Littérature scientifique sur le sujet « LHCGR gene »
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Articles de revues sur le sujet "LHCGR gene"
Montgomery, G. W., M. L. Tate, H. M. Henry, J. M. Penty et R. M. Rohan. « The follicle-stimulating hormone receptor and luteinizing hormone receptor genes are closely linked in sheep and deer ». Journal of Molecular Endocrinology 15, no 3 (décembre 1995) : 259–65. http://dx.doi.org/10.1677/jme.0.0150259.
Texte intégralCannon, Jennifer D., Srinivas V. Seekallu, Catherine A. VandeVoort et Charles L. Chaffin. « Association of luteinizing hormone receptor gene expression with cell cycle progression in granulosa cells ». American Journal of Physiology-Endocrinology and Metabolism 296, no 6 (juin 2009) : E1392—E1399. http://dx.doi.org/10.1152/ajpendo.90965.2008.
Texte intégralCheemakurthi, Ravi Krishna, Gottumukkala Achyuta Rama Raju, Thota Sivanaryana, Kalagara Madan, Kota Murali Krishna et Godi Sudhakar. « Case Report : A 54 base pair inactivating mutation of LHCGR in a 28-year old woman with poor ovarian response ». F1000Research 4 (18 mars 2015) : 72. http://dx.doi.org/10.12688/f1000research.6137.1.
Texte intégralLubis, Hilma Putri, Muhammad Fidel Ganis Siregar, Ichwanul Adenin, Binarwan Halim, Henry Salim Siregar et M. Oky Prabudi. « Association between Luteinizing Hormone/Choriogonadotropin Receptor Ins18LQ Gene Polymorphism and Polycystic Ovary Syndrome ». Open Access Macedonian Journal of Medical Sciences 8, A (10 août 2020) : 517–20. http://dx.doi.org/10.3889/oamjms.2020.4182.
Texte intégralXu, Yufei, Yulin Chen, Niu Li, Xuyun Hu, Guoqiang Li, Yu Ding, Juan Li, Yiping Shen, Xiumin Wang et Jian Wang. « Novel compound heterozygous variants in the LHCGR gene identified in a subject with Leydig cell hypoplasia type 1 ». Journal of Pediatric Endocrinology and Metabolism 31, no 2 (26 janvier 2018) : 239–45. http://dx.doi.org/10.1515/jpem-2016-0445.
Texte intégralDoroszko, Milena, Marcin Chrusciel, Joanna Stelmaszewska, Tomasz Slezak, Adolfo Rivero-Muller, Artur Padzik, Slawomir Anisimowicz et al. « Luteinizing Hormone and GATA4 Action in the Adrenocortical Tumorigenesis of Gonadectomized Female Mice ». Cellular Physiology and Biochemistry 43, no 3 (2017) : 1064–76. http://dx.doi.org/10.1159/000481718.
Texte intégralJeong, Hwal, Hae Lee et Jin Hwang. « LHCGR Gene Analysis in Girls with Non-Classic Central Precocious Puberty ». Experimental and Clinical Endocrinology & ; Diabetes 127, no 04 (5 mars 2018) : 234–39. http://dx.doi.org/10.1055/s-0043-125067.
Texte intégralWang, Peng, Han Zhao, Tao Li, Wei Zhang, Keliang Wu, Mei Li, Yuehong Bian et al. « Hypomethylation of the LH/Choriogonadotropin Receptor Promoter Region Is a Potential Mechanism Underlying Susceptibility to Polycystic Ovary Syndrome ». Endocrinology 155, no 4 (1 avril 2014) : 1445–52. http://dx.doi.org/10.1210/en.2013-1764.
Texte intégralKulkarni, Rewa, Maria E. Teves, Angela X. Han, Jan M. McAllister et Jerome F. Strauss. « Colocalization of Polycystic Ovary Syndrome Candidate Gene Products in Theca Cells Suggests Novel Signaling Pathways ». Journal of the Endocrine Society 3, no 12 (16 septembre 2019) : 2204–23. http://dx.doi.org/10.1210/js.2019-00169.
Texte intégralZhang, Zhiwei, Shuk-Wa Lau, Lingling Zhang et Wei Ge. « Disruption of Zebrafish Follicle-Stimulating Hormone Receptor (fshr) But Not Luteinizing Hormone Receptor (lhcgr) Gene by TALEN Leads to Failed Follicle Activation in Females Followed by Sexual Reversal to Males ». Endocrinology 156, no 10 (20 mai 2015) : 3747–62. http://dx.doi.org/10.1210/en.2015-1039.
Texte intégralThèses sur le sujet "LHCGR gene"
Kulkarni, Rewa M. « CO-LOCALIZATION OF POLYCYSTIC OVARY SYNDROME CANDIDATE GENE PRODUCTS IN HUMAN THECA CELLS SUGGESTS NOVEL SIGNALING PATHWAYS ». VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5741.
