Littérature scientifique sur le sujet « Neural mechanisms autism »
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Articles de revues sur le sujet "Neural mechanisms autism"
Watts, Timothy John. « The Pathogenesis of Autism ». Clinical medicine. Pathology 1 (janvier 2008) : CPath.S1143. http://dx.doi.org/10.4137/cpath.s1143.
Texte intégralMazefsky, Carla A., Amanda Collier, Josh Golt et Greg J. Siegle. « Neural features of sustained emotional information processing in autism spectrum disorder ». Autism 24, no 4 (28 février 2020) : 941–53. http://dx.doi.org/10.1177/1362361320903137.
Texte intégralBertone, Armando, Laurent Mottron, Patricia Jelenic et Jocelyn Faubert. « Motion Perception in Autism : A “Complex” Issue ». Journal of Cognitive Neuroscience 15, no 2 (1 février 2003) : 218–25. http://dx.doi.org/10.1162/089892903321208150.
Texte intégralRichey, J. Anthony, Cara R. Damiano, Antoinette Sabatino, Alison Rittenberg, Chris Petty, Josh Bizzell, James Voyvodic et al. « Neural Mechanisms of Emotion Regulation in Autism Spectrum Disorder ». Journal of Autism and Developmental Disorders 45, no 11 (25 janvier 2015) : 3409–23. http://dx.doi.org/10.1007/s10803-015-2359-z.
Texte intégralMosner, Maya G., R. Edward McLaurin, Jessica L. Kinard, Shabnam Hakimi, Jacob Parelman, Jasmine S. Shah, Joshua Bizzell et al. « Neural Mechanisms of Reward Prediction Error in Autism Spectrum Disorder ». Autism Research and Treatment 2019 (1 juillet 2019) : 1–10. http://dx.doi.org/10.1155/2019/5469191.
Texte intégralFailla, Michelle D., Estephan J. Moana-Filho, Greg K. Essick, Grace T. Baranek, Baxter P. Rogers et Carissa J. Cascio. « Initially intact neural responses to pain in autism are diminished during sustained pain ». Autism 22, no 6 (17 mai 2017) : 669–83. http://dx.doi.org/10.1177/1362361317696043.
Texte intégralSchmitz, Nicole, Katya Rubia, Therese van Amelsvoort, Eileen Daly, Anna Smith et Declan G. M. Murphy. « Neural correlates of reward in autism ». British Journal of Psychiatry 192, no 1 (janvier 2008) : 19–24. http://dx.doi.org/10.1192/bjp.bp.107.036921.
Texte intégralColizzi, Marco, Riccardo Bortoletto, Rosalia Costa, Sagnik Bhattacharyya et Matteo Balestrieri. « The Autism–Psychosis Continuum Conundrum : Exploring the Role of the Endocannabinoid System ». International Journal of Environmental Research and Public Health 19, no 9 (5 mai 2022) : 5616. http://dx.doi.org/10.3390/ijerph19095616.
Texte intégralGreene, Rachel K., Cara R. Damiano-Goodwin, Erin Walsh, Joshua Bizzell et Gabriel S. Dichter. « Neural Mechanisms of Vicarious Reward Processing in Adults with Autism Spectrum Disorder ». Autism Research and Treatment 2020 (21 mars 2020) : 1–12. http://dx.doi.org/10.1155/2020/8014248.
Texte intégralLeite, Júlio Fernandes, et Umberto Euzebio. « Anormalidades da formação cerebral e os transtornos de desenvolvimento neural ». STUDIES IN HEALTH SCIENCES 2, no 1 (17 septembre 2021) : 2–23. http://dx.doi.org/10.54018/shsv2n1-001.
Texte intégralThèses sur le sujet "Neural mechanisms autism"
Lefevre, Arthur. « Neural mechanisms of oxytocin and serotonin interaction in non-human primates and patients with autism ». Thesis, Lyon, 2016. http://www.theses.fr/2016LYSE1323/document.
