Littérature scientifique sur le sujet « Macrophages M2-Like »
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Articles de revues sur le sujet "Macrophages M2-Like"
Wen, Zhifa, Hongxiang Liu, Meng Zhou et Li-xin Wang. « Tumor released autophagosomes regulate M2-like macrophage polarization (TUM6P.974) ». Journal of Immunology 194, no 1_Supplement (1 mai 2015) : 141.22. http://dx.doi.org/10.4049/jimmunol.194.supp.141.22.
Texte intégralDraijer, Christina, Patricia Robbe, Carian E. Boorsma, Machteld N. Hylkema et Barbro N. Melgert. « Characterization of Macrophage Phenotypes in Three Murine Models of House-Dust-Mite-Induced Asthma ». Mediators of Inflammation 2013 (2013) : 1–10. http://dx.doi.org/10.1155/2013/632049.
Texte intégralLalor, Richard, et Sandra O’Neill. « Bovine κ-Casein Fragment Induces Hypo-Responsive M2-Like Macrophage Phenotype ». Nutrients 11, no 7 (23 juillet 2019) : 1688. http://dx.doi.org/10.3390/nu11071688.
Texte intégralLyu, Qingkang, Edwin J. A. Veldhuizen, Irene S. Ludwig, Victor P. M. G. Rutten, Willem van Eden, Alice J. A. M. Sijts et Femke Broere. « Characterization of polarization states of canine monocyte derived macrophages ». PLOS ONE 18, no 11 (8 novembre 2023) : e0292757. http://dx.doi.org/10.1371/journal.pone.0292757.
Texte intégralSánchez-Reyes, Karina, Alejandro Bravo-Cuellar, Georgina Hernández-Flores, José Manuel Lerma-Díaz, Luis Felipe Jave-Suárez, Paulina Gómez-Lomelí, Ruth de Celis, Adriana Aguilar-Lemarroy, Jorge Ramiro Domínguez-Rodríguez et Pablo Cesar Ortiz-Lazareno. « Cervical Cancer Cell Supernatants Induce a Phenotypic Switch from U937-Derived Macrophage-Activated M1 State into M2-Like Suppressor Phenotype with Change in Toll-Like Receptor Profile ». BioMed Research International 2014 (2014) : 1–11. http://dx.doi.org/10.1155/2014/683068.
Texte intégralZhu, Wenya, Qianqian Chen, Yi Li, Jun Wan, Jia Li et Shuai Tang. « HIF-1α-Overexpressing Mesenchymal Stem Cells Attenuate Colitis by Regulating M1-like Macrophages Polarization toward M2-like Macrophages ». Biomedicines 11, no 3 (8 mars 2023) : 825. http://dx.doi.org/10.3390/biomedicines11030825.
Texte intégralStrizova, Zuzana, Iva Benesova, Robin Bartolini, Rene Novysedlak, Eva Cecrdlova, Lily Koumbas Foley et Ilja Striz. « M1/M2 macrophages and their overlaps – myth or reality ? » Clinical Science 137, no 15 (août 2023) : 1067–93. http://dx.doi.org/10.1042/cs20220531.
Texte intégralLi, Dezhi, Min Yan, Fengfei Sun, Junmei Song, Xingsheng Hu, Sijia Yu, Lina Tang et Shishan Deng. « miR-498 inhibits autophagy and M2-like polarization of tumor-associated macrophages in esophageal cancer via MDM2/ATF3 ». Epigenomics 13, no 13 (juillet 2021) : 1013–30. http://dx.doi.org/10.2217/epi-2020-0341.
Texte intégralRonaghan, Natalie J., Mandy Soo, Uriel Pena, Marisa Tellis, Wenming Duan, Nooshin Tabatabaei-Zavareh, Philipp Kramer, Juan Hou et Theo J. Moraes. « M1-like, but not M0- or M2-like, macrophages, reduce RSV infection of primary bronchial epithelial cells in a media-dependent fashion ». PLOS ONE 17, no 10 (13 octobre 2022) : e0276013. http://dx.doi.org/10.1371/journal.pone.0276013.
