Bibliographies thématiques / Gene-nutrient interaction

Littérature scientifique sur le sujet « Gene-nutrient interaction »

Auteur : Grafiati
Publié le 11 mars 2023

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Articles de revues sur le sujet "Gene-nutrient interaction"

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López-Carrillo*, Lizbeth, Aubrey V. Herrera, R. Ulises Hernández-Ramirez, Walter Klimecki, A. Jay Gandolfi et Mariano E. Cebrián. « Nutrient-Gene Interaction in Arsenic Metabolism ». ISEE Conference Abstracts 2014, no 1 (20 octobre 2014) : 2026. http://dx.doi.org/10.1289/isee.2014.p3-767.

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Lee, Wai-Nang P., et Vay Liang W. Go. « Nutrient-Gene Interaction : Tracer-Based Metabolomics ». Journal of Nutrition 135, no 12 (1 décembre 2005) : 3027S—3032S. http://dx.doi.org/10.1093/jn/135.12.3027s.

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Go, Vay Liang W., Christine T. H. Nguyen, Diane M. Harris et Wai-Nang Paul Lee. « Nutrient-Gene Interaction : Metabolic Genotype-Phenotype Relationship ». Journal of Nutrition 135, no 12 (1 décembre 2005) : 3016S—3020S. http://dx.doi.org/10.1093/jn/135.12.3016s.

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Mocchegiani, Eugenio, Laura Costarelli, Robertina Giacconi, Catia Cipriano, Elisa Muti, Silvia Tesei et Marco Malavolta. « Nutrient–gene interaction in ageing and successful ageing ». Mechanisms of Ageing and Development 127, no 6 (juin 2006) : 517–25. http://dx.doi.org/10.1016/j.mad.2006.01.010.

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Smith, Daniel L., Crystal H. Maharrey, Christopher R. Carey, Richard A. White et John L. Hartman. « Gene-nutrient interaction markedly influences yeast chronological lifespan ». Experimental Gerontology 86 (décembre 2016) : 113–23. http://dx.doi.org/10.1016/j.exger.2016.04.012.

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Vainio, Harri. « Modification of lung cancer prevention by gene-nutrient interaction ». Scandinavian Journal of Work, Environment & ; Health 26, no 6 (décembre 2000) : 459–60. http://dx.doi.org/10.5271/sjweh.568.

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Luan, J. 'a, P. O. Browne, A. H. Harding, D. J. Halsall, S. O'Rahilly, V. K. K. Chatterjee et N. J. Wareham. « Evidence for Gene-Nutrient Interaction at the PPAR Locus ». Diabetes 50, no 3 (1 mars 2001) : 686–89. http://dx.doi.org/10.2337/diabetes.50.3.686.

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Reece, Richard J., Laila Beynon, Stacey Holden, Amanda D. Hughes, Karine Rébora et Christopher A. Sellick. « Nutrient-regulated gene expression in eukaryotes ». Biochemical Society Symposia 73 (1 janvier 2006) : 85–96. http://dx.doi.org/10.1042/bss0730085.

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The recognition of changes in environmental conditions, and the ability to adapt to these changes, is essential for the viability of cells. There are numerous well characterized systems by which the presence or absence of an individual metabolite may be recognized by a cell. However, the recognition of a metabolite is just one step in a process that often results in changes in the expression of whole sets of genes required to respond to that metabolite. In higher eukaryotes, the signalling pathway between metabolite recognition and transcriptional control can be complex. Recent evidence from the relatively simple eukaryote yeast suggests that complex signalling pathways may be circumvented through the direct interaction between individual metabolites and regulators of RNA polymerase II-mediated transcription. Biochemical and structural analyses are beginning to unravel these elegant genetic control elements.
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Shirazi-Beechey, S. P., G. A. Allison, I. S. Wood et J. Dyer. « NUTRIENT AND SUGAR TRANSPORTER GENE INTERACTION IN THE INTESTINAL EPITHELIA ». Biochemical Society Transactions 25, no 3 (1 août 1997) : 459S. http://dx.doi.org/10.1042/bst025459sd.

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Rubin, Jill, et Lars Berglund. « Apolipoprotein E and diets : a case of gene-nutrient interaction ? » Current Opinion in Lipidology 13, no 1 (février 2002) : 25–32. http://dx.doi.org/10.1097/00041433-200202000-00005.

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Thèses sur le sujet "Gene-nutrient interaction"

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Wilson, Carol Patricia. « The MTHFR C677T polymorphism and riboflavin : a novel gene-nutrient interaction affecting blood pressure ». Thesis, University of Ulster, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.554915.

