Littérature scientifique sur le sujet « Periodontal disease Microbiology »
Créez une référence correcte selon les styles APA, MLA, Chicago, Harvard et plusieurs autres
Consultez les listes thématiques d’articles de revues, de livres, de thèses, de rapports de conférences et d’autres sources académiques sur le sujet « Periodontal disease Microbiology ».
À côté de chaque source dans la liste de références il y a un bouton « Ajouter à la bibliographie ». Cliquez sur ce bouton, et nous générerons automatiquement la référence bibliographique pour la source choisie selon votre style de citation préféré : APA, MLA, Harvard, Vancouver, Chicago, etc.
Vous pouvez aussi télécharger le texte intégral de la publication scolaire au format pdf et consulter son résumé en ligne lorsque ces informations sont inclues dans les métadonnées.
Articles de revues sur le sujet "Periodontal disease Microbiology"
1
Moore, W. E. C. « Microbiology of periodontal disease ». Journal of Periodontal Research 22, no 5 (septembre 1987) : 335–41. http://dx.doi.org/10.1111/j.1600-0765.1987.tb01595.x.
Texte intégral
2
Jenkinson, Howard F., et David Dymock. « The Microbiology of Periodontal Disease ». Dental Update 26, no 5 (2 juin 1999) : 191–97. http://dx.doi.org/10.12968/denu.1999.26.5.191.
Texte intégral
3
Peros, William J., et Eugene D. Savitt. « The microbiology of periodontal disease ». Clinical Microbiology Newsletter 11, no 7 (avril 1989) : 49–51. http://dx.doi.org/10.1016/0196-4399(89)90009-3.
Texte intégral
4
Ledwon, Beata, Andrzej Miskiewicz, Ewa Grabowska, Jan Kowalski et Renata Górska. « The Relationship between Periodontal Disease and Motor Impairment in the Course of Parkinson’s Disease ». Postępy Higieny i Medycyny Doświadczalnej 74 (12 août 2020) : 340–47. http://dx.doi.org/10.5604/01.3001.0014.3516.
Texte intégralRésumé :
Introduction: The incidence of Parkinson’s disease and the severity of accompanying motor impairment increase significantly with age. The etiopathogenesis and progression of Parkinson’s disease at the molecular level is associated with the production of cytokines and acute phase proteins, which are also typical for inflammatory diseases, such as periodontitis and gingivitis. Objectives: The aim of the study was to assess the correlation between neurological parameters, the indices of periodontal status and systemic parameters of inflammation, as well as their change after treatment. Patients/Methods: The presented study is a retrospective analysis of data obtained from medical histories and patient charts. Charts of 93 patients diagnosed with Parkinson’s disease and periodontal diseases over the period 2015–2017 were selected. Sixty-one of these patients received periodontal treatment: professional scaling, root planning – SRP and periodontal pockets rinsing with 3% H2O2 and constituted a study group. Additionally, the patients were instructed to use a 0.2% chlorhexidine mouthwash. The other 32 patients, who were not periodontally treated, formed a control group. Both groups continued their anti-parkinsonian treatment. Results: The mean pocket depth at the baseline was 4.0 mm (SD 0.9 mm), mean bleeding index was 56.2%, and 63.9% of patients presented tooth mobility grade II or III. A significant correlation between periodontal and neurological parameters was observed at the baseline. After periodontal treatment, an improvement of both periodontal parameters and those related to the Parkinson’s disease was observed in the study group. Those periodontally treated exhibited lower number of anti-parkinsonian medicines, lower number of falls, as well as better results in10-m walk test and timed-up-and-go test, as compared to the control group. The improvement was observed both 3 and 9 months after the end of treatment.
5
Payne, M. A., A. Hashim, A. Alsam, S. Joseph, J. Aduse-Opoku, W. G. Wade et M. A. Curtis. « Horizontal and Vertical Transfer of Oral Microbial Dysbiosis and Periodontal Disease ». Journal of Dental Research 98, no 13 (27 septembre 2019) : 1503–10. http://dx.doi.org/10.1177/0022034519877150.
Texte intégralRésumé :
One of the hallmark features of destructive periodontal disease, well documented over the last 50 y, is a change to the quantitative and qualitative composition of the associated microbiology. These alterations are now generally viewed as transformational shifts of the microbial populations associated with health leading to the emergence of bacterial species, which are only present in low abundance in health and a proportionate decrease in the abundance of others. The role of this dysbiosis of the health associated microbiota in the development of disease remains controversial: is this altered microbiology the driving agent of disease or merely a consequence of the altered environmental conditions that invariably accompany destructive disease? In this work, we aimed to address this controversy through controlled transmission experiments in the mouse in which a dysbiotic oral microbiome was transferred either horizontally or vertically into healthy recipient mice. The results of these murine studies demonstrate conclusively that natural transfer of the dysbiotic oral microbiome from a periodontally diseased individual into a healthy individual will lead to establishment of the dysbiotic community in the recipient and concomitant transmission of the disease phenotype. The inherent resilience of the dysbiotic microbial community structure in diseased animals was further demonstrated by analysis of the effects of antibiotic therapy on periodontally diseased mice. Although antibiotic treatment led to a reversal of dysbiosis of the oral microbiome, in terms of both microbial load and community structure, dysbiosis of the microbiome was reestablished following cessation of therapy. Collectively, these data suggest that an oral dysbiotic microbial community structure is stable to transfer and can act in a similar manner to a conventional transmissible infectious disease agent with concomitant effects on pathology. These findings have implications to our understanding of the role of microbial dysbiosis in the development and progression of human periodontal disease.
