Littérature scientifique sur le sujet « Pathogen adaptation »
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Articles de revues sur le sujet "Pathogen adaptation"
Fonville, Judith M. « Expected Effect of Deleterious Mutations on Within-Host Adaptation of Pathogens ». Journal of Virology 89, no 18 (24 juin 2015) : 9242–51. http://dx.doi.org/10.1128/jvi.00832-15.
Texte intégralSánchez-Vallet, Andrea, Simone Fouché, Isabelle Fudal, Fanny E. Hartmann, Jessica L. Soyer, Aurélien Tellier et Daniel Croll. « The Genome Biology of Effector Gene Evolution in Filamentous Plant Pathogens ». Annual Review of Phytopathology 56, no 1 (25 août 2018) : 21–40. http://dx.doi.org/10.1146/annurev-phyto-080516-035303.
Texte intégralVanHook, Annalisa M. « Pathogen rewiring for host adaptation ». Science 370, no 6517 (5 novembre 2020) : 677.20–679. http://dx.doi.org/10.1126/science.370.6517.677-t.
Texte intégralSlev, Patricia R., et Wayne K. Potts. « Disease consequences of pathogen adaptation ». Current Opinion in Immunology 14, no 5 (octobre 2002) : 609–14. http://dx.doi.org/10.1016/s0952-7915(02)00381-3.
Texte intégralLaine, Anna-Liisa, Jeremy J. Burdon, Adnane Nemri et Peter H. Thrall. « Host ecotype generates evolutionary and epidemiological divergence across a pathogen metapopulation ». Proceedings of the Royal Society B : Biological Sciences 281, no 1787 (22 juillet 2014) : 20140522. http://dx.doi.org/10.1098/rspb.2014.0522.
Texte intégralHanford, Hannah E., Juanita Von Dwingelo et Yousef Abu Kwaik. « Bacterial nucleomodulins : A coevolutionary adaptation to the eukaryotic command center ». PLOS Pathogens 17, no 1 (21 janvier 2021) : e1009184. http://dx.doi.org/10.1371/journal.ppat.1009184.
Texte intégralFedderke, Johannes W., Robert E. Klitgaard et Valerio Napolioni. « Genetic adaptation to historical pathogen burdens ». Infection, Genetics and Evolution 54 (octobre 2017) : 299–307. http://dx.doi.org/10.1016/j.meegid.2017.07.017.
Texte intégralTASARA, T., et R. STEPHAN. « Cold Stress Tolerance of Listeria monocytogenes : A Review of Molecular Adaptive Mechanisms and Food Safety Implications ». Journal of Food Protection 69, no 6 (1 juin 2006) : 1473–84. http://dx.doi.org/10.4315/0362-028x-69.6.1473.
Texte intégralHenschen, Amberleigh E., Michal Vinkler, Marissa M. Langager, Allison A. Rowley, Rami A. Dalloul, Dana M. Hawley et James S. Adelman. « Rapid adaptation to a novel pathogen through disease tolerance in a wild songbird ». PLOS Pathogens 19, no 6 (9 juin 2023) : e1011408. http://dx.doi.org/10.1371/journal.ppat.1011408.
Texte intégralHoque, M. Mozammel, Parisa Noorian, Gustavo Espinoza-Vergara, Pradeep Manuneedhi Cholan, Mikael Kim, Md Hafizur Rahman, Maurizio Labbate et al. « Adaptation to an amoeba host drives selection of virulence-associated traits in Vibrio cholerae ». ISME Journal 16, no 3 (15 octobre 2021) : 856–67. http://dx.doi.org/10.1038/s41396-021-01134-2.
Texte intégralThèses sur le sujet "Pathogen adaptation"
Bacigalupe, Rodrigo. « Population genomic analysis of bacterial pathogen niche adaptation ». Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31266.
Texte intégralAmezaga, Herran Maria Rosario. « The adaptation of Listeria monocytogenes to osmotic stress ». Thesis, University of Aberdeen, 1996. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU602297.
Texte intégralBoixel, Anne-Lise. « Environmental heterogeneity, a driver of adaptation to temperature in foliar plant pathogen populations ? » Thesis, université Paris-Saclay, 2020. http://www.theses.fr/2020UPASA010.
