Littérature scientifique sur le sujet « Hemostasis »
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Articles de revues sur le sujet "Hemostasis"
Rathod, Dr Rohit Narendra. « Hemostasis in the Surgical Field ». EAS Journal of Medicine and Surgery 4, no 10 (16 novembre 2022) : 211–14. http://dx.doi.org/10.36349/easjms.2022.v04i10.003.
Texte intégralRomantsov, Mikhail N., Eugene F. Cherednikov, Aleksandr Anatolevich Glukhov et Constantine O. Fursov. « New technologies of endoscopic hemostasis in a treatment protocol of patients with gastroduodenal ulcer bleeding ». Vestnik of Experimental and Clinical Surgery 11, no 1 (8 avril 2018) : 16–23. http://dx.doi.org/10.18499/2070-478x-2018-11-1-16-23.
Texte intégralBudko, E. V., L. M. Yampolsky, D. A. Chernikova et A. A. Khabarov. « Comparative evaluation of the effectiveness of a local hemostatic agent modified with a bio-organic composition ». CARDIOMETRY, no 18 (18 mai 2021) : 100–112. http://dx.doi.org/10.18137/cardiometry.2021.18.100112.
Texte intégralWu, Baofeng, Ruixin Zhang, Chendi Liang, Chengjie Zhang et Gang Qin. « Study on the Safety of the New Radial Artery Hemostasis Device ». Journal of Interventional Cardiology 2022 (5 avril 2022) : 1–8. http://dx.doi.org/10.1155/2022/2345584.
Texte intégralHan, Wenli, et Shige Wang. « Advances in Hemostatic Hydrogels That Can Adhere to Wet Surfaces ». Gels 9, no 1 (22 décembre 2022) : 2. http://dx.doi.org/10.3390/gels9010002.
Texte intégralZhuk, S., et V. Vorobey-Vykhivska. « Dynamics of indicators of hemostatic system as prognostic criteria consequences of ART programs ». HEALTH OF WOMAN, no 5(111) (20 juin 2016) : 165–70. http://dx.doi.org/10.15574/hw.2016.111.165.
Texte intégralRavn, Hanne. « Hemostasis in Pediatric Cardiac Surgery ». Seminars in Thrombosis and Hemostasis 43, no 07 (8 juin 2017) : 682–90. http://dx.doi.org/10.1055/s-0037-1603365.
Texte intégralJin, Huiyang, et Zhengke Wang. « Advances in Alkylated Chitosan and Its Applications for Hemostasis ». Macromol 2, no 3 (27 juillet 2022) : 346–60. http://dx.doi.org/10.3390/macromol2030022.
Texte intégralPark, Jin-Seok, Byung Wook Bang, Su Jin Hong, Eunhye Lee, Kye Sook Kwon, Hyung Kil Kim, Yong Woon Shin et Don Haeng Lee. « Efficacy of a novel hemostatic adhesive powder in patients with refractory upper gastrointestinal bleeding : a pilot study ». Endoscopy 51, no 05 (10 janvier 2019) : 458–62. http://dx.doi.org/10.1055/a-0809-5276.
Texte intégralDambayev, G. Ts, A. N. Baikov, Ye V. Semichev, M. N. Shpisman, A. N. Aleinik, O. I. Deneko et P. S. Bushlanov. « The intraoperative methods of hemostasis during liver surgeries ». Bulletin of Siberian Medicine 10, no 4 (28 août 2011) : 89–92. http://dx.doi.org/10.20538/1682-0363-2011-4-89-92.
Texte intégralThèses sur le sujet "Hemostasis"
Keebaugh, Audrey Elizabeth. « Evaluation of hemostasis in hyperthyroid cats ». Thesis, Virginia Tech, 2020. http://hdl.handle.net/10919/99376.
