Littérature scientifique sur le sujet « In cellulo and in vivo models »
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Articles de revues sur le sujet "In cellulo and in vivo models"
Morant, Laura, Maria-Luise Petrovic-Erfurth et Albena Jordanova. « An Adapted GeneSwitch Toolkit for Comparable Cellular and Animal Models : A Proof of Concept in Modeling Charcot-Marie-Tooth Neuropathy ». International Journal of Molecular Sciences 24, no 22 (9 novembre 2023) : 16138. http://dx.doi.org/10.3390/ijms242216138.
Texte intégralDasnur Nanjappa, Madhusudan, Anup Pandith, Svetlana Sankaran, Dorothy Priyanka Dorairaj, Anusha Anjaneya Reddy et Hari Prasad Badubanahalli Ramesh. « Recent Advancements in Developments of Novel Fluorescent Probes : In Cellulo Recognitions of Alkaline Phosphatases ». Symmetry 14, no 8 (9 août 2022) : 1634. http://dx.doi.org/10.3390/sym14081634.
Texte intégralSailer, Alexander, Franziska Ermer, Yvonne Kraus, Rebekkah Bingham, Ferdinand H. Lutter, Julia Ahlfeld et Oliver Thorn-Seshold. « Potent hemithioindigo-based antimitotics photocontrol the microtubule cytoskeleton in cellulo ». Beilstein Journal of Organic Chemistry 16 (27 janvier 2020) : 125–34. http://dx.doi.org/10.3762/bjoc.16.14.
Texte intégralThompson, John W., Omar Elwardany, David J. McCarthy, Dallas L. Sheinberg, Carlos M. Alvarez, Ahmed Nada, Brian M. Snelling, Stephanie H. Chen, Samir Sur et Robert M. Starke. « In vivo cerebral aneurysm models ». Neurosurgical Focus 47, no 1 (juillet 2019) : E20. http://dx.doi.org/10.3171/2019.4.focus19219.
Texte intégralLepore Signorile, Martina, Valentina Grossi, Candida Fasano, Giovanna Forte, Vittoria Disciglio, Paola Sanese, Katia De Marco et al. « c-MYC Protein Stability Is Sustained by MAPKs in Colorectal Cancer ». Cancers 14, no 19 (4 octobre 2022) : 4840. http://dx.doi.org/10.3390/cancers14194840.
Texte intégralMode, Selma, Maren Ketterer, Maxime Québatte et Christoph Dehio. « Antibiotic persistence of intracellular Brucella abortus ». PLOS Neglected Tropical Diseases 16, no 7 (26 juillet 2022) : e0010635. http://dx.doi.org/10.1371/journal.pntd.0010635.
Texte intégralQiu, Xudong, Seth T. Gammon, James R. Johnson, Federica Pisaneschi, Steven W. Millward, Edward M. Barnett et David Piwnica-Worms. « Apoptosis Detection in Retinal Ganglion Cells Using Quantitative Changes in Multichannel Fluorescence Colocalization ». Biosensors 12, no 9 (28 août 2022) : 693. http://dx.doi.org/10.3390/bios12090693.
Texte intégralReinhardt, Christoph, et Heiko Rühl. « Animal and Cellular Models in Thrombosis and Hemostasis ». Hämostaseologie 43, no 05 (octobre 2023) : 319–20. http://dx.doi.org/10.1055/a-2031-7975.
Texte intégralIrrera, Pietro, Miriam Roberto, Lorena Consolino, Annasofia Anemone, Daisy Villano, Victor Navarro-Tableros, Antonella Carella, Walter Dastrù, Silvio Aime et Dario Livio Longo. « Effect of Esomeprazole Treatment on Extracellular Tumor pH in a Preclinical Model of Prostate Cancer by MRI-CEST Tumor pH Imaging ». Metabolites 13, no 1 (28 décembre 2022) : 48. http://dx.doi.org/10.3390/metabo13010048.