Texte intégralEnvironmental drivers, most notably temperature, affect the biology of phyllosphere microorganisms but also induce changes in their population dynamics, even in their evolutionary trajectories. The impact of climate on foliar plant disease epidemics is usually considered in forecasting models to inform management strategies. Such models focus on averages of environmental drivers but disregard both individual variation within populations and the scale and extent of biologically relevant environmental changes. These simplifications are glossing over substantial levels of individual variation that may have important consequences on the capacity of a population to adapt to environmental changes, and thus on the dynamics of epidemics in a fluctuating or changing climate. To examine the range of validity and consequences of these simplifying assumptions, I investigated how individual variation and environmental heterogeneity jointly affect fitness, phenotypic composition and resilience of populations of a foliar pathogen (Zymoseptoria tritici) inhabiting wheat canopies. Three complementary ways of exploration were adopted in this case study. First, an in vitro high-throughput phenotyping framework was developed, validated, and used to characterise the diversity in patterns of thermal responses existing across Z. tritici populations that were sampled over contrasted scales (spatial and seasonal variation of temperature). Second, the spatio-temporal thermal variations encountered in a wheat canopy, considered as a habitat exerting fluctuating selective pressures on these differential thermal sensitivities of individuals, were investigated in depth. Third, the way selection of “thermotypes” (functional groups of individuals displaying a similar thermal sensitivity) occurs and drives dynamics of Z. tritici populations was examined. To this end, both empirical (in vitro, in planta and in natura) and theoretical (in silico) competition experiments were conducted under increasingly complex selective environments. This research work demonstrates that glossing over the natural extent of individual phenotypic diversity in a phyllosphere microbial population and over the heterogeneity of selective pressures – from phyllo- to mesoclimate – leads to underestimate the resilience of this population, and thus its adaptive potential to environmental variations. In doing so, the results of this thesis, at the interface between epidemiology, micrometeorology, and ecology, improve our understanding of how important is individual variation to population dynamics and how environmental heterogeneity allows to maintain population diversity. Finally, this thesis provides insight into how large-scale patterns and local population processes are interlinked and display a “two-tier” adaptive dynamics
Guillemet, Martin. « The dynamics of viral adaptation : theoretical and experimental approaches ». Electronic Thesis or Diss., Université de Montpellier (2022-....), 2023. http://www.theses.fr/2023UMONG020.
Texte intégralMost living organisms on the tree of life can be infected by viruses. The ubiquity of viruses is driven by different factors including high mutation rates, high population sizes and low generation times, which allow for quick adaptation to very different host species. The dynamics of adaptation - the rate of change of the mean fitness of the viral population - results from the interplay between multiple evolutionary forces that may promote or hamper viral adaptation. But the interactions between these different factors may often be difficult to understand. During this PhD we developed a combination of theoretical and experimental approaches to disentangle the influence of some of these factors on viral adaptation.First, we explored the dynamics of viral adaptation to a homogeneous host population. We used Fisher’s Geometric Model of adaptation and studied the joint evolutionary and epidemiological dynamics of a viral population spreading in a host population. This modeled allowed us to explore the lethal mutagenesis hypothesis: is it possible to treat viral infections with mutagenic drugs to increase the mutation load of the viral population beyond a threshold that may result in the extinction of the within-host population? We show which parameters affect the critical mutation rate leading to viral extinction and we show how epidemiology and evolution can affect the transient within-host dynamics of the viral population when a single virus life-history trait (transmission rate) is under selection. We extend this modeling framework to study the joint evolution of transmission and virulence during the adaptation of an emerging pathogen. At the beginning of an epidemic, these two traits are expected to evolve independently but a trade-off may build up with viral adaptation.Second, we studied viral adaptation in heterogeneous host populations when the virus spreads among a diversified population of resistance host. We studied the evolutionary emergence of viruses: can viruses avoid extinction by the acquisition of escape mutations allowing them to infect some of the resistant hosts in the population? We developed a simple birth-death process to predict the probability of evolutionary emergence as a function of the composition of the host population. In particular, we show how the proportion of multiple resistant hosts can reduce the risk of pathogen evolutionary emergence. We put some of these predictions to the test using bacteriophages spreading in bacterial populations. We manipulate the diversity of CRISPR immunity in Streptococcus thermophilus bacteria and we confirm the key influence of multiple resistance on the risk of viral adaptation.Third, we also studied viral adaptation in time-varying environments where the host population is allowed to coevolve with the virus. In this experimental project we monitored the adaptation of bacteriophages as they coevolved with the CRISPR immunity of S. thermophilus bacteria. We track reciprocal adaptive changes in which bacteria acquire new layers of resistance (new spacers in the CRISPR array) and phages acquire new escape mutations in the corresponding protospacers. This experiment allows us to monitor the dynamics of viral adaptation across time and space. Interestingly, we find a significant asymmetries in competitive abilities among different bacterial strain in the absence of phage predation. This asymmetric competition has dramatic consequences on the maintenance of diversity of host resistance and on the coevolutionary dynamics with the virus. This thesis demonstrates the possibility to use experimental evolution with microbial microcosms to explore the validity of some theoretical predictions on the dynamics of viral adaptation. This experimental validation is particularly important if one wants to use evolutionary models to make public-health recommendations
Kastora, Stavroula. « Novel regulators that control the adaptation of a major fungal pathogen to combinations of host signals ». Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=228983.