Texte intégralMaster of Science
In feline hyperthyroidism, there is a predisposition for thrombus formation. An alteration of hemostasis has been documented in hyperthyroid humans, but despite reports of thrombus formation in hyperthyroid cats, the underlying mechanism is currently unknown. Hyperthyroidism can lead to cardiac abnormalities that could possibly contribute thrombus formation, although thrombus formation has occurred in hyperthyroid cats without detected abnormalities. The goal of this study was to evaluate markers of hemostasis in hyperthyroid cats presenting for radioiodine therapy to evaluate for presence of hypercoagulability. Twenty-five hyperthyroid cats were evaluated with hemostasis panels and echocardiograms. The results were compared to a group of 13 healthy cats. Markers of hemostasis and echocardiograms in 7 hyperthyroid cats were also compared to results 6 months or greater post-radioiodine therapy. There was evidence of altered hemostasis and hypercoagulability in hyperthyroid cats. The alterations noted resolved after radioiodine therapy and do not appear to be solely attributed to cardiac abnormalities seen in hyperthyroid cats.
Hormiga, Hernando Gonzalez [UNESP]. « Aplicação de diferentes pinças hemostáticas em veias de equinos : estudo morfológico ». Universidade Estadual Paulista (UNESP), 2016. http://hdl.handle.net/11449/144581.
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Foi realizada a avaliação morfológica e morfométrica da veia cefálica submetida à pinçamento de cinco equinos hígidos. Foram testadas as pinças hemostáticas: Diffenbach bulldog, De Bakey bulldog, Rochester reta e De Bakey com cremalheira. Após 15 minutos da aplicação das referidas pinças, foi realizada a flebectomia parcial e coletadas as amostras referentes a cada segmento do vaso pinçado e do segmento controle sem pinçamento. Das peças procedeu-se as preparações histológicas dos segmentos da veia nas colorações de Hematoxilina-Eosina e Tricrômio de Masson, os cortes histológicos foram avaliados por microscopia óptica. Foi realizada análise morfológica das veias, de forma qualitativa, utilizando para isto uma escala de avaliação de lesões vasculares após pinçamento; a avaliação morfométrica, quantificando em micrometros o achatamento produzido pelas pinças nas diferentes camadas vasculares. Em ambos os estudos, morfológico e morfométrico, as pinças com serrilhamento transversal e fechamento tipo cremalheira causaram as maiores alterações, observou se marcada vacuolização das células musculares e desarranjo na túnica media com perda marcada das células endoteliais da túnica intima do vaso estudado.
Morphometric and morphologic evaluation of the cephalic vein of five healthy horses submitted to clamping was done. Hemostatic clamps tested were Dieffenbach bulldog, De Bakey bulldog, Rochester straight and De Bakey with ratchets. 15 minutes after mentioned clamps were applied partial phlebotomy was performed and histologic sections of the veins were prepared and stained with Hematoxylin-Eosin and Masson Trichrome, after the stained preparations were evaluated by light microscopy. A qualitative morphological analysis of the veins was performed using a rating scale of vascular lesions after clamping; the morphometric evaluation consisted in quantifying in micrometers the flattening produced by the hemostatic clamps in the different vascular beds. In both studies, morphologic and morphometric, hemostats with transverse serration and ratcheted mechanism caused major changes, pronounced vacuolization of the muscle cells, derangement of the medium tunic and marked loss of endothelial cells of the intima tunic was observed in the vessel studied.
Gonçalves, Daniele Silvano. « Avaliação das alterações hemorrágicas e tromboembólicas em cães com doença renal crônica ». Botucatu, 2016. http://hdl.handle.net/11449/134372.
