Littérature scientifique sur le sujet « ISGs phenotype »
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Articles de revues sur le sujet "ISGs phenotype"
Morrison, Robert J., Nicolas-George Katsantonis, Kevin M. Motz, Alexander T. Hillel, C. Gaelyn Garrett, James L. Netterville, Christopher T. Wootten et al. « Pathologic Fibroblasts in Idiopathic Subglottic Stenosis Amplify Local Inflammatory Signals ». Otolaryngology–Head and Neck Surgery 160, no 1 (16 octobre 2018) : 107–15. http://dx.doi.org/10.1177/0194599818803584.
Texte intégralSwieboda, Dominika, Erica Johnson, Ioanna Skountzou et Rana Chakraborty. « Baby’s First Macrophage : How do placental macrophages (Hofbauer Cells, HCs) contribute to immune tolerance and infection response during pregnancy ? » Journal of Immunology 202, no 1_Supplement (1 mai 2019) : 126.28. http://dx.doi.org/10.4049/jimmunol.202.supp.126.28.
Texte intégralGluchowski, Marcel, Xiaoqiong Yu, Bernard Abrenica, Samantha Yao, Joshua Kimani, Renée N. Douville, Terry Blake Ball et Ruey-Chyi Su. « Transient Increases in Inflammation and Proapoptotic Potential Are Associated with the HESN Phenotype Observed in a Subgroup of Kenyan Female Sex Workers ». Viruses 14, no 3 (25 février 2022) : 471. http://dx.doi.org/10.3390/v14030471.
Texte intégralGupta, Sarthak, Shuichiro Nakabo, Luz P. Blanco, Liam J. O’Neil, Gustaf Wigerblad, Rishi R. Goel, Pragnesh Mistry et al. « Sex differences in neutrophil biology modulate response to type I interferons and immunometabolism ». Proceedings of the National Academy of Sciences 117, no 28 (29 juin 2020) : 16481–91. http://dx.doi.org/10.1073/pnas.2003603117.
Texte intégralJurczyszak, Denise, Lara Manganaro, Sofija Buta, Conor Gruber, Marta Martin-Fernandez, Justin Taft, Roosheel S. Patel et al. « ISG15 deficiency restricts HIV-1 infection ». PLOS Pathogens 18, no 3 (25 mars 2022) : e1010405. http://dx.doi.org/10.1371/journal.ppat.1010405.
Texte intégralHancock, Meaghan H., Karen L. Mossman et James R. Smiley. « Cell Fusion-Induced Activation of Interferon-Stimulated Genes Is Not Required for Restriction of a Herpes Simplex Virus VP16/ICP0 Mutant in Heterokarya Formed between Permissive and Restrictive Cells ». Journal of Virology 83, no 17 (17 juin 2009) : 8976–79. http://dx.doi.org/10.1128/jvi.00142-09.
Texte intégralChan, Jennie, Peter Liehl, Rosane DeOliveira, Shruti Sharma, Douglas Golenbock, Maria Mota et Katherine Fitzgerald. « Dual role of type I IFN during plasmodium infection (P3056) ». Journal of Immunology 190, no 1_Supplement (1 mai 2013) : 125.6. http://dx.doi.org/10.4049/jimmunol.190.supp.125.6.
Texte intégralThomas, Emmanuel, Mazen Noureddin, Yaron Rotman et T. Jake Liang. « Mechanism of induction and genotype-phenotype correlation of IL28B and ISG expression in HCV-infected primary human hepatocytes and liver (P1383) ». Journal of Immunology 190, no 1_Supplement (1 mai 2013) : 57.3. http://dx.doi.org/10.4049/jimmunol.190.supp.57.3.
Texte intégralFoxman, Ellen F., James A. Storer, Megan E. Fitzgerald, Bethany R. Wasik, Lin Hou, Hongyu Zhao, Paul E. Turner, Anna Marie Pyle et Akiko Iwasaki. « Temperature-dependent innate defense against the common cold virus limits viral replication at warm temperature in mouse airway cells ». Proceedings of the National Academy of Sciences 112, no 3 (5 janvier 2015) : 827–32. http://dx.doi.org/10.1073/pnas.1411030112.
