Littérature scientifique sur le sujet « Pulmonary arterial hyperreactivity »
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Articles de revues sur le sujet "Pulmonary arterial hyperreactivity"
Cardouat, G., M. Douard, L. Roubenne, C. Bouchet, Z. Kmecová, P. Robillard, C. Guignabert et al. « NGF induces pulmonary arterial hyperreactivity through Connexin 43 increased expression ». Revue des Maladies Respiratoires 38, no 6 (juin 2021) : 582. http://dx.doi.org/10.1016/j.rmr.2021.02.029.
Texte intégralCardouat, G., M. Douard, L. Roubenne, S. Kmecova, P. Robillard, C. Guignabert, L. Tu et al. « NGF induces pulmonary arterial hyperreactivity through Connexin 43 increased expression ». Archives of Cardiovascular Diseases Supplements 12, no 2-4 (octobre 2020) : 213. http://dx.doi.org/10.1016/j.acvdsp.2020.03.036.
Texte intégralBouchet, Clement, Guillaume Cardouat, Matthieu Douard, Paul Robillard, Roger Marthan, Christelle Guibert et Véronique Freund-Michel. « Short-term mechanisms activated by NGF to induce pulmonary arterial hyperreactivity ». Archives of Cardiovascular Diseases Supplements 14, no 2 (juin 2022) : 157–58. http://dx.doi.org/10.1016/j.acvdsp.2022.04.147.
Texte intégralBouchet, C., G. Cardouart, M. Douard, Z. Kmecova, P. Robillard, F. Delcambre, R. Marthan, P. Berger, C. Guibert et V. Freund-Michel. « Short-term mechanisms activated by NGF to induce pulmonary arterial hyperreactivity ». Revue des Maladies Respiratoires 40, no 2 (février 2023) : 129. http://dx.doi.org/10.1016/j.rmr.2022.11.041.
Texte intégralHinton, M., L. Mellow, A. J. Halayko, A. Gutsol et S. Dakshinamurti. « Hypoxia induces hypersensitivity and hyperreactivity to thromboxane receptor agonist in neonatal pulmonary arterial myocytes ». American Journal of Physiology-Lung Cellular and Molecular Physiology 290, no 2 (février 2006) : L375—L384. http://dx.doi.org/10.1152/ajplung.00307.2005.
Texte intégralPeták, Ferenc, Tibor Z. Janosi, Carole Myers, Fabienne Fontao et Walid Habre. « Impact of elevated pulmonary blood flow and capillary pressure on lung responsiveness ». Journal of Applied Physiology 107, no 3 (septembre 2009) : 780–86. http://dx.doi.org/10.1152/japplphysiol.00157.2009.
Texte intégralSmith, Kelly J., George B. Mallory, Steven H. Abman et Dunbar D. Ivy. « AIRWAY HYPERREACTIVITY AFTER ILOPROST INHALATION IN PEDIATRIC PATIENTS WITH PULMONARY ARTERIAL HYPERTENSION ». Chest 130, no 4 (octobre 2006) : 156S. http://dx.doi.org/10.1378/chest.130.4_meetingabstracts.156s-b.
Texte intégralMichel, R. P., et T. S. Hakim. « Increased resistance in postobstructive pulmonary vasculopathy : structure-function relationships ». Journal of Applied Physiology 71, no 2 (1 août 1991) : 601–10. http://dx.doi.org/10.1152/jappl.1991.71.2.601.
Texte intégralAzamar-Solis, Brizeida, Yahveth Cantero-Fortiz, Juan Carlos Olivares-Gazca, Jesús Mauricio Olivares-Gazca, Gisela Berenice Gómez-Cruz, Iván Murrieta-Álvarez, Guillermo J. Ruiz-Delgado et Guillermo J. Ruiz-Argüelles. « Primary Thrombophilia in Mexico XIII : Localization of the Thrombotic Events in Mexican Mestizos With the Sticky Platelet Syndrome ». Clinical and Applied Thrombosis/Hemostasis 25 (1 janvier 2019) : 107602961984170. http://dx.doi.org/10.1177/1076029619841700.
