Se connecter

Bibliographies thématiques / Osteoclasts / Livres

Livres sur le sujet « Osteoclasts »

Pour voir les autres types de publications sur ce sujet consultez le lien suivant : Osteoclasts.

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 6 juillet 2024

Créez une référence correcte selon les styles APA, MLA, Chicago, Harvard et plusieurs autres

Choisissez une source :

Consultez les 50 meilleurs livres pour votre recherche sur le sujet « Osteoclasts ».

À côté de chaque source dans la liste de références il y a un bouton « Ajouter à la bibliographie ». Cliquez sur ce bouton, et nous générerons automatiquement la référence bibliographique pour la source choisie selon votre style de citation préféré : APA, MLA, Harvard, Vancouver, Chicago, etc.

Vous pouvez aussi télécharger le texte intégral de la publication scolaire au format pdf et consulter son résumé en ligne lorsque ces informations sont inclues dans les métadonnées.

Parcourez les livres sur diverses disciplines et organisez correctement votre bibliographie.

1

R, Rifkin Barry, et Gay Carol V, dir. Biology and physiology of the osteoclast. Boca Raton : CRC Press, 1992.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

2

Yu, Hesheng. P2 purinoceptor-linked Ca2+ signaling and pH changes in osteoclasts. [Toronto : University of Toronto, Faculty of Dentistry], 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

3

Felix, Bronner, Farach-Carson Mary C. 1958- et Rubin Janet, dir. Bone resorption. London : Springer, 2005.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

4

Rodionova, N. V. Funkt͡s︡ionalʹnai͡a︡ morfologii͡a︡ kletok v osteogeneze. Kiev : Nauk. dumka, 1989.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

5

National Institute on Aging/National Institute of Dental Research Workshop on Human Models of Skeletal Aging (1994 Washington, D.C.). National Institute on Aging/National Institute of Dental Research Workshop on Human Models of Skeletal Aging : Washington, DC, March 1-2, 1994. Sous la direction de Robey Pamela Gehron 1952-, Sherman Sherry, National Institute of Dental Research (U.S.) et National Institute on Aging. New York, NY : Springer International, 1995.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

6

Maria, Bijvoet Olav Leonardus, Lipton Allan et International Cancer Congress (15th : 1990 : Hamburg, Germany), dir. Osteoclast inhibition in the management of malignancy-related bone disorders : An international symposium held during the 15th International Cancer Congress, Hamburg, Germany, August 1990. Seattle : Hogrefe & Huber, 1993.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

7

D, Rubens R., et European Conference on Clinical Oncology (5th : 1989 : London, England), dir. The Management of bone metastases and hypercalcaemia by osteoclast inhibition : An international symposium held during the 5th European Conference on Clinical Oncology (ECCO 5), London, September 1989. Toronto : Hogrefe & Huber, 1990.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

8

Shorey, Seema. Differences in the degree to which osteoclasts from different parts of the skeleton employ cathepsin K and matrix metalloproteinases for bone resorption. Ottawa : National Library of Canada, 2002.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

9

Holt, Ian. Control of osteoclast activity. Manchester : University of Manchester, 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

10

David, Evered, Harnett Sara et Ciba Foundation, dir. Cell and molecular biology of vertebrate hard tissues. Chichester, UK : Wiley, 1988.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

11

H, Helfrich Miep, et Ralston Stuart, dir. Bone research protocols. Totowa, N.J : Humana Press, 2003.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

12

H, Helfrich Miep, et Ralston Stuart, dir. Bone research protocols. Totowa, N.J : Humana Press, 2003.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

13

Tai, Victoria. The effects of leukotriene Bb4s on osteoclast formation and osteoclastic bone resorption and the role of osteoblastic cells in these processes. Ottawa : National Library of Canada = Bibliothèque nationale du Canada, 1999.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

14

Lorenzo, Joseph. Osteoimmunology : Interactions of the immune and skeletal systems. Amsterdam : Academic, 2011.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

15

R, Arnett Timothy, et Henderson Brian, dir. Methods in bone biology. London : Chapman & Hall, 1998.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

16

Green, Philip Macey. An experimental study of the osteoclast and its role in bone remodelling. Birmingham : University of Birmingham, 1988.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

17

Sutton, Michael Mark. The Influence of Microtubules and Microtubule-Based Structures on Osteoclast and CD4+ T Cell Function. [New York, N.Y.?] : [publisher not identified], 2022.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