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Hypertension IS a major risk factor for CVD and unequivocal evidence has demonstrated a continuous and linear relationship between elevated blood pressure (BP) and stroke. Among the many established risk factors for hypertension, a novel gene- nutrient interaction with a potential role in BP has recently emerged. A common polymorphism (677C---)oT) in the gene encoding the folate-metabolising enzyme methylenetetrahydrofolate reductase (MTHFR) produces a variant enzyme with decreased activity, and recent work at this centre in premature CVD patients reported that stabilisation of the variant enzyme by administration of its cofactor riboflavin may lower BP. The aim of this thesis was to further investigate the association between the MTHFR 677C---)o T polymorphism and BP and to evaluate the potential modulating role of riboflavin. The findings of this thesis demonstrated that riboflavin supplementation at the dietary level (1.6mg/dI16weeks) produced a genotype-specific lowering of BP that was clinically significant and that this effect was not confined solely to high-risk CVD patients but may in fact be applicable to hypertensive patients generally with the TT genotype. Preliminary work using 24-hour ambulatory blood pressure monitoring (ABPM) reported a non-significant trend towards higher BP in those with the TT genotype compared to those with the CC and CT genotypes. It also appeared to suggest that MTHFR genotype may have an effect on nocturnal BP characterised by non- dipping status, itself a cardiovascular risk factor independently of 24-hour blood pressure. In conclusion this thesis has confirmed that the MTHFR 677 TT genotype is a risk factor for hypertension and that optimisation of riboflavin status offers a targeted nutritional therapy with clinically relevant effects on BP specifically in this genotype group. Given the frequency of this polymorphism worldwide and the global burden of blood pressure-related disease, these findings could have important public health implications.
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LOTTO, VALENTINA. « Nutrient-gene interactions within one-carbon metabolism and effects on epigenetic regulation through dna methylation in peripheral blood mononuclear cells ». Doctoral thesis, Università degli Studi di Milano-Bicocca, 2010. http://hdl.handle.net/10281/18016.

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Epigenetics is a field of molecular biology that copes with the study of gene function regulation without variations in DNA structure or nucleotide sequences. Among the main epigenetic phenomema in eukaryotic cells there are DNA methylation and post-traslational mechanisms among which the major are histone methylation and acetylation. Epigenetic changes are potentially reversible phenomena that are controlled also by nutritional factors as the methyl-donors involved in the folate cycle. Plasma levels of B vitamins, among which “in primis” plasma folate concentrations, are implicated in epigenetic modulation so that it can be hypothesized that they may affect the modulation of gene expression through epigenetic mechanisms. Epigenetic modifications represent one of the earliest events in the genesis of some complex pathologies, therefore the study of the interaction between epigenetics and nutritional status is of great interest either to define the physiopathological mechanisms of development of some illnesses, and for possible personalized strategies of prevention. The present work has been articulated, at first, on the analysis of gene-nutritional interaction mechanisms within the folate cycle through the study of polymorphisms of enzymes involved in the metabolism of methyl-group donors; the aim was to study their possible role on the modulation of genomic DNA methylation in relationship to different plasma levels of idrosoluble B vitamins. In this regard, the most important functional polymorfisms known on the genes of one-carbon metabolism and their relationship with methylation status of polymorphonuclear cells DNA have been analyzed from a cohort of around 800 subjects within a clinical study, underlining the role of the key folate-related enzymes in the modulation of DNA methylation. Besides the function of genomic DNA methylation, the methylation status at specific sites has been also approached with the specific intent of considering a possible interrelationship between the role of promoter methylation and the co-presence of functional polymorphisms in the same genic site for a gene for which a precise functional effect is well-known. To address this issue the promoter region of coagulation factor VII gene was evaluated for both genetic and epigenetic modifications as a possible model of genetic-epigenetic interaction in the modulation of gene product regulation. The results showed the key importance of genetic-epigenetic interactions, so far unknowm, in modulating gene-expression at promoter gene sites. The role of other vitamins involved in one-carbon metabolism in major chronic diseases, and specifically the emerging role of B6 vitamin, have been also studied. Furthermore, a clinical study is now in progress to evaluate the function of gene-specific methylation in liver tissue where most of the folate cycle functions take place. The aim of this project is the evaluation of both genome-wide and gene-specific methylation status in the liver in comparison to that observed in peripheral blood mononuclear cells DNA to define whether methylation status of peripheral blood DNA may be regarded as a good systemic biomarker for this epigenetic feature of DNA in relation to B vitamins nutritional status in cancer disease. Results from this study may help to define possible functional markers of gene-nutrients interactions with effects on epigenetic modulation for future preventive or therapeutic strategies. With that purpose, a novel high-throughput array-based technique for the detection of gene-specific methylation at promoter sites has been optimized in our laboratory.
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Wu, Kelvin Kwan Hoe. « Gene-nutrient interactions and risk of coronary heart disease ». Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.614117.