6
Mahendra, Jaideep, Plato Palathingal, Little Mahendra, Janani Muralidharan, Khalid J. Alzahrani, Mohammed Sayed, Maryam H. Mugri et al. « Isolated Systolic Blood Pressure and Red-Complex Bacteria—A Risk for Generalized Periodontitis and Chronic Kidney Disease ». Microorganisms 10, no 1 (27 décembre 2021) : 50. http://dx.doi.org/10.3390/microorganisms10010050.
Texte intégralRésumé :
Hypertension is a risk factor for generalized periodontitis (GP) and chronic kidney diseases (CKD). However, the role of isolated systolic blood pressure as one of the major risks for these inflammatory diseases has not been explored. Very limited studies exist identifying the red-complex bacteria in association with the isolated systolic blood pressure. Hence, the main objective of this study was to assess the isolated systolic blood pressure and the red-complex bacteria along with the demographic variables, periodontal parameters, and renal parameters in patients with generalized periodontitis and chronic kidney disease. One hundred twenty participants (age 30–70 years) were divided into four groups—Group C: control (systemically and periodontally healthy subjects), Group GP: generalized periodontitis, Group CKD: subjects with CKD with good periodontal health, Group CKD + GP: subjects with both generalized periodontitis and CKD. Demographic variables and periodontal parameters were measured and recorded. Blood pressure measurements and a detailed history and renal parameters such as serum creatinine, eGFR, and fasting blood sugar were recorded. The red-complex bacteria (RCB) were assessed in the subgingival plaque samples of all four groups using RT-PCR. Older participants (above 50 years) showed worse periodontal scores in the CKD + GP group along with elevated isolated systolic blood pressure, higher serum creatinine, and fasting blood sugar. eGFR was significantly decreased compared to the other groups. Bacterial counts were higher in the GP + CKD group, suggesting that they may be at a higher risk for generalized periodontitis and chronic kidney disease. Isolated systolic blood pressure (ISBP) and RCB were significantly correlated with the renal and periodontal parameters. A log-linear relationship exists between periodontal disease, CKD, RCB, and isolated systolic hypertension levels.
7
K L Tsang, Annetta, Saso Ivanovski et Philip S Bird. « Caries and periodontal disease : Two diseases, one biofilm ». Microbiology Australia 26, no 3 (2005) : 110. http://dx.doi.org/10.1071/ma05110.
Texte intégralRésumé :
Dental plaque, a natural oral biofilm is involved in the aetiology of dental caries and periodontal disease. Despite decades of research, the microbiology, aetiology and pathogenesis of these diseases remain controversial. A number of factors interplay in these diseases, the indigenous microbes that inhabit the oral cavity, diet, host susceptibility and time. The ?Non-Specific Plaque Hypothesis? (NSPH) was proposed where the overall mass of plaque interacted with the host and caused disease. An alternative view was the ?Specific Plaque Hypothesis? (SPH) where, among the diverse microbial community, a limited subset of specific bacteria were associated with disease. In recent years, the ?Ecological Plaque Hypothesis? (EPH) has been proposed that it be recognised that the oral ecology as a whole contributes to the aetiology of dental caries and periodontal diseases, with shifts in the composition of microbial communities being of particular importance.
8
Peterson, D. E., G. E. Minah, C. D. Overholser, J. B. Suzuki, L. G. DePaola, D. M. Stansbury, L. T. Williams et S. C. Schimpff. « Microbiology of acute periodontal infection in myelosuppressed cancer patients. » Journal of Clinical Oncology 5, no 9 (septembre 1987) : 1461–68. http://dx.doi.org/10.1200/jco.1987.5.9.1461.
Texte intégralRésumé :
This study characterized the subgingival microbial flora associated with 27 acute exacerbations of preexistent periodontal disease in 24 patients with chemotherapy-induced myelosuppression. All but two acute periodontal infections developed at low granulocyte levels (less than 1,000/microL). Suspected pathogens were detected in high concentrations in subgingival plaque specimens in 17 episodes of acute periodontal infection; a single pathogen was recovered in ten acute infections, and more than one pathogen was recovered in seven acute infections. Staphylococcus epidermidis, Candida albicans, S aureus, and Pseudomonas aeruginosa predominated, with combinations of these detected in some patients. Concomitant bacteremias developed in two of these patients. The subgingival microflora associated with ten acute periodontal infections was characterized by predominantly indigenous microorganisms, which in nine episodes were in abnormal proportions compared with microbial profiles in noncancer patients with similar degrees of periodontal disease. These data demonstrate that pathogens normally associated with infections in myelosuppressed cancer patients, as well as indigenous oral flora, are associated with acute periodontal infections during granulocytopenia. This finding is important, since this body site has not commonly been recognized as a source for acute infection in these patients.
9
Hassell, Thomas M., et Emily L. Harris. « Genetic Influences in Caries and Periodontal Diseases ». Critical Reviews in Oral Biology & ; Medicine 6, no 4 (octobre 1995) : 319–42. http://dx.doi.org/10.1177/10454411950060040401.
