Littérature scientifique sur le sujet « Immunology, Neutrophil »
Créez une référence correcte selon les styles APA, MLA, Chicago, Harvard et plusieurs autres
Consultez les listes thématiques d’articles de revues, de livres, de thèses, de rapports de conférences et d’autres sources académiques sur le sujet « Immunology, Neutrophil ».
À côté de chaque source dans la liste de références il y a un bouton « Ajouter à la bibliographie ». Cliquez sur ce bouton, et nous générerons automatiquement la référence bibliographique pour la source choisie selon votre style de citation préféré : APA, MLA, Harvard, Vancouver, Chicago, etc.
Vous pouvez aussi télécharger le texte intégral de la publication scolaire au format pdf et consulter son résumé en ligne lorsque ces informations sont inclues dans les métadonnées.
Articles de revues sur le sujet "Immunology, Neutrophil"
Cavallaro, Elena C., Kar-Kate Liang, Kevin D. Forsyth et Dani-Louise Dixon. « Neutrophil polarization in the airways of infants with bronchiolitis ». Journal of Immunology 198, no 1_Supplement (1 mai 2017) : 55.30. http://dx.doi.org/10.4049/jimmunol.198.supp.55.30.
Texte intégralForlow, S. Bradley, Jill R. Schurr, Jay K. Kolls, Gregory J. Bagby, Paul O. Schwarzenberger et Klaus Ley. « Increased granulopoiesis through interleukin-17 and granulocyte colony-stimulating factor in leukocyte adhesion molecule–deficient mice ». Blood 98, no 12 (1 décembre 2001) : 3309–14. http://dx.doi.org/10.1182/blood.v98.12.3309.
Texte intégralMizgerd, J. P., B. B. Meek, G. J. Kutkoski, D. C. Bullard, A. L. Beaudet et C. M. Doerschuk. « Selectins and neutrophil traffic : margination and Streptococcus pneumoniae-induced emigration in murine lungs. » Journal of Experimental Medicine 184, no 2 (1 août 1996) : 639–45. http://dx.doi.org/10.1084/jem.184.2.639.
Texte intégralHarvath, L., K. B. Yancey et S. I. Katz. « Selective inhibition of human neutrophil chemotaxis to N-formyl-methionyl-leucyl-phenylalanine by sulfones. » Journal of Immunology 137, no 4 (15 août 1986) : 1305–11. http://dx.doi.org/10.4049/jimmunol.137.4.1305.
Texte intégralWang, Jun-Xia, et Peter Nigrovic. « CD177 participates in a novel mechanism for regulating neutrophil recruitment (P3093) ». Journal of Immunology 190, no 1_Supplement (1 mai 2013) : 43.9. http://dx.doi.org/10.4049/jimmunol.190.supp.43.9.
Texte intégralWang, Guoshun, et Hang Pong Ng. « Myeloid CFTR Loss-of-function Causes Persistent Neutrophilic Inflammation in Cystic Fibrosis ». Journal of Immunology 202, no 1_Supplement (1 mai 2019) : 187.33. http://dx.doi.org/10.4049/jimmunol.202.supp.187.33.
Texte intégralGadjeva, Mihaela, Abirami Kugadas, Anastasia Petenkova, Jennifer Geddes-McAlister, Michael K. Mansour et David Sykes. « Neutrophil maturation and their response to infectious pathogens are regulated by microbiota ». Journal of Immunology 202, no 1_Supplement (1 mai 2019) : 127.22. http://dx.doi.org/10.4049/jimmunol.202.supp.127.22.
Texte intégralBorges, Leandro, Tania Cristina Pithon-Curi, Rui Curi et Elaine Hatanaka. « COVID-19 and Neutrophils : The Relationship between Hyperinflammation and Neutrophil Extracellular Traps ». Mediators of Inflammation 2020 (2 décembre 2020) : 1–7. http://dx.doi.org/10.1155/2020/8829674.