Texte intégralKuhn, Deborah J., Qing Chen, Peter M. Voorhees, John S. Strader, Kevin D. Shenk, Congcong M. Sun, Susan D. Demo, Mark K. Bennett, Fred W. van Leeuwen et Robert Z. Orlowski. « The Novel, Irreversible Proteasome Inhibitor PR-171 Demonstrates Potent Anti-Tumor Activity in Pre-Clinical Models of Multiple Myeloma, and Overcomes Bortezomib Resistance. » Blood 108, no 11 (16 novembre 2006) : 3461. http://dx.doi.org/10.1182/blood.v108.11.3461.3461.
Texte intégralThèses sur le sujet "In cellulo and in vivo models"
Fischer, Jared Michael. « Mouse Models of Mutation in vivo ». University of Cincinnati / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1227214862.
Texte intégralPasseri, Elodie. « Use of nanoliposomes rich in omega-3 for the prevention of neurodegenerative diseases : bioavailability in vivo and in cortical neurons ». Electronic Thesis or Diss., Université de Lorraine, 2021. https://docnum.univ-lorraine.fr/ulprive/DDOC_T_2021_0337_PASSERI.pdf.
Texte intégralAlzheimer's disease (AD) is the most common cause of neurodegenerative disease and represents a major public health issue worldwide. Many therapeutic strategies have been explored for several decades, however, there is still no curative treatment and the priority remains prevention. In this thesis work, we focused on the preventive approach based on lipids, in particular omega-3 polyunsaturated fatty acids (n-3 PUFAs). N-3 PUFAs play an important role in the development, maintenance and function of the brain, and they have been the subject of particular interest in the prevention of cognitive deficits associated with neurodegenerative diseases. The objective of this work is to study the functionality and bioavailability of nanoliposomes (NL) rich in n-3 PUFAs, in order to assess their neuroprotective potential to develop new preventive strategies in aging-related diseases such as AD. To do this, a green extraction technique was used to prepare NL, from natural resources from salmon lecithin rich in n-3 PUFA. This thesis is based on three parts. The first part is devoted to a bibliographical review of new techniques to facilitate the access of molecules to the brain. NL shows a promising role, being able to improve the transport of molecules across the blood-brain barrier and reach relevant brain regions. In the second part of this work, the bioavailability of NL rich in n-3 PUFA was studied in a primary culture of cortical neuronal cells from rat embryos and in a mouse model. This study shows for the first time the brain bioavailability of NL rich in n-3 PUFA in in vitro and in vivo models. Finally, in the third part of this thesis, the physicochemical properties and transfer mechanisms of NL were studied in a primary culture of cortical neurons. These results provide new information on the interaction between NL and neurons and are promising with regards to the use of NL rich in n-3 PUFA, opening up new possibilities in the development of preventive and neuroprotective therapeutic strategies for neurodegenerative diseases such as AD
Rose, Nicolas. « New ex vivo models to study the mechanical interplay between muscle cells and their microenvironment ». Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS440.
Texte intégralEx vivo systems are under increasing interest for bioethical, legal and financial stakes. To study muscle function and regeneration, medical research requires functional analysis platforms for mature myotubes and unconventional muscle stem cells (MuSCs) culture tools integrating mechanical consraints. During my doctoral works, a 3D myotube culture chip with contraction monitoring capacity has been developed. Combining photopatterning technology and microsubstrate design allowed to obtain high culture yield in controlled physical and chemical microenvironment. Spontaneous contractions of human primary myoblast-derived 3D myotubes has been observed, their generated forces measured and their contraction pattern characterised. The impact of 3D culture on nuclear morphology has been analysed, confirming organizational similarities betaween obtained 3 myotubes and in vivo myofibers. Moreover, LMNA-related Congenital Muscular Dystrophy has been modelled in 3D mutants myotubes displaying typical laminopathy phenotype. Finally, MuSCs has ben cultured on hydrogels, demonstrating the effect of niche elasticity variations on MuSCs activation during muscle regeneration
BAZZINI, CHIARA. « STUDY OF MOLECULAR MECHANISMS AND NEW STRATEGIES AGAINST A CYTOTOXICITY AND NEUROINFLAMMATION IN EX VIVO CELLULAR MODELS FROM ALZHEIMER’S DISEASE PATIENTS ». Doctoral thesis, Università degli Studi di Milano-Bicocca, 2021. http://hdl.handle.net/10281/306480.