Texte intégralRésumé :
Deciphering the relative roles of heredity and environmental factors ("nature vs. nurture") in the pathogenesis of dental caries and diseases of the periodontium has occupied clinical and basic researchers for decades. Success in the endeavor has come more easily in the case of caries; the complex interactions that occur between host-response mechanisms and putative microbiologic pathogens in periodontal disease have made elucidation of genetic factors in disease susceptibility more difficult. In addition, during the 30-year period between 1958 and 1987, only meager resources were targeted toward the "nature" side of the nature/nurture dipole in periodontology. In this article, we present a brief history of the development of genetic epistemology, then describe the three main research mechanisms by which questions about the hereditary component of diseases in humans can be addressed. A critical discussion of the evidence for a hereditary component in caries susceptibility is next presented, also from a historical perspective. The evolution of knowledge concerning possible genetic ("endogenous", "idiotypic") factors in the pathogenesis of inflammatory periodontal disease is initiated with an analysis of some foreign-language (primarily German) literature that is likely to be unfamiliar to the reader. We identify a turning point at about 1960, when the periodontal research community turned away from genetics in favor of microbiology research. During the past five years, investigators have re-initiated the search for the hereditary component in susceptibility to common adult periodontal disease; this small but growing body of literature is reviewed. Recent applications of in vitro methods for genetic analyses in periodontal research are presented, with an eye toward a future in which persons who are at risk-genetically predisposed-to periodontal disease may be identified and targeted for interventive strategies. Critical is the realization that genes and environment do not act independently of each other; the appearance or magnitude of heritability may differ with various environments.
10
Darby, Ivan, et Michael Curtis. « Microbiology of periodontal disease in children and young adults ». Periodontology 2000 26, no 1 (juin 2001) : 33–53. http://dx.doi.org/10.1034/j.1600-0757.2001.2260103.x.
Texte intégralThèses sur le sujet "Periodontal disease Microbiology"
1
Martins, Angela Guimarães. « Avaliação clinica e microbiologica do uso de pontas sonicas diamantadas na descontaminação radicular (estudo longitudinal) ». [s.n.], 2005. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290413.
Texte intégralRésumé :
Orientador: Antonio Wilson SallumTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
Made available in DSpace on 2018-08-05T21:20:51Z (GMT). No. of bitstreams: 1 Martins_AngelaGuimaraes_D.pdf: 694133 bytes, checksum: 433f44b9064bb37a64b1dd3af169f058 (MD5) Previous issue date: 2005
Resumo: O objetivo deste estudo foi avaliar longitudinalmente a efetividade de pontas sônicas diamantadas (RootplanerTM), na descontaminação de superfícies radiculares de dentes unirradiculares sob parâmetros clínicos (índice de placa, sangramento à sondagem, profundidade de sondagem, nível de inserção clínica relativo, e retração gengival) e microbiológicos (cultura microbiana e PCR), comparados a instrumentação com curetas. Esta pesquisa trata-se de um teste clínico controlado randomizado com um delineamento em parcela subdividida, envolvendo 12 pacientes com moderada a avançada periodontite crônica apresentando profundidade de sondagem ³ 5 mm. Os pacientes foram submetidos a tratamento inicial com sessões de remoção de placa e cálculo supragengival e instruções de higiene bucal. Posteriormente, retalho cirúrgico foi realizado e a superfície dos dentes instrumentada. Os parâmetros clínicos e microbiológicos foram avaliados no tempo inicial (T0), e em 30 dias, 6, 12 e 24 meses após a cirurgia. Os dados obtidos foram submetidos à análise estatística através de Análise de variância (ANOVA), teste de Tukey, realizados para todos os períodos (a=0.05), Friedman e Wilcoxon foram usados na avaliação não paramétrica das unidades formadoras de colônias. Os resultados mostraram que quando as pontas sônicas diamantadas foram comparadas a instrumentos manuais nenhuma diferença estatística foi encontrada em redução de profundidade de sondagem, índice de placa, sangramento a sondagem, ganho de inserção, recessão gengival. Bem como, nas reduções das contagens microbiológicas das UFCs e diminuição de A.a., P.g. e T.f.. Os resultados não mostraram diferença significativa entre os tratamentos, apenas entre os períodos de avaliação. Concluiu-se, dentro das limitações do estudo que as pontas sônicas diamantadas podem ser uma boa alternativa terapêutica para instrumentação radicular, mantendo a estabilidade da saúde dos tecidos periodontais em longo prazo
Abstract: The aim of this study was to evaluate the long term the effectiveness of diamond-coated sonic scaler insert (RootplanerTM), in the root surface debridement in single-rooted teeth on clinical (plaque index, bleeding on probing, probing depth, relative attachment level clinical, and gingival recession) and microbiological parameters (culture and PCR), compared to hand instruments. The investigation was a test-controlled randomized clinical trial with a splith-mouth design involving 12 patients with moderate to advanced chronic periodontites with pocket depth ³ 5 mm. The patients were submitted to initial treatment sessions of supragingival plaque and cauculus removal and were also instructed in oral hygiene techniques. After, flap surgery was carried out and the tooth surface instrumented. Clinical and microbiological parameters, were evaluated at baseline (0), in the 30 days, 6, 12 and 24 months after surgery. Analysis of variance and Tukey test was done goes all periods (a=0.05) and the results showed that when diamond-coated sonic scaler insert was compared to manual scalers, showed no statistical differences in probing depth reduction, plaque index, bleeding on probing, relative attachment level gain, gingival recession. As well as, reductions of the microbial counts and colony-forming units using Friedman and Wilcoxon test, and decrease of A.a, P.g. and T.f.. There were no significant differences between regard with treatments for all parameters at each re-examination. Within the limits of this study, it can be concluded that the use the of diamond-coated sonic scaler insert can be a good alternative for root debridement, maintaining the stability of the periodontal tissues in long term
Doutorado
Periodontia
Doutor em Clínica Odontológica
2
Cruz, Gabriela Alessandra da. « Raspagem e alisamento radicular realizado em uma unica sessão, em pacientes com diabetes melito, portadores de periodontite cronica : avaliações clinica e laboratorial ». [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/289998.
