Littérature scientifique sur le sujet « Fibrosis »
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Articles de revues sur le sujet "Fibrosis"
&NA;. « Cystic fibrosis and fibrosing colonopathy ». Advances in Anatomic Pathology 3, no 2 (mars 1996) : 112. http://dx.doi.org/10.1097/00125480-199603000-00015.
Texte intégralSmyth, R. L. « Fibrosing colonopathy in cystic fibrosis. » Archives of Disease in Childhood 74, no 5 (1 mai 1996) : 464–68. http://dx.doi.org/10.1136/adc.74.5.464.
Texte intégralHernandez-Gonzalez, Fernanda, Rosa Faner, Mauricio Rojas, Alvar Agustí, Manuel Serrano et Jacobo Sellarés. « Cellular Senescence in Lung Fibrosis ». International Journal of Molecular Sciences 22, no 13 (29 juin 2021) : 7012. http://dx.doi.org/10.3390/ijms22137012.
Texte intégralAlbera, Carlo, Giulia Verri, Federico Sciarrone, Elena Sitia, Mauro Mangiapia et Paolo Solidoro. « Progressive Fibrosing Interstitial Lung Diseases : A Current Perspective ». Biomedicines 9, no 9 (16 septembre 2021) : 1237. http://dx.doi.org/10.3390/biomedicines9091237.
Texte intégralWaters, Brenda L. « Cystic Fibrosis with Fibrosing Colonopathy in the Absence of Pancreatic Enzymes ». Pediatric and Developmental Pathology 1, no 1 (janvier 1998) : 74–78. http://dx.doi.org/10.1007/s100249900009.
Texte intégralGibson, Sarah E., Carol F. Farver et Richard A. Prayson. « Multiorgan Involvement in Nephrogenic Fibrosing Dermopathy : An Autopsy Case and Review of the Literature ». Archives of Pathology & ; Laboratory Medicine 130, no 2 (1 février 2006) : 209–12. http://dx.doi.org/10.5858/2006-130-209-miinfd.
Texte intégralCarrino, David A., Sam Mesiano, Nichole M. Barker, William W. Hurd et Arnold I. Caplan. « Proteoglycans of uterine fibroids and keloid scars : similarity in their proteoglycan composition ». Biochemical Journal 443, no 2 (27 mars 2012) : 361–68. http://dx.doi.org/10.1042/bj20111996.
Texte intégralCottin, Vincent, Lutz Wollin, Aryeh Fischer, Manuel Quaresma, Susanne Stowasser et Sergio Harari. « Fibrosing interstitial lung diseases : knowns and unknowns ». European Respiratory Review 28, no 151 (27 février 2019) : 180100. http://dx.doi.org/10.1183/16000617.0100-2018.
Texte intégralYu, Guoying. « Fibrosis : A New Open-Access Journal to Share Your Research ». Fibrosis 1, no 1 (2023) : 1–2. http://dx.doi.org/10.35534/fibrosis.2023.10001.
Texte intégralTsomidis, Ioannis, George Notas, Argyro Voumvouraki, Dimitrios Samonakis, Mairi Koulentaki et Elias Kouroumalis. « Hepatic Lysosomal Enzyme Activity in Primary Biliary Cholangitis ». Fibrosis 1, no 1 (2023) : 1–12. http://dx.doi.org/10.35534/fibrosis.2023.10005.
Texte intégralThèses sur le sujet "Fibrosis"
Enes, Giovana da Silva Tavares 1982. « Fibrose cística = estreitando laços maternos = Cystic fibrosis : strengthening maternal ties ». [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308361.