Texte intégralAlzheimer's disease (AD) is a major public health concern and has been identified as a priority for research in Life Science. The two core pathological hallmarks of AD are extracellular amyloid plaques and intracellular neurofibrillary tangles which underlie microglial and neuronal damage, neuroinflammation and cognitive impairment. Soluble oligomers are the most toxic species of β-amyloid (Aβ) and interact with several protein kinases such as Ras/MAPK and PI3K/AKT pathways, which regulate many cellular processes and cognitive functions. These pathways mediate Aβ toxicity, regulating some molecular mechanisms involved in neuronal degeneration such as cytoskeletal impairment, glutamate excitotoxicity and neuroinflammation. In the last years much attention has been focused on the potential role of natural compounds as neuroprotective agents. Hop (Humulus Lupulus) contains flavonoids, aromatic molecules which have antioxidant, anti-inflammatory and anti-atherogenic properties. In fact, hop extract has anti-aggregating effects on Aβ, and it seems to prevent its production in cultured cells. Aβ induces also the activation of the pattern recognition receptor Nod-like receptor protein 3 (NLRP3) inflammasome complex in microglia and the consequent release of proinflammatory cytokines, playing a pivotal role in AD-associated neuroinflammation. NLRP3 activation results in the release of inflammatory mediators, including ASC protein complexes (ASC specks), IL-1β and IL-18, that facilitate Aβ deposition and neuroinflammation in a self-feeding pathogenic loop. Since specific therapeutical strategies are still lacking, the dampening of the inflammasome assembly and activation could be a new strategy for AD. The overall focus of this study is to investigate molecular mechanisms involved in neurodegenerative diseases and in neuroinflammation, using peripheral ex vivo cellular models from AD, to check new potential therapeutical targets. In order to characterize the complex interactions among Aβ, MAPK and AKT signaling, we used fibroblasts from sporadic AD patients with different disease severity. To evaluate any molecular mechanisms that could prevent or modulate Aβ-induced toxicity, the potential cytoprotective effects of Hop extract and related intracellular signaling were also investigated. Fibroblasts provide a useful cellular model for studying AD, since they could be differentiated into patient-specific neural cell lines, using iPSC technologies. Moreover, particular interest was given to NLRP3-inflammasome activation pathway. We investigated the involvement of NLRP3 inflammasome activation on intracellular pathways and their downstream targets, using a combination of in vitro studies and patient-derived samples. In particular, we used macrophage-derived THP-1 human monocytes and peripheral blood mononuclear cells (PBMC)-derived monocytes from healthy control (HC) subjects and AD patients, to analyse phagocytosis, autophagy and apoptosis modulation and the effects of the nucleoside reverse transcriptase inhibitor Stavudine (D4T), that reduces NLRP3 inflammasome activation blocking the purinergic receptor P2X7R. Furthermore, we analyzed the NLRP3 inflammasome pathway and the role of the selective NLRP3 inhibitor CRID3, to compare the effects of inflammasome inhibition through two different mechanisms. At this purpose, HC and AD-derived monocytes were differentiated into microglia-like cells (MDMIs) and characterized for myeloid surface and intracellular proteins expression. Key microglia functions such as inflammatory cytokines release, Aβ phagocytosis and degradation were evaluated upon exposure to NLRP3 inflammasome activators with or without CRID3. MDMIs reflected many features of microglia and, as fibroblasts-derived iPSCs, they are attractive cellular models helpful to understand AD pathogenesis, identify therapeutic targets and allow large-scale drug screening of the novel therapeutic candidates.