Texte intégralRésumé :
Orientadores: Sergio de Toledo, Enilson Antonio SallumTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
Made available in DSpace on 2018-08-08T01:22:04Z (GMT). No. of bitstreams: 1 Cruz_GabrielaAlessandrada_D.pdf: 559034 bytes, checksum: ebadda53606b61a7511fea24078716a2 (MD5) Previous issue date: 2007
Resumo: O objetivo deste estudo foi a avaliação clínica, hematológica e microbiológica do tratamento periodontal para ambos os grupos após realizar tratamento de raspagem e alisamento radicular em única sessão. Esse trabalho é um estudo prospectivo, paralelo e comparativo realizado em pacientes com doença periodontal crônica generalizada divididos em dois grupos, sendo 10 pacientes com diabetes melito insulino-dependente (DM) e 10 pacientes não diabéticos (NDM) acompanhados por 3 meses. Os dados obtidos nos períodos inicial e final foram divididos em parâmetros clínicos: índice de placa, Índice gengival, nível clínico de inserção, nível da margem gengival e profundidade de sondagem; parâmetros hematológicos: hemácias, hemoglobina, hematócrito, volume corpuscular médio, hemoglobina corpuscular média, concentração de hemoglobina corpuscular média, leucócitos, eosinófilos, metamielócitos, segmentados, linfócitos, monócitos, plaquetas, bastonetes, basófilos, glicemia, hemoglobina glicosilada (HbA1c), prova do laço, tempo de coagulação e sangramento e parâmetros microbiológicos, ¿pool¿ de bactéria por paciente, para os sítios com PS>5mm e regiões de bifurcação. As amostras foram obtidas com curetas tipo Gracey e analisadas através da técnica de reação de polimerase em cadeia ¿ PCR para verificação da freqüência de Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis e Tannarella forsythensis. As avaliações clinicas e hematológicas não demonstraram diferenças estatísticas significantes entre grupos NDM e DM nos períodos iniciais e final. A avaliação microbiológica demonstrou diminuição de Tannarella forsythensis estatisticamente significativa apenas para o grupo NDM (p=0,0313) após 3 meses. Este estudo sugere que os pacientes com DM apresentaram respostas clinica e laboratorial similares ao grupo NDM após a terapia de raspagem e alisamento radicular realizados em única sessão após o período de acompanhamento de 3 meses
Abstract: The aim of this study was to compare the clinical, hematological and microbiological responses of periodontal treatment for both groups after full-mouth scaling and root planning. This study is a prospective, parallel, comparative, clinical, hematological and microbiological study performed in patients with generalized chronic periodontal disease separated in two groups, with 10 patients with diabetes mellitus insulin-dependent (DM) and 10 patients non-diabetics (NDM) attended by 3 months. The data from baseline and 3 months were separated in clinical parameters, plaque Index, gingival index, clinical attachment level, gingival margin level and probing depth; hematological parameters, red blood cell count, hemoglobin, hematocrit, mean corpuscular volume, mean corpuscular hemoglobin, mean corpuscular hemoglobin concentration, leucocytes, eosinophils, metamielocytes, segmenteds, linfocytes, monocytes, platelets, bastonets, basophil, glucose, HbA1c, bleeding time and coagulation time and microbiological parameters, pool of bacterial per patients that were obtained from sites with PD>5mm and furcation sites. The samples were obtained with Gracey curettes and analyzed by technical of polymerase chain reaction ¿ PCR, to verify the frequency of Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis e Tannarella forsythensis. The clinical and hematological evaluations had no statistics significance changes between groups NDM and DM. The microbiological evaluation showed decrease of Tannarella forsythensis statistically significant for only NDM group (p=0,0313). This study suggest that diabetes mellitus patients respond to full-mouth scaling and root planning similar to non diabetics patients 3 months after therapy
Doutorado
Periodontia
Mestre em Clínica Odontológica
3
Irani, Dilshad Minocher. « Role of the surface associated material of Eikenella corrodens in bone resorption associated with periodontal disease : a research thesis submitted in fulfilment of the requirements for the degree of Master of Science in Dentistry ». Title page, contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09DSM/09dsmi65.pdf.
Texte intégral
4
Gully, Neville. « Studies on the growth and metabolism of Eikenella corrodens / ». Title page, summary and table of contents only, 2000. http://web4.library.adelaide.edu.au/theses/09PH/09phg973.pdf.
