Littérature scientifique sur le sujet « Permeabilità intestinale »
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Articles de revues sur le sujet "Permeabilità intestinale"
Bottaccioli, Anna Giulia. « La malattia infiammatoria cronica intestinale : cure integrate in ottica Pnei ». PNEI REVIEW, no 2 (novembre 2020) : 30–40. http://dx.doi.org/10.3280/pnei2020-002004.
Texte intégralBianchi, Alfio Ernesto, Antonio Maggi et Riccardo Raddino. « Il microbiota intestinale, tra salute e malattia : un vero attore a due facce ». CARDIOLOGIA AMBULATORIALE 30, no 2 (14 octobre 2021) : 85–91. http://dx.doi.org/10.17473/1971-6818-2021-2-1.
Texte intégralBertossi, Francesca. « Il ruolo del microbiota nell'aumento ponderale associato alla terapia antipsicotica ». PNEI REVIEW, no 2 (novembre 2022) : 108–22. http://dx.doi.org/10.3280/pnei2022-002010.
Texte intégralPabla, D., F. Akhlaghi, H. Zia, Marco Centanni et Nunzia Brusca. « Incremento della permeabilità intestinale alla levotiroxina sodica mediante acidi grassi a catena lineare studiata in linee di cellule epiteliali MDCK ». L'Endocrinologo 12, no 5 (octobre 2011) : 259–60. http://dx.doi.org/10.1007/bf03344845.
Texte intégralCavin, Jean-Baptiste, Hailey Cuddihey, Wallace K. MacNaughton et Keith A. Sharkey. « Acute regulation of intestinal ion transport and permeability in response to luminal nutrients : the role of the enteric nervous system ». American Journal of Physiology-Gastrointestinal and Liver Physiology 318, no 2 (1 février 2020) : G254—G264. http://dx.doi.org/10.1152/ajpgi.00186.2019.
Texte intégralDahlgren, David, Maria-Jose Cano-Cebrián, Tobias Olander, Mikael Hedeland, Markus Sjöblom et Hans Lennernäs. « Regional Intestinal Drug Permeability and Effects of Permeation Enhancers in Rat ». Pharmaceutics 12, no 3 (8 mars 2020) : 242. http://dx.doi.org/10.3390/pharmaceutics12030242.
Texte intégralPijls, Kirsten E., Ger H. Koek, Elhaseen E. Elamin, Hanne de Vries, Ad A. M. Masclee et Daisy M. A. E. Jonkers. « Large intestine permeability is increased in patients with compensated liver cirrhosis ». American Journal of Physiology-Gastrointestinal and Liver Physiology 306, no 2 (15 janvier 2014) : G147—G153. http://dx.doi.org/10.1152/ajpgi.00330.2013.
Texte intégralMarkovic, Milica, Moran Zur, Sapir Garsiani, Daniel Porat, Sandra Cvijić, Gordon L. Amidon et Arik Dahan. « The Role of Paracellular Transport in the Intestinal Absorption and Biopharmaceutical Characterization of Minoxidil ». Pharmaceutics 14, no 7 (27 juin 2022) : 1360. http://dx.doi.org/10.3390/pharmaceutics14071360.
Texte intégralWillems, D., S. Cadranel et W. Jacobs. « Measurement of urinary sugars by HPLC in the estimation of intestinal permeability : evaluation in pediatric clinical practice ». Clinical Chemistry 39, no 5 (1 mai 1993) : 888–90. http://dx.doi.org/10.1093/clinchem/39.5.888.
Texte intégralOscarsson, Elin, Tim Lindberg, Kathrin S. Zeller, Malin Lindstedt, Daniel Agardh, Åsa Håkansson et Karolina Östbring. « Changes in Intestinal Permeability Ex Vivo and Immune Cell Activation by Three Commonly Used Emulsifiers ». Molecules 25, no 24 (15 décembre 2020) : 5943. http://dx.doi.org/10.3390/molecules25245943.
Texte intégralThèses sur le sujet "Permeabilità intestinale"
Ponce, de Leon Rodriguez Maria del Carmen. « Développement d’un modèle in vitro d’inflammation intestinale par l’utilisation de lignées cellulaires humaines en co-culture pour l’étude des interactionsavec les micro-constituants alimentaires ». Thesis, Montpellier, 2019. http://www.theses.fr/2019MONTG009/document.
