Littérature scientifique sur le sujet « Neurological soft sign »
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Articles de revues sur le sujet "Neurological soft sign"
Dazzan, Paola, Tuhina Lloyd, Kevin D. Morgan, Jolanta Zanelli, Craig Morgan, Ken Orr, Gerard Hutchinson et al. « Neurological abnormalities and cognitive ability in first-episode psychosis ». British Journal of Psychiatry 193, no 3 (septembre 2008) : 197–202. http://dx.doi.org/10.1192/bjp.bp.107.045450.
Texte intégralThioritz, Wempy, Erlyn Limoa, J. C. Hutomo, Saidah Syamsuddin et Sonny T. Lisal. « Differentiation in Neurological Soft Sign Scores on Schizophrenic Patients with Antipsychotic Treatment ». Open Access Macedonian Journal of Medical Sciences 9, T3 (2 juillet 2021) : 249–53. http://dx.doi.org/10.3889/oamjms.2021.6356.
Texte intégralPhillips, P. H. « Intermittent exotropia increasing with near fixation : a "soft" sign of neurological disease ». British Journal of Ophthalmology 89, no 12 (1 décembre 2005) : 1120–22. http://dx.doi.org/10.1136/bjo.2004.063123.
Texte intégralPhillips, P. H., K. J. Fray et M. C. Brodsky. « Intermittent Exotropia Increasing with Near Fixation : A “Soft” Sign of Neurological Disease ». Journal of American Association for Pediatric Ophthalmology and Strabismus 10, no 2 (avril 2006) : 188. http://dx.doi.org/10.1016/j.jaapos.2006.03.006.
Texte intégralPicard, Hernàn, Anne Le Seac'h, Isabelle Amado, Raphael Gaillard, Marie-Odile Krebs et Cécile Beauvillain. « Impaired saccadic adaptation in schizophrenic patients with high neurological soft sign scores ». Psychiatry Research 199, no 1 (août 2012) : 12–18. http://dx.doi.org/10.1016/j.psychres.2012.04.039.
Texte intégralGARDNER, DAVID, PETER B. LUCAS et REX W. COWDRY. « Soft Sign Neurological Abnormalities in Borderline Personality Disorder and Normal Control Subjects ». Journal of Nervous and Mental Disease 175, no 3 (mars 1987) : 177–80. http://dx.doi.org/10.1097/00005053-198703000-00009.
Texte intégralKing, D. J., A. Wilson, S. J. Cooper et J. L. Waddington. « The Clinical Correlates of Neurological Soft Signs in Chronic Schizophrenia ». British Journal of Psychiatry 158, no 6 (juin 1991) : 770–75. http://dx.doi.org/10.1192/bjp.158.6.770.
Texte intégralAronowitz, Bonnie R., Concetta Decaria, Andrea Allen, Nicola Weiss, Audrey Saunders, Lisa Margolin, Serge Mosovich, Montebuchs Baum et Eric Hollander. « The Neuropsychiatry of Autism and Asperger's Disorder : Review of the Literature and Case Reports ». CNS Spectrums 2, no 5 (mai 1997) : 43–60. http://dx.doi.org/10.1017/s1092852900004892.
Texte intégralToro Espinoza, P., M. E. Ceballos, D. Valenzuela, M. F. Inostroza et J. Schröder. « Subtests of the heidelberg neurological soft sign scale that discriminate HIV patients with and without hand ». Journal of the Neurological Sciences 357 (octobre 2015) : e5. http://dx.doi.org/10.1016/j.jns.2015.08.096.
Texte intégralBajraktari, Mustafa, Majlinda Naco, Gentian Huti, Blerim Arapi et Rudin Domi. « Fat Embolism Syndrome Without Bone Fracture : Is It Possible ? » Open Access Macedonian Journal of Medical Sciences 10, no C (19 décembre 2022) : 331–35. http://dx.doi.org/10.3889/oamjms.2022.11169.
Texte intégralThèses sur le sujet "Neurological soft sign"
Chen, Eric Y. H. « Soft neurological signs in schizophrenia ». Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/24331.
Texte intégralOskamp, Andrea [Verfasser]. « Neurological Soft Signs in Stadien der Anorexia nervosa / Andrea Oskamp ». Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2013. http://d-nb.info/103563824X/34.
