Littérature scientifique sur le sujet « Human Induced neurons »
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Articles de revues sur le sujet "Human Induced neurons"
Wang, Gefei, Rui Li, Zhiwu Jiang, Liming Gu, Yanxia Chen, Jianping Dai et Kangsheng Li. « Influenza Virus Induces Inflammatory Response in Mouse Primary Cortical Neurons with Limited Viral Replication ». BioMed Research International 2016 (2016) : 1–7. http://dx.doi.org/10.1155/2016/8076989.
Texte intégralHäkli, Martta, Satu Jäntti, Tiina Joki, Lassi Sukki, Kaisa Tornberg, Katriina Aalto-Setälä, Pasi Kallio, Mari Pekkanen-Mattila et Susanna Narkilahti. « Human Neurons Form Axon-Mediated Functional Connections with Human Cardiomyocytes in Compartmentalized Microfluidic Chip ». International Journal of Molecular Sciences 23, no 6 (15 mars 2022) : 3148. http://dx.doi.org/10.3390/ijms23063148.
Texte intégralCresto, Noémie, Camille Gardier, Marie-Claude Gaillard, Francesco Gubinelli, Pauline Roost, Daniela Molina, Charlène Josephine et al. « The C-Terminal Domain of LRRK2 with the G2019S Substitution Increases Mutant A53T α-Synuclein Toxicity in Dopaminergic Neurons In Vivo ». International Journal of Molecular Sciences 22, no 13 (23 juin 2021) : 6760. http://dx.doi.org/10.3390/ijms22136760.
Texte intégralRawson, N. E., G. Gomez, B. Cowart, J. G. Brand, L. D. Lowry, E. A. Pribitkin et D. Restrepo. « Selectivity and Response Characteristics of Human Olfactory Neurons ». Journal of Neurophysiology 77, no 3 (1 mars 1997) : 1606–13. http://dx.doi.org/10.1152/jn.1997.77.3.1606.
Texte intégralGunewardene, Niliksha, Duncan Crombie, Mirella Dottori et Bryony A. Nayagam. « Innervation of Cochlear Hair Cells by Human Induced Pluripotent Stem Cell-Derived NeuronsIn Vitro ». Stem Cells International 2016 (2016) : 1–10. http://dx.doi.org/10.1155/2016/1781202.
Texte intégralKraskovskaya, Nina, Anastasia Bolshakova, Mikhail Khotin, Ilya Bezprozvanny et Natalia Mikhailova. « Protocol Optimization for Direct Reprogramming of Primary Human Fibroblast into Induced Striatal Neurons ». International Journal of Molecular Sciences 24, no 7 (5 avril 2023) : 6799. http://dx.doi.org/10.3390/ijms24076799.
Texte intégralPerego, M. Chiara, Benjamin D. McMichael, Nicholas R. McMurry, Scott W. Ventrello et Lisa J. Bain. « Arsenic Impairs Differentiation of Human Induced Pluripotent Stem Cells into Cholinergic Motor Neurons ». Toxics 11, no 8 (25 juillet 2023) : 644. http://dx.doi.org/10.3390/toxics11080644.
Texte intégralKarpe, Yashashree, Zhenyu Chen et Xue-Jun Li. « Stem Cell Models and Gene Targeting for Human Motor Neuron Diseases ». Pharmaceuticals 14, no 6 (12 juin 2021) : 565. http://dx.doi.org/10.3390/ph14060565.
Texte intégralTian, Jie L., Chia-Wei Huang, Farzad Eslami, Michael Philip Mannino, Rebecca Lee Mai et Gerald W. Hart. « Regulation of Primary Cilium Length by O-GlcNAc during Neuronal Development in a Human Neuron Model ». Cells 12, no 11 (31 mai 2023) : 1520. http://dx.doi.org/10.3390/cells12111520.
Texte intégralCheng, Xueyan, Zijian Tan, Xiao Huang, Yimin Yuan, Shangyao Qin, Yakun Gu, Dan Wang, Cheng He et Zhida Su. « Inhibition of Glioma Development by ASCL1-Mediated Direct Neuronal Reprogramming ». Cells 8, no 6 (11 juin 2019) : 571. http://dx.doi.org/10.3390/cells8060571.
Texte intégralThèses sur le sujet "Human Induced neurons"
Melo, de Farias Ana Raquel. « Probing the Alzheimer’s disease risk gene PTK2B using human-derived induced neurons ». Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS062.
