In a previous study we have shown that irradiation (IR) induced the polarization of cardiac macrophages into M1 in ApoE-/- mice and was associated with a high fibrosis score in ApoE-/- mice, suggesting that macrophage polarization could drive tissue sensitivity to ionizing radiation. This observation prompted us to investigate the role of macrophages in RIF using a classical experimental model of lung fibrosis developed in C57Bl/6 mice after 16Gy thorax-IR. We profiled both alveolar macrophages (AM) and interstitial macrophages (IM). During the acute phase we found AM depletion associated with CXCL1, MCP-1 and M-CSF secretion, followed by a repopulation phase mediated by recruitment and proliferation of monocytes/macrophages from the bone marrow. Interestingly, the newly recruited AM exhibited a yet never described hybrid polarization (M1/M2), associated with the up-regulation of both Th1 and Th2 cytokines. At delayed times points, IM were M2-polarized and associated with downregulation of Th1 cytokines and upregulation of Th2 cytokines in tissue lysates. These results suggest a differential contribution of hybrid AM vs M2 IM to fibrogenesis. Interestingly, in contrast to activated hybrid AM, activated M2 IM were able to induce fibroblast activation in vitro mediated by an enhanced TGF-β1 expression. Therefore, specific depletion of hybrid AM using intranasal administration of clodrosome increased RIF score and enhanced M2 IM infiltration. We next evaluated if the fibrogenic process can in turn affect macrophage polarization. Interestingly, after coculture of irradiated fibroblast with non-irradiated pulmonary macrophages, secretion of cytokines such as M-CSF and TIMP-1, which can stimulate macrophage activation, was observed. Furthermore, RIF inhibition using pravastatin treatment showed that fibrosis inhibition was associated with a decrease in M2 IM accompanied by an increase in M1 IM, but had no effect on polarization of AM. These present study shows a dual and opposite contribution of alevolar versus intertitial macrophages in RIF and the contribution of the fibrogenic process to IM polarization, resulting thereby in a chronical fibrogenic loop
Gibhardt, Christine S. [Verfasser], Gerhard [Akademischer Betreuer] Thiel et Marco [Akademischer Betreuer] Durante. « Radiation induced activation of potassium-channels : The role of ROS and calcium / Christine S. Gibhardt. Betreuer : Gerhard Thiel ; Marco Durante ». Darmstadt : Universitäts- und Landesbibliothek Darmstadt, 2014. http://d-nb.info/1110979487/34.
Texte intégralChishti, Arif Ali [Verfasser]. « The role of linear energy transfer in modulating radiation- induced NF-kB activation and its down-stream target genes / Arif Ali Chishti ». Bonn : Universitäts- und Landesbibliothek Bonn, 2014. http://d-nb.info/1057915106/34.
Texte intégralSheridan, Andrew Keith. « Kinetics and temperature- and pressure-induced polymorphic phase transformations in molecular crystals ». Thesis, King's College London (University of London), 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322597.
Texte intégralFahy, Katherine Erin. « Thermal Burn Injury Induced Microvesicle Particle Release ». Wright State University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=wright149383031006972.
Texte intégralLiu, Langni. « Ultraviolet-B radiation induces release of bioactive microvesicle particles in keratinocytes via platelet-activating factor and acid sphingomyelinase ». Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1598672100210086.
Texte intégralGibhardt, Christine S. « Radiation induced activation of potassium-channels : The role of ROS and calcium ». Phd thesis, 2014. https://tuprints.ulb.tu-darmstadt.de/4227/1/Doktorarbeit_Endversion.pdf.
Texte intégralLiu, Kuang-Hsin, et 柳光欣. « Curcumin Enhances Radiation-induced Cytotoxicity Via Suppression of NF-kB Activation in Breast Cancer MCF-7 Cells ». Thesis, 2014. http://ndltd.ncl.edu.tw/handle/87548230918557977897.