Texte intégralResumo: A doença renal crônica (DRC) acomete principalmente cães idosos e tem como característica principal a perda irreversível da função renal. A DRC em cães promove alterações metabólicas graves, caracterizadas frequentemente pela azotemia, hipoalbuminemia e anemia não regenerativa. Tanto a azotemia quanto a uremia predispõem a alterações hemostáticas que podem levar a quadros hemorrágicos. Além das disfunções plaquetárias, deficiência de anticoagulantes naturais e redução da fibrinólise são fatores que predispõem ao tromboembolismo. Este trabalho tem como objetivo avaliar as possíveis tendências hemorrágicas ou trombóticas em cães com DRC. Foram selecionados 20 cães saudáveis (grupo controle) com exames dentro da normalidade e 17 cães com DRC em estágios III ou IV classificados segundo a IRIS e a relação proteína/creatirina urinária maior que um (grupo DRC). As amostras de sangue para a realização da tromboelastometria (TEM), agregação plaquetária, tempo de protrombina (TP), tempo de tromboplastina parcial ativada (TTPA) e concentração de fibrinogênio foram colhidas em momento único para ambos os grupos após os critérios de inclusão serem confirmados. A análise estatística foi realizada de acordo com a distribuição das variáveis, ao nível de 5% de significância. No presente estudo foi possível observar um estado de hipercoagulabilidade sanguínea nos cães com DRC. Na TEM com o ativador de via extrínseca, observou-se encurtamento no tempo de coagulação e do tempo de formação do coá... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Chronic kidney disease (CKD) affects mostly older dogs and its main characteristic is the irreversible loss of kidney function. CKD in dogs promotes serious metabolic alterations, often characterized by azotemia, hypoalbuminemia and non-regenerative anemia. Azotemia and uremia predispose the hemostatic abnormalities that can lead to hemorrhagic cases. In addition to platelet dysfunction, deficiency of natural anticoagulants and reduced fibrinolysis are factors that predispose to thromboembolism. This work aims to evaluate the possible bleeding or thrombotic tendencies in dogs with CKD. 20 healthy dogs were selected (control group) with tests within normal limits and 17 dogs with CKD in stages III or IV classified according to IRIS and urine protein to creatinine ratio greater than one (CKD group). Blood samples for the realization of thromboelastometry (TEM), platelet aggregation, prothrombin time (PT), activated partial thromboplastin time (APTT) and fibrinogen concentration were collected at one time for both groups after the inclusion criteria had been confirmed. Statistical analysis performed according to the distribution of the variable at the 5% level of significance. In the present study, we observed a state of hypercoagulable blood in dogs with CKD. In TEM with the extrinsic pathway activator, there was shortening of the clotting time and clot formation time, increasing the alpha angle and the maximum clot firmness, and reducing the maximum lysis in dogs with CKD comp... (Complete abstract click electronic access below)
Mestre
Joesph, Wiencek R. « Regulating Hemostasis : The Factor Va Cofactor Effect ». Cleveland State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=csu1431514489.
Texte intégralLindfelt, Jan O. W. « Hepatic nerves in hemostasis and glucose metabolism : ». Lund : Dept. of Surgery, Lund University, 1993. http://catalog.hathitrust.org/api/volumes/oclc/39654187.html.
Texte intégralPeterle, Daniele. « Molecular Mechanism in the Alteration of Hemostasis ». Doctoral thesis, Università degli studi di Padova, 2018. http://hdl.handle.net/11577/3426350.
Texte intégralTeixeira, Bruno Costa. « Efeito de diferentes intensidades de exercício aeróbio prévio, sobre a curva lipêmica, inflamação e hemostasia de sujeitos submetidos à refeição hiperlipídica ». reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2016. http://hdl.handle.net/10183/142537.
Texte intégralBackground: Regular consumption of high-fat meals has been considered to play a role in the development of cardiovascular diseases. The increase of postprandial lipemia after a high-fat meal consumption can imbalance the relationship between coagulation and fibrinolysis and, by consequence, enhance an inflammatory response. Conversely, exercise has been considered an important intervention, once it may attenuate inflammatory responses and counterbalance hemostatic systems during the postprandial period. Purpose: Verify the subacute effect of two exercise bouts performed at different intensities on postprandial lipemia, inflammation and hemostasis after the consumption of a high-fat meal. Methods: Eleven healthy and physically active male subjects with average age of 23 ± 3 years completed 2-day trials in three conditions: Control, low-intensity exercise (LI) and moderate-intensity exercise (MI). Subjects performed an exercise bout (LI or MI) or no exercise (Control) on the evening of day 1. On the morning of day 2, a high-fat meal was provided (15 % of protein, 35 % of carbohydrates and 50 % of lipids). Blood was sampled at fasting (0 h) and every hour from 1 to 5 h for triglycerides (TG), total cholesterol, HDL, LDL and glucose. For plasminogen activator inhibitor-1 (PAI-1), plasminogen activator (tPA), tumor necrosis factor-alpha (TNFα), interleukin-6 (IL-6) and interleukin-10 (IL- 10), blood was sampled at 0, 1, 3 and 5 h. Results: TG area under the curve (AUC) was lower in LI and MI than Control (P<0.05). For PAI-1, there was a difference from LI to MI and Control at 1 h (P<0.05). For tPA, there was a difference from LI to Control at 1 h (P<0.05). For FVII the protocols MI and BI there was difference from Con in at 1h. For TNFα, there was a difference from MI to Control at 1 h (P<0.05). IL-10 concentration was different from MI to Control at 1 h and from MI to LI at 1, 3 and 5 h (P<0.05). Fasting IL-6 concentrations were different between all conditions (P<0.05). Conclusion: The consumption of a high-fat meal increases the inflammatory process and deregulates the balance between coagulation and fibrinolysis. Exercise, independent of the intensity, can reduce TG AUC compared to Control. MI can reduce TNFα and increases IL-10, while LI regulates coagulation and fibrinolysis balance, which can be explained by the increase in tPA and increase in PAI-1.