18

Graziano, Victor M. A. The ferret : A potential in vitro small animal model for the study of osteogenesis and osteoclasis. [Toronto : University of Toronto, Faculty of Dentistry], 1998.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

19

Poore, Charles T. Osteotomy and Osteoclasis for Deformities of the Lower Extremities. Creative Media Partners, LLC, 2018.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

20

Gabet, Yankel, Drorit Neumann, Ari Elson, Noam Levaot et Natalie A. Sims. Developmental Biology and Regulation of Osteoclasts. Frontier Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-867-2.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

21

Brown, Alexander J. Osteoclasts : Morphology, Functions and Clinical Implications. Nova Science Publishers, Incorporated, 2012.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

22

Reeves, Cecelia. Osteoclasts : Cell Biology, Functions and Related Diseases. Nova Science Publishers, Incorporated, 2015.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

23

Yu, Hesheng. P2 purinoceptor-linked Ca2+ signaling and pH changes in osteoclasts. 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

24

Bronner, Felix, Janet Rubin et Mary C. Farach-Carson. Bone Resorption. Springer London, Limited, 2006.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

25

Bronner, Felix, Janet Rubin et Mary C. Farach-Carson. Bone Resorption. Springer London, Limited, 2010.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

26

Anderson, Gail I. Osteoblast involvement in the formation and activation of osteoclasts in vitro. 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

27

Chandra, Divya. To evaluate differences in mRNA expression of c-fms, EP2 and EP4 between large and small osteoclasts. 2001.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

28

Osteoblasts : Morphology, Functions and Clinical Implications. Nova Science Pub Inc, 2013.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

29

Ruabens, Robert D. The Management of Bone Metastases and Hypercalcaemia by Osteoclast Inhibition : An International Symposium Held During the 5th European Conference on. Hogrefe & Huber Pub, 1990.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

30

Zaidi, Mone. Osteoclast-Osteoblast. Jai Pr, 1999.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

31

Clarke, Noel W. Metastatic disease in prostate cancer. Sous la direction de James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0068.

Texte intégral

Résumé :

Metastases are the predominant cause of morbidity and death from prostate cancer (CaP). The tendency for cells to migrate from the primary site, enter the vascular/lymphatic circulation, and implant/grow at secondary sites is the principal discriminator of aggressive form indolent disease. But this process is poorly understood. Cells enter the circulation in increasing number as the disease progresses, impinging on endothelial surfaces, particularly in red bone marrow where they bind and transmigrate, forming early cell colonies. This requires chemo-attractants and nutrients enabling cellular survival. Established metastases thrive independently, disrupting local tissue, as characterized by progressive replacement of red bone marrow and disruption of skeletal architecture. Bone disruption includes massive overstimulation of both osteoblasts and osteoclasts, inducing synchronous over-production of abnormal bone and gross osteolysis.

Styles APA, Harvard, Vancouver, ISO, etc.

32

Lories, Rik J., et Georg Schett. Pathology : bone. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0010.

Texte intégral

Résumé :

Axial spondyloarthritis is associated with different types of skeletal damage. Inflammation at the affected sites is linked with both loss of trabecular bone and new bone formation on the cortical side, potentially leading to joint or spine ankylosis. Both aspects of the disease can result in a significant burden for the patient. Bone loss is directly linked to proinflammatory cytokines and activation of osteoclasts. Control of inflammation is therefore the best strategy to prevent loss of bone. The nature of the new bone formation process is less defined. A prominent role for developmental signalling pathways has been proposed. Current therapies have limited or no impact on this process. However, emerging data suggest that early control of disease activity may be part of a window of opportunity to prevent structural damage, as biomechanical factors and instability following inflammation may also play a role.

Styles APA, Harvard, Vancouver, ISO, etc.

