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Littérature scientifique sur le sujet « Lateropulsion »

Auteur : Grafiati

Publié le 5 novembre 2024

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Articles de revues sur le sujet "Lateropulsion"

Delva, Iryna, Olga Oksak et Mykhaylo Delva. « TIME COURSE AND PREDICTORS OF RECOVERY FROM LATEROPULSION AFTER HEMISPHERIC STROKE (PROSPECTIVE STUDY) ». Eastern Ukrainian Medical Journal 12, n^o 1 (2024) : 174–82. http://dx.doi.org/10.21272/eumj.2024;12(1):174-182.

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Introduction. In recent years, considerable attention has been paid to the abnormality of body verticality perception in stroke patients. Most often, misperception of body verticality is manifested in the form of lateropulsion and repulsion syndrome. Objective: to study the timing of recovery from lateropulsion (pusher syndrome) and to determine the predictors of lateropulsion resolution in patients with hemispheric strokes. Material and methods. We included in the study patients with hemispheric strokes occurring within the last month. 61 patients were diagnosed with lateropulsion and 9 patients with pusher syndrome, according to the Scale for Contraversive Pushing. After initial examination, patients were subsequently invited for a weekly examination until the body's verticality was normalized. Results. Recovery time from pusher syndrome was significantly longer – 9.0 (95% confidence interval: 7.1–10.4) weeks compared to recovery time from lateropulsion – 5.9 (95% confidence interval: 5.5–6.3) weeks. Among all the studied factors, only spatial hemineglect was a significant independent predictor of a much longer resolution time of lateropulsion (hazard ratio 2.36; 95% confidence interval: 1.20–4.27). The mean duration of lateropulsion in patients with spatial hemineglect was 6.3 (95% confidence interval: 5.8–6.8) weeks, whereas in patients without spatial hemineglect, it was 4.8 (95% confidence interval: 4.3–5.4) weeks. In a subgroup of patients without spatial hemineglect, higher Fazekas scale values were a significant independent predictor of longer resolution time of lateropulsion (hazard ratio 2.38; confidence interval 95%: 1.25–4.48). Conclusions. After hemispheric strokes recovery time from pusher syndrome is much longer than recovery from lateropulsion. Recovery time from lateropulsion is determined by spatial hemineglect and leukoaraiosis severity.

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Lee, Kyoung Bo, Sang Won Yoo, Eun Kyu Ji, Woo Seop Hwang, Yeun Jie Yoo, Mi-Jeong Yoon, Bo Young Hong et Seong Hoon Lim. « Is Lateropulsion Really Related with a Specific Lesion of the Brain ? » Brain Sciences 11, n^o 3 (10 mars 2021) : 354. http://dx.doi.org/10.3390/brainsci11030354.

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Lateropulsion (pusher syndrome) is an important barrier to standing and gait after stroke. Although several studies have attempted to elucidate the relationship between brain lesions and lateropulsion, the effects of specific brain lesions on the development of lateropulsion remain unclear. Thus, the present study investigated the effects of stroke lesion location and size on lateropulsion in right hemisphere stroke patients. The present retrospective cross-sectional observational study assessed 50 right hemisphere stroke patients. Lateropulsion was diagnosed and evaluated using the Scale for Contraversive Pushing (SCP). Voxel-based lesion symptom mapping (VLSM) analysis with 3T-MRI was used to identify the culprit lesion for SCP. We also performed VLSM controlling for lesion volume as a nuisance covariate, in a multivariate model that also controlled for other factors contributing to pusher behavior. VLSM, combined with statistical non-parametric mapping (SnPM), identified the specific region with SCP. Lesion size was associated with lateropulsion. The precentral gyrus, postcentral gyrus, inferior frontal gyrus, insula and subgyral parietal lobe of the right hemisphere seemed to be associated with the lateropulsion; however, after adjusting for lesion volume as a nuisance covariate, no lesion areas were associated with the SCP scores. The size of the right hemisphere lesion was the only factor most strongly associated with lateropulsion in patients with stroke. These results may be useful for planning rehabilitation strategies of restoring vertical posture and understanding the pathophysiology of lateropulsion in stroke patients.

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Dai, Shenhao, Céline Piscicelli, Emmanuelle Clarac, Monica Baciu, Marc Hommel et Dominic Pérennou. « Lateropulsion After Hemispheric Stroke ». Neurology 96, n^o 17 (15 mars 2021) : e2160-e2171. http://dx.doi.org/10.1212/wnl.0000000000011826.