Texte intégralDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: A Fibrose Cística é uma doença autossômica recessiva, sistêmica, hereditária, crônica e progressiva e pode levar à morte. São características da doença as secreções mucosas espessas e viscosas que obstrui os ductos das glândulas exócrinas e contribuem para o aparecimento de doença pulmonar obstrutiva crônica, insuficiência pancreática com má digestão e má absorção e conseqüente desnutrição secundária, além de níveis elevados de eletrólitos no suor. Por ser uma doença crônica, ela exige cuidados sistemáticos pela vida toda, e na maioria dos casos quem exerce a função de cuidadora é a mãe. Além de viver uma nova experiência de ser mãe, ela terá que conviver com a frustração dele ser doente.Com este estudo foi possível compreender a relação que mãe e filho doente crônico constroem desde o momento do diagnóstico e conhecimento do tratamento, permeados por sentimentos como culpa e solidão. Assim, essas mães renunciam suas próprias vidas em função do cuidado do filho. Cuidados esse compartilhado com uma equipe de saúde multiprofissional ainda deficitária. Apesar de ter sido avaliado por elas como positivo, as sugestões por melhorias também surgiram: como uma melhor articulação entre os serviços de saúde nos diversos níveis, uma maior divulgação da doença e o aumento do número de dias de atendimento. Outro aspecto importante encontrado foi sobre importância do papel do psicólogo não só na atuação com o paciente e a família durante todo o tratamento; mas também na necessidade de oferecer um espaço para que os profissionais de saúde despreparados pudessem compartilhar suas angústias e frustrações o que reflete diretamente na assistência prestada
Abstract: The Cystic Fibrosis is a disease systemic, hereditary, chronic and progressive and it can lead to the death. There are characteristic of the disease the thick and viscous mucous secretions what it obstructs the ducts of the exocrine glands and contribute to the appearance of chronic obstructive pulmonary disease, pancreatic insufficiency with bad digestion and bad absorption and consequent secondary malnutrition, besides elevated levels of electrolytes in the sweat. Because of being a chronic disease, she demands systematic cares for the life completely, and in most of the cases who plays the function of care is the mother. Besides surviving a new experience of being a mother, she will have to coexist in spite of the fact that his frustration to be doente.Com this study there were possible understood the relation what mother and chronic sick son build from the moment of the diagnosis and knowledge of the treatment, permeated by feelings as fault and solitude. So, these mothers renounce his lives themselves in function of the care of the son. Taken care this shared one with a team of still deficient multiprofessional health. In spite of having been valued by them like positive, the suggestions for improvements also appeared: like a better articulation between the health services in several levels, a bigger spread of the disease and the increase of the number of service days. Another considered important aspect was on importance of the paper of the psychologist not alone in the acting with the patient and the family during the whole treatment; but also in the necessity of offering a space so that the unprepared health professionals could share his anguishes and frustrations what thinks straightly about the given presence
Mestrado
Saude da Criança e do Adolescente
Mestre em Ciências
Yi, Hao. « The Effect of Metformin on Inflammatory and Fibrotic Responses in Renal Fibrosis ». Thesis, The University of Sydney, 2019. https://hdl.handle.net/2123/21855.
Texte intégralCorreia, Cyntia Arivabeni de Araujo. « Estudo dos genes TNF alfa, ADIPOQ e STATH entre portadores de fibrose cistica ». [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308583.
Texte intégralTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: A Fibrose Cística (FC) possui uma grande variabilidade de expressão fenotípica, o que significa que crianças com o mesmo genótipo podem diferir quanto à sua apresentação. A proteína defeituosa formada é chamada CFTR (proteína reguladora da conductância iônica), causa transporte anormal de sódio e cloro através da membrana apical das células epiteliais das vias aéreas, pâncreas, intestino e aparelho reprodutor. Essa proteína é codificada por um único gene que recebe o mesmo nome da proteína, CFTR, e localiza-se no braço longo do cromossomo 7, região 7q3.1. Gêmeos monozigóticos apresentam maior concordância em relação à gravidade da doença pulmonar que os dizigóticos, sugerindo que a FC seja modulada por fatores genéticos secundários - genes modificadores - além do gene CFTR. A característica mais importante na FC é a sobrevida que é influenciada pela doença pulmonar. Portanto, genes que estejam envolvidos na imunidade, inflamação, reparação do epitélio e produção de muco são candidatos a genes modificadores da doença. Os objetivos foram: 1) determinar a prevalência dos polimorfismos -308G/A e -238G/A do gene TNF a entre portadores de FC e verificar existência de associação entre esses