Texte intégralDi Martile, Marta, Valentina Farini, Francesca Maria Consonni, Daniela Trisciuoglio, Marianna Desideri, Elisabetta Valentini, Simona D'Aguanno et al. « Melanoma-specific bcl-2 promotes a protumoral M2-like phenotype by tumor-associated macrophages ». Journal for ImmunoTherapy of Cancer 8, no 1 (avril 2020) : e000489. http://dx.doi.org/10.1136/jitc-2019-000489.
Texte intégralThèses sur le sujet "Macrophages M2-Like"
Eisel, David [Verfasser], et Viktor [Akademischer Betreuer] Umansky. « Reprogramming of M2-like macrophages by CD4+ T cells, transcription factor knockdown and miRNA transfection / David Eisel ; Betreuer : Viktor Umansky ». Heidelberg : Universitätsbibliothek Heidelberg, 2019. http://d-nb.info/1213029317/34.
Texte intégralAbdulhadi, Fatma Husien S. « DIFFERENTIATION OF U-937 MONOCYTES TO MACROPHAGE-LIKE CELLS POLARIZED INTO M1 OR M2 PHENOTYPES ACCORDING TO THEIR SPECIFIC ENVIRONMENT : A STUDY OF MORPHOLOGY, CELL VIABILITY, AND CD MARKERS OF AN IN VITRO MODEL OF HUMAN MACROPHAGES ». Wright State University / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=wright1401357388.
Texte intégralBouchard, Alexanne. « La protéine de stress du réticulum endoplasmique GRP94 dans le cancer du sein triple négatif, intérêt diagnostique et thérapeutique ». Electronic Thesis or Diss., Bourgogne Franche-Comté, 2023. https://nuxeo.u-bourgogne.fr/nuxeo/site/esupversions/8b1b931d-83a7-49fd-9779-012ad3949e79.
Texte intégralTriple-negative breast cancer (TNBC) is characterized by the absence of estrogen and progesterone receptors, as well as HER2, on tumor cells. It is the most aggressive subtype of breast cancer and is associated with a higher risk of metastasis. It accounts for 15-20% of all breast cancers. Due to the lack of specific targets, hormone therapy and HER2-targeted drugs are ineffective. TNBC represents a subgroup of heterogeneous tumors that can be classified according to their molecular characteristics. A better understanding of molecular mechanisms, particularly those involved in modulating the immune response, is needed to optimize the management of this cancer. In this context, molecular imaging can represent an interesting tool: it enables the non-invasive identification and in vivo visualization of specific targets in the tumor or tumor microenvironment (TME), thanks to selective molecular probes that can be used for diagnostic and/or therapeutic purposes. In this thesis work, two specific TME targets were studied using such probes: M2-like macrophages and GARP protein, a TGF-β anchoring receptor. M2-like macrophages are recognized as having a major pro-tumoral role. The results obtained enabled us to demonstrate the presence of CD206+ M2-like macrophages in our CSTN model using in vivo multimodal imaging. In this study, we validated the efficacy of 99mTc-Tilmanocept in SPECT/CT as a probe for imaging M2-like macrophages in the TME of our TNBC model. We also demonstrated co-expression of these CD206+ M2-like macrophages with the GRP94 protein, an important chaperone involved in immune responses. Finally, inhibition of GRP94 with a specific inhibitor, PU-WS13, significantly decreased the number of M2-like macrophages as well as tumor growth in our TNBC model. Thus, SPECT imaging with 99mTc-Tilmanocept could represent an innovative method for imaging CD206+ M2-like macrophages as a potential biomarker for prognosis, therapeutic prediction and/or monitoring of solid tumors. The second target studied, the GARP protein, is expressed at the membrane of Tregs and tumor cells and plays a key role in the activation of TGF-β, a major immunosuppressive cytokine in cancer development. The development of a theranostic approach targeting GARP combining imaging (111In-DOTAGA-GARP) and targeted radionuclide therapy (TRT) (177Lu-DOTAGA-GARP) has been achieved. We showed in our preclinical TNBC model that GARP expression was increased after external radiotherapy, a classic therapeutic strategy, and could be specifically detected and quantified in the TME using in vivo SPECT/CT imaging with the 111In-DOTAGA-GARP probe. Moreover, its use in its therapeutic form (177Lu-DOTAGA-GARP) limited tumor growth. This theranostic strategy could enable the personalization of cancer treatments by identifying and treating patients likely to respond to therapy targeting Tregs via TRT
Sharp, Bradley M. « Conversion of the U937 Monocyte into “Macrophage-like&#x201d ; Populations Exhibiting M1 or M2 Characteristics ». Wright State University / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=wright1368748415.