Boknäs, Niklas. « Studies on interfaces between primary and secondary hemostasis ». Doctoral thesis, Linköpings universitet, Avdelningen för mikrobiologi och molekylär medicin, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-132413.
Texte intégralKurdi, Mohamad. « Study of the clearance of proteins of hemostasis ». Paris 7, 2011. http://www.theses.fr/2012PA077071.
Texte intégralThe main objective of this thesis was to study the clearance mechanisms of glycoproteins playing a key role in the hemostatic process, factor X (FX) and the factor VIII (FVIII)/von Willebrand factor (VWF) couple which circulates in plasma in a tight non-covalent complex. In the first part of our work, we have studied the involvement of FX N-glycosylations on its clearance. It had been previously established by the team that these N-glycans were important for the long half-life of FX. We have now extended these data by showing that N-glycosylations also influence organ biodistribution and cellular interactions of the protein. Indeed, as compared to FX, a N-deglycosylated FX variant interacts with different cell types in the liver which is the main target organ for FX biodistribution. N-deglycosylated FX binds to and is degraded by hepatocytes. Conversely, FX binds to Kupffer cells. The binding of FX to Kupffer cells appears to be part of an original mechanism protecting FX from an accelerated clearance. Both VWF and FVIII are glycoproteins whose glycans are capped with sialic acids. In the second part of this thesis, we have studied the role of a receptor, Sialic acid-binding Ig-like lectin 5 (Siglec-5), in the clearance of the FVIII/VWF complex. Siglec-5 is expressed on the surface of macrophages, a cell type that is dominant in the clearance of this complex. Our results showed that FVIII and FW are ligands for Siglec-5. Furthermore, overexpression of Siglec-5 in vivo is associated with decreased endogenous levels of the two glycoproteins. These results suggest that Siglec-5 can play a role in the catabolism of the FVIII/FW complex
Shoffstall, Andrew J. « The Use of Synthetic Platelets to Augment Hemostasis ». Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1363775111.
Texte intégralLivres sur le sujet "Hemostasis"
F, Feldman Bernard, dir. Hemostasis. Philadelphia : W.B. Saunders, 1988.
Trouver le texte intégralBennett, Sterling T., Christopher M. Lehman et George M. Rodgers. Laboratory Hemostasis. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-08924-9.
Texte intégralMarcucci, Carlo Enrique, et Patrick Schoettker, dir. Perioperative Hemostasis. Berlin, Heidelberg : Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-55004-1.
Texte intégralThompson, Catherine, et Robert C. Blaylock, dir. Laboratory Hemostasis. Boston, MA : Springer US, 2007. http://dx.doi.org/10.1007/0-387-36840-x.
Texte intégralSaba, Hussain I., et Harold R. Roberts, dir. Hemostasis and Thrombosis. Oxford, UK : John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118833391.
Texte intégralKini, R. Manjunatha, Kenneth J. Clemetson, Francis S. Markland, Mary Ann McLane et Takashi Morita, dir. Toxins and Hemostasis. Dordrecht : Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-90-481-9295-3.