Texte intégralThe intestinal epithelium, main place of the absorption of (micro)-nutrients is also the first body's defense system. An imbalance in homeostasis can lead to an inflammatory reaction associated with defects in the intestinal barrier and immune function as well as malabsorption of nutrients, as seen in IBD (Inflammatory Bowel Diseases), in micronutrient fortification strategies and noncommunicable diseases (obesity). It is therefore important to find ways of action, for example through diet, to prevent or at least reduce the nutritional and pathological consequences of intestinal inflammation, and to understand the mechanisms involved. Among intestinal models, in vitro cell culture models are increasingly used and allow to evaluate the molecular mechanisms in a simple and reproducible way and to reduce animal experimentation.In this context and in order to study the interaction of dietary bioactive compounds with the intestine in state of inflammation, the first objective of this work was the development of an in vitro model of inflamed intestine combining in co-culture two human intestinal cell lines: Caco-2 TC7 (enterocytes) and HT29-MTX (goblet cells) and an immune cell line of macrophages (THP1). Several inflammation markers were evaluated and we were able to show that the tri-culture model responded to an inflammatory stimulus (LPS / IFNγ), by increasing the production of pro-inflammatory cytokines (TNF-α, IL6 and IL8) and enzymes (INOS and COX2) as well as the expression of their genes. In addition, an increase of epithelial permeability via tight junctions (TJs) alteration has also been demonstrated, as well as overproduction of mucus, which are recognized inflammation characteristics.The second objective was to study the interaction of β-cryptoxanthin (BCX), a lipophilic and antioxidant carotenoid of citrus, with the inflamed model. To solubilize BCX, we used two types of micelles (artificial and physiological) and studied markers of inflammation. Although it appears from the preliminary results that BCX micelles show a tendency to decrease the production of some cytokines (IL6 and IL8), the role of micelle constituents (Tween 40 or bile salts / phospholipids) in the phenomenon observed and in the epithelial permeability remains to be therefore clarified
Weaver, Laurence Trevelyan. « The intestinal permeability of infants ». Thesis, University of Cambridge, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289788.
Texte intégralSmethurst, Paul R. « Small intestinal permeability in man ». Thesis, Open University, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.315531.
Texte intégralLundin, Pål. « Intestinal permeability a parameter of mucosal dysfunction / ». Lund : Dept. of Animal Physiology, Lund University, 1997. http://books.google.com/books?id=wuNqAAAAMAAJ.
Texte intégralLang, Mia E. « Intestinal permeability in the irradiated ferret ». Thesis, University of Ottawa (Canada), 1991. http://hdl.handle.net/10393/7772.
Texte intégralAnderson, Alexander Douglas Gray. « Measurement of intestinal permeability in surgical patients ». Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/24575.
Texte intégralSalameh, Emmeline. « Développement d'un modèle murin de dénutrition avec entéropathie et évaluation de molécules d'intérêt permettant de contribuer au rétablissement de la fonction de barrière intestinale ». Thesis, Normandie, 2019. http://www.theses.fr/2019NORMR064.
Texte intégralBackground : Severe acute malnutrition (SAM) is a global health issue affecting 17 million children under the age of 5. SAM induces rapid weight loss and is often associated with environmental enteric dysfunction (EED). EED is characterized by intestinal hyperpermeability and inflammation, villus blunting and nutrient malabsorption. EED might, therefore, limit stabilization and re-nutrition protocol efficacy. Objectives : This thesis aimed to develop an undernutrition model with enteropathy to evaluate the effect of a therapeutic milk enriched with nutrients on gut barrier function. Results : During preclinical model development, several approaches were tested: calorie restriction, low-protein diet, use of lipopolysaccharides and indomethacin. Only daily indomethacin gavage during one week in protein-energy undernourished mice induced growth faltering associated with enteropathy. After preclinical model validation, we evaluated the effect of therapeutic milk supplemented with glutamine, leucine, gum arabic and/or selenium-enriched yeast on gut barrier function. Glutamine and leucine induce beneficial effects on gut barrier function. In ourexperimental conditions, therapeutic milk enriched with a combination of glutamine and leucine had a limited impact on this parameter. Gum arabic and selenium-enriched yeasts have prebiotic and probiotic properties on gut barrier function. Therapeutic milk supplemented with gum arabic and selenium-enriched yeast inhibited intestinal inflammation and enhanced specific bacteria abundance such as Faecalibacterium prausnitzii.Conclusion : The studies conducted during this thesis permitted to develop a new model of undernutrition with enteropathy. Therapeutic milk enriched with arabic gum and selenium-enriched yeast triggered beneficial effects on gut barrier function in our preclinical model
Yao, Shengtao. « The effect of denervation on intestinal permeability and function following small intestinal transplantation ». Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/mq22692.pdf.