Texte intégralBrinkworth, Jessica F., Kathrine Van Etten, Priya Bhatt, Keaton McClure, Negin Valizadegan, Minkyu Woo, Suvanthee Gunasekera, Yaravi Suarez et Brian Aldridge. « Functional comparison of human and non-human primate neutrophil responses ». Journal of Immunology 202, no 1_Supplement (1 mai 2019) : 73.21. http://dx.doi.org/10.4049/jimmunol.202.supp.73.21.
Texte intégralShelite, Thomas R., Nicole L. Mendell, Donald H. Bouyer, David Hughes Walker et Lynn Soong. « The role of neutrophils during Orientia infection. » Journal of Immunology 196, no 1_Supplement (1 mai 2016) : 66.28. http://dx.doi.org/10.4049/jimmunol.196.supp.66.28.
Texte intégralThèses sur le sujet "Immunology, Neutrophil"
Eckert, Rachael. « Molecular Mechanisms of Neutrophil Migration ». NCSU, 2007. http://www.lib.ncsu.edu/theses/available/etd-10312007-134315/.
Texte intégralRochon, Yvan P. (Yvan Pierre). « Dynamics of neutrophil aggregation ». Thesis, McGill University, 1991. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=70210.
Texte intégralMacdonald, Elizabeth A. « Bovine neutrophil functionality in mastitis resistance ». Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=68211.
Texte intégralBradford, Elaine Alison. « Proposed in vitro model of neutrophil swarming in a chronic, low-level inflammatory state ». Thesis, Virginia Tech, 2019. http://hdl.handle.net/10919/102737.
Texte intégralMaster of Science
Kirsch, Richard. « Characterisation of fibrinogen and fibrin proteolysis by the neutrophil membrane ». Doctoral thesis, University of Cape Town, 1999. http://hdl.handle.net/11427/26928.
Texte intégralHoenderdos, Kim. « Modulation of neutrophil degranulation by hypoxia ». Thesis, University of Cambridge, 2015. https://www.repository.cam.ac.uk/handle/1810/247459.
Texte intégralChen, Justin. « The effect of hyperleptinemia on polymorphonuclear neutrophil-endothelial interactions / ». Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=101709.
Texte intégralMale CD1 background mice (6-8 weeks) were divided to 3 treatment groups receiving once daily ip injections (1) sham (PBS); (2) low leptin (1mug/g); (3) high leptin (5mug/g). After 7 days of treatment, intravital microscopy was used to visualize post-capillary venule microcirculation of the cremaster muscle in the scrotum. Parameters such as neutrophil rolling, rolling velocity, preadherence, and adherence, were recorded and measured to assess PMN kinetics.
High doses of leptin resulted in increased preadherence and adherence of neutrophils in post-capillary venules. Serum leptin and TNFalpha levels were found not to correlate with this observation; consequently, potential pathways through which leptin increases PMN adhesion could not be elucidated. Conceivably, excessive adhesion could adversely affect neutrophil trafficking by producing a shift towards the marginal pool, limiting their ability to appropriately home into bacterial targets. This could parallel the situation in morbid obesity where high concentrations of leptin are also observed.
Boespflug, Nicholas. « ATF3 regulates neutrophil migration in mice ». University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1382372804.
Texte intégralLin, Yongqing. « Study of neutrophil diapedesis across a bovine mammary epithelium in vitro ». Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=22761.
Texte intégralChung, Henry Hung Li. « Engineered Microenvironment for Quantitative Studies of Neutrophil Migration ». Thesis, University of Rochester, 2015. http://pqdtopen.proquest.com/#viewpdf?dispub=3686523.
Texte intégralCell migration is present in virtually all life processes, including fertilization, embryogenic development, immune response, wound healing, and tumor metastasis. To improve the treatment of diseases associated with these various life processes, it is important to understand the underlying mechanisms of cell migration involved. This often requires that we recreate the environment that leads to and supports the continuous migration of cells. Here, we present two engineering approaches toward such a goal, with the additional emphasis that cell migration can be conducted in the absence of fluid flow, a mechanical stimulus that is known to influence cell behaviors. We chose the primary human neutrophil, which is highly motile and sensitive to both fluid flow and chemoattraction, as the model cell type for all our studies.
