Bibliographies thématiques / Post-Cancer

Littérature scientifique sur le sujet « Post-Cancer »

Auteur : Grafiati
Publié le 22 juin 2024

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Articles de revues sur le sujet "Post-Cancer"

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Mohamed Traore, Wend-Yam, Behyamet Onka, Ibrahima Dokal Diallo, Rachida Latib et Youssef Omor. « Post-radiotherapy recto-vaginal fistula in cervical cancer ». International Journal of Case Reports and Images 13, no 2 (3 octobre 2022) : 153–55. http://dx.doi.org/10.5348/101349z01wt2022ci.

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Belyea, James, Robert Hart, Jonathan Trites et Mark Taylor. « Post Cancer Treatment ». Otolaryngology–Head and Neck Surgery 143, no 2_suppl (août 2010) : P152. http://dx.doi.org/10.1016/j.otohns.2010.06.246.

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Madhu, Deepak, Syed Anjum Gardezi et Nadeem Tehami. « Is post-endoscopic ultrasound pancreatic cancer analogous to post-colonoscopy colorectal cancer ? » Endoscopy 55, no 01 (20 décembre 2022) : 102. http://dx.doi.org/10.1055/a-1881-4533.

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Sharma, Abha. « Predictors of Post traumatic growth among breast cancer patients in Nepal ». Asian Pacific Journal of Health Sciences 4, no 2 (30 juin 2017) : 9–17. http://dx.doi.org/10.21276/apjhs.2017.4.2.3.

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Green, Nathan, Dale Treible et Harvey Wallace. « Prostate Cancer : Post-Irradiation Incontinence ». Journal of Urology 144, no 2 Part 1 (août 1990) : 307–9. http://dx.doi.org/10.1016/s0022-5347(17)39438-7.

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Huang, Andy, George Tsavellas et Kirsten S. Hindle. « Colorectal cancer surveillance post-surgery ». Hospital Medicine 62, no 8 (août 2001) : 490–91. http://dx.doi.org/10.12968/hosp.2001.62.8.1626.

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Maher, Ian, et Jeremy Bordeaux. « Post-Skin Cancer Alar Reconstruction ». Facial Plastic Surgery 29, no 05 (13 septembre 2013) : 351–64. http://dx.doi.org/10.1055/s-0033-1353375.

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Audic, Yann, et Rebecca S. Hartley. « Post-transcriptional regulation in cancer ». Biology of the Cell 96, no 7 (septembre 2004) : 479–98. http://dx.doi.org/10.1016/j.biolcel.2004.05.002.

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Pradhan, S. A. « Post-cricoid cancer : An overview ». Seminars in Surgical Oncology 5, no 5 (1989) : 331–36. http://dx.doi.org/10.1002/ssu.2980050508.

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Elshagie Habib Abdelmalak, E. « Prostate cancer surgery post infection ». European Urology Open Science 59 (janvier 2024) : S136. http://dx.doi.org/10.1016/s2666-1683(24)00123-x.

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Thèses sur le sujet "Post-Cancer"

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Moore, Julie. « Cancer and post-traumatic growth ». Thesis, University of Southampton, 2010. https://eprints.soton.ac.uk/171959/.

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Cancer is a major medical problem and a leading cause of mortality in the UK. The experience of diagnosis and treatment can be a traumatic one for many people, with symptoms of anxiety, depression, and posttraumatic stress disorder (PTSD) common for many patients. Despite this, many survivors also report benefits and a sense of personal growth from their experience. Understanding this process and the influence of posttraumatic growth (PTG) on mental health outcomes for cancer patients may have far reaching implications for the promotion of psychological adjustment to this chronic illness. The literature review in this paper explores the predominant theories of PTG and the research on cancer-related PTG. The literature review explores links between the predictions of these general theories and research findings for cancer patients specifically. Establishing factors that predict PTG and its relationship with a range of mental health outcomes would help to build our understanding of emotional adjustment to chronic illness and inform the development of psychological interventions. The empirical paper investigates the role of trauma-related cognitive appraisals in the perception of PTG for breast cancer patients. More negative appraisals in relation to the event were associated with benefit finding.
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Holland, C. M. « Genomic and post-genomic studies in endometrial cancer ». Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604158.