Texte intégralAlzheimer's disease (AD) is the main type of dementia and poses a significant global public health challenge. It is characterized by a progressive decline in cognition, memory, and behavioral functions and affects more than 55 million people worldwide. At the molecular level, AD is defined by the presence of aggregated neurofibrillary tangles within neurons and the accumulation of amyloid-β (Aβ) plaques in the brain. These pathological features are associated with alterations in neuronal activity, synapse loss, gliosis, and neuroinflammation, leading to irreversible neurodegeneration. AD etiology and pathophysiology involves a complex interplay between genetic and environmental factors. Genome-Wide Association Studies have identified several loci carrying single nucleotide polymorphisms (SNPs) associated with AD risk. Among these loci, the one harboring the Protein Tyrosine Kinase 2β (PTK2B) is highlighted in the present work. This gene encodes a protein tyrosine kinase that is involved in calcium-induced regulation of ion channels and activation of numerous signaling pathways, such as MAP kinase. Non-synonimous genetic variations in the PTK2B locus have been associated with an increased risk of AD and are thought to regulate PTK2B expression. However, both the physiological and pathophysiological roles of PTK2B are not fully understood. In the human brain, PTK2B expression is mainly observed in glutamatergic neurons. Its expression declines during AD progression and may contribute to neuronal dysfunctions observed in the disease, such as increased electrical excitability and synaptic alterations. Therefore, understanding the role of PTK2B in human neurons may contribute to reveal the mechanisms of neuronal dysfunctions in AD. Considering that, the aims of this thesis are to uncover the cellular processes and molecular pathways regulated by PTK2B in human neurons. To that, we took advantage of isogenic human induced-pluripotent stem cells (hiPSCs) to generate neurons expressing different levels of PTK2B. Next, we employed functional and molecular assays to probe the consequences of altered PTK2B expression both in a physiological and in an AD-like context. We show that reduced PTK2B expression leads to increased TAU phosphorylation at various epitopes associated with AD pathology, suggesting a central role of PTK2B in regulating TAU aggregation. Using single-cell transcriptomics, we also show that reduced PTK2B expression leads to specific transcriptional alterations related to neuronal electrical activity and synaptic transmission mainly in glutamatergic neurons. Calcium imaging experiments indicate that PTK2B downregulation contributes to increased calcium spikes frequency without affecting synchronization, indicating an elevated neuronal electrical activity. Additionally, results from electrophysiological recordings from multi-electrode array (MEA) show increased electrical activity and disrupted bursting patterns in PTK2B mutant neurons. Overall, this work sheds light on the involvement of PTK2B in AD-related cellular processes, providing insights into the molecular mechanisms and functional alterations associated with PTK2B dysregulation in human iPSC-derived neural cells
Sánchez, Danés Adriana 1984. « Generation of human dopaminergic neurons from induced pluripotent stem cells to model Parkinson's disease ». Doctoral thesis, Universitat Pompeu Fabra, 2012. http://hdl.handle.net/10803/96912.
Texte intégralLa malaltia de Parkinson (MP) és una malaltia neurodegenerativa incurable que causa invalidesa i mort prematura. Els pacients de la malaltia de Parkinson presenten alteracions motores degudes a una degeneració gradual de les neurones dopaminèrgiques que projecten des de la substància nigra fins a l’estriat. A nivell microscòpic s’observa la presència d’agregats proteics insolubles en el citosol de les neurones coneguts com cossos o neurites de Lewy. Els mecanismes patològics responsables de la MP no es coneixen bé, possiblement a causa de la manca de models animals i cel•lulars adequats. Per tant, existeix una gran necessitat de desenvolupar models experimentals fiables que recapitulin les característiques bàsiques de la MP. El recent desenvolupament de les cèl•lules mare pluripotents induïdes (iPSC) ha permès la generació de iPSC específiques de pacient i el seu ús per modelar malalties humanes, ara bé, no és clar si aquesta estratègia es pot utilitzar per modelar exitosament malalties d’origen tardà, com ara la MP. És important destacar que el modelatge de malalties utilitzant iPSC, es basa, en gran mesura en l'existència de protocols eficients per a la diferenciació de les iPSC cap al tipus cel•lular rellevant per a la malaltia. Durant aquest període, per primera vegada, s’ha desenvolupat un protocol per a l’eficient diferenciació de les iPSC cap a neurones dopaminèrgiques amb les propietats característiques de neurones dopaminèrgiques nigrostriatals, mitjançant l’expressió forçada de LMX1A utilitzant vectors lentivirals. A continuació, s’ha generat un model cel•lular usant iPSC derivades de pacients de MP que recapitula les principals característiques fenotípiques de la malaltia, com ara la pèrdua de neurones dopaminèrgiques i l'acumulació de α-sinucleïna en les neurones dopaminèrgiques. En general, els nostres resultats demostren que hem desenvolupat una eina valuosa per a l’estudi dels mecanismes patològics que condueixen a la MP, així com una nova plataforma pel descobriment de nous fàrmacs encaminats a prevenir o evitar la neurodegeneració.