Texte intégralAlmeida, Cátia Alexandra Rebelo de. « Bevacizumab : more than an anti-angiogenic drug ». Master's thesis, 2018. http://hdl.handle.net/10362/65518.
Texte intégralDong-JerShiau et 蕭東哲. « NADPH oxidase regulates Toll-like receptor 2-dependent M2 tumor-associated macrophage polarization ». Thesis, 2015. http://ndltd.ncl.edu.tw/handle/02292645582092102396.
Texte intégral國立成功大學
微生物及免疫學研究所
103
Tumor-associated macrophages (TAM) are considered to be a major cell population in the tumor microenvironment and can be differentiated into M2 phenotype supporting tumor existence. However, the switching mechanisms of polarized TAM by tumor cells are yet to be determined. We previously showed that unknown factors in the hepatoma microenvironment trigger Toll like receptor 2 (TLR2) signaling to induce the degradation of NF-κB p65 via ERK1/2 activation and autophagy promoting the polarization of M2 macrophages. The underlying mechanism is yet to be determined. In this study we found hepatoma conditioned medium stimulates reactive oxygen species (ROS) to trigger ERK1/2 activation, autophagy-mediated NF-κB degradation and M2 macrophage polarization. This ROS-mediated NF-κB p65 degradation was attenuated in NADPH oxidase 2 (NOX2)-deficient cells. In addition, TLR2 signaling was found to be involved in NOX2-regulated M2 macrophage polarization. An endogenous TLR2 ligand, high-mobility group protein B1 (HMGB1), was found to be secreted by hepatoma cells. HMGB1 suppression by RNA-silencing or neutralizing antibody reduces ROS generation, ERK1/2 activation, autophagy-mediated NF-κB p65 degradation and M2 macrophage polarization. Our findings suggest that soluble HMGB1 can trigger TLR2 -dependent autophagy-mediated NF-κB p65 degradation and M2 macrophage polarization. Furthermore, we demonstrated that HMGB1 regulates tumor nodule formation and M2 macrophage recruitment in an in situ mice hepatoma model. In conclusion, we found a novel function of HMGB1 to regulate-TLR2/NOX2 dependent M2 polarization and TAM recruitment in hepatoma.
Livres sur le sujet "Macrophages M2-Like"
Tsai, Ching-Wei, Sanjeev Noel et Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.
Texte intégralChapitres de livres sur le sujet "Macrophages M2-Like"
Multhoff, G., E. A. Repasky et Peter Vaupel. « Mild Hyperthermia Induced by Water-Filtered Infrared A Irradiation : A Potent Strategy to Foster Immune Recognition and Anti-Tumor Immune Responses in Superficial Cancers ? » Dans Water-filtered Infrared A (wIRA) Irradiation, 129–39. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92880-3_10.
Texte intégralZhu, Lingyan, Yu Xiao, Yao Xiao, Yinan Jiang, Maha Adama et George K. Gittes. « Macrophages as a Target for Treating Diabetic Foot Ulcers ». Dans Diabetic Foot - Recent Advances [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.106613.
Texte intégralSanta, Kazuki. « Macrophages : Phagocytosis, Antigen Presentation, and Activation of Immunity ». Dans Phagocytosis - Main Key of Immune System [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.110832.
Texte intégralPedro Lapuente, Juan. « Immunomodulatory Effects of a M2-Conditioned Medium (PRS® CK STORM) : Theory on the Possible Complex Mechanism of Action through Anti-Inflammatory Modulation of the TLR System and the Purinergic System ». Dans Purinergic System [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104486.
Texte intégralActes de conférences sur le sujet "Macrophages M2-Like"
Eisel, David, Wolfram Osen, Krishna Das, Franziska Marie-Claire Hoerhold, Rainer König et Stefan B. Eichmüller. « Abstract A067 : Cognate interaction with CD4+ T-cells instructs M2-like macrophages to acquire M1-like phenotype ». Dans Abstracts : Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference : Translating Science into Survival ; September 30 - October 3, 2018 ; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-a067.