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ObjectiveTo test the hypothesis that lateropulsion is an entity expressing an impaired body orientation with respect to gravity in relation to a biased graviception and spatial neglect.MethodsData from the DOBRAS cohort (ClinicalTrials.gov: NCT03203109) were collected 30 days after a first hemisphere stroke. Lateral body tilt, pushing, and resistance were assessed with the Scale for Contraversive Pushing.ResultsAmong 220 individuals, 72% were upright and 28% showed lateropulsion (tilters [14%] less severe than pushers [14%]). The 3 signs had very high factor loadings (>0.90) on a same dimension, demonstrating that lateropulsion was effectively an entity comprising body tilt (cardinal sign), pushing, and resistance. The factorial analyses also showed that lateropulsion was inseparable from the visual vertical (VV), a criterion referring to vertical orientation (graviception). Contralesional VV biases were frequent (44%), with a magnitude related to lateropulsion severity: upright −0.6° (−2.9; 2.4), tilters −2.9° (−7; 0.8), and pushers −12.3° (−15.4; −8.5). Ipsilesional VV biases were less frequent and milder (p < 0.001). They did not deal with graviception, 84% being found in upright individuals. Multivariate, factorial, contingency, and prediction analyses congruently showed strong similarities between lateropulsion and spatial neglect, the latter encompassing the former.ConclusionsLateropulsion (pusher syndrome) is a trinity constituted by body tilt, pushing, and resistance. It is a way to adjust the body orientation in the roll plane to a wrong reference of verticality. Referring to straight above, lateropulsion might correspond to a form of spatial neglect (referring to straight ahead), which would advocate for 3D maps in the human brain involving the internal model of verticality.

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Gomez-Risquet, Maria, Anja Hochsprung, Eleonora Magni et Carlos Luque-Moreno. « Feedback Interventions in Motor Recovery of Lateropulsion after Stroke : A Literature Review and Case Series ». Brain Sciences 14, n^o 7 (5 juillet 2024) : 682. http://dx.doi.org/10.3390/brainsci14070682.

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Lateropulsion is a post-stroke phenomenon marked by an active push of the body across the midline towards the more affected side and/or a resistance of the weight shift towards the less affected side. Within the mechanisms of treatment, feedback systems have been shown to be effective. The aim of the present study was to create a body of knowledge by performing a literature review on the use of feedback mechanisms in the treatment of lateropulsion and to report two cases of lateropulsion patients who had undergone feedback-based treatment. Methods: The review was performed across five different databases (Embase, Medline/PubMed, Scopus, Web of Science, and PEDro) up to February 2024, and haptic feedback intervention was incorporated into the case series (with lateropulsion and ambulation capacity as the main variables). Results: In total, 211 records were identified and 6 studies were included after the review of the literature. The most used feedback modality was visual feedback. In the case series, positive results were observed from the intervention, particularly in the recovery of lateropulsion and balance, as well as in the improvement of gait for one patient. Patients demonstrated good adherence to the intervention protocol without adverse effects. Conclusions: Visual feedback is the most commonly used feedback modality in lateropulsion patients but other mechanisms such as haptic feedback also are feasible and should be taken into account. Larger sample sizes, extended follow-up periods, and the isolation of feedback mechanisms must be established to clarify evidence.

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Dai, Shenhao, Camille Lemaire, Céline Piscicelli et Dominic Pérennou. « Lateropulsion Prevalence After Stroke ». Neurology 98, n^o 15 (21 février 2022) : e1574-e1584. http://dx.doi.org/10.1212/wnl.0000000000200010.