Texte intégralWang, Chuan. « Pathologically and experimentally induced intestinal barrier changes evaluated by permeability measurements ». Lund : Dept. of Animal Physiology, Lund University, 1995. http://books.google.com/books?id=ddlqAAAAMAAJ.
Texte intégralBahlouli, Wafa. « Régulation de la perméabilité intestinale au cours du syndrome de l'intestin irritable : role du système ubiquitine-protéasome et impact de l'obésité ». Thesis, Normandie, 2019. http://www.theses.fr/2019NORMR047.
Texte intégralIrritable bowel syndrome (IBS) is a multifactorial functional disorder, involving environmental factors (stress and diet for instance), gut-brain-axis dysfunction, micro-inflammation, dysbiosis and an alteration of intestinal permeability. The role of the proteasome in the regulation of the intestinal barrier during IBS has been studied. In addition, these intestinal functional disorders have also been described in patients with obesity. Nevertheless, the mechanisms underlying an association of intestinal functional disorders in the obesity context, remain poorly understood and have therefore been investigated in this thesis. In this study, "IBS-like" mouse models such as water avoidance stress (WAS) and the post-inflammatory (post-TNBS) models, were used to study the impact of proteasome inhibition on the regulation of intestinal permeability. We found that the pharmacological inhibition of the proteasome (with PR-957) or the use of knock-out mice for a subunit of the proteasome (β2i -/-) limit intestinal hyperpermeability occured in IBS-Like models. Moreover, we found that oral supplementation with glutamine also reduces intestinal hyperpermeability, wich, thus, can be considered as a putative nutritional treatment for IBS. A colonic proteomic study of WAS mice and a study of colonic ubiquitoma in IBS patients with diarrheal profiles confirmed the involvement of proteasome in the pathophysiology of IBS. Therefore, the link between obesity and IBS was examined by combining models of obesity (ob/ob genetic and high-fat diet [HFD] models) with WAS model. Only HFD mice displayed enhanced intestinal hyperpermeability and higher plasma corticosterone levels in response to WAS. Further studies suggest that these results, themselve, are independent of leptin, glycaemia and gut microbiota. This study paves new ways of treating patients suffering from IBS, by nutritional intervention via glutamine or by using the proteasome as a therapeutic target. We also suggest a role of diet (high fat) in the development of intestinal functional disorders during obesity
Livres sur le sujet "Permeabilità intestinale"
Ford, Jayne. Non-steroidal anti-inflammatory drugs and intestinal permeability. Manchester : Universityof Manchester, 1994.
Trouver le texte intégralSinclair, David Graeme. Changes in pulmonary endothelial and intestinal permeability following cardiopulmonary bypass and in the critically ill. Birmingham : University of Birmingham, 1995.
Trouver le texte intégralIntestinal Permeability [Working Title]. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.82968.
Texte intégralNaturopath, Case Adams. The Science of Leaky Gut Syndrome : Intestinal Permeability and Digestive Health. Logical Books, 2014.
Trouver le texte intégralSingh, Marvin M., et Gerard E. Mullin. Diet, Environmental Chemicals, and the Gut Microbiome. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190490911.003.0006.
Texte intégralBazzan, Anthony J., et Daniel A. Monti. Diet, Gut, and Brain : A New Horizon. Sous la direction de Anthony J. Bazzan et Daniel A. Monti. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190690557.003.0001.
Texte intégralChapitres de livres sur le sujet "Permeabilità intestinale"
First, Michael B., Elizabeth Spencer, Elizabeth Spencer, Sander Begeer, Brynn Thomas, Danielle Geno Kent, Maria Fusaro et al. « Intestinal Permeability Studies ». Dans Encyclopedia of Autism Spectrum Disorders, 1650–52. New York, NY : Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_28.
Texte intégralReynolds, Ann. « Intestinal Permeability Studies ». Dans Encyclopedia of Autism Spectrum Disorders, 2549–51. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_28.
Texte intégralBjarnason, I., A. Macpherson et I. S. Menzies. « Intestinal permeability : the basics ». Dans Inflammatory Bowel Disease, 53–70. Dordrecht : Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-009-0371-5_6.
Texte intégralWesterbeek, E. A. M., B. Stahl et R. M. van Elburg. « Human milk and intestinal permeability ». Dans Handbook of dietary and nutritional aspects of human breast milk, 99–116. The Netherlands : Wageningen Academic Publishers, 2013. http://dx.doi.org/10.3920/978-90-8686-764-6_5.
Texte intégralWells, C. L., et S. L. Erlandsen. « Bacterial Translocation : Intestinal Epithelial Permeability ». Dans Update in Intensive Care and Emergency Medicine, 131–49. Berlin, Heidelberg : Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80224-9_9.