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Marley, Andrew Raymond. « The Association Between Citrus Consumption and Skin Cancer : An Analysis of Risk and Nutrient-Gene Interaction ». Diss., 2020. http://hdl.handle.net/1805/24802.

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Indiana University-Purdue University Indianapolis (IUPUI)
Purpose. In the US, melanoma and non-melanoma skin cancer (NMSC) rates have increased substantially in recent decades. While many skin cancer risk factors have been established, the impact of dietary citrus, which is naturally abundant in photocarcinogenic psoralens, remains enigmatic. The purpose of this research was to investigate associations between citrus consumption and risks of melanoma and NMSC, and to conduct a genome-wide study to identify genetic variants that may modify this association. Methods. Participants from the UK Biobank were leveraged for these analyses. Citrus consumption was collected via five rounds of 24-hour recall questionnaires, with complete citrus data available for n=210,126 participants. Ascertainment of melanoma and NMSC cases were identified by international classification of disease codes via linkage with national registries. Logistic regression was used to estimate odds ratios and 95% confidence intervals for the associations between citrus consumption and skin cancer outcomes. Individual citrus products were assessed for independent associations with skin cancer risk, and established skin cancer risk factors were tested for interaction. Joint 2-degree-of-freedom (df) and 1-df tests were used to assess interaction between total citrus consumption and genetic variants. Results. After controlling for covariates, high total citrus consumption was significantly associated with increased melanoma risk, an association primarily driven by orange and orange juice consumption. Skin color was found to be a significant effect modifier for the association between total citrus consumption and melanoma risk, but only before adjusting for multiple comparisons. No significant associations were observed for high total citrus consumption or consumption of any individual citrus products and NMSC risk. Significant associations for half a serving of citrus consumption and NMSC risk were likely due to chance or confounding. Index SNPs on chromosomes 3, 9, and 16 were significant according to the joint 2-df test, and 7 SNPs on chromosome 16 displayed evidence of a citrus-gene interaction. Conclusion. My analyses provide evidence in support of high citrus consumption significantly increasing risk of melanoma, but not NMSC. I also identified SNPs on AFG3L1P that may modify this association. Future research should further explore these associations, particularly for NMSC and to confirm my genetic findings.
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Livres sur le sujet "Gene-nutrient interaction"

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S, Meskin Mark, Bidlack Wayne R, Randolph R. Keith et International Phytochemical Conference (5th : 2004 : California State Polytechnic University, Pomona), dir. Phytochemicals : Nutrient-gene interactions. Boca Raton : CRC/Taylor & Francis, 2006.

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Naima, Moustaid-Moussa, et Berdanier Carolyn D, dir. Nutrient-gene interactions in health and disease. Boca Raton : CRC Press, 2001.

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Young-Joon, Surh, dir. Dietary modulation of cell signaling pathways. Boca Raton : Taylor & Francis, 2008.

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(Editor), Mark S. Meskin, Wayne R. Bidlack (Editor) et R. Keith Randolph (Editor), dir. Phytochemicals : Nutrient-Gene Interactions. CRC, 2006.

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Bidlack, Wayne R., R. Keith Randolph et Mark S. Meskin. Phytochemicals : Nutrient-Gene Interactions. Taylor & Francis Group, 2006.

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Meskin, Mark S. Phytochemicals : Nutrient-Gene Interactions. Taylor & Francis Group, 2010.

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Bidlack, Wayne R., R. Keith Randolph et Mark S. Meskin. Phytochemicals : Nutrient-Gene Interactions. Taylor & Francis Group, 2006.

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Choi, Sang-Woon. Nutrient-Gene Interactions in Cancer. Taylor & Francis Group, 2010.

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Choi, Sang-Woon, et Simonetta Friso, dir. Nutrient-Gene Interactions in Cancer. CRC Press, 2006. http://dx.doi.org/10.1201/9780849332296.

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Choi, Sang-Woon, et Simonetta Friso, dir. Nutrient-Gene Interactions in Cancer. CRC Press, 2006. http://dx.doi.org/10.1201/9781420004847.

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Chapitres de livres sur le sujet "Gene-nutrient interaction"

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Minihane, Anne Marie. « Nutrient-Gene Interactions ». Dans Nutrition Research Methodologies, 225–34. Chichester, UK : John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119180425.ch15.

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Lin, Dongxin, Hui Li, Wen Tan, Xiaoping Miao et Li Wang. « Genetic Polymorphisms in Folate- Metabolizing Enzymes and Risk of Gastroesophageal Cancers : A Potential Nutrient-Gene Interaction in Cancer Development ». Dans Nutrigenomics - Opportunities in Asia, 140–45. Basel : KARGER, 2007. http://dx.doi.org/10.1159/000107090.

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Muntoni, Sergio, et Sandro Muntoni. « Gene-Nutrient Interactions in Type 1 Diabetes ». Dans Nutrigenetics and Nutrigenomics, 188–209. Basel : KARGER, 2004. http://dx.doi.org/10.1159/000081259.