Texte intégralThe neurohormone oxytocin (OT) is increasingly studied for its therapeutic potential in social disorders, like autism, which are associated with the deregulation of several neurotransmission systems, including OT and serotonin (5-HT). Hence investigating OT’s interactions with other neurotransmitters is a relevant step towards mechanism-based treatments. Studies in rodents demonstrated that the interaction between OT and 5-HT, is critical for several aspects of social behaviour. Moreover, using PET-scan in humans we have recently found that 5-HT 1A receptor (5-HT1AR) function is modified after intra-nasal oxytocin intake. Thus I performed a first experiment in which intra-nasal OT was administered to patients with autism undergoing a [18F]MPPF (a 5-HT1AR radiotracer) PET scanner, in order to study their basal serotonergic system and to look if the oxytocin modulates the 5-HT1AR system. I found no differences of baseline 5-HT1AR concentration between 18 autistic subjects and 24 controls. Critically, in patients, OT did not induce changes on the 5-HT1AR system. Moreover, in controls, there was a correlation between 5-HT1AR and grey matter volume in the striatum, that was not observed in patients. These results suggest a subtle disruption of patients’ serotonergic system, that can only be seen at the functional level. Because PET scan does not tell us if the observed modification is due to a change in 5-HT1AR or 5-HT concentration, I performed a second PET scan experiment on 3 macaque monkeys, using [18F]MPPF and [11C]DASB, that marks the serotonin transporter. Compared to placebo, OT injections in the lateral ventricle significantly reduced [11C]DASB binding potential in right amygdala, insula and hippocampus whereas [18F]MPPF binding potential increased in right amygdala and insula. Thus we reproduced results obtained in healthy humans and extended it by suggesting that OT provokes the release of 5-HT in key limbic regions involved in socio-emotional processing. These results were confirmed with autoradiography.Taken together, these experiments indicate that OT modulates 5-HT release in primates, but this mechanism is disrupted in patients with autism. This opens ways to investigate combined OT/5-HT treatments, especially since FDA approved drugs targeting the two systems are already available for use in patients with autism
Lombardo, Michael. « Cognitive and neural mechanisms underlying self-referential and social cognition in autism and the general population ». Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608982.
Texte intégralCoffman, Marika Cerie. « Common and Distinct Neural Mechanisms of Fear Acquisition and Reversal in comorbid Autism with Social Anxiety and Social Anxiety Disorder uncomplicated by Autism ». Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/102409.
Texte intégralDoctor of Philosophy
Peng, Yun, Zhongming Lu, Guohui Li, Mariel Piechowicz, Miranda Anderson, Yasin Uddin, Jie Wu et Shengfeng Qiu. « The autism associated MET receptor tyrosine kinase engages early neuronal growth mechanism and controls glutamatergic circuits development in the forebrain ». NATURE PUBLISHING GROUP, 2016. http://hdl.handle.net/10150/617181.
Texte intégralLiao, Chuan-Ching, et 廖專晶. « Different Neural Mechanisms of Semantic Processing Among Youths with Autism Spectrum Disorder, their Unaffected Siblings and Typically Developing Youths ». Thesis, 2019. http://ndltd.ncl.edu.tw/handle/6upj59.
Texte intégral國立臺灣大學
心理學研究所
107
Autism spectrum disorder (ASD) is characterized as a highly heritable disorder, and aberrant semantic processing is one of common symptoms in individuals with ASD. Unaffected ASD siblings usually have similar genetic backgrounds and early-life environments with individuals with ASD, with a higher risk of developing the same disorder. However, little is known about genetic contributions to neural mechanisms of semantic processing in unaffected ASD siblings. Therefore, this study aimed to adopt an endophenotype approach to investigate the differences in underlying neural mechanisms of semantic processing in youths with ASD, their unaffected siblings, and typically developing (TD) youths. Endophenotypes for ASD were defined as similarly heritable traits that were correlated with ASD. This study recruited 39 ASD youths (mean age = 14.8 years, standard deviation [SD] = 3.9 years), their unaffected siblings (mean age = 15.7 years, SD= 5.2 years), and 40 TD youths (mean age = 14.6 years, SD= 4.7 years). These three groups of participants were matched with IQ, age, and handedness. Participants were instructed to judge whether two Chinese characters were related in meaning in an MRI scanner. Our behavioral result showed that there were no significant differences on accuracy and reaction time among these three groups. Brain imaging data revealed that unaffected siblings and TD youths showed greater brain activation in the left MTG as compared with ASD youths. Moreover, the unaffected siblings and ASD youths showed greater cuneus activation as compared to TD youths. Furthermore, unaffected siblings showed intermediate left IFG activation between TD and ASD youths, with the strongest activation in TD youths and the weakest in ASD youths. Our findings provided a supportive evidence that abnormal neural activation in the left IFG and the cuneus during semantic processing could serve as an endophenotype of ASD. For our unaffected ASD siblings, they may have intact lexical representation system as TD youths; however, because of shared genetic features with ASD, they adpoted the same perception-based strategies as ASD youths, and may have rather immature ability to manipulate semantic representations.
