Littérature scientifique sur le sujet « Lateral hypothalamu »
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Articles de revues sur le sujet "Lateral hypothalamu"
Wilent, W. Bryan, Michael Y. Oh, Catherine Buetefisch, Julian E. Bailes, Diane Cantella, Cindy Angle et Donald M. Whiting. « Mapping of microstimulation evoked responses and unit activity patterns in the lateral hypothalamic area recorded in awake humans ». Journal of Neurosurgery 115, no 2 (août 2011) : 295–300. http://dx.doi.org/10.3171/2011.3.jns101574.
Texte intégralTake, S., D. Uchimura, Y. Kanemitsu, T. Katafuchi et T. Hori. « Interferon-alpha acts at the preoptic hypothalamus to reduce natural killer cytotoxicity in rats ». American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 268, no 6 (1 juin 1995) : R1406—R1410. http://dx.doi.org/10.1152/ajpregu.1995.268.6.r1406.
Texte intégralYuan, C. S., et W. D. Barber. « Interactions of gastric vagal and peripheral nerves on single neurons of lateral hypothalamus in the cat ». American Journal of Physiology-Gastrointestinal and Liver Physiology 271, no 5 (1 novembre 1996) : G858—G865. http://dx.doi.org/10.1152/ajpgi.1996.271.5.g858.
Texte intégralGutterman, D. D., A. C. Bonham, G. F. Gebhart, M. L. Marcus et M. J. Brody. « Connections between hypothalamus and medullary reticular formation mediate coronary vasoconstriction ». American Journal of Physiology-Heart and Circulatory Physiology 259, no 3 (1 septembre 1990) : H917—H924. http://dx.doi.org/10.1152/ajpheart.1990.259.3.h917.
Texte intégralYuan, C. S., et W. D. Barber. « Hypothalamic unitary responses to gastric vagal input from the proximal stomach ». American Journal of Physiology-Gastrointestinal and Liver Physiology 262, no 1 (1 janvier 1992) : G74—G80. http://dx.doi.org/10.1152/ajpgi.1992.262.1.g74.
Texte intégralShimizu, Toru, Kevin Cox, Harvey J. Karten et Luiz R. G. Britto. « Cholera toxin mapping of retinal projections in pigeons (Columba livia), with emphasis on retinohypothalamic connections ». Visual Neuroscience 11, no 3 (mai 1994) : 441–46. http://dx.doi.org/10.1017/s0952523800002376.
Texte intégralBonham, A. C., D. D. Gutterman, J. M. Arthur, M. L. Marcus, G. F. Gebhart et M. J. Brody. « Electrical stimulation in perifornical lateral hypothalamus decreases coronary blood flow in cats ». American Journal of Physiology-Heart and Circulatory Physiology 252, no 3 (1 mars 1987) : H474—H484. http://dx.doi.org/10.1152/ajpheart.1987.252.3.h474.
Texte intégralMeylakh, Noemi, Kasia K. Marciszewski, Flavia Di Pietro, Vaughan G. Macefield, Paul M. Macey et Luke A. Henderson. « Altered regional cerebral blood flow and hypothalamic connectivity immediately prior to a migraine headache ». Cephalalgia 40, no 5 (12 mars 2020) : 448–60. http://dx.doi.org/10.1177/0333102420911623.
Texte intégralMondal, Muhtashan S., Masamitsu Nakazato et Shigeru Matsukura. « Orexins (hypocretins) : novel hypothalamic peptides with divergent functions ». Biochemistry and Cell Biology 78, no 3 (2 avril 2000) : 299–305. http://dx.doi.org/10.1139/o00-022.
Texte intégralIshii, Yuko, et Sebastien G. Bouret. « Embryonic Birthdate of Hypothalamic Leptin-Activated Neurons in Mice ». Endocrinology 153, no 8 (23 mai 2012) : 3657–67. http://dx.doi.org/10.1210/en.2012-1328.
Texte intégralThèses sur le sujet "Lateral hypothalamu"
Kosse, C. L. « Functional properties of GAD65 neurons in the lateral hypothalamus ». Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10044863/.
Texte intégralCarus-Cadavieco, Marta. « Coordination of innate behaviors by GABAergic cells in lateral hypothalamus ». Doctoral thesis, Humboldt-Universität zu Berlin, 2018. http://dx.doi.org/10.18452/19135.