Texte intégralGanguly, Payal, Agata Burska, Charlotte Davis, Jehan J. El-Jawhari, Peter V. Giannoudis et Elena Jones. « Intrinsic Type 1 Interferon (IFN1) Profile of Uncultured Human Bone Marrow CD45lowCD271+ Multipotential Stromal Cells (BM-MSCs) : The Impact of Donor Age, Culture Expansion and IFNα and IFNβ Stimulation ». Biomedicines 8, no 7 (15 juillet 2020) : 214. http://dx.doi.org/10.3390/biomedicines8070214.
Texte intégralThèses sur le sujet "ISGs phenotype"
RAUS, Svjetlana. « Reversion of anti-viral status in human tumors ». Doctoral thesis, 2014. http://hdl.handle.net/11562/694361.
Texte intégralOncolytic viral therapy is becoming an interesting alternative to standard therapies for incurable diseases. Our group has demonstrated the existence of two distinguishable phenotypes based on the expression of interferon-stimulated genes (ISGs) and Virus Stress Induced Genes (VSIG) with Myxovirus-resistance-A (MxA) protein as a marker. The existence of this ISGs phenotype has been proved to block viral oncolysis and viral mediated gene expression in cancer cells. We demonstrated that this caused in vivo an acquisition of a viral resistant phenotype in recurrent tumors cured in the first place with adeno-mediated oncolytic therapies. This is a phenomenon shared in both, solid and hematological tumors as well. The understanding of the mechanism that induces the expression of this interferon-related signature became of a key importance in optimizing oncolytic and gene therapies. In order to discover the pathways involved in the tumor-acquired ISGs phenotype, we focused our attention on tumor cell lines, and in particular pancreatic cancer and multiple myeloma cancer cell lines in vitro. Pancreatic ductal adenocarcinoma represents one of the most vicious cancers, for which no effective cure is optimised. On the other hand, multiple myeloma is the most frequent hematological tumor in humans and was often used as a possible target tumor for virus based therapies. The main aim of the present work was to confirm the existence of the dualism of ISGs phenotype in different cancer models and dissect the most important interferon pathways by specifically silencing different steps that could be responsible for the downstream ISGs up-regulation. We focused our attention to NFkB pathway in PDAC cancer cells and STAT3 pathway in multiple myeloma. We conclude that ASA and curcumin treatement can effectively revert the resistance in PDAC cell lines, while only resveratrol treatment can affect the infectivity of multiple myeloma cell lines to adenoviral vectors. Those findings can be considered the rational for a combinatory therapy with the aim of increasing the efficacy of oncolytic or gene therapy adeno-based approaches that might constitute the future for curing incurable cancers as pancreatic ductal adenocarcinoma.
Chapitres de livres sur le sujet "ISGs phenotype"
Berga, Sarah L. « The Brain Phenotype in Polycystic Ovary Syndrome (PCOS) : Androgens, Anovulation, and Gender ». Dans ISGE Series, 1–12. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63650-0_1.
Texte intégralIaremenko, Oleg, et Daria Koliadenko. « CLINICAL PHENOTYPES OF SYSTEMIC LUPUS ERYTHEMATOSUS WITH REGARD TO AGE AT DISEASE ONSET ». Dans Traditional medicine and pharmacology. Achievements, innovations, and alternatives, 56–61. International Science Group, 2021. http://dx.doi.org/10.46299/isg.2021.mono.med.ii-56-61.
Texte intégralRapports d'organisations sur le sujet "ISGs phenotype"
Blum, Abraham, et Henry T. Nguyen. Molecular Tagging of Drought Resistance in Wheat : Osmotic Adjustment and Plant Productivity. United States Department of Agriculture, novembre 2002. http://dx.doi.org/10.32747/2002.7580672.bard.
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