Texte intégralMelnyk, Anita. « The Evolution of Antibiotic Resistance in Experimental Populations of Bacteria ». Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/34556.
Texte intégralMcDougald, S. Diane School of Microbiology & Immunology UNSW. « Regulation of starvation and nonculturability in the marine pathogen, Vibrio vulnificus ». Awarded by:University of New South Wales. School of Microbiology and Immunology, 2000. http://handle.unsw.edu.au/1959.4/19118.
Texte intégralMaikova, Anna. « The CRISPR-Cas system of human pathogen Clostridium difficile : function and regulation ». Thesis, Université de Paris (2019-....), 2019. http://www.theses.fr/2019UNIP7091.
Texte intégralClostridium difficile (the novel name – Clostridioides difficile) is a Gram-positive, strictly anaerobic spore forming bacterium, found in soil and aquatic environments as well as in mammalian intestinal tracts. C. difficile is one of the major pathogenic clostridia. This bacterium has become a key public health issue associated with antibiotic therapy in industrialized countries. C. difficile-associated diarrhoea is currently the most frequently occurring nosocomial diarrhoea in Europe and worldwide. Since the last decade the number of severe infection forms has been rising due to emergence of the hypervirulent and epidemic strains as ribotype 027 R20291 strain. C. difficile infection causes diarrhoea, colitis and even death. Many aspects of C. difficile pathogenesis remain poorly understood. Particularly, the molecular mechanisms of its adaptation to changing conditions inside the host are to be scrutinized. During the infection cycle C. difficile survives in bacteriophage-rich gut communities possibly by relying on some special systems that control the genetic exchanges favored within these complex environments. During the last decade, CRISPR (clustered regularly interspaced short palindromic repeats)-Cas (CRISPR-associated) systems of adaptive prokaryotic immunity against exogenic genetic elements has become the center of interest among various anti-invader bacterial defense systems.Previous studies revealed the presence of abundant and diverse CRISPR RNAs in C. difficile. C. difficile has an original CRISPR system, which is characterized by the presence of an unusually large set of CRISPR arrays (12 arrays in the laboratory 630 strain and 9 ones in the hypervirulent R20291 strain), of two or three sets of cas genes conserved in the majority of sequenced C. difficile genomes and the prophage location of several CRISPR arrays. However, the role CRISPR-Cas plays in the physiology and infectious cycle of this important pathogen remains obscure.The general aims of this work run as follows: 1) to investigate the role and the functionality of C. difficile CRISPR-Cas system in the interactions with foreign DNA elements (such as plasmids), 2) to reveal the way C. difficile CRISPR-Cas system expression is regulated and functions in different states of bacterial culture, including its response to stresses. In the present PhD thesis the functionality of C. difficile CRISPR-Cas system was investigated (Chapter 2). Through conjugation efficiency assays defensive function (in interference) of C. difficile CRISPR-Cas system was demonstrated. The correlation between the previously known levels of expression of CRISPR RNAs and the observed levels of interference has also been shown. Moreover, through the series of interference experiments the functionality of PAMs (protospacer adjacent motifs) was confirmed, which have already been predicted in silico. Additionally, the general functional PAM consensus was determined using PAM libraries experiments. Furthermore, an adaptive function of C. difficile CRISPR-Cas system was shown for laboratory strain. The role of multiple cas operons in C. difficile CRISPR functionality is also demonstrated in this Chapter.In Chapter 3 the link between C. difficile CRISPR-Cas system and a new type I toxin-antitoxin system is demonstrated, as well as a possible co-regulation under biofilm and stress conditions of CRISPR-Cas system and these toxin-antitoxin modules. This Chapter also defines a possible role of c-di-GMP in regulation of C. difficile CRISPR-Cas system. Additionally, Chapter 4 describes the utilization of endogenous C. difficile CRISPR-Cas system as a novel tool for genome editing in C. difficile. Altogether, the obtained data highlight the original features of active C. difficile CRISPR-Cas system and demonstrate its biotechnological potential
Thézé, Julien. « Diversification et adaptation génomique des virus entomopathogènes ». Thesis, Tours, 2013. http://www.theses.fr/2013TOUR4006.