polimorfismos e a gravidade do quadro pulmonar, 2) identificar alterações de sequencia nos exons e junções exon/ intron dos genes ADIPOQ e STATH e verificar existência de associação entre possíveis variações nesses genes e a gravidade da FC. Foi realizada PCR seguida por digestão enzimática para o polimorfismo -308G/A do gene TNF a, reação em cadeia da polimerase ARMS para o polimorfismo -238G/A do gene TNF a, e para os genes ADIPOQ e STATH foi feita a triagem de mutações através de cromatografia líquida de alta resolução por desnaturação - DHPLC com posterior sequenciamento da região onde foi encontrada alteração. Foram analisados 49 pacientes com FC em seguimento no Ambulatório de Mucoviscidose do HC/UNICAMP, homozigotos para a mutação F508 ou heterozigotos compostos para mutações de classe I ou II ou homozigotos para mutações de classe II, que são alterações que não levam à formação de proteína funcional. Além disso, foram selecionados indivíduos que apresentem alteração de eletrólitos no suor. Para o polimorfismo -308G/A do gene TNFa os genótipos GG, AA e GA foram encontrados com as seguintes frequencias: 14,28, 67,35 e 18,36% respectivamente. Estes dados se opõem ao relatado na literatura. Tal diferença deve ocorrer pelas características populacionais da população brasileira. Para o polimorfismo -238G/A do gene TNFa, os genótipos GG e AG tiveram as seguintes frequencias: 79,59 e 20,41% respectivamente. O genótipo AA não foi encontrado na amostra analisada. A alta frequencia do genótipo GG comparado com o AA, concorda com a literatura. Não foi encontrada alteração na sequencia dos genes STATH e ADIPOQ. Não foi possível estabelecer uma associação entre a gravidade da FC e os genes TNFa, STATH e ADIPOQ, nas regiões analisadas.
Abstract: Cystic Fibrosis (CF) has a great variety expression, which means that the seriousness of the disease can vary a lot among people who have it. The defective protein, called CFTR (Cystic Fibrosis Transmenbrane Regulator), causes abnormal transportation of chloride and sodium through the apical membrane of the epithelial cells of the airway, liver, intestine and masculine reproductive tract. This protein is encoded by a single gene which has the same name, CFTR, and is located within the long arm of chromosome 7, region 7q3.1. CF is a disease which expressivity is much variable, with different degrees of damage and the age when the symptoms begins is also much variable, even within individuals of the same family, like twins. Because of it, it is been said that others genetic factors besides CFTR, can be modulating the clinical presentation. As the pulmonary state is the great responsible for the mortality of the disease genes that are involved in host defense, inflammation, epithelial repair, mucin production, and airway reponsiveness are of great interest. Base on this the objectives of this work were: determine the prevalence of the polymorphisms -308G/A e -238G/A from the TNF a gene and verify if there is an association between these polymorphisms pulmonary disease severity, and identify alterations on ADIPOQ and STATH genes and verify if there is an association between these polymorphisms and CF severity. PCR followed by restriction enzyme digestion was performed to detect the polymorphism -308G/A from the TNF a gene, ARMS PCR to the polymorphism -238G/A from the TNF a gene the DHPLC method associated to the sequencing to analyze ADIPOQ and STATH genes, were used. We performed analyses of 49 cystic fibrosis patients that are followed in a Cystic Fibrosis center from HC/UNICAMP, that are \F508 homozygous or compound heterozygous to mutations from class I or II, or that are homozygous to class II mutations, which are alterations that do not produce functional protein. Besides this, were selected individuals that have sweat test altered. To the polymorphism 308G/A from TNFa gene the genotypes GG, AA e AG were in the following frequencies: 14,28, 67,35 e 18,36%. This data is contradictory to the literature and may occur because of the racial admixture of the Brazilian population. To the polymorphism -238G/A from TNFa gene, the genotypes GG AG were in the following frequencies 79,59 e 20,41%. The genotype AA was not found in the analyzed group. The high frequency of the genotype GG is in agreement of the data. It was not possible to find any alteration on ADIPOQ and STATH genes. And also it was not possible to make any correlation between the severity of the CF disease and the genes TNFa, STATH and ADIPOQ between the analyzed regions.
Doutorado
Ciencias Biomedicas
Doutor em Ciências Médicas
Chadwick, Helen Kay. « Cognitive function in cystic fibrosis and cystic fibrosis related diabetes (CFRD) ». Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/16912/.
Texte intégralKarlas, Thomas, Joachim Berger, Nikita Garnov, Franziska Lindner, Harald Busse, Nicolas Linder, Alexander Schaudinn et al. « Estimating steatosis and fibrosis ». Universitätsbibliothek Leipzig, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-169692.
Texte intégralKatre, Ashwini A. « Ozone and lung fibrosis ». Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2009. https://www.mhsl.uab.edu/dt/2009m/katre.pdf.