Texte intégralGoldhahn, Klaus [Verfasser]. « Neurological Soft Signs bei Patientinnen mit Anorexia nervosa / Klaus Goldhahn ». Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2009. http://d-nb.info/1023956985/34.
Texte intégralDazzan, Paola. « Neurological soft signs in first episode psychoses : their clinical and neuroanatomical correlates ». Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.430827.
Texte intégralGalindo, Guarin Liliana. « Neurological soft signs, temperament and schizotypy in patients with schizophrenia and unaffected relatives : an FMRI study ». Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/403815.
Texte intégralSchizophrenia is a severe psychiatric disorder that has a profound effect on both the individuals affected and society. This common mental illness is a complex, heterogeneous behavioural and cognitive syndrome that seems to originate from disruption of brain development caused by genetic or environmental factors, or both. The genetic basis may be present in individuals without disease, as in the case of relatives of patients, being detectable through biological markers. Neurological soft signs (NSS) are discrete sensorimotor impairments associated with deviant brain development that were postulate as an endophenotype of schizophrenic spectrum disorder. Also the personality traits have been proposed as a vulnerability marker in schizophrenia. A specific profile of temperament and character and the schizotypal personality traits have also been correlated with schizotypal personality traits. These traits and some neurological abnormalities have been shown to aggregate in the relatives of schizophrenia patients. The etiopathogenesis of schizophrenia suggests it may be a "progressive neurodevelopmental disorder". This view postulates a disruption in functional circuits involving hetero modal association areas rather than a specific abnormality in a single brain region. The aim of this study is to explore the abnormalities in the functional connectivity of the default mode network related to the association between neurological soft signs and personality in schizophrenia. To investigate this a cross-sectional study is proposed, comparing a group of patients with schizophrenia, a group of unaffected relatives and a group of healthy controls. In order to explore the association of these potential biomarkers of schizophrenia the study was composed of two parts: a) To explore the association between neurological soft signs and personality traits in schizophrenia, two personality examinations (Temperament and Character Inventory and the Schizotypal Personality Questionnaire) and an evaluation of Neurological Soft Signs were performed. b) To explore the association between cerebral connectivity changes in the default mode network with the presence of neurological soft signs in schizophrenia a functional magnetic resonance scan was performed on participants in a resting state. The major finding in this study was that patients with schizophrenia and non-psychotic relatives display a unique profile of temperament and character and more schizotypal traits that correlate with higher presence of NSS. Our results reveal an association between these hypothesized vulnerability markers, as temperament (especially harm avoidance, reward dependence and persistence) and character (especially self-directedness and cooperativeness) correlated with the presence of NSS in the entire sample. Also the schizotypal traits (total scores and subscores) showed a very strong correlation with the presence of NSS in the entire sample. The results showed that susceptibility to NSS and to schizophrenia are both related to individual differences in personality features in non-psychotic relatives of patients with schizophrenia. These findings highlight the value of using both assessments to study high risk populations. The neuroimaging results showed connectivity changes in the default mode network with a possible association with the presence of neurological soft signs. These findings support the theory of cognitive dysmetria as a possible dysfunction in cortical-thalamic-cerebellar connectivity. This model also could explain the diversity of symptoms in schizophrenia and their associations (like this study that includes personality and sensory and motor functions). One strength of the study is that the relatives of patients with schizophrenia had no familial ties to the patients used, thus decreasing the possibility that similar upbringing would confound the results.
Negash, Alemayehu. « Bipolar disorder in rural Ethiopia : community-based studies in Butajira for screening, epidemiology, follow-up, and the burden of care ». Doctoral thesis, Umeå universitet, Psykiatri, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-21743.
Texte intégralRothman, David J. « An Investigation of Neurological soft signs as a discriminating factor between Veterans with Post-traumatic Stress Disorder, mild Traumatic Brain Injury, and co-occurring Post-traumatic Stress Disorder and mild Traumatic Brain Injury ». VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5915.
Texte intégralGay, Olivier. « Marqueurs neurodéveloppementaux en psychiatrie : intérêt dans les troubles schizophréniques ». Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB016/document.