Texte intégralAt different timescales, the purpose of my PhD was to understand insect virus evolution through the study of the genomic diversification and adaptation of insect large DNA viruses. Firstly, I was able to estimate the ages of baculovirus and nudivirus diversifications, and to propose a long-term coevolutionary scenario between these viruses and their insect hosts. Then, on a narrower timescale, I showed that insect hosts are the major factor in baculovirus diversification, and surprisingly, I also observed that the virus biotic environment, i.e. insect host plants, plays a central role in their evolution. Secondly, punctual mutations have been linked to the local adaptation of differentiated populations of the baculovirus SeMNPV. Finally, the study of convergent genomic adaptation between entomopoxviruses and baculoviruses highlighted that horizontal gene transfers are an important source of variability for large DNA viruses, for the adaption to the same ecological niches. Genes and mechanisms identified in this PhD work provide new insights to understand how genomes are shaped by ecology
Pawlik, Marie-Christin [Verfasser], et Ulrich [Akademischer Betreuer] Vogel. « Gene expression in the human pathogen Neisseria meningitidis : Adaptation to serum exposure and zinc limitation / Marie-Christin Pawlik. Betreuer : Ulrich Vogel ». Würzburg : Universitätsbibliothek der Universität Würzburg, 2013. http://d-nb.info/1036836509/34.
Texte intégralLivres sur le sujet "Pathogen adaptation"
Hsu, Ellen, et Louis Du Pasquier, dir. Pathogen-Host Interactions : Antigenic Variation v. Somatic Adaptations. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20819-0.
Texte intégral1966-, Dieckmann Ulf, dir. Adaptive speciation. Cambridge, UK : Cambridge University Press, 2004.
Trouver le texte intégralLe May, Christophe, Josselin Montarry, Cindy E. Morris, Omer Frenkel et Virginie Ravigné, dir. Plant Pathogen Life-History Traits and Adaptation to Environmental Constraints. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-530-6.
Texte intégralGriffiths, Mansel. Understanding Pathogen Behaviour : Virulence, Stress Response, and Resistance (Woodhead Publishing in Food Science and Technology). CRC Press, 2005.
Trouver le texte intégralPasquier, Louis Du, et Ellen Hsu. Pathogen-Host Interactions : Antigenic Variation v. Somatic Adaptations. Springer, 2015.
Trouver le texte intégralPasquier, Louis Du, et Ellen Hsu. Pathogen-Host Interactions : Antigenic Variation V. Somatic Adaptations. Springer London, Limited, 2015.
Trouver le texte intégralPasquier, Louis Du, et Ellen Hsu. Pathogen-Host Interactions : Antigenic Variation v. Somatic Adaptations. Springer, 2016.
Trouver le texte intégralSimões, Isaura, Daniel E. Voth et Luís Jaime Mota, dir. Obligate Intracellular Bacteria : Evasion and Adaptative Tactics Shaping the Host-Pathogen Interface. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-753-3.
Texte intégralBrüne, Martin, et Wulf Schiefenhövel, dir. Oxford Handbook of Evolutionary Medicine. Oxford University Press, 2019. http://dx.doi.org/10.1093/oxfordhb/9780198789666.001.0001.