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Genomic abnormalities lead to aberrant cell growth and signalling, contributing to the development of malignancy. Growth and metastasis also require the development of tumour vasculature. The VEGFs and angiopoietins are growth factor families central to this process. This thesis examines their expression in endometrial cancer. Genomic and post-genomic studies are used to broaden the investigation of angiogenesis and include related pathways. Using in situ hybridisation VEGF-A mRNA was detected in epithelial cancer cells but not pre-malignant and benign endometrium while VEGF-B was localised to both epithelial and stromal cells and was most abundant in benign endometrium. These findings suggest that VEGF-B may modulate the actions of VEGF-A by occupying receptors. Larger, gene microarray studies confirmed the differential expression of angiogenic factors and also identified a distinct set of coherently regulated genes in endometrial cancers. Within this gene set, PPARα, a transcription factor regulating fatty acid metabolism, was over-expressed in cancers unlike RXRβ, a heterodimerisation partner, which was down-regulated. A PPARα ligand altered proliferation and apoptosis of endometrial cancer cells suggesting that a normal PPARα/RXRβ ratio helps maintain cellular growth control. In conclusion, this work has defined the pattern of expression of the VEGF and angiopoieten families in endometrial cancer and identified VEGF-B as a possible modulator of VEGF-A action. Abnormalities of the fatty acid metabolic pathway have also been identified as a feature of endometrial cancer. Furthermore, an activating ligand for PPARα, a key factor in this pathway, exerted beneficial effects on the growth of endometrial cancer cells. PPARα ligands may therefore have therapeutic potential in endometrial cancer.
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Simmons, Kingsley Lorraine. « The uptake of post-surgical treatment in cancer patients ». Thesis, King's College London (University of London), 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416863.

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Ooi, Li Yin. « Post-GWAS functional characterisation of colorectal cancer risk loci ». Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/23514.

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Large bowel cancer, or colorectal cancer (CRC) is the third most common cause of cancer worldwide and the fourth biggest cause of cancer mortality. Twin studies have shown that the heritable contribution is ~35%, with ~5% of cases due to rare, high-penetrance mutations. In the last decade, the use of genome-wide association studies on large, well-characterised case-control cohorts of CRC has facilitated the identification of over 25 common genetic variants that carry with them an increased predisposition to colorectal cancer, invoking the common-disease common variant paradigm. As almost all of these variants lie within non-coding regions, the underlying causal mechanism is to-date poorly understood for the majority of these loci, and it is thought that they mediate risk by influencing gene expression levels. To test this hypothesis, an agnostic approach that utilises expression quantitative trait loci (eQTL) analysis was first carried on 115 normal colorectal mucosa samples and 59 peripheral blood mononuclear cells (PBMC). As these heritable variation on gene expression are likely to be subtle, there is a strong emphasis on the technical methodology to minimise experimentally-induced non-biological variations, including the extraction of high-quality RNA from primary tissue, the selection and validation of reference genes for normalisation of gene expression quantification, as well as internal validation of the samples and data processing. Thereafter, the association between the 25 CRC risk variants and the expression of their cis-genes were examined systematically, demonstrating that ten of these variants are also tissue-specific eQTLs. This intermediate phenotype strongly suggests that they confer risk, at least in part, by modifying regulatory mechanisms. One of the best eQTL associations (Xp22.2) is investigated in further detail to reveal a novel indel polymorphism (Indel24) at the distal promoter region of target gene SHROOM2 that influenced both transcript abundance and CRC risk more than the original tagging SNP. Functional verification with gene reporter assays indicated that Indel24 displays differential allelic control over transcriptional activity. Further in silico analysis and mutations to the reporter gene constructs provided evidence that Indel24 modulates transcription by modifying the spacing between CCAAT motifs and the consequent binding affinity of NF-Y transcription factor. siRNA depletion of NF-Y was associated with a reduction in transcriptional activity of the Indel24 gene construct as well as endogenous SHROOM2, which is strongly supportive of the interaction between Indel24 and NF-Y in the transcriptional activation of SHROOM2. Preliminary evidence is suggestive of SHROOM2 being expressed at the top of the intestinal epithelial crypt and playing a role in cell cycle regulation. Hypothesis-driven approaches can also be of utility in demonstrating functionality of CRC risk variants, complementing the hypothesis-free approach of eQTL analysis. Guided by a recently discovered gene-environment interaction between the 16q22.1 risk variant and circulating vitamin D levels, the influence of the rs9929218 SNP on CDH1 gene expression was examined, in relation to the expression of putative regulatory genes derived from in silico analysis and studies of other target genes. Although there was no direct association between rs9929218 and CDH1 expression, there were multiple two-way interactions that were together suggestive of rs9929218 influencing the VDR/FOXO4 regulation of CDH1. This provides functional support for the mechanism underlying the epidemiological observation of the gene-environment interaction between 16q22.1 and vitamin D, and demonstrates a candidate-based approach in deciphering the link between genetic locus and CRC susceptibility. In summary, the research presented in this thesis has validated the experimental rationale of utilising expression studies of normal colorectal mucosa to hone in on the molecular mechanisms and susceptibility genes underlying the association between common genetic variation and CRC risk.
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Swartzman, Samantha. « Psychosocial determinants of post-traumatic stress among cancer survivors ». Thesis, University of Dundee, 2017. https://discovery.dundee.ac.uk/en/studentTheses/10d5554d-8987-436b-9558-4e6be5f844f2.