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Cahill, Leah E., et Eric B. Rimm. « Diet–Gene Interactions : Haptoglobin Genotype and Nutrient Status ». Dans Preventive Nutrition, 115–29. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-22431-2_7.

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Ordovas, Jose M., Dolores Corella et James Kaput. « Nutrient-Gene Interactions in Lipoprotein Metabolism – An Overview ». Dans Nutrigenomics - Opportunities in Asia, 102–9. Basel : KARGER, 2007. http://dx.doi.org/10.1159/000107079.

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Desvergne, Béatrice, et Walter Wahli. « PPAR : a Key Nuclear Factor in Nutrient / Gene Interactions ? » Dans Inducible Gene Expression, Volume 1, 142–76. Boston, MA : Birkhäuser Boston, 1995. http://dx.doi.org/10.1007/978-1-4684-6840-3_5.

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Maugeri, Andrea, Martina Barchitta et Antonella Agodi. « Gene–nutrient interaction ». Dans Molecular Nutrition : Mother and Infant, 299–315. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-813862-5.00012-8.

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« Gene–Nutrient Interaction ». Dans Primary Care Nutrition, 297–308. Boca Raton : Taylor & Francis, 2017. : CRC Press, 2017. http://dx.doi.org/10.1201/9781315152165-17.

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Choi, Sang-Woon, et Simonetta Frisco. « Interaction between Folate and Methylenetetrahydrofolate Reductase Gene in Cancer ». Dans Nutrient-Gene Interactions in Cancer, 57–74. CRC Press, 2006. http://dx.doi.org/10.1201/9781420004847.ch4.

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Choi, Sang-Woon, et Simonetta Frisco. « Interaction between Folate and Methylenetetrahydrofolate Reductase Gene in Cancer ». Dans Nutrient-Gene Interactions in Cancer, 57–74. CRC Press, 2006. http://dx.doi.org/10.1201/9780849332296.ch4.

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Rapports d'organisations sur le sujet "Gene-nutrient interaction"

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Raghothama, Kashchandra G., Avner Silber et Avraham Levy. Biotechnology approaches to enhance phosphorus acquisition of tomato plants. United States Department of Agriculture, janvier 2006. http://dx.doi.org/10.32747/2006.7586546.bard.

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Abstract: Phosphorus is one of the least available macronutrient in the soil. The high affinity phosphate transporters are known to be associated with phosphate acquisition under natural conditions. Due to unique interactions of phosphate with soil particles, up to 80% of the applied phosphates may be fixed forcing the farmers to apply 4 to 5 times the fertilizers necessary for crop production. Efficient uptake and utilization of this essential nutrient is essential for sustainability and profitability of agriculture. Many predictions point to utilization/exhaustion of high quality phosphate rocks within this century. This calls for efforts to improve the ability of plants to acquire and utilize limiting sources of phosphate in the rhizosphere. Two important molecular and biochemical components associated with phosphate efficiency are phosphate transporters and phosphatases. This research project is aimed at defining molecular determinants of phosphate acquisition and utilization in addition to generating phosphate uptake efficient plants. The main objectives of the project were; Creation and analysis of transgenic tomato plants over-expressing phosphatases and transporters Characterization of the recently identified members (LePT3 and LePT4) of the Pi transporter family Generate molecular tools to study genetic responses of plants to Pi deficiency During the project period we have successfully identified and characterized a novel phosphate transporter associated with mycorrhizal symbiosis. The expression of this transporter increases with mycorrhizal symbiosis. A thorough characterization of mutant tomato lacking the expression of this gene revealed the biological significance of LePT3 and another novel gene LePT4. In addition we have isolated and characterized several phosphate starvation induced genes from tomato using a combination of differential and subtractive mRNA hybridization techniques. One of the genes, LePS2 belongs to the family of phospho-protein phosphatase. The functionality of the recombinant protein was determined using synthetic phosphor-peptides. Over expression of this gene in tomato resulted in significant changes in growth, delay in flowering and senescence. It is anticipated that phospho-protein phosphatase may have regulatory role in phosphate deficiency responses of plants. In addition a novel phosphate starvation induced glycerol 3-phosphate permease gene family was also characterized. Two doctoral research students are continuing the characterization and functional analysis of these genes. Over expression of high affinity phosphate transporters in tobacco showed increased phosphate content under hydroponic conditions. There is growing evidence suggesting that high affinity phosphate transporters are crucial for phosphate acquisition even under phosphate sufficiency conditions. This project has helped train several postdoctoral fellows and graduate students. Further analysis of transgenic plants expressing phosphatases and transporters will not only reveal the biological function of the targeted genes but also result in phosphate uptake and utilization efficient plants.
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