In the first approach, we used fluid flow to create a linear and time-invariant gradient of chemoattractants to guide the migration of neutrophils. A thin and porous membrane was used to screen off the associated flow forces while still permitting the diffusion of the gradient to the neutrophils. We showed that the membrane-based system is capable of directing neutrophil migration without the bias from fluid flow, and allowed within minutes the exchange of media to label and wash the migrated neutrophils. To assess the reduction of flow forces enabled by the membrane, we developed an analytical model to predict the direction and the magnitude of flow within the system. The validity of the model was verified both experimentally and numerically with particle tracking and computational fluid mechanic (CFM) simulations. We also performed total internal reflection fluorescence (TIRF) microscopy to verify the preservation of the gradient after v its diffusion through the membrane.
In the second approach, we created immobilized gradients of the chemoattractant interleukin 8 (IL-8) and the intercellular adhesion molecule 1 (ICAM-1) in the attempt to guide neutrophil migration. A gradient of soluble factors is first established, and the resulting difference of concentration over space leads to a bias in the binding of the soluble factors unto the substrate, forming an immobilized gradient. The immobilization is mediated by a combination of different physicochemical linkages, including electrostatic attraction, protein/protein interactions, and covalent bonding. We showed through labeling with fluorescent antibody that the number of IL-8 or ICAM-1 immobilized in a given area could be controlled, and varied over distances to form different gradient profiles. We further showed that our immobilization procedure does not affect the ability of IL-8 and ICAM-1 to activate and bind the neutrophils. However, with all the immobilized gradients that we have created so far, none were able to effectively promote the directed migration of neutrophils in long distances. Additional work is therefore required to establish if an immobilized gradient of either IL-8 or ICAM-1 alone can direct the migration of neutrophils in long distances, and if it does, what are the required conditions. Currently, our efforts suggest that the membrane-based chemotaxis system is a more attainable platform for promoting a directed migration that is shear-free.
The presented thesis work offers many potential applications. The membrane-based chemotaxis system, which has the general structure of two compartments separated by a membrane, resembled many physiological structures, including bone marrow, blood vessel, blood-brain barrier, hepatic portal vein, nephron in the kidneys, and alveolus in vi the lungs, and therefore serves as a versatile platform for understanding the transport phenomenon and the biochemical signaling in the aforementioned tissues. With improvements, the membrane-based system can also host larger-scale cell culture for protein production and tissue engineering. The protocols established for the gradient immobilization also provided many valuable references. These include: 1. A 1st order approximation of the reagents and the times required to fully saturate the substrate to be functionalized. 2. An automated image processing tool to measure the various parameters of cell motility. 3. A statistical framework to detect the presence of a directed migration. In theory, the standard operating procedures established are applicable to the surface functionalization with other peptides and proteins.
Livres sur le sujet "Immunology, Neutrophil"
A, Metcalf Julia, dir. Laboratory manual of neutrophil function. New York : Raven Press, 1986.
Trouver le texte intégralLilliehook, Inger. Studies of blood eosinophil and neutrophil granulocytes in health and diseased dogs. Uppsala : Sveriges Lantbruksuniversitet, 1999.
Trouver le texte intégralBerezhnai͡a, N. M. Neĭtrofily i immunologicheskiĭ gomeostaz. Kiev : Nauk. dumka, 1988.
Trouver le texte intégralAlonso-Fernández, Patricia. Neutrophils in biological age and longevity. New York : Nova Science Publishers, Inc., 2011.
Trouver le texte intégralThe Neutrophil : An Emerging Regulator of Inflammatory and Immune Response (Chemical Immunology). Not Avail, 2003.
Trouver le texte intégralDeLeo, Frank, et Mark T. Quinn. Neutrophil : Methods and Protocols. Springer, 2019.
Trouver le texte intégralDeLeo, Frank R., et Mark T. Quinn. Neutrophil : Methods and Protocols. Springer, 2020.