Texte intégral中臺科技大學
醫學影像暨放射科學系暨研究所
102
Breast cancer is the most common malignancy in women around the world. Radiotherapy is one of the treatment modality used in multidisciplinary strategy against breast cancer nowadays. Recurrence after radiotherapy is not uncommon and it may be resulted from induced radioresistance. Radiation-induced NF-B activity can upregulate anti-apoptotic genes in irradiated breast cancer cells. Curcumin is plant phytochemical derived from the root of Curcuma longa and known for the inhibitory effect on NF-B activity. The main goal of this study is to investigate whether curcumin is able to radiosensitize breast cancer cells through suppression of radiation-induced NF-B activity. In this study, the effects of curcumin combined with radiation on cell proliferation in MCF-7 cells were evaluated with clonogenic formation assay. Whether curcumin can increase radiation-induced apoptosis was investigated by using DNA fragmentation assay. We also determined the effects of curcumin on radiation-induced NF-B activity and increased the expression of NF-B regulated gene products with Western blotting assay. To confirm whether NF-B inhibition can sensitize breast cancer to radiation, the effects of NF-B inhibitor combined with radiation in MCF-7 cell were evaluated in this study. These results showed that curcumin can significantly increase radiation-inhibited cell proliferation as compared to radiation alone. Curcumin also enhanced radiation-induced cell apoptosis via dependent and independent mitochondrial pathways. We also found curcumin decreased radiation activated NF-B activity and increased the expression of NF-B regulated gene products (Bcl-2 and C-FLIP) from Western blotting assay. In addition, the results also showed NF-B inhibitor can increase radiosensitivity in MCF-7 cell.
Livres sur le sujet "Radiation Induced Activation"
Sobolev. Role of Cyclic Nucleotides in Radiobiology/The Functional Role of Radiation-induced Activation of Cholesterol and Lipogenesis in the Animal Body. Routledge, 1997.
Trouver le texte intégralHenriksen, Niels Engholm, et Flemming Yssing Hansen. Unimolecular Reactions. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198805014.003.0007.
Texte intégralChapitres de livres sur le sujet "Radiation Induced Activation"
Busi, F. « Redox Properties of Radiation Induced Free Radicals ». Dans Selective Activation of Drugs by Redox Processes, 63–78. Boston, MA : Springer US, 1990. http://dx.doi.org/10.1007/978-1-4615-3768-7_6.
Texte intégralKemp, C. J., R. Bremner et A. Balmain. « Molecular Genetics of Oncogene Activation and Gene Loss in Carcinogen-Induced Animal Tumours ». Dans Anticarcinogenesis and Radiation Protection 2, 117–22. Boston, MA : Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3850-9_17.
Texte intégralTang, D. G., C. A. Diglio et K. V. Honn. « 12(S)-Hete-Induced Microvascular Endothelial Cell Retraction is Mediated by Cytoskeletal Rearrangement Dependent on PKC Activation ». Dans Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury, 219–29. Boston, MA : Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_44.
Texte intégralFrohns, A., et F. Frohns. « Safety of Water-Filtered Infrared A (wIRA) on the Eye as a Novel Treatment Option for Chlamydial Infections ». Dans Water-filtered Infrared A (wIRA) Irradiation, 259–69. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92880-3_22.
Texte intégralSola, J., et L. Vila. « Platelet-Activating Factor Induces Receptor Mediated Leukocyte 5 Lipoxygenase Activation Through its Translocation to the Membrane ». Dans Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury, 35–38. Boston, MA : Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_8.
Texte intégralHanahan, D. J., L. Gregory, A. Tokumura, T. Nouchi et M. S. Olson. « Some Novel Observations on Inhibitors of Platelet Activating Factor Induced Reactions ». Dans Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury, 731–33. Boston, MA : Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_141.
Texte intégral« Photochemical and Radiation-Induced Activation of CO2 in Homogeneous Media ». Dans Greenhouse Gas Carbon Dioxide Mitigation, 411–30. CRC Press, 1998. http://dx.doi.org/10.1201/9781482227833-17.
Texte intégralZHIVOTOVSKY, B. D., et A. A. SEILIEV. « ROLE OF GENOME ACTIVATION PRODUCTS IN RADIATION-INDUCED INTERPHASE DEATH OF LYMPHOID CELLS ». Dans Radiation Research : A Twentieth-century Perspective, 216. Elsevier, 1991. http://dx.doi.org/10.1016/b978-0-12-168561-4.50774-5.
Texte intégralSARASIN, A., C. ROBERT, C. DROUGARD et L. DAYA-GROSJEAN. « ONCOGENE ACTIVATION IN ULTRAVIOLET-INDUCED SKIN TUMORS FROM NORMAL INDIVIDUALS AND XERODERMA PIGMENTOSUM PATIENTS ». Dans Radiation Research : A Twentieth-century Perspective, 42. Elsevier, 1991. http://dx.doi.org/10.1016/b978-0-12-168561-4.50068-8.