Texte intégral
5
Rocha, Marisa Manuela Pereira. « Probióticos e a Doença Periodontal ». Master's thesis, [s.n.], 2013. http://hdl.handle.net/10284/3918.
Texte intégralRésumé :
Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Medicina DentáriaCom o progresso da ciência nas últimas décadas, a Medicina Dentária tem evoluído de forma rápida, tendo vindo a assistir-se a um progressivo aumento e consequente utilização de novos produtos e métodos que visam optimizar e acrescentar soluções terapêuticas. Neste contexto, as bactérias, designadamente os “probióticos”, representam um campo de pesquisa promissor, além de um possível instrumento para auxílio na prática clínica diária. Ao lhes serem reconhecidas as suas capacidades de inibirem o crescimento de microrganismos patogénicos, os probióticos poderão ser, num futuro próximo, uma alternativa preventiva e de tratamento de múltiplas doenças, das quais a doença periodontal não é excepção. A doença periodontal ainda permanece, em todo o mundo, com alta prevalência e incidência, sendo aliás considerada um problema de saúde pública, que afecta a qualidade de vida de muitas pessoas. É considerada uma doença de elevada complexidade quanto à natureza da sua etiologia, classificação das diferentes formas, tratamento e sua manutenção. O objectivo deste trabalho foi avaliar, através de uma revisão bibliográfica, de que forma o uso de probióticos pode influenciar a condição periodontal. Apesar dos dados promissores, não é ainda possível, dadas as limitações dos estudos disponíveis, tecer conclusões definitivas sobre a influência dos probióticos na condição periodontal, pelo que mais estudos são necessários. With the progress of science in the last decades, Dentistry has evolved rapidly and it has been witnessed a progressive increase and consequent use of new products and methods in order to optimize and add new therapeutical solutions. In this context, bacteria, in particular probiotics, represent a promising search area besides being a possible instrument of aid in daily clinical practice. Being recognizable its capacities to inhibit pathogenic microrganisms´ growth, they are able to be, in the near future, an alternative of prevention and treatment of multiple diseases of which periodontal disease is not an exception. Periodontal disease still remains worldwide with high prevalence and incidence being in fact considered a public health problem which affects the quality of life of several people. It is considered a disease of high complexity relatively to its etiology, classification of its different forms, treatment and maintenance. The aim of this work was to evaluate trough a bibliographic revision, the way that the use of probiotics may influence the periodontal condition. Despite the promising data, it is not yet possible, given the limitations of the available studies, weaving definitive conclusions about the influence of probiotics in periodontal condition, so further studies are needed.
6
Tam, You-Cheuk. « The role of mucopolysaccharidase-producing anaerobic oral bacteria in the pathogenesis of inflammatory periodontal disease / ». Thesis, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=72044.
Texte intégralRésumé :
Mucopolysaccharidase-producing oral bacteria may contribute to the pathogenesis of inflammatory periodontal disease in several ways. Bacterial mucopolysaccharidases, in the area of the gingival crevice, can destroy important components of the ground substance of connective tissue leading to periodontal destruction on the one hand and enhancing the spread of bacterial toxins on the other. Partial breakdown of proteoglycan due to penetration of small amounts of these enzymes may also expose cryptic antigenic determinants, resulting in destructive autoimmune reactions. Finally, mucopolysaccharidase-producing oral bacteria may interact symbiotically with other pathogens of the gingival sulcus by enzymatic breakdown of tissue to release fermentable substrates for these pathogens.In the present investigation, it has been shown that anaerobic mucopolycaccharidase-producing bacteria are common inhabitants of gingival sulci of humans and that these microorganisms significantly increase in number in periodontal pockets of patients with inflammatory periodontal disease. Peptostreptococci probably are the most predominant producers of mucopolysaccharidases by virtue of their occurrence in subgingival plaque as well as the abundance of enzyme they produce. Peptostreptococci strains isolated from diseased periodontal pockets have been observed to convert from rough-colony-forming cells to smooth-colony-forming variants upon culturing in vitro. In dental plaque, hyaluronidase-producing peptostreptococci exist predominantly as the rough-colony-forming variants which produce higher amounts of hyaluronidase. Purified extracellular hyaluronidase from Peptostreptococcus strain 84H14S is different from previously reported bacterial hyaluronidases in several respects. It has different substrate specificity and optimum pH for activity. Also, the specific activity of this enzyme is much higher than any previously purified mucopolysaccharidases. Peptostreptococcus strain 84H14S is further shown to release potent cytotoxic factors into the culture medium, in addition to hyaluronidase, during its growth cycle. This may confer additional virulence to this bacterial genus in periodontal disease.
7
Sweet, Simon Paul. « Adherence, aggregation and hydrophobicity of oral bacteria : with particular reference to microorganisms implicated in periodontal disease ». Thesis, University of Glasgow, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236908.
Texte intégral
8
Silva, Emílio Barbosa e. [UNESP]. « Avaliação clínica e microbiológica de diferentes modalidades de terapia periodontal não-cirúrgica em pacientes com periodontite crônica ». Universidade Estadual Paulista (UNESP), 2004. http://hdl.handle.net/11449/104724.