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Background and ObjectivesLateropulsion is a deficit of active body orientation with respect to gravity in the frontal plane, mostly observed after a stroke. It magnifies mobility limitations and represents an emerging target in rehabilitation. Efforts to design specific interventional studies require some basic knowledge of epidemiology, which is insufficient today because many studies have focused on a few severe forms in individuals called pushers. The objectives of this study were to bridge this gap.MethodsWe systematically searched MEDLINE, EMBASE, CINAHL, and Cochrane Clinical Trials up to 31 May 2021 for original research reporting a prevalence or incidence of poststroke lateropulsion. We followed MOOSE and PRISMA guidelines. Eligibility for inclusion, data extraction, and study quality (Joanna Briggs Institute guidelines) were evaluated by 2 reviewers who used a standardized protocol (PROSPERO; CRD42020175037). A random-effects meta-analysis was used to obtain the pooled prevalence, whose heterogeneity was investigated by subgroup analysis (stroke locations and poststroke phases) and metaregression.ResultsWe identified 22 studies (5,125 individuals; mean age 68.5 years; 42.6% female; assessed 24 days, on average, after stroke), most published after 2000. The studies' quality was adequate, with only 8 (36.4%) showing risk of bias. The pooled lateropulsion prevalence was 55.1% (95% CI 35.9–74.2) and was consistent across assessment tools. After supratentorial stroke, lateropulsion prevalence was 41% (95% CI 33.5–48.5), and only 12.5% (95% CI 9.2–15.9) in individuals with severe lateropulsion, called pushers. Metaregression did not reveal any effect of age, sex, geographic region, publication year, or study quality. Lateropulsion prevalence progressively decreased from 52.8% (95% CI 40.7–65) in the acute phase to 37% (95% CI 26.3–47.7) in the early subacute phase and 22.8% (95% CI 0–46.3) in the late subacute phase. The ratio of right to left hemispheric stroke with lateropulsion increased as a function of time: 1.7 in the acute phase to 7.7 in the late subacute phase. After infratentorial stroke, lateropulsion prevalence was very high, reaching 83.2% (95% CI 63.9–100.3).DiscussionPoststroke lateropulsion prevalence is high, which appeals for its systematic detection to guide early interventions. Uprightness is predominantly controlled from the right hemisphere.

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Komiyama, Atsushi. « Isolated Body Lateropulsion ». Equilibrium Research 79, n^o 6 (31 décembre 2020) : 566–68. http://dx.doi.org/10.3757/jser.79.566.

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Dai, Shenhao, et Dominic Pérennou. « Renaissance of “lateropulsion” ». Annals of Physical and Rehabilitation Medicine 64, n^o 6 (novembre 2021) : 101595. http://dx.doi.org/10.1016/j.rehab.2021.101595.

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Gillespie, Jaime, Katelyn D. Bosteder, Radha Morar, Molly Trammell, Simon Driver et Chad Swank. « Physical therapist burden delivering gait training for a patient with lateropulsion after stroke during inpatient rehabilitation : a single-case design ». International Journal of Therapy and Rehabilitation 31, n^o 10 (2 octobre 2024) : 1–11. http://dx.doi.org/10.12968/ijtr.2024.0075.

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Background/Aims Gait training for patients with lateropulsion after stroke improves outcomes (eg reduced lateropulsion and improved function) but can be burdensome on the physical therapist. This study describes the physical therapist burden and performance of a patient with moderate lateropulsion during three gait training approaches during inpatient rehabilitation. Methods A physical therapist delivered gait training (one session each of overground robotic exoskeleton, overground supported walking, and body weight-supported treadmill training) for a patient with lateropulsion (scoring 8 out of 17 on the Burke Lateropulsion Scale). Outcomes were physiological burden (heart rate, metabolic equivalents, respiratory exchange ratio and energy expenditure), which were measured via a wearable metabolic system and perceptual burden (National Aeronautics and Space Administration Task Load Index) on the physical therapist. Patient performance (step count, time walking, time spent upright and time in moderate-to-vigorous intensity) was recorded. Results During overground robotic exoskeleton gait training, the physical therapist's physiological metrics included an average heart rate of 116 beats per minute (minimum–maximum: 98–127, time in moderate-to-vigorous intensity was 0%), average metabolic equivalents of 3.2 (minimum–maximum: 1.7–4.3), a respiratory exchange ratio of 0.79 (minimum–maximum: 0.70–0.93), an energy expenditure of 228 kcal/hour and a perceptual burden of 33.3. The patient walked 228 steps, spent 15.4 minutes upright, 8.7 minutes walking and achieved 0% in moderate-to-vigorous intensity. During overground supported walking, the physical therapist's metrics included an average heart rate of 145 beats per minute (minimum–maximum: 113–164, time in moderate-to-vigorous intensity was 87%), average metabolic equivalents of 4.7 (minimum–maximum: 2.7–6.0), a respiratory exchange ratio of 0.96 (minimum–maximum: 0.81–1.16), an energy expenditure of 343 kcal/hour and a perceptual burden of 60.8. The patient walked 588 steps, spent 19.6 minutes upright, 10.5 minutes walking and achieved 38% in moderate-to-vigorous intensity. During body weight-supported treadmill training, the physical therapist's metrics included an average heart rate of 112 beats per minute (minimum–maximum: 69–137, time in moderate-to-vigorous intensity was 34%), average metabolic equivalents of 3.9 (minimum–maximum: 3.2–4.4), a respiratory exchange ratio of 0.89 (minimum–maximum: 0.82–0.95), an energy expenditure of 281 kcal/hour and a perceptual burden of 32.5. The patient walked 682 steps, spent 16.0 minutes upright, 10.0 minutes walking and achieved 0% in moderate-to-vigorous intensity. Conclusions As concordance between physical therapist burden and patient gait performance was low in this study, future efforts to identify gait training approaches that minimise therapist burden while maximising outcomes for the patient with lateropulsion are necessary for the health of both. Implications for practice Physical therapists may consider advanced technology use such as overground robotic exoskeletons to reduce the burden during the provision of gait training for patients with lateropulsion. Gait training performance of patients with lateropulsion may vary across different gait training approaches with low correspondence to therapist burden.