Texte intégralThe term neurodevelopment in its broadest sense refers to all of the processes encompassing development of the nervous system from the earliest stages of formation in utero to later stages of maturation during adolescence to produce the fully functional adult nervous system. Work over the last thirty years has led to a neurodevelopmental model of human psychiatric disorders, including schizophrenia, based on genetic, epidemiological and imaging evidence. This model asserts that disease is fundamentally linked to or develops from abnormality(s) in the formation processes (early neurodevelopment) and maturation (late neurodevelopment) of the nervous system due to a combination of genetic and environmental factors. In this context this thesis aims to clarify the effects of neurodevelopmental abnormalities on psychiatric disorders, including schizophrenia, through the study of different markers. The first study aims to investigate correlations between markers of early brain development: a clinical marker (neurological soft signs) and an imaging marker (sulcation of the cerebral cortex) in a population of subjects with schizophrenia. A correlation between these two markers is presented: the sulcation index was found to be lower in subjects that had significant neurological soft signs. We concluded that the combined study of different markers may help to isolate subgroups of patients with greater early neurodevelopmental damage. The second study aims to characterize effects of different markers of early neurodevelopmental abnormalities on cognitive functioning in patients with schizophrenia. Effects on executive control (as measured by the Trail Making Test) were correlated with clinical markers (neurological soft signs, handedness) and imaging (sulcation of the anterior cingulate cortex and enlargment of the ventricles). We found interactions between different markers with a mainly non-linear summation effect. Our interpretation is that different markers reflect separate insults, though all early, on brain development with a common final effect on executive function. The third study aims to clarify the specificity of sulcation as a marker of early neurodevelopmental abnormalities by studying a population of adult subjects with autism spectrum disorder (ASD), a patholody beginning in early childhood and linked with evidence of early neurodevelopmental damage. Sulcation abnormalities of the anterior cingulate cortex, similar to those observed in patients with schizophrenia are detected in patients with ASD. These results suggest early neurodevelopmental abnormalities are shared by different psychiatric disorders and that changes in cortical sulcation are not specific to a given disorder but the early damage. In conclusion, we suggest that the study of neurodevelopmental abnormalities should be integrated into a dimensional approach in psychiatry
Aguirre, Pacheco Cándida Isabel. « Los signos neurológicos menores en la esquizofrenia : correlatos con las características clínicas, la función cognitiva y los cambios cerebrales estructurales ». Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/665695.
Texte intégralNeurological soft signs (NSS), defined as minor neurological abnormalities that do not have localizing value, are established as being present at a higher frequency in patients with schizophrenia than in the healthy population. They are also present at a higher than normal frequency in the relatives of patients with schizophrenia, although at a lower rate than in the patients themselves, leading to considerable interest in their potential role as a marker of vulnerability or predisposition to the disorder. Nevertheless, some aspects of the association of NSS with the clinical features of schizophrenia remain unclear. Other relevant issues concern the relationship between NSS and the cognitive impairment and brain structural changes that also characterize the disorder. The general aim of this thesis was to examine the relationship between NSS and selected aspects of schizophrenia in a relatively large sample of patients. The first study examined a) their association with the symptoms of the disorder, as classified into positive symptoms (delusions and hallucinations), the disorganization syndrome (mainly formal thought disorder) and negative symptoms; and b) with the cognitive impairment seen in the disorder, specifically two major specific deficits, executive function and memory. The second study evaluated the relationship of NSS with brain structural abnormality, specifically changes in grey matter. NSS were assessed in both studies using a detailed scale, the Neurological Evaluation Scale (NES) of Buchanan and co-workers, which provides a total score and three subscale scores: sensory integration, motor coordination, and sequencing of complex motor acts. In the first study, the frequency of NSS was rated in a sample of 78 subjects with chronic schizophrenia. A comparison sample of 36 healthy control subjects was also employed. Clinical symptoms were rated using the Positive and Negative Symptom Scale (PANSS). The Behavioural Assessment of the Dysexecutive Syndrome (BADS) and the Rivermead Behavioral Memory Test (RBMT) were used to measure executive function and memory, respectively; current WAIS IQ was used as a measure of general intellectual function. As