Texte intégralRésumé :
Made available in DSpace on 2014-06-11T19:33:28Z (GMT). No. of bitstreams: 0
Previous issue date: 2004-03-05Bitstream added on 2014-06-13T20:45:01Z : No. of bitstreams: 1
silva_eb_dr_arafo.pdf: 2283935 bytes, checksum: a7ddd656bfe4bd69726cb26dbb45597f (MD5)Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)
Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
O objetivo deste estudo foi avaliar em pacientes portadores de periodontite crônica avançada, os efeitos de diferentes modalidades de tratamento periodontal não-cirúrgico na composição microbiana subgengival e nos parâmetros clínicos de avaliação da doença periodontal em diferentes tempos de análise. Foram avaliados 30 pacientes com idade entre 25 e 68 anos com profundidade à sondagem inicial d 6mm e = 8 mm nos sítios selecionados. Estes pacientes foram divididos em três grupos, cada um contendo 10 pessoas de acordo com o tratamento realizado: Grupo 1 - realização de raspagem e alisamento radiculares supra e subgengivais concomitantemente no mesmo dia; Grupo 2 - realização de raspagem alisamento e polimento supragengival e 7 dias após realização de raspagem e alisamento radiculares subgengival; Grupo 3 - realização de raspagem alisamento e polimento somente supragengival por 30 dias e complementação da raspagem subgengival após este período. Os pacientes foram avaliados antes e após 7, 15, 30 e 60 dias de realizado o procedimento inicial pelos seguintes parâmetros clínicos: profundidade de sondagem, nível de inserção, sangramento à sondagem, presença de placa, inflamação gengival e supuração. Também foram realizadas coletas de amostras de placa subgengival nos mesmos sítios e períodos e analisadas a prevalência e os níveis de 36 espécies bacterianas pela técnica do checkerboard DNA-DNA hybridization. Como resultados deste estudo verificamos que a profundidade de sondagem foi significativamente reduzida pelos tratamentos de raspagem supra e subgengival concomitante e raspagem supra 7 dias prévios a subgengival. O grupo que recebeu somente raspagem supragengival até o 30º dia não apresentou melhora até este período, com uma redução significativa após a complementação da raspagem... .
The purpose of the present investigation was to evaluate the effects of different modalities of non-surgical periodontal treatment in 30 patients with advanced chronic periodontitis. Subjects were monitored clinically and microbiologically prior and 7, 15, 30 and 60 days after this three periodontal treatments: Group 1 - concomitant supra and subgingival scaling and root planning; Group 2 - supragingival scaling 7 days prior to subgingival scaling and root planning; Group 3 - supragingival scaling 30 days prior to subgingival scaling and root planning. Clinical assessment of plaque, redness, suppuration, bleeding on probing, pocket depth and clinical attachment level were made in two anterior teeth per patient. Subgingival plaque samples were taken at the same sites and the presence and levels of 36 subgingival bacteria species were determined using checkerboard DNA-DNA hybridization. The mean levels and % colonized by each species (prevalence) was computed for each subject at each visit. As results of this study was verified that pocket depth was significantly reduced for group 1 and 2. The group that received only supragengival scaling until the 30th day didn't improve until this period, with a significant reduction after the complementation of subgengival scaling and root planning. The clinical attachment level parameter didn't demonstrate significant alteration in none of the three groups along the time. However all the groups showed improvement or stability in this parameter, tends the group 1 presented the best results, followed for the group 2 and 3, respectively. All the groups didn't present differences between the initial period and the times of 30 and 60 days in relation to the other parameters of clinical evaluation, except for the gingival redness that had group 2 with better results, followed for the group 1 and 3... (Complete abstract, click electronic address below).
9
Silva, Emílio Barbosa e. « Avaliação clínica e microbiológica de diferentes modalidades de terapia periodontal não-cirúrgica em pacientes com periodontite crônica / ». Araraquara : [s.n.], 2004. http://hdl.handle.net/11449/104724.
Texte intégralRésumé :
Orientador: Benedicto Egbert Corrêa de ToledoBanca: Enilson Antônio Sallum
Banca: Márcio Fernando de Moraes Grisi
Banca: Silvana Regina Perez Orrico
Banca: Valdir Gouvêia Garcia
Resumo: O objetivo deste estudo foi avaliar em pacientes portadores de periodontite crônica avançada, os efeitos de diferentes modalidades de tratamento periodontal não-cirúrgico na composição microbiana subgengival e nos parâmetros clínicos de avaliação da doença periodontal em diferentes tempos de análise. Foram avaliados 30 pacientes com idade entre 25 e 68 anos com profundidade à sondagem inicial d 6mm e = 8 mm nos sítios selecionados. Estes pacientes foram divididos em três grupos, cada um contendo 10 pessoas de acordo com o tratamento realizado: Grupo 1 - realização de raspagem e alisamento radiculares supra e subgengivais concomitantemente no mesmo dia; Grupo 2 - realização de raspagem alisamento e polimento supragengival e 7 dias após realização de raspagem e alisamento radiculares subgengival; Grupo 3 - realização de raspagem alisamento e polimento somente supragengival por 30 dias e complementação da raspagem subgengival após este período. Os pacientes foram avaliados antes e após 7, 15, 30 e 60 dias de realizado o procedimento inicial pelos seguintes parâmetros clínicos: profundidade de sondagem, nível de inserção, sangramento à sondagem, presença de placa, inflamação gengival e supuração. Também foram realizadas coletas de amostras de placa subgengival nos mesmos sítios e períodos e analisadas a prevalência e os níveis de 36 espécies bacterianas pela técnica do "checkerboard DNA-DNA hybridization". Como resultados deste estudo verificamos que a profundidade de sondagem foi significativamente reduzida pelos tratamentos de raspagem supra e subgengival concomitante e raspagem supra 7 dias prévios a subgengival. O grupo que recebeu somente raspagem supragengival até o 30º dia não apresentou melhora até este período, com uma redução significativa após a complementação da raspagem... (Resumo completo, clicar acesso eletrônico abaixo).