Trouver le texte intégralMarzano, Angelo Valerio, Dan Lipsker et Massimo Cugno, dir. Neutrophil-mediated skin diseases : immunology and genetics. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88963-254-1.
Texte intégralImmunopharmacology of neutrophils. London : Academic Press, 1994.
Trouver le texte intégralHellewell, Paul G., Clive Page et Timothy J. Williams. Immunopharmacology of Neutrophils. Elsevier Science & Technology Books, 1994.
Trouver le texte intégralChapitres de livres sur le sujet "Immunology, Neutrophil"
Dockrell, David H., Emmet E. McGrath, Moria K. B. Whyte et Ian Sabroe. « The Neutrophil ». Dans Immunology of Fungal Infections, 51–73. Dordrecht : Springer Netherlands, 2007. http://dx.doi.org/10.1007/1-4020-5492-0_3.
Texte intégralEdwards, S. W., D. A. Moulding, M. Derouet et R. J. Moots. « Regulation of Neutrophil Apoptosis ». Dans Chemical Immunology and Allergy, 204–24. Basel : KARGER, 2003. http://dx.doi.org/10.1159/000071562.
Texte intégralHolland, Steven M. « Neutropenia and Neutrophil Defects ». Dans Manual of Molecular and Clinical Laboratory Immunology, 766–74. Washington, DC, USA : ASM Press, 2016. http://dx.doi.org/10.1128/9781555818722.ch78.
Texte intégralUciechowski, Peter, et Lothar Rink. « Basophil, Eosinophil, and Neutrophil Functions in the Elderly ». Dans Immunology of Aging, 47–63. Berlin, Heidelberg : Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-39495-9_5.
Texte intégralBaggiolini, M., A. Walz, T. Brunner, B. Dewald, V. von Tscharner, R. Zwahlen, C. Dahinden et al. « Novel Neutrophil-Activating Peptides and Their Role in Inflammation ». Dans Progress in Immunology, 765–71. Berlin, Heidelberg : Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83755-5_104.
Texte intégralHannigan, M. O., C. K. Huang et D. Q. Wu. « Roles of PI3K in Neutrophil Function ». Dans Current Topics in Microbiology and Immunology, 165–75. Berlin, Heidelberg : Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18805-3_6.
Texte intégralSha’afi, R. I., et T. F. P. Molski. « Activation of the Neutrophil (Part 1 of 4) ». Dans Chemical Immunology and Allergy, 1–16. Basel : KARGER, 1988. http://dx.doi.org/10.1159/000318681.
Texte intégralSha’afi, R. I., et T. F. P. Molski. « Activation of the Neutrophil (Part 2 of 4) ». Dans Chemical Immunology and Allergy, 17–32. Basel : KARGER, 1988. http://dx.doi.org/10.1159/000318683.
Texte intégralSha’afi, R. I., et T. F. P. Molski. « Activation of the Neutrophil (Part 3 of 4) ». Dans Chemical Immunology and Allergy, 33–49. Basel : KARGER, 1988. http://dx.doi.org/10.1159/000318684.
Texte intégralSha’afi, R. I., et T. F. P. Molski. « Activation of the Neutrophil (Part 4 of 4) ». Dans Chemical Immunology and Allergy, 50–64. Basel : KARGER, 1988. http://dx.doi.org/10.1159/000318685.
Texte intégralActes de conférences sur le sujet "Immunology, Neutrophil"
Wu, Julie, Anne Pipathsouk, A. Keizer-Gunnink, Wynand Alkema, Fabrizia Fusetti, Shanshan Liu, Steve Atschuler, Lani Wu, Arjan Kortholt et Orion Weiner. « Abstract B48 : Homer3 regulates the establishment of neutrophil polarity ». Dans Abstracts : AACR Special Conference : Tumor Immunology and Immunotherapy : A New Chapter ; December 1-4, 2014 ; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b48.