Texte intégralKahre, Tiina. « Cystic fibrosis in Estonia / ». Online version, 2004. http://dspace.utlib.ee/dspace/bitstream/10062/577/5/KahrePhD.pdf.
Texte intégralDwyer, Tiffany Jane. « Exercise in cystic fibrosis ». Thesis, The University of Sydney, 2010. http://hdl.handle.net/2123/6349.
Texte intégralDwyer, Tiffany Jane. « Exercise in cystic fibrosis ». Discipline of Physiotherapy, Faculty of Health Sciences, University of Sydney, 2010. http://hdl.handle.net/2123/6349.
Texte intégralExercise and physical activity have many benefits for adults with cystic fibrosis (CF), including the potential to aid mucus clearance, improve lung function, exercise capacity and quality of life. Despite the recommendations from consensus documents for CF adults to engage in regular physical activity, exercise participation amongst this population is often very low. No in-depth study has been undertaken to explore the determinants of exercise participation for this group and no studies have examined the benefits of habitual physical activity on the health status and quality of life of CF adults. As well, the current methods to quantify physical activity are problematic. The series of studies, involving CF adults, in this thesis was therefore undertaken in order to examine the physiological rationale for the use of exercise as an airway clearance technique, to understand the decision making process to engage in exercise, to determine if health status and quality of life were affected by exercise participation, and to establish the accuracy of a recently-developed objective measure of physical activity. The study in Chapter 2 provided some physiological rationale for choosing treadmill exercise to aid airway clearance in CF. The main findings were that a single bout of moderate intensity exercise increased the subjective ease of expectoration, most likely due to the increased ventilation with exercise, and that sputum viscoelasticity was favourably decreased immediately following treadmill exercise compared to cycle exercise and control. The studies in Chapters 3 and 4 identified the main beliefs regarding exercise for CF adults and highlighted that the main predictors of exercise intention and participation for this group were aspects of perceived and actual control to exercise, namely self-efficacy or confidence to exercise, feeling healthy, receiving encouragement to exercise, and rating exercise as a high priority treatment. Positive ratings of these aspects of control either increased exercise participation directly, indirectly by increasing intention, or strengthened the conversion of exercise intention to participation. Strategies aimed at targeting these aspects of control are therefore likely to be effective in increasing exercise participation for CF adults. The study in Chapter 5 demonstrated that CF adults, who reportedly performed at least 90 minutes of moderate to strenuous exercise per week, had significantly higher quality of life and fewer days in hospital over the following year than their peers, who exercised less. The difference in hospitalisation between the CF adults, who reportedly exercised more than 90 minutes per week and those who did not, was independent of baseline lung function, and the females who reportedly performed less than 90 minutes of exercise per week had three times as many days in hospital than their high-activity peers. The study in Chapter 6 showed that the SenseWear Pro3 Armband activity monitor provided a reasonable estimate of energy expenditure and step count. Also, its accuracy was not affected by diagnosis with CF, despite the potential for the high salt content in the sweat to interfere with the device’s physiological sensors placed on the skin. Overall, this series of studies adds to the growing evidence of the physical and psychological benefits from exercise participation for CF adults, as well as providing some empirical evidence upon which to base strategies to improve exercise participation for this group and support for an objective measure of physical activity.
Utley, Courtney, et Kristen L. McHenry. « Advances in Cystic Fibrosis ». Digital Commons @ East Tennessee State University, 2016. https://dc.etsu.edu/etsu-works/2546.
Texte intégralLivres sur le sujet "Fibrosis"
E, Hodson Margaret, Geddes Duncan M et Bush, Andrew, 1954 Apr. 24-, dir. Cystic fibrosis. 3e éd. London : Hodder Arnold, 2007.
Trouver le texte intégralDennis, Shale, dir. Cystic fibrosis. London : BMJ Publishing Group, 1996.
Trouver le texte intégralRittié, Laure, dir. Fibrosis. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8.
Texte intégralDavid, Evered, Whelan Julie, Ciba Foundation et Symposium on Fibrosis (1984 : Ciba Foundation), dir. Fibrosis. London : Pitman, 1985.
Trouver le texte intégralSymposium on Fibrosis (1984 : London), dir. Fibrosis. London : Pitman, 1985.
Trouver le texte intégralSymposium, Ciba Foundation. Fibrosis. London : Pitman, 1985.