Abstract: The purpose of the present investigation was to evaluate the effects of different modalities of non-surgical periodontal treatment in 30 patients with advanced chronic periodontitis. Subjects were monitored clinically and microbiologically prior and 7, 15, 30 and 60 days after this three periodontal treatments: Group 1 - concomitant supra and subgingival scaling and root planning; Group 2 - supragingival scaling 7 days prior to subgingival scaling and root planning; Group 3 - supragingival scaling 30 days prior to subgingival scaling and root planning. Clinical assessment of plaque, redness, suppuration, bleeding on probing, pocket depth and clinical attachment level were made in two anterior teeth per patient. Subgingival plaque samples were taken at the same sites and the presence and levels of 36 subgingival bacteria species were determined using checkerboard DNA-DNA hybridization. The mean levels and % colonized by each species (prevalence) was computed for each subject at each visit. As results of this study was verified that pocket depth was significantly reduced for group 1 and 2. The group that received only supragengival scaling until the 30th day didn't improve until this period, with a significant reduction after the complementation of subgengival scaling and root planning. The clinical attachment level parameter didn't demonstrate significant alteration in none of the three groups along the time. However all the groups showed improvement or stability in this parameter, tends the group 1 presented the best results, followed for the group 2 and 3, respectively. All the groups didn't present differences between the initial period and the times of 30 and 60 days in relation to the other parameters of clinical evaluation, except for the gingival redness that had group 2 with better results, followed for the group 1 and 3... (Complete abstract, click electronic address below).
Doutor
10
Bosco, Joseane Maria Dias [UNESP]. « Estudo longitudinal da terapia antibiótica local de bolsas periodontais residuais. Análise clínica e microbiológica ». Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/104702.
Texte intégralRésumé :
Made available in DSpace on 2014-06-11T19:33:27Z (GMT). No. of bitstreams: 0
Previous issue date: 2007-02-12Bitstream added on 2014-06-13T18:45:12Z : No. of bitstreams: 1
bosco_jmd_dr_arafo.pdf: 381181 bytes, checksum: 5b8b6929d9ffb9e26d5e951e6958d4a3 (MD5)Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Objetivos: Dois estudos controlados cegos aleatórios foram realizados para avaliar os resultados clínicos e microbiológicos iniciais e a longo prazo, da tetraciclina aplicada com o microbrush no tratamento de sítios com bolsa periodontal persistente 5mm e sangramento à sondagem, tratados previamente por raspagem e alisamento radicular (RAR). Material e método: Quarenta e dois pacientes com pelo menos 4 dentes com bolsas periodontais 5 mm e sangramento à sondagem foram divididos em 2 grandes grupos: tetraciclina e controle. Nos grupos tetraciclina, dois dentes receberam aplicação de solução de cloridrato de tetraciclina (100mg/mL) com microbrush uma vez por semana por 4 semanas (grupo T) e os outros dois dentes receberam o mesmo tratamento associado a uma sessão de RAR na primeira consulta (grupo RAR+T). Nos grupos controle, dois dentes receberam uma sessão de RAR (grupo RAR) e outros dois dentes receberam aplicação de solução salina com microbrush uma vez por semana por 4 semanas associada a uma sessão de RAR na primeira consulta (grupo RAR+S). Medidas clínicas de índice de placa visível (IP), índice de sangramento gengival (IG), sangramento à sondagem (SS), profundidade de sondagem (PS), nível de inserção clínica (NIC), recessão gengival (RG), e coletas microbiológicas para identificação das bactérias Aggregatibacter actinomycetemcomitans (Actinobacillus actinomycetemcomitans), Porphyromonas gingivais, Prevotella intermédia, Prevotella nigrescens, Tannerella forsythia, e Treponema denticola foram tomadas no baseline, 30, 90 e 180 dias após o tratamento. Amostras de biofilme subgengival foram analisadas por reação de polimerase em cadeia. Resultados: Todas as terapias resultaram em melhora na PS, NIC e SS, embora as diferenças clínicas entre os grupos não tenham alcançado significância estatística.