Texte intégralPecoraro, C., M. T. Saravo, P. Stanziale, M. M. Balletta, G. Parrilli et G. Budillon. « Abnormal Intestinal Permeability in IgA Nephropathy ». Dans Current Therapy in Nephrology, 65–67. Boston, MA : Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0865-2_17.
Texte intégralOami, Takehiko, et Craig M. Coopersmith. « Measurement of Intestinal Permeability During Sepsis ». Dans Methods in Molecular Biology, 169–75. New York, NY : Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1488-4_15.
Texte intégralCaspary, W. F. « Wirkung nichtsteroidaler Antirheumatika auf die intestinale Permeabilität ». Dans Ökosystem Darm VI, 92–102. Berlin, Heidelberg : Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-85187-2_10.
Texte intégralFu, Linglin, Bobby J. Cherayil, Haining Shi, Yanbo Wang et Yang Zhu. « Intestinal Permeability and Transport of Food Allergens ». Dans Food Allergy, 41–67. Singapore : Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-6928-5_3.
Texte intégralMielants, H., E. M. Veys, S. Goemaere, M. De Vos, C. Cuvelier, M. Maertens et C. Ackerman. « Intestinal mucosal permeability in inflammatory rheumatic diseases ». Dans Side-Effects of Anti-Inflammatory Drugs 3, 80–88. Dordrecht : Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2982-4_12.
Texte intégralActes de conférences sur le sujet "Permeabilità intestinale"
Guliaev, Sergei, Leonid Strizhakov, Sergey Moiseev, Ivan Gmoshinskii et Vladimir Mazo. « SAT0229 INTESTINAL PERMEABILITY IN IGA-VASCULITIS IN ADULTS ». Dans Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3722.
Texte intégralSprooten, Roy T. M., I. Grimbergen, D. Braeken, K. Lenaerts, E. Rutten, E. F. M. Wouters et G. G. U. Rohde. « Increased small intestinal permeability during severe acute exacerbations of COPD ». Dans ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.oa3525.
Texte intégralAubert, P., J. Chevalier, T. Durand, A. Bessard, O. Kelber, H. Abdel-Aziz et M. Neunlist. « Intestinal permeability induced by psychological stress : Action of STW 5 ». Dans GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608493.
Texte intégralSinghvi, D. G., B. Methe, K. Li, A. Fitch, Y. Zhang, F. C. Sciurba, S. M. Nouraie, A. Morris et J. M. Bon. « Influence of Smoking on Intestinal Microbiota Composition and Permeability in COPD ». Dans American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6283.
Texte intégralSeethaler, B., S. Beetz, M. Basrai, A. Schweinlin, J. Walter, M. Laville, NM Delzenne et SC Bischoff. « Auswirkungen einer Lebensstilveränderung auf die intestinale Permeabilität bei adipösen TeilnehmerInnen eines Gewichtsreduktionsprogramms ». Dans Kongress Ernährung 2020 – Medizin fürs Leben. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1710245.
Texte intégralAubert, P., J. Chevalier, T. Durand, A. Bessard, O. Kelber, H. Aziz-Kalbhenn et M. Neunlist. « Stress-induced changes in intestinal permeability in mice : Influence of STW 5 ». Dans Phytotherapiekongress 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1697324.
Texte intégralBarreto, Mario, Maurizio Simmaco, Luigi Principessa, Alfonso Lostia, Rosanna Zambardi, Susanna Bonafoni, Valentina Negro, Carla Olita et Maria P. Villa. « Asthma And Intestinal Permeability In Children : Role Of Age, Symptoms And Allergen Sensitization ». Dans American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6250.
Texte intégralAubert, P., J. Chevalier, T. Durand, A. Bessard, O. Kelber, H. Abdel-Aziz et M. Neunlist. « STW 5 prevents stress-induced changes in intestinal permeability in mice in-vivo ». Dans GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608490.
Texte intégralLuneva, O. E. « Food supplement “carrageenan” and its effect on the organism ». Dans VIII Information school of a young scientist. Central Scientific Library of the Urals Branch of the Russian Academy of Sciences, 2020. http://dx.doi.org/10.32460/ishmu-2020-8-0014.
Texte intégralNogueira, Fábio Dias, Ana Klara Rodrigues Alves, Barbara Beatriz Lira da Silva, Ana Kamila Rodrigues Alves, Marlilia Moura Coelho Sousa, Ana Karla Rodrigues Alves, Wanderson da Silva Nery, Breno Carvalho de Almeida, Flávia Dias Nogueira et Leiz Maria Costa Véras. « The autistic spectrum disorder and its relation to intestinal dysbiosis ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.283.
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