Texte intégralLateral hypothalamus (LH) is crucial for regulation of innate behaviors. However, it remained unknown whether and how temporal coordination of hypothalamic neuronal populations regulates behavioral transitions. This work combined optogenetics with neuronal recordings in behaving mice. LHVgat cells were optogenetically identified. LHVgat neurons increased firing rates upon transitions from non-REM (NREM) sleep to wakefulness, and their optogenetic stimulation during NREM sleep induced a fast transition to wakefulness. LHVgat cells project to the reticular thalamic nucleus (RTN). Optogenetic activation of LHVgat terminals in the RTN exerted a strong frequency-dependent inhibition of RTN cells and replicated state-dependent changes in RTN neurons activity. Recordings of LH neurons during exploration revealed that 65% of LH neurons increased their activity upon the onset of locomotion. Top-down forebrain innervation of LH is provided, to a great extent, by inhibitory inputs from the lateral septum (LS). During spontaneous exploration in a free-feeding model, LS and LH displayed prominent gamma oscillations (30-90 Hz) which entrained neuronal activity within and across the two regions. Optogenetic gamma-frequency stimulation of somatostatin-positive GABAergic projections to LH facilitated food-seeking, and increased the probability of entering the food zone. LS inhibitory input enabled separate signaling by LH neurons according to their feeding-related activity, making them fire at distinct phases of the gamma oscillation. In contrast to increased food intake during optogenetic stimulation of LHVgat cells, food intake during gamma-rhythmic LS-LH stimulation was not changed. Overall this works provides new insight into the function of LH circuitry, that employs signalling at different time scales, which, in coordination with upstream and downstream circuits, regulates transitions between innate behaviors.
Boucher-Thrasher, Annette. « Evidence for anatomical connectivity between lateral hypothalamic and lateral preoptic area brain stimulation sites ». Thesis, University of Ottawa (Canada), 1987. http://hdl.handle.net/10393/5246.
Texte intégralScollon, Jennifer Margaret. « The function of the lateral hypothalamus with regard to gustatory and reward related processes ». Thesis, University of St Andrews, 1999. http://hdl.handle.net/10023/14701.
Texte intégralOh, Jeremy Jaehwan. « Posterior lateral hypothalamus stimulation confirms brain reward site stimulation-induced hyperalgesia ». Thesis, Boston University, 2012. https://hdl.handle.net/2144/12548.
Texte intégralPain is known as one of the most important physical sensations that contribute to animal survival. Kornetsky and colleagues (201 0) established mesencephalic reticular formation (MRF) and midbrain forebrain bundle (MFB) thresholds separately and found that simultaneous stimulation of the sites significantly lowered MRF escape thresholds. They also found that the simultaneous stimulation of the sites eliminated the analgesic effect--that is, raising the MRF threshold--of morphine. In an effort to better understand the interaction between rewarding brain stimulation and nociceptive pain, this study expands upon the previous experiment by implanting an electrode in the ventral tegmental area (VTA). However, during the post-mortem histological review, it was found that the rewarding electrodes were implanted in the posterior lateral hypothalamus (PLH), not in the VTA. Our overall results of this experiment demonstrate similar findings as the previous study (Kornetsky et al., 2010). The simultaneous stimulation of the MRF and PLH significantly lowered MRF escape thresholds and eliminated the analgesic effects of morphine. A very low, subthreshold intensity of stimulation to the PLH yielded significantly increased sensitivity to MRF stimulation. Furthermore, the results show that morphine did not lower BSE threshold, when the MRF was simultaneously stimulated with the PLH. An examination of the rewarding electrode placements found in the simultaneous MRF and MFB stimulation experiment by Kornetsky and colleagues (2010) also show rewarding electrode in the PLH, which corroborates with the results in this experiment.
Morutto, Sara Lidia. « Role of the perifornical region of the lateral hypothalamus in appetitive conditioning ». Thesis, University of York, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288040.
Texte intégralVenner, Anne. « The electrical activity of lateral hypothalamic neurons and its regulation by nutrients and ethanol ». Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610286.
Texte intégralNieh, Edward H. (Edward Horng-An). « Lateral hypothalamic control of motivated behaviors through the midbrain dopamine system ». Thesis, Massachusetts Institute of Technology, 2016. http://hdl.handle.net/1721.1/106440.
Texte intégralCataloged from PDF version of thesis.
Includes bibliographical references (pages 209-231).
The lateral hypothalamus and ventral tegmental area are two brain regions that have long been known to be involved in processing reward and the control of feeding behaviors. We continue work in this area by identifying the functional connectivity between these two regions, providing evidence that LH neurons projecting to the VTA encode conditioned responses, while LH neurons innervated by the VTA encode conditioned and unconditioned stimuli. Activation of the LH-VTA projection can increase compulsive sugar seeking, while inhibition of the projection can suppress this behavior without altering normal feeding due to hunger. We can separate this projection into the GABAergic and glutamatergic components, and we show that the GABAergic component plays a role in promoting feeding and social interaction by increasing motivation for consummatory behaviors, while the glutamatergic component largely plays a role in the suppression of these behaviors. Finally, we show that activation of the GABAergic component causes dopamine release downstream in the nucleus accumbens via disinhibition of VTA dopamine neurons through VTA GABA neurons. Together, these experiments have profoundly elucidated the functional roles of the individual circuit components of the greater mesolimbic dopamine system and provided potential targets for therapeutic intervention of overeating disorders and obesity..
by Edward H. Nieh.