Objectives: Two randomized controlled single-blind studies were carried out to evaluate the initial and the long term clinical and microbiological results of tetracycline applied with a microbrush in treatment of sites with persistent periodontal pockets 5mm and bleeding on probing previous treated by scaling and root planning (SRP). Methods: For each patient in the test groups, two teeth received 4 applications of tetracycline solution (100mg/ml) with a microbrush once a week (T group) while the other two teeth received the same treatment plus one session of SRP at the first appointment (SRP+T group). For each patient in the control groups, two teeth received one session of SRP (SRP group) and the other two received 4 applications of saline with a microbrush once a week plus one session of SRP (SRP+S group). Clinical measurements were taken at baseline, 30, 90 and 180 days after last treatment. Subgingival biofilme samples were analyzed by polymerase chain reaction for Aggregatibacter actinomycetemcomitans (Actinobacillus actinomycetemcomitans), Porphyromonas gingivais, Prevotella intermédia, Prevotella nigrescens, Tannerella forsythia, e Treponema denticola identification. Results: All therapies provided significant improvement in probing depth, clinical attachment level and bleeding on probing, although the clinical difference did not reach statistically significance. At 1 month post-therapy the prevalence of P. gingivalis was significantly reduced in groups that received SRP plus additional therapy and T. forsythia was significantly reduced in only test groups. Until 6 months P. gingivalis, T. forsythensis,and A. actinomycetemcomitans significantly decrease in prevalence only for tetracycline groups. Conclusion: The results of the present study suggest that tetracycline solution applied with a microbrush may be an alternative in management of persistent periodontal pockets with further microbiologic benefits.
Livres sur le sujet "Periodontal disease Microbiology"
1
Periodontal disease. Basel : Karger, 2012.
Trouver le texte intégral
2
PhD, Henderson Brian, dir. Periodontal medicine and systems biology. Chichester, United Kingdom : Blackwell, 2009.
Trouver le texte intégral
3
Periodontal diseases in children and adolescents. New York : Nova Science Publishers, 2011.
Trouver le texte intégral
4
Jakubovics, Nicholas S., et Palmer Robert J. Oral microbial ecology : Current research and new perspectives. Norfolk, UK : Caister Academic Press, 2013.
Trouver le texte intégral
5
sman, Bjo rn A. Juvenile periodontitis : Generation of free oxygen radicals and elastase by peripheral PMN cells. Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1988.
Trouver le texte intégral
6
Adriaens, Patrick A. Bacterial invasion in hard tissues of periodontally diseased teeth : Structural and cultural studies. Ghent, Belgium : P.A. Adriaens, 1988.
Trouver le texte intégral
7
Gmür, Rudolf. Value of new serological probes for the study of putative periodontal pathogens : A survey after five years of application. Chicago : Quintessence Pub. Co., 1995.
Trouver le texte intégral
8
J, Genco Robert, dir. Molecular pathogenesis of periodontal disease. Washington, D.C : ASM Press, 1994.
Trouver le texte intégral
9
J, Genco Robert, dir. Molecular pathogenesis of periodontaldisease. Washington, D.C : ASM Press, 1994.
Trouver le texte intégral
10
Bo, Krasse, et Carlos James P, dir. Microbiologic diagnosis in dental caries and periodontal disease : Proceedings of a conference sponsored by the National Institute of Dental Research, Bethesda, Maryland, U.S.A., June 2-3, 1986. Copenhagen, 1986.
Trouver le texte intégralChapitres de livres sur le sujet "Periodontal disease Microbiology"
1
Dumitrescu, Alexandrina L., et Masaru Ohara. « Periodontal Microbiology ». Dans Etiology and Pathogenesis of Periodontal Disease, 39–76. Berlin, Heidelberg : Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03010-9_2.
Texte intégral
2
Marsh, Philip, et Michael Martin. « Periodontal diseases ». Dans Oral Microbiology, 167–97. Boston, MA : Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-7556-6_7.
Texte intégral
3
Mitchell, David A., Laura Mitchell et Lorna McCaul. « Restorative dentistry 1 : periodontology ». Dans Oxford Handbook of Clinical Dentistry, 171–216. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199679850.003.0005.
Texte intégralRésumé :
Contents. Classification. Epidemiology of periodontal disease. Oral microbiology. Aetiology of periodontal disease. Plaque biofilm. Calculus. Progression and risk factors. Pathogenesis of gingivitis and periodontitis. Clinical features of gingivitis and periodontitis. Diagnosis and monitoring. Aggressive periodontitis. Necrotizing periodontal diseases. Periodontal abscess. Periodontitis associated with endodontic lesions. Principles of treatment. Non-surgical treatment—plaque control. Non-surgical periodontal therapy—scaling and root surface debridement. Treatment with antimicrobials. Periodontal surgery—principles. Periodontal surgery—types of surgery. Periodontal surgery—regenerative techniques. Periodontal surgery—mucogingival surgery. Furcation involvement. Occlusion and splinting. Peri-implant mucositis and peri-implantitis. Supportive periodontal therapy.
4
Hashim, Nada Tawfig. « Oral Microbiology in Periodontal Health and Disease ». Dans Oral Microbiology in Periodontitis. InTech, 2018. http://dx.doi.org/10.5772/intechopen.75709.
Texte intégral
5
MacFarlane, T. Wallace, et Lakshman P. Samaranayake. « Microbiology of periodontal diseases ». Dans Clinical Oral Microbiology, 51–74. Elsevier, 1989. http://dx.doi.org/10.1016/b978-0-7236-0934-6.50010-0.
Texte intégral
6
Gupte, Satish. « Periodontal Diseases ». Dans The Short Textbook of Medical Microbiology for Dental Students, 201. Jaypee Brothers Medical Publishers (P) Ltd., 2012. http://dx.doi.org/10.5005/jp/books/11710_34.
Texte intégral
7
Könönen, Eija, et Purnima S. Kumar. « Bacteriology of Periodontal Diseases ». Dans Molecular Medical Microbiology, 957–68. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-12-397169-2.00053-6.
Texte intégral