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Bibliographies thématiques / Multiple disease resistance / Livres

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Livres sur le sujet « Multiple disease resistance »

Auteur : Grafiati

Publié le 10 mars 2023

Mis à jour le 31 juillet 2025

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Consultez les 18 meilleurs livres pour votre recherche sur le sujet « Multiple disease resistance ».

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1

Hopkins, Tanne Janice, ed. Timebomb: The global epidemic of multi-drug-resistant tuberculosis. McGraw-Hill, 2002.

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2

Stacey, Knobler, and Institute of Medicine (U.S.). Forum on Emerging Infections., eds. The resistance phenomenon in microbes and infectious disease vectors: Implications for human health and strategies for containment : workshop summary. National Academies Press, 2003.

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3

Border, Peter. Diseases fighting back: The growing resistance of TB and other bacterial diseases to treatment. Parliamentary Office of Science and Technology, 1994.

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4

Parliamentary Office of Science and Technology. Diseases fighting back. Parliamentary Office of Science and Technology, 1994.

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5

File, Thomas. New insights in the treatment of severe infections in the multiple-drug resistant situation: Proceedings of a satellite symposium to the 11th International Congress on Infectious Diseases, Cancun, Mexico, March 5, 2004. Karger, 2004.

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6

Fontanesi, Luca, ed. The genetics and genomics of the rabbit. CABI, 2021. http://dx.doi.org/10.1079/9781780643342.0000.

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Abstract The purpose of the book is to present in one location a comprehensive overview of the progress of genetics in the rabbit, with a modern vision that integrates genomics to obtain a complete picture of the state of the art and of the applications in this species, defined according to the multiple uses and multi-faceted places that this species has in applied and fundamental biology. The 18 chapters cover several fields of genetics and genomics: Chapters 1 and 2 present the rabbit within the evolutionary framework, including the systematics, its domestication and an overview of the genet

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7

Gillespie, Stephen H. Management of Multiple Drug-Resistant Infections (Infectious Disease). Humana Press, 2004.

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8

Young, Rick. Hunting the nightmare bacteria. 2017.

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9

Gillespie, Stephen H. Management of Multiple Drug-resistant Infections. Humana Press Inc.,U.S., 2004.

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10

Gillespie, Stephen H. Management of Multiple Drug-Resistant Infections. Humana Press, 2010.

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11

Sentissi, Kinza, and Stephanie Yacoubian. Physiologic Airflow Disruption. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0017.

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Airflow disruption can be triggered through multiple mechanisms. The obstruction can stem from within the airway lumen, airway walls, or the tissues surrounding it. This section focuses on airflow disruption initiated by bronchospasm, obstructive lung disease, asthma and status asthmaticus. Bronchospasm presents with increased airway resistance secondary to airway hyperreactivity or anaphylaxis. Asthma and chronic obstructive pulmonary disease (COPD) are obstructive and inflammatory lung pathologies. Airflow disruption in asthma is reversible between exacerbations. The airway obstruction in CO

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12

Paterson, David L., and Yoshiro Hayashi. Antimicrobial selection policies in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0286.

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Antibiotic selection is a crucial drug choice in critically-ill patients. Optimization of empiric antibiotic choice can be gained by knowledge of the site of infection and the probable causative organisms at that site. This should be linked with knowledge of the local epidemiology of antibiotic resistance in the actual intensive care unit housing the patient. Initial empiric antimicrobial choice may need to be broad in order to cover potential antibiotic-resistant pathogens. However, it is important to be prudent in antibiotic strategy since the selection of multiple-resistant organisms by exc

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13

MacIntyre, Iain M., and David J. Webb. Resistant hypertension. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0217_update_001.

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Resistant hypertension is defined as a blood pressure above target despite adherence to at least three different antihypertensive agents. The term can be used to identify patients with difficult-to-treat hypertension, who might benefit from specialist investigation and/or treatment. It likely affects 10–15% of patients with hypertension. ‘White coat’ hypertension should be excluded first by the use of out-of-office blood pressure monitoring. Risk factors include obesity, older age, chronic kidney disease, and diabetes.Treatment is based on identifying and treating any underlying cause and thro

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14

Summers, Louisa. The effects of resistance exercise on lower extremity power in women with multiple sclerosis. 1999.

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15

Levy, Jerrold H., and David Faraoni. Pathophysiology and causes of severe hypertension. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0162.

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Hypertension affects multiple groups of patients characterized by different clinical presentations and a spectrum of potential causes. The pathophysiology is complex and multifactorial. Although most patients are labelled ‘essential hypertension’, multiple mechanisms are involved in blood pressure regulation. Factors that influence blood pressure homeostasis include endothelial function, the renin-angiotensin system, and the sympathetic nervous system. In elderly patients, hypertension is common as the vascular system and arterial stiffness also contribute. Other important factors include infl

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16

Hastie, Nick, and Eve Miller-Hodges. WT1 and its disorders. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0329_update_001.

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Mutations in the Wilms tumour suppressor gene, WT1, are associated with Wilms tumour in childhood. However, in addition WT1 has a key role in renal development, emerging roles in podocyte function, and a potential role in tissue regeneration. An understanding of WT1 is of increasing importance to clinical practice. WT1 is a complex gene with multiple isoforms. It is crucial for normal embryonic development, especially kidney development, where it is necessary for mesenchymal-to-epithelial transition to form the nephron. WT1 mutations lead to abnormalities in renal and genitourinary development

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17

Carlucci, Annalisa, and Paolo Navalesi. Weaning failure in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0103.

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Weaning failure has been defined as failure to discontinue mechanical ventilation, as assessed by the spontaneous breathing trial, or need for re-intubation after extubation, so-called extubation failure. Both events represent major clinical and economic burdens, and are associated with high morbidity and mortality. The most important mechanism leading to discontinuation failure is an unfavourable balance between respiratory muscle capacity and the load they must face. Beyond specific diseases leading to loss of muscle force-generating capacity, other factors may impair respiratory muscle func

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18

Karmali, Mohamed A., and Jan M. Sargeant. Verocytotoxin-producing Escherichia coli (VTEC) infections. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0008.

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Verocytotoxin (VT)-producing Escherichia coli (VTEC), also known as Shiga toxin producing E. coli (STEC), are zoonotic agents, which cause a potentially fatal illness whose clinical spectrum includes diarrhoea, haemorrhagic colitis, and the haemolytic uraemic syndrome (HUS). VTEC are of serious public health concern because of their association with large outbreaks and with HUS, which is the leading cause of acute renal failure in children. Although over 200 different OH serotypes of VTEC have been associated with human illness, the vast majority of reported outbreaks and sporadic cases of VTE

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