GIANI, ALICE MARIA. « GENERATION OF AUTHENTIC HUMAN NEOCORTICAL NEURONS FROM INDUCED PLURIPOTENT STEM CELLS TO INVESTIGATE 7Q11.23 GENE DOSAGE IMBALANCES ». Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/561835.
Texte intégralThis research project has been aimed to investigate human neocortical development in healthy and diseased subjects by analyzing and comparing the transcriptional profiles and cellular morphologies of human neocortical cells derived from induced pluripotent stem cells (iPSCs). Given the importance to rely on a solid and highly reproducible iPSCs-based differentiation protocol that generates authentic neocortical neurons in vitro with high efficiency before applying it as a model system of human neurodevelopmental disorders, in the first phase of this study we performed a comprehensive transcriptional, cellular and physiological characterization of the in vitro neurodevelopmental paradigm. The transcriptional dynamics regulating in vitro neocortical development have been investigated by performing RNA-sequencing (RNA-seq) at both population and single- cell level in combination with several bioinformatics analyses including principal component analysis (PCA), differential gene expression analysis and weighted gene co-expression network analysis (WGCNA). The transcriptional results were corroborated by the widespread positivity for a selected panel of informative cell-fate and cell-stage specific markers detected through immunocytochemistry and the physiological maturity of our iPSCs-derived neocortical neurons was further confirmed by their ability to generate action potentials, develop complex firing patterns and sustain excitatory and inhibitory spontaneous synaptic activity. Overall, these results fully validated the reproducibility of the differentiation protocol and its efficiency and reliability in generating physiologically mature authentic neocortical neurons. Subsequently, we applied this extensively characterized neocortical differentiation paradigm to model in vitro two human neurodevelopmental disorders caused by symmetrical copy number variations (CNVs) of the Williams-Beuren syndrome chromosome region (WBSCR) located on the long arm (q) of chromosome 7 at position 11.23 (7q.11.23 locus). 7q11.23 CNVs are of special interest as the two disorders resulting from the deletion (Williams syndrome, WS) and duplication (7q.11.23 duplication syndrome, 7q11DUP) of this region exhibit cognitive and behavioral phenotypes marked by both similar features and symmetrically opposite traits. The association of 7q11DUP to complex neurodevelopmental disorders such as autism spectrum disorder and schizophrenia, while WS is a well-characterized syndrome without clear overlap to complex neurodevelopmental disorders make the study of this locus extremely interesting to identify the molecular mechanisms unique to each clinical condition, common to both syndromes and shared with other complex neurodevelopmental disorders. To this aim, we generated several iPSCs lines from a large cohort comprising WS individuals, 7q11DUP patients and healthy subjects and differentiated them into neocortical neurons by applying the previously in-depth characterized protocol. Having assessed the quality of our iPSCs-derived neocortical neurons, we are currently identifying neuronal subtypes specific genes and gene networks having the most statistically significant relationship to these disorders through single cell RNA-sequencing analysis. Furthermore, morphometric analysis of WS and control iPSCs-derived neocortical neurons has confirmed in humans many neuronal morphological abnormalities observed in a mouse knockout for Dnajc30, a previously uncharacterized gene contained in the 7q11.23 locus.
Fenske, Pascal [Verfasser]. « Characterization of synaptic transmission in autaptic cultured neurons derived from human induced pluripotent stem cells / Pascal Fenske ». Berlin : Freie Universität Berlin, 2021. http://d-nb.info/1234451611/34.