Texte intégralAhmed, Hafiz Uddin. Pathogenic variability and adaptation of Septoria tritici to different wheat cultivars. 1993.
Trouver le texte intégralChapitres de livres sur le sujet "Pathogen adaptation"
Heroven, Ann Kathrin, et Petra Dersch. « Metabolic Adaptation of Human PathogenicYersiniae ». Dans Host - Pathogen Interaction, 1–18. Weinheim, Germany : Wiley-VCH Verlag GmbH & Co. KGaA, 2016. http://dx.doi.org/10.1002/9783527682386.ch1.
Texte intégralJagathjothi, N., M. Deivamani, M. Yuvaraj, R. Sathya Priya, M. Saranya, R. Sharmila, K. S. Subramanian et al. « Plant Pathogen Mitigation and Adaptation to Climate Change ». Dans Plant Quarantine Challenges under Climate Change Anxiety, 53–78. Cham : Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-56011-8_3.
Texte intégralde la Fuente, José, et Margarita Villar. « Conflict and cooperation in tick-host-pathogen interactions contribute to increased tick fitness and survival. » Dans Climate, ticks and disease, 232–39. Wallingford : CABI, 2021. http://dx.doi.org/10.1079/9781789249637.0033.
Texte intégralKushwaha, Chanda, Neha Rani et Arun P. Bhagat. « Chapter 13. Nature, Dissemination and Epidemiological Consequences in Charcoal Rot Pathogen Macrophomina Phaseolina ». Dans The Phytopathogen Evolution and Adaptation, 357–80. 9 Spinnaker Way, Waretown, NJ 08758 USA : Apple Academic Press Inc., 2017. http://dx.doi.org/10.1201/9781315366135-17.
Texte intégralBakshi, Suman, Johar Singh et Sanjay J. Jambhulkar. « Isolation and characterization of yellow rust resistant mutants in wheat. » Dans Mutation breeding, genetic diversity and crop adaptation to climate change, 103–10. Wallingford : CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0010.
Texte intégralBurdon, J. J. « Genetic Variation in Pathogen Populations and its Implications for Adaptation to Host Resistance ». Dans Durability of Disease Resistance, 41–56. Dordrecht : Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-2004-3_4.
Texte intégralErnst, Florian D., Arnoud H. M. van Vliet, Manfred Kist, Johannes G. Kusters et Stefan Bereswill. « The Role of Nickel in Environmental Adaptation of the Gastric Pathogen Helicobacter pylori ». Dans Nickel and Its Surprising Impact in Nature, 545–79. Chichester, UK : John Wiley & Sons, Ltd, 2007. http://dx.doi.org/10.1002/9780470028131.ch15.
Texte intégralPförtner, Henrike, Maren Depke, Kristin Surmann, Frank Schmidt et Uwe Völker. « In vivo Proteomics Approaches for the Analysis of Bacterial Adaptation Reactions in Host–Pathogen Settings ». Dans Methods in Molecular Biology, 207–28. New York, NY : Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8695-8_15.
Texte intégralChalla, Surekha, et Nageswara Rao Reddy Neelapu. « Association Between Horizontal Gene Transfer and Adaptation of Gastric Human Pathogen Helicobacter pylori to the Host ». Dans Horizontal Gene Transfer, 257–67. Cham : Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-21862-1_10.
Texte intégralEpping, Lennard, Esther-Maria Antão et Torsten Semmler. « Population Biology and Comparative Genomics of Campylobacter Species ». Dans Current Topics in Microbiology and Immunology, 59–78. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65481-8_3.
Texte intégralActes de conférences sur le sujet "Pathogen adaptation"
Hulst, A. D., P. Bijma et M. C. M. De Jong. « 160. Can we prevent pathogen adaptation when breeding disease resistant livestock ? » Dans World Congress on Genetics Applied to Livestock Production. The Netherlands : Wageningen Academic Publishers, 2022. http://dx.doi.org/10.3920/978-90-8686-940-4_160.
Texte intégralYin, Chuntao. « Disease-induced changes in the rhizosphere microbiome reduced root disease ». Dans IS-MPMI Congress. IS-MPMI, 2023. http://dx.doi.org/10.1094/ismpmi-2023-5r.