Zangrossi, Manuela. « Study of the extra-telomeric functions of telomerase in in vitro and in vivo models ». Doctoral thesis, Università degli studi di Padova, 2018. http://hdl.handle.net/11577/3426233.
Texte intégralIl mantenimento dei telomeri, necessario per la proliferazione illimitata delle cellule tumorali, è esercitato dalla telomerasi, un complesso ribonucleoproteico contenente una trascrittasi inversa specializzata, codificata dal gene TERT, che utilizza un templato ad RNA per sintetizzare nuove sequenze telomeriche, svolgendo quindi un ruolo critico nella formazione e nella progressione dei tumori. TERT viene infatti solitamente represso in normali cellule somatiche, mentre è rilevabile nella maggior parte dei tumori. Studi recenti hanno suggerito che TERT è coinvolto in altre funzioni cellulari e può contribuire alla carcinogenesi anche attraverso meccanismi indipendenti dal mantenimento dei telomeri, quindi la sua inibizione potrebbe rappresentare una strategia promettente per migliorare il trattamento antitumorale, al di là dell’effetto sui telomeri. I possibili effetti terapeutici di BIBR1532 (BIBR), un inibitore specifico del TERT, sono stati valutati in diversi contesti cellulari, ma non sono attualmente disponibili dati ottenuti su modelli di neoplasie delle cellule B sia associate al virus di Epstein-Barr (EBV) che virus-indipendenti. Lo scopo di questo studio era di caratterizzare gli effetti biologici dell'inibizione di TERT a breve termine da parte del BIBR su linee cellulari linfoblastoidi immortalizzate da EBV (LCL) e su modelli in vitro di linfoma di Burkitt (BL); inoltre, sono stati studiati gli effetti del trattamento con BIBR a breve termine in vivo negli embrioni di zebrafish. I risultati ottenuti hanno dimostrato che l'inibizione a breve termine di TERT da parte di BIBR, in modelli in vitro di tumori delle cellule B, ha portato a una diminuzione della proliferazione cellulare, all'accumulo di cellule nella fase S e infine all'aumento dell'apoptosi. L'arresto del ciclo cellulare e l'apoptosi, conseguenti all'inibizione di TERT a breve termine, erano associati e probabilmente dipendenti dall'attivazione della risposta al danno del DNA, come evidenziato dall’aumento dei livelli di γH2AX e dall'attivazione dei pathway di ATM e ATR. L’analisi della media e del range di lunghezza dei telomeri e dei foci di danno al DNA ha indicato che la risposta al danno attivata in seguito all’inibizione TERT a breve termine non era legata a disfunzioni telomeriche, suggerendo quindi che TERT, oltre a stabilizzare il telomero, può proteggere il DNA tramite meccanismi telomero-indipendenti. In particolare, LCL-TERT positive trattate con BIBR in combinazione con fludarabina o ciclofosfamide hanno mostrato un aumento significativo del numero di cellule apoptotiche rispetto a quelle trattate con agenti chemioterapici da soli. In accordo con i risultati in vitro, l'inibizione a breve termine di Tert da parte del BIBR in embrioni di zebrafish ha ridotto la proliferazione cellulare, indotto un accumulo di cellule nella fase S, aumentato il tasso di apoptosi e innescato l'attivazione della risposta al danno al DNA. Questi effetti non erano legati a disfunzioni telomeriche, poiché il range di lunghezza dei telomeri non era influenzato dal trattamento a breve termine con BIBR e i foci di danno al DNA erano distribuiti casualmente, piuttosto che localizzati in modo specifico sui telomeri. Tutti questi effetti erano specificamente associati all'inibizione di Tert poiché il trattamento con BIBR non mostrava alcun effetto negli embrioni di zebrafish Tert-negativi. Nel complesso questi dati dimostrano che l'inibizione del TERT compromette la proliferazione cellulare e induce effetti pro-apoptotici non associati a disfunzioni telomeriche, rafforzando il concetto che TERT esercita di per sé funzioni pro-tumorali indipendenti dalla lunghezza del telomero e quindi supportando l'introduzione di inibitori di TERT per integrare le attuali modalità di trattamento antitumorale.