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Cancer survivors can experience a number of consequences of their disease, both physical and psychological. In this thesis, I aimed to clarify the psychosocial processes that reduce or increase post-traumatic stress after cancer. “Social support,” for instance, has often been linked to post-traumatic stress, but I describe several problems with this concept. Alternatively, inspired by the Social Identity Approach (SIA) to health, I proposed that group identification, or a sense of belonging and commonality with social groups, might predict levels of post-traumatic stress after cancer. In line with Social Cognitive Processing Theory (SCPT), I also proposed that cancer survivors’ perceptions that members of their social groups are closed, judgmental, and unreceptive in conversations about cancer (“group constraints”) might predict post-traumatic stress. I proposed that these constructs might act in tandem to predict levels of post-traumatic stress. To begin my programme of research, I conducted a meta-analysis that established factors contributing to post-traumatic stress disorder (PTSD). I also found an increased prevalence of PTSD among cancer survivors compared to controls without cancer. This meta-analysis demonstrated that cancer survivors can experience PTSD related to their illness. Subsequently, I undertook a series of quantitative questionnaire studies to establish psychosocial determinants of post-traumatic stress. My questionnaire included scales measuring post-traumatic stress, social support, group identification, and group constraints. In a pilot study, I used convenience sampling to test out the acceptability of the questionnaire and to provide a foundation for further hypothesising. Then, I distributed this piloted questionnaire to colorectal cancer survivors in Tayside. In initial “wave 1” cross-sectional analyses of this data, I found that social support was not an independent predictor of post-traumatic stress in multivariate analyses. However, group constraints consistently and independently predicted post-traumatic stress. Group identification exerted an effect on post-traumatic stress by reducing group constraints. I then distributed the same questionnaire a second time to the same respondents from wave 1 in order to collect longitudinal data. The results of the data from respondents at both waves 1 and 2 revealed that social support was, in fact, a likely causal predictor of post-traumatic stress. Constraints within the family also causally reduced post-traumatic stress. Family identification was a weak causal factor in reducing post-traumatic stress, but reciprocally, post-traumatic stress reduced group identifications. The longitudinal data also provided further, albeit not strong, support for the hypothesis that family identification reduces post-traumatic stress by reducing constraints on conversations within the family. Finally, in light of the consistent evidence from the quantitative studies above that conversational constraints are associated with increased post-traumatic stress, I conducted a qualitative interview study with a small number of cancer survivors aiming to characterise their experiences of conversations about cancer. The participants reported a broad range of experiences, beyond just constraints versus openness. These findings have broad implications for both theory and practice. For instance, in the final chapter, I discuss ways in which these results motivate further research on intersections between the Social Identity Approach and cancer-related post-traumatic stress. I also discuss implications for interventions going beyond traditional trauma exposure techniques.
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Patel, Uday. « Post chemoradiation re-staging of rectal cancer using MRI ». Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/23905.