33

Hall, Brian K. Bone, Volume II : The Osteoclast. CRC, 1991.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

34

Hall, Brian K. Bone, Volume II : The Osteoclast. Taylor & Francis Group, 1991.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

35

Bone Research Protocols (Methods in Molecular Medicine). Humana Press, 2003.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

36

Helfrich, Miep H., et Stuart H. Ralston. Bone Research Protocols. Humana Press, 2016.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

37

Helfrich, Miep H., et Stuart H. Ralston. Bone Research Protocols. Humana Press, 2010.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

38

Choi, Yongwon, Mark Horowitz, Joseph Lorenzo, Hiroshi Takayanagi et Georg Schett. Osteoimmunology : Interactions of the Immune and Skeletal Systems. Elsevier Science & Technology Books, 2015.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

39

Choi, Yongwon, Mark Horowitz, Joseph Lorenzo, Hiroshi Takayanagi et Georg Schett. Osteoimmunology : Interactions of the Immune and Skeletal Systems. Elsevier Science & Technology Books, 2015.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

40

Choi, Yongwon, Mark Horowitz, Joseph Lorenzo et Hiroshi Takayanagi. Osteoimmunology : Interactions of the Immune and Skeletal Systems. Elsevier Science & Technology Books, 2010.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

41

Folkins, Alia. Coloring Book - You Will Get Better - Osteoclast. Independently Published, 2021.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

42

Folkins, Alia. Coloring Book - You Will Get Better - Osteoclast. Independently Published, 2021.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

43

Poore, Charles T. Osteotomy and Osteoclasis for Deformities of the Lower Extremities. Franklin Classics Trade Press, 2018.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

44

Henderson, Brian, et T. Arnett. Methods in Bone Biology. Springer, 2013.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

45

Osteoimmunology Interactions Of The Immune And Skeletal Systems Ii. Springer, 2009.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

46

Lees, Rita L. Osteoclast heterogeneity : The importance of cell size and phase of activity. 2000.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

47

Sprague, Stuart M., et James M. Pullman. Spectrum of bone pathologies in chronic kidney disease. Sous la direction de David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0122.

Texte intégral

Résumé :

Histologic bone abnormalities begin very early in the course of chronic kidney disease. The KDIGO guidelines recommend that bone disease in patients with chronic kidney disease should be diagnosed on the basis of bone biopsy examination, with bone histomorphometry. They have also proposed a new classification system (TMV), using three key features of bone histology—turnover, mineralization, and volume—to describe bone disease in these patients. However, bone biopsy is still rarely performed today, as it involves an invasive procedure and highly specialized laboratory techniques. High-turnover bone disease (osteitis fibrosa cystica) is mainly related to secondary hyperparathyroidism and is characterized by increased rates of both bone formation and resorption, with extensive osteoclast and osteoblast activity, and a progressive increase in peritrabecular marrow space fibrosis. On the other hand, low-turnover (adynamic) bone disease involves a decline in osteoblast and osteoclast activities, reduced new bone formation and mineralization, and endosteal fibrosis. The pathophysiological mechanisms of adynamic bone include vitamin D deficiency, hyperphosphataemia, metabolic acidosis, inflammation, low oestrogen and testosterone levels, bone resistance to parathyroid hormone, and high serum fibroblast growth factor 23. Mixed uraemic osteodystrophy describes a combination of osteitis fibrosa and mineralization defect. In the past few decades, an increase in the prevalence of mixed uraemic osteodystrophy and adynamic bone disease has been observed.

Styles APA, Harvard, Vancouver, ISO, etc.

48

Sheraly, Aly R. The use of gastric proton pump inhibitors to modulate osteoclast-mediated resorption of calcium phosphate cements. 2005.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

49

J. Singh, Parminder, et Rohit Kotnis. The musculoskeletal system : structure and function. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.0003.

Texte intégral

Résumé :

♦ Structure of bone is comprised of cells, matrix, and water♦ Bone consists broadly of three surfaces (periosteal, endosteal, and Haversian) and two membranes (periosteum and endosteum)♦ The blood supply of bone is derived from four main routes (nutrient, metaphyseal, epiphyseal, and periosteal arteries)♦ There are three main types of cells in bone (osteoblast, osteocyte, and osteoclast)♦ The matrix is a composite material consisting of an organic and an inorganic component♦ Two types of bone formation are intramembranous and endochondral ossification♦ The skeleton is also involved in the vital homeostasis of calcium and phosphate.

Styles APA, Harvard, Vancouver, ISO, etc.

50

Bijvoet, O. L. M. Osteoclast Inhibition in the Management of Malignancy-Related Bone Disorders : An International Symposium Held During the 15th International Cancer Co. Hogrefe & Huber Pub, 1992.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

Nous offrons des réductions sur tous les plans premium pour les auteurs dont les œuvres sont incluses dans des sélections littéraires thématiques. Contactez-nous pour obtenir un code promo unique!