Ronayne, Rachel E. « Human Ependymin-1 Neurotrophic Factor Mimetics Reduce Tau Phosphorylation and Cellular Apoptosis in Vitro and in Vivo in Alzheimer’s Disease Models ». Digital WPI, 2008. https://digitalcommons.wpi.edu/etd-theses/1018.
Texte intégralLu, Yen-Zhen. « The effects of 670nm light on retinal Müller cell gliosis following retinal stress or injury : exploring the underlying cellular mechanisms using in vivo and in vitro models ». Phd thesis, Canberra, ACT : The Australian National University, 2018. http://hdl.handle.net/1885/154729.
Texte intégralToutain, Hervé. « Développement et caractérisation de modèles expérimentaux pour l'étude ex-vivo et in-vitro de la cellule tubulaire proximale de rein de lapin ». Rouen, 1989. http://www.theses.fr/1989ROUES036.
Texte intégralSibeko, Sengeziwe. « The impact of macrophage inflammatory protein-3 alpha and other innate immune markers on susceptibility/resistance to HIV infection in the female genital tract mucosa using cellular and ex vivo tissue models ». Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:a7ecf529-b94e-492f-8374-675cb495ef05.
Texte intégralBody, Simon. « Physiopathologie du lymphome à cellules du manteau : de la mécanistique aux modèles précliniques ». Thesis, Normandie, 2017. http://www.theses.fr/2017NORMC419/document.
Texte intégralMantle cell lymphoma (MCL) is a mature malignant hemopathy, belonging to the non-Hodgkin's lymphoma family. The MCL is characterized by the translocation t(11;14)(q13;q32) which causes an aberrant expression of cyclin D1. It is a rare disease but at high risk of relapse, and it is most often incurable due to the appearance of chemoresistant clones. The acquisition of resistance is intimately linked to the interactions between the tumor cells and their microenvironment. In order to mimic, in the most relevant way, these interactions, we have implemented a mouse xenograft model using the MCL cell lines JeKo1, REC1, Z138 and Granta-519 which we have modified so that they express a fluorophore (GFP or m-cherry) and / or the gene encoding the luciferase. After injection to the mice of the luciferase substrate, luciferin, we are able to follow over time the tumor progression. We can also assess the degree of tumor infiltration in bone marrow, spleen, brain and blood after euthanasia of animals, by flow cytometry and immunocytochemistry. This model allowed us to show the therapeutic interest of an inhibitor of exportin 1 (XPO1): the KPT 330 (or selinexor) which is able to contain cyclin D1 only on the nuclear level. We have shown that the subcellular localization of cyclin D1 is mainly cytoplasmic in some LCM (2/7) cell lines and in a number of patients (6/42, 14%), and is associated with a high potential Invasion, migration and an aggressive phenotype. Moreover, thanks to this model, we have been able to objectify the in vivo lack of efficacy of agonists to β-type estrogen receptors (ER β). These receptors, present on B lymphocytes, were thought to inhibit cell proliferation and cause cell death by apoptosis. The use of two ER β agonists, diarylpropionitrile (DPN) and ERB-041 showed an absence of effect of these molecules, when the tumor cells are in contact with their microenvironment. On the other hand, in order to better understand the mechanisms of resistance to chemotherapies, we studied the resistance of the REC-1 cell line treated with genotoxic agents. We have shown that this line has an abnormality of cyclin D1 degradation associated with decreased activity of the 26S proteasome. Finally, we have shown in preliminary work that the fused in sarcoma protein (FUS) could, when associated with cyclin D1, be able to regulate the repair pathways of DNA damage. Abnormalities of these pathways induce a great genetic instability responsible for the escape of tumors to treatments, the targeting of FUS could therefore be of therapeutic interest.Taken as a whole, these results reinforce or invalidate the interest of certain therapeutic targets in the hope of continuing to improve the management of patients. They also provide a tool for evaluating new molecules in a murine model that takes into account the interactions between the tumor cell and its microenvironment
Livres sur le sujet "In cellulo and in vivo models"
Ferrick, David A. Transgenic mice as a in vivo model for self reactivity. Austin : R.G. Landes Co., 1994.