Trouver le texte intégral1949-, Phan Sem Hin, et Thrall Roger S. 1947-, dir. Pulmonary fibrosis. New York : Marcel Dekker, 1995.
Trouver le texte intégralVarga, John, David A. Brenner et Sem H. Phan, dir. Fibrosis Research. Totowa, NJ : Humana Press, 2005. http://dx.doi.org/10.1385/1592599400.
Texte intégralSilverstein, Alvin. Cystic fibrosis. New York : F. Watts, 1994.
Trouver le texte intégralDavis, Stephanie Duggins, Margaret Rosenfeld et James Chmiel, dir. Cystic Fibrosis. Cham : Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42382-7.
Texte intégralChapitres de livres sur le sujet "Fibrosis"
Bährle-Rapp, Marina. « Fibrose, auch : Fibrosis ». Dans Springer Lexikon Kosmetik und Körperpflege, 206. Berlin, Heidelberg : Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_3985.
Texte intégralRosenbloom, Joel, Edward Macarak, Sonsoles Piera-Velazquez et Sergio A. Jimenez. « Human Fibrotic Diseases : Current Challenges in Fibrosis Research ». Dans Fibrosis, 1–23. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_1.
Texte intégralLi, Ian M. H., Amy L. Horwell, Grace Chu, Benoit de Crombrugghe et George Bou-Gharios. « Characterization of Mesenchymal-Fibroblast Cells Using the Col1a2 Promoter/Enhancer ». Dans Fibrosis, 139–61. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_10.
Texte intégralWeiskirchen, Sabine, Carmen G. Tag, Sibille Sauer-Lehnen, Frank Tacke et Ralf Weiskirchen. « Isolation and Culture of Primary Murine Hepatic Stellate Cells ». Dans Fibrosis, 165–91. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_11.
Texte intégralLiew, Lawrence J., Huan Ting Ong et Rodney J. Dilley. « Isolation and Culture of Adipose-Derived Stromal Cells from Subcutaneous Fat ». Dans Fibrosis, 193–203. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_12.
Texte intégralLeavitt, Tripp, Michael S. Hu et Michael T. Longaker. « Isolation of Live Fibroblasts by Fluorescence-Activated Cell Sorting ». Dans Fibrosis, 205–12. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_13.
Texte intégralPuebla-Osorio, Nahum, Seri N. E. Sarchio, Stephen E. Ullrich et Scott N. Byrne. « Detection of Infiltrating Mast Cells Using a Modified Toluidine Blue Staining ». Dans Fibrosis, 213–22. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_14.
Texte intégralEckes, Beate, Fang Wang, Laure Rittié, Gabriele Scherr et Paola Zigrino. « Cell-Populated Collagen Lattice Models ». Dans Fibrosis, 223–33. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_15.
Texte intégralXie, Tianfa, Jamar Hawkins et Yubing Sun. « Traction Force Measurement Using Deformable Microposts ». Dans Fibrosis, 235–44. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_16.
Texte intégralWorthen, Christal A., Laure Rittié et Gary J. Fisher. « Mechanical Deformation of Cultured Cells with Hydrogels ». Dans Fibrosis, 245–51. New York, NY : Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8_17.
Texte intégralActes de conférences sur le sujet "Fibrosis"
Holmes, Hal, Eli Vlaisavljevich, Ee Lim Tan, Keat G. Ong et Rupak M. Rajachar. « Magnetoelastic Materials as Novel Bioactive Coatings to Improve Integration of Percutaneous Implants ». Dans ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53308.
Texte intégralTorres Rojas, Aimee M., et Sylvie Lorente. « Vascular Model of Liver Fibrosis ». Dans ASME 2023 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2023. http://dx.doi.org/10.1115/imece2023-112123.
Texte intégralSewell-Loftin, M. K., et W. David Merryman. « The Role of SRC in Strain- and Ligand- Dependent Phenotypic Modulation of Mouse Embryonic Fibroblasts ». Dans ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53604.
Texte intégralŠkrbic, Dusan, Mirna Djuric, Jelena Papovic et Branislav Tusek. « COVID-19 vaccine and morbidity in the Adult Cystic Fibrosis Centre in Institute for Pulmonary Diseases of Vojvodina Sremska Kamenica, Serbia ». Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.47.