Texte intégralSloan, Steven R., et Angel Pellicer. « ras ONCOGENE ACTIVATION IN GAMMA- AND NEUTRON-RADIATION INDUCED MURINE THYMIC LYMPHOMAS ». Dans Congress Proceedings, 353–57. Elsevier, 1992. http://dx.doi.org/10.1016/b978-0-12-168562-1.50065-3.
Texte intégralActes de conférences sur le sujet "Radiation Induced Activation"
Honjo, Yoshio, Masahiro Furuya, Tomoji Takamasa et Koji Okamoto. « Interfacial Phenomena of Radiation-Induced and Photo-Induced ». Dans 16th International Conference on Nuclear Engineering. ASMEDC, 2008. http://dx.doi.org/10.1115/icone16-48320.
Texte intégralWennerberg, Erik, Noriko Kawashima et Sandra Demaria. « Abstract A21 : Adenosine is a rheostat for radiation therapy-induced immune activation ». Dans Abstracts : AACR Special Conference : Tumor Immunology and Immunotherapy : A New Chapter ; December 1-4, 2014 ; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-a21.
Texte intégralTamura, Naohisa, Tatsuya Hazuku, Tomoji Takamasa, Kaichiro Mishima, Toru Mitsutake et Sinichi Morooka. « Effect of Wall Surface Wettability Due to Radiation Induced Surface Activation on Quenching Process ». Dans 12th International Conference on Nuclear Engineering. ASMEDC, 2004. http://dx.doi.org/10.1115/icone12-49420.
Texte intégralAbernathy, Lisa M., Matthew D. Fountain, John M. David, Christopher K. Yunker, Michael C. Joiner et Gilda G. Hillman. « Abstract 277 : Radioprotection of lung tissue involves modulation of radiation-induced macrophage activation by soy isoflavones ». Dans Proceedings : AACR 106th Annual Meeting 2015 ; April 18-22, 2015 ; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-277.
Texte intégralGasparyan, Levon, Grigory Brill et Anu Makela. « Activation of VEGF and FGF induced angiogenesis under influence of low level laser radiation in vitro ». Dans Biomedical Optics 2006, sous la direction de Michael R. Hamblin, Ronald W. Waynant et Juanita Anders. SPIE, 2006. http://dx.doi.org/10.1117/12.645936.
Texte intégralChen, Genwen, Qianqian Zhao, Baoying Yuan et Zhaochong Zeng. « PWE-1 ALKBH5-modified HMGB1-STING activation contributes to radiation induced liver disease via innate immune response ». Dans Abstracts of the BSG Annual Meeting, 8–12 November 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-bsg.191.
Texte intégralZhang, Jian, Kaichiro Mishima, Naoki Sugitani, Masahiro Hino et Tomoji Takamasa. « Radiation Induced Surface Activation on SUS304 Rod Surface Coated With Thin Film Layer Formed by Vacuum Evaporation Method ». Dans 16th International Conference on Nuclear Engineering. ASMEDC, 2008. http://dx.doi.org/10.1115/icone16-48192.
Texte intégralVeeraraghavan, Jamunarani, Mohan Natarajan, Vibhudutta Awasthi, Terence S. Herman et Natarajan Aravindan. « Abstract 4203 : EF24, a novel curcumin analogue inhibits oncogenic activation induced by radiation abscopal effect in distant mice lungs ». Dans Proceedings : AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011 ; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4203.
Texte intégralBrannon, James H. « Chemical Etching of Silicon by CO2 Laser Induced Dissociation of NF3 ». Dans Microphysics of Surfaces, Beams, and Adsorbates. Washington, D.C. : Optica Publishing Group, 1987. http://dx.doi.org/10.1364/msba.1987.mb3.
Texte intégralWalker, Joshua M., Diego M. Barragan, Melissa J. Kasiewicz, Charles R. Thomas et William L. Redmond. « Abstract 1664 : Alpha-tocopheryloxyacetic acid (aTEA) induced immune activation synergizes with radiation therapy to treat primary and metastatic murine mammary carcinoma ». Dans Proceedings : AACR 107th Annual Meeting 2016 ; April 16-20, 2016 ; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1664.
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