Texte intégralToli, Diana Eleni. « Directed differentiation and purification of motor neurons from human induced pluripotent stem cells to model Amyotrophic Lateral Sclerosis ». Thesis, Paris 5, 2013. http://www.theses.fr/2013PA05T044/document.
Texte intégralAmyotrophic lateral sclerosis (ALS) is a fatal adult-onset neurodegenerative disorder primarily affecting motor neurons. Mechanisms leading to motor neuron death in ALS are poorly understood mostly because of disease heterogeneity and lack of access to affected cells. The induced pluripotent stem cell (iPSc) technology provides the opportunity to obtain and study human motor neurons and is therefore a promising tool for ALS modeling.IPSc clones from control subjects were generated, and we compared several protocols in order to set up an efficient protocol for iPSc differentiation into motor neurons. The obtained cultures were heterogenous, comprising different neuron subtypes and neural precursors. To allow investigation of intrinsic disease mechanisms in ALS motor neurons, we developed a new technique to purify motor neurons by FACS sorting. By combining the use of a lentiviral vector expressing a fluorescent protein under control of a motoneuron-specific promoter and of a monoclonal antibody directed against the p75 neurotrophin receptor, isolation of exquisitely pure motor neurons was achieved. In parallel, iPSc technology was used to establish cellular models of ALS. IPSc were generated from one patient with familial ALS carrying a mutation in the TARDBP gene (encoding a DNA-binding protein, TDP-43) and one patient with sporadic ALS. To validate our models, we investigated characteristic disease phenotypes during iPSc differentiation, including i) cytoplasmic aggregate formation, ii) motor neuron generation and survival defects, iii) neurite growth alterations
Hermann, Andreas, Jeong Beom Kim, Sumitra Srimasorn, Holm Zaehres, Peter Reinhardt, Hans R. Schöler et Alexander Storch. « Factor-Reduced Human Induced Pluripotent Stem Cells Efficiently Differentiate into Neurons Independent of the Number of Reprogramming Factors ». Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-203366.
Texte intégralMiyawaki, Yoshifumi. « Zonisamide promotes survival of human induced pluripotent stem cell-derived dopaminergic neurons in the striatum of female rats ». Kyoto University, 2020. http://hdl.handle.net/2433/259730.
Texte intégralBeevers, Joel Edward. « Investigating the function of microtubule-associated protein tau (MAPT) and its genetic association with Parkinson's using human iPSC-derived dopamine neurons ». Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:7a94919a-73a1-4a9f-b04d-cdf5b9c64be7.
Texte intégralBurton, Mark P., Declan J. McKeefry, Brendan T. Barrett, Chara Vakrou et A. B. Morland. « Disruptions to human speed perception induced by motion adaptation and transcranial magnetic stimulation ». Wiley, 2009. http://hdl.handle.net/10454/4731.
Texte intégralTo investigate the underlying nature of the effects of transcranial magnetic stimulation (TMS) on speed perception, we applied repetitive TMS (rTMS) to human V5/MT+ following adaptation to either fast- (20 deg/s) or slow (4 deg/s)-moving grating stimuli. The adapting stimuli induced changes in the perceived speed of a standard reference stimulus moving at 10 deg/s. In the absence of rTMS, adaptation to the slower stimulus led to an increase in perceived speed of the reference, whilst adaptation to the faster stimulus produced a reduction in perceived speed. These induced changes in speed perception can be modelled by a ratio-taking operation of the outputs of two temporally tuned mechanisms that decay exponentially over time. When rTMS was applied to V5/MT+ following adaptation, the perceived speed of the reference stimulus was reduced, irrespective of whether adaptation had been to the faster- or slower-moving stimulus. The fact that rTMS after adaptation always reduces perceived speed, independent of which temporal mechanism has undergone adaptation, suggests that rTMS does not selectively facilitate activity of adapted neurons but instead leads to suppression of neural function. The results highlight the fact that potentially different effects are generated by TMS on adapted neuronal populations depending upon whether or not they are responding to visual stimuli.
BBSRC
Kikuchi, Tetsuhiro. « Survival of human induced pluripotent stem cell-derived midbrain dopaminergic neurons in the brain of a primate model of Parkinson's disease ». Kyoto University, 2012. http://hdl.handle.net/2433/159389.
Texte intégralLivres sur le sujet "Human Induced neurons"
Nat, Roxana, et Andreas Eigentler. Cell Culture, iPS Cells and Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0013.