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The role of MRI after preoperative treatment of rectal cancer is unclear. This thesis investigates its diagnostic relevance in patient management. Matching pathology with MR images aimed to characterise post treatment rectal cancer appearances on T2 weighted MRI and verify the proposed MRI tumour regression grading system(mrTRG). Correlation of post-chemoradiation mrTRG, T stage(ymrT), N stage(ymrN) and Circumferential Resection Margin(ymrCRM) with histopathological T stage(ypT), N stage (ypN), and pathological CRM(pCRM) was investigated. These parameters were also compared against survival outcomes. The alternative response assessment methods of tumour volume and length change/RECIST as well as mrTRG and ymrT were evaluated against pathology. The accuracy and reproducibility of MRI parameters in assessing rectal cancer response to neoadjuvant chemotherapy was also investigated. Patients with good response to chemoradiation(assessed by mrTRG) were enrolled into a deferral of surgery trial. Serial MRI-based monitoring enabled evaluation of quantitative imaging methods such as Apparent Diffusion Co-efficient (ADC) measurements in distinguishing tumour vs. complete response. Pathological fibrosis correlated with low signal on MRI, tumour was intermediate signal. mrTRG showed good diagnostic accuracy against ypT&pTRG, ymrT's diagnostic accuracy was fair but improved when grouped into favourable and unfavourable categories. MRI Length&volume assessment were less consistently related. Negative ymrN and potentially clear ymrCRM correlated well with their respective pathological endpoints. Importantly, mrTRG and ymrCRM involvement predicted survival outcomes. mrTRG showed good diagnostic accuracy against pTRG when assessing neoadjuvant chemotherapy response, ymrT correlated less well. Overall mrTRG and ymrT were the most reproducible parameters. ADC values were significantly lower in patients with tumour vs. complete response with ADC measurement of <1.3X10-3/mm2/sec associated with tumour regrowth in 86% of cases. mrTRG and MRI CRM assessment appear the most important post treatment imaging parameters. Grouped ymrT may also be useful. These parameters could be used by the multidisciplinary team to tailor treatment pre-operatively.
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Wagner, Heidi. « Investigating post-translational modifications of c-Met in cancer ». Thesis, Queensland University of Technology, 2020. https://eprints.qut.edu.au/203995/1/Heidi_Wagner_Thesis.pdf.

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Longman, Michael Roy. « Combination of post-transcriptional and post-translational down-regulation of the oestrogen receptor in breast cancer ». Thesis, Cardiff University, 2011. http://orca.cf.ac.uk/54416/.

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Greater concentrations of fulvestrant are being employed within the clinic due to increased oestrogen receptor (ER) down-regulation and greater clinical benefit in ER+ breast cancer. However, complete ER down-regulation has not been achieved. The importance of residual ER is unknown and could allow cells to survive initial anti-hormone impact and progression to hormone insensitivity. This project aims to go further than current clinical therapy, using the MCF-7 cell model to target and assess the importance of residual ER. Cells were treated with fulvestrant aiming to achieve maximal ER down-regulation. The effect of any residual ER on signalling and growth was subsequently assessed. An alternative model for ER loss, ER siRNA, was employed to see whether this had a greater anti-ER effect. Finally, fulvestrant and ER siRNA were employed in combination to assess whether these agents work synergistically to give greater ER down-regulation and increased anti- tumour effect. With fulvestrant at 10"7M, ER levels were markedly reduced, although residual ER was observed that remained with increasing drug concentrations. There was significant reduction of ER signalling, proliferation and growth, but the inhibition was incomplete. When ER siRNA was assessed, similar results were obtained, with comparable and incomplete ER loss, residual signalling and growth. Following combination treatment of fulvestrant and ER siRNA, residual ER was almost undetectable, though this did not correspond to greater loss of ER signalling or growth when compared to either agent alone. While this work showed greater ER loss than previously recorded by targeting both protein and mRNA together, no greater anti-tumour activity was observed. Thus, while the mechanism underlying residual growth warrants future investigation (along with longer exposure), targeting ER alone, no matter how successfully, may not be the best treatment regimen and a combination of targets may be required as the optimum strategy to treat ER+ breast cancer.
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Djordjevic, Darja. « The Cancer War(d) : Onco-Nationhood in Post-Traumatic Rwanda ». Thesis, Harvard University, 2016. http://nrs.harvard.edu/urn-3:HUL.InstRepos:33493385.