Texte intégralBruun Jensen, Annette. « Fungal pathogen adaptations in social insects — Pandora’s Box ». Dans 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.92339.
Texte intégralAllen, Aideen C., Wladimir Malaga, Catherine Astarie-Dequeker, Ali Hassan, Céline Berrone, Flavie Moreau, Philip Supply, Roland Brosch et Christophe Guilhot. « From environmental bacteria to obligate pathogen : the study of adaptations enhancing the persistence of tuberculosis bacilli ». Dans ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.2802.
Texte intégralAllen, Aideen, Wladimir Malaga, Catherine Astarie-Dequeker, Ali Hassan, Céline Berrone, Flavie Moreau, Philip Supply, Roland Brosch et Christophe Guilhot. « From environmental bacteria to obligate pathogen : the study of adaptations enhancing the persistence of tuberculosis bacilli ». Dans ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4598.
Texte intégralMARSHALL, DAVID G., CHARLES J. DORMAN, FRANCES BOWE, CHRISTINE HALE et GORDON DOUGAN. « DNA TOPOLOGY AND ADAPTATION OF SALMONELLA TYPHIMURIUM TO AN INTRACELLULAR ENVIRONMENT ». Dans The Activities of Bacterial Pathogens in Vivo - Based on Contributions to a Royal Society Discussion Meeting. IMPERIAL COLLEGE PRESS, 2001. http://dx.doi.org/10.1142/9781848161610_0002.
Texte intégralRapports d'organisations sur le sujet "Pathogen adaptation"
Melotto, M., et S. Sela. NIFA-BARD collaborative, mechanisms of salmonella adaptation to the lettuce phyllosphere. Israel : United States-Israel Binational Agricultural Research and Development Fund, 2022. http://dx.doi.org/10.32747/2022.8134153.bard.
Texte intégralEldar, Avigdor, et Donald L. Evans. Streptococcus iniae Infections in Trout and Tilapia : Host-Pathogen Interactions, the Immune Response Toward the Pathogen and Vaccine Formulation. United States Department of Agriculture, décembre 2000. http://dx.doi.org/10.32747/2000.7575286.bard.
Texte intégralSela, Shlomo, et Michael McClelland. Desiccation Tolerance in Salmonella and its Implications. United States Department of Agriculture, mai 2013. http://dx.doi.org/10.32747/2013.7594389.bard.
Texte intégralFreeman, Stanley, Russell Rodriguez, Adel Al-Abed, Roni Cohen, David Ezra et Regina Redman. Use of fungal endophytes to increase cucurbit plant performance by conferring abiotic and biotic stress tolerance. United States Department of Agriculture, janvier 2014. http://dx.doi.org/10.32747/2014.7613893.bard.
Texte intégralShpigel, Nahum Y., Ynte Schukken et Ilan Rosenshine. Identification of genes involved in virulence of Escherichia coli mastitis by signature tagged mutagenesis. United States Department of Agriculture, janvier 2014. http://dx.doi.org/10.32747/2014.7699853.bard.
Texte intégralSplitter, Gary A., Menachem Banai et Jerome S. Harms. Brucella second messenger coordinates stages of infection. United States Department of Agriculture, janvier 2011. http://dx.doi.org/10.32747/2011.7699864.bard.
Texte intégralSela, Hanan, Eduard Akhunov et Brian J. Steffenson. Population genomics, linkage disequilibrium and association mapping of stripe rust resistance genes in wild emmer wheat, Triticum turgidum ssp. dicoccoides. United States Department of Agriculture, janvier 2014. http://dx.doi.org/10.32747/2014.7598170.bard.
Texte intégralPorat, Ron, Gregory T. McCollum, Amnon Lers et Charles L. Guy. Identification and characterization of genes involved in the acquisition of chilling tolerance in citrus fruit. United States Department of Agriculture, décembre 2007. http://dx.doi.org/10.32747/2007.7587727.bard.
Texte intégralChiel, Elad, et Christopher J. Geden. Development of sustainable fly management tools in an era of global warming. United States Department of Agriculture, janvier 2014. http://dx.doi.org/10.32747/2014.7598161.bard.
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