Texte intégralSeth, Rohit. Zinc deficiency induces apoptosis via mitochondrial p53- and caspase-dependent pathways in human neuronal precursor cells. Elseveir, 2014.
Trouver le texte intégralBisschops, Raf. Ligand & Electrically Induced Activation Patterns in Myenteric Neuronal Networks : Confocal Calcium Imaging As a Bridge Between Basic & Human Physiology (Acta Biomedica Lovaniensia). Leuven Univ Pr, 2005.
Trouver le texte intégralZiemann, Ulf. Pharmacology of TMS measures. Sous la direction de Charles M. Epstein, Eric M. Wassermann et Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0013.
Texte intégralChapitres de livres sur le sujet "Human Induced neurons"
Zhou-Yang, Lucia, Sophie Eichhorner, Lukas Karbacher, Lena Böhnke, Larissa Traxler et Jerome Mertens. « Direct Conversion of Human Fibroblasts to Induced Neurons ». Dans Methods in Molecular Biology, 73–96. New York, NY : Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1601-7_6.
Texte intégralLegault, Emilie M., et Janelle Drouin-Ouellet. « Generation of Induced Dopaminergic Neurons from Human Fetal Fibroblasts ». Dans Methods in Molecular Biology, 97–115. New York, NY : Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1601-7_7.
Texte intégralTofoli, Fabiano Araújo, Ana Teresa Silva Semeano, Ágatha Oliveira-Giacomelli, Maria Carolina Bittencourt Gonçalves, Merari F. R. Ferrari, Lygia Veiga Pereira et Henning Ulrich. « Midbrain Dopaminergic Neurons Differentiated from Human-Induced Pluripotent Stem Cells ». Dans Methods in Molecular Biology, 97–118. New York, NY : Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9007-8_8.
Texte intégralFernandes, Sofia R., Mariana Pereira, Sherif M. Elbasiouny, Yasin Y. Dhaher, Mamede de Carvalho et Pedro C. Miranda. « Interplay Between Electrical Conductivity of Tissues and Position of Electrodes in Transcutaneous Spinal Direct Current Stimulation (tsDCS) ». Dans Brain and Human Body Modelling 2021, 101–22. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-15451-5_7.
Texte intégralFalco, Amel, Rocío Bartolomé-Cabrero et Sergio Gascón. « Bcl-2-Assisted Reprogramming of Mouse Astrocytes and Human Fibroblasts into Induced Neurons ». Dans Methods in Molecular Biology, 57–71. New York, NY : Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1601-7_5.
Texte intégralSilva, M. Catarina, Ghata Nandi et Stephen J. Haggarty. « Differentiation of Human Induced Pluripotent Stem Cells into Cortical Neurons to Advance Precision Medicine ». Dans Methods in Molecular Biology, 143–74. New York, NY : Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1979-7_10.
Texte intégralBuchholz, Sarah, Michael Bell-Simons, Cagla Cakmak, Jennifer Klimek, Li Gan et Hans Zempel. « Cultivation, Differentiation, and Lentiviral Transduction of Human-Induced Pluripotent Stem Cell (hiPSC)-Derived Glutamatergic Neurons for Studying Human Tau ». Dans Methods in Molecular Biology, 533–49. New York, NY : Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3629-9_31.
Texte intégralSalvador, R., M. C. Biagi, O. Puonti, M. Splittgerber, V. Moliadze, M. Siniatchkin, A. Thielscher et G. Ruffini. « Personalization of Multi-electrode Setups in tCS/tES : Methods and Advantages ». Dans Brain and Human Body Modeling 2020, 119–35. Cham : Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-45623-8_7.
Texte intégralYoung, Jessica E., et Raul Delgado-Morales. « Human-Induced Pluripotent Stem Cell-Derived Neurons to Model and Gain Insights into Alzheimer’s Disease Pathogenesis ». Dans Stem Cell Genetics for Biomedical Research, 3–12. Cham : Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90695-9_1.
Texte intégralMehta, Arpan R., Siddharthan Chandran et Bhuvaneish T. Selvaraj. « Assessment of Mitochondrial Trafficking as a Surrogate for Fast Axonal Transport in Human Induced Pluripotent Stem Cell–Derived Spinal Motor Neurons ». Dans Methods in Molecular Biology, 311–22. New York, NY : Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1990-2_16.