Trouver le texte intégralMorgan, Douglas W., et Lisa A. Marshall, dir. In Vivo Models of Inflammation. Basel : Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-7775-6.
Texte intégralStevenson, Christopher S., Lisa A. Marshall et Douglas W. Morgan, dir. In Vivo Models of Inflammation. Basel : Birkhäuser Basel, 2006. http://dx.doi.org/10.1007/978-3-7643-7520-1.
Texte intégralStevenson, Christopher S., Lisa A. Marshall et Douglas W. Morgan, dir. In Vivo Models of Inflammation. Basel : Birkhäuser Basel, 2006. http://dx.doi.org/10.1007/978-3-7643-7760-1.
Texte intégral1974-, Stevenson Christopher S., Morgan Douglas W. 1951- et Marshall Lisa A. 1954-, dir. In vivo models of inflammation. 2e éd. Basel : Birkhäuser, 2006.
Trouver le texte intégral1951-, Morgan Douglas W., et Marshall Lisa A. 1954-, dir. In vivo models of inflammation. Basel : Birkhauser Verlag, 1999.
Trouver le texte intégralNtziachristos, Vasilis. Textbook of in vivo imaging in vertebrates. Chichester, West Sussex : J. Wiley, 2007.
Trouver le texte intégralFriedman, Herman, Steven Specter et Mauro Bendinelli, dir. In vivo Models of HIV Disease and Control. Boston, MA : Springer US, 2006. http://dx.doi.org/10.1007/b135975.
Texte intégralJ, Ellis Kenneth, Eastman Jerry D et International Symposium on In Vivo Body Composition Studies (1992 : Houston, Tex.), dir. Human body composition : In vivo methods, models, and assessment. New York : Plenum Press, 1993.
Trouver le texte intégralC, Sahu Saura, dir. Hepatotoxicity : From genomics to in vitro and in vivo models. Chichester, England : John Wiley & Sons, 2007.
Trouver le texte intégralChapitres de livres sur le sujet "In cellulo and in vivo models"
Andes, David R. « In Vivo Candida Device Biofilm Models ». Dans Candida albicans : Cellular and Molecular Biology, 93–113. Cham : Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50409-4_7.
Texte intégralCwykiel, Joanna, et Maria Z. Siemionow. « Cellular Therapy Models : Ex Vivo Chimera Model by Cell Fusion ». Dans Plastic and Reconstructive Surgery, 593–603. London : Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6335-0_72.
Texte intégralFlaherty, Lynne, John M. Harlan et Robert K. Winn. « Blockade of Leukocyte Adhesion in in Vivo Models of Inflammation ». Dans Cellular Adhesion, 153–66. Boston, MA : Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2466-3_9.
Texte intégralChernokal, Brea, Cailin R. Gonyea et Jason P. Gleghorn. « Lung Development in a Dish : Models to Interrogate the Cellular Niche and the Role of Mechanical Forces in Development ». Dans Advances in Experimental Medicine and Biology, 29–48. Cham : Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-26625-6_3.
Texte intégralWelsch, Clifford W. « Rodent Models To ExamineIn Vivo Hormonal Regulation of Mammary Gland Tumorigenesis ». Dans Cellular and Molecular Biology of Mammary Cancer, 163–79. Boston, MA : Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0943-7_10.
Texte intégralMazière, J. C., S. Salmon, C. Candide, C. Mazière, R. Santus, J. P. Reyftmann, P. Morlière et L. Dubertret. « Lipid Peroxidation and Cellular Functions : in Vitro Models and Relation to in Vivo Observations ». Dans Free Radicals, Lipoproteins, and Membrane Lipids, 327–42. New York, NY : Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-7427-5_31.