Texte intégralDeLoughery, Thomas G., dir. Hemostasis and Thrombosis. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-09312-3.
Texte intégralTakada, A., et A. Z. Budzynski, dir. Hemostasis and Circulation. Tokyo : Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-66925-8.
Texte intégralSzumita, Richard P., et Paul M. Szumita, dir. Hemostasis in Dentistry. Cham : Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-71240-6.
Texte intégralMa, Alice D., Harold R. Roberts et Miguel A. Escobar. Hemophilia and Hemostasis. Oxford : John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781118439289.
Texte intégralChapitres de livres sur le sujet "Hemostasis"
Sobanko, Joseph F. « Hemostasis ». Dans Safety in Office-Based Dermatologic Surgery, 63–75. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13347-8_8.
Texte intégralBloch, Michael H., Michael H. Bloch, Mark A. Geyer, David C. S. Roberts, Eileen M. Joyce, Jonathan P. Roiser, John H. Halpern et al. « Hemostasis ». Dans Encyclopedia of Psychopharmacology, 578. Berlin, Heidelberg : Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_802.
Texte intégralOrbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder et al. « Hemostasis ». Dans Encyclopedia of Behavioral Medicine, 957. New York, NY : Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_100792.
Texte intégralSaliba, Zakhia, Ramy C. Charbel et Tarek Smayra. « Hemostasis ». Dans Atlas of Cardiac Catheterization for Congenital Heart Disease, 53–61. Cham : Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-72443-0_6.
Texte intégralMacNeill, Amy L. « Hemostasis ». Dans Clinical Pathology and Laboratory Techniques for Veterinary Technicians, 75–94. Chichester, UK : John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119421351.ch3.
Texte intégralSaliba, Zakhia S., Sami G. Slaba et Elie B. Sawan. « Hemostasis ». Dans Cardiac Catheterization for Congenital Heart Disease, 181–91. Milano : Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5681-7_13.
Texte intégralRaimondi, Anthony J. « Hemostasis ». Dans Pediatric Neurosurgery, 175–96. New York, NY : Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4757-4202-2_9.
Texte intégralSainburg, Robert L., Andrew L. Clark, George E. Billman, Zachary J. Schlader, Toby Mündel, Kevin Milne, Earl G. Noble et al. « Hemostasis ». Dans Encyclopedia of Exercise Medicine in Health and Disease, 405–8. Berlin, Heidelberg : Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_47.
Texte intégralSaliba, Zakhia, et Ramy C. Charbel. « Hemostasis ». Dans Cardiac Catheterization for Congenital Heart Disease, 211–21. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-69856-0_15.
Texte intégralHoballah, Jamal J., et Rakan Nasser Eldine. « Hemostasis ». Dans Vascular Reconstructions, 139–52. New York, NY : Springer New York, 2021. http://dx.doi.org/10.1007/978-1-0716-1089-3_5.
Texte intégralActes de conférences sur le sujet "Hemostasis"
Crum, Lawrence. « Acoustic hemostasis ». Dans 15th international symposium on nonlinear acoustics : Nonlinear acoustics at the turn of the millennium. AIP, 2000. http://dx.doi.org/10.1063/1.1309175.
Texte intégralGrabowski, F. E. « RHEOLOGY AND PRIMARY HEMOSTASIS ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643986.
Texte intégralBrittain, S. B., J. L. Hajjar et S. S. Nidadavolu. « In-vitro hemostasis test platform ». Dans 2011 37th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2011. http://dx.doi.org/10.1109/nebc.2011.5778553.
Texte intégralBerdysh, D. S., S. G. Pavlenko et Evgeny Marchenko. « COMPARATIVE CHARACTERISTICS OF KIDNEY WOUND HEMOSTASIS WHEN USING PECTINS IN AN EXPERIMENT ». Dans NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2022. http://dx.doi.org/10.47501/978-5-6044060-2-1.142-145.
Texte intégralFarag, A. M., S. F. Bottoms, E. F. Mammen, M. Hosni et A. Ali. « EFFECT OF ORAL CONTRACEPTIVES ON HEMOSTASIS ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644283.