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Islam, Niaz, Gordon T. Plant et James F. Acheson. « Saccadic lateropulsion or ipsipulsion ». Acta Ophthalmologica 86, n^o 6 (26 août 2008) : 688–89. http://dx.doi.org/10.1111/j.1600-0420.2007.01040.x.

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Naoi, Tameto, Yuko Nakamura, Tomoaki Kameda, Ayako Ando et Tadataka Kawakami. « Imaging of ocular lateropulsion ». Neurology and Clinical Neuroscience 4, n^o 1 (6 août 2015) : 37. http://dx.doi.org/10.1111/ncn3.12019.

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Thèses sur le sujet "Lateropulsion"

Birnbaum, Melissa Ann. « Measuring lateropulsion following stroke in the clinical setting : exploring the measurement, nature and recovery of lateropulsion using Wii technologies and clinical measures ». Thesis, Curtin University, 2021. http://hdl.handle.net/20.500.11937/86725.

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This thesis explored the measurement of lateropulsion following stroke in clinical research, extending our knowledge about the nature and recovery of this postural control disorder. The thesis focused on three areas: (1) the measurement properties of clinical lateropulsion and sitting balance measures in stroke survivors; (2) the use of Wii Balance Board(s) as an instrumented postural control measure in sitting and standing in individuals with lateropulsion; and (3) the longer-term outcomes of individuals with lateropulsion.

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Chapitres de livres sur le sujet "Lateropulsion"

Straube, A., C. Helmchen, V. R. Robinson, A. Fuchs et U. Büttner. « Is Saccadic Lateropulsion in Wallenberg’s Syndrome Caused by a Cerebellar or a Brain-Stem Lesion ? » Dans Brain-Stem Localization and Function, 69–73. Berlin, Heidelberg : Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78172-8_7.

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Actes de conférences sur le sujet "Lateropulsion"

Tsatsis, Christopher G., Katherine E. Rice, Vera Protopopova, Darius Ramos, Jigar Jadav, Jean F. Coppola, Meg Broderick et David Putrino. « Lateropulsion rehabilitation using Virtual Reality for stroke patients ». Dans 2017 IEEE Long Island Systems, Applications and Technology Conference (LISAT). IEEE, 2017. http://dx.doi.org/10.1109/lisat.2017.8001960.

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Ximenes, Marcelo Tognato, Dan Mohamed Salman, Francisco Tomaz Meneses de Oliveira et Rubens José Gagliardi. « Downbeat Nystagmus Secondary to Chiari I Malformation ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.374.

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Context: Downbeat nystagmus are characterized by the rapid phase in the down direction in primary position of the eyes, in the most cases represents a cerebellar dysfunction, typically with injuries involving vestibulocerebellum (flocculus, paraflocculus, nodule and uvula), although most cases are due to primary lesions in the brain stem, usually involving paramedian tracts. This type of nystagmus is characteristic of craniocervical abnormalities, such as Chiari 1 malformation, also being seen in a wide variety of cerebellar diseases, including degenerative etiologies, toxic-metabolic and ischemic injury. Case report: A 45-year-old woman, who presented with dizziness started 6 years ago, related to rapid head movement, with progressive worsening, leading to difficulty in walking and a tendency to fall to the right, associated with mild to moderate intensity occipitonuchal headache, with improvement at rest and horizontal decubitus. Neurological examination showed downbeat nystagmus, intent tremor in the finger-to-nose-test, worse on the right arm, Romberg test presentes with anteroposterior and lateral instability, in addition to Tanden with bilateral lateropulsion, without other relevant changes. The magnetic resonance imaging showed platybasia and vertebrobasilar invagination, Chiari type 1 malformation with tonsillar herniation, without signs of cervical hydrosyringomyelia, signs of atrophy of both cerebellar hemispheres, a nonspecific nodule in the subcortical region of the left precuncle. Conclusion: The case described above is relevant to demonstrate that even in cases of acute changes in adulthood, screening for anatomical malformations in the posterior fossa should be considered.

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