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In Africa, the effects of the HIV/AIDS pandemic, rapidly expanding industrial and extractive economies, uncontrolled economic growth, environmental and lifestyle changes, and the rising age of populations with better access to medicine have occasioned rising rates of cancer. Rwanda’s national cancer program has been hailed as a unique example of how to build clinical oncology into a public healthcare infrastructure. Using ethnographic data, interviews, and historical archives, I address three sets of questions: 1. What historical, economic, social, and political factors have shaped the development of the country’s cancer program? 2. How do local clinicians and patients experience cancer as a treatable chronic disease? And how is that experience affected by the development of a national oncology infrastructure and new biomedical technologies? 3. As an instance of the transnational private-public partnerships characteristic of global health interventions in postcolonial Africa, what successes, limitations, and challenges does this cancer program present for envisioning oncology programs elsewhere in the global south? What are the ethical, political, and epistemological stakes involved in different models of cancer care? This project contributes to a new chapter in medical anthropology, one focused on rising rates of cancer in contemporary Africa. I argue that Rwanda’s cancer project is an exercise in the construction of a new sense of sovereignty, rendered through the politics of life as onco-nationhood; that it is an effort to create a postcolonial polity whose citizen body is gifted care of a international caliber provided by a paternal state. In a critical moment of post-traumatic social reconstruction, national biomedicine is becoming the entity through which government seeks to fuse sovereign statehood and nationhood in the cause of a healthy Rwandan future. Theorizing this relationship holds at least one key to developing an anthropology of cancer in contemporary Africa.
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Costanzo, Erin Susan. « Post-treatment adjustment and behavior change among women with breast cancer ». Diss., University of Iowa, 2006. http://ir.uiowa.edu/etd/56.

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Livres sur le sujet "Post-Cancer"

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Jurisica, Igor, Dennis A. Wigle et Bill Wong, dir. Cancer Informatics in the Post Genomic Era. Boston, MA : Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-69321-7.

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Crompvoets, Samantha. Breast Cancer and the Post-Surgical Body. London : Palgrave Macmillan UK, 2006. http://dx.doi.org/10.1057/9780230599789.

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Giordano, Antonio, et Nicola Normanno, dir. Breast Cancer in the Post-Genomic Era. Totowa, NJ : Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-945-1.

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Dennis, Wigle, Jurisica Igor et Wong Bill, dir. Cancer informatics in the post genomic era. New York : Springer, 2007.

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Sakharkar, Kishore R., Ramesh Chandra et Meena K. Sakharkar. Post-genomic Approaches in Cancer and Nano Medicine. New York : River Publishers, 2022. http://dx.doi.org/10.1201/9781003339083.

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O, Tollefsbol Trygve, dir. Cancer epigenetics. Boca Raton : CRC Press/Taylor & Francis Group, 2009.

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Limoges, Jacqueline Marie. Informational needs of women with breast cancer receiving chemotherapy post lumpectomy and nodal dissection. Ottawa : National Library of Canada = Bibliothèque nationale du Canada, 1992.

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Corporation, Market Intelligence Research, et Frost & Sullivan., dir. Post-therapeutic cancer support systems market : Diagnostic, regulatory, and demographic changes create new opportunities. Mountain View, CA : Market Intelligence Research Corp., 1991.

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Hubner, Donald M. Comparative effects of early versus late starvation on the development of the post-larval, megalopopa, stage of the crabs Hemigrapsus nudus, Cancer magister, and Cancer oregonensis. Bellingham, WA : Huxley College of Environmental Studies, Western Washington University, 2002.

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Steingraber, Sandra. Post-Diagnosis. Ithaca, USA : Firebrand Books, 1995.

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Chapitres de livres sur le sujet "Post-Cancer"

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Streicher, Lauren, et James A. Simon. « Sexual Function Post-Breast Cancer ». Dans Optimizing Breast Cancer Management, 167–89. Cham : Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-70197-4_11.

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Chahbazian, Chahin M. « Bone Metastases Post Breast Cancer ». Dans Practical Approaches to Cancer Invasion and Metastases, 98. Berlin, Heidelberg : Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-84885-8_29.

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Ward, Elizabeth. « Cancer Risk Post 9/11 ». Dans World Trade Center Pulmonary Diseases and Multi-Organ System Manifestations, 81–93. Cham : Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59372-2_6.

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Maclean, Julia. « Assessment of Function Post-cancer ». Dans Atlas of Extreme Facial Cancer, 447–54. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-88334-8_19.

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Rambaldi, Pier Francesco. « Breast Cancer : Post-Surgical Brachial Plexopathy ». Dans Whole-Body FDG PET Imaging in Oncology, 213–15. Milano : Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-5295-6_49.