Texte intégralWiegand, C., J. Tittelbach, U. C. Hipler et P. Elsner. « Water-Filtered Infrared A Irradiation : From Observations in Clinical Studies to Complex In Vitro Models ». Dans Water-filtered Infrared A (wIRA) Irradiation, 203–12. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92880-3_17.
Texte intégralSchaefer, H., et W. Schalla. « Human Cutaneous Pharmacokinetics In Vivo ». Dans Skin Models, 94–102. Berlin, Heidelberg : Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70387-4_12.
Texte intégralStephens, L. C., L. Milas, K. K. Ang, K. A. Mason et R. E. Meyn. « Apoptosis In Vivo ». Dans Tumor Models in Cancer Research, 625–40. Totowa, NJ : Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-968-0_25.
Texte intégralGarcia, Kristen, Marcus Hock, Vikrant Jaltare, Can Uysalel, Kimberly J. McCabe, Abigail Teitgen et Daniela Valdez-Jasso. « Investigating the Multiscale Impact of Deoxyadenosine Triphosphate (dATP) on Pulmonary Arterial Hypertension (PAH) Induced Heart Failure ». Dans Computational Physiology, 77–90. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-05164-7_7.
Texte intégralActes de conférences sur le sujet "In cellulo and in vivo models"
Boppart, Stephen A., Wolfgang Drexler, Uwe Morgner, Franz X. Kärtner et James G. Fujimoto. « Ultrahigh Resolution and Spectroscopic OCT Imaging of Cellular Morphology and Function ». Dans In Vivo optical Imaging at the NIH. Washington, D.C. : Optica Publishing Group, 1999. http://dx.doi.org/10.1364/ivoi.1999.msi56.
Texte intégralYasubumi Sakakibara, Hirotaka Nakagawa, Yusaku Nakashima et Katsuyuki Yugi. « Implementing in vivo cellular automata using toggle switch and inter-bacteria communication mechanism ». Dans 2007 2nd Bio-Inspired Models of Network, Information and Computing Systems (BIONETICS). IEEE, 2007. http://dx.doi.org/10.1109/bimnics.2007.4610137.
Texte intégralSakakibara, Yasubumi, Hirotaka Nakagawa, Yusaku Nakashima et Katsuyuki Yugi. « Implementing in vivo Cellular Automata using Toggle Switch and Inter-Bacteria Communication Mechanism ». Dans 2nd International ICST Conference on Bio-Inspired Models of Network, Information, and Computing Systems. IEEE, 2007. http://dx.doi.org/10.4108/icst.bionetics2007.2410.
Texte intégralWilliams, Amber, et Mark Niedre. « Detection of Circulating Tumor Cells and Clusters Using a 2-Fluorophore Diffuse in Vivo Flow Cytometer ». Dans Optical Molecular Probes, Imaging and Drug Delivery. Washington, D.C. : Optica Publishing Group, 2023. http://dx.doi.org/10.1364/omp.2023.om4e.4.
Texte intégralEhlert, Jan E., Bettina Mutschler, Melanie Müller, Andreas Lingnau, Steffen Hoffmann et Michael HG Kubbutat. « Abstract 3225 : Cellular and in vivo models for the analyses of B-Raf and c-Src inhibitors ». Dans Proceedings : AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010 ; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3225.
Texte intégralMarshall, Lauren, Isabel Löwstedt, Paul Gatenholm et Joel Berry. « Prevascularized, Co-Culture Model for Breast Cancer Drug Development ». Dans ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80409.
Texte intégralBoppart, Stephen A., Gary J. Tearney, Brett E. Bouma, James G. Fujimoto et Mark E. Brezinski. « Optical Coherence Tomography of Embryonic Morphology During Cellular Differentiation ». Dans Advances in Optical Imaging and Photon Migration. Washington, D.C. : Optica Publishing Group, 1996. http://dx.doi.org/10.1364/aoipm.1996.cit231.