Texte intégralKumagai, Yuko, Rihito Kanamaru, Hideyuki Ohzawa, Hisanaga Horie, Yoshinori Hosoya, Naohiro Sata et Joji Kitayama. « Abstract B77 : Low-density neutrophils (LDN) in circulating blood of postoperative patients may participate in the development of distant recurrence through the production of neutrophil extracellular traps (NETs) ». Dans Abstracts : AACR Special Conference on Tumor Immunology and Immunotherapy ; October 1-4, 2017 ; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-b77.
Texte intégralYazdani, Hamza O., Christof T. Kaltenmeier, David Geller et Samer Tohme. « Abstract A97 : Neutrophil extracellular traps (NETs) promote immune escape and metastatic growth after surgical stress ». Dans Abstracts : AACR Special Conference on Tumor Immunology and Immunotherapy ; November 17-20, 2019 ; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm19-a97.
Texte intégralMollaoglu, Gurkan, Alex Jones, Sarah Wait, Anandaroop Mukhopadhyay, Sangmin Jeong, Rahul Arya, Soledad Camolotto et al. « Abstract B72 : Lineage specifiers SOX2 and NKX2-1 inversely regulate tumor cell fate and neutrophil recruitment in lung cancer ». Dans Abstracts : AACR Special Conference on Tumor Immunology and Immunotherapy ; November 27-30, 2018 ; Miami Beach, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm18-b72.
Texte intégralSeitz, R., M. Wolf, R. Egbring et K. Havemann. « Neutrophil Elastase, Thrombin and Plasmin in Septic Shock : Influence on Prognosis ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643894.
Texte intégralRayes, Roni F., Alexandra Tinfow, Dorothy Antonatos, France Bourdeau, Betty Giannias et Jonathan D. Spicer. « Abstract B41 : Neutrophils modulate T-cell recruitment and promote hepatic metastases in lung cancer ». Dans Abstracts : AACR Special Conference on Tumor Immunology and Immunotherapy ; October 1-4, 2017 ; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-b41.
Texte intégralMishalian, Inbal, Rachel Bayuh, Lida Zolotarov, Liran Levy, Sunil Singhal, Steven M. Albelda et Zvi Gregorio Fridlender. « Abstract A68 : Tumor-associated neutrophils (TAN) develop protumorigenic properties during tumor progression. » Dans Abstracts : AACR Special Conference on Tumor Immunology : Multidisciplinary Science Driving Basic and Clinical Advances ; December 2-5, 2012 ; Miami, FL. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tumimm2012-a68.
Texte intégralSippel, Trisha R., Rae Russell, Timothy Ung, Marci Klaassen et Allen Waziri. « Abstract A44 : Disrupted transmigration of neutrophils in glioblastoma patients is augmented by steroid treatment. » Dans Abstracts : AACR Special Conference on Tumor Immunology : Multidisciplinary Science Driving Basic and Clinical Advances ; December 2-5, 2012 ; Miami, FL. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tumimm2012-a44.
Texte intégralHerbst, Brian, Elizabeth Jaffee et Lei Zheng. « Abstract PR10 : Inhibition of MEK1/2 overcomes resistance to aPD-1 blockade in pancreatic ductal adenocarcinoma through modulation of NETosis in tumor-associated neutrophils ». Dans Abstracts : AACR Virtual Special Conference : Tumor Immunology and Immunotherapy ; October 5-6, 2021. American Association for Cancer Research, 2022. http://dx.doi.org/10.1158/2326-6074.tumimm21-pr10.
Texte intégralPerobelli, Suelen Martins, Ana Carolina Terra Mercadante, Triciana Gonçalves-Silva, Rômulo Galvani, Antônio Pereira-Neves, Marlene Benchimol, Alberto Nobrega et Adriana Bonomo. « Abstract B39 : Neutrophils G-CSF stimulated promotes specific protection against graft vs. host disease and keeps the graft vs. leukemia effect ». Dans Abstracts : AACR Special Conference : Tumor Immunology and Immunotherapy : A New Chapter ; December 1-4, 2014 ; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b39.
Texte intégral