Texte intégralZderic, Vesna. « Thin-Profile Transducers for Intraoperative Hemostasis ». Dans 4TH INTERNATIONAL SYMPOSIUM ON THERAPEUTIC ULTRASOUND. AIP, 2005. http://dx.doi.org/10.1063/1.1901650.
Texte intégralSolovchuk, Maxim, et Wen-Hann Sheu. « Computational model for investigating acoustic hemostasis ». Dans The 2013 International Workshop on Computational Science and Engineering. Trieste, Italy : Sissa Medialab, 2014. http://dx.doi.org/10.22323/1.202.0019.
Texte intégralSapkota, A. « Metabolome Analysis for New Possible Hemostasis Biomarkers ». Dans 63rd Annual Meeting of the Society of Thrombosis and Haemostasis Research. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1680273.
Texte intégralSaleh, A. A., A. M. Farag, S. F. Bottoms, E. F. Mammen, M. Hosni et A. Ali. « SELECTED HEMOSTASIS PARAMETERS IN PREGNANCY AND HYPERTENSION ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644284.
Texte intégralBelov, V. P., et E. A. Vinokurova. « Perioperative period in the radical treatment of uterine cancer : features of management ». Dans Наука России : Цели и задачи. НИЦ "LJournal", 2021. http://dx.doi.org/10.18411/sr-10-08-2021-06.
Texte intégralRapports d'organisations sur le sujet "Hemostasis"
Andrew, Marilee, et Lawrence Crum. An Acoustic Hemostasis Device for Advanced Trauma Care. Fort Belvoir, VA : Defense Technical Information Center, octobre 2001. http://dx.doi.org/10.21236/ada398720.
Texte intégralCrum, Lawrence, Marilee Andrew, Shahram Vaezy, Peter Kaczkowski et Steven Kargl. An Acoustic Hemostasis Device for Acute Arterial Bleeding. Fort Belvoir, VA : Defense Technical Information Center, avril 2004. http://dx.doi.org/10.21236/ada422316.
Texte intégralCarlson, Mark A., William H. Velander, Gustavo Larsen, Luis Nunez et Wilson H. Burgess. Technologies for Hemostasis and Stabilization of the Acute Traumatic Wound. Fort Belvoir, VA : Defense Technical Information Center, octobre 2012. http://dx.doi.org/10.21236/ada602118.
Texte intégralCarlson, Mark A., William H. Velander, Gustavo Larsen, Luis Nunez et Wilson H. Burgess. Technologies for Hemostasis and Stabilization of the Acute Traumatic Wound. Fort Belvoir, VA : Defense Technical Information Center, octobre 2013. http://dx.doi.org/10.21236/ada602120.
Texte intégralZhigulina, K. V., et S. S. Spitsina. ASSESSMENT OF THE MAIN INDICATORS OF THE HEMOSTASIS SYSTEM IN PATIENTS WITH RHEUMATOID ARTHRITIS. DOI CODE, 2021. http://dx.doi.org/10.18411/wco-iof-esceo-2021-392.
Texte intégralMoore, II, et Bob M. Development of Hemostatic Agents. Fort Belvoir, VA : Defense Technical Information Center, août 2005. http://dx.doi.org/10.21236/ada454187.
Texte intégralMoore II, Bob M. Development of Hemostatic Agents. Fort Belvoir, VA : Defense Technical Information Center, janvier 2008. http://dx.doi.org/10.21236/ada476090.
Texte intégralLewis, Terry W. Hemostatic Activity of Chitosan in Wound Management. Fort Belvoir, VA : Defense Technical Information Center, mars 1989. http://dx.doi.org/10.21236/ada211370.
Texte intégralSnow, Carl, Cheryl Olson et Ted Melcer. The Navy Medical Technology Watch : Hemostatic Dressing Products for the Battlefield. Fort Belvoir, VA : Defense Technical Information Center, septembre 2006. http://dx.doi.org/10.21236/ada477227.
Texte intégralBode, Arthur P. Evaluation of Dried Storage of Platelets for Transfusion : Physiologic Integrity and Hemostatic Functionality. Fort Belvoir, VA : Defense Technical Information Center, juin 1994. http://dx.doi.org/10.21236/ada280665.
Texte intégral