Texte intégralCosta, Marcia Helena Soares. « Estudo da expressão dos receptores do peptídeo insulinotrópico dependente de glicose (GIPR) e do hormônio luteinizante (LHCGR) em tumores e hiperplasias do córtex adrenal ». Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-11092007-134837/.
Texte intégralIntroduction: The glucose- dependent insulinotropic peptide receptor (GIPR) and luteinizing hormone receptor (LHCGR) are G-protein coupled receptors with a wide tissue expression pattern. The aberrant expression of these receptors has been described in cases of ACTH-independent macronodular adrenal hyperplasia (AIMAH) and in some adenomas, resulting in the increase of adrenal cortex hormonal secretion (cortisol, androgens and aldosterone). The role of these receptors in other forms of adrenocortical hyperplasia, such as primary pigmented nodular adrenocortical disease (PPNAD), adrenal enlargement associated with multiple endocrine neoplasia type 1 (MEN1), and adrenocortical carcinoma has been scarcely investigated. Thus, the study of the expression of these receptors in patients with sporadical adrenocortical tumors, AIMAH, PPNAD and adrenal enlargement associated to MEN1 was considered important. Objectives: 1) Molecular study in patients with multiple endocrine neoplasia type 1 and PPNAD: mutation screening of MEN1 and PRKAR1A genes and analysis of the loss of heterozygosis (LOH) of these genes in the adrenal lesions of these patients. 2) To quantify the GIPR and LHCGR expression, in normal, tumor and hyperplasic tissue and to correlate the expression of these receptors with the adrenocortical tumor histology. Patients: 55 patients (30 adults) with adrenocortical tumors (37 adenomas and 18 carcinomas); 7 patients with AIMAH, 4 with MEN1, 1 with PPNAD and control tissue (adrenal, testis and pancreas). Methods: Extraction of genomic DNA, RNA and synthesis of complementary DNA (cDNA); PCR-amplification of the coding regions of MEN1 and PRKAR1A, followed by direct sequencing. LOH study using polymorphic marker amplification by PCR and GeneScan software analysis. Quantification of GIPR and LHCGR expression using realtime PCR -TaqMan method and GIPR immunohistochemistry study in adrenocortical tumors. Results: Identification of 3 mutations (893+ 1G>A, W183X and A68fsX118) and two polymorphic alterations (S145S and D418D) in MEN1 and a mutation (Y21X) in the PRKAR1A gene; LOH was not identified in adrenal tissue. The GIPR and LHCGR expression was identified in normal, tumor and hyperplasic adrenal tissues; the GIPR expression level was more elevated in malignant tumors compared to benign tumors in pediatric (median = 18.1 and 4.6, respectively; p <0.05) and adult patients (median = 4.8 and 1.3 respectively; p <0.001). The LHCGR expression in pediatric patients was elevated in benign as well as in malignant tumors (median = 6.4 and 4.3, respectively). In the adult group, the expression level of these receptors was extremely low in malignant tumors in relation to benign ones (median = 0.06 and 2.3, respectively; p <0.001). The GIPR immunohistochemistry was variable and did not correlate with GIPR gene expression. No difference between GIPR and LHCGR expression levels was observed in the different forms of hyperplasia. Conclusions: The presence of LOH and mutations in compound heterozygosis of MEN1 and PRKAR1A genes were ruled out as the mechanisms responsible for the adrenal enlargement in patients with multiple endocrine neoplasia type 1. GIPR overexpression is associated with malignant adrenocortical tumors in the adult and pediatric patients and low LHCGR expression is associated with malignant adrenocortical tumors only in the adult patients.
FORTUNATO, ANGELO. « Identification and characterization of genes involved in the development and progression of colorectal and endometrial cancers ». Doctoral thesis, 2012. http://hdl.handle.net/2158/794612.
Texte intégralChapitres de livres sur le sujet "LHCGR gene"
Gromoll, J., A. Richter-Unruh et N. Kossack. « A Novel Exon within the LH/CG Receptor Gene as Transcriptional Regulator of LHCGR Signalling. » Dans The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego, OR21–2—OR21–2. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part3.or1.or21-2.
Texte intégralActes de conférences sur le sujet "LHCGR gene"
Kovalchuk, Svetlana, Anna Arkhipova et Аrina Tagmazian. « Development of the Real-Time PCR method for detection of ss52050737 polymorphism of lhcgr gene in cattle ». Dans Proceedings of the International Scientific and Practical Conference “Digital agriculture - development strategy” (ISPC 2019). Paris, France : Atlantis Press, 2019. http://dx.doi.org/10.2991/ispc-19.2019.78.
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