Ph. D. in Neuroscience
Martianova, Ekaterina. « The role of the lateral hypothalamic neural outputs in motivated behaviour ». Doctoral thesis, Université Laval, 2020. http://hdl.handle.net/20.500.11794/67902.
Texte intégralUnderstanding how brain processes information is the one of the most intriguing questions that modern neuroscience faces. The current study aims to characterize how the lateral hypothalamus (LH) processes information to downstream brain targets to guide proper behavioral responses. The LH is a brain area that regulates vital behaviors such as autonomic and endocrine functions, homeostatic balance, regulation of metabolism, and sleep-wake cycles. Moreover, recent studies point out its importance in aversive and appetitive processing. The LH sends neural projections to many brain nuclei known to process signals that play important roles to guide and orchestrate proper behavior responses. Tracing experiments demonstrated that three important brain nuclei receive significant inputs from the LH, the lateral habenula (LHb), the ventral tegmental area (VTA), and the dorsal raphe nucleus (DRN). The LHb is the main disappointment center of the brain: its activity specifically increases when an animalis presented an aversive stimuli or in the absence of expected rewards, playing an important role in signaling punishment prediction error. The dopaminergic VTA is the main brain reward center. It plays important roles in reward-value encoding, reinforcement learning and motivation. The DRN is the main serotonin center playing and important role in emotion processing and adaptive responses. To specifically examine the contribution of LH neural outputs in freely moving mice, we used an advanced calcium imaging technique – fiber photometry system. Fiber photometry is a powerful approach that combines genetically encoded calcium indicators and multimode optical fibers to monitor neuronal activity in freely moving animals, which is critical to understand how specific groups of neurons play in directing or responding to an action or a stimulus. In the first chapter, I present a protocol that was developed for reliable detection of calcium signal using a camera-based multi-fiber photometry system. The protocol details the components of a multi-fiber photometry system, a method to access deep brain structures to deliverand collect light, and a method to account for motion artifacts before and during recordings. Additionally, I present an algorithm for processing of recorded signals that accounts common sources of artefacts that are inevitable during recordings. In the second chapter, I present results of the investigation of the functional role of three LH outputs to the DRN, VTA, and LHb. Using the protocol described in the first chapter, activity in the LH→DRN, LH→ VTA and LH→LHb pathways were simultaneously recorded during motivated responses in aversive and appetitive contexts. We found that these three LH neural outputs increased activity with aversive stimuli and cues predicting them. The neural activity also increased at onsets of spontaneous motivated behavior responses and decreased duringbehavioral immobility. Independent optogenetic activation of axon terminals in LHb, VTA,or DRN was sufficient to increase mobility, but had different effects in other behavioural tests. Altogether, we propose that LH sends complementary signals to the downstream targets to process information engaged in motivated behaviors. In the annex, I present a data analysis python package that was developed to process all fiber photometry recordings presented in the current study. The package allow to combine, store, and analyze recordings from multiple mice, trials, and different experiments with various measurements, behavioural events, and stimuli in a standardized way.
Clark, Judith. « Examinations of the nature of the deficits induced by n-methyl-D-aspartic acid lesions of the rat lateral hypothalamus ». Thesis, University of St Andrews, 1990. http://hdl.handle.net/10023/14703.
Texte intégralLivres sur le sujet "Lateral hypothalamu"
Kurt, Gizem, Hillary L. Woodworth, Gina M. Leinninger, D. Neil Granger et Joey P. Granger. Lateral Hypothalamic Control of Energy Balance. Morgan & Claypool Life Science Publishers, 2017.
Trouver le texte intégralKurt, Gizem, Hillary L. Woodworth, Gina M. Leinninger, D. Neil Granger et Joey P. Granger. Lateral Hypothalamic Control of Energy Balance. Morgan & Claypool Life Science Publishers, 2017.
Trouver le texte intégralKurt, Gizem, Hillary L. Woodworth, Gina M. Leinninger, D. Neil Granger et Joey P. Granger. Lateral Hypothalamic Control of Energy Balance. Morgan & Claypool Life Science Publishers, 2017.
Trouver le texte intégralTononi, Giulio, et Chiara Cirelli. The Neurobiology of Sleep. Sous la direction de Dennis S. Charney, Eric J. Nestler, Pamela Sklar et Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0011.
Texte intégralVenner, Anne, et Patrick M. Fuller. An overview of sleep–wake circuitry. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0005.