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Sultana, Asma, Robert Padbury et John Chen. « Incidental Gallbladder Cancer Post Laparoscopic Cholecystectomy ». Dans The Management of Gallstone Disease, 199–203. Cham : Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-63884-3_14.

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Bloom, Elizabeth S., Philip Poortmans, Marianne Aznar, Thomas A. Buchholz et Eric A. Strom. « Breast Cancer : Intact and Post Mastectomy ». Dans Medical Radiology, 641–84. Berlin, Heidelberg : Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/174_2011_163.

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Iavazzo, Christos, et Ioannis D. Gkegkes. « Vulvar cancer and post-vulvectomy complications ». Dans The Vulva, 106–17. Second edition. | Boca Raton, FL : CRC Press, [2018] : CRC Press, 2017. http://dx.doi.org/10.1201/9781315113739-13.

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Vaidya, Abhishek. « Post-Treatment Surveillance of Thyroid Cancer ». Dans Thyroid Surgery, 151–60. First edition. | Boca Raton : CRC Press, 2020. : CRC Press, 2020. http://dx.doi.org/10.1201/9780429086076-20.

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Goonewardene, Sanchia S., Karen Ventii, Amit Bahl, Raj Persad, Hanif Motiwala et David Albala. « Bladder Cancer Risk Post Pelvic Irradiation ». Dans Management of Urology, 165–67. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57915-9_22.

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Actes de conférences sur le sujet "Post-Cancer"

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Shatnawi, Sara, Esra'a Odat, Maram Thiabat et Aya A. Al-Badarneh. « Cancer in Jordan : Post-Hoc Comparative Analysis ». Dans 2022 13th International Conference on Information and Communication Systems (ICICS). IEEE, 2022. http://dx.doi.org/10.1109/icics55353.2022.9811179.

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Abdulqawi, Rayid, Matina Verykiou, Navjot Kaur et Elin Roddy. « Recurrence Post Surgical Resection Of Lung Cancer ». Dans American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4391.

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Attia, Monia, Henda Neji, Mariem Affes, Soumaya Ben Saad, Houda Gharsalli, Ines Baccouche, Saoussen Hantous et Khaoula Ben Miled. « Imaging features in post-operative lung cancer recurrence ». Dans ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa848.

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Al-Remawi, Mayyas, et Faisal Aburub. « Clinical Applications of AI in Post-Cancer Rehabilitation ». Dans 2024 2nd International Conference on Cyber Resilience (ICCR). IEEE, 2024. http://dx.doi.org/10.1109/iccr61006.2024.10533091.

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Sukik, Layan, Bushra Hoque, Linda Boutefnouchet et Vijay Ganji. « Serum vitamin D concentrations are non-linearly related to breast cancer risk in postmenopausal women ». Dans Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0138.

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Background: Post-menopausal women are at high risk for breast cancer. The association between serum 25-hydroxyvitamin D [25(OH)D] concentration and breast cancer in post-menopausal women is not well understood. Objectives: The aim of this study was to investigate the association between serum 25(OH)D and breast cancer using nationally representative sample surveys. Methodology: In this cross-sectional study, we used seven cycles of National Health and Nutrition Examination Surveys from 2001 through 2014. Participants were non-institutionalized post-menopausal women (n=8100). Logistic regression was performed to determine the association between serum 25(OH)D concentrations and breast cancer prevalence. A restricted cubic spline method was used to assess the non-linear association. Results: The prevalence of breast cancer was 3.3%, 4.0%, 4.6%, 6.4%, and 6.9% in the groups with serum 25(OH)D levels of <30, 30-<50, 50-<75, 75-<100, and ≥100 nmol/L, respectively. The risk of having breast cancer was significantly higher in the serum 25(OH)D 75-<100 nmol/L category compared to the 25(OH)D <30 nmol/L concentration [OR and 95% CI; 2.21 (1.23-3.98)]. Furthermore, a significant non-linear relationship between serum 25(OH)D concentrations (when used as a continuous variable) and breast cancer in all post-menopausal women (p for non-linear trend 0.032) was observed. Overall, the risk of breast cancer was highest (OR=1.5) between 70 nmol/L and 80 nmol/L of serum 25(OH)D concentration in all post-menopausal women. Conclusion: An adverse association was observed between serum 25(OH)D concentration and breast cancer in post-menopausal women. Further research is needed to elucidate the mechanism of vitamin D in cancer pathogenesis.
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Beaton, David, Matthew Rutter et Iosif Beintaris. « PTH-005 Reliability of post-colonoscopy colorectal cancer algorithms ». Dans British Society of Gastroenterology Annual Meeting, 17–20 June 2019, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2019. http://dx.doi.org/10.1136/gutjnl-2019-bsgabstracts.30.