Texte intégralPetrova, Guergana, Mila Baycheva, Dimitrinka Miteva, Vera Papochieva, Margarita Nikolova, Miglena Georgieva, Nadezhda Yaneva et Savov Alexey. « Late diagnosed homozygous delF508 patients - is it really rare ? » Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.2.
Texte intégralRemus, Natascha, Gaetan Leignadier, Elisa Thomas, Celine Delestrain, Michael Shum, Bernard Maitre, Ralph Epaud et Benoit Douvry. « The A-Step - a new incremental exercise test defying space and infection control measures ». Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.17.
Texte intégralDuplacie, Nele, Trudy Havermans, Janne Houben, Marianne Schulte, Linda Boulanger, Laura Moyens, Cindy Ruelens et Lieven Dupont. « Side-effects and ETI-treatment : a multidisciplinary challenge ». Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.37.
Texte intégralDaniels, Tanne, Kristel Van Calsteren et Lieven Dupont. « Maternal and obstetric outcomes in women with cystic fibrosis : a retrospective case series of patients in UZ Leuven ». Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.31.
Texte intégralSkogeland, Ulrika, Anna Hedborg, Cecilia Rodriguez, Adrienn Bánki et Tove Godskesen. « Adherence to medical regimens after lung transplantation among adults with Cystic Fibrosis increased during COVID-19 pandemic ». Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.4.
Texte intégralAsir, Nadine, Noor Al-Sulaiti, Abdusamea Shabani et Atqah Abdul Wahab. « First case of pott's disease in a cystic fibrosis adolescent with a homozygous CFTR mutation c.3700 A > ; G (p. Ile1234Val) ». Dans Adult Cystic Fibrosis Conference abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.acf-2023.29.
Texte intégralRapports d'organisations sur le sujet "Fibrosis"
Liu, Xiaopei, Dan Liu et Cong’e Tan. Gut microbiome-based machine learning for diagnostic prediction of liver fibrosis and cirrhosis : a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, mai 2022. http://dx.doi.org/10.37766/inplasy2022.5.0133.
Texte intégralHadsall, Katie. Genetic aspects of Pulmonary Fibrosis. Ames (Iowa) : Iowa State University, janvier 2020. http://dx.doi.org/10.31274/cc-20240624-784.
Texte intégralHua, Zi Bo, et Lv Yuan Chen. Human UCB MSC versus placebo for effect on kidney fibrosis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, octobre 2022. http://dx.doi.org/10.37766/inplasy2022.10.0104.
Texte intégralAgarwal, Sandeep K. Cadherin-11 Regulation of Fibrosis through Modulation of Epithelial-to-Mesenchymal Transition : Implications for Pulmonary Fibrosis in Scleroderma. Fort Belvoir, VA : Defense Technical Information Center, octobre 2013. http://dx.doi.org/10.21236/ada591380.
Texte intégralAgarwal, Sandeep K. Cadherin-11 Regulation of Fibrosis through Modulation of Epithelial-to-Mesenchymal Transition : Implications for Pulmonary Fibrosis in Scleroderma. Fort Belvoir, VA : Defense Technical Information Center, octobre 2014. http://dx.doi.org/10.21236/ada618226.
Texte intégralArtlett, Carol M. The Modulation of Fibrosis in Scleroderma by 3-Deoxyglucosone. Fort Belvoir, VA : Defense Technical Information Center, juin 2008. http://dx.doi.org/10.21236/ada485008.
Texte intégralArtlett, Carol M. The Modulation of Fibrosis in Scleroderma by 3-Deoxyglucosone. Fort Belvoir, VA : Defense Technical Information Center, juin 2010. http://dx.doi.org/10.21236/ada541210.
Texte intégralArtlett, Carol M. The Modulation of Fibrosis in Scleroderma by 3-deoxyglucosone. Fort Belvoir, VA : Defense Technical Information Center, juin 2009. http://dx.doi.org/10.21236/ada510087.
Texte intégralGarber, Alan, et Joseph Fenerty. Costs and Benefits of Prenatal Screening For Cystic Fibrosis. Cambridge, MA : National Bureau of Economic Research, octobre 1988. http://dx.doi.org/10.3386/w2749.
Texte intégralArtlett, Carol. The Modulation of Fibrosis In Scleroderma by 3-Deoxyglucosone. Fort Belvoir, VA : Defense Technical Information Center, juin 2011. http://dx.doi.org/10.21236/ada561077.
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