Texte intégralOhar, Jill A., Kemberly S. Waller et Thomas E. Dahms. « Platelet-activating Factor Induces Selective Pulmonary Arterial Hyperreactivity in Isolated Perfused Rabbit Lungs ». American Review of Respiratory Disease 148, no 1 (juillet 1993) : 158–63. http://dx.doi.org/10.1164/ajrccm/148.1.158.
Texte intégralThèses sur le sujet "Pulmonary arterial hyperreactivity"
Bouchet, Clement. « Expression et rôle du facteur de croissance des nerfs NGF dans l’hypertension pulmonaire ». Electronic Thesis or Diss., Bordeaux, 2024. http://www.theses.fr/2024BORD0092.
Texte intégralPulmonary hypertension (PH) is a severe disease characterized by an increase in mean pulmonary arterial pressure over 20mmHg at rest, compared with 14mmHg in healthy individuals. Ultimately, PH leads to right heart failure and death. In the absence of any curative treatment, identification of new therapeutic targets is a major challenge. In this context, a better understanding of PH development and progression is crucial. In our laboratory, one target of interest is the nerve growth factor or NGF, overexpressed in pulmonary arteries both in experimental and human PH, and playing a significant role in three major PH features: pulmonary arterial inflammation, reactivity and remodeling.First, we focused on possible mechanisms activated in PH that may contribute to NGF pulmonary arterial increased expression. We showed that pro-inflammatory cytokines whose expression are increased in PH, such as interleukin-1β (IL-1β) or Tumor necrosis factor-α, as well as oxidative stress can increase NGF expression and secretion by pulmonary arterial endothelial cells - EC- or smooth muscle cells -CML-. We showed that this secretion involved Transforming growth factor β1, which is secreted by both ECs and SMCs, and then acts in an autocrine manner on these cells to stimulate NGF secretion.We then studied the role of NGF in two major PH features: pulmonary arterial hyperreactivity and inflammation.Regarding hyperreactivity first, we showed that a chronic treatment with NGF (24h) led to pulmonary arterial hyperreactivity, by enhancing activity of connexin-43-dependent gap junctions in SMCs. In parallel, we showed that a shorter treatment with NGF (1h) also led to led to pulmonary arterial hyperreactivity, but in a connexin-43-independent manner, with deregulation of intracellular calcium signaling and increased sensitization of the contractile apparatus in SMCs.Regarding the link between NGF and inflammation, our results showed that in vivo, in a rat model of PH, a preventive treatment with anti-NGF blocking antibodies reduced pulmonary arterial monocyte/macrophage infiltration and inflammasome protein expression as well as both circulating and pulmonary levels of IL-1β. In vitro, we showed that NGF is able to attract monocytes, and that IL-1β plays an important role in pulmonary arterial remodeling, inflammation and endothelial dysfunction.In conclusion, this work has highlighted mechanisms contributing to NGF increased expression in PH and confirmed its involvement in the progression of the disease. The overall results obtained further support NGF as a therapeutic target of interest in this disease
Actes de conférences sur le sujet "Pulmonary arterial hyperreactivity"
Cardouat, guillaume, Matthieu Douard, Lukas Roubenne, Zuzana Kmecová, Paul Robillard, Christophe Guignabert, Ly Tu et al. « NGF induces pulmonary arterial hyperreactivity through connexin 43 increased expression ». Dans ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3565.
Texte intégralBOUCHET, Clement, Guillaume Cardouat, Matthieu Douart, Paul Robillard, Roger Marthan, Bernard Muller, Christelle Guibert et Veronique Freund-Michel. « Short-term mechanisms activated by NGF to induce pulmonary arterial hyperreactivity ». Dans ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa585.
Texte intégralSikarwar, Anurag S., K. T. Santhosh et Shyamala Dakshinamurti. « Inhibition Of Palmitoylation Normalizes Hyperreactivity Of The Pulmonary Arterial Thromboxane Receptor In Hypoxic Pulmonary Hypertension ». Dans American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5034.
Texte intégralDouard, Matthieu, Paul Robillard, Juliette Deweirdt, Isabelle Baudrimont, Mathilde Dubois, Roger Marthan, Jean-Pierre Savineau, Bernard Muller, Christelle Guibert et Veronique Freund-Michel. « Late Breaking Abstract - NGF induces pulmonary arterial hyperreactivity through increased connexin-43 expression : potential role in pulmonary hypertension ». Dans ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.oa3274.
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