expected, the schizophrenic patients showed a higher frequency of NSS than the control subjects. With respect to symptoms, no relationship was found between NSS total or subscale scores and positive or negative symptoms; there was only equivocal evidence for a correlation with the disorganization syndrome. NSS scores correlated inversely and strongly with scores on all the cognitive measures. The correlations with memory and executive function scores remained significant after controlling for the correlation with general intellectual impairment. The second study was carried out on a sample of 83 patients with schizophrenia and a group of 60 healthy controls. Grey matter volume was measured using MRI, and the images were analyzed using whole-brain voxel-based morphometry. The patients with schizophrenia showed a pattern of widespread volume reduction in the cortex, and also in several subcortical structures. They also showed significantly increased grey matter volume in the brainstem and midbrain and in a small region of the left cerebellum. No clusters of significant correlation between NSS total or subscale scores were observed in the patients. The findings are discussed with particular reference to the failure to replicate previous findings of associations between NSS and negative symptoms, as well as the second study’s failure to find an association with structural cerebral changes in schizophrenia, something that has been reported in almost all previous MRI studies. The potential implications of the findings for two current theories of NSS in schizophrenia are also discussed: Andreasen’s hypothesis of cognitive dysmetria and the proposal that they represent a trait marker or endophenotype for the disorder.
Martinez, Gilles. « Continuum autisme-schizophrénie : apport de l’étude de la cognition sociale et de marqueurs phénotypiques développementaux ». Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB065/document.
Texte intégralAutism and schizophrenia are both neurodevelopmental psychiatric disorders. Research on early-onset schizophrenia, commonly associated to autism spectrum disorders (ASD), suggested a possible developmental continuum between both of these disorders. Clinical and epidemiological evidence, and research from molecular genetics or brain imaging, come to support this hypothesis. In this context, social cognition is a matter of special interest. Impairments are reported both in the two disorders, but with inconsistent results, revealing common features as well as differences. Otherwise, links between social cognition impairments and neurodevelopmental burden have been until now poorly explored. Through the contribution of our three studies, we confirmed the importance of social cognition impairment in autism and schizophrenia. The MASC test (Movie for the Assessment of Social Cognition), an original tool which was by our findings validated in a French version, revealed higher overall impairment of mentalizing capabilities in ASD than in schizophrenia. Animated Shapes (non verbal test of attribution of intentions) revealed qualitative differences: whereas hypomentalizing is common both to ASD and schizophrenia, overmentalizing seemed to be more important in schizophrenia. Furthermore, along a continuum between autism and schizophrenia, social cognition impairment was linked to thought and language disorganization, and to neurological soft signs (a marker for neurodevelopmental load). In addition, in subjects with schizophrenia, overmentalizing was correlated to the precocity of onset of the disease. Altogether, our results highlight the need to screen developmental feature in adulthood. In that way, we presented preliminary results in order to validate a developmental disorders screening self-rated questionnaire. As a conclusion, our results bring evidence in favour of a hypothesis of a continuum between autism and schizophrenia, showing a social cognition impairment in both disorders, correlated to the neurodevelopmental load existing in both of them in a transnosographic way. We contributed to emphasize the sub-group of subjects with schizophrenia with early-onset of disease, characterized by a tendency to overmentalizing and presenting a marked disorganization. Our work provides avenue to further studies, integrating neuroimaging and genetic data, that will help to advance in a deeper comprehension of the pathophysiology of autism and schizophrenia. Furthermore, we used and validated in this work promising tools to improve finely psychopathological evaluation and differential diagnosis in adults suffering from autism and from schizophrenia
Livres sur le sujet "Neurological soft sign"
E, Tupper David, dir. Soft neurological signs. Orlando : Grune & Stratton, 1987.
Trouver le texte intégralSoft neurological signs in clumsy children : Indicators of timing and force control dysfunction. 1990.
Trouver le texte intégralChapitres de livres sur le sujet "Neurological soft sign"
Ojagbemi, Akin. « Neurological Soft Signs ». Dans Encyclopedia of Personality and Individual Differences, 3200–3204. Cham : Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_782.
Texte intégralOjagbemi, Akin. « Neurological Soft Signs ». Dans Encyclopedia of Personality and Individual Differences, 1–5. Cham : Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28099-8_782-1.