Texte intégralChapitres de livres sur le sujet "Lateral hypothalamu"
Kondoh, Takashi, Tentcho Voynikov, Eiichi Tabuchi, Takashi Yokawa, Taketoshi Ono et Kunio Torii. « Responses of Lateral Hypothalamic Neurons in Lysine-Deficient Rats ». Dans Olfaction and Taste XI, 534–35. Tokyo : Springer Japan, 1994. http://dx.doi.org/10.1007/978-4-431-68355-1_222.
Texte intégralBroberger, Christian, et Tomas Hökfelt. « Transmitter-Identified Neurons and Afferent Innervation of the Lateral Hypothalamic Area ». Dans Hypocretins, 95–120. Boston, MA : Springer US, 2005. http://dx.doi.org/10.1007/0-387-25446-3_7.
Texte intégralSaper, Clifford B. « The Prehistory of Orexin/Hypocretin and Melanin-Concentrating Hormone Neurons of the Lateral Hypothalamus ». Dans Narcolepsy, 205–9. New York, NY : Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8390-9_18.
Texte intégralOno, Taketoshi, Masaji Fukuda, Hisao Nishijo et Kiyomi Nakamura. « Plasticity in Inferotemporal Cortex-Amygdala-Lateral Hypothalamus Axis during Operant Behavior of the Monkey ». Dans Cellular Mechanisms of Conditioning and Behavioral Plasticity, 149–59. Boston, MA : Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-9610-0_15.
Texte intégralTabuchi, E., T. Kondoh, T. Voynikov, T. Yokawa, T. Ono et K. Torii. « Lateral Hypothalamic Neuron Response to Application of Amino Acids in Different Nutritive Conditions ». Dans Olfaction and Taste XI, 536. Tokyo : Springer Japan, 1994. http://dx.doi.org/10.1007/978-4-431-68355-1_223.
Texte intégralArita, H., I. Kita et M. Sakamoto. « Two Distinct Descending Inputs to the Cricothyroid Motoneuron in the Medulla Originating from the Amygdala and the Lateral Hypothalamic Area ». Dans Advances in Experimental Medicine and Biology, 53–58. Boston, MA : Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1933-1_10.
Texte intégralBernabeu, Ignacio, Monica Marazuela et Felipe F. Casanueva. « General concepts of hypothalamus-pituitary anatomy ». Dans Oxford Textbook of Endocrinology and Diabetes, 71–81. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.2004.
Texte intégral« Lateral tuberal nucleus ». Dans The Human Hypothalamus - Middle and Posterior Region, 339. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-820107-7.09999-7.
Texte intégralAhmed, Rebekah M., Frederik Steyn et Luc Dupuis. « Hypothalamus and weight loss in amyotrophic lateral sclerosis ». Dans The Human Hypothalamus - Middle and Posterior Region, 327–38. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-820107-7.00020-3.
Texte intégralOjeda, Sergio R. « The Anterior Pituitary and Hypothalamus ». Dans Textbook of Endocrine Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199744121.003.0008.
Texte intégralActes de conférences sur le sujet "Lateral hypothalamu"
Henningsen, Jo B., Barbara Baldo, Maria Björkqvist et Åsa Petersén. « A54 The role of excitotoxicity for neuropathology in the lateral hypothalamus in mouse models of huntington disease ». Dans EHDN 2018 Plenary Meeting, Vienna, Austria, Programme and Abstracts. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/jnnp-2018-ehdn.52.
Texte intégralOliveira, Jefferson Borges de, Caroline Berthier Zanin, Gustavo Carreira Henriques, Maiévi Liston, Rafael Glória Zatta, Rodrigo de Faria Martins et Tatiana Pizzolotto Bruch. « Pallister-Hall Syndrome - case report ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.575.
Texte intégralShabanov, P. D., S. V. Azarenko, V. V. Lukashkova et A. A. Lebedev. « Effects of peptides orexin and ghrelin on self-stimulation of the lateral hypothalamus in rats after corticoliberin receptors blockade ». Dans II Международная конференция, посвящеенная 100- летию И.А. Држевецкой. СКФУ, 2022. http://dx.doi.org/10.38006/9612-62-6.2022.336.340.
Texte intégralLi, Guoshi, Stacy Cheng, Frank Ko, Scott L. Raunch, Gregory Quirk et Satish S. Nair. « Computational Modeling of Lateral Amygdala Neurons During Acquisition and Extinction of Conditioned Fear, Using Hebbian Learning ». Dans ASME 2006 International Mechanical Engineering Congress and Exposition. ASMEDC, 2006. http://dx.doi.org/10.1115/imece2006-15078.
Texte intégralRapports d'organisations sur le sujet "Lateral hypothalamu"
Meiri, Noam, Michael D. Denbow et Cynthia J. Denbow. Epigenetic Adaptation : The Regulatory Mechanisms of Hypothalamic Plasticity that Determine Stress-Response Set Point. United States Department of Agriculture, novembre 2013. http://dx.doi.org/10.32747/2013.7593396.bard.
Texte intégral