Texte intégralBaker, Jonathan, Genki Kimura, Yuki Nishimoto, Shyreen Hassibi, Yasuo Kizawa et Kazuhiro Ito. « The senolytic effect of Dasatinib and Quercetin on cellular senescence in COPD in vitro and in vivo models ». Dans ERS International Congress 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/13993003.congress-2023.pa4046.
Texte intégralUddin, Sardar M. Zia, et Yi-Xian Qin. « Anabolic Effects of Ultrasound as Countermeasures of Simulated Microgravity in In-Vitro and In-Vivo Functional Disuse Models ». Dans ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53796.
Texte intégralZwick, H., B. E. Stuck, W. R. Elliott, D. J. Lund, S. T. Schuschereba et G. Li. « An Animal Model for In-Vivo Characterization of Laser Induced Retinal cellular Pathology and Recovery Processes ». Dans In Vivo optical Imaging at the NIH. Washington, D.C. : Optica Publishing Group, 1999. http://dx.doi.org/10.1364/ivoi.1999.msi31.
Texte intégralRapports d'organisations sur le sujet "In cellulo and in vivo models"
Eldar, Avigdor, et Donald L. Evans. Streptococcus iniae Infections in Trout and Tilapia : Host-Pathogen Interactions, the Immune Response Toward the Pathogen and Vaccine Formulation. United States Department of Agriculture, décembre 2000. http://dx.doi.org/10.32747/2000.7575286.bard.
Texte intégralParada, Luis F. In Vivo Models of NF-1 : The Nervous System and Tumorigenesis. Fort Belvoir, VA : Defense Technical Information Center, septembre 2000. http://dx.doi.org/10.21236/ada386287.
Texte intégralParada, Luis. In Vivo Models of NF-1 : The Nervous System and Tumorigenesis. Fort Belvoir, VA : Defense Technical Information Center, septembre 1999. http://dx.doi.org/10.21236/ada391279.
Texte intégralGeisbert, Thomas W. Pathogenesis of Ebola Hemorrhagic Fever in Primate Models In Vivo and In Vitro. Fort Belvoir, VA : Defense Technical Information Center, janvier 2003. http://dx.doi.org/10.21236/ad1012627.
Texte intégralYoung, Pamela R. Advanced Imaging Approaches to Characterize Stromal and Metabolic Changes in In Vivo Mammary Tumor Models. Fort Belvoir, VA : Defense Technical Information Center, mars 2013. http://dx.doi.org/10.21236/ada580941.
Texte intégralSpiers, Donald, Arieh Gertler, Harold Johnson et James Spain. An In Vitro and In Vivo Investigation of the Diverse Biological Activities of Bovine Placental Lactogen. United States Department of Agriculture, août 1993. http://dx.doi.org/10.32747/1993.7568087.bard.
Texte intégralKopeć, Aneta, Ewa Piątkowska et Joanna Skoczylas. Sardines and sprats as the potential source of nutrients required for supporting proper function of immune system in in vitro and in vivo models. Publishing House of the University of Agriculture in Krakow, 2024. http://dx.doi.org/10.15576/repourk/2024.1.01.
Texte intégralLioy, P. J., M. Gallo, P. Georgopoulos, R. Tate et B. Buckley. Comparison of the bioavailability of elemental waste laden soils using in vivo and in vitro analytical methodology and refinement of exposure/dose models. 1998 annual progress report. Office of Scientific and Technical Information (OSTI), juin 1998. http://dx.doi.org/10.2172/13580.
Texte intégralBoisclair, Yves R., et Arieh Gertler. Development and Use of Leptin Receptor Antagonists to Increase Appetite and Adaptive Metabolism in Ruminants. United States Department of Agriculture, janvier 2012. http://dx.doi.org/10.32747/2012.7697120.bard.
Texte intégralCao, Siyang, Yihao Wei, Tiantian Qi, Peng Liu, Yingqi Chen, Fei Yu, Hui Zeng et Jian Weng. Stem cell therapy for peripheral nerve injury : An up-to-date meta-analysis of 55 preclinical researches. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, octobre 2022. http://dx.doi.org/10.37766/inplasy2022.10.0083.
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