Livres sur le sujet "Neural mechanisms autism"
Westberg, Lars, et Hasse Walum. Oxytocin and Vasopressin Gene Variation and the Neural Basis of Social Behaviors. Sous la direction de Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.011.
Texte intégralPatisaul, Heather B., et Scott M. Belcher. Endocrine Disruptors and Neurobehavioral Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780199935734.003.0006.
Texte intégralBeauchaine, Theodore P., et Sheila E. Crowell, dir. The Oxford Handbook of Emotion Dysregulation. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190689285.001.0001.
Texte intégralGaitanis, John, Phillip L. Pearl et Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Sous la direction de Donald L. Schomer et Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.
Texte intégralFriston, Karl J., et Raymond J. Dolan. Computational Psychiatry and the Bayesian Brain. Sous la direction de Dennis S. Charney, Eric J. Nestler, Pamela Sklar et Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0072.
Texte intégralChapitres de livres sur le sujet "Neural mechanisms autism"
Anagnostou, Evdokia, Deepali Mankad, Joshua Diehl, Catherine Lord, Sarah Butler, Andrea McDuffie, Lisa Shull et al. « Neural Mechanisms in Autism ». Dans Encyclopedia of Autism Spectrum Disorders, 1994–2007. New York, NY : Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_572.
Texte intégralCasanova, Manuel. « Neural Mechanisms in Autism ». Dans Encyclopedia of Autism Spectrum Disorders, 3102–15. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_572.
Texte intégralIbrahim, Karim, Gregory McCarthy et Denis G. Sukhodolsky. « Neural Mechanisms of Emotional Dysregulation ». Dans Encyclopedia of Autism Spectrum Disorders, 1–4. New York, NY : Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-6435-8_102453-1.
Texte intégralIbrahim, Karim, Gregory McCarthy et Denis G. Sukhodolsky. « Neural Mechanisms of Emotional Dysregulation ». Dans Encyclopedia of Autism Spectrum Disorders, 3115–17. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_102453.
Texte intégralFolstein, Susan E., Michael Dowd, Raymond Mankoski et Ovsanna Tadevosyan. « How Might Genetic Mechanisms Operate in Autism ? » Dans Autism : Neural Basis and Treatment Possibilities, 70–83. Chichester, UK : John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470869380.ch5.
Texte intégralNakai, Nobuhiro, Eric T. N. Overton et Toru Takumi. « Optogenetic Approaches to Understand the Neural Circuit Mechanism of Social Deficits Seen in Autism Spectrum Disorders ». Dans Advances in Experimental Medicine and Biology, 523–33. Singapore : Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-8763-4_36.
Texte intégralHirsch, Emily, et Leslie Hulvershorn. « Neural Findings in Pediatric Irritability ». Dans Irritability in Pediatric Psychopathology, sous la direction de Amy Krain Roy, Melissa A. Brotman et Ellen Leibenluft, 171–94. Oxford University Press, 2019. http://dx.doi.org/10.1093/med-psych/9780190846800.003.0009.
Texte intégralFriedman, Hagit. « Autism Spectrum Disorder (ASD) : From Molecular Mechanism to Novel Therapeutic Approach ». Dans Learning Disabilities - Neurobiology, Assessment, Clinical Features and Treatments. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.100537.
Texte intégralChristian, Kimberly M., Song Hongjun et Ming Guo-li. « Application of Stem Cells to Understanding Psychiatric Disorders ». Dans Neurobiology of Mental Illness, sous la direction de Karl Deisseroth, 123–26. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0009.
Texte intégralSagata, Noriaki, Yasunari Sakai et Takahiro A. Kato. « Clarifying the Pathophysiological Mechanisms of Neuronal Abnormalities of NF1 by Induced-Neuronal (iN) Cells from Human Fibroblasts ». Dans Neurofibromatosis [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.98817.
Texte intégralActes de conférences sur le sujet "Neural mechanisms autism"
Nikolaeva, Elena I. « Genetics and psychophysiology of ADHD and autism ». Dans 2nd International Neuropsychological Summer School named after A. R. Luria “The World After the Pandemic : Challenges and Prospects for Neuroscience”. Ural University Press, 2020. http://dx.doi.org/10.15826/b978-5-7996-3073-7.12.
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