Texte intégralActes de conférences sur le sujet "Human Induced neurons"
Schmieder, F., R. Habibey, V. Busskamp, J. W. Czarske et L. Büttner. « Adaptive Holographic Optogenetic Illumination for Human Neural Network Analysis ». Dans Digital Holography and Three-Dimensional Imaging. Washington, D.C. : Optica Publishing Group, 2022. http://dx.doi.org/10.1364/dh.2022.w4a.7.
Texte intégralPitta, Marina Galdino da Rocha, Jordy Silva de Carvalho, Luzilene Pereira de Lima et Ivan da Rocha Pitta. « iPSC therapies applied to rehabilitation in parkinson’s disease ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.022.
Texte intégralMurata, Masaaki, Hidekatsu Ito, Teppei Taenaka et Suguru N. Kudoh. « Modification of activity pattern induced by synaptic enhancements in a semi-artificial network of living neurons ». Dans 2011 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2011. http://dx.doi.org/10.1109/mhs.2011.6102187.
Texte intégralKomatsu, Masaaki, Heather E. Wheeler, Claudia Wing, Shannon Delaney et M. Eileen Dolan. « Abstract 3489 : A novel investigation into chemotherapy-induced peripheral neuropathy using iPSC-derived human neurons ». Dans Proceedings : AACR Annual Meeting 2014 ; April 5-9, 2014 ; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3489.
Texte intégralFoisset, F., C. Lehalle, A. Nasri, C. Bourdais, I. Vachier, S. Assou, Q. Muller et al. « Development of a bronchial epithelium model innervated by sensory neurons from human induced pluripotent stem cells ». Dans ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2394.
Texte intégralFerreira, João, Manuel de Sousa Ribeiro, Ricardo Gonçalves et João Leite. « Looking Inside the Black-Box : Logic-based Explanations for Neural Networks ». Dans 19th International Conference on Principles of Knowledge Representation and Reasoning {KR-2022}. California : International Joint Conferences on Artificial Intelligence Organization, 2022. http://dx.doi.org/10.24963/kr.2022/45.
Texte intégralCourellis, Hristos, Juri Minxha, Araceli Cardenas, Taufik Valiante, Adam Mamelak, Ralph Adolphs, Stefano Fusi et Ueli Rutishauser. « Abstract representations encoded by human hippocampal neurons support behavioral inference and can be induced through verbal instruction. » Dans 2023 Conference on Cognitive Computational Neuroscience. Oxford, United Kingdom : Cognitive Computational Neuroscience, 2023. http://dx.doi.org/10.32470/ccn.2023.1668-0.
Texte intégralCotter, Laurent, Romane A. Lahaye, Wei Chou Tseng, Yuanjing Liu, Aude Lemesle, Sophie Lenoir, Elena Dale, Frédéric Saudou et Anselme L. Perrier. « I07 Selective lowering of mutant-huntingtin by antisense oligonucleotides (ASOs) in human cortical neurons derived from patient-specific induced pluripotent stem cells (HD-IPSC) ». Dans EHDN 2022 Plenary Meeting, Bologna, Italy, Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jnnp-2022-ehdn.233.
Texte intégral« Human induced stem cells (hiPSCs)-derived motor neurons model applied for the study of the aryl hydrocarbon receptor mediated signaling by the induction of environmental pollutant ». Dans INTERNATIONAL CONFERENCE ON BIOLOGICAL RESEARCH AND APPLIED SCIENCE. Jinnah University for Women, 2024. http://dx.doi.org/10.37962/ibras/2024/57.
Texte intégralMartens, William L., Philip Poronnik et Darren Saunders. « Hypothesis-Driven Sonification of Proteomic Data Distributions Indicating Neurodegredation in Amyotrophic Lateral Sclerosis ». Dans The 22nd International Conference on Auditory Display. Arlington, Virginia : The International Community for Auditory Display, 2016. http://dx.doi.org/10.21785/icad2016.024.
Texte intégralRapports d'organisations sur le sujet "Human Induced neurons"
Yu, Mei, Pengyu Wang, Binbin Li, Qiaoling Ruan, Jingzi ZhangBao, Lei Wu, Xiaoshuang Zhang, Zhaolin Liu et Fang Huang. NRSF Negatively Regulates Microglial Pro-Inflammatory Activation. Progress in Neurobiology, mai 2024. http://dx.doi.org/10.60124/j.pneuro.2024.20.02.
Texte intégral