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Lange, Marta, Szabolcs Bozsányi, Emilija Vija Plorina, Norbert Kiss, Nora Noemi Varga, Ilze Lihacova et Alexey Lihachev. « Skin cancer post-operative scar evaluation using autofluorescence features ». Dans Applications of Digital Image Processing XLV, sous la direction de Andrew G. Tescher et Touradj Ebrahimi. SPIE, 2022. http://dx.doi.org/10.1117/12.2633932.

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Sivaranjini, S., et K. Nirmala. « Breast cancer response post neoadjuvant chemotherapy using MRI measurements ». Dans 2017 Fourth International Conference on Signal Processing,Communication and Networking (ICSCN). IEEE, 2017. http://dx.doi.org/10.1109/icscn.2017.8085419.

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Dawson, B., et T. Mason. « 261. Dupont Bladder Cancer Screening for Post-Exposure Cohorts ». Dans AIHce 2005. AIHA, 2005. http://dx.doi.org/10.3320/1.2758723.

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Baile-Maxía, S., C. Mangas-Sanjuan, M. Alustiza, L. Medina-Prado, O. Murcia, N. Traversi, P. Zapater et al. « POST-COLONOSCOPY COLORECTAL CANCER : INCIDENCE, CHARACTERISTICS AND PREDICTIVE FACTORS ». Dans ESGE Days 2022. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1744934.

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Rapports d'organisations sur le sujet "Post-Cancer"

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Edwards, Dean P. Ph.D. Post-Doctoral Training Program in Breast Cancer Research. Fort Belvoir, VA : Defense Technical Information Center, juillet 2001. http://dx.doi.org/10.21236/ada396800.

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Edwards, Dean P. Ph.D. Post-Doctoral Training Program in Breast Cancer Research. Fort Belvoir, VA : Defense Technical Information Center, juillet 2002. http://dx.doi.org/10.21236/ada408784.

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Edwards, Dean P. Ph.D. Post-Doctoral Training Program in Breast Cancer Research. Fort Belvoir, VA : Defense Technical Information Center, juillet 2004. http://dx.doi.org/10.21236/ada428190.

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Workman, Sarah, et Maddy Thompson. Breaking down barriers : Empowering Black women in breast cancer care. Royal Geographical Society (with IBG), mars 2022. http://dx.doi.org/10.55203/vvdj9112.

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Bovbjerg, Dana. Post-Doctoral Training Program in Bio-Behavioral Breast Cancer Research. Fort Belvoir, VA : Defense Technical Information Center, mai 2005. http://dx.doi.org/10.21236/ada442252.

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Bovbjerg, Dana. Post-Doctoral Training Program in Bio-Behavioral Breast Cancer Research. Fort Belvoir, VA : Defense Technical Information Center, juin 2002. http://dx.doi.org/10.21236/ada407804.

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Bovbjerg, Dana H. Post-Doctoral Training Program in Bio-Behavioral Breast Cancer Research. Fort Belvoir, VA : Defense Technical Information Center, juin 2003. http://dx.doi.org/10.21236/ada418207.

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Jin, Hongjun. Early Detection Of Breast Cancer using Post-Translationally Modified Biomarkers. Fort Belvoir, VA : Defense Technical Information Center, mars 2012. http://dx.doi.org/10.21236/ada559414.

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Vijayakumar, Srinivasan. Prevention of Post-Radiotherapy Failure in Prostate Cancer by Vitamin D. Fort Belvoir, VA : Defense Technical Information Center, mars 2006. http://dx.doi.org/10.21236/ada455585.

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Vijayakumar, Srinivasan. Prevention of Post-Radiotherapy Failure in Prostate Cancer by Vitamin D. Fort Belvoir, VA : Defense Technical Information Center, mars 2005. http://dx.doi.org/10.21236/ada433996.

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