Texte intégralShaffer, David, Cornelius S. Stokman, Patricia A. O’Connor, Stephen Shafer, Joseph E. Barmack, Suzanne Hess, D. Spalten et Irvin S. Schonfeld. « Early Soft Neurological Signs and Later Psychopathology ». Dans Life-Span Research on the Prediction of Psychopathology, 31–48. London : Routledge, 2021. http://dx.doi.org/10.4324/9781003165187-3.
Texte intégralNeeper, Ronald, et Robert S. Greenwood. « On the Psychiatric Importance of Neurological Soft Signs ». Dans Advances in Clinical Child Psychology, 217–58. Boston, MA : Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-9826-4_6.
Texte intégral« Neurological Soft Signs ». Dans Encyclopedia of Clinical Neuropsychology, 1754. New York, NY : Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_4874.
Texte intégralSchröder, Johannes, et Christina J. Herold. « Neurological Soft Signs in Schizophrenia Spectrum Disorders ». Dans Movement Disorders in Psychiatry, sous la direction de Antonio L. Teixeira, Erin Furr Stimming et William G. Ondo, 169—C10.P105. Oxford University PressNew York, 2022. http://dx.doi.org/10.1093/med/9780197574317.003.0010.
Texte intégralMehta, Gautam, et Bilal Iqbal. « Central Nervous System ». Dans Clinical Medicine for the MRCP PACES. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780199542550.003.0011.
Texte intégralSchröder, Johannes, et Christina J. Herold. « Neurological Soft Signs – A Transdiagnostic Phenomenon in Neuropsychiatric Conditions ». Dans Reference Module in Neuroscience and Biobehavioral Psychology. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-809324-5.24016-9.
Texte intégralSechi, Elia, et Dean M. Wingerchuk. « Rapidly Progressive Numbness and Weakness After Soft-Tissue Abscess ». Dans Mayo Clinic Cases in Neuroimmunology, sous la direction de Andrew McKeon, B. Mark Keegan et W. Oliver Tobin, 7–9. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197583425.003.0002.
Texte intégralActes de conférences sur le sujet "Neurological soft sign"
Silva, Bruno Custódio, Fernanda Silva dos Santos, Victória Porcher Simioni, Ana Luíza Kolling Konopka, Paulo Ricardo Gazzola Zen et Rafael Fabiano Machado Rosa. « Type 1 neurofibromatosis and its relation to the occurrence of cerebral vascular accident ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.077.
Texte intégralSchülke, Rasmus, Kyra Liepach, AnnaL Brömstrup, Thorsten Folsche, Maximilian Deest, Stefan Bleich, Alexandra Neyazi, Helge Frieling et Hannah Maier. « Neurological soft signs are increased in major depressive disorder irrespective of antidepressant treatment ». Dans Abstracts of the 3rd Symposium of the Arbeitsgemeinschaft für Neuropsychopharmakologie und Pharmakopsychiatrie (AGNP) and Deutsche Gesellschaft für Biologische Psychiatrie (DGBP). Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0042-1757665.
Texte intégralKaneko, Miki, Keiji Iramina, Takashi Ohya, Yushiro Yamashita, Yuichiro Kamei, Yoshinori Katayama et Sachio Takashima. « A measurement of soft neurological signs by pronosupination using wireless acceleration and angular velocity sensors ». Dans 2011 Biomedical Engineering International Conference (BMEiCON) - Conference postponed to 2012. IEEE, 2012. http://dx.doi.org/10.1109/bmeicon.2012.6172050.
Texte intégralRapports d'organisations sur le sujet "Neurological soft sign"
Pitman, Roger K. Can Post mTBI Neurological Soft Signs Predict Postconcussive and PTSD Symptoms : A Pilot Study. Fort Belvoir, VA : Defense Technical Information Center, juin 2013. http://dx.doi.org/10.21236/ada602492.
Texte intégralPitman, Roger K. Can Post mTBI Neurological Soft Signs Predict Postconcussive and PTSD Symptoms : A Pilot Study. Fort Belvoir, VA : Defense Technical Information Center, juin 2012. http://dx.doi.org/10.21236/ada602679.
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