Texte intégralSarkar, Sandipto, Prashant K. Thakkar, Heinz Lenz, Peter Enzinger, Andrew H. Ko, Allyson J. Ocean, Yao Lu et al. « Abstract 2011 : HER2 expression and M2-like tumor infiltrating macrophages associated with Cabazitaxel activity in gastric cancer ». Dans Proceedings : AACR Annual Meeting 2020 ; April 27-28, 2020 and June 22-24, 2020 ; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-2011.
Texte intégralZhao, Hailan, Jing Xu, Jiaqi Wang, Haoming Xu, Wenqi Huang, Xue Guo, Yong Zhang, Yandi Liu, Hongli Huang et Youlian Zhou. « IDDF2022-ABS-0079 Short-chain fatty acid-producing mixed bacteria improve experimental colitis via promoting M2-like macrophages polarization ». Dans Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 2–4 September 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-iddf.44.
Texte intégralEl Kasmi, Karim C., Suzette R. Riddle, Aimee Anderson, Min Li, Amanda Flockton, Megan Noe, Adil Anwar et al. « Activated Adventitial Pulmonary Artery Fibroblasts Drive Macrophages Toward A Distinct M2-Like Phenotype Through STAT3 Signaling In Pulmonary Hypertension ». Dans American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4745.
Texte intégralBruno, Antonino, Denisa Baci, Matteo Gallazzi, Annalisa Bosi, Angelo Naselli, Andrea Guarneri, Lorenzo Mortara, Douglas Noonan et Adriana Albini. « Abstract 1581 : Prostate tumor associated NK cells (PTANKs) acquire the decidual-like/pro-angiogenic phenotype and polarize macrophages towards the M2-like/TAM subset ». Dans Proceedings : AACR Annual Meeting 2020 ; April 27-28, 2020 and June 22-24, 2020 ; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-1581.
Texte intégralLee, Kim Yee, Siew Meng Chong et Yaw Chyn Lim. « Abstract A10 : L1236, a Hodgkin and Reed-Sternberg cell line, stimulates monocytes in vitro to differentiate into M2-like macrophages ». Dans Abstracts : Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011 ; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-a10.
Texte intégralHan, Zhen, Chingyu Su, Xiwei Wu, Hanjun Qin, Steven T. Rosen et Christiane Querfeld. « Abstract 2759 : Reprogramming of PD1+ M2-like tumor-associated macrophages with anti-PD-L1 and Lenalidomide in cutaneous T cell lymphoma ». Dans Proceedings : AACR Annual Meeting 2021 ; April 10-15, 2021 and May 17-21, 2021 ; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-2759.
Texte intégralSable, Rushikesh, Jesse Jaynes, Michael Ronzetti, Wendy Guzman, Zachary Knotts, Natalia de Val, Juan Morgan, Clayton Yates, Baljinnyam Bolormaa et Udo Rudloff. « Abstract B49 : Precision targeting of M2-like macrophages by the innate defense regulator RP-182 in pancreatic cancer and noncancerous diseases ». Dans Abstracts : AACR Special Conference on Pancreatic Cancer : Advances in Science and Clinical Care ; September 6-9, 2019 ; Boston, MA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.panca19-b49.
Texte intégralDai, M.-S., H.-C. Lo, L.-C. Chen, A. Janckilla et S.-F. Tseng. « Abstract P2-01-07 : M2-like tumor-associated macrophages require Tartrate-resistant acid phosphatase as overall function to promote breast cancer metastasis ». Dans Abstracts : 2018 San Antonio Breast Cancer Symposium ; December 4-8, 2018 ; San Antonio, Texas. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-p2-01-07.
Texte intégralAlbini, Adriana, Denisa Baci, Matteo Gallazzi, Federico Dehò, Angelo Naselli, Andrea Guernieri, Lorenzo Mortara, Douglas M. Noonan et Antonino Bruno. « Abstract LT006 : NK cells from prostate cancer patients acquire a pro-angiogenic phenotype and polarize macrophages towards a M2-like/TAM subset ». Dans Abstracts : AACR Virtual Special Conference : The Evolving Tumor Microenvironment in Cancer Progression : Mechanisms and Emerging Therapeutic Opportunities ; in association with the Tumor Microenvironment (TME) Working Group ; January 11-12, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.tme21-lt006.
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